Relevant bibliographies by topics / V-Myb

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'V-Myb'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "V-Myb"

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Symonds, G., K. H. Klempnauer, M. Snyder, G. Moscovici, C. Moscovici, and J. M. Bishop. "Coordinate regulation of myelomonocytic phenotype by v-myb and v-myc." Molecular and Cellular Biology 6, no. 5 (1986): 1796–802. http://dx.doi.org/10.1128/mcb.6.5.1796.

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Both avian myeloblastosis virus (by the action of v-myb) and avian myelocytomatosis virus MC29 (by the action of v-myc) transform cells of the myelomonocytic lineage. Whereas avian myeloblastosis virus elicits a relatively immature phenotype, cells transformed by MC29 resemble mature macrophages. When cells previously transformed by v-myb were superinfected with MC29, their phenotype was rapidly altered to that of a more mature cell. These superinfected cells expressed both v-myb (at a level similar to that found before superinfection) and v-myc. It therefore appears that the expression of v-m
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Symonds, G., K. H. Klempnauer, M. Snyder, G. Moscovici, C. Moscovici, and J. M. Bishop. "Coordinate regulation of myelomonocytic phenotype by v-myb and v-myc." Molecular and Cellular Biology 6, no. 5 (1986): 1796–802. http://dx.doi.org/10.1128/mcb.6.5.1796-1802.1986.

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Both avian myeloblastosis virus (by the action of v-myb) and avian myelocytomatosis virus MC29 (by the action of v-myc) transform cells of the myelomonocytic lineage. Whereas avian myeloblastosis virus elicits a relatively immature phenotype, cells transformed by MC29 resemble mature macrophages. When cells previously transformed by v-myb were superinfected with MC29, their phenotype was rapidly altered to that of a more mature cell. These superinfected cells expressed both v-myb (at a level similar to that found before superinfection) and v-myc. It therefore appears that the expression of v-m
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Lipsick, Joseph S., and Duen-Mei Wang. "Transformation by v-Myb." Oncogene 18, no. 19 (1999): 3047–55. http://dx.doi.org/10.1038/sj.onc.1202745.

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Ness, Scott A., Hartmut Beug, and Thomas Graf. "v-myb dominance over v-myc in doubly transformed chick myelomonocytic cells." Cell 51, no. 1 (1987): 41–50. http://dx.doi.org/10.1016/0092-8674(87)90008-0.

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Guo, K., C. Anjard, A. Harwood, H. J. Kim, P. C. Newell, and J. D. Gross. "A myb-related protein required for culmination in Dictyostelium." Development 126, no. 12 (1999): 2813–22. http://dx.doi.org/10.1242/dev.126.12.2813.

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The avian retroviral v-myb gene and its cellular homologues throughout the animal and plant kingdoms contain a conserved DNA binding domain. We have isolated an insertional mutant of Dictyostelium unable to switch from slug migration to fruiting body formation i.e. unable to culminate. The gene that is disrupted, mybC, codes for a protein with a myb-like domain that is recognized by an antibody against the v-myb repeat domain. During development of myb+ cells, mybC is expressed only in prestalk cells. When developed together with wild-type cells mybC- cells are able to form both spores and sta
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Mink, S., U. Kerber, and K. H. Klempnauer. "Interaction of C/EBPbeta and v-Myb is required for synergistic activation of the mim-1 gene." Molecular and Cellular Biology 16, no. 4 (1996): 1316–25. http://dx.doi.org/10.1128/mcb.16.4.1316.

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The retroviral oncogene v-myb encodes a transcription factor (v-Myb) which activates the myelomonocyte-specific mim-1 gene, a natural myb target gene, by cooperating with members of the C/EBP transcription factor family. The finding that v-Myb, together with C/EBP, is sufficient to activate the mim-1 gene in heterologous cell types has implicated Myb and C/EBP as a bipartite molecular switch, which regulates the expression of myelomonocyte-specific genes. To understand the relationship between v-Myb and C/EBP in more detail, we have examined the molecular basis of the activation of the mim-1 p
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Chen, R. H., and J. S. Lipsick. "Differential transcriptional activation by v-myb and c-myb in animal cells and Saccharomyces cerevisiae." Molecular and Cellular Biology 13, no. 7 (1993): 4423–31. http://dx.doi.org/10.1128/mcb.13.7.4423.

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The v-myb oncogene and its cellular homolog c-myb encode sequence-specific DNA-binding proteins which regulate transcription from promoters containing Myb-binding sites in animal cells. We have developed a Saccharomyces cerevisiae system to assay transcriptional activation by v-Myb and c-Myb. In yeast strains containing integrated reporter genes, activation was strictly dependent upon both the Myb DNA-binding domain and the Myb recognition element. BAS1, an endogenous Myb-related yeast protein, was not required for transactivation by animal Myb proteins and by itself had no detectable effect o
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Chen, R. H., and J. S. Lipsick. "Differential transcriptional activation by v-myb and c-myb in animal cells and Saccharomyces cerevisiae." Molecular and Cellular Biology 13, no. 7 (1993): 4423–31. http://dx.doi.org/10.1128/mcb.13.7.4423-4431.1993.

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The v-myb oncogene and its cellular homolog c-myb encode sequence-specific DNA-binding proteins which regulate transcription from promoters containing Myb-binding sites in animal cells. We have developed a Saccharomyces cerevisiae system to assay transcriptional activation by v-Myb and c-Myb. In yeast strains containing integrated reporter genes, activation was strictly dependent upon both the Myb DNA-binding domain and the Myb recognition element. BAS1, an endogenous Myb-related yeast protein, was not required for transactivation by animal Myb proteins and by itself had no detectable effect o
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Grässer, F. A., T. Graf, and J. S. Lipsick. "Protein truncation is required for the activation of the c-myb proto-oncogene." Molecular and Cellular Biology 11, no. 8 (1991): 3987–96. http://dx.doi.org/10.1128/mcb.11.8.3987.

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The protein product of the v-myb oncogene of avian myeloblastosis virus, v-Myb, differs from its normal cellular counterpart, c-Myb, by (i) expression under the control of a strong viral long terminal repeat, (ii) truncation of both its amino and carboxyl termini, (iii) replacement of these termini by virally encoded residues, and (iv) substitution of 11 amino acid residues. We had previously shown that neither the virally encoded termini nor the amino acid substitutions are required for transformation by v-Myb. We have now constructed avian retroviruses that express full-length or singly trun
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Grässer, F. A., T. Graf, and J. S. Lipsick. "Protein truncation is required for the activation of the c-myb proto-oncogene." Molecular and Cellular Biology 11, no. 8 (1991): 3987–96. http://dx.doi.org/10.1128/mcb.11.8.3987-3996.1991.

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The protein product of the v-myb oncogene of avian myeloblastosis virus, v-Myb, differs from its normal cellular counterpart, c-Myb, by (i) expression under the control of a strong viral long terminal repeat, (ii) truncation of both its amino and carboxyl termini, (iii) replacement of these termini by virally encoded residues, and (iv) substitution of 11 amino acid residues. We had previously shown that neither the virally encoded termini nor the amino acid substitutions are required for transformation by v-Myb. We have now constructed avian retroviruses that express full-length or singly trun
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Dissertations / Theses on the topic "V-Myb"

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Rivière, Didier. "Etude structurale par RMN de la protéine v-Myb." Rouen, 2004. http://www.theses.fr/2004ROUES040.

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Les protéines Myb sont des facteurs de transcription capables de contrôler la différentiation et la prolifération cellulaire. Il ne fait aujourd'hui aucun doute que les activités importantes des protéines Myb sont déterminées par leurs domaines fonctionnels. La spectroscopie RMN hétéronucléaire multidimensionnelle a été utilisée pour étudier le domaine de liaison minimale à l'ADN de la protéine v-Myb (R2R3 v-Myb) en phase aqueuse. Nous avons ainsi attribué la quasi-totalité de cette protéine. A l'aide des effets structurants secondaires, nous avons pu estimer les régions hélicoi͏̈dales de cett
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SORET, JOHANN. "Specificite du potentiel transformant de l'oncogene v-myb du virus de la myeloblastose aviaire (amv) et mecanismes d'activation du proto-oncogene c-myb." Paris 6, 1989. http://www.theses.fr/1989PA066467.

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L'oncogene v-myb d'amv possede un potentiel transformant restreint aux cellules hematopoietiques. Nos etudes suggerent que l'alteration des regions impliquees dans la constitution du domaine dna binding de v-myb puisse modifier cette specificite et permettre la transformation de fibroblastes de poule. L'analyse des proprietes biologiques de deux mutants qui expriment des sequences v-myb tronquees indique que les produits d'expression correspondants stimulent la proliferation des fibroblastes de poule et pourraient intervenir dans les mecanismes de replication de l'adn. Pour aborder l'etude de
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MERZAK, ABDERRAHIM. "Transformation partielle de fibroblastes d'embryon de poule par des mutants de l'oncogene v-myb et cooperation avec l'oncogene ej-h-ras pour la transformation de cellules d'embryon de hamster." Paris 7, 1992. http://www.theses.fr/1992PA077128.

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L'oncogene v-myb, du virus de la myeloblastose aviaire amv, code pour une proteine nucleaire la p48#v##m#y#b. Cette proteine est constituee de trois domaines importants: 1) un domaine amino-terminal de liaison a l'adn formee d'une sequence repetee trois fois (r1,r2,r3); 2) un domaine central transactivateur de la transcription et 3) un domaine de regulation negative de la transcription. L'etude par des mutants de deletion, du role de differents domaines de la p48#v##m#y#b dans sa specificite de transformation a montre que: a) la deletion de la premiere repetition r1 du domaine de liaison a l'a
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Li, Ruoping. "Effets de mutations ponctuelles ou de délétions dans l'oncongène v-myb des virus AMW et E26 sur la transformation des cellules myéloi͏̈des et sur la prolifération des cellules de neurorétine de poulet." Lille 1, 1989. http://www.theses.fr/1989LIL10072.

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Clonage moléculaire et séquençage du gène v-myb du rétrovirus aviaire thermosensible ts 143 E26 et mise en évidence d'une mutation impliquée dans la fixation de la protéine myb à l'ADN. Démonstration par mutagénèse du virus E26 que cette mutation est responsable du phénotype thermosensible des cellules transformées par ce virus. Mise en évidence du rôle du gène v-myb dans la stimulation in vitro des cellules de neurorétine de poulet
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Bechade, Catherine. "Expression des oncogènes v-myc et v-mil dans les cellules de neurorétine d'embryon de poulet." Paris 7, 1985. http://www.theses.fr/1985PA07F026.

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Le virus de carcinomatose aviaire MH2 est capable d'induire la multiplication des cellules de NR d'embryon de poulet qui normalement ne se divisent pas in vitro. Le génome de MH2 contient deux oncogènes : v-myc et v-mil. Des mutants de MH2 délétés dans le gène v-mil ont perdu la capacité d'induire la prolifération des cellules de NR. Un mutant de MH2 délété dans le gène v-myc a conservé cette propriété. Le produit du gène v-mil est donc nécessaire et suffisant à l'expression de la proprieté mitogène du MH2.
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Bachnou, Naïma. "Transdifferenciation in vivo et in vitro de myeoblastes cardiaques sous l'effet des oncogenes v-myc et v-erba associes." Paris 7, 1992. http://www.theses.fr/1992PA077219.

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Le virus de la myelocytomatose aviaire mc29 (porteur de l'oncogene v-myc) provoque chez l'embryon d'oiseau infecte a 3 jours d'incubation des tumeurs cardiaques et des anomalies cutanees. L'induction de ces tumeurs nous a incitee a etudier l'effet de v-myc associe a d'autres oncogenes. Avec la construction maheva (v-myc associe a v-erba sous le controle des ltr de mh2) nous confirmons que v-myc induit des rhabdomyomes cardiaques. De plus nous mettons en evidence chez l'embryon de poulet un effet de cooperation in vivo et in vitro entre v-myc et v-erba. Cette cooperation induit une transdiffere
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James, Leonard Philip. "Myc and Mad target genes /." Thesis, Connect to this title online; UW restricted, 2000. http://hdl.handle.net/1773/5093.

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Bechade, Catherine. "Role des oncogenes v-mil et v-myc dans la multiplication et la transformation des cellules embryonnaires de neuroretine en culture." Paris 7, 1989. http://www.theses.fr/1989PA077176.

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Le retrovirus aviaire mh2 induit la multiplication et la transformation des cellules de neuroretine (nr) d'embryons de poulet qui normalement ne se divisent pas. Le genome du virus mh2 contient deux oncogenes: v-mil et v-myc. L'isolement de mutants de mh2, deletes dans v-mil ou v-myc, nous a permis de preciser les proprietes biologiques de chacun des oncogenes et d'analyser les relations entre l'induction de la division cellulaire et la transformation. Le gene v-mil est responsable de la division des cellules de nr. Par contre, il induit un phenotype de transformation reduit dans les cellules
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Turque, Nathalie. "Effet de l'oncogène V-MYC sur la différenciation de la neurorétine d'oiseau in vitro." Lille 1, 1996. http://www.theses.fr/1996LIL10204.

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Par criblage d'une banque d'adnc construite à partir d'arnm provenant de cellules de neuroretine de caille infectées par le rétrovirus mc29 porteur de l'oncogène v-myc, ont été isoles deux clones l'un correspondant au gène pax-qnr, orthologue aviaire du gène pax-6/pax6 codant un facteur de transcription essentiel pour la formation des yeux et le deuxième, qnr-71, codant une protéine du mélanome apparentée au produit du gène silver gène. A cote de son expression déjà documentée dans le système nerveux central, nous avons mis en évidence une expression de pax-6 dans le pancréas, et plus particul
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Enikeev, Amir. "Monitorovací systém pro zjištění motility a polohy laboratorních zvířat po anestézii." Master's thesis, Vysoké učení technické v Brně. Fakulta elektrotechniky a komunikačních technologií, 2019. http://www.nusl.cz/ntk/nusl-401008.

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This diplom work entitled "Monitoring system for the detection of motility and position of laboratory animals after anesthesia" focuses on the design and implementation of non-contact detection of the rat or mouse position in the enclosure with a transparent cover. The aim of this semester paper is to find suitable methods of realizing contactless detection of the position of a laboratory rat or mouse. This automatic positioning of the animal will be used as the basis for controlling the irradiator in the next follow-up work, which will "shade" animal movement and aim at the scar on the animal
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Books on the topic "V-Myb"

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Lanux, Veronique Marie De. Immortalization of early hematopoietic progenitors by v-myb overexpression. National Library of Canada, 1994.

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Moncure, Jane Belk. My "v" book. Child's World, 1991.

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Moncure, Jane Belk. My v book. The Child's World, 1991.

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Chernyak, V. G. My igraem v shakhmaty. "Fizkul'tura i sport", 1986.

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Cherni͡ak, V. G. My igraem v shakhmaty. "Fizkulʹtura i sport", 1986.

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My v prave znatʹ. Nestor-Istorii︠a︡, 2010.

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D, Orlova R., ред. My zhili v Këlʹne. Fortuna limited, 2003.

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My vstretilisʹ v rai͡u︡. Vita Nova, 2001.

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Shoĭkhet, Aleksandr. "--Priekhali my v Izrailʹ--". Morii︠a︡, 1995.

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Ermakova, S. O. (Svetlana Olegovna) and Kulikova V. N, eds. My zhili v SSSR. BMM, 2011.

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Book chapters on the topic "V-Myb"

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Vázquez-Cedeira, Marta, Diana M. Monsalve, Marta Sanz-García, et al. "V-Myb Myeloblastosis Viral Oncogene Homolog." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_101460.

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Vázquez-Cedeira, Marta, Diana M. Monsalve, Marta Sanz-García, et al. "v-myc Myelocytomatosis Viral Oncogene Homolog." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_101461.

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Troppmair, J., J. L. Cleveland, D. S. Askew, and U. R. Rapp. "v-Raf/v-Myc Synergism in Abrogation of IL-3 Dependence: v-Raf Suppresses Apoptosis." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77633-5_57.

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Gautschi, Walter. "My Collaboration with Gradimir V. Milovanović." In Approximation and Computation. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6594-3_2.

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Weizsäcker, F. "Kooperation der Onkogene v-myc und v-mil bei der Transformation von Hühnermakrophagen." In Wachstumsfaktoren und Onkogenprodukte bei Entstehung und Regression der Arteriosklerose. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-662-02553-6_7.

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Sullivan, N., C. Green, M. Pasdar, and R. Watt. "Characterization and Nuclear Localization of the v- and c-myc Proteins." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71562-4_52.

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Humphries, E. H., and E. J. Filardo. "The Transforming Activity of PP59C-MYCis Weaker Than That of v-myc." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75889-8_32.

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Harris, A. W., A. Strasser, M. L. Bath, A. G. Elefanty, and S. Cory. "Lymphomas and Plasmacytomas in Transgenic Mice Involving Bcl2, Myc and v-Abl." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60801-8_22.

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Morse, H. C., J. W. Hartley, T. N. Fredrickson, et al. "Tumors of Newborn NFS/N Mice Infected with Murine Retroviruses Containing Avian v-Myc." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71562-4_3.

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Principato, M., S. P. Klinken, J. L. Cleveland, et al. "In Vitro Transformation of Murine Bone Marrow Cells with a v-raf/v-myc Retrovirus Yields Clonally Related Mature B Cells and Macrophages." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-74006-0_6.

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Conference papers on the topic "V-Myb"

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Subbotin, Andrej. "My recollections about G. V. Dluzhnevskaya." In Monuments of archaeology in studies and photographs (in the memory of Galina Vatslavna Dluzhnevskaya). Institute for the History of Material Culture Russian Academy of Sciences, 2018. http://dx.doi.org/10.31600/978-5-907053-08-3-2018-47-50.

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B.B., Alikhanov. "TO WAY TO SAVE THE PLANET LAND RESOURCE." In International Conference Europe, science and we evropa, v da a my. Publishing House "Education and Science" s.r.o., 2020. http://dx.doi.org/10.37057/ch_1_1.

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А.Н., Тургунов. "Использование биологически активных и экологически безопасных веществ при хранении фруктов". У International Conference Europe, science and we evropa, v da a my #2. Publishing House "Education and Science" s.r.o., 2020. http://dx.doi.org/10.37057/ch_2_1.

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Maximiano, António, Guilherme Vaz, and Jule Scharnke. "CFD Verification and Validation Study for a Captive Bullet Entry in Calm Water." In ASME 2017 36th International Conference on Ocean, Offshore and Arctic Engineering. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/omae2017-61666.

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As a step towards complex impact loads cases, e.g. lifeboat drop tests or ship/platform slamming in waves, a verification and validation (V&V) study is carried out with an open-usage community based CFD code ReFRESCO for a simple impact load test case: a captive axisymmetric generic lifeboat shape (bullet) that penetrates the water surface at a constant velocity and angle of attack. The quantities of interest are the body fixed longitudinal force FX, vertical force FZ, and pitch moment MYY. The influence of the iterative convergence level, domain size and free surface modelling are investi
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Yamamoto, Hidetake. "Development of Minimal Invasive SMA Multi-Transducer Unit for Blood Analysis or Drug Delivery." In ASME 2010 5th Frontiers in Biomedical Devices Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/biomed2010-32008.

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This research purpose is to develop minimal medical units applying heated actuations of the Shape Memory Alloy (SMA) transducers using the medical Ti-Ni material, in order to enable minimal hypodermic invasive microvolume either blood suction or drug delivery by equipping nontoxic and minimal edged microneedle to be created in my laboratory. I have focused on lymphocytes for immunotoxin and erythrocytes for glucose level in blood. This paper has reported double-action mechanisms of the compact unit in originally developing and its actions by low DC inputs. The Joule’s heating of the SMA coil s
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Tuzhilova, Elena Nikolaevna. "Organizatsiia detsko-roditel'skogo kluba "Vmeste my smozhem vsio..." v gruppe kratkovremennogo prebyvaniia "Lekoteka" dlia detei s OVZ." In International Research-to-practice conference. TSNS Interaktiv Plus, 2018. http://dx.doi.org/10.21661/r-471434.

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Cole, David E. "An Historical Overview of the Development of the Chevrolet Small-Block V-8 Engine." In ASME 2014 Internal Combustion Engine Division Fall Technical Conference. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/icef2014-5695.

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The Chevrolet Small-Block engine was officially introduced to the public as one of the two engines in the 1955 Chevrolet passenger car. Its companion engine powering Chevrolet products was the very dated “Blue-Flame” 6. This six-cylinder driven powertrain was a key contributor to the reputation at that time that Chevrolet cars were dull and, at best, average. The team: In late 1952, my father, Ed Cole, assumed the role as Chevrolet Chief Engineer. A few years earlier he was the Chief Engineer at Cadillac and led the development of a new, advanced design overhead valve V-8. The expectation at C
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Larionova galina vasilevna, Galina Vasilevna, and Tat'iana Vladimirovna Mostarova. "Metodicheskaia razrabotka po organizatsii dukhovno-prosvetitel'skoi raboty s roditel'skoi obshchestvennost'iu "Igry-sostiazaniia chuvashskikh bogatyrei" v ramkakh detsko-roditel'skogo kluba "My vmeste"." In International Research-to-practice conference. Publishing house Sreda, 2020. http://dx.doi.org/10.31483/r-75024.

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Yanko, T. E. "RUSSIAN ADVERB DAVNO ‚LONG AGO, FOR A LONG TIME‘ REVISITED FROM A CORPUS PERSPECTIVE." In International Conference on Computational Linguistics and Intellectual Technologies "Dialogue". Russian State University for the Humanities, 2020. http://dx.doi.org/10.28995/2075-7182-2020-19-773-783.

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Abstract:
During the last twenty years, the Russian adverb davno ‘long ago, for a long time’ was widely discussed in literature. It was recognized that the unique parameter of davno is its inability to be the theme of a sentence. Moreover, if davno functions in the context of aspectual forms relating to the past it can only be the rheme. In the context of the aspectual verbal forms relating to the past but preserving the connection with the moment of speech, davno can be either the rheme proper, or a component of the rheme. A classic example of an aspectual verb form referring to the past is the general
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