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Journal articles on the topic 'Vagus nerve; Optic nerve'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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1

Frohlich, S., and C. A. Franco. "The neuropsychological function of the 12 cranials nerves." European Psychiatry 26, S2 (2011): 417. http://dx.doi.org/10.1016/s0924-9338(11)72125-3.

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The cranial nerves can be an important key for research in Neuropsychology. Our hypothesis is that they can be organized in three groups and then, related to specifics attitudes.The Cochlear Nerve (VII pair), the Optic Nerve (II pair) and the olfactory nerve (I pair) have special translators that process the sensorial information from the environment to the brain, to form a clue. They are the first cranial nerve group: the cognitive nerves that incite the nervous system in an endogenous way. The second cranial nerve group stimulates muscles: the spinal nerve (XI pair) that regulates the postur

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2

Stern, Yoram, Gideon Marshak, Karl Segal, Thomas Shpitzer, and Raphael Feinmesser. "Vocal Cord Palsy: Possible Late Complication of Radiotherapy for Head and Neck Cancer." Annals of Otology, Rhinology & Laryngology 104, no. 4 (1995): 294–96. http://dx.doi.org/10.1177/000348949510400407.

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Cranial nerve palsies are uncommon complications of radiotherapy for head and neck cancer. A review of the literature reveals that cranial nerve damage after radiotherapy has been reported for the optic, oculomotor, trigeminal, abducens, cochlear, vagus, spinal accessory, and hypoglossal nerves. The hypoglossal nerve appears to be the most commonly affected, and the recurrent laryngeal nerve is seldom involved. The case histories of three patients who developed vocal cord palsy from 21 to 34 years after a course of curative or postoperative radiotherapy for carcinoma of the head and neck are p

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3

Al.Khateeb, MO, S. Mirsattari, D. Diosy, and R. McLachlan. "P.020 Septo-optic dysplasia plus manifesting with medically refractory epilepsy." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 43, S2 (2016): S26. http://dx.doi.org/10.1017/cjn.2016.126.

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Background: Septo-Optic Dysplasia is a rare disorder with developmental malformations that was first reported by De Morsier.SOD associated with refractory epilepsy has not been well studied. We report six cases with SOD in patients with malformation of cortical development(MCD) and medically refractory epilepsy that underwent video-EEG telemetry. Methods: Six cases of SOD plus were admitted to the Epilepsy Monitoring Unit at London Health Sciences Centre because of medically refractory epilepsy. Functional hemispherectomy in one patient resulted in significant reduction of her seizures while i

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4

Dhital, Kumud, Jill Lincoln, Otto Appenzeller, and Geoffrey Burnstock. "Adrenergic innervation of vasa and nervi nervorum of optic, sciatic, vagus and sympathetic nerve trunks in normal and streptozotocin-diabetic rats." Brain Research 367, no. 1-2 (1986): 39–44. http://dx.doi.org/10.1016/0006-8993(86)91576-3.

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5

Grover, Donald A., and Seymour Zigman. "Funduscopic morphology of the light- and dark-adapted eye of the toadfish (Opsanus tau) in vivo." Canadian Journal of Zoology 69, no. 9 (1991): 2498–500. http://dx.doi.org/10.1139/z91-353.

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Intraocular funduscopy and photography permitted serial examination and documentation of time-related changes in the living toadfish (Opsanus tau) eye as it went from the dark-adapted to the light-adapted state. In the present study, the fully dark-adapted eye showed virtually no choroidal, pigment epithelial, or retinal vascular detail. The optic nerve was chalky white with indistinct borders. Grayish brown to chalky white retinal streaks radiated from the optic nerve into the periphery. As light adaptation began, the retinal streaks faded, with the retinal vasculature and the underlying chor

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6

Cyriac, Jiji Tresa, Tambi Cherian, Wasna Ali Hadi, and Joyce Jose. "Optic neuropathy due to allergic fungal rhinosinusitis." Journal of Neurosciences in Rural Practice 02, no. 02 (2011): 180–82. http://dx.doi.org/10.4103/0976-3147.83589.

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ABSTRACTAn uncommon case of allergic fungal rhinosinusitis presented to the ophthalmology outpatient department of our hospital with complaints of blurred vision in the right eye of a few days duration and vague complaints of pain around the eyes. The visual acuity on examination was grossly reduced in the right eye and normal in the left eye. Color vision was normal. Anterior segment examination including pupils was normal. Dilated fundus examination was normal except for temporal pallor in the right optic disc. Automated perimetry and magnetic resonance imaging (MRI) scan of brain and orbit

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7

Voldman, Alexander, Breanna Durbin, John Nguyen, Brian Ellis, and Monique Leys. "Fulminant Idiopathic Intracranial Hypertension and Venous Stasis Retinopathy Resulting in Severe Bilateral Visual Impairment." European Journal of Ophthalmology 27, no. 2 (2017): e25-e27. http://dx.doi.org/10.5301/ejo.5000918.

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Purpose To report a complicated case of fulminant idiopathic intracranial hypertension and concomitant venous stasis retinopathy leading to postpapilledema optic atrophy. Methods Case report. Results A 34-year-old morbidly obese woman with a history of idiopathic intracranial hypertension (IIH) presented with a 1-month history of bilateral vision loss, diplopia, and left eye pain after being lost to follow-up for 6 years. Fundus examination revealed florid papilledema with venous tortuosity bilaterally. Brain and orbit magnetic resonance imaging showed bilateral globe flattening, intraocular o

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8

Wang, Ningli, and Diya Yang. "Cerebrospinal fluid pressure and glaucoma." Modeling and Artificial Intelligence in Ophthalmology 1, no. 3 (2017): 4–8. http://dx.doi.org/10.35119/maio.v1i3.49.

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Cerebrospinal fluid pressure (CSFP) and glaucoma relationship has aroused great interest in recent years. Before that, Elevated intraocular pressure (IOP) has long being considered as the major risk factors for the development and progression of glaucomatous optic nerve damage [1]. However, numerous studies have shown that a relatively large number of patients with typical glaucomatous optic neuropathy (Normal Tension Glaucoma, NTG), in whom the IOP measurements have always been in the normal range (<21mmHg) [2]. Thus, it was illusive that why do NTG patients develop into glaucoma without h

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9

Seabrook, G. R., L. A. Fieber, and D. J. Adams. "Neurotransmission in neonatal rat cardiac ganglion in situ." American Journal of Physiology-Heart and Circulatory Physiology 259, no. 4 (1990): H997—H1005. http://dx.doi.org/10.1152/ajpheart.1990.259.4.h997.

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The intrinsic cardiac ganglia of the neonatal rat heart in situ were studied using electrophysiological and histochemical techniques. The vagal branches innervating the atrial myocardium and cardiac ganglia were identified and individual ganglion cells visualized using Hoffman modulation contrast optics. Histochemical studies revealed the presence of acetylcholinesterase activity associated with neuronal cell bodies and fibers, catecholamine-containing, small intensely fluorescent cells, and cell bodies and nerve fibers immunoreactive for vasoactive intestinal polypeptide. Intracellular record

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10

Firsching, Raimund, Michael Schütze, Markus Motschmann, and Wolfgang Behrens-Baumann. "Venous ophthalmodynamometry: a noninvasive method for assessment of intracranial pressure." Journal of Neurosurgery 93, no. 1 (2000): 33–36. http://dx.doi.org/10.3171/jns.2000.93.1.0033.

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Object. The goal of this study was to examine the potential use of ophthalmodynamometry in the noninvasive assessment of intracranial pressure (ICP). Under normal conditions, pressure within the central retinal vein is equal to or greater than ICP, because the central retinal vein passes through the optic nerve before it drains into the cavernous sinus. The optic nerve sheath is the place where ICP affects retinal venous pressure. Suction ophthalmodynamometry is an established method of investigation in ophthalmology to determine the pressure of the central retinal artery. Although observation

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11

Roohipourmoallai, Ramak, Samaneh Davoudi, Seyed Majid Hosseinian Benvidi, and Siva S. R. Iyer. "Peripheral Retinal Neovascularization in a Patient with Sarcoidosis and Cocaine-Associated Autoimmunity." Case Reports in Ophthalmological Medicine 2021 (June 1, 2021): 1–4. http://dx.doi.org/10.1155/2021/9923260.

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A 63-year-old African-American female with history of sarcoidosis (lymph node biopsy proven) and cocaine abuse for 8 years was referred to us because of new floater. Her ocular history was unremarkable except for vague complaints of visual disturbance during a hospital admission in 2016. On presentation, her visual acuity was 20/400 in the right eye and 20/30 in the left eye. Dilated fundus exam and multimodal imaging showed thick epiretinal membrane (ERM) superior to optic nerve head causing a lamellar macular hole and intra retinal edema in the right eye, a full thickness macular hole, and p

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12

Lulic, Dzenan, Amir Ahmadian, Ali A. Baaj, Selim R. Benbadis, and Fernando L. Vale. "Vagus nerve stimulation." Neurosurgical Focus 27, no. 3 (2009): E5. http://dx.doi.org/10.3171/2009.6.focus09126.

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Vagus nerve stimulation (VNS) is a key tool in the treatment of patients with medically refractory epilepsy. Although the mechanism of action of VNS remains poorly understood, this modality is now the most widely used nonpharmacological treatment for drug-resistant epilepsy. The goal of this work is to review the history of VNS and provide information on recent advances and applications of this technology.

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13

Aaronson, Scott T., and Charles R. Conway. "Vagus Nerve Stimulation." Psychiatric Clinics of North America 41, no. 3 (2018): 409–18. http://dx.doi.org/10.1016/j.psc.2018.05.001.

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14

George, Mark S., Harold A. Sackeim, Lauren B. Marangell, et al. "VAGUS NERVE STIMULATION." Psychiatric Clinics of North America 23, no. 4 (2000): 757–83. http://dx.doi.org/10.1016/s0193-953x(05)70196-9.

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15

Ross, Ian B., Tatiana Maleeva, and William W. Sutherling. "Vagus Nerve Stimulation." Journal of Neurosurgery: Pediatrics 2, no. 5 (2008): 375. http://dx.doi.org/10.3171/ped.2008.2.11.375.

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16

De Tiège, Xavier, Benjamin Legros, Marc Op de Beeck, Serge Goldman, and Patrick Van Bogaert. "Vagus Nerve Stimulation." Journal of Neurosurgery: Pediatrics 2, no. 5 (2008): 375–77. http://dx.doi.org/10.3171/ped.2008.2.11.377.

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17

Jellinger, K. A. "Vagus Nerve Stimulation." European Journal of Neurology 11, no. 10 (2004): 721. http://dx.doi.org/10.1111/j.1468-1331.2004.00852.x.

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18

Bhat, Venkat, and Sidney H. Kennedy. "Vagus Nerve Stimulation." Cognitive And Behavioral Neurology 31, no. 2 (2018): 99–100. http://dx.doi.org/10.1097/wnn.0000000000000155.

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19

Ghia, J. E., P. Blennerhassett, H. K. Ondiveeran, E. Verdu, and S. M. Collins. "The Vagus Nerve." Inflammatory Bowel Diseases 12 (April 2006): S22. http://dx.doi.org/10.1097/00054725-200604002-00049.

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20

Yang, Eric J., Sahil Sekhon, Kristen M. Beck, Isabelle M. Sanchez, Tina Bhutani, and John Koo. "Vagus Nerve Stimulation." Journal of Psoriasis and Psoriatic Arthritis 3, no. 2 (2018): 54–58. http://dx.doi.org/10.1177/2475530318761399.

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Treatments for psoriasis and psoriatic arthritis have progressed at a rapid rate over the past 20 years, but treating patients with recalcitrant disease still remains a difficult task. Current therapies for these diseases involve topical agents, phototherapy, and systemic immunosuppression. However, the role of the nervous system in psoriasis and psoriatic arthritis remains largely unexplored. Recent animal studies and clinical trials have demonstrated that vagus nerve stimulation can decrease inflammatory processes in rheumatoid arthritis and inflammatory bowel disease. In this article, we ou

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21

Amar, A. P., M. L. Levy, C. Y. Liu, and M. L. J. Apuzzo. "Vagus Nerve Stimulation." Proceedings of the IEEE 96, no. 7 (2008): 1142–51. http://dx.doi.org/10.1109/jproc.2008.922569.

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22

Scherrmann, Judith, Christian Hoppe, Thomas Kral, Johannes Schramm, and Christian E. Elger. "Vagus Nerve Stimulation." Journal of Clinical Neurophysiology 18, no. 5 (2001): 408–14. http://dx.doi.org/10.1097/00004691-200109000-00004.

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23

Schachter, Steven C., and Clifford B. Saper. "Vagus Nerve Stimulation." Epilepsia 39, no. 7 (1998): 677–86. http://dx.doi.org/10.1111/j.1528-1157.1998.tb01151.x.

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24

Kernich, Catherine A. "Vagus Nerve Stimulation." Neurologist 10, no. 1 (2004): 57–58. http://dx.doi.org/10.1097/01.nrl.0000106923.10813.92.

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25

Howland, Robert H. "Vagus Nerve Stimulation." Current Behavioral Neuroscience Reports 1, no. 2 (2014): 64–73. http://dx.doi.org/10.1007/s40473-014-0010-5.

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26

George, M. "Vagus nerve stimulation." Journal of Affective Disorders 107 (March 2008): S22—S23. http://dx.doi.org/10.1016/j.jad.2007.12.152.

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27

Yuan, Hsiangkuo, and Stephen D. Silberstein. "Vagus Nerve and Vagus Nerve Stimulation, a Comprehensive Review: Part I." Headache: The Journal of Head and Face Pain 56, no. 1 (2015): 71–78. http://dx.doi.org/10.1111/head.12647.

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28

Yuan, Hsiangkuo, and Stephen D. Silberstein. "Vagus Nerve and Vagus Nerve Stimulation, a Comprehensive Review: Part III." Headache: The Journal of Head and Face Pain 56, no. 3 (2015): 479–90. http://dx.doi.org/10.1111/head.12649.

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29

Yuan, Hsiangkuo, and Stephen D. Silberstein. "Vagus Nerve and Vagus Nerve Stimulation, a Comprehensive Review: Part II." Headache: The Journal of Head and Face Pain 56, no. 2 (2015): 259–66. http://dx.doi.org/10.1111/head.12650.

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30

Beale, John P. "Optic Nerve Ischemia: Optic Nerve Sheath Decompression Alone or With Optic Nerve Decompression." Archives of Ophthalmology 113, no. 4 (1995): 406. http://dx.doi.org/10.1001/archopht.1995.01100040020011.

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31

Ellrich, Jens. "Transcutaneous Vagus Nerve Stimulation." European Neurological Review 6, no. 4 (2011): 254. http://dx.doi.org/10.17925/enr.2011.06.04.254.

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Invasive vagus nerve stimulation (VNS) is an approved treatment for drug-resistant epilepsy. Besides its recognised clinical efficacy, there are major drawbacks, such as invasiveness and a great many side effects. Therefore there is a medical demand for transcutaneous VNS (t-VNS®), which combines selective, non-invasive access to vagus nerve afferents with a low risk profile. Both treatments excite thick myelinated fibres of vagus nerve branches that project to the nucleus of the solitary tract in the brainstem. Preclinical data emphasise the equivalent anticonvulsive effects of both methods.

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32

Hachinski, Vladimir. "Vagus Nerve Stimulation Therapy." Archives of Neurology 55, no. 2 (1998): 234. http://dx.doi.org/10.1001/archneur.55.2.234.

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33

Koo, Betty, Steve D. Ham, Sandeep Sood, and Brent Tarver. "Human Vagus Nerve Electrophysiology." Journal of Clinical Neurophysiology 18, no. 5 (2001): 429–33. http://dx.doi.org/10.1097/00004691-200109000-00007.

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34

Wheless, J. W., and J. Baumgartner. "Vagus nerve stimulation therapy." Drugs of Today 40, no. 6 (2004): 501. http://dx.doi.org/10.1358/dot.2004.40.6.850483.

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35

Wood, Sarah. "Optic Nerve." Oxford Literary Review 26, no. 1 (2004): 139–53. http://dx.doi.org/10.3366/olr.2004.008.

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36

Park, Kyu Hyung, Se Joon Woo, and Jeong-Min Hwang. "Optic Nerve Head Drusen Mimicking Optic Nerve Tumor." Korean Journal of Ophthalmology 29, no. 3 (2015): 207. http://dx.doi.org/10.3341/kjo.2015.29.3.207.

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37

Liauw, L., G. J. Vielvoye, R. J. W. de Keizer, and S. G. van Duinen. "Optic nerve glioma mimicking an optic nerve meningioma." Clinical Neurology and Neurosurgery 98, no. 3 (1996): 258–61. http://dx.doi.org/10.1016/0303-8467(96)00022-4.

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38

Wax, Martin B. "Optic Nerve Ischemia: Optic Nerve Sheath Decompression Alone or With Optic Nerve Decompression-Reply." Archives of Ophthalmology 113, no. 4 (1995): 407. http://dx.doi.org/10.1001/archopht.1995.01100040020012.

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39

Verlinden, T. J. M., K. Rijkers, G. Hoogland, and A. Herrler. "Morphology of the human cervical vagus nerve: implications for vagus nerve stimulation treatment." Acta Neurologica Scandinavica 133, no. 3 (2015): 173–82. http://dx.doi.org/10.1111/ane.12462.

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40

Labuschagne, Jason J. "Vagus Nerve Stimulation: Lead Revision on a Naïve Segment of the Vagus Nerve." Neuromodulation: Technology at the Neural Interface 23, no. 6 (2019): 883–85. http://dx.doi.org/10.1111/ner.13049.

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41

Mahapatra, AK. "Rationale of optic nerve decompression in optic nerve injury." Indian Journal of Neurotrauma 1, no. 1 (2004): 33–36. http://dx.doi.org/10.1016/s0973-0508(04)80024-8.

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42

Thakar, A., A. K. Mahapatra, and D. A. Tandon. "Delayed Optic Nerve Decompression for Indirect Optic Nerve Injury." Laryngoscope 113, no. 1 (2003): 112–19. http://dx.doi.org/10.1097/00005537-200301000-00021.

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43

Behbehani, Raed S., Noel Vacarezza, Robert C. Sergott, Jurij R. Bilyk, Fred Hochberg, and Peter J. Savino. "Isolated Optic Nerve Lymphoma Diagnosed by Optic Nerve Biopsy." American Journal of Ophthalmology 139, no. 6 (2005): 1128–30. http://dx.doi.org/10.1016/j.ajo.2004.12.006.

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44

Ogino, Hiroyuki, Masaki Hara, Motoharu Satake, et al. "Malignant Peripheral Nerve Sheath Tumors of Intrathoracic Vagus Nerve." Journal of Thoracic Imaging 16, no. 3 (2001): 181–84. http://dx.doi.org/10.1097/00005382-200107000-00008.

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45

Kawai, Kensuke, Kenichi Usami, and Nobuhito Saito. "Vagus nerve stimulation for epilepsy." Rinsho Shinkeigaku 51, no. 11 (2011): 990–92. http://dx.doi.org/10.5692/clinicalneurol.51.990.

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46

Millichap, J. Gordon. "Vagus Nerve Stimulation for Epilepsy." Pediatric Neurology Briefs 11, no. 7 (1997): 55. http://dx.doi.org/10.15844/pedneurbriefs-11-7-13.

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47

Howland, Robert H. "What is Vagus Nerve Stimulation?" Journal of Psychosocial Nursing and Mental Health Services 44, no. 8 (2006): 11–14. http://dx.doi.org/10.3928/02793695-20060801-02.

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48

Rowland, Neil E. "The vagus nerve and thirst." Physiology & Behavior 82, no. 1 (2004): 75–80. http://dx.doi.org/10.1016/j.physbeh.2004.04.039.

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49

Kehagias, Dimitris T., Eric C. Bourekas, and Gregory A. Christoforidis. "Schwannoma of the Vagus Nerve." American Journal of Roentgenology 177, no. 3 (2001): 720–21. http://dx.doi.org/10.2214/ajr.177.3.1770720.

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50

Uthman, Basim M. "Vagus Nerve Stimulation for Seizures." Archives of Medical Research 31, no. 3 (2000): 300–303. http://dx.doi.org/10.1016/s0188-4409(00)00060-6.

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