Academic literature on the topic 'Valva Tricuspide'

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Journal articles on the topic "Valva Tricuspide"

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Das, Debasish, Nishant Debta, and Manas Ranjan Mohapatra. "Myxomatous Tricuspid Valve." Journal of Cardiovascular Medicine and Surgery 4, no. 1 (2018): 60–61. http://dx.doi.org/10.21088/jcms.2454.7123.4118.10.

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McElhinney, Doff B., Norman H. Silverman, Michael M. Brook, Frank L. Hanley, and Paul Stanger. "Asymmetrically short tendinous cords causing congenital tricuspid regurgitation: improved understanding of tricuspid valvar dysplasia in the era of color flow echocardiography." Cardiology in the Young 9, no. 3 (May 1999): 300–304. http://dx.doi.org/10.1017/s1047951100004972.

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AbstractBackgroundTricuspid regurgitation as a manifestation of an isolated congenital anomaly of the tricuspid valve is rare. Cross-sectional and color Doppler echocardiography allow improved evaluation of tricuspid valvar function. As a result, the heterogeneous category of congenital tricuspid valvar dysplasia may be better understood from a functional point of view. We are reporting a distinct entity in which tricuspid valvar regurgitation results from failure of coaptation due to short tendinous cords tethering the septal leaflet.Patients and ResultsThree children with significant primary tricuspid regurgitation were evaluated, treated, and followed. On echocardiographic evaluation, a central regurgitant jet of moderate or severe degree was directed toward the atrial septum through poorly coapting tricuspid valvar leaflets, which did not approximate due to tethering of the septal leaflet by abnormally short cords. In one patient, the tricuspid valve was otherwise normal; in the other two the leaflets and cords were also thickened. Two patients underwent surgery at 9 and 11 years of age. The cords tethering the septal leaflet were augmented by interposing appropriate lengths of expanded polytetrafluoroethylene suture and performing commissural annuloplasty. Both patients are asymptomatic 33 and 42 months postoperatively, with mild residual tricuspid regurgitation that has not changed since surgery. The other patient, an 8 month-old infant, has not yet undergone surgery.ConclusionsAsymmetric tendinous cords of the tricuspid valve causing tethering of the septal leaflet is a distinct cause of tricuspid regurgitation that can be recognized with echocardiography. Although rare, the importance of recognizing this lesion lies in its being readily amenable to surgical repair.
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Mortazavi, Ali, Ross M. Reul, Leon Cannizzaro, and Kathryn G. Dougherty. "Transvenous Transcatheter Valve-in-Valve Implantation after Bioprosthetic Tricuspid Valve Failure." Texas Heart Institute Journal 41, no. 5 (October 1, 2014): 507–10. http://dx.doi.org/10.14503/thij-14-4302.

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We describe the case of a 38-year-old man with a history of metastatic testicular cancer who had undergone multiple thoracic surgical procedures, including tricuspid valve replacement with a bioprosthetic valve as a result of tricuspid involvement of his malignancy. He presented at our outpatient cardiology clinic with worsening fatigue, shortness of breath, and peripheral edema, investigation of which revealed severe tricuspid bioprosthesis stenosis with central regurgitation. Because of the patient's medical history, he was considered to be a high-risk surgical candidate. Therefore, transcatheter tricuspid valve-in-valve implantation of a 26-mm Edwards Sapien® valve was attempted through a transjugular approach. The procedure restored tricuspid valvar competence and substantially improved the patient's symptoms. We discuss the technical aspects of this case and briefly review the usefulness of the valve-in-valve technique in the tricuspid position.
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Wong, Kenny K., Duncan I. Farquharson, and Walter J. Duncan. "Unguarded tricuspid valvar orifice in the fetus." Cardiology in the Young 14, no. 5 (October 2004): 557–59. http://dx.doi.org/10.1017/s1047951104005141.

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The unguarded tricuspid valve is uncommon. We describe herein a fetus with a grossly dilated right ventricle and atrium, with severe tricuspid and pulmonary valvar regurgitation. The right ventricle was akinetic, and no tricuspid tissue or valvar apparatus was identified. Colour Doppler showed a highly unusual retrograde flow of blood through the right heart. The pregnancy was terminated, and necropsy examination confirmed the gross dilation of the right heart chambers, with severely dysplastic valvar tissue at the right atrioventricular junction effectively giving an unguarded orifice. There was no valvar displacement, and the left heart was normal. The fetus had a normal karyotype, albeit with absent kidneys.
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El Asmar, Bechara, Michael Acker, Jean Paul Couetil, P. H. Penther, and Alain Carpentier. "Tricuspid valve myxoma: A rare indication for tricuspld valve repair." Annals of Thoracic Surgery 52, no. 6 (December 1991): 1315–16. http://dx.doi.org/10.1016/0003-4975(91)90020-q.

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Du, Zhong-Dong, Nathan Roguin, Mila Barak, and Elliot Milgram. "Prevalence of valvar regurgitation by color Doppler echocardiography in neonates with normal hearts." Cardiology in the Young 6, no. 3 (July 1996): 216–21. http://dx.doi.org/10.1017/s1047951100003693.

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AbstractValvar regurgitation has been commonly found by echocardiography in adults and older children with normal hearts, but there is limited knowledge in neonates. To evaluate the prevalence of valvar regurgitation and analyze risk factors, echocardiography was performed on 975 consecutive neonates (1–7 days of age) with normal hearts. Regurgitation was detected and graded with pulsed, continuous-wave and color Doppler imaging techniques. Thirty-five cases with valvar regurgitation were followed for one month. The results showed that 369 (37.8%) neonates had evidence of regurgitation in one or more valves. Tricuspid regurgitation was most commonly found and was detected in 345 (35.4%) neonates, mitral regurgitation in 51 (5.2%), pulmonic regurgitation in 25 (2.6%), and aortic regurgitation in eight (0.8%). Most of them were trivial or mild and involved just one valve. Moderate or severe regurgitation was detected only in the tricuspid valve (3%) and the mitral valve (0.1 %). Five (0.5%) neonates with severe tricuspid regurgitation had heart murmurs, and two (0.2%) of them presented with cardiorespiratory distress. The risk factors for tricuspid and mitral regurgitation included lower chronologic age, gestational age and whether the neonates had a patent arterial duct; whereas those for tricuspid regurgitation also related to lower Apgar scores, birth weight and whether the neonates had a patent oval foramen. About 90% of the tricuspid regurgitation disappeared and the remaining 10% decreased in one month. We concluded that trivial or mild valvar regurgitation is commonly found in neonates with normal hearts. Moderate or severe regurgitation is relatively rare and occurs only in the tricuspid and mitral valves with a few cases causing clinical attention.
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Bharucha, Tara, Robert H. Anderson, Zek S. Lim, and Joseph J. Vettukattil. "Multiplanar review of three-dimensional echocardiography gives new insights into the morphology of Ebstein’s malformation." Cardiology in the Young 20, no. 1 (January 19, 2010): 49–53. http://dx.doi.org/10.1017/s1047951109991971.

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AbstractIntroductionWe aimed to assess the ability of the multiplanar review modality of three-dimensional echocardiography to examine the dynamic morphology and the functional characteristics of malformed tricuspid valves in patients previously identified as having Ebstein’s malformation. Based on these characteristics, we attempted to differentiate Ebstein’s malformation from tricuspid valvar dysplasia.MethodsUsing three-dimensional multiplanar review, analysed with either Qlab 6.0 or Tomtech Image Arena 3.0, we studied 23 patients, aged from 1 day to 70 years, previously diagnosed using cross-sectional echocardiography as having Ebstein’s malformation.ResultsUsing the features of rotational abnormality, and the orientation, of the effective tricuspid valvar orifice as diagnostic features of Ebstein’s malformation, we reclassified 11 patients (48 per cent) as exhibiting tricuspid valvar dysplasia. In addition, we studied the dynamic morphology as well as the function of the tricuspid valve. Surgical treatment was undertaken on 10 patients, revealing good correlation with the findings obtained using three-dimensional multiplanar review. In those with Ebstein’s malformation, we found varying degrees of rotation, with the effective valvar orifice always directed towards the right ventricular outflow tract. The opening of the orifice of dysplastic tricuspid valves, in contrast, was towards the apex of the right ventricle. The degree of delamination, and abnormalities of subcordal apparatus, were similar in the two groups.DiscussionThree-dimensional multiplanar review permits accurate definition of the dynamic morphology of Ebstein’s malformation, permitting clear differentiation from tricuspid valvar dysplasia.
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Pinto, Robin J., Jaya Deshpande, and Bharat V. Dalvi. "Mitral value anomalies in tricuspid atresia: an autopsy study of 54 hearts." Cardiology in the Young 7, no. 2 (April 1997): 163–71. http://dx.doi.org/10.1017/s1047951100009410.

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AbstractAnatomic study of 54 autopsied cases of tricuspid atresia revealed mitral valvar anomalies in 9 (16.7%). The morphologic findings in these 9 cases are presented and compared with 45 cases having a normal mitral valvar apparatus. The anomalies included a cleft valve in 2, parachute valve in 2, straddling valve in 3, and a valve with dual orifice in 2.Two of these cases had more than one anomaly. Data from echocardiography and cardiac catheterisation, when available, had failed to diagnose the anomalies during life. The anomalies were more commonly associated with discordant ventriculo-arterial connections (33.3% versus 13.3%, p < 0.01), were associated significantly with a large ventricular septal defect (33.3% versus 4.4%, p < 0.01), and with pulmonary hypertension (33.3% versus 2.2%, p < 0.01). Two of 3 post operative deaths among the 9 were due to hemodynamic consequences arising from the uncorrected mitral valvar anomaly (parachute valve in both ). Diagnosis of these anomalies prior to surgical correction is imperative, as their presence may necessitate repair or replacement of the mitral valve. Failure to do so may result in increased perioperative mortality, congestive cardiac failure and arrhythmias at a later date. Mitral valvar anomalies may be a contributory factor to the early onset of cardiac failure seen in patients with tricuspid atresia undergoing palliative shunts.
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Serraf, Alain, Richard A. Jonas, Redmond P. Burke, Aldo R. Castaneda, and John E. Mayer. "Univentricular repair for complex double right ventricle and transposed great arteries." Cardiology in the Young 7, no. 2 (April 1997): 207–14. http://dx.doi.org/10.1017/s1047951100009483.

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AbstractThe Fontan operation was initially designed to provide a “physiological correction” of tricuspid atresia, but its indications have been extended to more complex forms of congenitally malformed hearts. In selected patients with two adequately sized ventricles, but highly complex intra cardiac anatomy which makes a biventricular repair more hazardous and which would require multiple reoperations, a univentricular repair using Fontan's principle has been proposed. Between 1978 and 1992, a modified Fontan operation was carried out in 34 such patients. Of the patients. 13 had double outlet right ventricle and 21 had transposed great arteries. Anomalies of ventricular loop were present in 14 patients. A non-committed ventricular septal defect was found in 26 whilst multiple defects were present in 10. All patients had anomalies of the atrioventricular valves, with straddling of the tricuspid valve,11and/or of the mitral valve11, and abnormal tricuspid5, or mitral4cordal attachments. Two patients had mild tricuspid hypoplasia, 1 had mitral hypoplasia and 3 had a cleft mitral valve. Fourteen patients had anomalies of both atrioventricular valve but only 6 had mild atrioventricular valvar incompetence. Pulmonary stenosis or atresia was seem in 26. All the patients had at least one or more (mean 2.35) intracardiac anomalies that preculded a biventricular repair. The Fontan operation was performed at a median age of 4.75 years. There were 4 early (11.4%, 70% CL: 5.1–19.8) and 2 late deaths, with a 4 year actuarial survival rate of 79.6±8.1%. Univariate analysis did not reveal any risk factor for early of late death. Follow-up at a mean interval of 3.6 ±2.6 years was available in all but 1 survivor. There were 6 early and 2 late reoperations, and 3 patients required implantation of a pacemaker. In conclusion, the modified Fontan operation can provide satisfactory early and mid term results in patients with two ventricles but with complex intracardiac anatomy which precludes a biventricular repair.
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Radford, Dorothy J., R. Bruce Garlick, and Peter G. Pohlner. "Multiple valvar replacements for hypereosinophilic syndrome." Cardiology in the Young 12, no. 1 (January 2002): 67–70. http://dx.doi.org/10.1017/s1047951102000136.

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A boy with familial eosinophilia had the hypereosinophilic syndrome, with involvement of mitral and tricuspid valves. Between the ages of 11 and 20 years, he underwent eight surgical procedures on his atrioventricular valves. The pathology included recurrent thrombotic vegetative masses related to hypereosinophilia. Initial repair of the mitral valve was shortlived, but recurrent repairs of the tricuspid valve were helpful. Mechanical prostheses inserted in the mitral position thrombosed despite anticoagulant therapy, and bioprosthetic valves deteriorated with thrombus, fibrosis, or tearing. The hypereosinophilic syndrome is unusual in children, and produces additional problems with valvar surgery.
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Dissertations / Theses on the topic "Valva Tricuspide"

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Antoniali, Fernando. "Determinação da proporção entre os segmentos do anel da valva tricuspide : estudo anatomico em corações de humanos." [s.n.], 2006. http://repositorio.unicamp.br/jspui/handle/REPOSIP/311156.

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Orientador: Domingo Marcolino Braile
Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
Made available in DSpace on 2018-08-07T03:55:43Z (GMT). No. of bitstreams: 1 Antoniali_Fernando_M.pdf: 4775200 bytes, checksum: 8361f227d501574e0c98c3965065c58b (MD5) Previous issue date: 2006
Resumo: Objetivo: Determinar a proporção existente entre os segmentos do anel da valva tricúspide normal em humanos. Método: Foram estudados 30 corações de cadáveres humanos não formolizados, com menos de 6h de período ¿post-mortem¿, sem lesões congênitas ou adquiridas e com valvas tricúspides sem deformidades e continentes. A continência desta valva foi confirmada por injeção de água sob pressão no interior do ventrículo direito estando a valva pulmonar fechada. Foram realizadas fotos digitais da valva tricúspide com o anel valvar íntegro e após secção na comissura póstero-septal e retificação do anel valvar. Estas fotos contendo escalas milimetradas foram avaliadas por programa específico de computador. Foram feitas medidas computadorizadas do perímetro, segmento septal e segmento ântero-posterior do anel valvar íntegro. Nesta condição também foram feitas medidas da distância linear entre as comissuras ântero-septal e póstero-septal. Na condição de anel valvar retificado, foram realizadas medidas computadorizadas e manuais do perímetro e dos segmentos septal, anterior e posterior do anel valvar tricuspídeo. Compararam-se as medidas médias e as razões entre elas nas condições de anel íntegro e retificado. Compararam-se, também, a forma computadorizada e manual de mensuração do anel. Resultados: Nas medidas computadorizadas realizadas com imagens digitais do anel valvar íntegro, os valores médios do perímetro, segmento septal e ântero-posterior foram 105mm (±12,7), 30,6mm (±3,7) e 74mm (±9,4), respectivamente. A distância linear média entre as comissuras ântero-septal e póstero-septal foi de 28,9mm (±3,4). Nas medidas computadorizadas realizadas com imagens digitais do anel valvar retificado, os valores médios foram 117,5mm (±13,3), 32mm (±3,7), 46,3mm (±8,3) e 39,1mm (±8,5), respectivamente para perímetro, segmento septal, anterior e posterior. A razão média entre o segmento ântero-posterior e o septal foi 2,43 (±0,212) e 2,67 (±0,304) respectivamente em anéis íntegros e retificados. Houve diferenças significantes entre as medidas do perímetro (p<0,0001), do segmento septal (p=0,003) e do segmento ântero-posterior (p<0,0001) quando realizadas em anéis íntegros e retificados. As razões entre segmento ântero-posterior e septal também apresentaram diferença significante (p=0,0005). As medidas manuais do anel valvar retificado apresentaram os valores médios de 118,5mm (±12,7), 32,6mm (±3,4), 46,6mm (±7,7) e 39,3mm (±7,9), respectivamente para perímetro, segmento septal, anterior e posterior. Não houve diferenças significantes entre medidas manuais e computadorizadas. Conclusões: A proporção existente entre os segmento septal e o segmento ântero-posterior, do anel da valva tricúspide normal em humanos, é igual a 1 : 2,43. A secção e retificação do anel tricuspídeo altera as medidas de seus segmentos e suas relações
Abstract: Objective: The purpose of this study was to determine the proportion among the segments of the human tricuspid valve annulus. Methods: Descriptive autopsy study of 30 human hearts, without fixation, with less than six hours of post-mortem period, without congenital or acquired lesions and without tricuspid regurgitation. The tricuspid valve insufficiency was excluded by infusion of pressured water in the right ventricle with closed pulmonary valve. Digital images of the tricuspid ring on anatomical position and on flattened state were analyzed by specific software. Computerized measurements of the perimeter, septal segment, anteroposterior segment and the linear distance between the anteroseptal and posteroseptal commissures were obtained on anatomical position. Computerized and manual measurements of the perimeter, septal, anterior and posterior segments were obtained on flattened state. The measurements were demonstrated and compared on the two different situations, anatomical position and flattened. The computerized measurements were compared with the manual ones. Results: The mean values of the perimeter, septal and anteroposterior segments of the tricuspid ring, obtained by computerized measurements on anatomical position were: 105mm (±12.7), 30.6mm (±3.7) e 74mm (±9.4), respectively. The mean linear distance between the anteroseptal and posteroseptal commissures was 28.9mm (±3.4). On the flattened state and by computerized measurements, the mean value of the perimeter was 117.5mm (±13.3) and of the septal, anterior e posterior segments were respectively: 32mm (±3.7), 46.3mm (±8.3) e 39.1mm (±8.5). The mean ratio between the antero-posterior and septal segments was 2.43 (±0.212) on the anatomical position and on flattened state was 2.67 (±0.304). Statistical differences were observed in the measurements of perimeter (p<0.0001), septal segment (p=0.003) e antero-posterior segment (p<0.0001) on the two situations. Statistical difference also occurred on the ratios between the antero-posterior and septal segments (p=0.0005). The mean values obtained by manual measurements of the tricuspid ring on flattened state were: 118.5mm (±12.7), 32.6mm (±3.4), 46.6mm (±7.7) e 39.3mm (±7.9), respectively for perimeter, septal, anterior and posterior segments. There weren¿t statistical differences on computerized and manual measurements. Conclusions: The proportion between the septal and antero-posterior segments of the normal human tricuspid valve is 1 : 2.43. The attitude of flatting the tricuspid ring to measure the segments, changes their values and the ratios between them
Mestrado
Cirurgia
Mestre em Cirurgia
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Silva, José Pedro da. "Nova técnica cirúrgica para a correção da anomalia de Ebstein: resultados imediatos e em longo prazo." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/5/5156/tde-28012009-154640/.

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Introdução: As principais operações para correção da anomalia de Ebstein baseiam-se na reconstrução da valva atrioventricular direita (AVD) em formato monovalvular, tendo resultados comprometidos pela necessidade de substituição ou alta reincidência de insuficiência valvar. Uma nova técnica foi desenvolvida, diferenciado-se das anteriores, pela correção anatômica da valva AVD, resultando na coaptação plena das válvulas no fechamento valvar. O objetivo deste estudo é avaliar a aplicabilidade dessa técnica, estudando os seus efeitos na evolução clínica, função da valva, restauração do ventrículo direito funcional e remodelamento reverso do coração no pós-operatório imediato (POI) e no pós-operatório em longo prazo (POL). Métodos: Estudo retrospectivo de 52 pacientes consecutivos, com idade média de 18,5±13,8anos, submetidos técnica do cone para correção da anomalia de Ebstein, entre novembro 1993 e dezembro de 2006, cujos principais detalhes cirúrgicos são: as válvulas anterior e posterior da valva AV direita são mobilizadas das suas implantações anômalas no ventrículo direito (VD), a borda livre desse conjunto é rodada no sentido horário para ser suturada à, previamente mobilizada, borda septal da válvula anterior, formando um cone cujo vértice permanece fixo ao ápice do VD e a base é suturada no nível do anel atrioventricular verdadeiro, reduzido ao mesmo tamanho da base do cone. A válvula septal, sempre que possível, é incorporada à parede do cone. A porção atrializada do VD é reduzida por plicatura longitudinal. Os dados clínicos, ecocardiográficos e os índices cardiotorácicos, obtidos nos períodos pré-operatório (PREOP) e pós-operatório, foram analisados. Resultados: Houve dois óbitos hospitalares (3,8 %) e dois óbitos no seguimento em longo prazo A melhora clínica foi significante, sendo a distribuição dos pacientes em classes funcionais de insuficiência cardíaca (NYHA), IV = 4, III = 27, II = 11 e I = 5 no PREOP, modificada para IV = 0, III = 1, II = 2 e I = 44 no pós-operatório em longo prazo (POL) (p<0,0001), com seguimento médio de 57 meses. Quatro pacientes foram reoperados, sendo realizada nova plastia valvar. O índice cardiotorácico de 0,66±0,09 no PREOP diminuiu para 0,54±0,06 no POL (p<0,0001). Os ecocardiogramas mostraram redução dos graus de insuficiência da valva AV direita (p<0,001), sendo a distribuição dos pacientes no pré-operatório, grau 1 = 0, grau 2 = 1, grau 3 = 15, grau 4 = 24, modificada para grau 1 = 19, grau 2 = 17, grau 3 = 4, grau 4 = 0 no POI, com pequena alteração no POL (grau 1 = 11, grau 2 = 22, grau 3 = 7, grau 4 = 0). A cavidade funcional do VD foi restaurada pela operação, ocorrendo aumento da área do VD funcional indexada de 8,53± 7,02 cm2/m2 no PREOP para 21,01±6,87 cm2/m2 no POI (p<0,001), e ficando inalterada em 20,28±5,26 cm2/m2 no POL (p>0,05). Conclusões: Esta técnica foi aplicável com baixa mortalidade hospitalar e sem necessidade de substituição valvar. Houve melhora clínica pós-operatória e baixa incidência de reoperações em longo prazo. A correção da insuficiência valvar foi eficaz e duradoura na maioria dos pacientes. Houve restauração da área funcional do VD e remodelamento reverso do coração.
Background: The main operations for Ebsteins anomaly repair are conceived to reconstruct the tricuspid valve (TV) in a monocusp format, but their results are restricted either by the need for valve replacement or by high incidence of postoperative valve regurgitation. A new surgical technique was developed, that performs an anatomical reconstruction of the tricuspid valve, realizing a leaflet-to-leaflet coaptation at the TV closure. The objective of this study is to access the feasibility of this technique, evaluating its effects in clinical outcome, tricuspid valve function, right ventricle (RV) morphology and reverse remodeling of the heart.Methods: Retrospective study on 52 consecutive patients, mean age of 18,5+- 13,8 years, treated with a new surgical technique for Ebsteins anomaly repair (the cone technique), between November 1993 and December 2006, which principal details are: a) the anterior and posterior tricuspid valve leaflets re mobilizedfrom their anomalous attachments in the RV, the free edge of this complex is rotated clockwise to be sutured to the septal border of anterior leaflet, creatind a cone which vertex remains fixed at RV apex and whose base is the sutured to a true tricuspid annulus, plicated to match it to base of said cone. The septal leaflet is incorporated into the cone wall ewhenever possible. The atrialized chamber is reduced by longitudinal placation. The clinical and echocardiographic data and the patients cardiothoracic ratios, collected at the preoperative, early and late postoperative periods, were analyzed. Results: There were two hospital deaths (3.8 %) and two more deaths in the long term followup. The significant clinical improvement was evident by the change of patients functional class of heart failure (NYHA) from IV=4, III=27, II=11 and I=5, in the preoperative to IV =0, III = 1, II = 2 e I = 44 at 57 months mean long term follow-up (p<0,0001). Four patients required late TV re-repair. Atrioventricular block did not occur and there was no need for tricuspid valve replacement at any time. The cardiothoracic ratio decreased from 0,66+-0,09, preoperatively, to 0,54+-0,06 in long term follow-up (p<0,001). Echocardiographic studies showed significant TV insufficiency reduction from the preoperative patient distribution of: grade 1 = 0, grade 2 = 1, grade 3 = 15, grade 4 = 24, modified to: grade 1 = 19, grade 2 = 17, grade 3 = 4, grade 4 = 0 on early postoperative period (p<0.001), with little change afterwards (grade 1 = 11, grade 2 = 22, grade 3 = 7, grade 4 = 0). The normal RV morphology was surgically restored, indicated by the enlargement of RV indexed area from 8.53+-7.02 cm2/m2, preoperatively to 21.01+-6.87 cm2/m2 in the early perioperative period (p<0.001), remaining unchanged, 20.28+-5.26 cm2/m2 in long term echocardiogram (p>0,05). Conclusions: This operative technique was feasible with low hospital mortality and no need for TV replacement. There was improvement in the patients clinical status and low incidence of reoperations in long term follow-up. The TV repair was efficacious and durable for the great marjority of patients and there was immediate RV morphology restoration and reverse remodeling of heart in long term follow-up
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Amini, Khoiy Keyvan. "Biomechanical Characterization and Simulation of the Tricuspid Valve." University of Akron / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=akron1542651986497595.

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THOMAS, VINEET SUNNY. "A Multiscale Framework to Analyze Tricuspid Valve Biomechanics." University of Akron / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=akron1542255754172363.

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Spinner, Erin M. "Tricuspid valve mechanics: understanding the effect of annular dilatation and papillary muscle displacement." Diss., Georgia Institute of Technology, 2011. http://hdl.handle.net/1853/45754.

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Tricuspid regurgitation (TR), back flow of blood from the right ventricle to the right atrium, has been reported in approximately 85% of the population, with 16% having mild or severe TR. Patients with untreated moderate to severe TR are likely to experience decreased exercise capacity and have increased morbidity and mortality, thus affecting the patient's quality of life. Current methods of repair offer limited rates of success, and many patients require further operations to correct returning levels of TR. Incomplete repair may be due to incomplete understanding of the functional anatomy and mechanics of the TV and the underlying causes of TR. It was hypothesized that alterations in the geometry of tricuspid valve annular and subvalvular apparatus induced by ventricular dilatation determine the severity of TR. In vivo measurements of papillary muscle (PM) position in patients with single or biventricular dilatation revealed PM displacement away from the center of the annulus as compared to control patients. Additionally, pulmonary arterial pressure, annulus area, ventricular size and apical displacement of the anterior PM were highly correlated with the severity of TR. An in vitro right-heart simulator was developed to investigate isolated mechanics of TR. Through these in vitro studies it was demonstrated that the tricuspid valve begins to leak at only 40% dilation, much lower than the mitral valve. Additionally, it was shown that isolated PM displacement resulted in significant TR. The highest levels of TR were achieved with a combination of annular dilatation and PM displacement. Alterations in leaflet coaptation, as quantified by measuring the amount of leaflet available for coaptation and leaflet mobility were observed with annular dilatation and PM displacement, both isolated and combined. The changes in leaflet coaptation resulted in redistribution of the forces on the chords originating from the anterior PM and inserting into the anterior and posterior leaflets. The findings herein provide the clinical and scientific community with a mechanistic understanding of the tricuspid valve to further improve intervention and repair of TV disease.
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Warsi, Mohammed Ali. "Ebstein anomaly of the tricuspid valve in an adult cohort." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0003/MQ46203.pdf.

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Roczek, Emily [Verfasser]. "Influence of Right Ventricular Leads of Cardiac Devices on Tricuspid Valve Function and Occurrence of Tricuspid Regurgitation / Emily Roczek." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2021. http://d-nb.info/1228859469/34.

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FIGUEROA, AGUILAR GABRIELA. "“DISPLASIA VALVULAR TRICUSPIDEA EN PERRO RAZA BULLDOG INGLES” REPORTE DE CASO." Tesis de Licenciatura, Universidad Autónoma del Estado de México, 2016. http://hdl.handle.net/20.500.11799/66356.

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The development of a clinical case of a canine breed Bulldog ingles, 3.5 months old male having difficulty breathing, impaired physical activity as well as an abdominal protuberance described. To a breath chest auscultation with an intensity of 6/6, arrhyth- mias and death rattles perceived. A radiographic study was conducted as an adjunct to physical examination, by which, generalized cardiomegaly, dorsal displacement of the trachea, lung radiopacity with alveolar pattern is observed, plus an echocardiogram in which ventricular septal defect was observed, volume overload, artery dilated pulmonary among other hallazgos and an electrocar- diogram which is interpreted during disease management, advanced deterioration of the patient’s health is evident, the cualle death occurred.
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Lourenço, Magali Gaspar. "Análise da morfologia e da vascularização do aparelho valvar das valvas mitral e tricúspide do homem: analogia com o coração canino." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-25062009-095303/.

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Objetivou-se, neste trabalho, estudar a morfologia e vascularização do aparelho valvar das valvas mitral e tricúspide em corações humanos, comparando os resultados com aqueles obtidos em cães. A estereologia foi usada como ferramenta que permitiu avaliar os volumes entre as cúspides valvares, cordas tendíneas e mm. papilares, para o conhecimento das relações anatômicas desta estrutura. Foram analisados 15 corações humanos (10 homens e cinco mulheres, com idades variando de 9 a 77 anos) e 15 corações caninos (Canis familiaris - SRD 10 machos e cinco fêmeas, adultos). Os corações, sem alterações macroscópicas, foram dissecados, pesados e seu volume aferido com vistas a análise da morfologia. O estudo da irrigação e drenagem foi realizado por meio de moldes vasculares, em 10 corações de cada espécime. Fundamentados nos resultados obtidos, concluiu-se que as estruturas do aparelho valvar do coração canino e humano apresentam semelhanças tanto morfológicas como em relação a vascularização.
In this study we performed a comparative analysis of the morphology and vascularization of the valvar apparatus of the mitral and tricuspid valve of the human and canine heart. The volumes of the heart and valvar structures (valvar cuspids, chordae tendineae and papillary muscle) were investigated using stereological method. Fifteen human (10 men and 5 women) and 15 canine (mongrel dogs, 10 males and 5 females) hearts of different ages were studied. Ten hearts of each specimen were perfused with vascular cast material to assess and describe the vascularization. Our findings indicate that human and canine valvar apparatus of the mitral and tricuspid valves and its vascularization are similar.
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Conklin, Colleen. "Disparities in Survival and Mortality among Infants with Congenital Aortic, Pulmonary, and Tricuspid Valve Defects by Maternal Race/Ethnicity and Infant Sex." Scholar Commons, 2011. http://scholarcommons.usf.edu/etd/3046.

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Background: The etiology of congenital heart valve defects is not well understood; little is known about the risk factors that contribute to the survival and mortality outcomes of children with these defects. Methods: Using data from the Texas Birth Defects Registry (TBDR) we conducted a retrospective cohort study of 2070 singleton infants with congenital aortic, pulmonary, or tricuspid valve atresia or stenosis born in Texas between January 1, 1996 and December 31, 2007 to Hispanic, Non-Hispanic (NH) black, and NH white women. TBDR data were death-to-birth matched by the Texas Vital Statistics Unit for deaths between January 1, 1996 and December 31, 2008. Using Kaplan-Meier survival estimates with log rank tests and Cox proportional hazards regression model hazard ratios (HR) with 95% confidence intervals (CI), we examined whether infant sex and maternal race/ethnicity affected early childhood survival or risk of mortality for children with congenital heart valve defects. Covariates included birth weight and gestational age, maternal age, maternal education, and number of co-occurring birth defects. Results: In children with aortic valve atresia and aortic valve stenosis, we found males had higher early childhood survival than females (55.0% vs. 41.5%, P=0.0451 and 91.6% vs. 82.5%, P=0.0492, respectively). Early childhood survival for males (94.9%) with pulmonary valve stenosis was slightly lower than females (97.1%, P=0.0116), and was also lower for NH black (94.1%) and Hispanic (95.3%) children than NH white children (97.8%, P=0.0340). After adjusting for covariates, early childhood mortality in children with pulmonary valve atresia with hypoplastic right ventricle was greater in NH black than NH white children (HR=2.93, CI 1.09-7.85, P=0.0329) and greater in NH black males than NH white males (HR=4.63, CI 1.12-19.19, P=0.0349). For children with tricuspid valve atresia, early childhood survival was lower in NH black males (35.7%) and Hispanic males (64.0%) than NH white males (81.0%, P=0.0269); after adjusting for covariates, risk for early childhood mortality was higher in NH black than NH white children (HR=3.39, CI 1.41-8.13, P=0.0062), and higher in NH black males than NH white males (HR=5.23, CI 133-20.58, P=0.0179). Conclusions: Our findings demonstrate there are disparities in early childhood survival and risk of mortality by infant sex and maternal race/ethnicity for children with congenital heart valve defects. These findings provide a foundation for further investigation to better understand why these disparities exist and what can be done to improve the outcomes for children with these defects.
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Books on the topic "Valva Tricuspide"

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Soliman, Osama I., and Folkert J. ten Cate, eds. Practical Manual of Tricuspid Valve Diseases. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-58229-0.

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Moorjani, Narain, Bushra S. Rana, and Francis C. Wells. Operative Mitral and Tricuspid Valve Surgery. London: Springer London, 2018. http://dx.doi.org/10.1007/978-1-4471-4204-1.

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Giamberti, Alessandro, and Massimo Chessa, eds. The Tricuspid Valve in Congenital Heart Disease. Milano: Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5400-4.

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Warsi, Mohammed Ali. Ebstein anomaly of the tricuspid valve in an adult cohort. Ottawa: National Library of Canada, 1999.

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Alain, Carpentier. Carpentier's reconstructive valve surgery. Maryland Heights, Mo: Saunders/Elsevier, 2010.

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1957-, Adams David H., and Filsoufi Farzan, eds. Carpentier's reconstructive valve surgery. Maryland Heights, Mo: Saunders/Elsevier, 2010.

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Pneumonia, malignant endocarditis of tricuspid valve. [S.l: s.n., 1985.

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1941-, Rao P. Syamasundar, ed. Tricuspid atresia. 2nd ed. Mt. Kisco, NY: Futura, 1992.

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Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Tricuspid valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0377_update_003.

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Muraru, Denisa, Ashraf M. Anwar, and Jae-Kwan Song. Heart valve disease: tricuspid valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0037.

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The tricuspid valve is currently the subject of much interest from echocardiographers and surgeons. Functional tricuspid regurgitation is the most frequent aetiology of tricuspid valve pathology, is characterized by structurally normal leaflets, and is due to annular dilation and/or leaflet tethering. A primary cause of tricuspid regurgitation with/without stenosis can be identified only in a minority of cases. Echocardiography is the imaging modality of choice for assessing tricuspid valve diseases. It enables the cause to be identified, assesses the severity of valve dysfunction, monitors the right heart remodelling and haemodynamics, and helps decide the timing for surgery. The severity assessment requires the integration of multiple qualitative and quantitative parameters. The recent insights from three-dimensional echocardiography have greatly increased our understanding about the tricuspid valve and its peculiarities with respect to the mitral valve, showing promise to solve many of the current problems of conventional two-dimensional imaging. This chapter provides an overview of the current state-of-the-art assessment of tricuspid valve pathology by echocardiography, including the specific indications, strengths, and limitations of each method for diagnosis and therapeutic planning.
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Book chapters on the topic "Valva Tricuspide"

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Henein, Michael Y., Mary Sheppard, John R. Pepper, and Michael Rigby. "Tricuspid Valve." In Clinical Echocardiography, 63–79. London: Springer London, 2011. http://dx.doi.org/10.1007/978-1-84882-521-5_3.

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Berdajs, Denis, and Marko I. Turina. "Tricuspid Valve." In Operative Anatomy of the Heart, 383–406. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-540-69229-4_8.

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Henein, Michael Y., Mary Sheppard, John Pepper, and Michael Rigby. "Tricuspid Valve." In Clinical Echocardiography, 61–76. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3785-6_3.

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Felder, Laura, Kathryn S. King, and Naudereh Noori. "Tricuspid Valve: Tricuspid Regurgitation." In Learning Cardiac Auscultation, 205–15. London: Springer London, 2015. http://dx.doi.org/10.1007/978-1-4471-6738-9_17.

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Dominik, Jan, and Pavel Zacek. "Tricuspid Valve Surgery." In Heart Valve Surgery, 309–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-12206-4_7.

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Yaku, Hitoshi, Kiyoshi Doi, Sachiko Yamazaki, and Satoshi Numata. "Tricuspid Aortic Valve." In Aortic Valve Preservation, 85–91. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-2068-2_12.

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Hahn, Rebecca T. "Tricuspid Valve Disease." In Heart Valve Disease, 127–45. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-23104-0_9.

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Friedewald, Vincent E. "Tricuspid Valve Stenosis." In Clinical Guide to Cardiovascular Disease, 1271–79. London: Springer London, 2016. http://dx.doi.org/10.1007/978-1-4471-7293-2_96.

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Rivera, R. "Tricuspid Valve Reconstructions." In Cardiac Reconstructions, 74–81. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74629-1_13.

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Tate, David A., and George A. Stouffer. "The tricuspid valve." In Cardiovascular Hemodynamics for the Clinician, 163–70. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781119066491.ch13.

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Conference papers on the topic "Valva Tricuspide"

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Bhattacharya, Shamik, and Zhaoming He. "Tricuspid Valve Annulus Tension." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53088.

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Functional tricuspid regurgitation is a direct outcome of right ventricular dilatation and tricuspid annulus dilatation. The mechanism underlying functional tricuspid regurgitation is believed to be multifactorial and related to abnormalities in right ventricular volume, function and shape. Changes in the right ventricle geometry may lead to alterations in the positions of the papillary muscles (PM) of the tricuspid valve (TV). PM displacement happens in right ventricular dilatation but its correlation with tricuspid annulus dilatation is still unknown. The unique structure and orientation of tricuspid PM has role to play in TV annulus mechanics and right ventricular mechanics (Fig.1). It has been already shown that annulus tension (AT) is a parameter to evaluate left ventricular function that, previously, was evaluated via the left ventricular geometry and pressure [1–3].
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Subbotina, I., M. Bernhardt, E. Girdauskas, C. Sinning, H. Reichenspurner, and B. Sill. "Outcome of Concomitant Tricuspid Valve Surgery in Patients with Mild or Moderate Tricuspid Valve Regurgitation." In 48th Annual Meeting German Society for Thoracic, Cardiac, and Vascular Surgery. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678892.

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Zhingre Sanchez, Jorge D., and Paul A. Iaizzo. "A Novel Transcatheter Edge-to-Edge Suturing Technique and Prototype for Repairing Tricuspid Valve Regurgitation." In 2020 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/dmd2020-9033.

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Abstract Tricuspid valve regurgitation is a major clinical issue that continues to attract interest from interventional cardiologists and medical device designers due to its rising prevalence and progressive nature. This disease impact is exacerbated among the aging population, considered as high risk of mortality for open-heart surgical procedures. Furthermore, early intervention for tricuspid regurgitation following left-sided heart procedures continues to increase. Thus, percutaneous or transcatheter interventions have emerged as the new frontier for tricuspid valve therapy. Specifically, tricuspid leaflet plication, or edge-to-edge repair, is a valvular procedure to enhance the coaptation of the leaflets and reduce regurgitation. The current landscape of approved transcatheter devices for leaflet coaptation are exclusive to the mitral valve or being investigated for tricuspid treatment. However, most of these transcatheter systems are designed with high procedure specificities, are expensive, and require extensive procedural training. Hence, there is an opportunity to percutaneously plicate the tricuspid leaflets using commonly available right-heart catheter equipment. This study details a novel transcatheter repair procedure that can plicate the tricuspid valve leaflets solely using current market released catheters and/or surgical equipment. Testing and evaluation of this prototype procedure was performed using Visible Heart® methodologies.
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Graf, H., F. Herrmann, P. Wellmann, S. Sadoni, C. Hagl, and G. Juchem. "Permanent Pacemaker Requirement after Tricuspid Valve Surgery." In 48th Annual Meeting German Society for Thoracic, Cardiac, and Vascular Surgery. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678950.

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Edriss, H., E. Karem, and H. Mohammadzadeh. "A Novel Endovascular Tricuspid Valve Endocarditis Management." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3399.

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Dolensky, Joseph R., Lauren D. C. Casa, and Ajit P. Yoganathan. "The Effect of Pulmonary Hypertension on Tricuspid Valve Coaptation in Normal and Pathologic Valve Geometries: An In Vitro Study." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80184.

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Pulmonary hypertension (PHTN) is a pathological condition defined as a mean pulmonary artery pressure (mPAP) greater than 25 mmHg. PHTN can result from a number of lung and heart pathologies, including abnormalities of the pulmonary vasculature, left heart disease, chronic lung disease, and chronic thrombotic disease [1]. Regardless of the cause, the increased afterload on the right heart results in right ventricle (RV) hypertrophy and dilatation and tricuspid regurgitation (TR) [2]. RV dilatation is thought to result in the displacement of the tricuspid valve (TV) papillary muscles (PM) and dilatation of the TV annulus, negatively impacting TV function.
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Spinner, Erin M., Dana Buice, Stamatios Lerakis, and Ajit P. Yoganathan. "Alterations in Tricuspid Valve Mechanics as a Result of Annular Dilatation and Papillary Muscle Displacement: An In Vitro Study." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19043.

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Current information as to the mechanism which causes malcoaptation of the tricuspid leaflets and leads to regurgitation is lacking. This study investigated the effect isolated and combined annular dilatation and PM displacement on valve mechanics. 3D echocardiography was used to quantify the changes in valve mechanics in relation to changes in tricuspid regurgitation.
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Govindarajan, V., J. Mousel, S. C. Vigmostad, H. S. Udaykumar, M. M. Levack, J. H. Gorman, B. M. Jackson, R. C. Gorman, and K. B. Chandran. "Patient-Specific Valve Dynamics Using 3D Fluid-Structure Interaction Modeling: Comparison Between Bicuspid and Tricuspid Aortic Valves." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14563.

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Aortic valve diseases such as congenital bicuspid aortic valve (BAV) and progressive calcification in tricuspid valves affect the hemodynamics in the aortic arch. In addition to leaflet calcification, BAVs are associated with other ailments such as aortic coarctation, aneurysm and dissection [1]. It has also been observed that progressive calcification is accelerated in the case of BAVs compared to normal tricuspid valves. While it is not yet known whether the geometric distortion in BAVs is the main cause of calcification [2] in these valves, the distortion in the leaflets may give rise to altered stresses during the deformation processes which might play a role in accelerating the calcification process in BAVs. In addition, the altered flow caused by the change in geometry could alter the local fluid stresses during the opening phase, which might affect the endothelial lining of the aortic wall. Analyzing and comparing BAV and tricuspid aortic valves as a fluid-structure interaction problem will help determine the stress distribution on the leaflets during opening phase, and enable the examination of altered flow dynamics in the ascending aorta. In this study, the opening phase of a patient-specific bicuspid aortic valve is analyzed at physiological conditions and compared with the opening phase of a tricuspid aortic valve.
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Gavazzoni, M., A. Pozzoli, L. Vicentini, M. Miura, M. Zuber, F. Maisano, and M. Taramasso. "Single-Center Experience with Catheter-Based Tricuspid Valve Replacement with NaviGate Bioprosthesis for Tricuspid Regurgitation." In 48th Annual Meeting German Society for Thoracic, Cardiac, and Vascular Surgery. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678846.

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Zhingre Sanchez, Jorge D., Lars M. Mattison, Michael G. Bateman, and Paul A. Iaizzo. "Computational Simulations of Ventricular Outflow Tract Obstructions Associated With Varied Replacement Valve Geometries." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6916.

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Transcatheter replacement therapies for the atrioventricular (AV) valves are considered as the next frontier for the treatment of valvular regurgitation. The AV valves, tricuspid and mitral, are the regulators of blood flow from the atria into the ventricles. During diastole, blood flows through the open tricuspid and mitral valves to fill the right and left ventricles, respectively. During systole, the ventricles contract, closing the AV valves, and forcing the blood to exit through their respective ventricular outflow tracts (VOTs) to the arterial circulations. Although the current gold standard for the treatment of valvular regurgitation is surgical replacement or repair, the field of transcatheter therapies is rapidly expanding as new treatment options for patients; especially for those individuals considered to be at greater risks for surgical complications. Market released bioprosthetic devices for replacing the aortic and pulmonary valves have shown great promise and success. However, the advancement of similar therapies for either the mitral and tricuspid valves remain in the early stages of development. This slower progress is attributed to the high complexities and variabilities of the AV valves, which present challenges for both device design and post-implantation functions.
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Reports on the topic "Valva Tricuspide"

1

Zhao, Xueshan, Kaibo Sun, Siwei Bi, and Zhong Wu. A systematic review and meta-analysis of aortic valve repair for bicuspid versus tricuspid aortic valves. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2020. http://dx.doi.org/10.37766/inplasy2020.12.0079.

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