Relevant bibliographies by topics / Valve tricuspide

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  2. Dissertations / Theses
  3. Books
  4. Book chapters
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Academic literature on the topic 'Valve tricuspide'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Valve tricuspide"

1

Fondard, O. "Lésions acquises de la valve tricuspide." EMC - Cardiologie 1, no. 4 (2006): 1–14. http://dx.doi.org/10.1016/s1166-4568(06)39343-6.

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Chauvaud, S. "Chirurgie des lésions acquises de la valve tricuspide." EMC - Techniques chirurgicales - Thorax 4, no. 3 (2009): 1–11. http://dx.doi.org/10.1016/s1241-8226(09)50082-5.

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Campelo-Parada, F., L. Cheddadi, and D. Carrié. "La valve tricuspide en 2017 : les premiers pas des traitements percutanés." Archives des Maladies du Coeur et des Vaisseaux - Pratique 2017, no. 260 (2017): 10–17. http://dx.doi.org/10.1016/j.amcp.2017.06.001.

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Bigué Mar, Ndeye, Issa Dior Seck, Karim Garba Yakouba, Racky Wade, Ablaye Ndiaye, and Assane Ndiaye. "Étude anatomique par dissection de la valve tricuspide chez l’homme (à propos de 21 cas)." Morphologie 105, no. 350 (2021): S30. http://dx.doi.org/10.1016/j.morpho.2021.05.086.

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Chetboul, V., D. Tran, C. Carlos, D. Tessier, and J. L. Pouchelon. "Les malformations congénitales de la valve tricuspide chez les carnivores domestiques: étude rétrospective de 50 cas." Schweizer Archiv für Tierheilkunde 146, no. 6 (2004): 265–75. http://dx.doi.org/10.1024/0036-7281.146.6.265.

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Loudot, C., C. Baeteman, M. Catanese, et al. "343 Analyse en tomographie à cohérence optique haute définition de foyers rétiniens secondaires à une endocardite de la valve tricuspide." Journal Français d'Ophtalmologie 31 (April 2008): 117–18. http://dx.doi.org/10.1016/s0181-5512(08)70941-0.

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Das, Debasish, Nishant Debta, and Manas Ranjan Mohapatra. "Myxomatous Tricuspid Valve." Journal of Cardiovascular Medicine and Surgery 4, no. 1 (2018): 60–61. http://dx.doi.org/10.21088/jcms.2454.7123.4118.10.

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El Asmar, Bechara, Michael Acker, Jean Paul Couetil, P. H. Penther, and Alain Carpentier. "Tricuspid valve myxoma: A rare indication for tricuspld valve repair." Annals of Thoracic Surgery 52, no. 6 (1991): 1315–16. http://dx.doi.org/10.1016/0003-4975(91)90020-q.

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D, Kishore Naick, Sreekanth C, Thyagaraju K, and Subhadra Devi Velichety. "MORPHOMETRY OF TRICUSPID VALVE IN HUMAN FOETAL CADAVERS." International Journal of Anatomy and Research 3, no. 2 (2015): 1114–20. http://dx.doi.org/10.16965/ijar.2015.173.

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Allen, Margaret D., Frank Slachman, A. Craig Eddy, Daniel Cohen, Catherine M. Otto, and Alan S. Pearlman. "Tricuspid valve repair for tricuspid valve endocarditis: Tricuspid valve “recycling”." Annals of Thoracic Surgery 51, no. 4 (1991): 593–98. http://dx.doi.org/10.1016/0003-4975(91)90317-j.

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Dissertations / Theses on the topic "Valve tricuspide"

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Antoniali, Fernando. "Determinação da proporção entre os segmentos do anel da valva tricuspide : estudo anatomico em corações de humanos." [s.n.], 2006. http://repositorio.unicamp.br/jspui/handle/REPOSIP/311156.

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Orientador: Domingo Marcolino Braile<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas<br>Made available in DSpace on 2018-08-07T03:55:43Z (GMT). No. of bitstreams: 1 Antoniali_Fernando_M.pdf: 4775200 bytes, checksum: 8361f227d501574e0c98c3965065c58b (MD5) Previous issue date: 2006<br>Resumo: Objetivo: Determinar a proporção existente entre os segmentos do anel da valva tricúspide normal em humanos. Método: Foram estudados 30 corações de cadáveres humanos não formolizados, com menos de 6h de período ¿post-mortem¿, sem lesões congênitas ou adquiridas e com valvas tricúspides sem deformidades e continentes. A continência desta valva foi confirmada por injeção de água sob pressão no interior do ventrículo direito estando a valva pulmonar fechada. Foram realizadas fotos digitais da valva tricúspide com o anel valvar íntegro e após secção na comissura póstero-septal e retificação do anel valvar. Estas fotos contendo escalas milimetradas foram avaliadas por programa específico de computador. Foram feitas medidas computadorizadas do perímetro, segmento septal e segmento ântero-posterior do anel valvar íntegro. Nesta condição também foram feitas medidas da distância linear entre as comissuras ântero-septal e póstero-septal. Na condição de anel valvar retificado, foram realizadas medidas computadorizadas e manuais do perímetro e dos segmentos septal, anterior e posterior do anel valvar tricuspídeo. Compararam-se as medidas médias e as razões entre elas nas condições de anel íntegro e retificado. Compararam-se, também, a forma computadorizada e manual de mensuração do anel. Resultados: Nas medidas computadorizadas realizadas com imagens digitais do anel valvar íntegro, os valores médios do perímetro, segmento septal e ântero-posterior foram 105mm (±12,7), 30,6mm (±3,7) e 74mm (±9,4), respectivamente. A distância linear média entre as comissuras ântero-septal e póstero-septal foi de 28,9mm (±3,4). Nas medidas computadorizadas realizadas com imagens digitais do anel valvar retificado, os valores médios foram 117,5mm (±13,3), 32mm (±3,7), 46,3mm (±8,3) e 39,1mm (±8,5), respectivamente para perímetro, segmento septal, anterior e posterior. A razão média entre o segmento ântero-posterior e o septal foi 2,43 (±0,212) e 2,67 (±0,304) respectivamente em anéis íntegros e retificados. Houve diferenças significantes entre as medidas do perímetro (p<0,0001), do segmento septal (p=0,003) e do segmento ântero-posterior (p<0,0001) quando realizadas em anéis íntegros e retificados. As razões entre segmento ântero-posterior e septal também apresentaram diferença significante (p=0,0005). As medidas manuais do anel valvar retificado apresentaram os valores médios de 118,5mm (±12,7), 32,6mm (±3,4), 46,6mm (±7,7) e 39,3mm (±7,9), respectivamente para perímetro, segmento septal, anterior e posterior. Não houve diferenças significantes entre medidas manuais e computadorizadas. Conclusões: A proporção existente entre os segmento septal e o segmento ântero-posterior, do anel da valva tricúspide normal em humanos, é igual a 1 : 2,43. A secção e retificação do anel tricuspídeo altera as medidas de seus segmentos e suas relações<br>Abstract: Objective: The purpose of this study was to determine the proportion among the segments of the human tricuspid valve annulus. Methods: Descriptive autopsy study of 30 human hearts, without fixation, with less than six hours of post-mortem period, without congenital or acquired lesions and without tricuspid regurgitation. The tricuspid valve insufficiency was excluded by infusion of pressured water in the right ventricle with closed pulmonary valve. Digital images of the tricuspid ring on anatomical position and on flattened state were analyzed by specific software. Computerized measurements of the perimeter, septal segment, anteroposterior segment and the linear distance between the anteroseptal and posteroseptal commissures were obtained on anatomical position. Computerized and manual measurements of the perimeter, septal, anterior and posterior segments were obtained on flattened state. The measurements were demonstrated and compared on the two different situations, anatomical position and flattened. The computerized measurements were compared with the manual ones. Results: The mean values of the perimeter, septal and anteroposterior segments of the tricuspid ring, obtained by computerized measurements on anatomical position were: 105mm (±12.7), 30.6mm (±3.7) e 74mm (±9.4), respectively. The mean linear distance between the anteroseptal and posteroseptal commissures was 28.9mm (±3.4). On the flattened state and by computerized measurements, the mean value of the perimeter was 117.5mm (±13.3) and of the septal, anterior e posterior segments were respectively: 32mm (±3.7), 46.3mm (±8.3) e 39.1mm (±8.5). The mean ratio between the antero-posterior and septal segments was 2.43 (±0.212) on the anatomical position and on flattened state was 2.67 (±0.304). Statistical differences were observed in the measurements of perimeter (p<0.0001), septal segment (p=0.003) e antero-posterior segment (p<0.0001) on the two situations. Statistical difference also occurred on the ratios between the antero-posterior and septal segments (p=0.0005). The mean values obtained by manual measurements of the tricuspid ring on flattened state were: 118.5mm (±12.7), 32.6mm (±3.4), 46.6mm (±7.7) e 39.3mm (±7.9), respectively for perimeter, septal, anterior and posterior segments. There weren¿t statistical differences on computerized and manual measurements. Conclusions: The proportion between the septal and antero-posterior segments of the normal human tricuspid valve is 1 : 2.43. The attitude of flatting the tricuspid ring to measure the segments, changes their values and the ratios between them<br>Mestrado<br>Cirurgia<br>Mestre em Cirurgia
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Bonhomme, Stéphanie. "Insuffisance tricuspide, première manifestation d'une tumeur carnicoi͏̈de de l'ovaire : à propos d'un cas et revue de la littérature." Paris 13, 2004. http://www.theses.fr/2004PA130035.

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Roubertie, François. "Identification de substrats arythmogènes et des mécanismes de décompensation dans une population de tétralogie de Fallot à l’âge adulte et perspectives de prise en charge ultérieure." Thesis, Bordeaux, 2015. http://www.theses.fr/2015BORD0421/document.

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Le nombre d’adultes porteurs d’une tétralogie de Fallot opérée dans l’enfance est en constante augmentation. Initialement, ces patients étaient considérés comme guéris. A l’âge adulte, ils présentent en fait des complications d’ordre rythmique, responsables de morts subites, et des complications d’ordre mécanique : dilatation du ventricule droit (VD) liée à l’insuffisance pulmonaire chronique, séquellaire de la première chirurgie de réparation de la cardiopathie. Les mécanismes de l’arythmie ainsi qu’une éventuelle interaction entre la dysfonction VD et la survenue de ces arythmies ne restent que partiellement élucidés. Dans ce travail, en couplant les données d’études cliniques et les données expérimentales issues d’un modèle animal (MA) mimant une tétralogie de Fallot réparée, nous avons montré que 1) l’échocardiographie ne pouvait pas se substituer à l’IRM pour la surveillance des patients avec tétralogie de Fallot réparée 2) la valvulation pulmonaire restait une intervention à risque de mortalité 3) une bioprothèse non stentée était une bonne solution pour effectuer cette valvulation 4) en cas de fuite tricuspidienne sévère lors de cette valvulation, une plastie était indispensable 5) plusieurs gènes participaient au remodelage ventriculaire droit (analyse génétique effectuée sur le MA) 6) le remodelage électrophysiologique du VD (MA) s’accompagnait de propriétés pro-arythmogènes. Les mécanismes de décompensation sont intriqués : un lien entre dysfonction VD et arythmie paraît bien établi. D’autres analyses électrophysiologiques sont en cours au niveau du ventricule gauche (MA), pour rechercher d’autres mécanismes pro-arythmogènes<br>The number of adults with a repaired tetralogy of Fallot is increasing. In the past, those patients were considered healed. Nonetheless, they present arrhythmogenic issues, with frequent sudden death, and mechanical complications: right ventricular dilation due to long lasting pulmonary valve regurgitation, secondary to surgical repair. The origin of arrhythmia and its interaction with right ventricular dysfunction is only partially understood. In this study, combining clinical with experimental data, we pointed out: 1) concerning the follow-up of this population, echocardiography is not a substitute to MRI 2) operative mortality of pulmonary valve replacement (PVR) still exists 3) a stentless bioprosthesis represents a valid solution for PVR 4) a valve repair is mandatory for severe tricuspid valve regurgitation at PVR 5) the genetic analysis carried out in an animal model of repaired tetralogy of Fallot, demonstrated the involvement of numerous genes in right ventricular remodeling 6) remodeling of the right ventricle in this animal model generates pro-arrhythmic substrate. Heart failure mechanisms in repaired tetralogy of Fallot are complex: a link between right ventricular dysfunction and arrhythmias is demonstrated. Further studies are needed to investigate other pro-arrhythmic mechanisms involving the left ventricle
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Amini, Khoiy Keyvan. "Biomechanical Characterization and Simulation of the Tricuspid Valve." University of Akron / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=akron1542651986497595.

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THOMAS, VINEET SUNNY. "A Multiscale Framework to Analyze Tricuspid Valve Biomechanics." University of Akron / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=akron1542255754172363.

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Spinner, Erin M. "Tricuspid valve mechanics: understanding the effect of annular dilatation and papillary muscle displacement." Diss., Georgia Institute of Technology, 2011. http://hdl.handle.net/1853/45754.

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Tricuspid regurgitation (TR), back flow of blood from the right ventricle to the right atrium, has been reported in approximately 85% of the population, with 16% having mild or severe TR. Patients with untreated moderate to severe TR are likely to experience decreased exercise capacity and have increased morbidity and mortality, thus affecting the patient's quality of life. Current methods of repair offer limited rates of success, and many patients require further operations to correct returning levels of TR. Incomplete repair may be due to incomplete understanding of the functional anatomy and mechanics of the TV and the underlying causes of TR. It was hypothesized that alterations in the geometry of tricuspid valve annular and subvalvular apparatus induced by ventricular dilatation determine the severity of TR. In vivo measurements of papillary muscle (PM) position in patients with single or biventricular dilatation revealed PM displacement away from the center of the annulus as compared to control patients. Additionally, pulmonary arterial pressure, annulus area, ventricular size and apical displacement of the anterior PM were highly correlated with the severity of TR. An in vitro right-heart simulator was developed to investigate isolated mechanics of TR. Through these in vitro studies it was demonstrated that the tricuspid valve begins to leak at only 40% dilation, much lower than the mitral valve. Additionally, it was shown that isolated PM displacement resulted in significant TR. The highest levels of TR were achieved with a combination of annular dilatation and PM displacement. Alterations in leaflet coaptation, as quantified by measuring the amount of leaflet available for coaptation and leaflet mobility were observed with annular dilatation and PM displacement, both isolated and combined. The changes in leaflet coaptation resulted in redistribution of the forces on the chords originating from the anterior PM and inserting into the anterior and posterior leaflets. The findings herein provide the clinical and scientific community with a mechanistic understanding of the tricuspid valve to further improve intervention and repair of TV disease.
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Warsi, Mohammed Ali. "Ebstein anomaly of the tricuspid valve in an adult cohort." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0003/MQ46203.pdf.

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Roczek, Emily [Verfasser]. "Influence of Right Ventricular Leads of Cardiac Devices on Tricuspid Valve Function and Occurrence of Tricuspid Regurgitation / Emily Roczek." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2021. http://d-nb.info/1228859469/34.

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Silva, José Pedro da. "Nova técnica cirúrgica para a correção da anomalia de Ebstein: resultados imediatos e em longo prazo." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/5/5156/tde-28012009-154640/.

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Introdução: As principais operações para correção da anomalia de Ebstein baseiam-se na reconstrução da valva atrioventricular direita (AVD) em formato monovalvular, tendo resultados comprometidos pela necessidade de substituição ou alta reincidência de insuficiência valvar. Uma nova técnica foi desenvolvida, diferenciado-se das anteriores, pela correção anatômica da valva AVD, resultando na coaptação plena das válvulas no fechamento valvar. O objetivo deste estudo é avaliar a aplicabilidade dessa técnica, estudando os seus efeitos na evolução clínica, função da valva, restauração do ventrículo direito funcional e remodelamento reverso do coração no pós-operatório imediato (POI) e no pós-operatório em longo prazo (POL). Métodos: Estudo retrospectivo de 52 pacientes consecutivos, com idade média de 18,5±13,8anos, submetidos técnica do cone para correção da anomalia de Ebstein, entre novembro 1993 e dezembro de 2006, cujos principais detalhes cirúrgicos são: as válvulas anterior e posterior da valva AV direita são mobilizadas das suas implantações anômalas no ventrículo direito (VD), a borda livre desse conjunto é rodada no sentido horário para ser suturada à, previamente mobilizada, borda septal da válvula anterior, formando um cone cujo vértice permanece fixo ao ápice do VD e a base é suturada no nível do anel atrioventricular verdadeiro, reduzido ao mesmo tamanho da base do cone. A válvula septal, sempre que possível, é incorporada à parede do cone. A porção atrializada do VD é reduzida por plicatura longitudinal. Os dados clínicos, ecocardiográficos e os índices cardiotorácicos, obtidos nos períodos pré-operatório (PREOP) e pós-operatório, foram analisados. Resultados: Houve dois óbitos hospitalares (3,8 %) e dois óbitos no seguimento em longo prazo A melhora clínica foi significante, sendo a distribuição dos pacientes em classes funcionais de insuficiência cardíaca (NYHA), IV = 4, III = 27, II = 11 e I = 5 no PREOP, modificada para IV = 0, III = 1, II = 2 e I = 44 no pós-operatório em longo prazo (POL) (p<0,0001), com seguimento médio de 57 meses. Quatro pacientes foram reoperados, sendo realizada nova plastia valvar. O índice cardiotorácico de 0,66±0,09 no PREOP diminuiu para 0,54±0,06 no POL (p<0,0001). Os ecocardiogramas mostraram redução dos graus de insuficiência da valva AV direita (p<0,001), sendo a distribuição dos pacientes no pré-operatório, grau 1 = 0, grau 2 = 1, grau 3 = 15, grau 4 = 24, modificada para grau 1 = 19, grau 2 = 17, grau 3 = 4, grau 4 = 0 no POI, com pequena alteração no POL (grau 1 = 11, grau 2 = 22, grau 3 = 7, grau 4 = 0). A cavidade funcional do VD foi restaurada pela operação, ocorrendo aumento da área do VD funcional indexada de 8,53± 7,02 cm2/m2 no PREOP para 21,01±6,87 cm2/m2 no POI (p<0,001), e ficando inalterada em 20,28±5,26 cm2/m2 no POL (p>0,05). Conclusões: Esta técnica foi aplicável com baixa mortalidade hospitalar e sem necessidade de substituição valvar. Houve melhora clínica pós-operatória e baixa incidência de reoperações em longo prazo. A correção da insuficiência valvar foi eficaz e duradoura na maioria dos pacientes. Houve restauração da área funcional do VD e remodelamento reverso do coração.<br>Background: The main operations for Ebsteins anomaly repair are conceived to reconstruct the tricuspid valve (TV) in a monocusp format, but their results are restricted either by the need for valve replacement or by high incidence of postoperative valve regurgitation. A new surgical technique was developed, that performs an anatomical reconstruction of the tricuspid valve, realizing a leaflet-to-leaflet coaptation at the TV closure. The objective of this study is to access the feasibility of this technique, evaluating its effects in clinical outcome, tricuspid valve function, right ventricle (RV) morphology and reverse remodeling of the heart.Methods: Retrospective study on 52 consecutive patients, mean age of 18,5+- 13,8 years, treated with a new surgical technique for Ebsteins anomaly repair (the cone technique), between November 1993 and December 2006, which principal details are: a) the anterior and posterior tricuspid valve leaflets re mobilizedfrom their anomalous attachments in the RV, the free edge of this complex is rotated clockwise to be sutured to the septal border of anterior leaflet, creatind a cone which vertex remains fixed at RV apex and whose base is the sutured to a true tricuspid annulus, plicated to match it to base of said cone. The septal leaflet is incorporated into the cone wall ewhenever possible. The atrialized chamber is reduced by longitudinal placation. The clinical and echocardiographic data and the patients cardiothoracic ratios, collected at the preoperative, early and late postoperative periods, were analyzed. Results: There were two hospital deaths (3.8 %) and two more deaths in the long term followup. The significant clinical improvement was evident by the change of patients functional class of heart failure (NYHA) from IV=4, III=27, II=11 and I=5, in the preoperative to IV =0, III = 1, II = 2 e I = 44 at 57 months mean long term follow-up (p<0,0001). Four patients required late TV re-repair. Atrioventricular block did not occur and there was no need for tricuspid valve replacement at any time. The cardiothoracic ratio decreased from 0,66+-0,09, preoperatively, to 0,54+-0,06 in long term follow-up (p<0,001). Echocardiographic studies showed significant TV insufficiency reduction from the preoperative patient distribution of: grade 1 = 0, grade 2 = 1, grade 3 = 15, grade 4 = 24, modified to: grade 1 = 19, grade 2 = 17, grade 3 = 4, grade 4 = 0 on early postoperative period (p<0.001), with little change afterwards (grade 1 = 11, grade 2 = 22, grade 3 = 7, grade 4 = 0). The normal RV morphology was surgically restored, indicated by the enlargement of RV indexed area from 8.53+-7.02 cm2/m2, preoperatively to 21.01+-6.87 cm2/m2 in the early perioperative period (p<0.001), remaining unchanged, 20.28+-5.26 cm2/m2 in long term echocardiogram (p>0,05). Conclusions: This operative technique was feasible with low hospital mortality and no need for TV replacement. There was improvement in the patients clinical status and low incidence of reoperations in long term follow-up. The TV repair was efficacious and durable for the great marjority of patients and there was immediate RV morphology restoration and reverse remodeling of heart in long term follow-up
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FIGUEROA, AGUILAR GABRIELA. "“DISPLASIA VALVULAR TRICUSPIDEA EN PERRO RAZA BULLDOG INGLES” REPORTE DE CASO." Tesis de Licenciatura, Universidad Autónoma del Estado de México, 2016. http://hdl.handle.net/20.500.11799/66356.

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The development of a clinical case of a canine breed Bulldog ingles, 3.5 months old male having difficulty breathing, impaired physical activity as well as an abdominal protuberance described. To a breath chest auscultation with an intensity of 6/6, arrhyth- mias and death rattles perceived. A radiographic study was conducted as an adjunct to physical examination, by which, generalized cardiomegaly, dorsal displacement of the trachea, lung radiopacity with alveolar pattern is observed, plus an echocardiogram in which ventricular septal defect was observed, volume overload, artery dilated pulmonary among other hallazgos and an electrocar- diogram which is interpreted during disease management, advanced deterioration of the patient’s health is evident, the cualle death occurred.
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Books on the topic "Valve tricuspide"

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Soliman, Osama I., and Folkert J. ten Cate, eds. Practical Manual of Tricuspid Valve Diseases. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-58229-0.

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Moorjani, Narain, Bushra S. Rana, and Francis C. Wells. Operative Mitral and Tricuspid Valve Surgery. Springer London, 2018. http://dx.doi.org/10.1007/978-1-4471-4204-1.

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Giamberti, Alessandro, and Massimo Chessa, eds. The Tricuspid Valve in Congenital Heart Disease. Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5400-4.

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Alain, Carpentier. Carpentier's reconstructive valve surgery. Saunders/Elsevier, 2010.

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1957-, Adams David H., and Filsoufi Farzan, eds. Carpentier's reconstructive valve surgery. Saunders/Elsevier, 2010.

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Warsi, Mohammed Ali. Ebstein anomaly of the tricuspid valve in an adult cohort. National Library of Canada, 1999.

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Pneumonia, malignant endocarditis of tricuspid valve. s.n., 1985.

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1941-, Rao P. Syamasundar, ed. Tricuspid atresia. 2nd ed. Futura, 1992.

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Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Tricuspid valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0377_update_003.

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Muraru, Denisa, Ashraf M. Anwar, and Jae-Kwan Song. Heart valve disease: tricuspid valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0037.

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The tricuspid valve is currently the subject of much interest from echocardiographers and surgeons. Functional tricuspid regurgitation is the most frequent aetiology of tricuspid valve pathology, is characterized by structurally normal leaflets, and is due to annular dilation and/or leaflet tethering. A primary cause of tricuspid regurgitation with/without stenosis can be identified only in a minority of cases. Echocardiography is the imaging modality of choice for assessing tricuspid valve diseases. It enables the cause to be identified, assesses the severity of valve dysfunction, monitors the right heart remodelling and haemodynamics, and helps decide the timing for surgery. The severity assessment requires the integration of multiple qualitative and quantitative parameters. The recent insights from three-dimensional echocardiography have greatly increased our understanding about the tricuspid valve and its peculiarities with respect to the mitral valve, showing promise to solve many of the current problems of conventional two-dimensional imaging. This chapter provides an overview of the current state-of-the-art assessment of tricuspid valve pathology by echocardiography, including the specific indications, strengths, and limitations of each method for diagnosis and therapeutic planning.
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Book chapters on the topic "Valve tricuspide"

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Felder, Laura, Kathryn S. King, and Naudereh Noori. "Tricuspid Valve: Tricuspid Regurgitation." In Learning Cardiac Auscultation. Springer London, 2015. http://dx.doi.org/10.1007/978-1-4471-6738-9_17.

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Henein, Michael Y., Mary Sheppard, John R. Pepper, and Michael Rigby. "Tricuspid Valve." In Clinical Echocardiography. Springer London, 2011. http://dx.doi.org/10.1007/978-1-84882-521-5_3.

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Berdajs, Denis, and Marko I. Turina. "Tricuspid Valve." In Operative Anatomy of the Heart. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-540-69229-4_8.

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Henein, Michael Y., Mary Sheppard, John Pepper, and Michael Rigby. "Tricuspid Valve." In Clinical Echocardiography. Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3785-6_3.

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Dominik, Jan, and Pavel Zacek. "Tricuspid Valve Surgery." In Heart Valve Surgery. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-12206-4_7.

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Yaku, Hitoshi, Kiyoshi Doi, Sachiko Yamazaki, and Satoshi Numata. "Tricuspid Aortic Valve." In Aortic Valve Preservation. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-2068-2_12.

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Hahn, Rebecca T. "Tricuspid Valve Disease." In Heart Valve Disease. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-23104-0_9.

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Friedewald, Vincent E. "Tricuspid Valve Stenosis." In Clinical Guide to Cardiovascular Disease. Springer London, 2016. http://dx.doi.org/10.1007/978-1-4471-7293-2_96.

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Rivera, R. "Tricuspid Valve Reconstructions." In Cardiac Reconstructions. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74629-1_13.

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Tate, David A., and George A. Stouffer. "The tricuspid valve." In Cardiovascular Hemodynamics for the Clinician. John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781119066491.ch13.

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Conference papers on the topic "Valve tricuspide"

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Bhattacharya, Shamik, and Zhaoming He. "Tricuspid Valve Annulus Tension." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53088.

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Functional tricuspid regurgitation is a direct outcome of right ventricular dilatation and tricuspid annulus dilatation. The mechanism underlying functional tricuspid regurgitation is believed to be multifactorial and related to abnormalities in right ventricular volume, function and shape. Changes in the right ventricle geometry may lead to alterations in the positions of the papillary muscles (PM) of the tricuspid valve (TV). PM displacement happens in right ventricular dilatation but its correlation with tricuspid annulus dilatation is still unknown. The unique structure and orientation of tricuspid PM has role to play in TV annulus mechanics and right ventricular mechanics (Fig.1). It has been already shown that annulus tension (AT) is a parameter to evaluate left ventricular function that, previously, was evaluated via the left ventricular geometry and pressure [1–3].
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Govindarajan, V., J. Mousel, S. C. Vigmostad, et al. "Patient-Specific Valve Dynamics Using 3D Fluid-Structure Interaction Modeling: Comparison Between Bicuspid and Tricuspid Aortic Valves." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14563.

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Aortic valve diseases such as congenital bicuspid aortic valve (BAV) and progressive calcification in tricuspid valves affect the hemodynamics in the aortic arch. In addition to leaflet calcification, BAVs are associated with other ailments such as aortic coarctation, aneurysm and dissection [1]. It has also been observed that progressive calcification is accelerated in the case of BAVs compared to normal tricuspid valves. While it is not yet known whether the geometric distortion in BAVs is the main cause of calcification [2] in these valves, the distortion in the leaflets may give rise to altered stresses during the deformation processes which might play a role in accelerating the calcification process in BAVs. In addition, the altered flow caused by the change in geometry could alter the local fluid stresses during the opening phase, which might affect the endothelial lining of the aortic wall. Analyzing and comparing BAV and tricuspid aortic valves as a fluid-structure interaction problem will help determine the stress distribution on the leaflets during opening phase, and enable the examination of altered flow dynamics in the ascending aorta. In this study, the opening phase of a patient-specific bicuspid aortic valve is analyzed at physiological conditions and compared with the opening phase of a tricuspid aortic valve.
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Subbotina, I., M. Bernhardt, E. Girdauskas, C. Sinning, H. Reichenspurner, and B. Sill. "Outcome of Concomitant Tricuspid Valve Surgery in Patients with Mild or Moderate Tricuspid Valve Regurgitation." In 48th Annual Meeting German Society for Thoracic, Cardiac, and Vascular Surgery. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678892.

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Färber, G., J. Marx, M. Diab, and T. Doenst. "The Value of EuroSCORE II for Mortality Prediction in Isolated Tricuspid Valve Surgery." In 50th Annual Meeting of the German Society for Thoracic and Cardiovascular Surgery (DGTHG). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725760.

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Zhingre Sanchez, Jorge D., and Paul A. Iaizzo. "A Novel Transcatheter Edge-to-Edge Suturing Technique and Prototype for Repairing Tricuspid Valve Regurgitation." In 2020 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/dmd2020-9033.

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Abstract Tricuspid valve regurgitation is a major clinical issue that continues to attract interest from interventional cardiologists and medical device designers due to its rising prevalence and progressive nature. This disease impact is exacerbated among the aging population, considered as high risk of mortality for open-heart surgical procedures. Furthermore, early intervention for tricuspid regurgitation following left-sided heart procedures continues to increase. Thus, percutaneous or transcatheter interventions have emerged as the new frontier for tricuspid valve therapy. Specifically, tricuspid leaflet plication, or edge-to-edge repair, is a valvular procedure to enhance the coaptation of the leaflets and reduce regurgitation. The current landscape of approved transcatheter devices for leaflet coaptation are exclusive to the mitral valve or being investigated for tricuspid treatment. However, most of these transcatheter systems are designed with high procedure specificities, are expensive, and require extensive procedural training. Hence, there is an opportunity to percutaneously plicate the tricuspid leaflets using commonly available right-heart catheter equipment. This study details a novel transcatheter repair procedure that can plicate the tricuspid valve leaflets solely using current market released catheters and/or surgical equipment. Testing and evaluation of this prototype procedure was performed using Visible Heart® methodologies.
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Graf, H., F. Herrmann, P. Wellmann, S. Sadoni, C. Hagl, and G. Juchem. "Permanent Pacemaker Requirement after Tricuspid Valve Surgery." In 48th Annual Meeting German Society for Thoracic, Cardiac, and Vascular Surgery. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678950.

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Edriss, H., E. Karem, and H. Mohammadzadeh. "A Novel Endovascular Tricuspid Valve Endocarditis Management." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3399.

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Dolensky, Joseph R., Lauren D. C. Casa, and Ajit P. Yoganathan. "The Effect of Pulmonary Hypertension on Tricuspid Valve Coaptation in Normal and Pathologic Valve Geometries: An In Vitro Study." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80184.

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Pulmonary hypertension (PHTN) is a pathological condition defined as a mean pulmonary artery pressure (mPAP) greater than 25 mmHg. PHTN can result from a number of lung and heart pathologies, including abnormalities of the pulmonary vasculature, left heart disease, chronic lung disease, and chronic thrombotic disease [1]. Regardless of the cause, the increased afterload on the right heart results in right ventricle (RV) hypertrophy and dilatation and tricuspid regurgitation (TR) [2]. RV dilatation is thought to result in the displacement of the tricuspid valve (TV) papillary muscles (PM) and dilatation of the TV annulus, negatively impacting TV function.
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Zhingre Sanchez, Jorge D., Emma A. Schinstock, Michael G. Bateman, and Paul A. Iaizzo. "The Development and Testing of a Fixation Apparatus for Inducing the Coaptation of the Cardiac Atrioventricular Valves." In 2019 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/dmd2019-3298.

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As the prevalence of mitral and tricuspid valvular disease continues to grow with the aging population [1,2], there is a growing critical need to treat high mortality risk patients using minimally invasive and/or non-surgical percutaneous procedures. However, these transcatheter procedures, especially those aimed at repairing or replacing the mitral and tricuspid valves, are mostly still in development and/or early clinical testing. Catheter delivery, prosthesis fixation, and/or demonstrating device efficacy are major challenges currently being addressed [3,4]. Although in situ animal models can assess catheter systems with clinical imaging, direct visualization of tissue-device interactions in real human heart anatomies are desired. In vitro delivery and implantations of valvular prototypes in human heart specimens can be instrumental for accurate device testing and gaining important design insights. Such investigations can be performed on a pulsatile flow apparatus, utilizing perfusion fixed human hearts with mitral and/or tricuspid valves eliciting coaptation and relative function. The employment of endoscopic cameras provides direct visualization and can be coupled with echocardiography, providing novel insights relative to these transcatheter devices in a dynamic environment. However, these investigative approaches require appropriately fixed human heart specimens that will allow for dynamic valve movement. This study discusses the design, construction, and implementation of a novel fixation apparatus to promote the coaptation of the mitral and tricuspid valves in swine and fresh human heart specimen.
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Gavazzoni, M., A. Pozzoli, L. Vicentini, et al. "Single-Center Experience with Catheter-Based Tricuspid Valve Replacement with NaviGate Bioprosthesis for Tricuspid Regurgitation." In 48th Annual Meeting German Society for Thoracic, Cardiac, and Vascular Surgery. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678846.

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Reports on the topic "Valve tricuspide"

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Zhao, Xueshan, Kaibo Sun, Siwei Bi, and Zhong Wu. A systematic review and meta-analysis of aortic valve repair for bicuspid versus tricuspid aortic valves. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.12.0079.

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