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Books on the topic 'Valve tricuspide'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

Soliman, Osama I., and Folkert J. ten Cate, eds. Practical Manual of Tricuspid Valve Diseases. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-58229-0.

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2

Moorjani, Narain, Bushra S. Rana, and Francis C. Wells. Operative Mitral and Tricuspid Valve Surgery. Springer London, 2018. http://dx.doi.org/10.1007/978-1-4471-4204-1.

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3

Giamberti, Alessandro, and Massimo Chessa, eds. The Tricuspid Valve in Congenital Heart Disease. Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5400-4.

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4

Alain, Carpentier. Carpentier's reconstructive valve surgery. Saunders/Elsevier, 2010.

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5

1957-, Adams David H., and Filsoufi Farzan, eds. Carpentier's reconstructive valve surgery. Saunders/Elsevier, 2010.

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6

Warsi, Mohammed Ali. Ebstein anomaly of the tricuspid valve in an adult cohort. National Library of Canada, 1999.

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7

Pneumonia, malignant endocarditis of tricuspid valve. s.n., 1985.

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8

1941-, Rao P. Syamasundar, ed. Tricuspid atresia. 2nd ed. Futura, 1992.

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9

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Tricuspid valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0377_update_003.

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10

Muraru, Denisa, Ashraf M. Anwar, and Jae-Kwan Song. Heart valve disease: tricuspid valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0037.

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The tricuspid valve is currently the subject of much interest from echocardiographers and surgeons. Functional tricuspid regurgitation is the most frequent aetiology of tricuspid valve pathology, is characterized by structurally normal leaflets, and is due to annular dilation and/or leaflet tethering. A primary cause of tricuspid regurgitation with/without stenosis can be identified only in a minority of cases. Echocardiography is the imaging modality of choice for assessing tricuspid valve diseases. It enables the cause to be identified, assesses the severity of valve dysfunction, monitors the right heart remodelling and haemodynamics, and helps decide the timing for surgery. The severity assessment requires the integration of multiple qualitative and quantitative parameters. The recent insights from three-dimensional echocardiography have greatly increased our understanding about the tricuspid valve and its peculiarities with respect to the mitral valve, showing promise to solve many of the current problems of conventional two-dimensional imaging. This chapter provides an overview of the current state-of-the-art assessment of tricuspid valve pathology by echocardiography, including the specific indications, strengths, and limitations of each method for diagnosis and therapeutic planning.
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11

Anwar, Ashraf M., and Folkert Jan ten Cate. Tricuspid and pulmonary valves. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0016.

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Right-sided heart valves are complex anatomical structures. Studies describing the morphological and functional assessment of both valves are lacking. Most echocardiographic modalities provide a qualitative rather than quantitative approach.Echocardiography has a central role in the assessment of tricuspid regurgitation through estimation of severity, understanding the mechanism, assessment of pulmonary artery pressure, evaluation of right ventricular function, guidance towards surgery versus medical therapy, and assessment of valve competence after surgery.Transoesophageal echocardiography is an accurate method providing a qualitative assessment of right-sided heart valves. However, the lack of good validation makes it difficult to recommend its use for a quantitative approach. Hopefully, the future will provide refinements in instrumentation and techniques leading to increased accuracy in reporting and cost-effectiveness in making clinical decisions.
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12

Soliman, Osama I., and Folkert J. ten Cate. Practical Manual of Tricuspid Valve Diseases. Springer, 2017.

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13

Moorjani, Narain, Bushra S. Rana, and Francis C. Wells. Operative Mitral and Tricuspid Valve Surgery. Springer, 2019.

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14

Moorjani, Narain, Bushra S. Rana, and Francis C. Wells. Operative Mitral and Tricuspid Valve Surgery. Springer, 2019.

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15

Praz, Fabien, Stefan Stortecky, and Maurizio Taramasso, eds. Transcatheter Treatment of Tricuspid Valve Regurgitation. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-908-3.

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16

Waggoner, Alan, and Lori Green. Pulmonary Hypertension And Tricuspid Valve Disease. Gulfcoast Ultrasound Institute, 1998.

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17

Chessa, Massimo, and Alessandro Giamberti. The Tricuspid Valve in Congenital Heart Disease. Springer, 2014.

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18

Selton-Suty, Christine, Olivier Huttin, Clément Venner, and Yves Juillière. Functional anatomy of right heart valves. Edited by Yen Ho. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0019.

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This chapter summarizes the anatomical features of right heart valves. The tricuspid valve usually comprises three leaflets (anterior, septal, and posterior) separated by three commissures (anteroseptal, posteroseptal, and anteroposterior). The tricuspid valve apparatus consists of papillary muscles and chordae with high variability in number, length, shape, and arrangement of papillary muscles. The tricuspid valve leaflets are attached to a fibrous annulus with an ovoid saddle shape. The pulmonary valve and the aortic valve have identical architecture: three cusps (anterior, left, and right) separated by commissures, a fibrous ring, sinuses, and the sinotubular junction. Good knowledge of tricuspid and pulmonary valve functional anatomy is of clinical importance in the understanding of primary and secondary valvular dysfunction and in the development of new surgical and percutaneous therapeutic strategies.
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19

Transesophageal Echocardiography of the Tricuspid and Pulmonary Valves. Jaypee Brothers Medical Publishers (P) Ltd., 2014. http://dx.doi.org/10.5005/jp/books/12111.

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20

Rigo, Fausto, Covadonga Fernández-Golfín, and Bruno Pinamonti. Familial cardiomyopathies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0047.

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The tricuspid valve is currently the subject of much interest from echocardiographers and surgeons. Functional tricuspid regurgitation is the most frequent aetiology of tricuspid valve pathology, is characterized by structurally normal leaflets, and is due to annular dilation and/or leaflet tethering. A primary cause of tricuspid regurgitation with/without stenosis can be identified only in a minority of cases. Echocardiography is the imaging modality of choice for assessing tricuspid valve diseases. It enables the cause to be identified, assesses the severity of valve dysfunction, monitors the right heart remodelling and haemodynamics, and helps decide the timing for surgery. The severity assessment requires the integration of multiple qualitative and quantitative parameters. The recent insights from three-dimensional echocardiography have greatly increased our understanding about the tricuspid valve and its peculiarities with respect to the mitral valve, showing promise to solve many of the current problems of conventional two-dimensional imaging. This chapter provides an overview of the current state-of-the-art assessment of tricuspid valve pathology by echocardiography, including the specific indications, strengths, and limitations of each method for diagnosis and therapeutic planning.
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21

Bianchi, Giacomo. Rediscovered Tricuspid Valve: Structure, Function and Clinical Significance in Health and Disease. Nova Science Publishers, Incorporated, 2019.

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22

Henderson, Deborah J., Bill Chaudhry, and José Luis de la Pompa. Development of the arterial valves. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0018.

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The arterial valves guarding the entrances to the aorta and pulmonary trunk have many similarities to the mitral and tricuspid valves in the atrioventricular region of the heart. Despite these similarities, there are significant differences in the formation and structure of the arterial and atrioventricular valves. The most fundamental of these relate to the lineage origins of the cells forming the primitive cushions. Although the fate of the different lineages remains unclear, each makes a permanent contribution to the mature valve. Arterial valve formation is intrinsically linked to cushion formation and outflow tract septation; therefore abnormalities in these processes have a profound impact on development of the valve leaflets. In this chapter we highlight the main differences in the development and structure of the arterial valves, compared with the atrioventricular valves, show how abnormalities in these developmental processes can result in arterial valve anomalies, and discuss controversies within the literature.
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23

Rahimi, Kazem. Miscellaneous valvar pathology: Mitral stenosis, pulmonary stenosis, and tricuspid regurgitation. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0096.

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Mitral stenosis is obstruction to inflow of blood from left atrium to left ventricle at the level of the mitral valve. Non-valvar causes of left ventricular inflow obstruction include left atrial tumours and cor triatriatum.
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24

Archer, Nick, and Nicky Manning. Right-sided abnormalities. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198766520.003.0011.

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This chapter explores right-sided abnormalities, discussing the venoatrial junction (including both azygous/hemiazygous connections and abnormal systemic venous drainage), the atrioventricular junction (including tricuspid atresia and Ebstein’s anomaly/tricuspid valve dysplasia), and the ventriculoarterial junction (including pulmonary stenosis, pulmonary atresia with intact septum, tetralogy of Fallot, tetralogy of Fallot with absent pulmonary valve, pulmonary atresia with ventricular septal defect, and double outlet right ventricle), and arterial abnormalities.
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25

Tourneau, Thierry Le, Luis Caballero, and Tsai Wei-Chuan. Right atrium. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0024.

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The right atrium (RA) is located on the upper right-hand side of the heart and has relatively thin walls. From an anatomical point of view, the RA comprises three basic parts, the appendage, the vestibule of the tricuspid valve, and the venous component (superior and inferior vena cava, and the coronary sinus) receiving the deoxygenated blood. The RA is a dynamic structure dedicated to receive blood and to assist right ventricular (RV) filling. The three components of atrial function are the reservoir function during ventricular systole, the conduit function which consists in passive blood transfer from veins to the RV in diastole, and the booster pump function in relation to atrial contraction in late diastole to complete ventricular filling. Right atrial function depends on cardiac rhythm (sinus or atrial fibrillation), pericardial integrity, RV load and function, and tricuspid function. Right atrial dimension assessment is limited in two-dimensional (2D) echocardiography. Right atrial planimetry in the apical four-chamber view is commonly used with an upper normal value of 18-20 cm2. Minor and major diameters can also be measured. Three-dimensional (3D) echocardiography could overcome the limitation of conventional echocardiography in assessing RA size. Right atrial function has been poorly explored by echocardiography both in physiological and pathological contexts. Although tricuspid inflow and tissue Doppler imaging of tricuspid annulus can be used in the exploration of RA function, 2D speckle tracking and 3D echocardiography appear promising tools to dissect RA function and to overcome the limitations of standard echocardiography.
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26

Lancellotti, Patrizio, and Bernard Cosyns. Adult Congenital Heart Disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713623.003.0013.

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Echocardiography has a fundamental role in patients with adult congenital heart disease. This chapter identifies the role of echocardiography in atrial septal defects, ventricular septal defects, atrioventricular septal defects, patent ductus arteriosus, and persistent left superior vena cava. For each condition, the role of transthoracic and transoesophagael echocardiogram are shown alongside examples of main types and features and haemodynamic effect. Echocardiographic findings of LV outflow tract obstruction, supravalvular aortic stenosis, aortic stenosis, and aortic coarction are covered, as well as an examination of complex congenital lesions, including the tetralogy of Fallot and Ebstein’s anomaly of the tricuspid valve.
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27

Orchard, Liz. Congenital heart disease in adults. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0088.

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Congenital heart disease is the most common congenital abnormality, affecting 0.8% of births. There have been major advances in both the surgical and interventional treatment of congenital heart disease, with about 85% of patients now surviving into adulthood. Congenital heart disease can be divided into simple and complex lesions. Simple lesions include atrial septal defect, ventricular septal defect, patent ductus arteriosus, coarctation of the aorta, and left ventricular outflow tract lesions. Complex lesions include tetralogy of Fallot, complete transposition of great arteries, single ventricle/Fontan physiology, pulmonary atresia, and Ebstein’s anomaly of the tricuspid valve. This chapter reviews the clinical features and management of those congenital cardiac disorders seen in adolescents and adults.
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28

Badano, Luigi P., and Denisa Muraru. Assessment of right heart function and haemodynamics. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0011.

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Assessment of right ventricular (RV) size, function, and haemodynamics has been challenging because of its unique cavity geometry. Conventional two-dimensional assessment of RV function is often qualitative. Doppler methods involving tricuspid inflow and pulmonary artery flow velocities, which are influenced by changes in pre- and afterload conditions, may not provide robust prognostic information for clinical decision making. Recent advances in echocardiographic assessment of the RV include tissue Doppler imaging, speckle-tracking imaging, and volumetric three-dimensional imaging, but they need specific training, expensive dedicated equipment, and extensive clinical validation. However, assessment of RV function is crucial, especially in patients with signs of right-sided failure and those with congenital or mitral valve diseases. This chapter aims to address the role of the various echocardiographic modalities used to assess RV and pulmonary vascular bed function. Special emphasis has been placed on technical considerations, limitations, and pitfalls of image acquisition and analysis.
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29

Jones, Michael, Norman Qureshi, and Kim Rajappan. Atrial flutter. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0117.

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Atrial flutter is the term given to one of the four types of supraventricular tachycardia; in it, atrial activation occurs as a consequence of a continuous ‘short circuit’: a defined and fixed anatomical route, resulting in a fairly uniform atrial rate, and uniform atrial flutter waves on the ECG. The ventricles are not a part of this arrhythmia circuit, and ventricular activation is variable, dependent on atrioventricular (AV) nodal conduction. Given that the atrial rate is essentially uniform (e.g. 300 min−1), ventricular activation tends to be regular (i.e. 150 min−1, 100 min−1, 75 min−1, etc., if the atrial rate is 300 mins−1), or regularly irregular if changes are occurring in the fraction of conducted impulses to the ventricles. When AV nodal conduction permits only 4:1 conduction or less, atrial flutter is usually obvious, but when ventricular rates are higher (150 min−1 or more) the flutter waves can be obscured by the QRS complexes, making diagnosis more difficult. Atrial flutter is of two types, typical and atypical. Typical atrial flutter is a right atrial tachycardia, with electrical activation proceeding around the tricuspid valve annulus. This arrhythmia is dependent on a zone of slow electrical conduction through the cavotricuspid isthmus (the tissue lying between the origin of the inferior vena cava and the posterior tricuspid valve). The resulting circuit can be either anticlockwise (activation proceeds up the inter-atrial septum, across the atrial roof, down the free wall, and then through the cavotricuspid isthmus to the basal septum) or clockwise (down the inter-atrial septum and around the circuit in the opposite direction). Anticlockwise typical atrial flutter is more common. Atypical atrial flutter refers to all other atrial flutters, and this includes other right atrial flutters (e.g. pericristal flutter), left atrial flutters, post-ablation or post-surgical flutters, and pulmonary vein flutters. The feature common to all types of flutter and which differentiates flutter from other types of supraventricular tachycardia is the presence of a macro-re-entrant anatomical circuit around which the electrical impulse travels continuously and repeatedly, thereby generating the flutter. Even though typical atrial flutter has a fairly obvious and specific appearance on the ECG, atypical flutters do not, and often it is only possible to differentiate atypical flutter from atrial tachycardias by invasive electrophysiology studies, as the ECG alone may be insufficient.
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30

Rigo, Fausto, Covadonga Fernández-Golfín, and Bruno Pinamonti. Dilated cardiomyopathy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0043.

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Dilated cardiomyopathy (DCM) is characterized by a globally dilated and dysfunctioning left ventricle (LV). Therefore, echocardiographic diagnostic criteria for DCM are a LV end-diastolic diameter greater than 117% predicted value corrected for age and body surface area and a LV ejection fraction less than 45% (and/or fractional shortening less than 25%). Usually, the LV is also characterized by a normal or mildly increased wall thickness with eccentric hypertrophy and increased mass, a spherical geometry (the so-called LV remodelling), a dyssynchronous contraction (typically with left bundle branch block), and diastolic dysfunction with elevated LV filling pressure. Other typical echocardiographic features of DCM include functional mitral and tricuspid regurgitation, right ventricular dysfunction, atrial dilatation, and secondary pulmonary hypertension. Several echocardiographic parameters, measured both at baseline and at follow-up, are valuable for prognostic stratification of DCM patients. Furthermore, re-evaluation of echocardiographic parameters during the disease course under optimal medical therapy is valuable for tailoring medical treatment and confirming indications for invasive treatments at follow-up. The stress echo can play a pivotal role in the different phases of DCM helping us in stratifying the prognosis of these patients. Finally, familial screening is an important tool for early diagnosis of DCM in asymptomatic patients.
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