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Books on the topic 'Valve tricuspide'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Soliman, Osama I., and Folkert J. ten Cate, eds. Practical Manual of Tricuspid Valve Diseases. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-58229-0.

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2

Moorjani, Narain, Bushra S. Rana, and Francis C. Wells. Operative Mitral and Tricuspid Valve Surgery. Springer London, 2018. http://dx.doi.org/10.1007/978-1-4471-4204-1.

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3

Giamberti, Alessandro, and Massimo Chessa, eds. The Tricuspid Valve in Congenital Heart Disease. Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5400-4.

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4

Alain, Carpentier. Carpentier's reconstructive valve surgery. Saunders/Elsevier, 2010.

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5

1957-, Adams David H., and Filsoufi Farzan, eds. Carpentier's reconstructive valve surgery. Saunders/Elsevier, 2010.

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6

Warsi, Mohammed Ali. Ebstein anomaly of the tricuspid valve in an adult cohort. National Library of Canada, 1999.

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7

Pneumonia, malignant endocarditis of tricuspid valve. s.n., 1985.

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8

1941-, Rao P. Syamasundar, ed. Tricuspid atresia. 2nd ed. Futura, 1992.

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9

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Tricuspid valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0377_update_003.

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10

Muraru, Denisa, Ashraf M. Anwar, and Jae-Kwan Song. Heart valve disease: tricuspid valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0037.

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The tricuspid valve is currently the subject of much interest from echocardiographers and surgeons. Functional tricuspid regurgitation is the most frequent aetiology of tricuspid valve pathology, is characterized by structurally normal leaflets, and is due to annular dilation and/or leaflet tethering. A primary cause of tricuspid regurgitation with/without stenosis can be identified only in a minority of cases. Echocardiography is the imaging modality of choice for assessing tricuspid valve diseases. It enables the cause to be identified, assesses the severity of valve dysfunction, monitors th

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11

Anwar, Ashraf M., and Folkert Jan ten Cate. Tricuspid and pulmonary valves. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0016.

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Right-sided heart valves are complex anatomical structures. Studies describing the morphological and functional assessment of both valves are lacking. Most echocardiographic modalities provide a qualitative rather than quantitative approach.Echocardiography has a central role in the assessment of tricuspid regurgitation through estimation of severity, understanding the mechanism, assessment of pulmonary artery pressure, evaluation of right ventricular function, guidance towards surgery versus medical therapy, and assessment of valve competence after surgery.Transoesophageal echocardiography is

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12

Soliman, Osama I., and Folkert J. ten Cate. Practical Manual of Tricuspid Valve Diseases. Springer, 2017.

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13

Moorjani, Narain, Bushra S. Rana, and Francis C. Wells. Operative Mitral and Tricuspid Valve Surgery. Springer, 2019.

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14

Moorjani, Narain, Bushra S. Rana, and Francis C. Wells. Operative Mitral and Tricuspid Valve Surgery. Springer, 2019.

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15

Praz, Fabien, Stefan Stortecky, and Maurizio Taramasso, eds. Transcatheter Treatment of Tricuspid Valve Regurgitation. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-908-3.

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16

Waggoner, Alan, and Lori Green. Pulmonary Hypertension And Tricuspid Valve Disease. Gulfcoast Ultrasound Institute, 1998.

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17

Chessa, Massimo, and Alessandro Giamberti. The Tricuspid Valve in Congenital Heart Disease. Springer, 2014.

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18

Selton-Suty, Christine, Olivier Huttin, Clément Venner, and Yves Juillière. Functional anatomy of right heart valves. Edited by Yen Ho. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0019.

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This chapter summarizes the anatomical features of right heart valves. The tricuspid valve usually comprises three leaflets (anterior, septal, and posterior) separated by three commissures (anteroseptal, posteroseptal, and anteroposterior). The tricuspid valve apparatus consists of papillary muscles and chordae with high variability in number, length, shape, and arrangement of papillary muscles. The tricuspid valve leaflets are attached to a fibrous annulus with an ovoid saddle shape. The pulmonary valve and the aortic valve have identical architecture: three cusps (anterior, left, and right)

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19

Transesophageal Echocardiography of the Tricuspid and Pulmonary Valves. Jaypee Brothers Medical Publishers (P) Ltd., 2014. http://dx.doi.org/10.5005/jp/books/12111.

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20

Rigo, Fausto, Covadonga Fernández-Golfín, and Bruno Pinamonti. Familial cardiomyopathies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0047.

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The tricuspid valve is currently the subject of much interest from echocardiographers and surgeons. Functional tricuspid regurgitation is the most frequent aetiology of tricuspid valve pathology, is characterized by structurally normal leaflets, and is due to annular dilation and/or leaflet tethering. A primary cause of tricuspid regurgitation with/without stenosis can be identified only in a minority of cases. Echocardiography is the imaging modality of choice for assessing tricuspid valve diseases. It enables the cause to be identified, assesses the severity of valve dysfunction, monitors th

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21

Bianchi, Giacomo. Rediscovered Tricuspid Valve: Structure, Function and Clinical Significance in Health and Disease. Nova Science Publishers, Incorporated, 2019.

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22

Henderson, Deborah J., Bill Chaudhry, and José Luis de la Pompa. Development of the arterial valves. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0018.

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The arterial valves guarding the entrances to the aorta and pulmonary trunk have many similarities to the mitral and tricuspid valves in the atrioventricular region of the heart. Despite these similarities, there are significant differences in the formation and structure of the arterial and atrioventricular valves. The most fundamental of these relate to the lineage origins of the cells forming the primitive cushions. Although the fate of the different lineages remains unclear, each makes a permanent contribution to the mature valve. Arterial valve formation is intrinsically linked to cushion

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23

Rahimi, Kazem. Miscellaneous valvar pathology: Mitral stenosis, pulmonary stenosis, and tricuspid regurgitation. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0096.

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Mitral stenosis is obstruction to inflow of blood from left atrium to left ventricle at the level of the mitral valve. Non-valvar causes of left ventricular inflow obstruction include left atrial tumours and cor triatriatum.

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24

Archer, Nick, and Nicky Manning. Right-sided abnormalities. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198766520.003.0011.

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This chapter explores right-sided abnormalities, discussing the venoatrial junction (including both azygous/hemiazygous connections and abnormal systemic venous drainage), the atrioventricular junction (including tricuspid atresia and Ebstein’s anomaly/tricuspid valve dysplasia), and the ventriculoarterial junction (including pulmonary stenosis, pulmonary atresia with intact septum, tetralogy of Fallot, tetralogy of Fallot with absent pulmonary valve, pulmonary atresia with ventricular septal defect, and double outlet right ventricle), and arterial abnormalities.

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25

Tourneau, Thierry Le, Luis Caballero, and Tsai Wei-Chuan. Right atrium. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0024.

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The right atrium (RA) is located on the upper right-hand side of the heart and has relatively thin walls. From an anatomical point of view, the RA comprises three basic parts, the appendage, the vestibule of the tricuspid valve, and the venous component (superior and inferior vena cava, and the coronary sinus) receiving the deoxygenated blood. The RA is a dynamic structure dedicated to receive blood and to assist right ventricular (RV) filling. The three components of atrial function are the reservoir function during ventricular systole, the conduit function which consists in passive blood tra

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26

Lancellotti, Patrizio, and Bernard Cosyns. Adult Congenital Heart Disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713623.003.0013.

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Echocardiography has a fundamental role in patients with adult congenital heart disease. This chapter identifies the role of echocardiography in atrial septal defects, ventricular septal defects, atrioventricular septal defects, patent ductus arteriosus, and persistent left superior vena cava. For each condition, the role of transthoracic and transoesophagael echocardiogram are shown alongside examples of main types and features and haemodynamic effect. Echocardiographic findings of LV outflow tract obstruction, supravalvular aortic stenosis, aortic stenosis, and aortic coarction are covered,

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27

Orchard, Liz. Congenital heart disease in adults. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0088.

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Congenital heart disease is the most common congenital abnormality, affecting 0.8% of births. There have been major advances in both the surgical and interventional treatment of congenital heart disease, with about 85% of patients now surviving into adulthood. Congenital heart disease can be divided into simple and complex lesions. Simple lesions include atrial septal defect, ventricular septal defect, patent ductus arteriosus, coarctation of the aorta, and left ventricular outflow tract lesions. Complex lesions include tetralogy of Fallot, complete transposition of great arteries, single vent

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28

Badano, Luigi P., and Denisa Muraru. Assessment of right heart function and haemodynamics. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0011.

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Assessment of right ventricular (RV) size, function, and haemodynamics has been challenging because of its unique cavity geometry. Conventional two-dimensional assessment of RV function is often qualitative. Doppler methods involving tricuspid inflow and pulmonary artery flow velocities, which are influenced by changes in pre- and afterload conditions, may not provide robust prognostic information for clinical decision making. Recent advances in echocardiographic assessment of the RV include tissue Doppler imaging, speckle-tracking imaging, and volumetric three-dimensional imaging, but they ne

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29

Jones, Michael, Norman Qureshi, and Kim Rajappan. Atrial flutter. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0117.

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Atrial flutter is the term given to one of the four types of supraventricular tachycardia; in it, atrial activation occurs as a consequence of a continuous ‘short circuit’: a defined and fixed anatomical route, resulting in a fairly uniform atrial rate, and uniform atrial flutter waves on the ECG. The ventricles are not a part of this arrhythmia circuit, and ventricular activation is variable, dependent on atrioventricular (AV) nodal conduction. Given that the atrial rate is essentially uniform (e.g. 300 min−1), ventricular activation tends to be regular (i.e. 150 min−1, 100 min−1, 75 min−1, e

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30

Rigo, Fausto, Covadonga Fernández-Golfín, and Bruno Pinamonti. Dilated cardiomyopathy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0043.

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Dilated cardiomyopathy (DCM) is characterized by a globally dilated and dysfunctioning left ventricle (LV). Therefore, echocardiographic diagnostic criteria for DCM are a LV end-diastolic diameter greater than 117% predicted value corrected for age and body surface area and a LV ejection fraction less than 45% (and/or fractional shortening less than 25%). Usually, the LV is also characterized by a normal or mildly increased wall thickness with eccentric hypertrophy and increased mass, a spherical geometry (the so-called LV remodelling), a dyssynchronous contraction (typically with left bundle

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