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Books on the topic 'Vascular cell adhesion molecule'

Author: Grafiati

Published: 28 May 2022

Last updated: 30 July 2025

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1

D, Pearson Jeremy, ed. Vascular adhesion molecules and inflammation. Birkhäuser, 1999.

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2

D, Pearson Jeremy, ed. Vascular adhesion molecules and inflammation. Birkhäuser, 1999.

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3

L, Gordon J., ed. Vascular endothelium: Interactions with circulating cells. Elsevier, 1991.

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4

Bereta, Joanna. Regulation of expression of vascular cell adhesion molecule-1 and inducible nitric oxide synthase in murine brain microvascular endothelium. Wydawn. Uniwersytetu Jagiellońskiego, 1997.

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5

Dominique, Bagnard, ed. Neuropilin: From nervous system to vascular and tumor biology. Kluwer Academic/Plenum Pub., 2002.

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6

Dominique, Bagnard, ed. Neuropilin: From nervous system to vascular and tumor biology. Kluwer Academic/Plenum Pub., 2002.

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7

Dominique, Bagnard, ed. Neuropilin: From nervous system to vascular and tumor biology. Kluwer Academic/Plenum Pub., 2002.

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8

Gallagher, Helen Christine. Regulation of neural cell adhesion molecule polysialylation state. University College Dublin, 1998.

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9

Berezin, V. A. Structure and function of the neural cell adhesion molecule NCAM. Springer, 2010.

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10

Berezin, Vladimir, ed. Structure and Function of the Neural Cell Adhesion Molecule NCAM. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-1170-4.

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11

Kunz, Beat. Function blocking monoclonal antibodies against the neural cell adhesion molecule axonin-1. [s.n.], 1996.

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12

Ma, Patrick Chi-Chung. Mechanism of developmental regulation of the cell adhesion molecule gp80 in Dictyostelium discoideum. National Library of Canada, 1990.

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13

Fox, Gerard B. Behavioural, functional and pharmacological modulation of rodent neural cell adhesion molecule mediated neuroplasticity. University College Dublin, 1995.

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14

Kulka, Irena. Developmental changes of neural cell adhesion molecule (N-CAM) in the chick retino-tectal projection. [s.n.], 1990.

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15

Zhao, Xiaoning. Identification and characterization of a homophilic binding and neuritogenic site in the cell adhesion molecule L1. National Library of Canada = Bibliothèque nationale du Canada, 1998.

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16

Nualláin, Brian Ó. An investigation of ligand interactions with apoflavodoxin from Desulfovibrio vulgaris and the cell adhesion molecule LFA-3. University College Dublin, 1998.

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17

Vascular Adhesion Molecules and Inflammation (Pir (Series).). Birkhauser, 2000.

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18

Bagnard, Dominique. Neuropilin: From Nervous System to Vascular and Tumor Biology. Springer, 2012.

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19

Bagnard, Dominique. Neuropilin: From Nervous System to Vascular and Tumor Biology. Springer London, Limited, 2011.

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20

Neuropilin: From Nervous System to Vascular and Tumor Biology. Springer, 2012.

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21

Neuropilin: From Nervous System to Vascular and Tumor Biology (Advances in Experimental Medicine and Biology). Springer, 2007.

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22

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0040.

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Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the extracellular matrix and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and further transmigration across

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23

LAMBERT M. TIMPLEDON, MIRIAM T. MARSEKEN, SUSAN F. SURHONE. NEURAL CELL ADHESION MOLECULE. BETASCRIPT PUBLISHING, 2010.

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24

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0040_update_001.

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Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the intimal layer and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles attached to the extracellular matrix suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and

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25

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0040_update_002.

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Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the intimal layer and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles attached to the extracellular matrix suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and

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26

Power, Christine, and Rod Pigott. The Adhesion Molecule Factsbook. Academic Pr, 1993.

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27

Alharbi, Yousef, Manish S. Patankar, and Rebecca J. Whelan. Antibody-Based Therapy for Ovarian Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0006.

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With their role in connecting disease-associated antigens to the cellular immune response, antibodies hold considerable promise as therapeutic agents. This chapter discusses three classes of therapeutic antibodies that have been developed for use in ovarian cancer therapy. The first includes antibodies selected against tumor-associated antigens such as MUC16/CA125, mesothelin, epithelial cell adhesion molecule, and folate receptor α‎. Antibodies in the second class target proteins such as CTLA-4 and PD1 that act as immune response checkpoint receptors. The third class of antibodies target secr

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28

Power, Christine, and Rod Pigott. The Adhesion Molecule Factsbook. Academic Pr, 1993.

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29

Tsai, Ching-Wei, Sanjeev Noel, and Hamid Rabb. Pathophysiology of Acute Kidney Injury, Repair, and Regeneration. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0030.

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Acute kidney injury (AKI), regardless of its aetiology, can elicit persistent or permanent kidney tissue changes that are associated with progression to end-stage renal disease and a greater risk of chronic kidney disease (CKD). In other cases, AKI may result in complete repair and restoration of normal kidney function. The pathophysiological mechanisms of renal injury and repair include vascular, tubular, and inflammatory factors. The initial injury phase is characterized by rarefaction of peritubular vessels and engagement of the immune response via Toll-like receptor binding, activation of

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30

(Editor), Clare Isacke, and Michael A. Horton (Editor), eds. The Adhesion Molecule: FactsBook (Factsbook). 2nd ed. Academic Press, 2000.

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31

(Editor), Clare Isacke, and Michael A. Horton (Editor), eds. The Adhesion Molecule: FactsBook (Factsbook). Academic Press, 2000.

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32

Berezin, Vladimir. Structure and Function of the Neural Cell Adhesion Molecule Ncam. Springer, 2010.

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33

Structure and function of the neural cell adhesion molecule NCAM. Springer, 2010.

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34

Heiz, Monika. Regulation of ectodomain shedding of the L1 cell adhesion molecule. 2004.

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35

Rao, Yong. Molecular analysis of homophilic interactions of the neural cell adhesion molecule NCAM. 1994.

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36

Desbarats, Laurie Ann. The regulation of expression of the cell adhesion molecule gp80 in Dictyostelium discoideum. 1994.

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37

Wong, Lu Min. Molecular cloning and characterization of the cell adhesion molecule gp80 of dictyostelium discoideum. 1988.

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38

Jun, Wang. Structural and functional characterization of the cell adhesion molecule GP150 in dictyostelium discoideum. 2002.

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39

Wong, Estella F. S. Characterization of the calcium-dependent cell adhesion molecule DdCAD-1 in Dictyostelium discoideum. 2002.

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40

Brar, Simuran Kaur. The purification, cloning and characterization of the cell adhesion molecule gp24 in Dictyostelium discoideum. 1995.

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41

Levi, Marcel, and Tom van der Poll. Coagulation and the endothelium in acute injury in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0307.

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Vascular endothelial cells play a pivotal mediatory role in many responses to systemic inflammation, including the cross-talk between coagulation and inflammation in sepsis. Endothelial cells respond to the cytokines expressed and released by activated leukocytes, but can also release cytokines themselves. Furthermore, endothelial cells are able to express adhesion molecules and growth factors that may not only promote the inflammatory response further, but also affect a myriad of downstream responses. It has recently become clear that, in addition to these mostly indirect effects of the endot

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42

Berezin, Vladimir. Structure and Function of the Neural Cell Adhesion Molecule NCAM: Advances in Experimental Medicine and Biology. Springer, 2012.

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43

Mohajeri, Hasan. Ectopic expression of the neural cell adhesion molecule L1 in differentiated CNS glial cells of adult mice. 1993.

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44

Appel, Frank Burkhard Bernhard. Importance of the immunoglobulin-like domains and the fibronectin type III homologous repeats of the neural cell adhesion molecule L1 for neurite outgrowth, cell body adhesion and signal transduction. 1993.

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45

Stork, Oliver. Roles of the neural cell adhesion molecule (NCAM) and its synapse-specific isoform (NCAM 180) in information processing in the central nervous system and in control of behaviour. 1997.

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46

Xu, Yaping. The Expression of matrix metalloproteinase -2, -9, tissue inhibitor-1 of matrix metalloproteinases, cell adhesion molecule CD44 variant 6, oncogene HER2/neu and tumor-supressor gene p53 in head and neck squamous cell carcinoma and their clinical relevance. 2001.

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