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1

O'DWYER, J. P., J. E. KING, C. E. WOOD, B. L. TAYLOR, and G. B. SMITH. "Continuous measurement of systemic vascular resistance." Anaesthesia 49, no. 7 (February 22, 2007): 587–90. http://dx.doi.org/10.1111/j.1365-2044.1994.tb14225.x.

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2

Abbas, Amr E., F. David Fortuin, Bhavesh Patel, Carlos A. Moreno, Nelson B. Schiller, and Steven J. Lester. "Noninvasive measurement of systemic vascular resistance using Doppler echocardiography." Journal of the American Society of Echocardiography 17, no. 8 (August 2004): 834–38. http://dx.doi.org/10.1016/j.echo.2004.04.008.

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3

Schrijen, F., C. Saunier, and F. Chabot. "Peripheral pulmonary vascular resistance." Journal of Applied Physiology 74, no. 2 (February 1, 1993): 613–16. http://dx.doi.org/10.1152/jappl.1993.74.2.613.

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The pressure-flow relationship has been studied in a peripheral portion of the lung vasculature in anesthetized dogs with use of a double-lumen catheter wedged in a distal pulmonary artery. One lumen was used to infuse mixed venous blood in the wedged area and the other to measure the corresponding perfusion pressure. Flow ranged from 0 to 9.2 ml/min, and the mean volume of the wedged area (n = 59) was 0.75 +/- 0.05 (SE) ml. In the areas where the distal pulmonary artery was in the same direction as the catheter ("coaxial"), the mean pressure-flow curve showed a negligible gamma-intercept and
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4

Moriyasu, Fuminori, Osamu Nishida, Nobuyuki Ban, Takefumi Nakamura, Kensuke Miura, Masahiko Sakai, Takeo Miyake, and Haruto Uchino. "Measurement of Portal Vascular Resistance in Patients With Portal Hypertension." Gastroenterology 90, no. 3 (March 1986): 710–17. http://dx.doi.org/10.1016/0016-5085(86)91127-3.

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5

Lautt, W. W., C. V. Greenway, D. J. Legare, and H. Weisman. "Localization of intrahepatic portal vascular resistance." American Journal of Physiology-Gastrointestinal and Liver Physiology 251, no. 3 (September 1, 1986): G375—G381. http://dx.doi.org/10.1152/ajpgi.1986.251.3.g375.

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The pressure drop from the portal vein to the vena cava occurs primarily across a postsinusoidal site localized to a narrow segment (less than 0.5 cm) of hepatic veins (roughly 1.5 mm diam) in the anesthetized cat. Portal venous pressure (PVP = 8.9 +/- 0.3 mmHg) and lobar hepatic venous pressure (LVP = 8.7 +/- 0.4 mmHg) are insignificantly different, and pressure changes imposed from the presinusoidal or postsinusoidal side are equally transmitted to both pressure sites. Several types of experiments were done to validate the LVP measurement. The portal vein, hepatic sinusoids, and hepatic vein
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6

Allison, R. C., B. Rippe, V. R. Prasad, J. C. Parker, and A. E. Taylor. "Pulmonary vascular permeability and resistance measurements in control and ANTU-injured dog lungs." American Journal of Physiology-Heart and Circulatory Physiology 256, no. 6 (June 1, 1989): H1711—H1718. http://dx.doi.org/10.1152/ajpheart.1989.256.6.h1711.

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Because questions have arisen regarding pulmonary vascular permeability and resistance measurements in isolated, perfused lungs, we sought to determine the 1) stability of repeated measurements of permeability and resistance in control lungs; and 2) magnitude of change in these measurements when permeability was greatly increased. Using blood-perfused dog lungs, we measured filtration coefficient (Kf) and isogravimetric capillary pressure (Pci) as indexes of vascular permeability, and we also determined total vascular resistance (Rt) as well as the segmental resistances using the double-occlus
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7

Parker, James C., Mark N. Gillespie, Aubrey E. Taylor, and Sherri L. Martin. "Capillary filtration coefficient, vascular resistance, and compliance in isolated mouse lungs." Journal of Applied Physiology 87, no. 4 (October 1, 1999): 1421–27. http://dx.doi.org/10.1152/jappl.1999.87.4.1421.

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Although many recently produced transgenic mice possess gene alterations affecting pulmonary vascular function, there are few baseline measurements of vascular resistance and permeability. Therefore, we excised the lungs of C57/BL6 mice and perfused them with 5% bovine serum albumin in RPMI-1640 culture medium at a nominal flow of 0.5 ml/min and ventilated them with 20% O2-5% CO2-75% N2. The capillary filtration coefficient, a sensitive measurement of hydraulic conductivity, was unchanged over 2 h (0.33 ± 0.03 ml ⋅ min−1 ⋅ cmH2O−1 ⋅ 100 g−1) in a control group ventilated with low peak inflatio
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8

Giesen, Leonie A., Michelle White, and Robert M. R. Tulloh. "Comparison of the effect of inhaled anaesthetic with intravenous anaesthetic on pulmonary vascular resistance measurement during cardiac catheterisation." Cardiology in the Young 25, no. 2 (February 19, 2014): 368–72. http://dx.doi.org/10.1017/s1047951114000195.

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AbstractBackground: Children with pulmonary hypertension routinely undergo pulmonary vascular resistance studies to assess the disease severity and vasodilator responsiveness. It is vital that results are accurate and reliable and are not influenced by the choice of anaesthetic agent. However, there are anecdotal data to suggest that propofol and inhalational agents have different effects on pulmonary vascular resistance. Methods: A total of 10 children with pulmonary hypertension were selected sequentially to be included in the study. To avoid confounding because of baseline anatomic or demog
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9

Parvin, S. D., D. H. Evans, and P. R. F. Bell. "Peripheral resistance measurement in the assessment of severe peripheral vascular disease." British Journal of Surgery 72, no. 9 (September 1985): 751–53. http://dx.doi.org/10.1002/bjs.1800720928.

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10

Rothe, C. F., and R. Maass-Moreno. "Gastrointestinal hemodynamics during compensation for hemorrhage and measurement of Pmcf." American Journal of Physiology-Heart and Circulatory Physiology 266, no. 3 (March 1, 1994): H1242—H1250. http://dx.doi.org/10.1152/ajpheart.1994.266.3.h1242.

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To quantify the degree of autonomic reflex control of the gastrointestinal vasculature, we studied the responses to a 10-ml/kg hemorrhage or transfusion and autonomic blockade in fentanyl- and pentobarbital-anesthetized dogs. The active total blood volume was estimated by indocyanine green dilution. Transfusion and hemorrhage did not significantly change gastrointestinal vascular compliance [1.82 +/- 0.68 (SD) ml/mmHg], but autonomic blockade with hexamethonium and atropine increased it by 0.57 +/- 0.37 ml/mmHg. Neither hemorrhage nor autonomic blockade significantly changed gastrointestinal v
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11

Bishop, R., D. McLeod, S. McIlveen, R. Blake, R. Gunther, J. Davis, L. Talken, et al. "Long-Term Measurement of Bronchial Vascular Resistance in Awake Sheep and Dogs." Archives of Physiology and Biochemistry 111, no. 4 (January 2003): 315–16. http://dx.doi.org/10.3109/13813450312331337478.

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12

R., Bishop, McLeod D., McIlveen S., Blake R., Gunther R., Davis J., Talken L., et al. "Long-Term Measurement of Bronchial Vascular Resistance in Awake Sheep and Dogs." Archives of Physiology and Biochemistry 111, no. 4 (April 1, 2003): 315–16. http://dx.doi.org/10.1080/13813450312331337478.

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13

Tinkanen, Helena, and Erkki Kujansuu. "The Reproducibility of the Doppler Ultrasound Measurement of Uterine Artery Vascular Resistance." Gynecologic and Obstetric Investigation 39, no. 3 (1995): 188–91. http://dx.doi.org/10.1159/000292406.

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14

Aleksic, M., J. Heckenkamp, M. Gawenda, and J. Brunkwall. "Pulsatility Index Determination by Flowmeter Measurement: A New Indicator for Vascular Resistance?" European Surgical Research 36, no. 6 (2004): 345–49. http://dx.doi.org/10.1159/000081642.

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15

Davies, A. H., T. R. Magee, R. N. Baird, and M. Horrocks. "Intraoperative measurement of vascular graft resistance as a predictor of early outcome." British Journal of Surgery 80, no. 7 (July 1993): 854–57. http://dx.doi.org/10.1002/bjs.1800800712.

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16

Krohova, J., L. Faes, B. Czippelova, R. Pernice, Z. Turianikova, R. Wiszt, N. Mazgutova, A. Busacca, and M. Javorka. "Vascular resistance arm of the baroreflex: methodology and comparison with the cardiac chronotropic arm." Journal of Applied Physiology 128, no. 5 (May 1, 2020): 1310–20. http://dx.doi.org/10.1152/japplphysiol.00512.2019.

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Baroreflex response consists of several arms, but the cardiac chronotropic arm (blood pressure changes evoking heart rate response) is usually analyzed. This study introduces a method to assess the vascular baroreflex arm with the continuous noninvasive measurement of peripheral vascular resistance as an output considering causality in the interaction between oscillations and slower dynamics of vascular tone changes. We conclude that although vascular baroreflex arm involvement becomes dominant during orthostasis, gain of this interaction is relatively stable.
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17

Raj, J. U., P. Kaapa, and J. Anderson. "Effect of pulsatile flow on microvascular resistance in adult rabbit lungs." Journal of Applied Physiology 72, no. 1 (January 1, 1992): 73–81. http://dx.doi.org/10.1152/jappl.1992.72.1.73.

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We have determined the effect of pulsatile flow on segmental vascular resistance in lungs from 29 adult rabbits. In group I (n = 4), II (n = 8), and III (n = 8) lungs were isolated. In group IV (n = 9) rabbits were anesthetized, their chests were opened, and lungs were studied in vivo. Group I and II lungs had steady-flow perfusion: group I with intact vasotonus and group II with papaverine treatment. Group III lungs (papaverine treated) were perfused for two consecutive 45-min periods with steady and pulsatile flow. In all isolated lungs and in lungs of five anesthetized rabbits, we measured
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18

Parkinson, S., C. Belleau, J. Homan, and B. Richardson. "Flow and velocity waveform indices in the ovine fetal abdominal aorta with changes in behavioural state." Reproduction, Fertility and Development 7, no. 5 (1995): 1299. http://dx.doi.org/10.1071/rd9951299.

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The purpose of this study was to determine the extent to which flow and velocity waveform indices are comparable as measured from the ovine fetal abdominal aorta and reflect downstream vascular resistance with changes in electrocortical activity. Nine chronically catheterized fetal sheep were studied near term with continuous measurement of electrocortical activity, perfusion pressure, and fetal heart rate. A cuffed Doppler crystal and a Transonic flow probe on the descending abdominal aorta were used for the simultaneous measurement of velocity waveforms and flow waveforms, with the Resistanc
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19

Bush, A., C. M. Busst, W. B. Knight, J. S. Carvalho, M. L. Rigby, and E. A. Shinebourne. "Preoperative measurement of pulmonary vascular resistance in complete transposition of the great arteries." Heart 63, no. 5 (May 1, 1990): 300–303. http://dx.doi.org/10.1136/hrt.63.5.300.

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20

Spokes, Rachel A., and Vicki C. Middlefell. "Simultaneous measurement of plasma protein extravasation and carotid vascular resistance in the rat." European Journal of Pharmacology 281, no. 1 (July 1995): 75–79. http://dx.doi.org/10.1016/0014-2999(95)00231-9.

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21

Bates, D. O., and F. E. Curry. "Vascular endothelial growth factor increases hydraulic conductivity of isolated perfused microvessels." American Journal of Physiology-Heart and Circulatory Physiology 271, no. 6 (December 1, 1996): H2520—H2528. http://dx.doi.org/10.1152/ajpheart.1996.271.6.h2520.

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These experiments report the first direct measurement of microvessel permeability coefficients after exposure to vascular endothelial growth factor (VEGF). The Landis technique was extended to enable measurement of the resistance of the microvessel wall to water flow, hydraulic conductivity (Lp), on the same microvessel in the frog mesentery during the initial exposure to VEGF (acute) and 24 and 72 h after initial exposure (chronic). Control measurements of Lp showed no change either acutely or chronically. Exposure to 1 nM VEGF rapidly and transiently increased microvessel Lp within 30 s (to
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22

Ghorbani-Shirkouhi, Samaneh, Fatemeh Ashouri, Saeideh Aghayari Sheikh Neshin, Alia Saberi, Bahador Hasanzadeh, and Parisa Shahshahani. "The prevalence and associated factors of aspirin resistance among prophylactic aspirin users." Romanian Journal of Neurology 20, no. 1 (March 31, 2021): 50–56. http://dx.doi.org/10.37897/rjn.2021.1.7.

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Background. Aspirin is an antiplatelet used for the secondary prevention after vascular events. It is also suggested for the primary prevention of vascular events in high risk people, however, despite using standard prophylactic doses, aspirin resistance may result in therapeutic failure and arterial thrombosis. Since the prevalence of aspirin resistance and its associated factors were heterogeneous in different studies, this study was conducted to determine the prevalence and associated factors of aspirin resistance in an Iranian population under aspirin for primary prevention of vascular eve
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23

Polska, Elzbieta, Karl Kircher, Paulina Ehrlich, Pia V. Vecsei, and Leopold Schmetterer. "RI in central retinal artery as assessed by CDI does not correspond to retinal vascular resistance." American Journal of Physiology-Heart and Circulatory Physiology 280, no. 4 (April 1, 2001): H1442—H1447. http://dx.doi.org/10.1152/ajpheart.2001.280.4.h1442.

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The aim of the present study was to investigate the association between ultrasound Doppler measurements of resistive index (RI) in the central retinal artery and retinal vascular resistance ( R) assessed with laser Doppler velocimetry, vessel size measurement, and calculation of ocular perfusion pressure (PP) in healthy subjects. An increase in vascular resistance was induced by inhalation of 100% O2. During hyperoxia no significant changes in PP were observed. Mean flow velocity in main retinal veins was reduced by −27.5 ± 2.0%. The average decrease in diameter was −11.5 ± 1.0%. R, which was
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24

Zanaboni, P. B., J. D. Bradley, R. O. Webster, and T. E. Dahms. "Cyclooxygenase inhibition prevents PMA-induced increase in pulmonary vascular permeability to albumin." Journal of Applied Physiology 73, no. 5 (November 1, 1992): 2011–15. http://dx.doi.org/10.1152/jappl.1992.73.5.2011.

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In a previous study, we demonstrated that phorbol myristate acetate-(PMA) induced injury in isolated blood-perfused rabbit lungs was characterized by increased pulmonary vascular resistance (PVR) and permeability to water as measured by fluid filtration coefficient (Kf). The Kf increase was prevented by pretreatment with three cyclooxygenase inhibitors, indomethacin, ibuprofen, and meclofenamate. Other studies have shown that PMA causes a decrease in pulmonary vascular surface area, probably due to the increase in arterial resistance. Measurement of Kf requires increased microvascular pressure
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25

Evlakhov, Vadim I., Ilya Z. Poyassov, and Tatiana P. Berezina. "The peculiarities of pulmonary macro- and microhemodynamics changes after treatment with agonists and blockers of cholinoceptors." Medical academic journal 20, no. 4 (March 18, 2021): 35–44. http://dx.doi.org/10.17816/maj55326.

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Background. The pulmonary arterial and venous vessels are innervated by parasympathetic cholinergic nerves. However, the studies, performed on the isolated rings of pulmonary vessels, can not give answer to the question about the role of cholinergic mechanisms in the changes of pulmonary circulation in full measure.
 Aim. The comparative analysis of the changes of the pulmonary macro- and microhemodynamics after acetylcholine, atropine, pentamine and nitroglycerine treatment.
 Materials and methods. The study was carried out on the anesthetized rabbits in the condition of intact circ
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26

Constable, Peter D., Geoffrey W. Smith, George E. Rottinghaus, Mike E. Tumbleson, and Wanda M. Haschek. "Fumonisin-induced blockade of ceramide synthase in sphingolipid biosynthetic pathway alters aortic input impedance spectrum of pigs." American Journal of Physiology-Heart and Circulatory Physiology 284, no. 6 (June 1, 2003): H2034—H2044. http://dx.doi.org/10.1152/ajpheart.00155.2002.

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The sphingolipid signaling pathway appears to play an important role in regulating vascular tone. We examined the effect of fumonisin B1, a fungal toxin in corn that blocks ceramide synthase in the sphingolipid signaling pathway, on the ascending aortic impedance spectrum of pigs. Sixteen pigs were fed culture material containing fumonisin B1 (20 mg/kg body wt) ( n = 7) or a control diet ( n = 9) daily for 3 days and then instrumented under α-chloralose anesthesia for measurement of ascending aortic pressure and flow. Fumonisin ingestion increased serum sphinganine and sphingosine concentratio
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27

Parker, R. E., and K. L. Brigham. "Effects of endotoxemia on pulmonary vascular resistances in unanesthetized sheep." Journal of Applied Physiology 63, no. 3 (September 1, 1987): 1058–62. http://dx.doi.org/10.1152/jappl.1987.63.3.1058.

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Ten experiments were conducted on nine sheep to determine the effects of endotoxemia (1.0 microgram/kg iv over 15 min) on the vascular resistances of two segments of the pulmonary circulation. The first segment (S1) was from the main pulmonary artery to the site in the pulmonary veins corresponding to the pressure measured with a deflated and wedged 7-Fr Swan-Ganz catheter. The second segment (S2) was from the wedge pressure measurement site to the left atrium. Endotoxemia caused both pulmonary arterial pressure and pulmonary arterial wedge pressure to increase significantly during early (phas
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28

Fisher, Lyle E., Attila Cziraki, Curt M. Steinhart, and John D. Catravas. "Unaltered pulmonary capillary surface area in the presence of changing arterial resistance." American Journal of Physiology-Lung Cellular and Molecular Physiology 274, no. 2 (February 1, 1998): L264—L269. http://dx.doi.org/10.1152/ajplung.1998.274.2.l264.

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We hypothesized that capillary recruitment may not be solely dependent on extracapillary factors. To test this hypothesis, rabbits were anesthetized and placed on total cardiac bypass at a constant, physiological pulmonary blood flow. Vascular occlusion techniques were combined with measurement of the transpulmonary metabolism of an angiotensin-converting enzyme substrate, allowing the concomitant assessment of changes in segmental resistances and dynamically perfused capillary surface area. Intra-arterial serotonin infusion increased upstream pulmonary vascular resistances without affecting d
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29

Cochrane, E., and I. D. McCarthy. "Rapid effects of parathyroid hormone(1–34) and prostaglandin E2 on bone blood flow and strontium clearance in the rat in vivo." Journal of Endocrinology 131, no. 3 (December 1991): 359–65. http://dx.doi.org/10.1677/joe.0.1310359.

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ABSTRACT The vascular effects of noradrenaline, ATP, parathyroid hormone (PTH) and prostaglandin E2 (PGE2) were investigated in the rat. Additionally, the exchange of mineral ions between bone and blood was assessed by measuring strontium clearance, with the aim of investigating whether the vascular effects of these agents altered uptake of mineral ions or if this exchange could be changed independently of blood flow. Radioactive microspheres and 85Sr were used to establish bone blood flow and mineral clearance. Measurements of bone blood flow and arterial pressure were made in each animal and
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30

Ulett, Kimberly B., and Thomas H. Marwick. "Incorporation of Pulmonary Vascular Resistance Measurement into Standard Echocardiography: Implications for Assessment of Pulmonary Hypertension." Echocardiography 24, no. 10 (November 2007): 1020–22. http://dx.doi.org/10.1111/j.1540-8175.2007.00539.x.

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31

Siegel, Lawrence G., and Ronald G. Pearl. "Measurement of the Longitudinal Distribution of Pulmonary Vascular Resistance from Pulmonary Artery Occlusion Pressure Profiles." Anesthesiology 68, no. 2 (February 1, 1988): 305–6. http://dx.doi.org/10.1097/00000542-198802000-00036.

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32

Collee, George. "Measurement of the Longitudinal Distribution of Pulmonary Vascular Resistance from Pulmonary Artery Occlusion Pressure Profiles." Anesthesiology 68, no. 2 (February 1, 1988): 307. http://dx.doi.org/10.1097/00000542-198802000-00037.

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33

Godden, D. J., E. M. Wagner, P. D. Pare, W. Mitzner, and E. M. Baile. "Measurement of airway wall blood flow in sheep by laser-Doppler flowmetry: interpretation and problems." Journal of Applied Physiology 70, no. 2 (February 1, 1991): 641–49. http://dx.doi.org/10.1152/jappl.1991.70.2.641.

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This study investigated the pulmonary vascular response to endothelin (ET) in rats. In conscious rats, an incremental intravenous bolus of ET-1 (100-1,000 pM) caused, after an initial drop in systemic arterial pressure (Psa), a secondary dose-dependent increase of Psa concomitant with a decrease of cardiac output (CO) and heart rate (HR). Pulmonary arterial pressure (Ppa) remained unchanged, and pulmonary vascular resistance (PVR) increased significantly only after 1,000 pM (+ 40.0 +/- 10.4 at 15 min). Meclofenamate (6 mg/kg iv) did not alter hemodynamic response to ET (300 pM). After autonomi
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34

Bell, L. B., E. J. Zuperku, and J. P. Kampine. "Technique for continuous measurement of compliance in isolated vascular segments." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 250, no. 1 (January 1, 1986): R142—R149. http://dx.doi.org/10.1152/ajpregu.1986.250.1.r142.

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A method has been developed that provides an on-line (1/s) measurement of elastance [E = 1/compliance (C)] in a perfused isolated vessel segment in situ. Values of E can be obtained as a function of perfusion pressure (P) or in response to drug interventions. Noncompliant stainless steel tubes are used as inflow and outflow cannulas. Segment P is obtained from a short small-bore stainless steel catheter and miniature pressure transducer. During constant flow through the inflow cannula, an adjustable outflow resistance is used to set the segment P. Simultaneous activation of solenoid pinch valv
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35

Geierlehner, Alexander, Raymund E. Horch, Ingo Ludolph, and Andreas Arkudas. "Intraoperative Blood Flow Analysis of DIEP vs. ms-TRAM Flap Breast Reconstruction Combining Transit-Time Flowmetry and Microvascular Indocyanine Green Angiography." Journal of Personalized Medicine 12, no. 3 (March 16, 2022): 482. http://dx.doi.org/10.3390/jpm12030482.

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Background: Vascular patency is the key element for high flap survival rates. The purpose of this study was to assess and compare the blood flow characteristics of deep inferior epigastric perforator (DIEP) and muscle-sparing transverse rectus abdominis musculocutaneous (ms-TRAM) flaps for autologous breast reconstruction. Methods: This prospective clinical study combined Transit-Time Flowmetry and microvascular Indocyanine Green Angiography for the measurement of blood flow volume, vascular resistance, and intrinsic transit time. Results: Twenty female patients (mean age, 52 years) received 2
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36

Huonker, M., Y. O. Schumacher, A. Ochs, S. Sorichter, J. Keul, and M. Rössle. "Cardiac function and haemodynamics in alcoholic cirrhosis and effects of the transjugular intrahepatic portosystemic stent shunt." Gut 44, no. 5 (May 1, 1999): 743–48. http://dx.doi.org/10.1136/gut.44.5.743.

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BACKGROUNDA portosystemic stent shunt may impair cardiac function and haemodynamics.AIMSTo investigate the effects of a transjugular intrahepatic portosystemic shunt (TIPS) on cardiac function and pulmonary and systemic circulation in patients with alcoholic cirrhosis.PATIENTS/METHODS17 patients with alcoholic cirrhosis and recent variceal bleeding were evaluated by echocardiography and catheterisation of the splanchnic and pulmonary circulation before and after TIPS. The period of catheter measurement was extended to nine hours in nine of the patients. The portal vein was investigated by Dopp
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37

Charan, N. B., G. M. Turk, and R. Ripley. "Measurement of bronchial arterial blood flow and bronchovascular resistance in sheep." Journal of Applied Physiology 59, no. 2 (August 1, 1985): 305–8. http://dx.doi.org/10.1152/jappl.1985.59.2.305.

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We studied the bronchial arterial blood flow (Qbr) and bronchial vascular resistance (BVR) in sheep prepared with carotid-bronchial artery shunt. Nine adult sheep were anesthetized, and through a left thoracotomy a heparinized Teflon-tipped Silastic catheter was introduced into the bronchial artery. The other end of the catheter was brought out through the chest wall and through a neck incision was introduced into the carotid artery. A reservoir filled with warm heparinized blood was connected to this shunt. The height of blood column in the reservoir was kept constant at 150 cm by adding more
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38

Ichinose, Masashi, Shunsaku Koga, Naoto Fujii, Narihiko Kondo, and Takeshi Nishiyasu. "Modulation of the spontaneous beat-to-beat fluctuations in peripheral vascular resistance during activation of muscle metaboreflex." American Journal of Physiology-Heart and Circulatory Physiology 293, no. 1 (July 2007): H416—H424. http://dx.doi.org/10.1152/ajpheart.01196.2006.

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Continuous measurement of leg blood flow (LBF) using Doppler ultrasound with simultaneous noninvasive mean arterial blood pressure (MAP) measurement permits beat-to-beat estimates of leg vascular resistance (LVR) in humans. We tested the hypothesis that the beat-to-beat fluctuations in LVR and the dynamic relationship between MAP and LVR are modulated by the activation of muscle metaboreflex. Twelve healthy subjects performed a 1-min isometric handgrip exercise at 50% maximal voluntary contraction, which was followed by a period of imposed postexercise muscle ischemia (PEMI). We then employed
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39

Abe, Michiaki, Tae Yamamoto, Ryusuke Inoue, Hideyasu Kiyomoto, Hiroshi Sato, and Sadayoshi Ito. "728 ULTRASOUND DOPPLER MEASUREMENT OF AORTIC BLOOD FLOW AND INTRARENAL VASCULAR RESISTANCE IN CHRONIC KIDNEY DISEASE." Journal of Hypertension 30 (September 2012): e211. http://dx.doi.org/10.1097/01.hjh.0000420404.67000.81.

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40

Durkin, Robert J., Timothy W. Evans, and Stephen M. Winter. "Noninvasive Estimation of Pulmonary Vascular Resistance by Stroke Index Measurement With an Inert Gas Rebreathing Technique." Chest 106, no. 1 (July 1994): 59–66. http://dx.doi.org/10.1378/chest.106.1.59.

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Kanda, Takashi, Masashi Fujita, Osamu Iida, Masaharu Masuda, Shin Okamoto, Takayuki Ishihara, Kiyonori Nanto, Tatsuya Shiraki, and Masaaki Uematsu. "The Accurate Measurement of Pulmonary Vascular Resistance Using Echocardiography in Patients with Mild Left Heart Failure." Journal of Cardiac Failure 21, no. 10 (October 2015): S196. http://dx.doi.org/10.1016/j.cardfail.2015.08.288.

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42

Crissinger, K. D., P. R. Kvietys, and D. N. Granger. "Developmental intestinal vascular responses to venous pressure elevation." American Journal of Physiology-Gastrointestinal and Liver Physiology 254, no. 5 (May 1, 1988): G658—G663. http://dx.doi.org/10.1152/ajpgi.1988.254.5.g658.

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The response to venous pressure elevation is an important criterion for determining whether metabolic or myogenic mechanisms are involved in local vasoregulation. We studied the effects of venous pressure elevation on intestinal hemodynamics and oxygenation in 20 mixed-breed piglets, divided equally among 1-day-, 3-day-, 2-wk-, and 1-mo-old animals. A venous circuit was established between the superior mesenteric and jugular veins, which allowed measurement of superior mesenteric blood flow, venous pressure, capillary pressure, and arteriovenous oxygen difference at venous pressures between 0
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43

Axelsson, M., and S. Nilsson. "Blood pressure control during exercise in the Atlantic cod, Gadus morhua." Journal of Experimental Biology 126, no. 1 (November 1, 1986): 225–36. http://dx.doi.org/10.1242/jeb.126.1.225.

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Atlantic cod were subjected to 12–15 min swimming exercise at 2/3 body lengths s-1 in a Blazka-type swim tunnel. Pre- and postbranchial blood pressures, cardiac output (ventral aortic blood flow) and heart rate were continuously recorded, and blood samples for measurement of arterial and mixed venous oxygen tension were taken before and at the end of the exercise period. In a second group of fish, subjected to similar exercise regimes, blood samples were taken for analysis of the plasma concentrations of catecholamines. Pre- and postbranchial blood pressures and cardiac output increase during
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44

Yin, Tan Ying, Farhanahani Mahmud, and Nur Ilyani Ramli. "Near-Infrared Spectroscopy (NIRS)-based Digit Skin Tissue Blood Flow Measurement System." International Journal of Engineering & Technology 7, no. 4.30 (November 30, 2018): 131. http://dx.doi.org/10.14419/ijet.v7i4.30.22076.

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The tissue blood flow (BF) and vascular resistance are the important information for consult peripheral vascular system which related to cardiovascular disease. Unfortunately, most of the current BF monitors are costly, built in huge size and preferable use in hospital and clinic. In the present study, a portable digit skin tissue BF measurement system had been developed using Near-infrared spectroscopy (NIRS) method with simple circuitry and low cost that can be afforded by patients to monitor their cardiovascular information. This system consists of a self-developed NIRS probe; LED and a pho
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HIRATA, Kumiko, Kadirvelu AMUDHA, Raja ELINA, Takeshi HOZUMI, Junichi YOSHIKAWA, Shunichi HOMMA, and Chim C. LANG. "Measurement of coronary vasomotor function: getting to the heart of the matter in cardiovascular research." Clinical Science 107, no. 5 (October 26, 2004): 449–60. http://dx.doi.org/10.1042/cs20040226.

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Measurement of endothelial function in patients has emerged as a useful tool for cardiovascular research. Although no gold standard for the measurement of endothelial function exists, the measurement of flow-mediated dilation in the brachial artery, assessed with Doppler ultrasonography, is the most studied method. However, the assumption that endothelial dysfunction detected in brachial arteries is a manifestation of systemic endothelial dysfunction including the coronary circulation may not be entirely valid. Brachial and myocardial circulations differ in terms of the microvascular architect
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Balasubramanian, Lavanya, Kay-Pong Yip, Tai-Hsin Hsu, and Chun-Min Lo. "Impedance analysis of renal vascular smooth muscle cells." American Journal of Physiology-Cell Physiology 295, no. 4 (October 2008): C954—C965. http://dx.doi.org/10.1152/ajpcell.00009.2008.

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Impedance of renal vascular smooth muscle cells (VSMCs) cultured on microelectrodes was measured by electric cell-substrate impedance sensing. Changes in measured impedance as a function of frequency were compared with the calculated values obtained from an extended cell-electrode model to estimate the junctional resistance, distance between the ventral cell surface and the substratum, and apical and basolateral membrane capacitances of renal VSMCs. This cell-electrode model was derived to accommodate the slender and rectangular shape of VSMCs. The calculated changes in impedance ( Zcal) based
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MORIYASU, Fuminori, Osamu NISHIDA, Nobuyuki BAN, Takefumi NAKAMURA, Yasunari SOH, Jiroh OCHI, Kensuke MIURA, et al. "Measurement of portal vascular resistance in patients with chronic liver diseases by simultaneous measurement of portal blood flow and portal venous pressure." Kanzo 26, no. 4 (1985): 485–92. http://dx.doi.org/10.2957/kanzo.26.485.

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48

Ju, Bin, Yun Tao Qian, and Huo Jie Ye. "Wavelet Based Measurement on Photoplethysmography by Smartphone Imaging." Applied Mechanics and Materials 380-384 (August 2013): 773–77. http://dx.doi.org/10.4028/www.scientific.net/amm.380-384.773.

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[Purpose] Smartphones video cameras can be used to detect the photoplethysmograph (PPG) signal.The pulse wave signal detected by smartphone always mixed mass noise because of finger moving, unevenness of pressure and outer light interference. Previous studies limit to the filtering algorithm that denoising signals, without considering characteristics information of pulse wave itself. [Method] In this paper, we propose an algorithm based on wavelet to detect qualified PPG, which captures three critical characteristic quantities through wavelet high frequency coefficient. [Results] Experiment il
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Baines, A. D. "Is there a role for renal α2-adrenoceptors in the pathogenesis of hypertension?" Canadian Journal of Physiology and Pharmacology 65, № 8 (1 серпня 1987): 1638–43. http://dx.doi.org/10.1139/y87-257.

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Current information suggests that α2-adrenoceptors do not directly influence vascular resistance or Na reabsorption in the rat kidney. To reexamine the effects of α2-agonists we used isolated rat kidneys perfused at 37.5 °C with precise measurement of renal artery pressure and flow. The recirculating perfusate contained pyruvate as the sole metabolic substrate which enabled us to use gluconeogenesis as an index of proximal tubular α1-responses. Clonidine and guanfacine in 100 nM concentrations decreased phosphate excretion without altering Na, Cl, or K reabsorption or gluconeogenesis; 500 nM c
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Cassano, Velia, Daniele Crescibene, Marta Letizia Hribal, Corrado Pelaia, Giuseppe Armentaro, Marcello Magurno, Alfredo Toscani, et al. "Uric Acid and Vascular Damage in Essential Hypertension: Role of Insulin Resistance." Nutrients 12, no. 9 (August 19, 2020): 2509. http://dx.doi.org/10.3390/nu12092509.

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Increased levels of uric acid (UA) have been shown to be correlated with many clinical conditions. Uric acid may adversely affect the insulin signalling pathway inducing insulin resistance (IR). Several studies report the association between arterial stiffness (AS), an early indicator of atherosclerosis, and UA. The purpose of the present study was to evaluate the association between UA and AS, considering the potential role of IR. We enrolled 1114 newly diagnosed, never-treated hypertensive patients. Insulin resistance was assessed by the homeostatic model assessment (HOMA) index. Arterial st
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