Relevant bibliographies by topics / Vasocontriction

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  1. Journal articles
  2. Dissertations / Theses

Academic literature on the topic 'Vasocontriction'

Author: Grafiati
Published: 31 May 2025
Last updated: 31 July 2025

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Journal articles on the topic "Vasocontriction"

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Mahesh, Karthik, Chirag Ahuja, Abeer Goel, and Sukriya Saravanan. "Reversible cerebral vasocontriction syndrome in amphetamine abuse." Journal of the Neurological Sciences 429 (October 2021): 119680. http://dx.doi.org/10.1016/j.jns.2021.119680.

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Myers, Jeff L., Patrick W. Domkowski, Yining Wang, and Richard A. Hopkins. "Sympathetic blockade blunts hypercapnic pulmonary arterial vasocontriction in newborn piglets." European Journal of Cardio-Thoracic Surgery 13, no. 3 (1998): 298–305. http://dx.doi.org/10.1016/s1010-7940(98)00007-4.

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Spain, David A., Mark A. Wilson, R. Neal Garrison, and Sponsor J. David Richardson. "NITRIC OXIDE SYNTHASE INHBITION ACCREVATES INTESTINAL MICROVASCULAR VASOCONTRICTION AND HYPOPERFUSION OF BACTERIMIA." Journal of Trauma: Injury, Infection, and Critical Care 35, no. 1 (1993): 171. http://dx.doi.org/10.1097/00005373-199307000-00080.

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Hwang, Jung Won, Chul Min Kim, Yong Seok Oh, and Yong Lak Kim. "Effects of Isoflurane on Hypoxic Pulmonary Vasocontriction in Human during One Lung Ventilation." Korean Journal of Anesthesiology 30, no. 2 (1996): 147. http://dx.doi.org/10.4097/kjae.1996.30.2.147.

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Lee, Na Kyung, Han Suk Park, Chan Jong Chung, and Young Jhoon Chin. "Effects of Isoflurane on Hypoxic Pulmonary Vasocontriction in Human during One Lung Ventilation." Korean Journal of Anesthesiology 30, no. 2 (1996): 154. http://dx.doi.org/10.4097/kjae.1996.30.2.154.

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Madrid, María Isabel, Miguel García-Salom, Jerónimo Tornel, Marc De Gasparo, and Francisco J. Fenoy. "Effect of interactions between nitric oxide and angiotensin II on pressure diuresis and natriuresis." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 273, no. 5 (1997): R1676—R1682. http://dx.doi.org/10.1152/ajpregu.1997.273.5.r1676.

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The present study examined the effect of an angiotensin II AT1 or AT2 receptor antagonist on the impairment of the pressure diuresis and natriuresis response produced by nitric oxide (NO) synthesis blockade. N ω-nitro-l-arginine methyl ester (l-NAME, 37 nmol ⋅ kg−1 ⋅ min−1) lowered renal blood flow and reduced the slopes of the pressure diuresis and natriuresis responses by 44 and 40%, respectively. Blockade of AT1 receptors with valsartan increased slightly sodium and water excretion at low renal perfusion pressure (RPP). Blockade of AT2 receptors with PD-123319 had no effect on renal functio
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7

Dahniarti, Dhany, Irfan Idris, and Nasrudin Am. "Pengaruh Kepatuhan Suplementasi Tablet Kalsium Modifikasi terhadap Kadar Kalsium dan Tekanan Darah Pada Ibu Hamil." PALAPA 6, no. 1 (2018): 19–33. http://dx.doi.org/10.36088/palapa.v6i1.56.

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A low intake of calcium leads to an increase in high blood pressure by stimulating the release of paratiroid and renin hormones that lead to increase in intracellular calcium concentration in the vasculer smooth muscle cells and result in vasocontriction. This study aims to determine the influence of compliance to supply modified calcium tablet toward calcium level and blood pressure in pregnant mothers. This research was conducted at RSIA Sitti Khadijah I Maternity Hospital Makassar with research design of quasy experiment with prepost test group control design. There were 30 pregnant mother
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Maharani, Laksmi. "PROTEIN SOLUBLE SUPPRESSION OF TUMORIGENICITY 2 (sST2) SEBAGAI PENANDA KERUSAKAN KARDIOVASKULAR PADA PREEKLAMSIA." Jurnal Akta Trimedika 1, no. 2 (2024): 187–97. http://dx.doi.org/10.25105/aktatrimedika.v1i2.19706.

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Preeclampsia as a pregnancy complication was still a direct cause of maternal death, especially in developing countries. The process of abnormal trophoblast invasion as a pathophysiology of preeclampsia would cause an ischemic environment in the placenta. This situation caused the placenta to release pro-inflammatory cytokines and anti-angiogenic factors into the maternal circulation, resulting in the vasocontriction of blood vessels, endothelial dysfunction in the vasculature of maternal organs, and the production of different clinical features depending on the organs affected. Vascular and m
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9

Jerez, Susana, Liliana Sierra, and María Peral de Bruno. "17-Octadecynoic acid improves contractile response to angiotensin II by releasing vasocontrictor prostaglandins." Prostaglandins & Other Lipid Mediators 97, no. 1-2 (2012): 36–42. http://dx.doi.org/10.1016/j.prostaglandins.2011.07.008.

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Moe, Gordon W., Jean L. Rouleau, Quang T. Nguyen, Peter Cernacek, and Duncan J. Stewart. "Role of endothelins in congestive heart failure." Canadian Journal of Physiology and Pharmacology 81, no. 6 (2003): 588–97. http://dx.doi.org/10.1139/y03-011.

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Despite major advances in conventional medical therapy, patients with heart failure continue to experience significant morbidity and mortality. Endothelin-1 (ET-1) is a potent vasocontrictor and mitogenic peptide that is activated in heart failure. There is increasing experimental and clinical evidence in support of an important role of ET-1 in the pathophysiology of heart failure. Manipulation of the activity of ET-1, especially using endothelin receptor blockers, has allowed for the further elucidation of the role of this neurohormonal system and development of novel therapeutic strategies i
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Dissertations / Theses on the topic "Vasocontriction"

1

Mohamed, Boubacar. "Contrôle bidirectionnel du couplage neurovasculaire par les interneurones à somatostatine." Electronic Thesis or Diss., Sorbonne université, 2024. http://www.theses.fr/2024SORUS408.

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Le couplage neurovasculaire, qui relie l'activité neuronale au débit sanguin cérébral, est altéré précocement dans plusieurs troubles neurologiques et sous-tend l'imagerie cérébrale fonctionnelle. Ce processus complexe implique de multiples acteurs cellulaires, les interneurones inhibiteurs en particulier faisant l'objet d'une attention croissante, mais les mécanismes qui sous-tendent la façon dont ils contrôlent le flux sanguin cérébral restent insaisissables. Cette étude élucide les mécanismes par lesquels les interneurones à somatostatine contrôlent de manière bidirectionnelle le couplage n
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2

Šnorek, Michal. "Význam produkce oxidu dusnatého a reaktivních forem kyslíku při akutním koronárním syndromu." Doctoral thesis, 2013. http://www.nusl.cz/ntk/nusl-326750.

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In cardiology, there are different conditions associated with the release of free radicals in some forms of hypoxia, such as ventilatory hypoxia or reduced perfusion. The role of free radicals during hypoxia in cardiology is the key point of our interest. In presented thesis, we have focused on hypoxia-induced pulmonary vasoconstriction and acute myocardial ischemia. Hypoxic pulmonary vasoconstriction (HPV), an important physiological mechanism, is regulated by changes in the production of and interactions among reactive oxygen species (ROS). There is controversy, however, over whether HPV is
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Journal articles
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