Relevant bibliographies by topics / Vector Borne

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Vector Borne'

Author: Grafiati
Published: 4 June 2021
Last updated: 4 February 2022

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Journal articles on the topic "Vector Borne"

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GUBLER, D. J. "Vector-borne diseases." Revue Scientifique et Technique de l'OIE 28, no. 2 (August 1, 2009): 583–88. http://dx.doi.org/10.20506/rst.28.2.1904.

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Rosenberg, Ronald, and C. Ben Beard. "Vector-borne Infections." Emerging Infectious Diseases 17, no. 5 (May 2011): 769–70. http://dx.doi.org/10.3201/eid1705.110310.

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Abdullah; YAZAR, INCI. "Vectors and Vector-Borne Diseases in Turkey." Ankara Üniversitesi Veteriner Fakültesi Dergisi 60, no. 4 (2013): 281–96. http://dx.doi.org/10.1501/vetfak_0000002593.

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Onmaz, A. C., R. G. Beutel, K. Schneeberg, A. N. Pavaloiu, A. Komarek, and R. van den Hoven. "Vectors and vector-borne diseases of horses." Veterinary Research Communications 37, no. 1 (September 30, 2012): 65–81. http://dx.doi.org/10.1007/s11259-012-9537-7.

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Dhopte, Pragati, and Irrusappan Hari. "VECTOR-BORNE DISEASES IN INDIA." International Journal of Advanced Research 8, no. 10 (October 31, 2020): 1055–67. http://dx.doi.org/10.21474/ijar01/11933.

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Vectors are transmitted diseases from person to person that diseases are known as vactor borne diseases. There are mainly six vector borne diseases present in India, tropical and subtropical rigion also. As per current medical importance, geographic distribution, epidemiology and potential spreading of vector borne diseases, Malaria total cases were 29340 and deaths 2 and Japanese encephalitis total cases were 111. Chikungunya and Kala azar total cases were 700 and no deaths were found in 2020 respectively. 87.25% of MDA were supplied to total population and the dengue cases were 136422 and deaths 132 were observed in 2019. The vector borne diseases in India are reviewed in this article.
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REITER, P. "The standardised freight container: vector of vectors and vector-borne diseases." Revue Scientifique et Technique de l'OIE 29, no. 1 (April 1, 2010): 57–64. http://dx.doi.org/10.20506/rst.29.1.1960.

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Rajagopalan, P. K. "Aspects of Vector Borne Disease Control." Journal of Communicable Diseases 50, no. 01 (March 29, 2018): 28–31. http://dx.doi.org/10.24321/0019.5138.201806.

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Blaustein, Leon, Richard S. Ostfeld, and Robert D. Holt. "A Community-Ecology Framework for Understanding Vector and Vector-Borne Disease Dynamics." Israel Journal of Ecology and Evolution 56, no. 3-4 (May 6, 2010): 251–62. http://dx.doi.org/10.1560/ijee.56.3-4.251.

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The integration of community ecology into the understanding and management of vectors and vector-borne diseases has largely occurred only recently. This compendium examines a variety of community interactions that can affect vector or vector-borne disease dynamics. They include: the importance of risk of predation, risk of ectoparasatism, competition, interactions of competition with transgenic control, apparent competition mediated through vectors, indirect effects of pesticides, vector diversity, and parasite diversity within a vector. In this paper, we summarize these studies and introduce several additional important questions in need of further exploration.
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Choi, Young Hwa. "Vector-borne infectious diseases." Journal of the Korean Medical Association 60, no. 6 (2017): 449. http://dx.doi.org/10.5124/jkma.2017.60.6.449.

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Wang, Penghua, Fengwei Bai, Gong Cheng, Jianfeng Dai, and Michael J. Conway. "Vector-Borne Viral Diseases." BioMed Research International 2015 (2015): 1. http://dx.doi.org/10.1155/2015/582045.

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Dissertations / Theses on the topic "Vector Borne"

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Harrison, Eleanor Margaret. "Epidemiology and evolution of vector borne disease." Thesis, University of Bath, 2013. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.619145.

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In recent years the incidence of many vector borne-diseases has increased worldwide. We investigate the epidemiology and evolution of vector-borne disease, focussing on the neglected tropical disease leishmaniasis to determine suitable strategies for control and prevention. We develop a compartmental mathematical model for leishmaniasis, and examine the dependence of disease spread on model parameters. We perform an elasticity analysis to establish the relative impact of disease parameters and pathways on infection spread and prevalence. We then use optimal control theory to determine optimal vaccination and spraying strategies for leishmaniasis, and assess the dependence of control on disease relapse. We investigate the evolution of virulence in vector-borne disease using adaptive dynamics and both non-spatial and metapopulation models for disease spread. Using our metapopulation model we also determine the impact of land-use change such as urbanisation and deforestation on disease spread and prevalence. We find that in the absence of evolution, control techniques which directly reduce the rate of vector transmission lead to the greatest reduction in potential disease spread. Although the spraying of insecticide can reduce the basic reproductive number $R_{0}$, we find that vaccination is more effective. Disease relapse is the driving force behind infection at endemic equilibrium and greatly increases the level of control required to prevent a disease epidemic. When a trade-off is in place between transmission and virulence we find that control techniques which reduce the duration of transmission lead to the fixation of pathogen strains with heightened virulence. Control techniques such as spraying can therefore be counterproductive, as increasing virulence increases human infection prevalence. This holds true when virulence is in either the host or vector and suggests that virulence within the vector should not be ignored. Urbanisation and deforestation can also lead to increases in both transmission and virulence, as reducing the distance between urban settlements and the vector natural habitat alters disease incidence.
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Morin, Cory William. "Climate and Environmental Influences on the Ecology of Vectors and Vector-borne Diseases." Diss., The University of Arizona, 2012. http://hdl.handle.net/10150/241951.

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Recently researchers have recognized the potential effects of climate variability and climate change on infectious disease ecology. Mosquito-borne diseases are of considerable concern due to their reliance on temperature to regulate vector reproduction, survival, and vector and agent development. Precipitation is also influential because it helps maintain habitat for immature mosquitoes. The interactions between climate, vector, and agent are complex, however, and thus assessing the overall impact of climate on disease occurrence is difficult. Discerning the influence of climate on mosquito-borne diseases requires an interdisciplinary synthesis of knowledge about the relationships between components of the disease system and analysis techniques that account for the individual and interacting roles that each element contributes to the ecology of the disease. In this dissertation, climate and climate change influences on dengue fever and West Nile virus are identified through process based modeling to simulate changes in vector and viral transmission dynamics. Analysis of the literature pertaining to climate influences on dengue virus ecology reveals that climate variables often interact interdependently to influence dengue virus transmission. Statistical techniques correlating or modeling climate-dengue relationships are often inconsistent and location specific. Process based modeling has been employed to better simulate the intricacies and non-linear dynamics involved, but most models focus only on vector populations. Therefore, models should incorporate viral development and transmission components to better simulate dengue virus ecology. A model of West Nile virus vector dynamics across the southern United States reveals that impacts from climate change are very location and context-specific. While temperatures generally increase the season length of vector activity, changes in precipitation and evapotranspiration dynamics often lead to lower summer mosquito populations and limited population development in water-stressed areas. A simulation of dengue fever cases in San Juan County, Puerto Rico with a coupled vector-epidemiological model showed strong agreement when compared with reported case data (Willmott's d = 0.90 and r2 = 0.71). The model indicates that certain climate variables became disease limiting during specific times of the year. Temperature limits virus transmission during the winter by slowing viral development while lower precipitation limits spring transmission by suppressing vector populations.
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El, Moustaid Fadoua. "Modeling Temperature Effects on Vector-Borne Disease Dynamics." Diss., Virginia Tech, 2019. http://hdl.handle.net/10919/102579.

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Vector-borne diseases (VBDs) cause significant harm to humans, plants, and animals worldwide. For instance, VBDs are very difficult to manage, as they are governed by complex interactions. VBD transmission depends on the pathogen itself, vector-host movement, and environmental conditions. Mosquito-borne diseases are a perfect example of how all these factors contribute to changes in VBD dynamics. Although vectors are highly sensitive to climate, modeling studies tend to ignore climate effects. Here, I am interested in the arthropod small vectors that are sensitive to climate factors such as temperature, precipitation, and drought. In particular, I am looking at the effect of temperature on vector traits for two VBDs, namely, dengue, caused by a virus that infects humans and bluetongue disease, caused by a virus that infects ruminants. First, I collect data on mosquito traits' response to temperature changes, this includes adult traits as well as juvenile traits. Next, I use these traits to model mosquito density, and then I incorporate the density into our mathematical models to investigate the effect it has on the basic reproductive ratio R0, a measure of how contagious the disease is. I use R0 to determine disease risk. For dengue, my results show that using mosquito life stage traits response to temperature improves our vector density approximation and disease risk estimates. For bluetongue, I use midge traits response to temperature to show that the suitable temperature for bluetongue risk is between 21.5 �C and 30.7 �C. These results can inform future control and prevention strategies.
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Uzcategui, Cuello Nathalie Yumari. "Evolution and dispersal of mosquito-borne flaviviruses." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288520.

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Costa, Maria Carolina Regateiro Machado e. "Vector-borne pathogens found in carnivores in wild Namibia." Master's thesis, Universidade de Lisboa, Faculdade de Medicina Veterinária, 2019. http://hdl.handle.net/10400.5/18037.

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Dissertação de Mestrado Integrado em Medicina Veterinária
This dissertation aimed to identify and molecularly characterize vector-borne pathogens from several parasite families, all possessing stages found in peripheral blood, from a wide variety of free-ranging carnivores living in Namibia, in the southern part of Africa. Blood samples collected from 9 bat-eared foxes (Otocyon megalotis), 17 brown hyenas (Parahyaena brunnea), 19 spotted hyenas (Crocuta crocuta) and 85 cheetahs (Acinonyx jubatus) were screened by Polymerase Chain Reactions (PCRs) and tested for pathogens of the Onchocercidae family, the order Piroplasmida, bacteria from the Anaplasmataceae and the Rickettsiaceae families and, lastly, the Hepatozoidae family. The PCRs targeted both the ITS-2 and 12S, 18S, 16S, 18S and 18S rRNA genes respectively and were followed by nucleotide sequencing. In total, sampled animals showed a 43.1% rate of Onchocercidae infection, 67.7% of Piroplasmida, 60% of them were positive for Anaplasmataceae, 10% for Rickettsiaceae and Hepatozoidae were detected in 47.7% of them. Obtained filaroid sequences showed high homologies with both Acanthocheilonema reconditum and Acanthocheilonema dracunculoides and further phylogenetic analysis were performed in both brown and spotted hyenas, with the construction of a phylogenetic tree. Piroplasmida results were not studied any further. For Anaplasmataceae, subsequent sequencing results indicated high similarity with both Anaplasma phagocytophilum and Anaplasma platys and varied PCR protocols were conducted in order to differentiate between these organisms, but no conclusions were reached. The Rickettsiaceae found displayed high homologies with Rickettsia raoultii. And finally, the Hepatozoidae infection showed to be a mixed one with both Hepatozoon canis and Hepatozoon felis. These results are important not only on a conservation level for the infected host species, but are also relevant for domestic animals coexisting in the surrounding areas, as well as humans, especially since a few of the parasites found may have zoonotic potential. Future studies should focus on understanding vectors, transmission routes, infection dynamics and host specificity in order to better evaluate the possible danger these infections may withhold.
RESUMO - Agentes patogénicos transmitidos por vetores presentes em carnívoros na Namíbia - Esta dissertação teve como principal objetivo identificar e caracterizar molecularmente agentes patogénicos transmitidos por vetores de várias famílias parasitárias, com o aspeto em comum de todas possuírem fases do desenvolvimento encontradas no sangue, de espécies variadas de carnívoros selvagens que habitam na Namíbia, no Sul de África. Foram testadas amostras sanguíneas de 9 raposas-orelhas-de-morcego (Otocyon megalotis), 17 hienas-castanhas (Parahyaena brunnea), 19 hienas-malhadas (Crocuta crocuta) e 85 chitas (Acinonyx jubatus) por PCR e analisadas para pesquisa de parasitas da família Onchocercidae, da ordem Piroplasmida, bactérias das famílias Anaplasmataceae e Rickettsiaceae e, finalmente, da família Hepatozoidae. Os PCRs foram direcionados aos genes do rRNA ITS-2 e 12S, 18S, 16S, 18S e 18S respetivamente e foram seguidos de sequenciação de nucleótidos. Na totalidade, os animais testados mostraram uma taxa de infeção de 43.1% por Onchocercidae, de 67.7% de Piroplasmida, 60% deles tiveram resultados positivos para Anaplasmataceae, 10% para Rickettsiaceae e Hepatozoidae foram detetados em 47.7% da população. As sequências obtidas de filarídeos, mostraram possuir elevadas homologias com Acanthocheilonema reconditum e Acanthocheilonema dracunculoides, e estudos filogenéticos mais intensivos foram realizados, nomeadamente uma árvore filogenética que inclui ambas as espécies de hienas. Os resultados relativos a Piroplasmida não foram aprofundados. Para as Anaplasmataceae, as sequenciações subsequentes indicaram elevada similaridade com Anaplasma phagocytophilum e Anaplasma platys e múltiplos protocolos de PCRs foram efetuados, com o intuito de diferenciar entre estas duas espécies, mas não foram retiradas quaisquer conclusões. As Rickettsiaceae presentes evidenciaram fortes semelhanças com Rickettsia raoultii. E finalmente, as infeções por Hepatozoidae mostraram ser uma infeção mista por ambos Hepatozoon canis e Hepatozoon felis. A importância destes resultados não se limita apenas à conservação das espécies animais em causa, mas são também relevantes em termos dos animais domésticos coabitantes na mesma região, assim como humanos, especialmente tendo em conta o possível potencial zoonótico de algumas espécies parasitárias. Estudos futuros devem ter como principais objetivos o estudo dos vetores respetivos, tipo de transmissão, dinâmica da infeção e especificidade parasitária, para melhor avaliar os possíveis perigos que podem advir da presença destes parasitas.
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McOdimba, Francis Awuor. "Epidemiology of vector-borne diseases in cattle from SE Uganda." Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/30498.

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Institutions involved in vector-borne diseases research, epidemiological studies as well as vaccine development require reliable and sensitive assays to support the development of vaccine products and new drugs for treatment. These diagnostic assays also aid in identifying disease control target populations, and to monitor infection during trials for assessing the efficacy of preventive or curative drug. Molecular techniques such as the polymerase chain reaction (PCR) amplification have been used in detecting parasites of several species, sub-species and types and are favoured over microscopic examination of blood or the immunological methods because of their superior sensitivity and higher throughput. Two of the most commonly used diagnostic methods, microscopy and molecular techniques for pathogen detection and species characterization, were evaluated for their sensitivity and specificity and subsequently used in screening cattle for parasites in the blood of cattle kept under traditional mixed farming management system. Molecular methods revealed higher VBD prevalence in the cattle from the villages of Tororo and Busia districts of SE Uganda. The prevalence of trypanosome species pathogenic to livestock was found to be higher than previously documented in this area. Based on the data obtained by PCR amplification the effect of prophylactic drug intervention against trypanosomiasis was assessed over a period of six months. While isometamidium chloride treatment of cattle appeared to control trypanosomiasis in areas with low prevalence, the drug had no effect in controlling the disease in high prevalence areas. It would therefore be necessary to combine the use of drug intervention with other methods such as vector control, to reduce the prevalence, in order to realize effective control of trypanosomiasis.
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Shearer, Freya. "Improving geospatial models of risk for vector-borne, zoonotic diseases." Thesis, University of Oxford, 2017. http://ora.ox.ac.uk/objects/uuid:cfe8ffa9-453b-4e10-9009-e387a39db6de.

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Public health surveillance data are often incomplete, particularly where resources are lacking, but geospatial models can help to fill the gaps by providing estimates where data are sparse. By combining information on locations where diseases have been recorded with geographic data on environmental and socioeconomic covariates known to affect disease transmission using machine-learning models (such as boosted regression trees), niche modelling can generate fine-resolution, evidence-based risk maps for a variety of diseases of public health importance. This thesis investigates the geographical distribution of two vector-borne, zoonotic diseases of public health importance: Plasmodium knowlesi malaria and yellow fever (YF). A number of new methodological approaches to niche modelling are developed for: mapping diseases whose distributions are impacted by multiple host and vector species, ameliorating spatial bias in disease reporting rates, and accounting for human vaccination coverage. Chapter 2 investigates spatial variation in risk of human P. knowlesi infection across Southeast Asia. The infection risk model for P. knowlesi malaria is based on improvements to a standard niche modelling approach, and incorporates a novel joint distribution model to leverage data from a number of host species. Chapter 3 estimates YF vaccination coverage through time across all age cohorts in every district/municipality of countries at risk of YF, globally. These estimates are used to estimate the additional vaccination coverage needed to prevent further YF outbreaks, and they provide information needed to account for population immunity when estimating YF infection risk. Chapter 4 describes the development of a novel Poisson point process niche model, which is then used to predict YF infection risk in humans and demonstrates how vaccination coverage can be efficiently accounted for in disease niche models. The disease risk maps of P. knowlesi malaria and YF produced through this thesis will act as resources to improve the targeting, implementation and evaluation of disease prevention, surveillance and control strategies. Methods developed to account for vaccination coverage, reporting rate biases, and complex transmission systems will be applicable to risk mapping for a range of vector-borne, zoonotic diseases of public health importance.
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Xue, Ling. "Modeling and analysis of vector-borne diseases on complex networks." Diss., Kansas State University, 2013. http://hdl.handle.net/2097/16788.

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Doctor of Philosophy
Department of Electrical and Computer Engineering
Caterina Scoglio
Vector-borne diseases not only cause devastating economic losses, they also significantly impact human health in terms of morbidity and mortality. From an economical and humane point of view, mitigation and control of vector-borne diseases are essential. Studying dynamics of vector-borne disease transmission is a challenging task because vector-borne diseases show complex dynamics impacted by a wide range of ecological factors. Understanding these factors is important for the development of mitigation and control strategies. Mathematical models have been commonly used to translate assumptions concerning biological (medical, demographical, behavioral, immunological) aspects into mathematics, linking biological processes of transmission and dynamics of infection at population level. Mathematical analysis translates results back into biology. Classical deterministic epidemic models do not consider spatial variation, assuming space is homogeneous. Spatial spread of vector-borne diseases observed many times highlights the necessity of incorporating spatial dynamics into mathematical models. Heterogeneous demography, geography, and ecology in various regions may result in different epidemiological characteristics. Network approach is commonly used to study spatial evolution of communicable diseases transmitted among connected populations. In this dissertation, the spread of vector-borne diseases in time and space, is studied to understand factors that contribute to disease evolution. Network-based models have been developed to capture different features of disease transmission in various environments. Network nodes represent geographical locations, and the weights represent the level of contact between regional pairings. Two competent vector populations, Aedes mosquitoes and Culex mosquitoes, and two host populations, cattle and humans were considered. The deterministic model was applied to the 2010 Rift Valley fever outbreak in three provinces of South Africa. Trends and timing of the outbreak in animals and humans were reproduced. The deterministic model with stochastic parameters was applied to hypothetical Rift Valley fever outbreak on a large network in Texas, the United States. The role of starting location and size of initial infection in Rift Valley fever virus spread were studied under various scenarios on a large-scale network. The reproduction number, defined as the number of secondary infections produced by one infected individual in a completely susceptible population, is typically considered an epidemic threshold of determining whether a disease can persist in a population. Extinction thresholds for corresponding Continuous-time Markov chain model is used to predict whether a disease can perish in a stochastic setting. The network level reproduction number for diseases vertically and horizontally transmitted among multiple species on heterogeneous networks was derived to predict whether a disease can invade the whole system in a deterministic setting. The complexity of computing the reproduction number is reduced because the expression of the reproduction number is the spectral radius of a matrix whose size is smaller than the original next generation matrix. The expression of the reproduction number may have a wide range of applications to many vector-borne diseases. Reproduction numbers can vary from below one to above one or from above one to below one by changing movement rates in different scenarios. The observations provide guidelines on executing movement bans in case of an epidemic. To compute the extinction threshold, corresponding Markov chain process is approximated near disease free equilibrium. The extinction threshold for Continuous-time Markov chain model was analytically connected to the reproduction number under some assumptions. Numerical simulation results agree with analytical results without assumptions, proposing a mathematical problem of proving the existence of the relationships in general. The distance of the extinction threshold were shown to be closer to one than the reproduction number. Consistent trends of probability of extinction varying with disease parameters observed through numerical simulations provide novel insights into disease mitigation, control, and elimination.
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Kumsa, Bersissa. "Molecular investigation of arthropods and vector-borne bacteria from Ethiopia." Thesis, Aix-Marseille, 2014. http://www.theses.fr/2014AIXM5054/document.

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En outre, nous avons fait une expérience sur les nouveaux outils pour identifier les tiques par MALDI-TOF MS protéines profilage et des méthodes moléculaires. Notre étude visant à explorer les bactéries dans les ixodidae prélevés sur des animaux domestiques en Éthiopie a révélé une prévalence globale de 6% (46/767) des rickettsies de SFG, 3,8% (29/767) ADN de Borrelia et 6,4% (54/842) de C. burnetii dans différentes espèces de tiques. L'étude pour étudier les bactéries dans 303 puces prélevés sur des chiens et des chats domestiques en Ethiopie qui ont été identifiés comme étant morphologiquement Ctenocephalides felis felis, Ctenocephalides canis, Pulex irritans et Echidnophaga gallinacé montré Rickettsia felis dans 21% des puces, principalement dans Ctenocephalides felis, avec un semblable prévalence dans les puces de chiens et de chats. La présence d'Acinetobacter spp. dans M. ovinus, Heterodoxus spiniger, Bovicola ovis et Linognathus vituli. La séquence du gène rpoB partiel a révélé la présence de A. soli, A. lowffii, A. Pitti et 3 nouveaux Acinetobacter spp. dans les poux et Keds. Bartonella melophagi a été identifié par une PCR standard, suivi par un séquençage du fragment de la gltA et gène rpoB chez M. ovinus. Dans l'ensemble, nos résultats alerte les médecins en charge des patients avec fièvre d'étiologie inconnue en Ethiopie et ceux qui se soucient de voyageurs en provenance de l'Ethiopie à prendre en compte la présence de plusieurs espèces zoonotiques à transmission vectorielle de bactéries, y compris SFG rickettsies, C. burnetii, R. felis, B. henselae et B. melophagi comme agents pathogènes potentiels
Our study to explore bacteria in ixodid ticks collected from domestic animals in Ethiopia revealed an overall prevalence of 6% (46/767) SFG rickettsiae, 3.8% (29/767) Borrelia DNA and 6.4% (54/842) C. burnetii in different tick species. The study to investigate bacteria in 303 fleas collected from domestic dogs and cats in Ethiopia that were morphologically identified as Ctenocephalides felis felis, Ctenocephalides canis, Pulex irritans and Echidnophaga gallinacean showed Rickettsia felis in 21% of fleas, mainly in Ctenocephalides felis, with a similar prevalence in fleas from dogs and cats. The study to investigate bacteria in lice and sheep ked (Melophagus ovinus) revealed Acinetobacter spp. in M. ovinus, Heterodoxus spiniger, Bovicola ovis and Linognathus vituli. Partial rpoB gene sequence revealed A. soli, A. lowffii, A. pitti and 3 new Acinetobacter spp. in the lice and keds. Molecular identification of lice using an 18S rRNA gene analysis confirmed the morphological methods of lice identification. Bartonella melophagi was identified by standard PCR followed by sequencing of fragments of the gltA and rpoB genes in M. ovinus.Overall, our findings alert physicians managing patients with fever of unknown aetiology in Ethiopia and those who care for travellers from Ethiopia to consider the presence of several vector-borne zoonotic species of bacteria including SFG rickettsiae, C. burnetii, R. felis, B. henselae and B. melophagi as potential causative agents
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Twiddy, Sally Susanna. "The molecular epidemiology and evolution of dengue virus." Thesis, University of Oxford, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269490.

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Books on the topic "Vector Borne"

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Society for General Microbiology. Symposium. Microbe-vector interactions in vector-borne diseases. Cambridge [Eng.]: Cambridge University Press, 2004.

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Integrated vector management: Controlling vectors of malaria and other insect vector borne diseases. Chichester, West Sussex, UK: Wiley-Blackwell, 2011.

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International Symposium of Vectors & Vector-borne Diseases (8th 2006 Madurai, India). Vector-borne diseases: Epidemiology and control. Edited by Tyagi B. K and Indian Council of Medical Research. Centre for Research in Medical Entomology. Jodhpur: Scientific Publishers, India on behalf of Centre for Research in Medical Entomology, ICMR, 2008.

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Mack, Alison, ed. Global Health Impacts of Vector-Borne Diseases. Washington, D.C.: National Academies Press, 2016. http://dx.doi.org/10.17226/21792.

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Aksoy, Serap, ed. Transgenesis and the Management of Vector-Borne Disease. New York, NY: Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-78225-6.

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Takken, Willem, and Bart G. J. Knols, eds. Emerging pests and vector-borne diseases in Europe. The Netherlands: Wageningen Academic Publishers, 2007. http://dx.doi.org/10.3920/978-90-8686-626-7.

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India) Symposium on Vectors and Vector Borne Diseases (11th 2011 Jabalpur. XI Symposium on Vectors and Vector Borne Diseases, 15th - 17th October 2011: Abstracts. Jabalpur: Regional Medical Research Centre for Tribals, Indian Council of Medical Research, 2011.

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WHO Study Group on Vector Control for Malaria and Other Mosquito-borne Diseases. Vector control for malaria and other mosquito-borne diseases. Geneva: World Health Organization, 1995.

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K, Panigrahi Srikanta, and Anand Mona, eds. Vector borne diseases in India: Environmental health & policy perspectives. New Delhi: Manak Publications, 2007.

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Garros, Claire, Jérémy Bouyer, Willem Takken, and Renate C. Smallegange, eds. Pests and vector-borne diseases in the livestock industry. The Netherlands: Wageningen Academic Publishers, 2018. http://dx.doi.org/10.3920/978-90-8686-863-6.

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Book chapters on the topic "Vector Borne"

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Sharma, Satyavan. "Vector-borne diseases." In Progress in Drug Research / Fortschritte der Arzneimittelforschung / Progrès des recherches pharmaceutiques, 365–485. Basel: Birkhäuser Basel, 1990. http://dx.doi.org/10.1007/978-3-0348-7133-4_8.

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Martcheva, Maia. "Vector-Borne Diseases." In Texts in Applied Mathematics, 67–89. Boston, MA: Springer US, 2015. http://dx.doi.org/10.1007/978-1-4899-7612-3_4.

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Wilder-Smith, Annelies. "Vector-borne diseases." In Essential Travel Medicine, 65–73. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118597361.ch7.

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Seifert, Horst S. H. "Vector-borne Diseases." In Tropical Animal Health, 149–270. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-0147-6_5.

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Zolnikov, Tara Rava. "Vector-Borne Disease." In Autoethnographies on the Environment and Human Health, 113–26. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-69026-1_9.

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Müller, Ruth, Friederike Reuss, Vladimir Kendrovski, and Doreen Montag. "Vector-Borne Diseases." In Biodiversity and Health in the Face of Climate Change, 67–90. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-02318-8_4.

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Dantas-Torres, Filipe, and Domenico Otranto. "Vector-Borne Zoonoses." In Zoonoses - Infections Affecting Humans and Animals, 683–95. Dordrecht: Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-017-9457-2_27.

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Balakrishnan, Indran, and Stephen H. Gillespie. "Vector-Borne Parasitic Diseases." In Principles and Practice of Travel Medicine, 91–124. Chichester, UK: John Wiley & Sons, Ltd, 2002. http://dx.doi.org/10.1002/0470842512.ch8.

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Goddard, Jerome. "Miscellaneous Vector-Borne Diseases." In Infectious Diseases and Arthropods, 153–76. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-400-5_7.

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Wall, Emma C., and Peter L. Chiodini. "Vector-Borne Parasitic Diseases." In Principles and Practice of Travel Medicine, 112–25. Oxford, UK: Wiley-Blackwell, 2013. http://dx.doi.org/10.1002/9781118392058.ch9.

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Conference papers on the topic "Vector Borne"

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ROGERS, DAVID. "NEW APPROACHES FOR STUDYING VECTORS AND VECTOR-BORNE DISEASES." In International Seminar on Nuclear War and Planetary Emergencies 40th Session. WORLD SCIENTIFIC, 2009. http://dx.doi.org/10.1142/9789814289139_0033.

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Barker, Michelle, Donald Brower, and Natalie Meyers. "Vector-Borne Disease Network digital library." In 2014 IEEE/ACM Joint Conference on Digital Libraries (JCDL). IEEE, 2014. http://dx.doi.org/10.1109/jcdl.2014.6970212.

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Gerardo, Elizabeth. "Combating vector borne disease in the Pacific." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.106690.

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Ajraldi, Valerio, Andrea Costamagna, and Ezio Venturino. "A Simple Model for Vector‐Borne Ecoepidemics." In NUMERICAL ANALYSIS AND APPLIED MATHEMATICS: International Conference on Numerical Analysis and Applied Mathematics 2008. American Institute of Physics, 2008. http://dx.doi.org/10.1063/1.2991097.

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Anguelov, Roumen, Jean Lubuma, and Yves Dumont. "Mathematical analysis of vector-borne diseases on plants." In 2012 IEEE 4th International Symposium on Plant Growth Modeling, Simulation, Visualization and Applications (PMA). IEEE, 2012. http://dx.doi.org/10.1109/pma.2012.6524808.

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Raizada, Sandali, Shuchi Mala, and Achyut Shankar. "Vector Borne Disease Outbreak Prediction by Machine Learning." In 2020 International Conference on Smart Technologies in Computing, Electrical and Electronics (ICSTCEE). IEEE, 2020. http://dx.doi.org/10.1109/icstcee49637.2020.9277286.

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NETESOV, SERGEY. "VECTOR-BORNE DISEASES IN THE ASIAN PART OF RUSSIA." In International Seminar on Nuclear War and Planetary Emergencies 40th Session. WORLD SCIENTIFIC, 2009. http://dx.doi.org/10.1142/9789814289139_0037.

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Ghaffari, Meysam, Ashok Srinivasan, Anuj Mubayi, Xiuwen Liu, and Krishnan Viswanathan. "Next-generation high-resolution vector-borne disease risk assessment." In ASONAM '19: International Conference on Advances in Social Networks Analysis and Mining. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3341161.3343694.

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Bowden, Sarah. "Community ecology of mosquito vectors: Linking larval competition, climate change, and vector-borne disease." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.94716.

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McAllister, Janet. "Entomology at CDC: Protecting the public from vector-borne diseases." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.103181.

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Reports on the topic "Vector Borne"

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Ross, S. G., M. C. Thomson, and T. Pultz. RADARSAT-1 for Monitoring Vector-borne Diseases in Tropical Environments: A Review. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2001. http://dx.doi.org/10.4095/219826.

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VanderNoot, Victoria A., Deanna Joy Curtis, Chung-Yan Koh, Benjamin H. Brodsky, and Todd Lane. Enhanced vector borne disease surveillance of California Culex mosquito populations reveals spatial and species-specific barriers of infection. Office of Scientific and Technical Information (OSTI), August 2014. http://dx.doi.org/10.2172/1154713.

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Ibáñez, Ana María, Sandra Rozo, and Maria J. Urbina. Forced Migration and the Spread of Infectious Diseases. Inter-American Development Bank, November 2020. http://dx.doi.org/10.18235/0002894.

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Abstract:
We examine the role of Venezuelan forced migration on the propagation of 15 infectious dis-eases in Colombia. For this purpose, we use rich municipal-monthly panel data. We exploit the fact that municipalities closer to the main migration entry points have a disproportionate ex-posure to infected migrants when the cumulative migration flows increase. We find that higher refugee inflows are associated with increments in the incidence of vaccine-preventable dis-eases, such as chickenpox and tuberculosis, as well as sexually transmitted diseases, including AIDS and syphilis. However, we find no significant effects of migration on the propagation of vector-borne diseases. Contact with infected migrants upon arrival seems to be the main driving mechanism.
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