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Journal articles on the topic 'Venous thromboembolism'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 November 2024

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1

Nitta, Satoshi, Koji Kawai, Tomokazu Kimura, Takashi Kawahara, Shuya Kandori, Akio Hoshi, Takahiro Kojima, and Hiroyuki Nishiyama. "Predictors of venous thromboembolism development before and during chemotherapy for advanced germ cell tumor." Japanese Journal of Clinical Oncology 50, no. 3 (February 9, 2020): 338–43. http://dx.doi.org/10.1093/jjco/hyz177.

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Abstract Objective We retrospectively analyzed the incidence and localization of venous thromboembolism in patients undergoing chemotherapy for advanced germ cell tumor and separately evaluated the risk factors for venous thromboembolism development before and during chemotherapy. Methods We included 121 patients treated with cisplatin-based chemotherapy between 2005 and 2018. Venous thromboembolism was defined as venous thrombosis diagnosed using radiological imaging with or without thromboembolic symptoms. We analyzed the clinical parameters for identifying the possible venous thromboembolism risk factors. Khorana score was used to calculate the venous thromboembolism risk. Results Thirteen patients showed prechemotherapy venous thromboembolism and 13 developed venous thromboembolism during chemotherapy. The most common venous thromboembolism was deep vein thrombosis (10 patients), followed by inferior vena cava thrombus (eight patients) and pulmonary thrombus (six patients). Compared to the group without venous thromboembolism, the group with prechemotherapy venous thromboembolism showed higher proportion of patients with tumors originating in the right testis (10 out of 13), significantly higher lactate dehydrogenase levels (828 IU/L versus 436 IU/L, P = 0.013), significantly higher proportion of patients with retroperitoneal lymph node (RPLN) metastases >5 cm in diameter (76.9% versus 33.7%, P = 0.003) and slightly higher proportion of patients with high-risk Khorana score (≥ 3; 30.8% versus 11.6%). No significant differences were observed between the clinical characteristics of patients with venous thromboembolism developed during chemotherapy and patients without venous thromboembolism. Conclusions We show that both RPLN mass > 5 cm and high lactate dehydrogenase levels are significant risk factors for prechemotherapy venous thromboembolism but not for venous thromboembolism development during chemotherapy.

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2

Shigematsu, K. "Diagnosis of Venous Thromboembolism(Venous Thromboembolism)." Iryou kikigaku (The Japanese journal of medical instrumentation) 78, no. 11 (2008): 866–72. http://dx.doi.org/10.4286/jjmi.78.866.

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3

Sakon, M. "Treatment of Venous Thromboembolism(Venous Thromboembolism)." Iryou kikigaku (The Japanese journal of medical instrumentation) 78, no. 11 (2008): 873–77. http://dx.doi.org/10.4286/jjmi.78.873.

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4

Marinkovic, Jovana, and Olivera Tarabar. "Multiple myeloma and deep vein thrombosis/pulmonary thromboembolism - incidence and risk factors." Medical review 75, no. 7-8 (2022): 230–36. http://dx.doi.org/10.2298/mpns2208230m.

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Introduction. Deep vein thrombosis and pulmonary thromboembolism are among the most important causes of morbidity and mortality in cancer patients. They are common and serious complications that are affected by various risk factors. The aim of this study was to determine the incidence of thromboembolic complications and their association with risk factors in patients with newly diagnosed multiple myeloma. Material and Methods. A retrospective study included 32 patients and the following variables were collected: age, C-reactive protein, beta-2 microglobulin, hemoglobin, platelets, total proteins, nephrotic syndrome, fibrinogen, D-dimer, albumin, lactate dehydrogenase, creatinine, calcium, gender, performance status, type of multiple myeloma, clinical stage, and applied therapy. All variables were compared between two groups of patients: group with thromboembolic complications (deep venous thrombosis and pulmonary thromboembolism), and the group without these complications. Results. The study sample included 18 men and 14 women. The patients? age ranged from 36 to 73 years. Of the 32 patients, 6 had deep venous thrombosis of the lower extremities, and 2 patients had deep venous thrombosis and pulmonary thromboembolism. The only two biomarkers that showed an association with venous thromboembolism in our patients with myeloma were elevated levels of C-reactive protein and D-dimer. Conclusion. The incidence of thromboembolic complications in patients with newly diagnosed multiple myeloma was 25%. When making the diagnosis of myeloma, risk factors that were significantly associated with venous thromboembolism were elevated levels of C-reactive protein and D-dimer. The occurrence of venous thromboembolism in patients with multiple myeloma was not associated with significantly higher mortality compared to patients without venous thromboembolism.

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5

Kambouche, Faouzia. "Pulmonary Tuberculosis and Venous Thromboembolism." International Journal of Science and Research (IJSR) 12, no. 8 (August 5, 2023): 1891. http://dx.doi.org/10.21275/sr23821151939.

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6

Kurokami, Tsunehiko, Reiko Takasawa, Sayaka Takeda, Masashi Kurobe, Kei Takasawa, Masato Nishioka, and Masayuki Shimohira. "Venous thromboembolism in two adolescents with Down syndrome." Turkish Journal of Pediatrics 60, no. 4 (August 25, 2018): 429–32. http://dx.doi.org/10.24953/turkjpediatr.2018.879.

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Although venous thromboembolic events are relatively rare in children, they are an increasingly recognized clinical entity in pediatric tertiary care hospitals. Although vascular disorders are prevalent with Down syndrome, it remains unclear whether Down syndrome patients are at higher risk for venous thromboembolic events. We report two adolescent cases with Down syndrome who unexpectedly developed venous thromboembolism in a general care unit. Our cases had a few risk factors; laparoscopic radical surgery for Hirschsprung's disease with central venous catheterisation in Case 1, and bacterial hepatic abscess in Case 2. Despite preventive heparinization with catheterisation and minor surgery in Case 1 and non-sepsis in Case 2, bed rest for only a few days triggered sudden onset of deep vein thrombosis in lower limbs with pulmonary thromboembolism in both cases. We speculate that the characteristics of Down syndrome, including physical and behavioural problems, might cause venous thromboembolic events. Thus, we should pay more attention to the relationship specifically between venous thromboembolism and Down syndrome, especially in adolescents, and increase prevention, early detection and treatment efforts.

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7

Armstrong, Emily M., Jessica M. Bellone, Lori B. Hornsby, Sarah Treadway, and Haley M. Phillippe. "Pregnancy-Related Venous Thromboembolism." Journal of Pharmacy Practice 27, no. 3 (April 17, 2014): 243–52. http://dx.doi.org/10.1177/0897190014530425.

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Pregnancy is associated with an increased risk of venous thromboembolism (VTE), with a reported incidence ranging from 0.49 to 2 events per 1000 deliveries. Risk factors include advanced maternal age, obesity, smoking, and cesarian section. Women with a history of previous VTE are at a 4-fold higher risk of recurrent thromboembolic events during subsequent pregnancies. Additionally, the presence of concomitant thrombophilia, particularly factor V Leiden (homozygosity), prothrombin gene mutation (homozygosity), or antiphospholipid syndrome (APS), increases the risk of pregnancy-related VTE. Low-molecular-weight heparin (LMWH) and unfractionated heparin (UFH) are the drugs of choice for anticoagulation during pregnancy. LMWH is preferred due to ease of use and lower rates of adverse events. Women with high thromboembolic risk particularly those with a family history of VTE should receive antepartum thromboprophylaxis. Women with low thromboembolic risk or previous VTE caused by a transient risk factor (ie, provoked), who have no family history of VTE, may undergo antepartum surveillance. Postpartum anticoagulation can be considered in women with both high and low thromboembolic risk.

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8

DÜZ, Özge Arıcı, Oktay OLMUŞÇELİK, Ali İhsan GEMİCİ, and Özlem SAATÇİ SANCAKTEPE. "Evaluation of pai-1 polymorphisms in central and peripheral thromboembolies." Journal of Experimental and Clinical Medicine 38, no. 2 (March 14, 2021): 167–71. http://dx.doi.org/10.52142/omujecm.38.2.20.

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Thromboembolism is a clinical finding that occurs due to thrombus; formed in the vascular system and has various etiological factors. It can be classified as central and peripheral thromboembolism. Our objective in this study is to explore genetic risk factors in central and peripheral thromboembolism and reveal the differences. 342 thromboembolism patients were retrospectively included to the study between January 2016 and December 2019. Demographic characteristics, risk factors for thromboembolism and genetic mutations in central and peripheral thromboembolism groups were overviewed. The genetic mutations evaluated in patients were Factor V Leiden G1691A, Factor V HR1299R, Factor II (Prothrombin) G20210A, MTHFR (Methylenetetrahydrofolate reductase) C677T, MTHFR A1298C, PAI 4G/5G. Within the scope of the study, genetic analyzes of 106 patients were reached and included in the study. Seventy-two central thromboembolism (69.8%), 34 (31.2%) peripheral thromboembolisms were detected. Sixty-three of the central thromboembolisms were from arterial and nine were from venous origin. There was no significant difference between age, gender and risk factors of central thromboembolism and peripheral thromboembolism patients (p˃0.05), but smoking was more common in central thromboembolism patients (p: 0.041). 4G/5G polymorphism was observed more frequently in patients with central thromboembolism (p: 0.039). Thromboembolism is a multifactorial disease, PAI-1 4G/5G polymorphism is a medium risk factor for thromboembolism. We conclude that PAI-1 4G/5G polymorphism is more frequent in central thromboembolism than peripheral thromboembolism and its evaluation can give more information about the thromboembolic risk analyze.

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9

Katı, O., O. K. Bakkaloglu, I. Hatemi, A. F. Celik, E. Seyahi, and Y. Erzin. "P1029 Prevalance and risk factors of thromboembolism in Inflammatory Bowel Disease." Journal of Crohn's and Colitis 18, Supplement_1 (January 1, 2024): i1857. http://dx.doi.org/10.1093/ecco-jcc/jjad212.1159.

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Abstract Background Inflammatory bowel disease (IBD), as a chronic inflammatory condition, can affect atherosclerotic process, arterial events, and increase the formation of venous thromboembolism. The aim of this study was to determine the frequency of thromboembolism and the risk factors associated with acute thromboembolism in our IBD cohort. Methods A total of 3133 patients with 1414 Crohn's Disease (CD), 1667 Ulcerative Colitis (UC) and 52 Indeterminate Colitis who were admitted between 1999 and 2021 were retrospectively analyzed. Patients with acute arterial events and venous thromboembolism (n=39) during the follow-up period were compared with a control group (n=78) of the same gender and same diagnosis. Patients with Behçet's syndrome (n=126),systemic vasculitis (n=16) and hereditary thrombophilia (n=5) were excluded. Results Among 3133 IBD patients, number of patients with arterial events or venous thromboembolism at any time was 124, and the frequency was 3,95%. A total of 132 thromboembolic events, 86 arterial (65,2%) and 46 venous (34,8%) were recorded.Coronary artery disease was significantly higher in the UC group (2,3% vs. 1,2%; p=0.028). During follow-up, 40 acute thromboembolic events, 25 arterial and 15 venous, were seen in 39 patients (frequency 1,24%). There was no significant difference between the CD and UC groups in terms of acute thromboembolism (p=0.871). The presence of exacerbation, the presence of hospitalization, the number of priot hospitalizations and clinical activity at the last visit were associated with both arterial and venous thromboembolism; smoking history, age at diagnosis, body-mass index, non-mucosal CD (Montreal B2-B3) and higher basal CRP were associated with arterial thromboembolism only; higher CRP at the last visit, annual exacerbation frequency, presence of an additional inflammatory disease, presence of complications (surgery, abcess), longer total steroid traetment duration and also recent steroid therapy were associated only with venous thromboembolism. In regression analysis, recent steroid therapy and additional inflammatory disease were considered as independent risk factors for venous thromboembolism whereas age at diagnosis of IBD was considered as an independent risk factor for arterial events. Conclusion In addition to risk factors such as age, smoking and obesity, parameters indicating disease activity, inflammatory comorbidities and recent steroid therapy appear to be associated with acute thromboembolism in IBD patients.

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10

Morshed, Ghada, and Nader Zaki. "Adequate timing of thromboembolic prophylaxis in colorectal cancer surgery." International Surgery Journal 5, no. 2 (January 25, 2018): 576. http://dx.doi.org/10.18203/2349-2902.isj20180355.

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Background: It has been found that patients with colorectal cancer are at increased risk for postoperative venous thromboembolism. The aim of this prospective study is to evaluate the incidence of venous thromboembolism and major bleeding complications in patients undergoing colorectal cancer who are treated with preoperative or postoperative venous thromboprophylaxis.Methods: This prospective study included 30 patients from September 2013 to November 2017. There were 22 males and 8 females; mean age was 66.7±5.5 years (range 44-78). Author divided the patients randomly into two groups (group A=15 cases with preoperative and group B = 15 cases with postoperative venous thromboprophylaxis).Results: There was no significant difference in preoperative versus postoperative thromboembolic prophylaxis regarding postoperative DVT 0/15(0%) vs 1/15 (6.6%), P=0.69, no bleeding complications and no pulmonary embolism.Conclusions: Preoperative and postoperative thromboembolic prophylaxis are equally safe in venous thromboembolism protection.

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11

Khan, Faizan, Tobias Tritschler, Susan R. Kahn, and Marc A. Rodger. "Venous thromboembolism." Lancet 398, no. 10294 (July 2021): 64–77. http://dx.doi.org/10.1016/s0140-6736(20)32658-1.

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12

MacKellar, Rachel. "VENOUS THROMBOEMBOLISM." Journal of the Australasian Society of Aerospace Medicine 12 (2020): 25–30. http://dx.doi.org/10.21307/asam-2020-005.

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13

Brumfield, Elisha J. "Venous Thromboembolism." Kansas Journal of Medicine 1, no. 1 (November 8, 2007): 20–21. http://dx.doi.org/10.17161/kjm.v1i1.10738.

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14

Thomas, Dwayne A., Bennett P. deBoisblanc, and Warren R. Summer. "Venous thromboembolism." Postgraduate Medicine 102, no. 4 (October 1997): 179–94. http://dx.doi.org/10.3810/pgm.1997.10.340.

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15

Holman, Russell L. "Venous thromboembolism." Postgraduate Medicine 108, no. 4 (September 15, 2000): 19–20. http://dx.doi.org/10.3810/pgm.2000.09.15.1229.

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16

Anderson, Frederick A., and H. Brownell Wheeler. "VENOUS THROMBOEMBOLISM." Clinics in Chest Medicine 16, no. 2 (June 1995): 235–51. http://dx.doi.org/10.1016/s0272-5231(21)01089-3.

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17

Shapiro, Susan, Tamara Everington, Lara Roberts, and Roopen Arya. "Venous thromboembolism." Clinical Medicine 19, no. 3 (May 2019): 262.1–262. http://dx.doi.org/10.7861/clinmedicine.19-3-262a.

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18

Broccard, Alain F. "VENOUS THROMBOEMBOLISM." Shock 21, no. 5 (May 2004): 493. http://dx.doi.org/10.1097/00024382-200405000-00018.

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19

Moser, Kenneth M. "Venous Thromboembolism." American Review of Respiratory Disease 141, no. 1 (January 1990): 235–49. http://dx.doi.org/10.1164/ajrccm/141.1.235.

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20

Skolnik, Neil S. "Venous Thromboembolism." American Review of Respiratory Disease 141, no. 6 (June 1990): 1603–4. http://dx.doi.org/10.1164/ajrccm/141.6.1603b.

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21

Sakuma, Masahito, Mashio Nakamura, Norikazu Yamada, Satoshi Ota, Kunio Shirato, Takeshi Nakano, Masaaki Ito, and Takao Kobayashi. "Venous Thromboembolism." Circulation Journal 73, no. 2 (2009): 305–9. http://dx.doi.org/10.1253/circj.cj-08-0372.

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22

Blann, Andrew D., and Gregory Y. H. Lip. "Venous thromboembolism." BMJ 332, no. 7535 (January 26, 2006): 215–19. http://dx.doi.org/10.1136/bmj.332.7535.215.

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23

Otoya, Jorge, Albert A. Nemcek, and David Green. "Venous Thromboembolism." Chest 96, no. 5 (November 1989): 1169–74. http://dx.doi.org/10.1378/chest.96.5.1169.

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24

Zhu, Tienan, Isabelle Martinez, and Joseph Emmerich. "Venous Thromboembolism." Arteriosclerosis, Thrombosis, and Vascular Biology 29, no. 3 (March 2009): 298–310. http://dx.doi.org/10.1161/atvbaha.108.182428.

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25

Wolberg, Alisa S., and Nigel Mackman. "Venous Thromboembolism." Arteriosclerosis, Thrombosis, and Vascular Biology 29, no. 3 (March 2009): 296–97. http://dx.doi.org/10.1161/atvbaha.109.184580.

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26

Nelson Worel, Jane. "Venous Thromboembolism." Journal of Cardiovascular Nursing 24, Supplement (November 2009): S14—S19. http://dx.doi.org/10.1097/jcn.0b013e3181b85c7b.

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27

Long, Janet B. "Venous Thromboembolism." Journal of Cardiovascular Nursing 24, Supplement (November 2009): S8—S13. http://dx.doi.org/10.1097/jcn.0b013e3181b85c91.

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28

Merli, Geno J. "Venous Thromboembolism." Journal of Cardiovascular Nursing 24, Supplement (November 2009): S1—S3. http://dx.doi.org/10.1097/jcn.0b013e3181b85ca6.

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29

Mason, Carol. "Venous Thromboembolism." Journal of Cardiovascular Nursing 24, Supplement (November 2009): S4—S7. http://dx.doi.org/10.1097/jcn.0b013e3181b85cbb.

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30

Bakkum-Gamez, Jamie N., and Sean C. Dowdy. "Venous Thromboembolism." Obstetrics & Gynecology 129, no. 6 (June 2017): 971–73. http://dx.doi.org/10.1097/aog.0000000000002073.

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31

Harris, Caroline, Christine Sulmers, Kathleen Groesch, Teresa Wilson, Kristin Delfino, and Funminiyi Taylor. "Venous Thromboembolism." Obstetrics & Gynecology 127 (May 2016): 88S. http://dx.doi.org/10.1097/01.aog.0000483795.00678.28.

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32

Davis, Brittany, and Anita Hadpawat-Lee. "Venous Thromboembolism." Obstetrics & Gynecology 129 (May 2017): 114S. http://dx.doi.org/10.1097/01.aog.0000514605.93806.28.

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33

Sanford, David B. "Venous thromboembolism." OR Nurse 7, no. 4 (July 2013): 32–39. http://dx.doi.org/10.1097/01.orn.0000431585.98335.02.

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&NA;. "Venous thromboembolism." OR Nurse 7, no. 4 (July 2013): 39–40. http://dx.doi.org/10.1097/01.orn.0000432341.19720.5c.

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35

Brenner, David W., Martin A. Fogle, and Paul F. Schellhammer. "Venous Thromboembolism." Journal of Urology 142, no. 6 (December 1989): 1403–11. http://dx.doi.org/10.1016/s0022-5347(17)39111-5.

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36

SKOLNIK, NEIL S., and MATTHEW R. GERSTBERGER. "Venous Thromboembolism." Family Practice News 37, no. 23 (December 2007): 10. http://dx.doi.org/10.1016/s0300-7073(07)71397-x.

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37

Brauer, Sandra. "Venous thromboembolism." Journal of Physiotherapy 56, no. 4 (2010): 281. http://dx.doi.org/10.1016/s1836-9553(10)70063-1.

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38

HYERS, THOMAS M. "Venous Thromboembolism." American Journal of Respiratory and Critical Care Medicine 159, no. 1 (January 1999): 1–14. http://dx.doi.org/10.1164/ajrccm.159.1.9803109.

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39

Chertow, Glenn M., and Kenneth W. Mahaffey. "Venous Thromboembolism." Circulation 126, no. 16 (October 16, 2012): 1937–38. http://dx.doi.org/10.1161/circulationaha.112.138057.

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40

Dalen, James E., John A. Paraskos, Ira S. Ockene, Joseph S. Alpert, and Jack Hirsh. "Venous Thromboembolism." Chest 89, no. 5 (May 1986): 370S—373S. http://dx.doi.org/10.1378/chest.89.5_supplement.370s.

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&NA;. "Venous Thromboembolism." Journal of Neuroscience Nursing 33, no. 4 (August 2001): 220. http://dx.doi.org/10.1097/01376517-200108000-00013.

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42

Becker, Daniel M. "Venous thromboembolism." Journal of General Internal Medicine 1, no. 6 (November 1986): 402–11. http://dx.doi.org/10.1007/bf02596427.

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43

Markel, David C., Sally York, Michael J. Liston, Jeffrey C. Flynn, C. Lowry Barnes, and Charles M. Davis. "Venous Thromboembolism." Journal of Arthroplasty 25, no. 1 (January 2010): 3–9. http://dx.doi.org/10.1016/j.arth.2009.07.021.

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44

Gera, Renuka, and Roshni Kulkarni. "Venous thromboembolism." Indian Journal of Pediatrics 66, no. 2 (March 1999): 163–69. http://dx.doi.org/10.1007/bf02761196.

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Tapson, Victor F. "Venous thromboembolism." Clinics in Chest Medicine 24, no. 1 (March 2003): xi—xiii. http://dx.doi.org/10.1016/s0272-5231(02)00080-1.

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46

Bounameaux, Henri, and Frits R. Rosendaal. "Venous Thromboembolism." Circulation 123, no. 20 (May 24, 2011): 2189–91. http://dx.doi.org/10.1161/circulationaha.111.031690.

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47

Gorski, Lisa A. "Venous Thromboembolism." Home Healthcare Nurse: The Journal for the Home Care and Hospice Professional 25, no. 2 (February 2007): 94–100. http://dx.doi.org/10.1097/00004045-200702000-00009.

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&NA;. "Venous Thromboembolism." Home Healthcare Nurse: The Journal for the Home Care and Hospice Professional 25, no. 2 (February 2007): 101–2. http://dx.doi.org/10.1097/00004045-200702000-00010.

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49

Fitzgerald, Jan. "Venous Thromboembolism." Orthopaedic Nursing 29, no. 4 (July 2010): 226–34. http://dx.doi.org/10.1097/nor.0b013e3181e517af.

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&NA;. "Venous Thromboembolism." Orthopaedic Nursing 29, no. 4 (July 2010): 235–36. http://dx.doi.org/10.1097/nor.0b013e3181eb1aeb.

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