Academic literature on the topic 'Viral co-infection'

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Dissertations / Theses on the topic "Viral co-infection"

1

Falconer, Karolin. "HIV-1/HCV co-infection immunity and viral dynamics /." Stockholm, 2010. http://diss.kib.ki.se/2010/978-91-7409-762-7/.

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2

Lissauer, Samantha Mary. "Modelling hepatitis C viral host interaction and co-infection." Thesis, University of Birmingham, 2018. http://etheses.bham.ac.uk//id/eprint/8774/.

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Hepatitis C Virus (HCV) is a clinically important infection that leads to chronic liver disease and Human Immunodeficiency Virus (HIV) co-infected patients have more rapid progression to severe liver disease and show higher rates of HCV vertical transmission. Hepatocytes are a highly differentiated cell type and support low level HCV replication. Most studies of the viral life cycle use de-differentiated hepatoma cell lines, which are highly permissive. The mechanism behind this difference is poorly understood. We show that dimethylsulfoxide (DMSO) differentiated Huh-7 cells have a 100-fold re
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Danta, Mark. "Acute hepatitis C infection in HIV co-infection : epidemiology, natural history and the host-viral responses." Thesis, University College London (University of London), 2006. http://discovery.ucl.ac.uk/1445482/.

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Aim: To analyse the epidemiological, clinical and immunological aspects of a recent epidemic of acute HCV in HIV co-infected individuals.;Methods: The epidemiology was characterised using combined molecular and clinical studies. A phylogenetic analysis of the E1/E2 region of the HCV genome was performed to determine HCV transmission. Transmission risk factors were then explored in a multicentre case-control study. Data was collected on the early clinical course of HCV in HIV. Longitudinal cell-mediated responses to HCV and HCV evolution were studied with PBMCs and serum collected during the ac
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4

Powell, Eleanor A. "Viral Factors Associated with Occult HBV in HIV-positive South Africans." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439561912.

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5

Long, Elizabeth Michelle. "Genetic and co-receptor characterization of viral diversity early in human immunodeficiency virus type 1 infection /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/4997.

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6

Auma, Ann Winniefred Nangobi. "THE IMPACT OF DIRECT-ACTING ANTI-VIRAL THERAPY ON NAIVE CD4+ T CELL LYMPHOPENIA AND CELLULAR IMMUNE ACTIVATION IN HCV INFECTION AND HCV/HIV CO-INFECTION." Case Western Reserve University School of Graduate Studies / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1625764728651756.

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7

Deka, Srilekha. "Induction of the Chlamydia trachomatis Persistent State by Herpes simplex Virus Type 2 Does Not Require Productive Viral Replication." Digital Commons @ East Tennessee State University, 2005. https://dc.etsu.edu/etd/1065.

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Clinical and epidemiological studies have shown that multiple infection with HSV-2 and Chlamydia trachomatis occurs in vivo. We hypothesize that (i) HSV-2 co-infection of C. trachomatis infected cervical epithelial cells induces chlamydial persistence; (ii) productive HSV-2 replication is required for induction of persistence in C. trachomatis; (iii) co-infection with C. trachomatis and HSV-2 alters accumulation of immuno-modulatory molecules in infected cells. To test these hypotheses, a cell culture model of co-infection was established in HeLa cells. Transmission electron microscopic (TEM)
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Young, Sean Gregory. "Landscape genetics of avian influenza (H5N1 and H9N2) in Egyptian poultry from 2006-2015: co-infection, key substitutions, and viral diffusion." Diss., University of Iowa, 2017. https://ir.uiowa.edu/etd/5687.

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With a case fatality rate higher than the 1918 Spanish Flu pandemic, H5N1 highly pathogenic avian influenza represents a threat to global public health. Efforts to identify locations with the greatest potential for pandemic emergence, as well as how the virus is spreading, may help minimize this threat. First detected in Egypt in 2006, H5N1 viruses have resulted in the deaths of millions of birds in both commercial and backyard poultry flocks, and more than 350 human infections, the most of any country, have been confirmed. Human outbreaks have been so far constrained by poor viral adaptation
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9

Tenckhoff, Solveig. "Einfluss der GBV-C-Infektion auf die HIV-1-Replikation." Doctoral thesis, Universitätsbibliothek Leipzig, 2012. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-89518.

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Das 1995 entdeckte GB-Virus C (GBV-C) gehört als Pegivirus zur Familie der Flaviviridae und ist nichtpathogen. In Industrieländern sind 2 bis 12,5 % der gesunden Bevölkerung und bis zu 45 % der Personen aus Risikokollektiven, z.B. Patienten mit Infektionen mit dem humanen Immundefizienzvirus Typ 1 (HIV-1) oder dem Hepatitis-C-Virus (HCV), virämisch. Die Mehrzahl der klinischen Studien und Metaanalysen zu GBV-C/HIV-1-Koinfektionen zeigten, dass GBV-C mit einem verlangsamten Krankheitsverlauf und einer erhöhten Überlebenswahrscheinlichkeit von GBV-C/HIV-1-koinfizierten Patienten korreliert. In d
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10

Vicente, Sobrinho Ludmila Silva [UNESP]. "Leishmaniose felina e sua associação com imunodeficiência viral e toxoplasmose em gatos provenientes de área endêmica para leishmaniose visceral." Universidade Estadual Paulista (UNESP), 2010. http://hdl.handle.net/11449/94718.

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Made available in DSpace on 2014-06-11T19:27:18Z (GMT). No. of bitstreams: 0 Previous issue date: 2010-08-20Bitstream added on 2014-06-13T19:55:44Z : No. of bitstreams: 1 sobrinho_lsv_me_araca.pdf: 921356 bytes, checksum: 29d3e58899a35e5de14b32e263c2fe77 (MD5)<br>Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)<br>O objetivo do presente estudo foi determinar em uma população de 302 gatos provenientes de área endêmica para leishmaniose visceral, a prevalência da infecção por Leishmania spp. e a presença de coinfecção pelo Toxoplasma gondii, vírus da imunodeficiência felina (FIV)
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