Relevant bibliographies by topics / Viral quasi-species

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Academic literature on the topic 'Viral quasi-species'

Author: Grafiati
Published: 7 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "Viral quasi-species"

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Lim, Seng Gee, Yan Cheng, Stephane Guindon, et al. "Viral Quasi-Species Evolution During Hepatitis Be Antigen Seroconversion." Gastroenterology 133, no. 3 (2007): 951–58. http://dx.doi.org/10.1053/j.gastro.2007.06.011.

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Moussaoui, Ali, and Vitaly Volpert. "The impact of immune cell interactions on virus quasi-species formation." Mathematical Biosciences and Engineering 21, no. 11 (2024): 7530–53. http://dx.doi.org/10.3934/mbe.2024331.

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<p>The process of viral infection spreading in tissues was influenced by various factors, including virus replication within host cells, transportation, and the immune response. Reaction-diffusion systems provided a suitable framework for examining this process. In this work, we studied a nonlocal reaction-diffusion system of equations that modeled the distribution of viruses based on their genotypes and their interaction with the immune response. It was shown that the infection may persist at a certain level alongside a chronic immune response, exhibiting spatially uniform or oscillator
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R., Jaisankar, and Deepa S. "On Estimating the Time from HIV Infection to AIDS - A Stochastic Modeling Approach." Indian Journal of Science and Technology 18, no. 15 (2025): 1170–76. https://doi.org/10.17485/IJST/v18i15.63.

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Abstract <strong>Objectives:</strong>&nbsp;To construct a statistical model through which one can estimate the mean time to get AIDS condition in a HIV infected individual.&nbsp;<strong>Methods:</strong>&nbsp;The methodology adopted for the construction of the statistical model for estimating the time to get AIDS condition considers the antigenic diversity threshold and the antigenic diversity induced by the virus entered through every individual infected sexual contact and the antigenic diversity induced by their corresponding quasi &ndash; species. A comparison has been made with the existin
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Nieto-Rabiela, F., G. Suzán, A. Wiratsudakul, and O. Rico-Chávez. "Viral metacommunities associated to bats and rodents at different spatial scales." Community Ecology 19, no. 2 (2018): 168–75. https://doi.org/10.5281/zenodo.13531157.

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(Uploaded by Plazi for the Bat Literature Project) One of the main goals of community ecology is to measure the relative importance of environmental filters to understand patterns of species distribution at different temporal and spatial scales. Likewise, the identification of factors that shape symbiont metacommunity structures is important in disease ecology because resulting structures drive disease transmission. We tested the hypothesis that distributions of virus species and viral families from rodents and bats are defined by shared responses to host phylogeny and host functional characte
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Nieto-Rabiela, F., G. Suzán, A. Wiratsudakul, and O. Rico-Chávez. "Viral metacommunities associated to bats and rodents at different spatial scales." Community Ecology 19, no. 2 (2018): 168–75. https://doi.org/10.5281/zenodo.13531157.

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(Uploaded by Plazi for the Bat Literature Project) One of the main goals of community ecology is to measure the relative importance of environmental filters to understand patterns of species distribution at different temporal and spatial scales. Likewise, the identification of factors that shape symbiont metacommunity structures is important in disease ecology because resulting structures drive disease transmission. We tested the hypothesis that distributions of virus species and viral families from rodents and bats are defined by shared responses to host phylogeny and host functional characte
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Fahim, Qanita, Irfan Ali Mirza, Ayesha Khalid, Anum Imtiaz, and Asad Ahmad. "THE HEPATITIS C UNTYPE-ABLE GENOTYPES, AN EMERGENCE OF QUASI-SPECIES IN HCV INFECTED PATIENTS." PAFMJ 71, Suppl-1 (2021): S106–12. http://dx.doi.org/10.51253/pafmj.v71isuppl-1.2603.

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Objective: To determine the frequency of Type-able and untype-able genotypes in hepatitis C infected patients,and to observe their association with gender, age, Alanine Aminotranferease and viral load.&#x0D; Study Design: Cross-sectional analytical study.&#x0D; Place and Duration of Study: Department of Microbiology, Combined Military Hospital, and Lahore, Pakistanfrom Sep 2017 to Mar 2018.&#x0D; Methodology: Six hundred forty seven anti HCV antibodies positive serum samples by Enzyme Linked ImmunoSorbant Assay were received from a total of 6791 serum samples. The positive sera were subjected
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Alonso, Julia, and Hugo Fort. "Error catastrophe for viruses infecting cells: analysis of the phase transition in terms of error classes." Philosophical Transactions of the Royal Society A: Mathematical, Physical and Engineering Sciences 368, no. 1933 (2010): 5569–82. http://dx.doi.org/10.1098/rsta.2010.0274.

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RNA viruses offer a very exciting arena in which to study evolution in ‘real time’ owing to both their high replication rate—many generations per day are possible—and their high mutation rate, leading to a large phenotypic variety. They can be regarded as a swarm of genetically related mutants around a dominant or master genetic sequence. This system is called a ‘viral quasi-species’. Thus, a common framework to describe RNA viral dynamics is by means of the quasi-species equation (QSE). The QSE is in fact a system of a very large number of nonlinear coupled equations. Here, we consider a simp
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Lee, Guinevere Q. "Chemistry and Bioinformatics Considerations in Using Next-Generation Sequencing Technologies to Inferring HIV Proviral DNA Genome-Intactness." Viruses 13, no. 9 (2021): 1874. http://dx.doi.org/10.3390/v13091874.

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HIV persists via integration of the viral DNA into the human genome. The HIV DNA pool within an infected individual is a complex population that comprises both intact and defective viral genomes, each with a distinct integration site, in addition to a unique repertoire of viral quasi-species. Obtaining an accurate profile of the viral DNA pool is critical to understanding viral persistence and resolving interhost differences. Recent advances in next-generation deep sequencing (NGS) technologies have enabled the development of two sequencing assays to capture viral near-full- genome sequences a
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9

Wu, Shuang, Fumio Imazeki, Fuat Kurbanov, et al. "Evolution of hepatitis B genotype C viral quasi-species during hepatitis B e antigen seroconversion." Journal of Hepatology 54, no. 1 (2011): 19–25. http://dx.doi.org/10.1016/j.jhep.2010.06.018.

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10

Mandary, Madiiha Bibi, Malihe Masomian, Seng-Kai Ong, and Chit Laa Poh. "Characterization of Plaque Variants and the Involvement of Quasi-Species in a Population of EV-A71." Viruses 12, no. 6 (2020): 651. http://dx.doi.org/10.3390/v12060651.

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Viral plaque morphologies in human cell lines are markers for growth capability and they have been used to assess the viral fitness and selection of attenuated mutants for live-attenuated vaccine development. In this study, we investigate whether the naturally occurring plaque size variation reflects the virulence of the variants of EV-A71. Variants of two different plaque sizes (big and small) from EV-A71 sub-genotype B4 strain 41 were characterized. The plaque variants displayed different in vitro growth kinetics compared to the parental wild type. The plaque variants showed specific mutatio
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Dissertations / Theses on the topic "Viral quasi-species"

1

"High-throughput discovery and detection of viral mutations in hepatitis B virus quasi-species for patients undergoing antiviral therapy." Thesis, 2009. http://library.cuhk.edu.hk/record=b6074765.

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HBV DNA replicates through a genomic RNA intermediate. The HBV reverse transcriptase lacks proof-reading activity, resulting in a much higher mutation rate for the HBV genome compared with other DNA viruses. HBV DNA thus is often present in quasi-species in an individual. One or more species may be favorably selected by factors like host immune clearance and use of antiviral drugs.<br>Hepatitis B virus (HBV) infected millions of people worldwide. Chronic HBV infection is the leading cause of liver cirrhosis and hepatocellular carcinoma (HCC).<br>In summary, this study developed and validated t
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