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Relevant bibliographies by topics / Viris diseases

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Academic literature on the topic 'Viris diseases'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Viris diseases"

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Richard Martis, Pascaline Vilash. "Zika Virus Disease." Community and Public Health Nursing 1, no. 2 (2016): 159–61. http://dx.doi.org/10.21088/cphn.2455.8621.1216.16.

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Ahmad, Nadeem, Rubeena Bano, and Priyanka Singh. "Ebola Virus Disease." Indian Journal of Medical & Health Sciences 3, no. 2 (2016): 131–34. http://dx.doi.org/10.21088/ijmhs.2347.9981.3216.10.

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Cárdenas-Alonso, M. R. "Las enfermedades causadas por virus en ornamentales en México y alternativas de solución." Revista Chapingo Serie Horticultura I, no. 01 (January 1994): 124–30. http://dx.doi.org/10.5154/r.rchsh.1993.04.035.

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Ettehadi, Amirhossein. "Molecular detection of Marek's disease virus antigen A in fowls infected with Marek’s disease." Journal of Coastal Life Medicine 4, no. 1 (January 2016): 24–29. http://dx.doi.org/10.12980/jclm.4.2016j5-105.

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Marsh, Glenn A. "Bat-associated diseases." Microbiology Australia 38, no. 1 (2017): 3. http://dx.doi.org/10.1071/ma17002.

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Emerging infectious diseases pose a significant threat to human and animal health. Increasingly, emerging and re-emerging infectious diseases are of zoonotic origin and are derived from wildlife. Bats have been identified as an important reservoir of zoonotic viruses belonging to a range of different virus families including SARSCoronavirus, Rabies virus, Hendra virus, Nipah virus, Marburg virus and Ebola virus.

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PISI, A. "Strawberry virus diseases." EPPO Bulletin 16, no. 2 (June 1986): 353–58. http://dx.doi.org/10.1111/j.1365-2338.1986.tb00288.x.

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Elisha, A., and B. Adegboro. "Ebola Virus Diseases." African Journal of Clinical and Experimental Microbiology 15, no. 3 (September 9, 2014): 117. http://dx.doi.org/10.4314/ajcem.v15i3.1.

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B Wasnik, Seema, Mohandeep Kaur, Mala Chabbra, Tarun Kumar, Vinodbala Dhir, Rajeshth Mittal, and Nandini Duggal. "Ebola Virus Disease in the year 2014-2015: Retrospective Study of Suspected Cases of Ebola Virus Disease at Intensive Care Unit of Tertiary Care Center." Indian Journal of Anesthesia and Analgesia 6, no. 1 (2019): 103–9. http://dx.doi.org/10.21088/ijaa.2349.8471.6119.15.

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Ahmed, A. I., S. M. Odisho, and R. N. Al-Gafari. "Comparison of the immune response between local manufactured and commercial inactivated Newcastle Disease Virus vaccine in a challenge trail with field isolated Newcastle Disease Virus." Iraqi Journal of Veterinary Medicine 42, no. 1 (June 28, 2018): 46–51. http://dx.doi.org/10.30539/009.

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Owens, Gregory P., R. Anthony Williamson, Mark P. Burgoon, Omar Ghausi, Dennis R. Burton, and Donald H. Gilden. "Cloning the Antibody Response in Humans with Chronic Inflammatory Disease: Immunopanning of Subacute Sclerosing Panencephalitis (SSPE) Brain Sections with Antibody Phage Libraries Prepared from SSPE Brain Enriches for Antibody Recognizing Measles Virus Antigens In Situ." Journal of Virology 74, no. 3 (February 1, 2000): 1533–37. http://dx.doi.org/10.1128/jvi.74.3.1533-1537.2000.

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ABSTRACT In central nervous system (CNS) infectious and inflammatory diseases of known cause, oligoclonal bands represent antibody directed against the causative agent. To determine whether disease-relevant antibodies can be cloned from diseased brain, we prepared an antibody phage display library from the brain of a human with subacute sclerosing panencephalitis (SSPE), a chronic encephalitis caused by measles virus, and selected the library against SSPE brain sections. Antibodies that were retrieved reacted strongly with measles virus cell extracts by enzyme-linked immunosorbent assay and were specific for the measles virus nucleocapsid protein. These antibodies immunostained cells in different SSPE brains but not in control brain. Our data provide the first demonstration that diseased brain can be used to select in situ for antibodies directed against the causative agent of disease and point to the potential usefulness of this approach in identifying relevant antibodies in chronic CNS or systemic inflammatory diseases of unknown cause.

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Dissertations / Theses on the topic "Viris diseases"

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Yassin, Khaled. "Unravelling the mystery of liver diseases in Egypt : the burden of disease /." Lage : Jacobs, 2001. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=009222709&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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com, khalesi20022002@yahoo, and Bahman Khalesi. "Studies of beak and feather disease virus infection." Murdoch University, 2007. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20071119.90905.

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The circovirus Beak and feather disease virus (BFDV) causes psittacine beak and feather disease (PBFD) that is characterised by a chronic disease process associated with feather abnormalities, beak deformities and eventual death in various species of birds in the order Psittaciformes. This disease is seen in captive and wild psittacine species in Australia and several other countries and is a significant threat to the survival of some endangered psittacine species. This thesis reports on genetic studies that have furthered the understanding of the diversity of BFDV present within Australia. These studies have optimised methods of detecting BFDV. They have also resulted in the production of an immunogenic and antigenic recombinant BFDV Capsid protein that could lead to alternate methods of producing viral antigen for serological tests and the development of a BFDV vaccine. To assess the optimal method of the detection of BFDV infection, feather and blood samples were submitted by referring veterinarians throughout Australia from psittacine birds tentatively diagnosed with PBFD or with a history of being in contact with PBFD-affected birds. These samples were examined by 3 procedures commonly used to detect BFDV infection: a polymerase chain reaction (PCR) assay and haemagglutination (HA) for the detection of virus, and haemagglutination inhibition (HI) tests for the detection of virus antibody in response to infection. Of the samples examined from 623 psittacine birds, the prevalence of BFDV DNA in feather samples detected by PCR was 18.85%. There was a strong correlation between PCR and HA testing of feather samples, although possible false-positive and false-negative PCR and HA results were obtained in some samples. Of the 143 birds that were PCR feather-positive only 2 had detectable HI antibody and these birds were also HA feather-negative, which suggests that they were developing immunity to recent infection. All birds with HI antibody were feather HA negative. Despite the rare occurrence of PBFD in cockatiels (Nymphicus hollandicus), 2 of the 13 samples collected from this species were PCR and HA positive indicating that this species can be infected with BFDV. Three studies were undertaken to further our understanding of the genetics of BFDV in Australian avifauna: sequence analysis of the BFDV detected in a grey cockatiel (Nymphicus hollandicus), a species normally considered resistant to infection with BFDV; analysis of the genome of BFDV present in lorikeets (Trichoglossus sp.) in Australia; and analysis of the genome of BFDV detected in endangered swift parrots (Lathamus discolor). Sequence analysis of the entire genome of the cockatiel BFDV isolate revealed that it clustered phylogenetically with 2 other viruses, one from a sulphur crested cockatoo (SCC1-AUS) and one from a Major Mitchell cockatoo (MMC-AUS), which suggests that this isolate from the grey cockatiel was not a cockatiel-specific biotype. Phylogenetic analysis of the ORF V1 of BFDV detected in 7 lorikeets demonstrated these 7 isolates clustered phylogenetically with other BFDV isolates obtained from Loriidae species elsewhere in the world and confirmed the presence of a loriid-specific genotype. Phylogenetic analysis of the sequence data generated from ORF V1 of virus detected in 2 endangered swift parrots provided evidence they were also infected with BFDV genotypes derived from other species of birds, one isolate clustering with viruses from a Loriidae genotype and the other with isolates derived from species of Cacatuidae and Psittacidae. As part of this research, a baculovirus expression system was successfully developed for the production of recombinant BFDV Capsid protein. Inoculation of this protein into chickens resulted in the development of HI antibody, which demonstrated its immunogenicity. When used as an antigen in HI tests it detected antibody in virus-infected birds, which demonstrated its antigenicity. This protein offers potential application as an antigen for the development of serological tests and as an immunogen for incorporation into vaccines for control of PBFD.

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Nelson, M. R., A. Nadeem, W. Ahmed, and T. V. Orum. "Cotton Virus Diseases." College of Agriculture, University of Arizona (Tucson, AZ), 1998. http://hdl.handle.net/10150/210398.

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Virus diseases of cotton have historically been of only sporadic importance to global cotton production. Recent devastating epidemics in Pakistan and other areas have brought new awareness to the potential for disaster of a pathogen once considered to be of a minor importance. Under changing conditions this pathogen (cotton leaf curl virus) has emerged as a serious problem in Pakistan and India. Cotton leaf curl virus does not occur in the United States or the rest of the western hemisphere but recent experience worldwide is a reminder that pathogens, such as this geminivirus, can be moved easily from one part of the world to another and therefor we need to be aware of the potential impact of such pathogens on local crops.

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Tsang, Chiu-shun Peter. "Oral biology of human immunodeficiency virus-infected individuals in Hong Kong /." [Hong Kong : Faculty of Dentistry, University of Hong Kong], 1997. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19900661.

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Engel, Marie. "Eradication of Aujeszky's disease (pseudorabies) virus from pig herds : alternatives to depopulation /." Uppsala : Swedish Univ. of Agricultural Sciences (Sveriges lantbruksuniv.), 1999. http://epsilon.slu.se/avh/1999/91-576-5437-9.pdf.

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Zaid, Ali, and n/a. "IMMUNE EVASION AND DISEASE MECHANISMS IN ROSS RIVER VIRUS INFECTION." University of Canberra. Biomedical Sciences, 2008. http://erl.canberra.edu.au./public/adt-AUC20091216.122508.

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Ross River virus (RRV) is an Alphavirus distributed throughout Australia. It is transmitted by mosquitoes and is known to cause moderate to severe disease symptoms in humans. Along with other alphaviruses such as Sindbis virus and Chikungunya virus, RRV is known to cause arthritic symptoms, characterised by muscle and joint inflammation. Several investigations have established the role of macrophage cells and pro-inflammatory host factors in the development of RRV-induced disease. In this study, we attempted to determine differences between RRV passaged in mammalian and mosquito cells. There is strong evidence that arthropod-borne viruses are able to display enhanced infectivity when passaged into arthropod cell line. We showed that mosquito cell-derived RRV (mos-RRV) was able to replicate to higher titres than mammalian cell-derived RRV. We also showed that mos-RRV failed to induce Type I IFN-associated antiviral responses. The second aim of this study was to investigate the role of TNF-ᬠa pro-inflammatory cytokine implicated in arthritic diseases, in the development of RRV disease. We treated RRV-infected C57BL/6J mice with a commercially available TNF-ᠩnhibitor drug and monitored disease signs. We found that the TNF-ᠩnhibitor does not ameliorate RRV disease (RRVD) symptoms, and that it does not prevent muscle and joint inflammation. We analysed histological sections of muscle and joint tissue of Enbrel-treated and untreated, RRV-infected cells. We also determined and compared host cytokine expression profiles. Finally, we sought to determine the requirement for natural killer (NK) cells in RRV disease. NK cells have been detected in the synovium of RRV-infected patients since early studies, but their role in disease pathogenesis remains unclear. Using a NK-dysfunctional mouse (C57BL/6J-Lystbg), we showed that mice lacking a functional NK system are more susceptible to RRV disease than wildtype, C57BL/6J mice. We monitored disease symptoms following RRV infection and assessed muscle and joint inflammation in Lystbg and C57BL/6J mice. This thesis examines mechanisms of viral infection and immune evasion employed by RRV, as well as into the role of host cells and cytokines in RRVD pathogenesis disease mechanisms. We showed that a functional NK cell system is required for the regulation of RRV-induced muscle and joint inflammation. Our characterisation of the use of a commercial TNF-ᠩnhibitor in RRV-induced disease in mice may provide information on the role of TNF-ᠩn viral arthritis, and may help towards developing safe and effective treatment.

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Schmidt, Madelyn R. "Virus-Lymphocyte Interactions: Virus Expression Is Differentially Modulated by B Cell Activation Signals: A Dissertation." eScholarship@UMMS, 1991. https://escholarship.umassmed.edu/gsbs_diss/51.

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It is shown here that the ability of B lymphocytes to act as supportive host cells for virus infections requires they be activated from the resting Gostage of the cell cycle. I have used a series of activation regimens, which allow B cells to progress to different stages in their activation/differentiation pathway toward antibody secretion, in order to evaluate the extent of activation required to support vesicular stomatitis or Newcastle disease virus infections. At least three distinct phases during B cell activation which affected VSV infection were defined. Freshly isolated resting murine splenic B cells in the Go phase of the cell cycle do not support VSV, assessed by protein synthesis, infectious center formation, and PFU production. Small B cells cultured for 48 hours without stimulation still do not support VSV. B cells stimulated with the lymphokines found in Con A activated supernatants from splenic T cells or cloned T cell lines transited into the G1 phase of the cell cycle but remain refractory to VSV. These VSV non-supportive B cell populations do take up virus particles and transcribe viral mRNAs which can be translated in vitro, suggesting a translational block to VSV. B cells stimulated into the S phase of the cell cycle with anti-immunoglobulin synthesize VSV proteins and increased numbers of infectious centers, but only low level PFU synthesis (center) is observed. Co-stimulation with anti-Ig and lymphokines, which supports differentiation to antibody secretion, enhanced PFU synthesis without further increasing the number of infected B cells. LPS, which activates B cells directly to antibody secretion by a pathway different from anti-Ig, induced infectious centers, and PFUs at levels comparable to those seen when stably transformed permissive cell lines are infected. Co-stimulation of LPS activated B cells with the same lymphokine populations that enhance PFU production when anti-Ig is used as a stimulator suppresses PFU production completely, suggesting that anti-Ig and LPS activated B cells are differentially responsive to lymphokines. NDV infection of murine B cells differed markedly from VSV infection, as all B cell populations examined gave a similar response pattern. NDV viral proteins were synthesized by B cells in each of the activation states previously described, even freshly isolated B cells. Infectious center formation increased up to 5-fold over the levels observed with unstimulated B cells after anti-Ig or LPS activation. However, PFU synthesis was low (center) for all B cell populations. These results suggest that these two similar viruses may be dependent on different host cell factors and that these factors are induced for VSV but not NDV by the B cell activators employed here or that the process of infection of B cell by these two viruses induces different cellular responses.

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Choi, Wai-yee Junet. "Serum neopterin for early assessment of severity of severe acute respiratory syndrome and Dengue virus infection." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B32031579.

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Oladele, Oluwafemi. "Characterization of feline borna disease virus /." Uppsala : Department of Biomedical Sciences and Veterinary Public Health, Swedish University of Agricultural Sciences, 2006. http://epsilon.slu.se/10454915.pdf.

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Tsang, Chiu-shun Peter, and 曾昭舜. "Oral biology of human immunodeficiency virus-infected individuals in Hong Kong." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1997. http://hub.hku.hk/bib/B31237770.

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Books on the topic "Viris diseases"

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Carbone, Kathryn M., ed. Borna Disease Virus and its Role in Neurobehavioral Diseases. Washington, DC, USA: ASM Press, 2002. http://dx.doi.org/10.1128/9781555817909.

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Frank and Bobbie Fenner Conference on Medical Research. (1st 1988 John Curtin School of Medical Research). Immunology of virus diseases. [Canberra]: John Curtin School of Medical Research, 1989.

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da Silva, Suzane Ramos, Fan Cheng, and Shou-Jiang Gao. Zika Virus and Diseases. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2018. http://dx.doi.org/10.1002/9781119408673.

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Food, Ontario Ministry of Agriculture and. Virus diseases of soybeans. S.l: s.n, 1988.

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Lapierre, Hervé. Virus and virus diseases of Poaceae (Gramineae). Edited by Signoret Pierre A and Institut national de la recherche agronomique (France). Paris: Institut National de la Recherche Agronomique (France), 2004.

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Bechter, Karl. Borna Disease Virus. Heidelberg: Steinkopff, 1998. http://dx.doi.org/10.1007/978-3-642-95999-8.

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Lado, Marta, ed. Ebola Virus Disease. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-94854-6.

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Dybing, Jody Kay. Sequence analysis of infectious bursal disease virus of stereotype 2 and expression of the VP5 cDNA. Charlottetown: University of Prince Edward Island, 1992.

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Dybing, Jody Kay. Sequence analysis of infectious bursal disease virus of stereotype 2 and expression of the VP5 cDNA. Ottawa: National Library of Canada, 1992.

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Sastry, K. Subramanya. Seed-borne plant virus diseases. India: Springer India, 2013. http://dx.doi.org/10.1007/978-81-322-0813-6.

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Book chapters on the topic "Viris diseases"

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Chen, Ren-Gui, Ping Li, Chen Wang, Ming-Yu Xia, Xin-Feng Wu, Cheng Tan, and Ru-Zhi Zhang. "Virus Diseases." In Atlas of Skin Disorders, 3–10. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8037-1_1.

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Neve, R. A. "Virus diseases." In Hops, 175–93. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3106-3_8.

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Taylor, N. L., and K. H. Quesenberry. "Virus Diseases." In Red Clover Science, 91–96. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-015-8692-4_7.

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Libbey, Jane E., and Robert S. Fujinami. "Virus-Induced Immunosuppression." In Polymicrobial Diseases, 375–87. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555817947.ch19.

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Díaz-Menéndez, Marta, and Clara Crespillo-Andújar. "The Disease." In Zika Virus Infection, 43–53. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-59406-4_6.

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Davenport, Andrew, Todd W. Costantini, Raul Coimbra, Marc M. Sedwitz, A. Brent Eastman, David V. Feliciano, David V. Feliciano, et al. "Virus Disease." In Encyclopedia of Intensive Care Medicine, 2458. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_2424.

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Klein, M. "Phytoplasma Diseases." In Virus and Virus-like Diseases of Potatoes and Production of Seed-Potatoes, 145–58. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-007-0842-6_17.

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Samson, A. C. R. "Virus Structure." In Newcastle Disease, 23–44. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-1759-3_3.

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Kishi, Masahiko, Keizo Tomonaga, Patrick Lai, and Juan Carlos de la Torre. "Borna Disease Virus Molecular Virology." In Borna Disease Virus and its Role in Neurobehavioral Diseases, 23–43. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555817909.ch2.

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Planz, Oliver, Karl A. Bechter, and Martin Schwemmle. "Human Borna Disease Virus Infection." In Borna Disease Virus and its Role in Neurobehavioral Diseases, 179–225. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555817909.ch6.

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Conference papers on the topic "Viris diseases"

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Tareq HAMMOODI, Zeyad. "CORONA EPIDEMIC (COVD 19) BETWEEN SHARIA AND MEDICINE." In International Research Congress of Contemporary Studies in Social Sciences (Rimar Congress 2). Rimar Academy, 2021. http://dx.doi.org/10.47832/rimarcongress2-7.

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The Corona epidemic is a wide group of viruses that include viruses that can cause a group of illnesses in humans, ranging from the common cold to severe acute respiratory syndrome, as there is no definitive and specific treatment for the epidemic. The medicines used are helpful and supportive, and they mostly aim to reduce the patient’s temperature with the use of pulmonary resuscitation devices, as the body’s resistance depends on autoimmunity, as it is the main factor in preventing this epidemic, and here we must know the role of medical and forensic scholars in preventing and treating With what appears from this epidemic and other epidemics, we do not know when and how they will appear to the world. The emergence of this disease is an extension of several diseases before it and the so-called (contemporary diseases), which are contagious communicable diseases, including bird flonza disease, swine flonza, sass and AIDS, mad cow disease, Ebola, human papillomavirus, herpes simplex virus, yellow fever, and many others These diseases are epidemic.

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Gür, A., M. Karakoç, MF Geyik, K. Nas, R. Çevik, AJ Saraç, S. Em, and F. Erdogan. "SAT0135 Association between hepatitis c virus antibody, hepatitis b virus antigen and fibromiyalgia." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.594.

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Luz, Jeniffer, Scenio De Araujo, Caio Abreu, Juvenal Silva Neto, and Carlos Gulo. "Formation of a cooperation network in Mato Grosso on Machine Learning and Image Analysis: Diagnosis of COVID-19 in X-ray images." In Computer on the Beach. São José: Universidade do Vale do Itajaí, 2021. http://dx.doi.org/10.14210/cotb.v12.p523-524.

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Since the beginning of the COVID-19 outbreak, the scientific communityhas been making efforts in several areas, either by seekingvaccines or improving the early diagnosis of the disease to contributeto the fight against the SARS-CoV-2 virus. The use of X-rayimaging exams becomes an ally in early diagnosis and has been thesubject of research by the medical image processing and analysiscommunity. Although the diagnosis of diseases by image is a consolidatedresearch theme, the proposed approach aims to: a) applystate-of-the-art machine learning techniques in X-ray images forthe COVID-19 diagnosis; b) identify COVID-19 features in imagingexamination; c) to develop an Artificial Intelligence model toreduce the disease diagnosis time; in addition to demonstrating thepotential of the Artificial Intelligence area as an incentive for theformation of critical mass and encouraging research in machinelearning and processing and analysis of medical images in the Stateof Mato Grosso, in Brazil. Initial results were obtained from experimentscarried out with the SVM (Support Vector Machine) classifier,induced on a publicly available image dataset from Kaggle repository.Six attributes suggested by Haralick, calculated on the graylevel co-occurrence matrix, were used to represent the images. Theprediction model was able to achieve 82.5% accuracy in recognizingthe disease. The next stage of the studies includes the study of deeplearning models.

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Davi, Caio, André Pastor, Thiego Oliveira, Fernando B. Lima Neto, Ulisses Braga-Neto, Abigail W. Bigham, Michael Bamshad, Ernesto T. A. Marques, and Bartolomeu Acioli-Santos. "Computational Intelligence applied to Human Genome Data for the Dengue Severity Prognosis." In XI Simpósio Brasileiro de Bioinformática. Sociedade Brasileira de Computação - SBC, 2019. http://dx.doi.org/10.5753/bsb_estendido.2018.8800.

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Dengue has become one of the most important worldwide arthropodborne diseases around the world. Here, one hundred and two Brazilian dengue virus (DENV) III patients and controls were genotyped for 322 innate immunity gene loci. All biological data (including age, sex and genome background) were analyzed using Machine Learning techniques to discriminate tendency to severe dengue phenotype development. Our current approach produces median values for accuracy greater than 86%, with sensitivity and specificity over 98% and 51%, respectively. Genome data information from 13 key immune polymorphic SNPs was used under different dominant or recessive models. Our approach is a valuable tool for early diagnosis of the severe form of dengue infection and can be used to identify individuals at high risk of developing this form of the disease even in uninfected individuals. The model also identifies various genes involved dengue severity.

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Mukhina, Aleksandra Alekseevna. "Developing a Differential Zone for Virus Disease Prevention using Phytoterapy." In International Scientific and Practical Conference, chair Olga Vladimirovna Nesterova and Nadezhda Viktorovna Nesterova. TSNS Interaktiv Plus, 2020. http://dx.doi.org/10.21661/r-541016.

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Pulmonology is a branch of medicine that treats diseases of the respiratory system. An important problem in the modern world is chronic obstructive pulmonary diseases: bronchial asthma, emphysema, chronic bronchitis, including obstructive and others. This group of diseases with a transient or permanent violation of airway patency with the development and further progression of respiratory failure.

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"DATA MINING ON DENGUE VIRUS DISEASE." In 13th International Conference on Enterprise Information Systems. SciTePress - Science and and Technology Publications, 2011. http://dx.doi.org/10.5220/0003422000320041.

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Chen, Ling. "Engineered bioluminescent influenza viruses shed light on defense against influenza virus infection (Conference Presentation)." In Photonic Diagnosis and Treatment of Infections and Inflammatory Diseases, edited by Tianhong Dai. SPIE, 2018. http://dx.doi.org/10.1117/12.2291867.

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Ertenli, Ý., S. Kiraz, S. Apras, MA Oztürk, V. Çobankara, Z. Balkancý, and M. Çalgüner. "THU0228 Prevalence of hepatitis e virus in behÇet’s disease." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.760.

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Michtchenko, A., A. V. Budagovsky, and O. N. Budagovskaya. "Optical Diagnostics Fungal and Virus Diseases of Plants." In 2015 12th International Conference on Electrical Engineering, Computing Science and Automatic Control (CCE). IEEE, 2015. http://dx.doi.org/10.1109/iceee.2015.7357968.

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Welch, David, Henry M. Spotnitz, David J. Brenner, Gerhard Randers-Pehrson, Manuela Buonanno, and Igor Shuryak. "Far-UVC light applications: sterilization of MRSA on a surface and inactivation of aerosolized influenza virus." In Photonic Diagnosis and Treatment of Infections and Inflammatory Diseases, edited by Tianhong Dai. SPIE, 2018. http://dx.doi.org/10.1117/12.2309424.

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Reports on the topic "Viris diseases"

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Kamp, Jan, Pieter Blok, Gerrit Polder, Jan van der Wolf, and Henk Jalink. Smart disease detection seed potatoes 2015-2018 : Detection of virus and bacterial diseases using vision and sensor technology. Wageningen: Stichting Wageningen Research, Wageningen Plant Research, Business Unit Field Corps, 2020. http://dx.doi.org/10.18174/494707.

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Sjogren, Maria H., and Kent Holtzmuller. Hepatitis C Virus Infection: Mechanism of Disease Progression. Fort Belvoir, VA: Defense Technical Information Center, October 2001. http://dx.doi.org/10.21236/ada406083.

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Sjogren, Maria H. Hepatitis C. Virus Infection: Mechanism of Disease Progression. Fort Belvoir, VA: Defense Technical Information Center, October 2004. http://dx.doi.org/10.21236/ada433067.

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Dr. Shawn Carbonell, Dr Shawn Carbonell. Developing a new treatment for Ebola Virus Disease. Experiment, April 2015. http://dx.doi.org/10.18258/4932.

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Sjogren, Maria H., and Brooke Huntley. Hepatitis C. Virus Infection: Mechanisms of Disease Progression. Fort Belvoir, VA: Defense Technical Information Center, October 2007. http://dx.doi.org/10.21236/ada477987.

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Gardner, Murray B. Genetic Immunization for Lentiviral Immunodeficiency Virus Infection and Disease. Fort Belvoir, VA: Defense Technical Information Center, November 1997. http://dx.doi.org/10.21236/ada338248.

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Gardner, Murray B. Genetic Immunization for Lentiviral Immunodeficiency Virus Infection and Disease. Fort Belvoir, VA: Defense Technical Information Center, October 1998. http://dx.doi.org/10.21236/ada361721.

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Zhang, Jibin, Michael G. Kaiser, Melissa S. Herrmann, Rodrigo A. Gallardo, David A. Bunn, Terra R. Kelly, Jack C. M. Dekkers, Huaijun Zhou, and Susan J. Lamont. Different Genetic Resistance Resulted in Distinct Response to Newcastle Disease Virus. Ames (Iowa): Iowa State University, January 2017. http://dx.doi.org/10.31274/ans_air-180814-324.

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Lai, Ching J. Development of Safe, Effective Vaccines for Dengue Virus Disease by Recombinant Baculovirus. Fort Belvoir, VA: Defense Technical Information Center, April 1993. http://dx.doi.org/10.21236/ada266829.

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Herrmann, Melissa S., Rodrigo Gallardo, David A. Bunn, Huaijun Zhou, and Susan J. Lamont. Do Two Distinct Chicken Lines Differ in Their Response to Newcastle Disease Virus? Ames (Iowa): Iowa State University, January 2016. http://dx.doi.org/10.31274/ans_air-180814-228.

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