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Books on the topic 'Virus de Hepatitis VHC'

Author: Grafiati

Published: 11 September 2021

Last updated: 3 February 2022

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1

Ruggeri, Franco Maria, Ilaria Di Bartolo, Fabio Ostanello, and Marcello Trevisani. Hepatitis E Virus. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7522-4.

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2

Handa, Hiroshi, and Yuki Yamaguchi. Hepatitis Delta Virus. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/0-387-35103-5.

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3

Guo, Haitao, and Andrea Cuconati, eds. Hepatitis B Virus. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6700-1.

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4

Wang, Youchun, ed. Hepatitis E Virus. Dordrecht: Springer Netherlands, 2016. http://dx.doi.org/10.1007/978-94-024-0942-0.

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5

M, Feinstone Stephen, ed. Hepatitis A. Boca Raton, Fla: CRC Press, 1988.

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6

Law, Mansun, ed. Hepatitis C Virus Protocols. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-8976-8.

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7

Tang, Hong, ed. Hepatitis B Virus Infection. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-13-9151-4.

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8

Jirillo, Emilio, ed. Hepatitis C Virus Disease. New York, NY: Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-71376-2.

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9

Miyamura, Tatsuo, Stanley M. Lemon, Christopher M. Walker, and Takaji Wakita, eds. Hepatitis C Virus I. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-56098-2.

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10

Miyamura, Tatsuo, Stanley M. Lemon, Christopher M. Walker, and Takaji Wakita, eds. Hepatitis C Virus II. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-56101-9.

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11

Chayama, Kazuaki, ed. Hepatitis C Virus Treatment. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-2416-0.

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12

Shiffman, Mitchell L., ed. Chronic Hepatitis C Virus. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-1192-5.

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13

1968-, Zhang Weiying, and Ye Lihong, eds. Hepatitis B virus research focus. New York: Nova Science Publishers, 2008.

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14

Sánchez, Glòria. Hepatitis A Virus in Food. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7104-2.

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15

Hirsh, Jennifer Lee. Hepatitis B virus and Hepatitis A virus in Kingston, Jamaica: Maternal-child immunity. [New Haven: s.n.], 1989.

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16

Liaw, Yun-Fan, and Fabien Zoulim, eds. Hepatitis B Virus in Human Diseases. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-22330-8.

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17

Kao, Jia-Horng, and Ding-Shinn Chen, eds. Hepatitis B Virus and Liver Disease. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-4843-2.

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18

Feitelson, Mark. Molecular Components of Hepatitis B Virus. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2573-4.

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19

Berenguer, Marina, ed. Hepatitis C Virus and Liver Transplantation. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-8438-7.

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20

Molecular components of hepatitis B virus. Boston: Nijhoff, 1985.

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21

Kao, Jia-Horng, ed. Hepatitis B Virus and Liver Disease. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-3615-8.

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22

International Symposium on Enterically-Transmitted Hepatitis Viruses (1995 Paris, France). Enterically-transmitted hepatitus viruses: Proceedings of the International Symposium on Enterically-Transmitted Hepatitus Viruses, Paris, Ecole du Val-de-Grace, October 16-17, 1995. Edited by Buisson Yves, Coursaget P, and Kane Mark. Tours, France: La Simarre, 1996.

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23

Alsharif, Fadwa Muzahim. Serum pooling in hepatitis C virus serodiagnosis. Manchester: University of Manchester, 1995.

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24

International Symposium on HCV (3rd 1991 Strasbourg, France). Hepatitis C virus: Scientific and clinical status. Edited by Deinhardt Friedrich, Bradley Daniel W, Houghton Michael, and Advanced Therapeutics Communications (Firm). Secaucus, NJ, USA (400 Plaza Dr., Secaucus 07904): Advanced Therapeutics Communications, 1992.

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25

Tilston, Peter John. Diagnostic methods for Hepatitis C virus infection. Manchester: University of Manchester, 1994.

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26

Sulaiman, H. Ali. Virus hepatitis A sampai E di Indonesia. [Jakarta]: Yayasan Penerbitan Ikatan Dokter Indonesia, 1995.

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27

Hepatitis B: The hunt for a killer virus. Princeton, N.J: Princeton University Press, 2002.

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28

Maine Hepatitis C Infection Needs Assessment Steering Committee. At the crossroads: Hepatitis C infection in Maine : a comprehensive statewide needs assessment. [Augusta, Me.?]: Published by the Maine Center for Public Health for the Maine Hepatitis C Infection Needs Assessment Steering Committee, 2001.

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29

Mathet, Verónica L. Genetic diversity & variability of hepatitis B virus (HBV). New York: Nova Science Publishers, 2008.

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30

Mathet, Verónica L. Genetic diversity & variability of hepatitis B virus (HBV). New York: Nova Science Publishers, 2009.

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31

C. L. van der Poel. Hepatitis C virus: Studies on transmission and epidemology. Amsterdam: Babeliowsky, 1991.

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32

Protocolo para estimar la mortalidad por cirrosis y por carcinoma hepatocelular atribuible a las hepatitis virales B y C. Pan American Health Organization, 2021. http://dx.doi.org/10.37774/9789275323762.

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Una de las metas a alcanzar para el 2030 de la Estrategia mundial del sector de la salud contra las hepatitis víricas 2016-202: hacia el fin de las hepatitis víricas es reducir la mortalidad por el virus de la hepatitis B (VHB) y el virus de la hepatitis C (VHC). Para medir este indicador y darle seguimiento, los países requieren poner en funcionamiento un proceso sistemático para generar estimaciones nacionales de mortalidad por hepatitis virales. Esta publicación está dirigida a las instituciones y ministerios encargados de monitorizar los avances en cada país. El objetivo principal de este protocolo es presentar métodos sencillos para estimar la proporción de pacientes con cirrosis y con carcinoma hepatocelular que tienen infección por el VHB y el VHC, para luego calcular la mortalidad nacional por estas secuelas atribuibles a hepatitis virales, de preferencia dentro de un sistema de vigilancia. Además, se proporciona un marco general sobre cómo debe funcionar el sistema de vigilancia, cómo recopilar los datos y consideraciones éticas para tener en cuenta. El sistema de vigilancia se diseña sobre la base de centros centinela donde se recopilará información de pacientes con cirrosis y con carcinoma hepatocelular. Estos datos servirán para estimar la fracción de cirrosis y de carcinoma hepatocelular atribuible a la infección por los virus de las hepatitis B y C. Por otro lado, se recopilarán datos sobre el número de muertes a nivel nacional por cirrosis y por carcinoma hepatocelular. Con esta información, se calculará la mortalidad por cirrosis y por carcinoma hepatocelular atribuible a la infección por el VHB y el VHC.

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33

Handa, Hiroshi. Hepatitis Delta Virus. Landes Bioscience, 2005.

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34

Casey, John L. Hepatitis Delta Virus. Springer, 2010.

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35

Wang, Youchun. Hepatitis E Virus. Springer, 2018.

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36

Handa, Hiroshi, and Yuki Yamaguchi. Hepatitis Delta Virus. Springer, 2011.

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37

W, Reesink H., ed. Hepatitis C virus. Basel: Karger, 1994.

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38

Meng, X. J. Hepatitis E virus. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0048.

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Hepatitis E virus (HEV) is a small, non-enveloped, single-strand, positive-sense RNA virus of approximately 7.2 kb in size. HEV is classified in the family Hepeviridae consisting of four recognized major genotypes that infect humans and other animals. Genotypes 1 and 2 HEV are restricted to humans and often associated with large outbreaks and epidemics in developing countries with poor sanitation conditions, whereas genotypes 3 and 4 HEV infect humans, pigs and other animal species and are responsible for sporadic cases of hepatitis E in both developing and industrialized countries. The avian HEV associated with Hepatitis-Splenomegaly syndrome in chickens is genetically and antigenically related to mammalian HEV, and likely represents a new genus in the family. There exist three open reading frames in HEV genome: ORF1 encodes non-structural proteins, ORF2 encodes the capsid protein, and the ORF3 encodes a small phosphoprotein. ORF2 and ORF3 are translated from a single bicistronic mRNA, and overlap each other but neither overlaps ORF1. Due to the lack of an efficient cell culture system and a practical animal model for HEV, the mechanisms of HEV replication and pathogenesis are poorly understood. The recent identification and characterization of animal strains of HEV from pigs and chickens and the demonstrated ability of cross-species infection by these animal strains raise potential public health concerns for zoonotic HEV transmission. It has been shown that the genotypes 3 and 4 HEV strains from pigs can infect humans, and vice versa. Accumulating evidence indicated that hepatitis E is a zoonotic disease, and swine and perhaps other animal species are reservoirs for HEV. A vaccine against HEV is not yet available.

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39

Reesink, H. W., ed. Hepatitis C Virus. S. Karger AG, 1998. http://dx.doi.org/10.1159/isbn.978-3-318-00249-2.

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40

Casey, John L., ed. Hepatitis Delta Virus. Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/3-540-29802-9.

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41

Kourtis, Athena P., Shruti Chandramouli, Gonzague Jourdain, and Marc Bulterys. Hepatitis B Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0004.

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Hepatitis B virus (HBV) is the most common cause of chronic viral hepatitis and hepatocellular carcinoma in the world. Worldwide, more than 250 million people are chronically infected with HBV, causing nearly 780,000 deaths each year, and mother-to-child transmission (MTCT) accounts for more than one-third of chronic HBV infections. Universal vaccination in neonates is the most effective strategy for eliminating infections worldwide. Maternal antiviral treatment during the antepartum/postpartum period for mothers with high HBV viral loads is effective in preventing HBV MTCT. Full immunization coverage is currently the only way to reach the goal of eradicating HBV infection. Operational research and, in some resource-limited settings, international funding may be essential to bring the vaccine where neonates and infants need it, including remote locations where home births are common. Continued improvements in the coverage and timeliness of HBV vaccination and education of clinicians about its importance are needed.

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42

Honegger, Jonathan R. Hepatitis C Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0005.

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An estimated 185 million individuals have been infected with hepatitis C virus (HCV) worldwide. Although often clinically silent for decades, chronic HCV infection predisposes to late-onset complications, including liver cirrhosis and hepatocellular carcinoma. Mother-to-child transmission (MTCT) of HCV affects approximately 5% of children born to viremic mothers and is the primary route of HCV infection in young children. While some vertically acquired HCV infections are resolved during the first years of life, many persist indefinitely. Chronically infected children tend to be asymptomatic and have mild liver disease, but they face a risk of progression to advanced liver disease in adulthood. Current diagnostic and management strategies leave most infected children undiagnosed and untreated. Widespread use of newly-available direct-acting antiviral therapies has the potential to substantially reduce the global burden of HCV, including vertically acquired HCV, but an effective vaccine likely will be required to achieve this ultimate goal.

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43

Krain, Lisa J., and Kenrad E. Nelson. Hepatitis E Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0006.

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Hepatitis E virus (HEV) poses serious risks to pregnant women and their developing fetuses, including increased risk of pregnancy loss, stillbirth, preterm delivery, and early infant death. Supportive care is currently the standard treatment for pregnant women with HEV infection, but in some cases, ribavirin treatment or early delivery may be indicated. Infants born with acute HEV infection face increased risk of complications and death. Intensive monitoring and support may be required in the neonatal period, particularly for preterm infants. Infants who survive the early neonatal period are likely to recover fully and clear the virus. Immunoassays and molecular methods for diagnosis of HEV have improved markedly over the past decade. New HEV vaccines may provide an opportunity to prevent both maternal illness and mother-to-child transmission (vertical transmission) (MTCT).

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44

Koshy, Rajen, and Wolfgang H. Caselmann. Hepatitis B Virus. PUBLISHED BY IMPERIAL COLLEGE PRESS AND DISTRIBUTED BY WORLD SCIENTIFIC PUBLISHING CO., 1998. http://dx.doi.org/10.1142/p005.

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45

W, Reesink H., ed. Hepatitis C virus. 2nd ed. Basel: Karger, 1998.

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46

Hepatitis A. Taylor & Francis Group, 2017.

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47

Hepatitis C Virus Infection. Blackwell Publishing, 1992.

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48

Chronic Hepatitis B. W.B. Saunders Company, 2010.

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49

Gail, Dinter-Gottlieb, ed. The unique hepatitis delta virus. New York: Springer, 1995.

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50

Gail, Dinter-Gottlieb, ed. The unique hepatitis delta virus. Austin: R.G. Landes Co., 1995.

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