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Books on the topic 'Virus encephalitis'

Author: Grafiati
Published: 24 April 2022

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1

Diamond, Michael S. West Nile Encephalitis Virus Infection. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-79840-0.

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2

Booss, John. Viral encephalitis in humans. Washington, D.C: ASM Press, 2003.

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3

Booss, John. Viral encephalitis in humans. Washington, D.C: ASM Press, 2003.

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4

Das, Bina Pani. Mosquito Vectors of Japanese Encephalitis Virus from Northern India. India: Springer India, 2013. http://dx.doi.org/10.1007/978-81-322-0861-7.

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5

Diamond, Michael S. West Nile encephalitis virus infection: Viral pathogenesis and the host immune response. New York, NY: Springer, 2009.

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6

Jacobs, Susan Catherine. Characterisation and analysis of the NS1 gene of tick-borne encephalitis virus. Oxford: Oxford Brookes University, 1992.

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7

Murphy, Karen. Helpers simplex virus encephalitis: Establishment and evaluation of the polymerase chain reaction to overcome present diagnostic difficulties. [S.l: The Author], 1995.

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8

Current issues in clinical neurovirology: Pathogenesis, diagnosis and treatment. Philadelphia, Pa: Saunders, 2008.

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9

Foley, Paul Bernard. Encephalitis Lethargica: The Mind and Brain Virus. Springer, 2019.

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10

Foley, Paul Bernard. Encephalitis Lethargica: The Mind and Brain Virus. Springer, 2018.

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11

Japanese encephalitis and West Nile viruses. Berlin: Springer, 2002.

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12

Encephalitis: Diagnosis and treatment. New York: Informa Healthcare, 2008.

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13

Viral Encephalitis in Humans. Margaret M. Esiri, 2003.

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14

Nuttall, Patricia A. Tick-borne encephalitides. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0044.

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Tick-borne encephalitides are caused by three different viruses transmitted by ticks and belonging to the Flaviviridae virus family: tick-borne encephalitis virus (Far Eastern, Siberian, and European subtypes), louping ill virus, and Powassan virus (including deer tick virus). These viruses cause encephalitis affecting humans in Eurasia and North America. In nature, they are maintained in transmission cycles involving Ixodes tick species and small or medium-sized wild mammals. The tick-borne flavivirus group is one of the most intensely studied groups of tick-borne pathogens.
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15

Jackson, Michael Keven. Nucleotide sequence of S reading frame and viral-induced trans-activation of caprine arthritis-encephalitis virus. 1990.

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16

Papageorges, Marc. Studies on quantitative joint scintigraphy and radioimmunodetection of caprine arthritis-encephalitis virus-induced arthritis. 1989.

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17

Encephalitis: Diagnosis and Treatment (Neurological Disease and Therapy). Informa Healthcare, 2007.

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18

Finn, Matthew. West Nile Virus. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0053.

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West Nile virus (WNV) is a single-stranded RNA virus of the Flavivirus family that is transmitted via a mosquito vector, typically causing fever and capable of causing meningoencephalitis. Although mortality is low, it can lead to debilitating neuroinvasive disease in some patients. WNV is a leading cause of domestically-acquired arboviral disease and most commonly occurs in late August and early September. Consider WNV in otherwise unexplained cases of meningitis or encephalitis. Initial testing should consist of cerebrospinal fluid (CSF) analysis and West Nile immunoglobulin M enzyme-linked immunosorbent assay in serum and/or CSF. WNV is a nationally notifiable disease. Prevention remains the key to controlling this disease. Reducing the breeding grounds of the Culex mosquito and using insect repellant to prevent bites are two important strategies.
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19

Michaels, Frank H. Studies of the effects of infection by caprine arthritis-encephalitis virus of synovial macrophages: Activation, loss of accessory cell capability and increased expression of virus. 1989.

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20

S, Diamond Michael, ed. West Nile encephalitis virus infection: Viral pathogenesis and the host immune response. New York: Springer, 2008.

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21

Das, Bina Pani. Mosquito Vectors of Japanese Encephalitis Virus from Northern India: Role of BPD hop cage method. Springer, 2012.

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22

Lichtensteiger, Carol A. Antibody neutralization and MHC-restricted lymphocyte-mediated cytotoxicity of variants of caprine arthritis-encephalitis lentivirus. 1992.

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23

West Nile virus: Field hearing before the Committee on Environment and Public Works, United States Senate, One Hundred Sixth Congress, first session, to conduct oversight of the state and federal response to the 1999 outbreak of the West Nile Virus in Connecticut and New York, December 14, 1999, Fairfield CT. Washington, U.S. G.P.O, 2000.

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24

West Nile virus: Field hearing before the Committee on Environment and Public Works, United States Senate, One Hundred Sixth Congress, first session, to conduct oversight of the state and federal response to the 1999 outbreak of the West Nile Virus in Connecticut and New York, December 14, 1999, Fairfield CT. Washington, U.S. G.P.O: United States Government Printing Office, 2000.

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25

Knowles, Donald P. Studies of the pathogenesis of viral-induced arthritis and the transcriptional organization of caprine arthritis-encephalitis virus. 1988.

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26

Petersen, Ruby. Antigenic cross-reactivity between caprine arthritis-encephalitis, visna and progressive pneumonia viruses involves all virion-associated proteins and glycoproteins. 1985.

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27

Hullinger, Gordon A. A gp135 cellular receptor mediates infection of caprine synovial membrane cells by the lentivirus caprine arthritis-encephalitis virus. 1993.

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28

Gould, E. A. Mosquito-borne arboviruses. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0039.

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The arboviruses are all single-stranded RNA viruses, although they belong to four different viral families. Several important human pathogens belong to the mosquito-borne arboviruses including yellow fever, Japanese encephalitis and Rift Valley Fever. They cause a wide range of illnesses from unrecognised infection to severe systemic disease with hemorrhagic complications and encephalitis with a high mortality similar range of illnesses is seen in infected animals.Arboviruses have several unique characteristics, these include; an ability to infect and be transmitted by mosquitos, ticks, midges, sand flies, bugs, fleas, blackflies and horseflies. They infect vertebrate hosts which may amplify virus for invertebrate vectors that feed on infected vertebrates. An ability to replicate in anthropods, with little pathology and in vertebrates often with significant pathology. Many arboviruses are Zoonotic.Control methods depend on the epidemiology of particular viruses, but epidemic vector control through control of insect breeding sites and the use of insecticide spraying have been successfully used in the past. Effective vaccines are available for yellow fever and Japanese encephalitis.
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29

Henggeller, Michelle. Infections in the HIV Patient. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0055.

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The hallmark of the human immunodeficiency virus (HIV) patient with a cluster of differentiation 4 (CD4) T lymphocyte count below 200 is the development of opportunistic infections. Although the use of antiretroviral therapy (ART) has decreased the incidence of these infections, they continue to be a major case of morbidity and mortality in the patient with HIV. These infections can be respiratory in nature and present with cough or shortness of breath: Pneumocystis pneumonia (PCP), tuberculosis (TB), aspergillosis, and coccidioidomycosis. Neurological infections, which can present with change in mental status, include toxoplasmosis encephalitis (TE), meningoencephalitis, John Cunningham (JC) virus, and progressive multifocal leukoencephalopathy (PML). Gastrointestinal infections, such as Cryptosporidium, present with abdominal pain and diarrhea. Viral changes can result from cytomegalovirus retinitis. Fever or nonspecific symptoms can result from disseminated Mycobacterium Avium complex disease, histoplasmosis, bartonellosis, and cytomegalovirus.
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30

Solomon, Tom, and Benedict Michael. Neurological infection. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0229.

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Neurological infections can be broadly subdivided into chronic/subacute and acute. Chronic/subacute infection usually presents with global cognitive decline, with the prototypical disease being progressive multifocal leucoencephalopathy due to infection with the JC virus in immunocompromised patients. Acute neurological infections can be defined microbiologically, by the nature of the pathogen; clinically, by the presenting signs and symptoms and initial CSF findings; or anatomically. The anatomical definitions are those occurring intracranially (‘meningitis’, where infection involves the meninges overlying the brain; ‘encephalitis’, where the brain parenchyma is involved; or ‘cerebral abscesses’) and those affecting the spinal cord (‘myelitis’). However, there is often both clinical and histological overlap between these syndromes; consequently, the terms ‘meningoencephalitis’ and ‘encephalomyelitis’ are often used. Patients with acute intracranial CNS infections provide the greatest challenge to general physicians, because urgent investigation and appropriate treatment can save lives; they therefore form the focus of this chapter.
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31

Swanepoel, R., and J. T. Paweska. Rift Valley fever. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0043.

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Rift Valley fever (RVF) is an acute disease of domestic ruminants in mainland Africa and Madagascar, caused by a mosquito borne virus and characterized by necrotic hepatitis and a haemorrhagic state. Large outbreaks of the disease in sheep, cattle and goats occur at irregular intervals of several years when exceptionally heavy rains favour the breeding of the mosquito vectors, and are distinguished by heavy mortality among newborn animals and abortion in pregnant animals. Humans become infected from contact with tissues of infected animals or from mosquito bite, and usually develop mild to moderately severe febrile illness, but severe complications, which occur in a small proportion of patients, include ocular sequelae, encephalitis and fatal haemorrhagic disease. Despite the occurrence of low case fatality rates, substantial numbers of humans may succumb to the disease during large outbreaks. Modified live and inactivated vaccines are available for use in livestock, and an inactivated vaccine was used on a limited scale in humans with occupational exposure to infection. The literature on the disease has been the subject of several extensive reviews from which the information presented here is drawn, except where indicated otherwise (Henning 1956; Weiss 1957; Easterday 1965; Peters and Meegan 1981; Shimshony and Barzilai 1983; Meegan and Bailey 1989; Swanepoel and Coetzer 2004; Flick and Bouloy 2005). In September 2000, the disease appeared in south-west Saudi Arabia and adjacent Yemen, and the outbreak lasted until early 2001 (Al Hazmi et al. 2003; Madani et al. 2003; Abdo-Salem et al. 2006). The virus was probably introduced with infected livestock from the Horn of Africa, and it remains to be determined whether it has become endemic on the Arabian Peninsula.
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32

US GOVERNMENT. 21st Century Collection Centers for Disease Control (CDC) Emerging Infectious Diseases (EID): Comprehensive Collection from 1995 to 2002 with Accurate and Detailed Information on Dozens of Serious Virus and Bacteria Illnesses ¿ Hantavirus, Influenza, AIDS, Malaria, TB, Pox, Bioterrorism, Smallpox, Anthrax, Vaccines, Lyme Disease, Rabies, West Nile Virus, Hemorrhagic Fevers, Ebola, Encephalitis (Core Federal Information Series). Progressive Management, 2002.

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33

Karen, Bellenir, ed. Infectious diseases sourcebook: Basic consumer health information about non-contagious bacterial, viral, prion, fungal, and parasitic diseases spread by food and water, insects and animals, or environmental contact, including botulism, E. coli, encephalitis, Legionnaires' disease, Lyme disease, malaria, plague, rabies, salmonella, tetanus, and others, and facts about newly emerging diseases, such as hantavirus, mad cow disease, monkeypox, and West Nile virus, along with information about preventing disease transmission, the threat of bioterrorism, and current research initiatives, with a glossary and directory of resources for more information. Detroit, MI: Omnigraphics, 2004.

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