Relevant bibliographies by topics / Visceral

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Visceral'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Visceral"

1

Gebhart, GF. "Peripheral Contributions to Visceral Hyperalgesia." Canadian Journal of Gastroenterology 13, suppl a (1999): 37A—41A. http://dx.doi.org/10.1155/1999/730765.

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Hyperalgesia has long been recognized clinically as a consequence of tissue injury. Primary hyperalgesia (arising from the site of injury) is generally considered to be due to sensitization of sensory receptors (eg, nociceptors) and perhaps activation of so-called ‘silent nociceptors’ by mediators released, synthesized or attracted to the site of tissue injury. Key questions associated with understanding visceral hyperalgesia relate to whether the viscera are innervated by nociceptors (ie, sensory receptors that respond selectively to noxious intensities of stimulation), whether visceral receptors and/or afferent fibres sensitize after tissue injury and whether silent nociceptors exist in the viscera. Studies in nonhuman animals have revealed that hollow organs such as the esophagus, gall bladder, stomach, urinary bladder, colon and uterus are innervated by mechanically sensitive receptors with low or high thresholds for response. Accordingly, it appears that the viscera are innervated by nociceptors, although the issue is far from settled. One characteristic of cutaneous nociceptors is their ability to be sensitized when tissue is injured. Mechanosensitive visceral receptors also sensitize when the viscera are experimentally inflamed, but both visceral receptors with low thresholds and those with high thresholds for response are sensitized. Moreover, it is often not appreciated that visceral receptors are likely polymodal rather than unimodal – that is, mechanically sensitive visceral receptors typically are also sensitive to chemical and/or thermal stimuli. In this sense, visceral receptors may be considered evolutionarily older than more highly developed, specialized cutaneous receptors. Finally, there are mechanically insensitive receptors that innervate the viscera and, when tissue is injured, develop spontaneous activity and acquire sensitivity to mechanical stimuli. In the aggregrate, visceral receptors change their behaviour in the presence of tissue injury and, along with activated mechanically insensitive receptors, increase the afferent barrage into the spinal cord, contributing to the development of visceral hyperalgesia.
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Cabioglu, Mehmet Tugrul, and Gülnaz Arslan. "Neurophysiologic Basis of Back-Shu and Huatuo-Jiaji Points." American Journal of Chinese Medicine 36, no. 03 (January 2008): 473–79. http://dx.doi.org/10.1142/s0192415x08005916.

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Acupuncture, a method of traditional Chinese medicine that uses Back-Shu and Huatuo-Jiaji points, is especially effective for treating diseases of the visceral organs. Applying acupuncture on Back-Shu points affects visceral organs in many ways. For example, it dilates the bronchus, affects the heartbeat, stomach motility, urinary bladder contractions and so on. Acupuncture's effects can be explained as viscero-cutaneous, cutaneo-visceral, cutaneo-muscular, and viscero-muscular reflexes. Segmental dispersion of the sympathetic and parasympathetic systems is related to the location of Back-Shu points. Changes in visceral organs caused by application of acupuncture can be explained as modulation of the sympathetic and parasympathetic systems.
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Tao, Zhuo-Ying, Yang Xue, Jin-Feng Li, Richard J. Traub, and Dong-Yuan Cao. "Do MicroRNAs Modulate Visceral Pain?" BioMed Research International 2018 (October 10, 2018): 1–10. http://dx.doi.org/10.1155/2018/5406973.

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Visceral pain, a common characteristic of multiple diseases relative to viscera, impacts millions of people worldwide. Although hundreds of studies have explored mechanisms underlying visceral pain, it is still poorly managed. Over the past decade, strong evidence emerged suggesting that microRNAs (miRNAs) play a significant role in visceral nociception through altering neurotransmitters, receptors and other genes at the posttranscriptional level. Under pathological conditions, one kind of miRNA may have several target mRNAs and several kinds of miRNAs may act on one target, suggesting complex interactions and mechanisms between miRNAs and target genes lead to pathological states. In this review we report on recent progress in examining miRNAs responsible for visceral sensitization and provide miRNA-based therapeutic targets for the management of visceral pain.
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Jouet, D., H. Ferté, C. Hologne, M. L. Kaltenbach, and J. Depaquit. "Avian schistosomes in French aquatic birds: a molecular approach." Journal of Helminthology 83, no. 2 (June 2009): 181–89. http://dx.doi.org/10.1017/s0022149x09311712.

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AbstractThe prevalence of human cercarial dermatitis (HCD) caused by bird schistosomes appears to be increasing in France, in light of the impact of tourism combined with high densities of wild aquatic hosts in freshwater areas. The present work expands our knowledge of schistosome systematics by including samples of bird schistosomes collected from their natural hosts in France. Heads (318) and viscera (81) of aquatic birds belonging to 16 species from five orders, collecting during the hunting seasons or found dead, were autopsied for nasal and visceral schistosomes. Eggs and/or adults were analysed by molecular methods using the D2 domain and the second internal transcribed spacer (ITS-2) region of rDNA to determine species. Even if nasal eggs were polymorphic according to the host, all haplotypes were similar to that of Trichobilharzia regenti. Marked diversity of visceral species was observed. Final hosts under natural conditions were reported. For the first time, Trichobilharzia franki is reported in its natural bird hosts, Anas platyrhynchos, Anas crecca, Aythya fuligula and Cygnus olor. We also identified T. szidati in A. crecca and Anas clypeata. Bilharziella polonica was found in six species of aquatic birds, including Grus grus. This finding is the first record of bird schistosomes in this aquatic bird. Three new taxa of visceral schistosomes in Anser anser are strongly suspected according to their haplotypes. Futhermore, a new haplotype of visceral schistosomes isolated in Cygnus olor and similar to Allobilharzia visceralis was identified.
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Gebhart, G. F., and T. J. Ness. "Central mechanisms of visceral pain." Canadian Journal of Physiology and Pharmacology 69, no. 5 (May 1, 1991): 627–34. http://dx.doi.org/10.1139/y91-093.

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Deep pain arising from muscle, joints, connective tissue, and the viscera is different in character and quality from pain arising from cutaneous structures. Deep pains, particularly visceral pain, are poorly localized, typically referred or transferred to a cutaneous site, and generally produce strong emotional and autonomic responses and tonic muscle contractions. Despite the prevalence and clinical importance of deep pains, it is only relatively recently that investigative efforts have begun to focus on the mechanisms of deep pain. The present report briefly reviews the development and use of a model of visceral pain that employs constant pressure distension of the colon and rectum as a noxious stimulus. Converging behavioral, pharmacological, and physiological evidence that colorectal distension is a valid, reliable, noxious, visceral stimulus is presented.Key words: visceral pain, colorectal distension, pain modulation, pain model, adequate visceral stimuli.
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Berkley, K. J., G. Guilbaud, J. M. Benoist, and M. Gautron. "Responses of neurons in and near the thalamic ventrobasal complex of the rat to stimulation of uterus, cervix, vagina, colon, and skin." Journal of Neurophysiology 69, no. 2 (February 1, 1993): 557–68. http://dx.doi.org/10.1152/jn.1993.69.2.557.

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1. Previous studies in the rat and other species have shown that neurons in and near the ventrobasal complex (VB) can be activated by various visceral as well as somatic stimuli. 2. This study examined the responses of 84 single neurons in and near the rostral 2/3 of VB in 19 adult female rats in estrus to mechanical stimulation of the skin (brush, pressure, noxious pinch) and 4 different visceral stimuli, as follows: distension of both uterine horns, mechanical probing of the vagina, gentle pressure against the cervix, and distension of the colon. The rats were studied while under moderate gaseous anesthesia (33% O2-67% N2O + 0.5% halothane) and paralyzed (pancuronium bromide). 3. Of 77 neurons tested with both somatic and visceral stimuli, 70 were responsive to one type and/or the other. Responses to somatic stimuli were immediate with brief afterdischarges to the pinch stimuli. In contrast, responses to visceral stimuli were delayed an average of 9 s with long afterdischarges averaging 2 min. Most viscerally responsive neurons (74%) had somatic receptive fields, often (44%) to noxious pinch. 4. Of the 70 responsive neurons, 43 (61%) responded to 1 or more of the 4 visceral stimuli, primarily with excitation. Most of these 43 neurons (71%) were responsive to uterine distension, whereas fewer responded to stimulation of the cervix (45%), vagina (29%), or colon (34%). 5. Viscerally responsive neurons were preferentially located in regions bordering or near VB. Only 6 of 22 neurons within the core of VB (27%) responded to visceral stimuli, in contrast with 37 of 48 neurons bordering or near VB (77%). 6. The six viscerally responsive neurons within VB all had somatic receptive fields located primarily on the caudal part of the body and were responsive to only one or two of the four visceral stimuli, usually the uterus. The 37 viscerally responsive neurons bordering or near VB were of 3 types. Neurons of the first type (n = 15) were scattered throughout the areas bordering VB and responded to both somatic and visceral stimuli much like VB neurons, except that they showed more visceral convergence. Neurons of the second type (n = 11) were concentrated at the rostral and dorsal borders of VB and responded only to visceral stimuli, mainly the uterus. Neurons of the third type (n = 11) were concentrated ventrally and had very complex, long-lasting and history-dependent response characteristics to both visceral and somatic stimuli.(ABSTRACT TRUNCATED AT 400 WORDS)
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Heineman, Katherine. "An Osteopathic Manipulative Treatment (OMT) Evaluation and Treatment Protocol to Improve Gastrointestinal Function." AAO Journal 32, no. 2 (June 1, 2022): 34–44. http://dx.doi.org/10.53702/2375-5717-32.2.34.

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Abstract As a hands-on approach to patient care diagnosis and management, osteopathic manipulative medicine (OMM) can be utilized to modulate the autonomic input to the gastrointestinal system. Palpatory findings of tissue texture changes at predictable body regions may correspond to visceral dysfunction related to the gastrointestinal (GI) system.1 Osteopathic manipulative treatment (OMT) of the viscero-somatic segment or viscero-visceral reflex can remove the feedback related to the somatic or visceral component, thereby affecting nociceptive facilitation at the spinal or visceral level and helping to restore autonomic balance.1,2 The purpose of this thesis is to describe an evaluation and treatment protocol to address somatic and visceral dysfunction found in many patients with impaired gastrointestinal function. A retrospective analysis of 5 patients will be outlined using the evaluation and treatment protocol. The safety of an OMT evaluation and treatment protocol as applied to address gastrointestinal function and as outlined in the current literature will also be addressed.
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Blackshaw, L. Ashley, Stuart M. Brierley, Andrea M. Harrington, and Patrick A. Hughes. "TRP Channels in Visceral Pain." Open Pain Journal 6, no. 1 (March 8, 2013): 23–30. http://dx.doi.org/10.2174/1876386301306010023.

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Visceral pain is both different and similar to somatic pain - different in being poorly localized and usually referred elsewhere to the body wall, but similar in many of the molecular mechanisms it employs (like TRP channels) and the specialization of afferent endings to detect painful stimuli. TRPV1 is sensitive to low pH. pH is lowest in gastric juice, which may cause severe pain when exposed to the oesophageal mucosa, and probably works via TRPV1. TRPV1 is found in afferent fibres throughout the viscera, and the TRPV1 agonist capsaicin can recapitulate symptoms experienced in disease. TRPV1 is also involved in normal mechanosensory function in the gut. Roles for TRPV4 and TRPA1 have also been described in visceral afferents, and TRPV4 is highly enriched in them, where it plays a major role in both mechanonociception and chemonociception. It may provide a visceral-specific nociceptor target for drug development. TRPA1 is also involved in mechano-and chemosensory function, but not as selectively as TRPV4. TRPA1 is colocalized with TRPV1 in visceral afferents, where they influence each other's function. Another modulator of TRPV1 is the cool/mint receptor TRPM8, which, when activated can abrogate responses mediated via TRPV1, suggesting that TRPM8 agonists may provide analgesia via this pathway. In all, the viscera are rich in TRP channel targets on nociceptive neurones which we hope will provide opportunities for therapeutic analgesia.
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Fankboner, Peter V., and J. Lane Cameron. "Seasonal atrophy of the visceral organs in a sea cucumber." Canadian Journal of Zoology 63, no. 12 (December 1, 1985): 2888–92. http://dx.doi.org/10.1139/z85-432.

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The gut, gonad, respiratory trees, and circulatory system of the commercial sea cucumber Parastichopus californicus are annually lost as a result of atrophy of these organs and not, as originally supposed, through spontaneous, seasonal evisceration. Visceral loss is preceded by cessation of feeding–locomotory behaviour. Torpor ensues, and the visceral tissues are absorbed through a progressive process which includes phagocytosis by the sea cucumber's coelomocytes and, in some instances, the scavenging activities of endosymbionts. Regeneration of the viscera occurs within several weeks. Similar seasonal atrophy of the visceral organs has not been reported to occur in other coelomate organisms. We hypothesize that visceral atrophy in P. californicus is an expression of seasonal diapause induced by reduced food availability.
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Lim, Robert K. S. "VISCERAL RECEPTORS AND VISCERAL PAIN." Annals of the New York Academy of Sciences 86, no. 1 (December 15, 2006): 73–89. http://dx.doi.org/10.1111/j.1749-6632.1960.tb42791.x.

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Dissertations / Theses on the topic "Visceral"

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Siddiqui, Mahveen. "Asymptomatic visceral leishmaniasis." Thesis, London School of Hygiene and Tropical Medicine (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.313394.

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McGuire, Cian. "Human visceral afferent recordings : a pre-clinical human model of visceral pain." Thesis, Queen Mary, University of London, 2014. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8955.

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Aim: We have recently developed electrophysiological recordings of human visceral afferent (HVA) activity in isolated gastrointestinal tissues. The aim of the this study was 1) test the mechano- and chemosensitivity of HVAs, 2) characterise subpopulations of HVAs based on their response to mechanical stimuli, 3) test the effect of drugs that have/are in clinical trials on the mechanosensitivity (von Frey hair (VFH) probing and appendix distension) of HVAs. Methods: All experiments were performed in accordance with UK human ethics regulations [NREC09/H0704/2]. Surgically resected human ileum, colon, and appendix were obtained from consenting patients undergoing bowel resection. Tissues were pinned in a tissue bath, or cannulated (appendix), and superfused with carbongenated Krebs buffer, at 6ml/min, 32-34°C. Mesenteric nerve bundles were carefully dissected and afferent activity was recorded using suction electrodes. Tissues were tested for mechanosensitivity (VFHs, stretching, mucosal stroking, distension) and chemosensitivity (bradykinin (BK), ATP (adenosine trisphosphate), PGE2 (prostaglandin E2), serotonin (aka 5-hydroxytryptamine (5-HT)), histamine, adenosine). The receptors involved in the activation of HVAs by BK, or ATP were also investigated. The response of HVAs to VFH probing or distension was tested before and after the application of tegaserod, STa endotoxin, or a transient receptor potential vanilloid 4 (TRPV4) agonist (GSK1016790A) or antagonist (HC067047). Results and Conclusion: HVAs were characterised as mesenteric, serosal, muscular, or muscular-mucosal. HVAs were chemosensitive to all mediators. Bradykinin B2 receptors are the most important receptors involved in the activation of HVAs by BK. P2Y receptors may play an important role in the activation of HVAs by ATP. Application of tegaserod, HC067047 or STa endotoxin reduced the HVA response to mechanical stimuli. HVA recordings are feasible and practical and are suitable for both basic scientific mechanistic studies, and could potentially be used as a pre-clinical model, in conjunction with animal experiments, to help predict the efficacy of novel compounds before clinical trials.
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Shiwlochan, Amrita G. "Prenatal androgens and visceral fat." Honors in the Major Thesis, University of Central Florida, 2009. http://digital.library.ucf.edu/cdm/ref/collection/ETH/id/1327.

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This item is only available in print in the UCF Libraries. If this is your Honors Thesis, you can help us make it available online for use by researchers around the world by following the instructions on the distribution consent form at http://library.ucf.edu/Systems/DigitalInitiatives/DigitalCollections/InternetDistributionConsentAgreementForm.pdf You may also contact the project coordinator, Kerri Bottorff, at kerri.bottorff@ucf.edu for more information.
Bachelors
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Anthropology
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Perry, Meghan Rose. "Arsenic, antimony and visceral leishmaniasis." Thesis, University of Dundee, 2014. https://discovery.dundee.ac.uk/en/studentTheses/14edf50b-4943-4ec8-8556-8aaecf3a9f49.

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In Bihar state, India, the cure rate of antimonial compounds in the treatment of visceral leishmaniasis (VL) has declined from over 85% to less than 50%. This has been attributed to prolonged, widespread misuse of antimonials within the Indian private healthcare system. An alternative resistance hypothesis is that exposure to arsenic in drinking water in this region has resulted in antimony-resistant Leishmania parasites. Leishmania donovani were serially passaged in mice exposed to environmentally-relevant levels of arsenic in drinking water. Arsenic accumulation in organs of these mice was proportional to exposure. After five passages, isolated parasites were refractory to SbV in drug sensitivity assays. Treatment of infected mice with SbV confirmed that these parasites retained resistance in vivo, supporting this hypothesis. A retrospective field study on a cohort of antimony treated VL patients was performed in an arsenic contaminated area of Bihar to evaluate the presence of an increased risk of treatment failure and death in those exposed to arsenic. It demonstrated a significant increased risk of death from VL in arsenic exposed patients but did not indicate a significant relationship between arsenic exposure and antimonial treatment failure. Collectively these data suggest that it is biochemically possible that arsenic contamination may have contributed to the development of antimonial resistance in Bihar although issues of underpower and the retrospective nature of our epidemiological study made it difficult to conclusively demonstrate this. Further research in to the relationships between arsenic exposure and antimonial treatment failure and death in the leishmaniases is warranted.
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Schröder, Guilherme. "Deseducação visceral : compostagens e decomposições." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2017. http://hdl.handle.net/10183/170345.

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Esta dissertação se apresenta por três movimentos básicos. Crítico: os resquícios do militante, ainda insistente em vociferar contra a escola e contra as aberrações da internação compulsória da ensinagem. Poético: um descabimento encantado, ainda meio romântico, que beira o sem sentido. Mas que encontra suas potências justamente no não saber, no mistério e na dúvida. Caósmico: imersão na criação suja, porca, fecal, destrutiva, shivaísta de mundos. Ao mesmo tempo experimentação potência, em êxtase, gozo intenso, vibrante de soltura. Através de fragmentos compostados a pesquisa se mistura com as decomposições institucionais. Percebe pequenos restos, descreve breves abandonos. E olha enquanto vive, para a vasta compostagem acontecendo nos conceitos educativos.
Three basic movements present this dissertation Critic: the militant remmants, still loudly yelling against school and against compulsory teaching aberrations. Poetic: a delighted disregard, still romantic, in the nonsense edge. That find your powers in don't knowing, in the mystery and in doubt. Caosmic: worlds dirty creation immersion, hog, faecal, destructive, shivaish. At the same time powerful experimentation, in bliss, intense joy, vibrant release. Trough composted fragments the research is mixed with the institutional decompositions. Look at small scraps, describes brief neglects. And looks while living, to the large education concepts composting.
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Genari, Isabel Cristina Contel [UNESP]. "Conhecimento de escolares sobre leishmaniose visceral." Universidade Estadual Paulista (UNESP), 2009. http://hdl.handle.net/11449/94726.

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A leishmaniose visceral (LV) vem se tornando um problema de saúde pública e atividades de educação em saúde podem resultar em maior participação da comunidade para o controle desta zoonose, pois tem forte potencial epidemiológico. Este estudo teve como objetivo avaliar o conhecimento sobre LV de escolares de 6º e 7º anos do Ensino Fundamental de três escolas públicas do município de Birigui-SP e a variação deste conhecimento após realização de um programa educativo. Um questionário foi aplicado antes (Etapa I) e após (Etapa II) a realização do projeto educativo que se constituiu de aula expositiva, palestra realizada por profissional de saúde, confecção de história em quadrinhos e de cruzadinha sobre o tema. Um total de 711 escolares foi entrevistado na Etapa I e 693 na Etapa II. Adotou-se critério de conhecimento de LV como “Bom”, “Regular” e “Ruim” quando, de 10 questões analisadas pela Teoria de Resposta ao Item, os escolares tiveram 10- 8, 7-4, e 3-0 acertos, respectivamente. Verificou-se que houve aumento estatisticamente significante do nível de conhecimento, com o número de escolares com conceito Bom variando de 35,7% (Etapa I) para 59,7% (Etapa II), com conceito Regular de 54,3% (Etapa I) para 35,3% (Etapa II) e com conceito Ruim de 10,0% (Etapa I) para 4,9% (Etapa II). As atividades educativas realizadas geraram ganhos de conhecimento entre os escolares, o que permite concluir que programas educativos de forma continuada podem trazer resultados para a saúde pública.
Visceral leishmaniasis (VL) is a public health problem and education activities in health can result in greater community participation for the control of zoonoses. We aimed at evaluating the 6th and 7th grade students’ knowledge on visceral leishmaniasis from three public schools of Birigui,SP, Brazil. We also evaluated the knowledge gaining before and after an educative project were carried out by using a questionnaire before (Phase I) and after (Phase II) these activities that comprehended one speech, making one comic and one crossword about VL. We interviewed 711 students in Phase I and 693 in Phase II. A criterion of knowledge of VL was adopted as “Good”, “Medium” and “Bad” when, out of 10 questions analyzed by Item Response Theory, 10-8, 7-4, and 3-0 were right, respectively. We observed a statistically significant increase in the students’ level of knowledge after the educational project, since the number of students with “Good” concept changed from 35.7% (Phase I) to 59.7% (Phase II); the “Medium” concept changed from 54.3% to 35.3% and the “Bad” concept, from 10,0% to 4.9%. The educational activities carried out led to gains in knowledge among students suggesting that continuing education can bring important results to public health.
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Almeida, Breno Fernando Martins de [UNESP]. "Estresse oxidativo na leishmaniose visceral canina." Universidade Estadual Paulista (UNESP), 2013. http://hdl.handle.net/11449/121938.

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Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
Oxidative stress occurs due to an imbalance between oxidants and antioxidant defenses. Considering that dogs with leishmaniasis may develop uremia, which affects the oxidative metabolism and apoptosis of neutrophils, we proposed to investigate the hypothesis that oxidative stress, alteration of oxidative metabolism and apoptosis of neutrophils in dogs varies according to stage of leishmaniasis and that uremia, commonly occurs in late stages of disease, would contribute to such variations. To this, we evaluated plasma markers of oxidative stress (total antioxidant capacity, lipid peroxidation, glutathione, uric acid, bilirubin and albumin), superoxide production (method of nitroblue tetrazolium reduction and flow cytometry with the probe hydroethidine), and viability and apoptosis rate (morphological method and flow cytometry using annexin V-PE system) on neutrophils from dogs with leishmaniasis classified according to the LeishVet Consensus in moderate stage of the disease, very severe stage when uremia is installed and dogs with uremia negative for leishmaniasis, comparing them to healthy dogs. Leishmaniasis regardless of stage and uremia cause oxidative stress with reduced total antioxidant capacity, which may be related to greater induced production of superoxide and stimulated apoptosis observed in these groups. While spontaneously, neutrophils of dogs in very severe stage and with uremia had lower viability, increased apoptosis and higher lipid peroxidation, suggesting that uremia is the cause of such changes. Can be concluded that the intensity of oxidative stress does not vary with the stage of leishmaniasis, however uremic condition contributes to reduce the viability of neutrophils in late-stage dogs. Changes of neutrophil oxidative metabolism occur concomitant with oxidative stress and increased neutrophil apoptosis in both leishmaniasis and in uremia alone
FAPESP: 2011/14083-0
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Humphrey, Jay Dowell. "Mechanical behavior of excised visceral pleura." Diss., Georgia Institute of Technology, 1985. http://hdl.handle.net/1853/19973.

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Ahmed, Ahmed Ali. "Study of visceral leishmaniasis in Ethiopia." Thesis, University of Liverpool, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.316716.

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Dourado, Cibele Marie Ribeiro. "Immunodiagnosis of visceral leishmaniasis in Brazil." Thesis, University of Liverpool, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.357398.

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Books on the topic "Visceral"

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Montanari, Sergio Ariel. Visceral. Rosario: Editorial Ciudad Gótica, 2010.

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Parish, Simon. Visceral reality. [Amsterdam]: Parish, 1993.

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Bellman, Richard. Visceral vision. [Montreal: The Author], 1989.

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Barral, J. P. Visceral manipulation. Seattle: Eastland Press, 1988.

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Ádám, György. Visceral Perception. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4757-2903-0.

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Visceral reason. Oxon, U.K: Taylor & Francis, 2005.

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Cavanagh, David. Ethic, visceral: Poems. Burlington, VT: Wood Thrush Books, 1987.

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Barral, J. P. Visceral vascular manipulations. Edinburgh: Churchill Livingstone/Elsevier, 2011.

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Cameron, Oliver G. Visceral sensory neuroscience: Interoception. New York, N.Y: Oxford University Press, 2002.

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Shea, Michael J. Visceral manipulation illustrated notes. Juno Beach, FL (13878 Oleander Ave., Juno Beach, 33408): Shea Educational Group, 1996.

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Book chapters on the topic "Visceral"

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Gebhart, G. F. "Visceral Pain and Visceral Hyperalgesia." In Pain Management and Anesthesiology, 75–79. Dordrecht: Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-011-5145-0_7.

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Oderich, Gustavo S., and Karina S. Kanamori. "Visceral Artery Revascularization Visceral artery revascularization." In PanVascular Medicine, 3989–4014. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-37078-6_126.

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Ádám, György. "The Subject of This Monograph." In Visceral Perception, 3–6. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4757-2903-0_1.

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Ádám, György. "Visceroception A Borderline Sensory System." In Visceral Perception, 119–33. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4757-2903-0_10.

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Ádám, György. "Visceroception and Cognition." In Visceral Perception, 135–59. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4757-2903-0_11.

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Ádám, György. "Visceral Perception and Symptom Report An Epilogue." In Visceral Perception, 161–67. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4757-2903-0_12.

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Ádám, György. "Definitions and Terminology." In Visceral Perception, 7–16. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4757-2903-0_2.

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Ádám, György. "Historical Roots and Evolution." In Visceral Perception, 17–27. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4757-2903-0_3.

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Ádám, György. "Conceptual Preliminaries." In Visceral Perception, 31–35. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4757-2903-0_4.

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Ádám, György. "Functional Properties of Receptor Structures." In Visceral Perception, 37–55. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4757-2903-0_5.

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Conference papers on the topic "Visceral"

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Quintana, Rebecca M., Yuanru Tan, Kathryn Gabriele, and Noni Korf. ""It's Just that Visceral"." In CHI '18: CHI Conference on Human Factors in Computing Systems. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3170427.3188650.

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Forehand, Leslie. "Visceral Data – Observations of Point Clouds." In 107th ACSA Annual Meeting. ACSA Press, 2019. http://dx.doi.org/10.35483/acsa.am.107.8.

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Dalegrave, Suélen, João Pedro Grassi de Araujo, and Maurício Orlando Wilnsen. "LEISHMANIOSE VISCERAL CANINA – RELATO DE CASO." In I CONGRESSO NACIONAL DE RESIDÊNCIAS EM SAÚDE (ONLINE). EDITORA OMNIS SCIENTIA, 2021. http://dx.doi.org/10.47094/iconres.2021/7.

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But, Michal. "SP15 Sympathetic blocks for visceral malignancies." In ESRA Abstracts, 39th Annual ESRA Congress, 22–25 June 2022. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/rapm-2022-esra.16.

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Batista, Erika Rodrigues de Senna, Júlia Rodrigues de Senna Mendonça, André Rodrigues de Senna Batista Filho, Maria Eduarda Vieira de Senna Batista, Ana Letícia Cunha Faria, Fernanda Quadros Mendonça, Divino Urias Mendonça, and Welberth Fernandes de Souza. "SYSTEMIC LUPUS ERYTHEMATOSUS OVERLAPPING VISCERAL LEISHMANIASIS." In XXXIX Congresso Brasileiro de Reumatologia. Sociedade Brasileiro de Reumatologia, 2022. http://dx.doi.org/10.47660/cbr.2022.1996.

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Brasil, Salomão Bruno Dos Santos, Matteus Gomes De Oliveira, Luis Felipe Ferreira Carneiro, Osmar Júnior Da Silva Silva, and Josuelem Portela Castro. "LEISHMANIOSE VISCERAL E SUAS CONSEQUÊNCIAS HEMATOLÓGICAS." In II Congresso Brasileiro de Hematologia Clínico-laboratorial On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/hematoclil/130.

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Introdução: A leishmaniose visceral (LV) é uma doença endêmica brasileira, com origem histórica advinda da região Nordeste, tendo importante incidência estimada de 500.000 casos e 50.000 mortes por ano no mundo inteiro. A patologia se caracteriza pela infecção do sistema fagocítico mononuclear (SFM) a partir do protozoário intracelular obrigatório Leishmania chagasi (no Brasil), tendo como agente de transmissão os flebotomíneos Lutzomyia longipalpis e Lutzomyia cruzi. As manifestações hematológicas são amplas e de importância clínica, tendo como principais características anemia e neutropenia sempre presentes. Objetivo: Evidenciar os aspectos hematológicos adquiridos por meio do quadro de leishmaniose visceral e expor as conclusões chegadas a partir da pesquisa. Metodologia: O levantamento da bibliografia foi realizado usando os bancos de dados científicos PubMed e SciELO. Foram usados os seguintes descritores para se desenvolver o seguinte trabalho, tanto em língua portuguesa, como inglesa: “visceral leishmaniasis”, “anemia”, “thrombocytopenia” e “leukopenia”. Resultados: É encontrado na literatura que as manifestações hematológicas da LV se apresentam principalmente com pancitopenia, fibrinólise, hemólise, entre outras que estão diretamente ligadas a fisiopatogenia da doença ao se considerar que a medula óssea ,neste quadro, se encontra altamente parasitada e com aspecto celular hiperplásico. O sequestro esplênico, por vezes, se encontra associado a anemia característica, apesar de ser eminentemente multifatorial. Conclusão: Chega-se a dedução, portanto, de que alguns aspectos hematológicos podem resultar em aumento da letalidade para a leishmaniose visceral. Mesmo que haja a possibilidade de se identificar as alterações por meio de exames complementares, é essencial se ter atenção para as manifestações clínicas e histórico pessoal, para se ter uma abordagem terapêutica efetiva no âmbito do tempo e farmacológico.
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SOUZA, CAIO CEZAR NOGUEIRA DE, JACQUELINE DA SILVA BRITO, MARCELY KAREN SANTOS DO ROSÁRIO, ALZIRA ALCANTARA MENDES QUEIROZ NETA, and ALEXANDRE DO ROSÁRIO CASSEB. "LEISHMANIOSE VISCERAL CANINA - RELATO DE CASO." In I Congresso Nacional de Especialidades Veterinárias On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/convesp/5843.

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Introdução: A leishmaniose visceral é uma zoonose parasitária que no Brasil é transmitida pelo vetor Lutzomyia longipalpis. O cão tem importância na epidemiologia da doença devido ser o reservatório doméstico e na clínica, pelo fato de manifestar sintomatologia semelhante a infecção em humanos, diferindo apenas no que tange a lesões dérmicas encontradas em animais infectados e sintomáticos. Objetivo: Dessa forma, o objetivo desse estudo é relatar um caso de Leishmaniose Visceral em um cão e as consequências oriundas dessa infecção. Relato de caso: Foi atendido no dia 14/06 no Hospital Veterinário da Universidade Federal Rural da Amazônia um animal que foi adquirido em um canil há 2 anos, local onde a mãe veio a óbito pouco tempo depois devido a leishmaniose. Há 7 meses havia sido diagnosticado com a doença e no momento da consulta apresentava prurido, lesões de pele e grandes áreas de alopecia, durante o exame físico apresentou uma característica incomum nos olhos, foram solicitados hemograma, bioquímicos, PCR, RIFI e ELISA para leishmania e parasitológico de pele em fita. Resultados: Os resultados evidenciaram anemia, linfopenia, trombocitopenia, leucopenia, enzimas hepáticas e renais sem alterações, PCR negativo, RIFI reagente, ELISA reagente e parasitológico em fita negativo. Após a confirmação do diagnóstico, foram receitados Miltefosina 2%, Alopurinol 300mg, Domperidona 10mg, e tratamento de suporte, além disso foi usado coleira repelente Seresto, Sebolytic para banhos terapêuticos e Allenderm Spot 4ml na pele após banho. Conclusão: Após 30 dias de tratamento foi possível observar considerável melhora nos parâmetros hematológicos, além de redução do prurido e retomada do crescimento da pelagem. O animal passou a ser acompanhado mensalmente até ter alta clínica.
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Cordeiro, Camilla Natacha Correia, Pedro Henrique dos Santos Silva, Luana Helen Gonçalves Slauta, Rafaela Oliveira de Arruda, Paola Menezes Ribeiro, and Joslaine Adrian da Silva. "REVISÃO DE LITERATURA: LEISHMANIOSE VISCERAL CANINA." In IV Congresso Iberoamericano de Saúde Pública Veterinária. Congresse.me, 2022. http://dx.doi.org/10.54265/hwse9279.

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Victor, UN, RJ Schwab, SJ Ratcliffe, EA Beothy, BE Staley, and GW Pien. "Visceral Fat Measurements and AHI in Women." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5407.

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Goksel, Orcun, Valery Vishnevsky, Alvaro Gomariz Carrillo, and Christine Tanner. "Imaging of sliding visceral interfaces during breathing." In 2016 IEEE 13th International Symposium on Biomedical Imaging (ISBI 2016). IEEE, 2016. http://dx.doi.org/10.1109/isbi.2016.7493268.

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Reports on the topic "Visceral"

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Ozdenoren, Emre, Stephen Salant, and Dan Silverman. Willpower and the Optimal Control of Visceral Urges. Cambridge, MA: National Bureau of Economic Research, June 2006. http://dx.doi.org/10.3386/w12278.

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Carlson, Dawn E., William G. Cioffi, Mason Jr., McManus Arthur D., Pruitt William F., and Jr Basil A. Evaluation of Serum Visceral Protein Levels as Indicators of Nitrogen Balance in Thermally Injured Patients. Fort Belvoir, VA: Defense Technical Information Center, August 1991. http://dx.doi.org/10.21236/ada244867.

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Ismaiel, Abdulrahman, Ayman Jaaouani, Daniel-Corneliu Leucuta, Stefan-Lucian Popa, and Dan-Lucian Dumitrascu. The Visceral Adiposity Index in Non-Alcoholic Fatty Liver Disease and Liver Fibrosis — Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2021. http://dx.doi.org/10.37766/inplasy2021.12.0056.

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Review question / Objective: The objective of the study was to compare the mean difference and AUROC of Visceral Adiposity Index (VAI) in NAFLD/NASH/liver fibrosis patients and controls in observational studies. Condition being studied: Nonalcoholic fatty liver disease (NAFLD) is a multi-system disease, being mainly a liver pathology involving excessive hepatic fat accumulation unrelated to alcohol consumption or other secondary causes of hepatic steatosis. It is an emerging cause of concern and increasing clinical burden, imposing a public health challenge. NAFLD is the most common chronic liver disease and is predicted to be the most common indication for a liver transplant in Western countries by 2030, owing to a prevalence of 25% worldwide. The visceral adiposity index (VAI) is a scoring system based on body mass index, triglycerides, high-density lipoproteins (HDLs), and waist circumferences (WCs).
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Harmon, David L., Israel Bruckental, Gerald B. Huntington, Yoav Aharoni, and Amichai Arieli. Influence of Small Intestinal Protein on Carbohydrate Assimilation in Beef and Dairy Cattle. United States Department of Agriculture, August 1995. http://dx.doi.org/10.32747/1995.7570572.bard.

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The long term goal of the proposed research, "Influence of small intestinal protein on carbohydrate assimilation and metabolism in beef and dairy cattle" was to define the limits of small intestinal starch digestion and clarify regulatory mechanisms involved in starch assimilation in cattle. It was hypothesized that dietary protein plays a critical role in the regulation of intestinal digestion; however, studies clearly identifying this role were lacking. The first two experiments quantified starch digestion (disappearance from the small intestine) in response to known increments in duodenal protein supply and found that the quantity of DM, OM and starch disappearing from the small intestine increased linearly (P <.01) with protein infusion. A follow-up experiment also demonstrated that casein infusion linearly increased pancreatic a-amylase concentration and secretion rate. The final experiment provided critical data on metabolic fates of glucose derived from intestinal starch digestion. These data demonstrated that increasing postruminal starch supply does increase the metabolism of glucose by visceral tissues: however, this increase is minor (20%) compared with the increase in portal production (70%). These changes can have a dramatic impact on the glucose economy of the animal and result in large increases in the amount of glucose reaching peripheral tissues.
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Yahav, Shlomo, John McMurtry, and Isaac Plavnik. Thermotolerance Acquisition in Broiler Chickens by Temperature Conditioning Early in Life. United States Department of Agriculture, 1998. http://dx.doi.org/10.32747/1998.7580676.bard.

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The research on thermotolerance acquisition in broiler chickens by temperature conditioning early in life was focused on the following objectives: a. To determine the optimal timing and temperature for inducing the thermotolerance, conditioning processes and to define its duration during the first week of life in the broiler chick. b. To investigate the response of skeletal muscle tissue and the gastrointestinal tract to thermal conditioning. This objective was added during the research, to understand the mechanisms related to compensatory growth. c. To evaluate the effect of early thermo conditioning on thermoregulation (heat production and heat dissipation) during 3 phases: (1) conditioning, (2) compensatory growth, (3) heat challenge. d. To investigate how induction of improved thermotolerance impacts on metabolic fuel and the hormones regulating growth and metabolism. Recent decades have seen significant development in the genetic selection of the meat-type fowl (i.e., broiler chickens); leading to rapid growth and increased feed efficiency, providing the poultry industry with heavy chickens in relatively short growth periods. Such development necessitates parallel increases in the size of visceral systems such as the cardiovascular and the respiratory ones. However, inferior development of such major systems has led to a relatively low capability to balance energy expenditure under extreme conditions. Thus, acute exposure of chickens to extreme conditions (i.e., heat spells) has resulted in major economic losses. Birds are homeotherms, and as such, they are able to maintain their body temperature within a narrow range. To sustain thermal tolerance and avoid the deleterious consequences of thermal stresses, a direct response is elicited: the rapid thermal shock response - thermal conditioning. This technique of temperature conditioning takes advantage of the immaturity of the temperature regulation mechanism in young chicks during their first week of life. Development of this mechanism involves sympathetic neural activity, integration of thermal infom1ation in the hypothalamus, and buildup of the body-to-brain temperature difference, so that the potential for thermotolerance can be incorporated into the developing thermoregulation mechanisms. Thermal conditioning is a unique management tool, which most likely involves hypothalamic them1oregulatory threshold changes that enable chickens, within certain limits, to cope with acute exposure to unexpected hot spells. Short-tem1 exposure to heat stress during the first week of life (37.5+1°C; 70-80% rh; for 24 h at 3 days of age) resulted in growth retardation followed immediately by compensatory growth" which resulted in complete compensation for the loss of weight gain, so that the conditioned chickens achieved higher body weight than that of the controls at 42 days of age. The compensatory growth was partially explained by its dramatic positive effect on the proliferation of muscle satellite cells which are necessary for further muscle hypertrophy. By its significant effect of the morphology and functioning of the gastrointestinal tract during and after using thermal conditioning. The significant effect of thermal conditioning on the chicken thermoregulation was found to be associated with a reduction in heat production and evaporative heat loss, and with an increase in sensible heat loss. It was further accompanied by changes in hormones regulating growth and metabolism These physiological responses may result from possible alterations in PO/AH gene expression patterns (14-3-3e), suggesting a more efficient mechanism to cope with heat stress. Understanding the physiological mechanisms behind thermal conditioning step us forward to elucidate the molecular mechanism behind the PO/AH response, and response of other major organs. The thermal conditioning technique is used now in many countries including Israel, South Korea, Australia, France" Ecuador, China and some places in the USA. The improvement in growth perfom1ance (50-190 g/chicken) and thermotolerance as a result of postnatal thermal conditioning, may initiate a dramatic improvement in the economy of broiler's production.
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