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Journal articles on the topic 'Visceroptosis'

Author: Grafiati

Published: 4 June 2025

Last updated: 23 June 2025

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1

Bokovoj, Sergej P. "Surgical treatment of right-sized visceroptosis." Urologicheskie vedomosti 7, no. 4 (2017): 30–38. http://dx.doi.org/10.17816/uroved7430-38.

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This article is devoted to the surgical treatment of right-sided visceroptosis. The original methods of nephropexy, colopexy and hepatopexy are described in details. The indications and peculiarities of the preparation for these operations are given as well as the peculiarities of postoperative management of such patients. Long-term results of surgical treatment of right-sided visceroptosis in 103 patients are presented in the article. A good result was found in 93 (90.3%) patients and satisfactory result in 10 (9.7%) patients. (For citation: Bokovoj SP. Surgical treatment of right-sized visceroptosis. Urologicheskie vedomosti. 2017;7(4):30-38. doi: 10.17816/uroved7430-38).

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2

Ellis, Harold. "Surgery for visceroptosis." Journal of Perioperative Practice 28, no. 12 (2018): 366–67. http://dx.doi.org/10.1177/1750458918790178.

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3

Bokovoy, Sergej P. "Symptomatology and diagnostics of the right-sided visceroptosis." Urologicheskie vedomosti 7, no. 2 (2017): 16–24. http://dx.doi.org/10.17816/uroved7216-24.

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In this The results of examination of 442 patients admitted to the hospital with a preliminary diagnosis of “right-sided nephroptosis, visceroptosis” are presented. Variants of combined and isolated omissions of the right abdominal organs, diagnostic methods and clinical manifestations of nephroptosis, coloptosis and hepatoptosis are described. An algorithm for examining such patients is presented, which shows the most complete view of patients and choose the optimal method for their treatment. (For citation: Bokovoy SP. Symptomatology and diagnostics of the right-sided visceroptosis. Urologicheskie vedomosti. 2017;7(2):-24. doi: 10.17816/uroved72-24).

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4

Takakura, Will, Yaniv Raphael, Richard Sukov, and Ali Rezaie. "S3153 Hypermobile Ehlers-Danlos Syndrome and Visceroptosis." American Journal of Gastroenterology 115, no. 1 (2020): S1656—S1657. http://dx.doi.org/10.14309/01.ajg.0000714660.32906.3b.

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5

Pandey, Krishna, Nishtha Singh, Amit Nayak, and C. Mohanty. "Gross congenital abnormalities induced by leflunomide in mice embryos." European Journal of Anatomy 27, no. 4 (2023): 375–81. http://dx.doi.org/10.52083/wnei1659.

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Leflunomide is an antirheumatic drug commonly used by females, as this disease is common in females and there are chances of pregnancy while taking this medication in initial months of pregnancy this commonly prescribed drug lacks studies related to its teratogenic potential. Present study was conducted to know about its teratogenicity in mice embryos. Pregnant mice were exposed to Leflunomide by oral route on gestational days 6 to 11 either as single dose in one of the gestational days or continuous doses. The embryos were collected on day 19 of gestation, were measured and examined for external anomalies. Findings suggested that Leflunomide was embryo lethal when given as continuous dose as there were 100% resorption of embryos. In the single dose group, maximum resorptions were found when was given in early pregnancy. Other anomalies included malrotated limbs, open eyes, kinking of tails, defect in anterior abdominal wall and visceroptosis and anencephaly. these anomalies were noted in embryos exposed to leflunomide only on gestational days 7 and 8. The above findings suggest that leflunomide interferes with embryonic growth It also interferes with neural tube closure leading to anencephaly. Findings of open eyes and kinking tails suggest that the drug may affect epithelial and mesodermal growth. Leflunomide perhaps interferes with the lateral folding of the embryo leading to defect in the anterior abdominal wall and visceroptosis. The present study concludes that Leflunomide is teratogenic and embryolethal in mice and should be avoided in human pregnancy.

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6

Dordoni, Chiara, Marco Ritelli, Marina Venturini, et al. "Recurring and Generalized Visceroptosis in Ehlers-Danlos Syndrome Hypermobility Type." American Journal of Medical Genetics Part A 161, no. 5 (2013): 1143–47. http://dx.doi.org/10.1002/ajmg.a.35825.

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7

Saxena, Aaditya, Sangeetha Balumani, and Sanjeev Khera. "Visceroptosis: a rare clinical manifestation of nutritional Vitamin D deficiency rickets." BMJ Case Reports 17, no. 10 (2024): e262569. http://dx.doi.org/10.1136/bcr-2024-262569.

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8

Rezaie, Ali, Yaniv Raphael, Richard Sukov, and Xiaochen Liu. "Ehlers-Danlos Syndrome Type III (EDS) and Visceroptosis: Getting to the Bottom of This Diagnosis." American Journal of Gastroenterology 113, Supplement (2018): S270—S271. http://dx.doi.org/10.14309/00000434-201810001-00469.

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9

Baron, J. "The wax and wane of intestinal autointoxication and visceroptosis—historical trends of real versus apparent new digestive diseases." American Journal of Gastroenterology 97, no. 11 (2002): 2695–99. http://dx.doi.org/10.1016/s0002-9270(02)05467-9.

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10

Baron, J. H., and A. Sonnenberg. "The wax and wane of intestinal autointoxication and visceroptosis-historical trends of real versus apparent new digestive diseases." American Journal of Gastroenterology 97, no. 11 (2002): 2695–99. http://dx.doi.org/10.1111/j.1572-0241.2002.07050.x.

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11

Chan, Yin, Bianca Chang, Yaniv Raphael, Richard Sukov, and Ali Rezaie. "S527 Radiologic Identification of Visceroptosis in Patients With Hypermobile Ehlers-Danlos Syndrome (hEDS) With Functional Gastrointestinal (GI) Symptoms Compared to Healthy Subjects." American Journal of Gastroenterology 117, no. 10S (2022): e372-e372. http://dx.doi.org/10.14309/01.ajg.0000858748.17063.63.

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12

Reinstein, Eyal, Mark Pimentel, Mitchel Pariani, Stephen Nemec, Thomas Sokol, and David L. Rimoin. "Visceroptosis of the bowel in the hypermobility type of Ehlers–Danlos syndrome: Presentation of a rare manifestation and review of the literature." European Journal of Medical Genetics 55, no. 10 (2012): 548–51. http://dx.doi.org/10.1016/j.ejmg.2012.06.012.

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13

Shashmurina, V. R., O. L. Mishutina, M. A. Postnikov, A. B. Shashmurina, E. V. Dmitrieva, and R. V. Parfenov. "Phenotypic signs of connective tissue dysplasia in adolescents." Russian Osteopathic Journal, no. 2 (June 27, 2023): 119–33. http://dx.doi.org/10.32885/2220-0975-2023-2-119-133.

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Introduction. Connective tissue dysplasia (СTD) is a group of polymorphic pathological conditions caused by hereditary or congenital defects in collagen synthesis and accompanied by a violation of the functions of the musculoskeletal system and internal organs, which is considered as a factor in the development of pathology of the maxillofacial region in children: dystrophic forms of periodontal diseases; anomalies of shape, size, teething; pathology of the temporomandibularthe mandibular joint. There are diffi culties in determining the pathogenetic relationship between CTD and individual nosological forms of dental diseases, which is important for determining the prognosis of their course and the choice of methods of rehabilitation of children. It is important to improve the methodology of preventive examinations in relation to the early detection of phenotypic signs of CTD, including in the maxillofacial region. Substantiation of the connection of CTD with dental diseases will make it possible to develop examination and medical examination schemes, increase the effectiveness of comprehensive prevention of dental diseases and rehabilitation of adolescents with combined pathology.The aim is to study the incidence of CTD in adolescents and its relationship with dental diseases in order to develop a scheme of examinations and medical examinations, increase the effectiveness of comprehensive prevention of dental diseases and rehabilitation of adolescents with combined pathology.Materials and methods. A survey of 140 male adolescents aged 15–18 years, students of schools in Smolensk was conducted. When examining the somatic status, generally accepted markers of CTD were taken into account: bone (asthenic type of constitution, scoliotic deformity of the spine, hallux valgus, chest deformities), skin (increased skin extensibility of more than 3 cm), articular (hypermobility of joints) and visceral (changes from the cardiovascular system — prolapses of the heart valves, abnormally located chords; changes from the respiratory organs — polycystic lung disease, trachiobronchial dyskinesia; changes from the urinary system — nephroptosis, kidney doubling; changes from the gastrointestinal tract — visceroptosis, gallbladder anomaly; changes from the blood system — thrombocytopathy, hemoglobinopathy). When examining the dental status, the following generally accepted markers of CTD were taken into account: malocclusion (prognathia, deep bite, their combination), narrowing of the dentition, anomalies of the position of individual teeth, anomalies of soft tissue attachment, gum recession, chronic gingivitis. The diagnosis of CTD and the health group was established by a pediatrician on the basis of clinical recommendations and the order of the Ministry of Health of the Russian Federation № 621 dated 30.12.2003 «On a comprehensive assessment of the health status of children». Factor analysis was used to form a set of signs combining signs of CTD and dental diseases.Results. Of the phenotypic signs of СTD, bone and joint changes were diagnosed in 79,3 % (95 % CI 72,6– 86) of adolescents. In second place in frequency 35 % (95 % CI 27,1–42,9) were signs of СTD of the cardiovascular system and the organ of vision (myopia). 40 % (95 % CI 31,9–48,1) of the examined adolescents were diagnosed with chronic gingivitis, gum recession. Deep bite and prognathia were more common than other malocclusions: in 40,7 % (95 % CI 32,6–48,9) and 17,1 % (95 % CI 10,9–23,4), respectively. The use of factor analysis allowed us to identify four most informative factors (the fi rst of them is the most signifi cant) to characterize the relationship between the signs of CTD and its dental manifestations.Conclusion. The incidence of CTD in males in the age group of 15–18 years is 43 %. The most informative prognostic factor was the relationship of tracheobronchial dyskinesia with prognathia, deep bite, narrowing of the jaws, generalized gingivitis. The interrelation of signs of СTD of the cardiovascular system and bone-joint changes with gum recession, chronic gingivitis, prognathia and deep bite was also revealed.

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14

Matias, Mariana, and Madara Kronberga. "EP.TU.425Visceroptosis - obsolete condition or missed opportunity to intervene early?" British Journal of Surgery 108, Supplement_7 (2021). http://dx.doi.org/10.1093/bjs/znab311.054.

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Abstract Visceroptosis is defined as displacement or prolapse of abdominal organs below their natural position, most commonly stomach – gastroptosis – or bowel - enteroptosis. It is also known as Glénard’s disease, first described in 1885, when there is evidence of generalised visceroptosis. The aetiology of visceroptosis remains unknown, however some authors have suggested this condition to result from weakness of the ligaments that hold the abdominal organs in their anatomical place. Although patients with visceroptosis are often asymptomatic, this condition can also present with various gastrointestinal symptoms depending on its severity. The most common symptoms reported include anorexia, severe constipation or diarrhoea, abdominal distention, gastroparesia, nausea, intractable abdominal pain. The aim of this study is to review the most updated literature reporting cases of visceroptosis, the diagnostic approach and its subsequent management, encouraging all surgeons to be more attentive towards and accurately identify visceroptotic habitus. Method Literature review using PubMed, Web of science and other reliable sources using the possible combinations of the following key words: visceroptosis, gastroptosis, enteroptosis, gastrointestinal complications, surgery. Results Current literature is scant regarding this unusual presentation and it is often referred as a rare complication of Ehlers-Danlos Syndrome. Due to its rarity, it often remains underdiagnosed potentially leading to sub-optimal treatment of patients, and ultimately significant morbidity and mortality. Conclusion We believe this condition should be repopularised as serious complications such as visceral torsion might arise from it which may require immediate surgical treatment, affecting quality of life and a high rate of relapses after surgical procedures.

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15

Chan, Yin, Bianca W. Chang, Amrit K. Kaboj, Yaniv Raphael, Richard Sukov, and Ali Rezaie. "Radiographic identification of visceroptosis in hypermobile Ehlers-Danlos syndrome patients with functional gastrointestinal symptoms compared to healthy subjects." Clinical and Translational Gastroenterology, March 3, 2025. https://doi.org/10.14309/ctg.0000000000000834.

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INTRODUCTION: Visceroptosis is a potential cause of gastrointestinal symptoms in hypermobile Ehlers-Danlos syndrome (hEDS). METHODS: We systematically examined the prolapse of abdominal organs below their natural supine position (visceroptosis) during upright small bowel barium study in healthy and hEDS subjects with irritable bowel syndrome. RESULTS: Comparison of age- and sex-matched healthy (n=20) and hEDS (n=10) subjects did not show any significant difference in dynamic movement of the viscera. Subgroup analysis did not demonstrate any correlation between the degree of prolapse, clinical symptoms, and hypermobility clinical (Beighton) scores. The interobserver reliability for 3 out the 4 anatomical landmarks showed ‘moderate’ or ‘good’ correlation based on their interclass correlation coefficients. DISCUSSION: hEDS patients do not appear to have a significantly increased incidence of visceroptosis.

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16

S.M., Khusanov, Botirov A.K., Mamajonova D.M., and Botirov J.A. "SURGICAL TREATMENT RESULTS OF VISCEROPTOSIS." April 18, 2025. https://doi.org/10.5281/zenodo.15243196.

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<em>The authors report that manifestations of chronic dysfunctions of the colon in visceroptosis attract the attention of surgeons only at the stage of sub- and decompensated colostasis. However, prolonged conservative treatment and allowing the disease to progress to the sub- and decompensation stage is unjustified, as it often leads to a high recurrence rate—ranging from 17.6% to 45.9%. The authors conclude that complications in the early postoperative period were caused by certain shortcomings in preoperative preparation, determining the volume of the large intestine to be resected, the method of intestinal anastomosis formation, and postoperative management. This has necessitated adjustments in the surgical strategy for patients with visceroptosis combined with chronic colostasis.</em>

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17

Ljaljukov, A., E. Loginova, G. Nechaeva, et al. "Abdominal hemodynamics in patients with visceroptosis." European Heart Journal 42, Supplement_1 (2021). http://dx.doi.org/10.1093/eurheartj/ehab724.2724.

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Abstract Background Abdominal circulation plays the important physiological role for structure and function of the digestive system, maintenance of nutrient homeostasis. Purpose To study the features of abdominal hemodynamics in patients with visceroptosis. Methods We studied 69 patients (mean age 22.28±3.7 years) with splanchnoptosis (visceroptosis) and 52 age- and sex- matched patients without splanchnoptosis (controls). Exclusion criteria: a history of digestive system surgery of taking blood circulation activating drugs. Doppler ultrasonography of the common hepatic artery, splenic artery, superior mesenteric artery, portal vein was performed on an empty stomach and 30 minutes after a food sample (standardized for proteins (14 g), fats (10 g) and carbohydrates (45 g)) using the Sonoace-8000 ultrasound scanner (Medison, South Korea). The data were analyzed using the Statistica-6 packages. Results There were no differences in hemodynamic parameters of fasting abdominal blood flow (ABF). After a food testing, in the postprandial period the ABF in all vessels in patients with splanchnoptosis was lower than in controls: the portal vein blood flow (BF) 1124,0 [1030,0–1419,0] ml/min vs 1373,0 [1136,0–1567,5] ml/min respectively (U=433,5; Z=−2,1; p=0,0342); the common hepatic artery BF 341,0 [295,0–394,0] ml/min vs 412,0 [331,0–521,0] ml/min respectively (U=335,0; Z=−2,3; p=0,0218); the splenic artery BF 396,0 [292,0–538,0] ml/min vs 502,0 [394,0–594,0] ml/min respectively (U=328,0; Z=−2,1; p=0,0399); the superior mesenteric artery BF 988,0 [837,0–1272,0] ml/min vs 1136,5 [992,0–1465,0] ml/min respectively (U=1625,5; Z=−2,2; p=0,0314). Changes in ABF were correlated with splanchnoptosis: the portal vein BF with any ptosis (rs=−0,21; p&lt;0.05), the common hepatic artery BF with gastroptosis (rs=−0,38; p&lt;0.05), the superior mesenteric artery BF with colonoptosis (rs=−0,86; p&lt;0.05). The peripheral vascular resistance was correlated with the common hepatic artery BF (rs=−0,46; p&lt;0.05), with the splenic artery BF (rs=−0,33; p&lt;0.05) and with the superior mesenteric artery BF (rs=−0.79; p&lt;0.05). Conclusions Patients with splanchnoptosis in the postprandial period have low volumetric BF in abdominal aorta vessels. This can be associated with the length of the mesenteric vessels, which undergoes the greatest changes in splanchnoptosis. Food testing reveals latent BF deficit in patients with splanchnoptosis. Funding Acknowledgement Type of funding sources: None.

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18

Kucera, Stephen, and Stephen Sullivan. "Visceroptosis and the Ehlers-Danlos Syndrome." Cureus, November 8, 2017. http://dx.doi.org/10.7759/cureus.1828.

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19

Chikako, Shimizu. "Changes in the bulge of the rump in the Chamber and IVC groups." March 12, 2020. https://doi.org/10.5281/zenodo.3707215.

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20

Olusola, Siji, Tobias Jarman, Chetan Parmar, and Manikandan Kathirvel. "HPB SO14 - Surprise Inside: Gallbladder Herniation in a Parastomal Hernia - A Rare Case Report and Management Insight." British Journal of Surgery 111, Supplement_9 (2024). http://dx.doi.org/10.1093/bjs/znae271.247.

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Abstract Background Parastomal hernias are a common complication of stoma formation, affecting approximately 40% of patients within two years. Gallbladder herniation within a parastomal hernia is a rare phenomenon, with only 18 cases previously reported. Patients are typically elderly females, with the condition attributed to ageing factors such as loss of visceral fat and elastic tissue, liver shrinkage, and the increased length and loss of elasticity of the gallbladder mesentery. These factors contribute to visceroptosis of the gallbladder, increasing its mobility and the risk of herniation. Method We present the case of a 68-year-old female with a four-day history of severe parastomal hernia pain and swelling. She had an emergency left hemicolectomy and transverse colostomy in the right upper quadrant 10 years prior for ischaemic colitis and perforation. Her stoma was functioning well. Vital signs and biochemistry results were normal. On examination her hernia was severely tender but partially reducible. CT revealed a right upper abdominal parastomal hernia containing loops of colon and surprisingly the gallbladder with a long stretched cystic duct. There was no evidence of cholecystitis, bowel obstruction, or compromise within the hernial sac. Results With analgesia, the patient's pain improved, though her hernia remained irreducible. She was discharged after two days of conservative management and referred back to the hospital where she had the initial operation. On enquiry with that hospital it was reported that an elective parastomal hernia repair with completion right colectomy and end ileostomy was performed. The gallbladder was mobilised and reduced back into the peritoneal cavity, and the hernial defect was closed. The patient made an excellent recovery and was discharged home three days after surgery. Conclusion Cholecystic parastomal herniation is a very rare phenomenon and should be considered as a differential in similar cases. Management should be based on clinical presentation, radiological diagnosis, and a multidisciplinary team approach. No standardised classification or approach exists. We propose classifying gallbladder herniations as either being simple (without inflammatory sequelae) or complicated (with cholecystitis, gallbladder torsion, incarceration or perforation). Simple herniations, such as the case presented, can be managed electively with intra-operative reduction of the gallbladder sufficing. Complicated herniations during acute admissions indicate the need for emergency surgical intervention with combined cholecystectomy and parastomal hernia repair.

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21

Potekaev, Nikolai N., Olga B. Borzykh, Elena I. Karpova, et al. "Connective Tissue Dysplasia As A Predictor Of Premature Skin Aging." Russian Open Medical Journal 12, no. 4 (2023). http://dx.doi.org/10.15275/rusomj.2023.0409.

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Background — One of the manifestations of connective tissue dysplasia (CTD) is premature skin aging. The latter can have different etiological factors. The objective of our study was to investigate the effect of predictors of premature skin aging, especially CTD, on the severity of involutional changes in the skin, as well as to develop the approach for the management of patients with premature aging. Material and Methods — We included in our study 78 women 35-45 years of age with no substantial anti-aging treatment procedures in their anamneses. We considered genealogical and cosmetological anamneses, and life history (including somatic pathology), physical examination; determined prevailing type of facial skin aging; identified predictors of CTD, assessed psychoemotional state of the patient, and performed sonography of the skin and biochemical examination of patient serum. At a clinical stage, we conducted a randomized comparative study of biorevitalizant efficacy in patients with normal and premature patterns of skin aging. Results — Our study established the role of CTD in assessing the risk of premature skin aging. In addition to the presence of CTD, the importance of identifying isolated phenotypic manifestations of CTD (such as arachnodactyly, hypermobility syndrome, low relative weight of the patient, and skin manifestations of CTD) was shown. Other predictors were also analyzed; their identification can help assessing the risk of premature skin aging. Among them, somatic pathology (varicose veins, herniated discs, visceroptosis, autonomic vascular dystonia) and physical examination data (such as pallor of the facial skin, swelling, skin hyperelasticity) were noted. When assessing the clinical efficacy of biorevitalization, the greatest satisfaction of patients with premature skin aging regarding its results was characteristic for the group of patients after 7% collagen treatment. The clinical efficacy according to sonography and histological examination in patients with premature skin aging was significantly higher after the use of collagen or a complex hyaluronic acid (HA) preparation, compared with native HA. Patients with premature skin aging exhibited high level of anxiety and/or depression, which in turn increased the risk of underestimating the outcome of procedures. Conclusion — The presented study confirmed the role of CTD in assessing the risk of premature skin aging. Predictors were revealed (somatic pathology, type of facial aging, physical examination data of the patient, etc.) that could be used to assess the risk of premature skin aging. In patients with premature aging, preparation with native HA (as a biorevitalizant) had a low clinical efficacy comparable to using placebo. A collagen-based preparation and a complex HA-based preparation exhibited high clinical efficacy. Also, patients with premature aging of the skin had a higher score of anxiety and depression, while patients with high levels of anxiety and depression were more likely to underestimate the satisfaction with the results of their aesthetic treatment.

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