Academic literature on the topic 'Visual pathway'

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Journal articles on the topic "Visual pathway"

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Kosmorsky, G. S. "Albino visual pathway." Neurology 47, no. 1 (1996): 311. http://dx.doi.org/10.1212/wnl.47.1.311.

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Mesiwala, Ali H., and John D. Loeser. "Visual Pathway Glioma." Pediatric Neurosurgery 36, no. 3 (2002): 161. http://dx.doi.org/10.1159/000048373.

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Koppens, Joanna. "Visual Fields via the Visual Pathway." Clinical & Experimental Ophthalmology 35, no. 3 (2007): 284. http://dx.doi.org/10.1111/j.1442-9071.2007.01460.x.

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DiMartino, Robert. "Visual Fields via the Visual Pathway." Optometry and Vision Science 84, no. 7 (2007): 551. http://dx.doi.org/10.1097/opx.0b013e3180de4f74.

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Rowe, Fiona, and Thomas Meigen. "Visual Fields via the Visual Pathway." Current Eye Research 32, no. 7-8 (2007): 729–30. http://dx.doi.org/10.1080/02713680701471057.

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Spry, P. G. D. "Visual Fields via the Visual Pathway." British Journal of Ophthalmology 91, no. 6 (2007): 845. http://dx.doi.org/10.1136/bjo.2006.107987.

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Thompson, Charlotte R., and Simmons Lessell. "Anterior Visual Pathway Gliomas." International Ophthalmology Clinics 37, no. 4 (1997): 261–79. http://dx.doi.org/10.1097/00004397-199703740-00021.

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Park, Soo-Hyun, and Nam-Hee Kim. "Visual Evoked Potentials for Detecting Visual Pathway Abnormality." Korean Journal of Neuromuscular Disorders 13, no. 2 (2021): 21–27. http://dx.doi.org/10.46518/kjnmd.2021.13.2.21.

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Visual evoked potentials (VEPs) are especially useful for evaluating patients with visual pathway involvement but no objective findings on ophthalmic examination. To apply VEPs appropriately in clinical practice, clinicians should be well aware of the standard test techniques and various factors affecting the interpretation of VEPs to detect visual pathway abnormalities. This article summarizes the method for recording VEPs and the technical and physiologic factors associated with VEPs.
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Hines, Pamela J. "Dating the ipsilateral visual pathway." Science 372, no. 6538 (2021): 141.4–141. http://dx.doi.org/10.1126/science.372.6538.141-d.

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Shuper, Avinoam, Liora Kornreich, Shalom Michowitz, Michael Schwartz, Isaac Yaniv, and Ian J. Cohen. "VISUAL PATHWAY TUMORS AND HYDROCEPHALUS." Pediatric Hematology and Oncology 17, no. 6 (2000): 463–68. http://dx.doi.org/10.1080/08880010050120818.

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Dissertations / Theses on the topic "Visual pathway"

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Ardila, Jimenez Silvia. "Analysis of visual responses in the mouse early visual pathway." Thesis, Imperial College London, 2016. http://hdl.handle.net/10044/1/43376.

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Many animals, including humans, rely on visual input to guide their behaviour and interact with their environment. The study of the visual system is prevalent in neuroscience, however, given the highly complex nature of the brain, we are yet to understand the full functionality of the system. In this study, we set out to explore different aspects of visual processing in the mouse early visual pathway, and how they compare to those in other mammals. In this thesis, we study two major brain structures in the early visual pathway, the dorsal lateral geniculate nucleus (dLGN) in the thalamus, and the primary visual area (V1 or area 17) in the cortex. We aim to explore 3 different aspects of visual information processing in these areas. Firstly, the functional response characteristics of single neurons in the visual thalamus. Secondly, whether additional communication channels are used in thalamo-cortical interactions in the mouse. And lastly, the correlation of behaviour within neuronal population activity in visual cortex. We use data from two different experimental paradigms. One involves an anaesthetised preparation, recording extracellular potentials from the visual thalamus in isolation, or from the visual thalamus and the primary visual cortex simultaneously. The second involves an awake preparation in which animals were trained on a 'Go'/'NoGo' discrimination task and extracellular potentials were recorded from the primary visual cortex during behaviour. This project combines time-series analysis and information theoretical methods to analyse high dimensional multi-electrode array recordings. In addition to the analysis of experimental data, we also explore the practical and methodological implications of measuring communication through cross-frequency coupling and propose an alternative method to measure this phenomenon.
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Ather, Sarim. "Imaging the visual pathway in human albinism." Thesis, University of Leicester, 2016. http://hdl.handle.net/2381/37816.

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Albinism refers to a group of genetic abnormalities that are associated with profound defects throughout the visual pathway. These include foveal hypoplasia, optic nerve anomalies, chiasmal misrouting, visual cortex reorganisation, and nystagmus. This study utilises optical coherence tomography and magnetic resonance imaging to assess the visual pathway in a large cohort of patients. We find that in albinism, there is maldevelopment of the fovea, with a continuation of the inner retinal layers and a failure of the photoreceptor layers to specialize. The latter abnormality is the biggest contributor to reduced visual acuity seen in albinism. The optic nerve head is characterized by presence of excess glial tissue within the optic rim indicating incomplete maturation. In addition, there is reduced peripapillary retinal never fibre layer thickness consistent with previous histology reports of reduced ganglion cell numbers in albinism. We demonstrate the ability of diffusion tractography to quantify abnormal chiasmal decussation for the first time. Moreover, we find that cortical abnormalities are related to melanin levels within the retinal pigment epithelium and axonal disorganisation. Our results show that nystagmus severity is related to the degree of foveal maldevelopment. This finding adds credence to the increasing recognition of the importance of sensory abnormalities in generating nystagmus. In conclusion, we find that in albinism, normal development of the visual pathway appears to have halted prior to reaching completion. Patients with albinism show a spectrum of anomalies ranging from resembling normality to being grossly atypical. This spectrum closely resembles stages in normal visual development. Our findings represent a step forward in the scientific understanding of visual deficits associated with albinism and are likely to aid clinicians in the management of affected patients.
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Henderson, A. P. D. "Imaging the anterior visual pathway in neurological disease." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1386992/.

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Axonal loss is an important cause of irreversible disability in multiple sclerosis. Whilst recovery of neurological function due to restoration of neuronal conduction is usual after multiple sclerosis relapse, some relapses are followed by fixed disability. Quantitative measures of axons in the retina, and in their continuation in the optic nerve allow measurement of the effect upon axons of a prototypical multiple sclerosis relapse, optic neuritis. Using optical coherence tomography, axonal and neuronal loss were observable in the retina of patients with progressive multiple sclerosis, even when they were clinically unaf- fected by optic neuritis. With follow-up, these changes were found to be stable. After optic neuritis, 99% of retinal nerve fibre layer thinning was detectable by 4.75 months, and 99% of macular volume loss was detectable by 11 months. The earliest time to detect retinal nerve fibre layer thinning was 1.64 months, and the earliest time to detect macular volume loss was 0.99 months. Sample sizes for clinical trials generated for trials of neuro-protective agents, using multiple sclerosis thickness as an endpoint, showed that 35 subjects per arm of a randomised trial would be needed to detect an 50% effect, with 80% power. Early measures of colour vision and visual evoked potential delay were strongly related to the eventual degree of axonal loss. Comparisons of two methods of measuring multiple sclerosis thinning after optic neuritis suggested that optical coherence tomography would be superior to scanning laser polarimetry. Comparisons of optic nerve mean area and multiple sclerosis thickness showed that whilst initial swelling was similar, the degree of subsequent atrophy was less in the optic nerve. Measures of axonal loss in the retina will be an important method for assessing the efficacy of therapies that propose to prevent axonal loss in multiple sclerosis and optic neuritis.
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Wang, Jiaqiong. "DIFFUSE TRAUMATIC AXONAL INJURY WITHIN THE VISUAL SYSTEM: IMPLICATIONS FOR VISUAL PATHWAY REORGANIZATION." VCU Scholars Compass, 2012. http://scholarscompass.vcu.edu/etd/2911.

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Traumatic brain injury is a major health problem with much of its morbidity associated with traumatic axonal injury (TAI). To date, significant insight has been gained into the initiating pathogenesis of TAI. However, the specific anterograde and retrograde sequelae of TAI are poorly understood because the diffuse nature of TAI complicates data analysis. To overcome this limitation, we subjected transgenic mice expressing yellow fluorescent protein (YFP) within the visual system to central fluid percussion injury, and consistently generated diffuse TAI within the optic nerve that could easily be followed in the organized YFP positive fibers. We demonstrated progressive axonal swelling, disconnection and proximal and distal axonal dieback, with regression and reorganization of the proximal swellings, and the persistence of the distal disconnected and degenerating swellings. Antibodies targeting the C-terminus of amyloid precursor protein, a marker of TAI, mapped to the proximal axonal segments without distal targeting. Antibodies targeting microglia/macrophages, revealed activated microglia/ macrophages closely encompassing the distal disconnected, degenerating axonal segments at 7 - 28 days post injury, suggesting their role in the delayed axonal degeneration. In contrast, in the proximal reorganizing axonal segments, microglia/macrophages appeared less reactive with their processes paralleling preserved axonal profiles. Concomitant with these events, YFP fluorescence quenching also occurred, complicating data analysis. This quenching mapped to Texas-Red-conjugated-IgG immunoreactive loci, suggesting that blood–brain barrier disruption and its attendant edema participated in fluorescence quenching. This was confirmed through antibodies targeting endogenous YFP, which identified the retention of intact axons despite YFP fluorescent loss. Paralleling these events, TAI was not accompanied by retrograde retinal ganglion cell (RGC) death. Specifically, no TUNEL+ or cleaved caspase-3 immunoreactive RGCs were observed from 2 days to 3 months post-TBI. Further, Brn3a immunoreactive RGC quantification revealed no significant RGC loss. This RGC preservation was accompanied by the persistent phospho-c-Jun expression for up to 3 months post-TBI, a finding linked to neuronal survival and potential axonal repair. Parallel ultrastructural study again failed to identify RGC death. Collectively, this study provides unprecedented insight into the evolving pathobiology associated with TAI, and offers advantages for future studies focusing on its therapeutic management and neuronal reorganization.
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Fritsch, Dennis Manuel. "Visual impairment in the absence of ON-pathway signal." Thesis, University College London (University of London), 2018. http://discovery.ucl.ac.uk/10048856/.

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Congenital retinal diseases are a major cause of childhood and lifelong visual im- pairment. Such conditions can manifest a variable array of severe and subtle ef- fects on vision. Assessment of visual function in children can be challenging; yet, knowledge about phenotype, genotype and impact of these disorders is crucial for providing appropriate support, tailored diagnostics and for developing treatments. ON-and OFF-pathways are separately transmitting information on brightness and darkness from the retina to the cortex, where their interplay is crucial in visual perception. This project investigated the effects of retinal ON-pathway dysfunction on vision. A cohort of 109 patients with ON-pathway dysfunction was examined from four subgroups of visual electrophysiological phenotypes (incomplete and complete Congenital Stationary Night Blindness - CSNB, Duchenne Muscular Dystrophy - DMD, and congenital disorders of N-glycosylation - PMM2-CDG). Using spe- cialised visual evoked potential stimuli, designed to distinguish the ON-and OFF- pathway signal arrival at the striate cortex, marked ON system delays were revealed in patients with subtypes of CSNB, DMD mutations post exon 30 and PMM2-CDG. A child-friendly psychophysical software called LumiTrack was developed to assess motion and contrast perception, two important qualities conveyed by ON-and OFF-pathways. Patients with subtypes of CSNB and PMM2-CDG showed abnormalities in motion perception and subnormal contrast sensitivity, while patients with DMD performed at the level of healthy volunteers. These impairments may occur due to a delay of signal transmission through the retina, resulting in an ON/OFF signal asymmetry within the visual system. A genotype-phenotype comparison suggested a trend of increasing ON/OFF asymmetry associated with genetic defects affecting proteins placed later within the photoreceptor / ON bipolar cell signalling cascade. This systematic study of cortical and behavioural visual function in patients with ON-pathway dysfunction highlights the impairments encountered by patients in visual qualities important for everyday life.
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Eguchi, Akihiro. "Neural network modelling of the primate ventral visual pathway." Thesis, University of Oxford, 2017. https://ora.ox.ac.uk/objects/uuid:99277b9c-00ee-45e3-8adb-47190d716912.

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The aim of this doctoral research is to advance understanding of how the primate brain learns to process the detailed spatial form of natural visual scenes. Neurons in successive stages of the primate ventral visual pathway encode the spatial structure of visual objects and faces. However, it remains a difficult challenge to understand exactly how these neurons develop their response properties through visually guided learning. This thesis approaches this problem through the use of computational modelling. In particular, I first show how the brain may learn to represent the spatial structure of objects and faces through a series of processing stages along the ventral visual pathway. Then I propose how understanding the two complementary unsupervised learning mechanisms of translation invariance may have useful applications in clinical psychology. Next, the potential functional role of top-down (feedback) propagation of visual information in the brain in driving the development of border ownership cells, which are thought to play a role in binding visual features such as boundary edges to their respective objects, is investigated. In particular, the limitations of traditional rate-coded neural networks in modelling these cells are identified. Finally, a general solution to such binding problems with the use of a more biologically realistic spiking neural network is presented. This work is set to make an important contribution towards understanding how the visual system learns to encode the detailed spatial structure of objects and faces within scenes, including representing the binding relations between the visual features that comprise those objects and faces.
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Simons, Emily Sue. "Impact of amyloid-beta on the primary visual pathway." Kent State University / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=kent1626687149999739.

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Colello, Raymond J. "The development of the retinofugal pathway in rodents." Thesis, University of Oxford, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.253313.

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Edgar, Graham K. "Temporal processing in the normal and demyelinated human visual pathway." Thesis, Keele University, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.375925.

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Khawaja, Farhan. "Hierarchical transformations of sensory inputs along the primate dorsal visual pathway." Thesis, McGill University, 2014. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=121159.

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The dorsal pathway of macaque visual cortex is involved in the processing of motion signals that are useful for perception and behaviour. This involves multiple stages, with each stage integrating information from its predecessors. Along these stages, motion information is first measured by the primary visual cortex (V1), which sends specialized projections to extrastriate regions such as the middle temporal area (MT). Previous work with plaid stimuli has shown that most V1 neurons respond to the individual components of moving stimuli, while some MT neurons are capable of estimating the global motion of the pattern. The observation of this pattern-selectivity has led to two-stage models, in which MT neurons integrate the outputs of component-selective V1 neurons. In this thesis we first show (Chapter 2) thatthe majority of neurons in the medial temporal area (MST), which receives input from MT, have this pattern-selective property. We also show that increased pattern selectivity in MST is associated with greater prevalence of the mechanisms (i.e. motion opponency and component integration) implemented by two-stage MT models. Thus the relationship between MST and MT is qualitatively similar to that between MT and V1, as repeated application of basic motion mechanisms leads to novel selectivities at each stage along the pathway. The second and third chapters of this thesis use dissociation of spikes and local field potentials (LFPs) through stimulus selectivity and inactivation of spiking output, respectively, to investigate the anatomical source of the LFP. These experiments suggest that the β and γLband of the LFP exhibit properties similar to the presumptive feedforward input to each area while the γH band reflects local spiking output. Together theresults of this thesis suggest a strong link between the sensory tuning of cortical LFPs and afferent inputs, with important implications for the interpretation of the BOLD signal, which has been shown to correlate with the spike-independent component of the LFP. The thesis also has important implications for the design of Brain-Machine Interfaces, which use the LFP response to decode brain function.<br>Chez le macaque, la voie dorsale du cortex visuel est impliquée dans le traitement des signaux relatifs au mouvement qui sont utiles à la fois pour la perception et le comportement. Ce processus nécessite plusieurs étapes d'intégrations de l'information avec chacune des étapes intégrant l'information acheminée lors des étapes précédentes. Le long de ces étapes, l'information relative au mouvement est d'abord traitée par le cortex visuel primaire (V1) qui envoie des projections spécialisées aux régions extra-striées comme la région moyenne temporale (ou MT). En utilisant un stimulus visuel complexe de type 'plaid', des travaux antérieurs ont montré que la plupart des neurones V1 répondent aux composantes individuelles du stimulus en mouvement, alors que certains neurones du MT sont plutôt capables de traiter l'information concernant le mouvement et/ou le pattern du stimulus dans sa globalité. L'observation de ce type de modèle sélectif (ou modèle-sélectivité) à conduit au modèle à deux étapes au cours duquel les neurones du MT intègrent les informations sortantes sélectives des neurones V1. Dans cette thèse nous montrons tout d'abord (chapitre 2) que la majorité des neurones dans la région médiane temporale (ou MST), qui reçoivent des projections du MT, ont cette propriété de modèle-sélectivité. Nous avons montré également que l'augmentation du modèle-sélectivité dans le MST est associée à la prévalence des mécanismes implémentés par le modèle MT à deux étapes (c'est-à-dire mouvement dans des sens opposés et composante du processus d'intégration). Ainsi, la relation entre le MST et le MT est qualitativement semblable à celle entre le MT et le V1, étant donné qu'une application répétée des mécanismes de base du mouvement conduit à des sélectivités nouvelles à chaque étape le 10 long de cette voie. Dans le deuxième et le troisième chapitre de cette thèse nous avons utilisé les techniques de dissociation des potentiels d'actions ainsi que les potentiels de champs locaux (LFPs) en ayant recours, respectivement, à la stimulation sélective et l'inactivation de décharge neuronale à la sortie afin de déterminer l'origine anatomique du LFP. Nos résultats suggèrent que la bande β et γL de la LFP présente des propriétés semblables à celles des projections entrantes capables de rétroaction anticipative tandis que la bande γH reflète le pattern de décharge des projections sortantes. Ensemble, les résultats de cette thèse suggèrent un lien important entre l'affinement des entrées sensorielles des LFPs corticaux et les autres afférences, avec des implications importantes pour l'interprétation du signal "FORT", qui semble correler avec la composante indépendante du pic du LFP. La thèse a également des implications importantes dans la conception des Interfaces Cerveau- Machine, qui utilisent la réponse du LFP pour décoder le fonctionnement du cerveau.
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Books on the topic "Visual pathway"

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Rowe, Fiona, ed. Visual Fields via the Visual Pathway. Blackwell Publishing Ltd, 2006. http://dx.doi.org/10.1002/9780470759271.

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Rowe, Fiona J. Visual fields via the visual pathway. Blackwell Pub., 2006.

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Baert, A. L., K. Sartor, and Wibke S. Müller-Forell, eds. Imaging of Orbital and Visual Pathway Pathology. Springer-Verlag, 2006. http://dx.doi.org/10.1007/3-540-27989-x.

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Jang, Jaeson, and Se-Bum Paik. Emergence of Functional Circuits in the Early Visual Pathway. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-0031-0.

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Samuel, Sokol, ed. Electrophysiologic testing in disorders of the retina, optic nerve, and visual pathway. American Academy of Ophthalmology, 1990.

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A pathway to reality: Visual and aural concepts in Dorothy Richardson's "Pilgrimage". Francke Verlag, 2000.

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1935-, Voth D., and Glees Paul, eds. The Orbit and the visual pathway: Anatomical and pathological aspects and detailed clinical accounts. W. de Gruyter, 1994.

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Software development pathway for the iPRO certificate for IT practitioners, with Visual Basic and Java. Heinemann, 2005.

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Tovée, Martin J. An introduction to the visual system. 2nd ed. Cambridge University Press, 2008.

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Tovée, M. J. An introduction to the visual system. Cambridge University Press, 1996.

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Book chapters on the topic "Visual pathway"

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Righi, Giulia, and Jean Vettel. "Dorsal Visual Pathway." In Encyclopedia of Clinical Neuropsychology. Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_1358.

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Righi, Giulia, and Jean Vettel. "Ventral Visual Pathway." In Encyclopedia of Clinical Neuropsychology. Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_1409.

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Righi, Giulia, and Jean Vettel. "Dorsal Visual Pathway." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_1358-2.

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Righi, Giulia, and Jean Vettel. "Ventral Visual Pathway." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_1409-2.

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Righi, Giulia, and Jean Vettel. "Dorsal Visual Pathway." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_1358.

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Righi, Giulia, and Jean Vettel. "Ventral Visual Pathway." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_1409.

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Snell, Richard S., and Michael A. Lemp. "The Visual Pathway." In Clinical Anatomy of the Eye. Blackwell Science Ltd,., 2013. http://dx.doi.org/10.1002/9781118690987.ch13.

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Packer, Roger J., Peter J. Savino, Gary R. Diamond, and Leslie N. Sutton. "Visual Pathway Gliomas." In Management of Childhood Brain Tumors. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4613-1501-8_9.

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Joukal, Marek. "Anatomy of the Human Visual Pathway." In Homonymous Visual Field Defects. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-52284-5_1.

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Marsalek, Petr, Marek Hajný, and Martin Vokurka. "Pathological Physiology of the Visual Pathway." In Homonymous Visual Field Defects. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-52284-5_2.

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Conference papers on the topic "Visual pathway"

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Verostko, Roman. "Pathway series." In ACM SIGGRAPH 97 Visual Proceedings: The art and interdisciplinary programs of SIGGRAPH '97. ACM Press, 1997. http://dx.doi.org/10.1145/259081.259161.

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Verostko, Roman. "Carnival, pathway series." In ACM SIGGRAPH 97 Visual Proceedings: The art and interdisciplinary programs of SIGGRAPH '97. ACM Press, 1997. http://dx.doi.org/10.1145/259081.259160.

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Zheng, Lingxiang, Xianchao Zheng, Zhanjian Lin, Weiwei Tang, and Changle Zhou. "A dorsal pathway guided visual attention model." In 2013 International Joint Conference on Awareness Science and Technology & Ubi-Media Computing (iCAST-UMEDIA). IEEE, 2013. http://dx.doi.org/10.1109/icawst.2013.6765517.

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Wang, Chong, Junge Zhang, PeiPei Yang, and Kaiqi Huang. "Robust Object Recognition via Visual Pathway Feedback." In 2014 22nd International Conference on Pattern Recognition (ICPR). IEEE, 2014. http://dx.doi.org/10.1109/icpr.2014.114.

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Barraza, Jose F., and Andrew M. Derrington. "About the delay in the visual pathway." In ICO XVIII 18th Congress of the International Commission for Optics, edited by Alexander J. Glass, Joseph W. Goodman, Milton Chang, Arthur H. Guenther, and Toshimitsu Asakura. SPIE, 1999. http://dx.doi.org/10.1117/12.354854.

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MOYA, KENNETH L., ALVIN W. LYCKMAN, and ANNAMARIA CONFALONI. "MOLECULAR CHANGES DURING PRIMARY VISUAL PATHWAY DEVELOPMENT." In Proceedings of the International School of Biophysics. WORLD SCIENTIFIC, 2001. http://dx.doi.org/10.1142/9789812799975_0006.

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Lesica, N. A., and G. B. Stanley. "Signal detection of salient visual features by the early visual pathway." In 2005 IEEE Engineering in Medicine and Biology 27th Annual Conference. IEEE, 2005. http://dx.doi.org/10.1109/iembs.2005.1616436.

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Sasi, Swapna, Taher Yunus Lilywala, and Basabdatta Sen Bhattacharya. "Optimising hyperparameter search in a visual thalamocortical pathway model." In 2022 International Joint Conference on Neural Networks (IJCNN). IEEE, 2022. http://dx.doi.org/10.1109/ijcnn55064.2022.9892380.

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Meng, Fan, and Sumei Li. "No-Reference Stereoscopic Image Quality Assessment Based on The Visual Pathway of Human Visual System." In 2021 International Conference on Visual Communications and Image Processing (VCIP). IEEE, 2021. http://dx.doi.org/10.1109/vcip53242.2021.9675346.

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Sobreira, F., C. Tremmel, and D. J. Krusienski. "Modeling the Visual Pathway for Stimulus Optimization in Brain-Computer Interfaces." In 2018 26th European Signal Processing Conference (EUSIPCO). IEEE, 2018. http://dx.doi.org/10.23919/eusipco.2018.8553194.

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Reports on the topic "Visual pathway"

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Dale, Naomi, Aneesa Khan, and Sophie Dale. Early intervention for vision and neurodevelopment in infants and very young children with visual impairment: a systematicreview. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.8.0080.

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Review question / Objective: Research question - What is the effectiveness of Early Childhood Intervention (ECI) in the first 3 years of life? Population (P) Infants and very young children with diagnosed visual impairment. Intervention (I) ECI programmes that includes vision and developmental stimulation, play, learning and responsive parenting Comparison (C) Standard care or control Outcomes (O) Primary: Vision function or and/or neurodevelopment and/or parent-child interaction outcomes Secondary: Parental context factors eg parental wellbeing and mental health, parental satisfaction with service provision. Condition being studied: Childhood congenital or very early visual impairment arising from congenital disorders of the peripheral or anterior visual system or cerebral-based vision disorders. This includes all vision disorders of the globe, retina and anterior optic nerve and all vision disorders that are considered cerebral based along visual pathways that are retro-chiasmatic and include central brain regions and networks involved in vision processing.
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Harris, L. B., P. Adiban, and E. Gloaguen. The role of enigmatic deep crustal and upper mantle structures on Au and magmatic Ni-Cu-PGE-Cr mineralization in the Superior Province. Natural Resources Canada/CMSS/Information Management, 2021. http://dx.doi.org/10.4095/328984.

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Aeromagnetic and ground gravity data for the Canadian Superior Province, filtered to extract long wavelength components and converted to pseudo-gravity, highlight deep, N-S trending regional-scale, rectilinear faults and margins to discrete, competent mafic or felsic granulite blocks (i.e. at high angles to most regional mapped structures and sub-province boundaries) with little to no surface expression that are spatially associated with lode ('orogenic') Au and Ni-Cu-PGE-Cr occurrences. Statistical and machine learning analysis of the Red Lake-Stormy Lake region in the W Superior Province confirms visual inspection for a greater correlation between Au deposits and these deep N-S structures than with mapped surface to upper crustal, generally E-W trending, faults and shear zones. Porphyry Au, Ni, Mo and U-Th showings are also located above these deep transverse faults. Several well defined concentric circular to elliptical structures identified in the Oxford Stull and Island Lake domains along the S boundary of the N Superior proto-craton, intersected by N- to NNW striking extensional fractures and/or faults that transect the W Superior Province, again with little to no direct surface or upper crustal expression, are spatially associated with magmatic Ni-Cu-PGE-Cr and related mineralization and Au occurrences. The McFaulds Lake greenstone belt, aka. 'Ring of Fire', constitutes only a small, crescent-shaped belt within one of these concentric features above which 2736-2733 Ma mafic-ultramafic intrusions bodies were intruded. The Big Trout Lake igneous complex that hosts Cr-Pt-Pd-Rh mineralization west of the Ring of Fire lies within a smaller concentrically ringed feature at depth and, near the Ontario-Manitoba border, the Lingman Lake Au deposit, numerous Au occurrences and minor Ni showings, are similarly located on concentric structures. Preliminary magnetotelluric (MT) interpretations suggest that these concentric structures appear to also have an expression in the subcontinental lithospheric mantle (SCLM) and that lithospheric mantle resistivity features trend N-S as well as E-W. With diameters between ca. 90 km to 185 km, elliptical structures are similar in size and internal geometry to coronae on Venus which geomorphological, radar, and gravity interpretations suggest formed above mantle upwellings. Emplacement of mafic-ultramafic bodies hosting Ni-Cr-PGE mineralization along these ringlike structures at their intersection with coeval deep transverse, ca. N-S faults (viz. phi structures), along with their location along the margin to the N Superior proto-craton, are consistent with secondary mantle upwellings portrayed in numerical models of a mantle plume beneath a craton with a deep lithospheric keel within a regional N-S compressional regime. Early, regional ca. N-S faults in the W Superior were reactivated as dilatational antithetic (secondary Riedel/R') sinistral shears during dextral transpression and as extensional fractures and/or normal faults during N-S shortening. The Kapuskasing structural zone or uplift likely represents Proterozoic reactivation of a similar deep transverse structure. Preservation of discrete faults in the deep crust beneath zones of distributed Neoarchean dextral transcurrent to transpressional shear zones in the present-day upper crust suggests a 'millefeuille' lithospheric strength profile, with competent SCLM, mid- to deep, and upper crustal layers. Mechanically strong deep crustal felsic and mafic granulite layers are attributed to dehydration and melt extraction. Intra-crustal decoupling along a ductile décollement in the W Superior led to the preservation of early-formed deep structures that acted as conduits for magma transport into the overlying crust and focussed hydrothermal fluid flow during regional deformation. Increase in the thickness of semi-brittle layers in the lower crust during regional metamorphism would result in an increase in fracturing and faulting in the lower crust, facilitating hydrothermal and carbonic fluid flow in pathways linking SCLM to the upper crust, a factor explaining the late timing for most orogenic Au. Results provide an important new dataset for regional prospectively mapping, especially with machine learning, and exploration targeting for Au and Ni-Cr-Cu-PGE mineralization. Results also furnish evidence for parautochthonous development of the S Superior Province during plume-related rifting and cannot be explained by conventional subduction and arc-accretion models.
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