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Books on the topic 'Vitamin A deficiency in children. Vitamin A in the body. Children'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

P, West Keith, ed. Vitamin A deficiency: Health, survival, and vision. New York: Oxford University Press, 1996.

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2

Herbers, Mary Ruth. Vitamin A: An urgent nutritional need for the world's children. Washington: U.S. G.P.O., 1985.

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3

Herbers, Mary Ruth. Vitamin A: An urgent nutritional need for the world's children. Washington: U.S. G.P.O., 1985.

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4

Herbers, Mary Ruth. Vitamin A: An urgent nutritional need for the world's children. Washington: U.S. G.P.O., 1985.

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5

National Nutrition Monitoring Bureau (India). Prevalence of Vitamin A deficiency among preschool children in rural areas. Hyderabad: National Institute of Nutrition, Indian Council of Medical Research, 2006.

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6

East, Central, and Southern African Regional Workshop on Vitamin A Interventions and Child Survival (1990 Lusaka, Zambia). East, Central, and Southern African Regional Workshop on Vitamin A Interventions and Child Survival, Lusaka, Zambia, 21-24 June 1990: Proceedings. New York: Vitamin A Technical Assistance Program, Helen Keller International, 1990.

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7

Demeke, Teshome. Vitamin A status of pre-school children in Ethiopia: (an estimate of national prevalence). Addis Abeba [i.e. Ababa], Ethiopia: Ethiopian Nutrition Institute, 1985.

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8

1942-, Sommer Alfred, and World Health Organization, eds. Vitamin A deficiency and its consequences: A field guide to detection and control. 3rd ed. Geneva: World Health Organization, 1995.

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9

Amouzou, Kou'santa Sabiba. Evaluation des marqueurs nutritionnels et génétiques du statut en coenzymes B (cobalamines et folates) et de l'homocystéinemie plasmatique dans une population d'Afrique de l'Ouest (Benin-Togo). Lomé: [s.n., 2003.

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10

Palmer, Amanda, Nita Dalmiya, and United Nations Children's Fund. (UNICEF). Vitamin A Supplementation: A Decade of Progress. United Nations Publications, 2007.

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11

Getting to the roots: Mobilizing community volunteers to combat Vitamin A deficiency disorders in Nepal. Kathmandu: UNICEF Regional Office for South Asia, 2003.

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12

J, Haselow Nancy, Rosen David S. 1960-, Sloan Nancy L, and Helen Keller International. Vitamin A Technical Assistance Program., eds. How to use the HKI food frequency method to assess community risk of vitamin A deficiency. Campinas: Helen Keller International, 1993.

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13

Rosen, David S., and Nancy J. Haselow. How to Use the Hki Food Frequency Method to Assess Community Risk of Vitamin a Deficiency. Helen Keller Intl, 1997.

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14

O, Olum I., Mirza N. M, Muhudhia S. O, Kenya Paediatric Association, and UNICEF, eds. Vitamin A and child health: A review of the effects of deficiency & the benefits of supplementation. [Nairobi]: Kenya Paediatric Association with Support from UNICEF, 1990.

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15

Effectiveness of vitamin A supplementation in the control of young child morbidity and mortality in developing countries. Toronto, Ontario, Canada: Department of Nutritional Sciences, Faculty of Medicine, University of Toronto, 1993.

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16

P, West Keith, Helen Keller International, and Charles A. Dana Foundation, eds. Bellagio meeting on vitamin A deficiency & childhood mortality: Proceedings of "Public health significance of vitamin A deficiency andits control," Bellagio Study and Conference Center of the Rockefeller Foundation, February 3-7, 1992. Capanis [Brazil]: Helen Keller International, 1993.

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17

A, Underwood Barbara, Olson James A, and International Vitamin A Consultative Group., eds. A Brief guide to current methods of assessing Vitamin A status. Washington, DC: International Vitamin A Consultative Group (IVACG)Secretariat, Nutrition Foundation, 1993.

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18

Stuart, Jeffrey J., 1965- author, ed. Could it be B12?: What every parent needs to know about vitamin B12 deficiency. 2016.

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19

Cannell, John Jacob. Autism causes, prevention and treatment: Vitamin D deficiency and the explosive rise of autism spectrum disorder. 2015.

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20

R, Tatala S., Shirika la Chakula Bora Tanzania., and Chalmers tekniska hogskola. Dept. of Food Science., eds. Prevalence of iron deficiency anaemia in preschool children and the effect of vitamin C supplementation: A study carried out in Msoga Village, Coast Region, Tanzania, 1992/1993. Dar es Salaam, Tanzania: Tanzania Food and Nutrition Centre, 1993.

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21

Organisation de coordination pour la lutte contre les endémies en Afrique centrale., United States. Agency for International Development. Office of Nutrition., and International Science and Technology Institute. Vitamin A Field Support Project., eds. Vitamin A status of Bulu and Pygmy children in Southern Cameroon, January 21-30, 1993. Arlington, VA: Vitamin A Field Support Project (VITAL), International Science and Technology Institute, Inc., 1994.

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22

What's Wrong with My Child?: From Neurological and Developmental Disabilities to Autism...How to Protect Your Child from B12 Deficiency. Quill Driver Books, 2015.

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23

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Micronutrients and minerals. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0008.

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• Vitamin deficiency 58• Mineral deficiency 61• Trace element deficiency 62• Vitamin supplementation for infants and young children 64The term ‘micronutrients’ includes two main classes of nutrient substances required in the diet in very small amounts: the essential organic micronutrients (vitamins) and the essential inorganic micronutrients (trace elements). Vitamin and mineral deficiencies may complicate malnutrition arising from underlying disease or inadequate diet. Key features are given below. However, micronutrients have wide-ranging effects, far beyond the simple prevention of deficiency states....
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24

Jadon, Deepak R., Tehseen Ahmed, and Ashok K. Bhalla. Disorders of bone mineralization—osteomalacia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199642489.003.0146_update_001.

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Disorders of bone mineralization cause rickets in children and osteomalacia in adults. Both remain common in developing countries. Incidence in Western countries had declined since the fortification of foodstuffs, but appears to be increasing again. Calcium and inorganic phosphate are the key precursors for bone mineralization and growth. The commonest aetiology of osteomalacia is vitamin D deficiency, primarily due to low dietary intake and inadequate sun exposure. In the last decade gene mutations have been identified that are responsible for inherited rickets and osteomalacia, particularly those that result in phosphate deficiency, hypophosphatasia, and vitamin D receptor or metabolizing enzyme mutations. Additionally, the pathogenesis of tumour-induced osteomalacia is becoming better understood. Osteomalacia may present as bone pain and tenderness, muscle pain and weakness, and skeletal deformity or fracture. Key investigations include biochemical assessment and plain radiographs. Radioisotope bone scans and bone biopsy may be considered in selected cases. Differential diagnoses include osteoporosis, seronegative arthritides, and localized soft tissue disorders. Treatment, guided by the underlying aetiology, aims to reduce symptoms, fracture risk, bone deformity and sequelae. Vitamin D deficient patients require vitamin D and calcium replacement.
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25

Jadon, Deepak R., Tehseen Ahmed, and Ashok K. Bhalla. Disorders of bone mineralization—osteomalacia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0146.

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Abstract:
Disorders of bone mineralization cause rickets in children and osteomalacia in adults. Both remain common in developing countries. Incidence in Western countries had declined since the fortification of foodstuffs, but appears to be increasing again. Calcium and inorganic phosphate are the key precursors for bone mineralization and growth. The commonest aetiology of osteomalacia is vitamin D deficiency, primarily due to low dietary intake and inadequate sun exposure. In the last decade gene mutations have been identified that are responsible for inherited rickets and osteomalacia, particularly those that result in phosphate deficiency, hypophosphatasia, and vitamin D receptor or metabolizing enzyme mutations. Additionally, the pathogenesis of tumour-induced osteomalacia is becoming better understood. Osteomalacia may present as bone pain and tenderness, muscle pain and weakness, and skeletal deformity or fracture. Key investigations include biochemical assessment and plain radiographs. Radioisotope bone scans and bone biopsy may be considered in selected cases. Differential diagnoses include osteoporosis, seronegative arthritides, and localized soft tissue disorders. Treatment, guided by the underlying aetiology, aims to reduce symptoms, fracture risk, bone deformity and sequelae. Vitamin D deficient patients require vitamin D and calcium replacement.
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26

Wright, Charlotte M. Promoting healthy nutrition. Edited by Alan Emond. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198788850.003.0011.

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Key nutritional issues affecting preschool children discussed in this chapter are breastfeeding, complementary feeding, and promoting a healthy diet. The evidence on iron deficiency and vitamin D deficiency is reviewed. Recommendations are made for commissioning of services to support breastfeeding, and evidence-based advice given for practitioners.
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27

Pitt, Matthew. Pathophysiological correlations in neuropathies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198754596.003.0004.

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This chapter begins with an explanation of the pathophysiological correlations between the recorded changes and the underlying diagnosis which allow classification into demyelinating and axonal neuropathy. Demyelinating neuropathies are discussed first. The extensive and ever expanding literature in hereditary neuropathies is highlighted. The different variants of the acute inflammatory demyelinating polyneuropathy encountered in children are discussed along with the electrodiagnostic criteria for the diagnosis. Chronic inflammatory demyelinating polyneuropathy is then covered, both in its clinical presentation and electrodiagnosis. Other causes such as MNGIE and Lyme disease are highlighted. In the section on axonal neuropathy, division into hereditary and acquired is made. The diagnosis of sensorimotor hereditary neuropathies is discussed along with primarily sensory neuropathies including ataxia telangiectasia, Friedreich’s ataxia, and abetalipoproteinaemia, finishing with discussion of the hereditary sensory and autonomic neuropathies. The many different causes of acquired axonal neuropathy are listed and discussed including neoplasia, endocrine disturbances, metabolic conditions, infective agents, autoimmune conditions, mitochondrial disease, drugs, and vitamin deficiency, finishing with critical illness neuromyopathy.
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