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Journal articles on the topic 'Vlgr1'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 February 2022

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1

Knapp, Barbara, Deva Krupakar Kusuluri, Nicola Horn, Karsten Boldt, Marius Ueffing, and Uwe Wolfrum. "Identification of Protein Complexes Associated with the Usher Syndrome 2C and Epilepsy-Associated Protein VLGR1 Applying Affinity Proteomics." Genomics and Computational Biology 4, no. 1 (2017): 100051. http://dx.doi.org/10.18547/gcb.2018.vol4.iss1.e100051.

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Authors aimed to identify novel VLGR1-associated protein networks to shed light on its integration into signaling pathways and the cellular compartments in which VLGR1 functions using high-resolution affinity proteomics based on tandem affinity purifications (TAPs).

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2

Yagi, Hideshi, Yoshihiro Takamura, Takunari Yoneda, et al. "Vlgr1 knockout mice show audiogenic seizure susceptibility." Journal of Neurochemistry 92, no. 1 (2005): 191–202. http://dx.doi.org/10.1111/j.1471-4159.2004.02875.x.

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3

Yagi, Hideshi, Hisashi Tokano, Mitsuyo Maeda, et al. "Vlgr1 is required for proper stereocilia maturation of cochlear hair cells." Genes to Cells 12, no. 2 (2007): 235–50. http://dx.doi.org/10.1111/j.1365-2443.2007.01046.x.

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4

Sun, Jin-Peng, Rong Li, Hong-Ze Ren, An-Ting Xu, Xiao Yu, and Zhi-Gang Xu. "The Very Large G Protein Coupled Receptor (Vlgr1) in Hair Cells." Journal of Molecular Neuroscience 50, no. 1 (2012): 204–14. http://dx.doi.org/10.1007/s12031-012-9911-5.

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5

Zallocchi, M., D. Delimont, D. T. Meehan, and D. Cosgrove. "Regulated Vesicular Trafficking of Specific PCDH15 and VLGR1 Variants in Auditory Hair Cells." Journal of Neuroscience 32, no. 40 (2012): 13841–59. http://dx.doi.org/10.1523/jneurosci.1242-12.2012.

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6

Gibert, Yann, D. Randy McMillan, Kathleen Kayes-Wandover, Axel Meyer, Gerrit Begemann, and Perrin C. White. "Analysis of the very large G-protein coupled receptor gene (Vlgr1/Mass1/USH2C) in zebrafish." Gene 353, no. 2 (2005): 200–206. http://dx.doi.org/10.1016/j.gene.2005.05.015.

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7

Yagi, Hideshi, Yoshihiro Noguchi, Ken Kitamura, and Makoto Sato. "Deficiency of Vlgr1 resulted in deafness and susceptibility to audiogenic seizures while the degree of hearing impairment was not correlated with seizure severity in C57BL/6- and 129-backcrossed lines of Vlgr1 knockout mice." Neuroscience Letters 461, no. 2 (2009): 190–95. http://dx.doi.org/10.1016/j.neulet.2009.06.012.

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8

Weston, Michael D., Mirjam W. J. Luijendijk, Kurt D. Humphrey, Claes Möller, and William J. Kimberling. "Mutations in the VLGR1 Gene Implicate G-Protein Signaling in the Pathogenesis of Usher Syndrome Type II." American Journal of Human Genetics 74, no. 2 (2004): 357–66. http://dx.doi.org/10.1086/381685.

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9

Kusuluri, Deva K., Baran E. Güler, Barbara Knapp, et al. "Adhesion G protein-coupled receptor VLGR1/ADGRV1 regulates cell spreading and migration by mechanosensing at focal adhesions." iScience 24, no. 4 (2021): 102283. http://dx.doi.org/10.1016/j.isci.2021.102283.

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10

Hu, Qiao-Xia, Jun-Hong Dong, Hai-Bo Du та ін. "Constitutive GαiCoupling Activity of Very Large G Protein-coupled Receptor 1 (VLGR1) and Its Regulation by PDZD7 Protein". Journal of Biological Chemistry 289, № 35 (2014): 24215–25. http://dx.doi.org/10.1074/jbc.m114.549816.

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11

van Wijk, Erwin, Bert van der Zwaag, Theo Peters, et al. "The DFNB31 gene product whirlin connects to the Usher protein network in the cochlea and retina by direct association with USH2A and VLGR1." Human Molecular Genetics 15, no. 5 (2006): 751–65. http://dx.doi.org/10.1093/hmg/ddi490.

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12

McMillan, D. Randy, and Perrin C. White. "Loss of the transmembrane and cytoplasmic domains of the very large G-protein-coupled receptor-1 (VLGR1 or Mass1) causes audiogenic seizures in mice." Molecular and Cellular Neuroscience 26, no. 2 (2004): 322–29. http://dx.doi.org/10.1016/j.mcn.2004.02.005.

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13

McGee, J., R. J. Goodyear, D. R. McMillan, et al. "The Very Large G-Protein-Coupled Receptor VLGR1: A Component of the Ankle Link Complex Required for the Normal Development of Auditory Hair Bundles." Journal of Neuroscience 26, no. 24 (2006): 6543–53. http://dx.doi.org/10.1523/jneurosci.0693-06.2006.

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14

Rayamajhi, Min B., Paul D. Pratt, Philip W. Tipping, et al. "Seasonal Growth, Biomass Allocation, and Invasive Attributes Manifested by Dioscorea bulbifera L. (Air-Potato) Plants Generated from Bulbils in Florida." Invasive Plant Science and Management 9, no. 3 (2016): 195–204. http://dx.doi.org/10.1614/ipsm-d-16-00022.1.

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Dioscorea bulbifera is a serious invader of various ecosystems in Florida, where plants generated by its two morphotypes climb aggressively and smother supporting vegetation. There is a dearth of published research on its invasive biological attributes including vine growth and biomass production by plants generated from bulbils. Herein, we assessed these parameters in common garden studies by planting bulbils from four biomass categories (PBBCs I–IV) of both morphotypes. Vine lengths, longevity-based growth rates (VLGR), biomass, and quantities of leaves and daughter bulbils in both morphotyp

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15

Fleet, Benjamin, Jenna Malone, Christopher Preston, and Gurjeet Gill. "Target-Site Point Mutation Conferring Resistance to Trifluralin in Rigid Ryegrass (Lolium rigidum)." Weed Science 66, no. 2 (2017): 246–53. http://dx.doi.org/10.1017/wsc.2017.67.

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Populations of rigid ryegrass suspected of resistance to trifluralin due to control failures exhibited varying levels of susceptibility to trifluralin, with 15 out of 17 populations deemed resistant (>20% plant survival). Detailed dose–response studies were conducted on one highly resistant field-evolved population (SLR74), one known multiply resistant population (SLR31), and one susceptible population (VLR1). On the basis of the dose required to kill 50% of treated plants (LD50), SLR74 had 15-fold greater resistance than VLR1, whereas, the multiply resistant SLR31 had 10-fold greater resis

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16

Burnet, Michael W. M., Andrew R. Barr, and Stephen B. Powles. "Chloroacetamide Resistance in Rigid Ryegrass (Lolium rigidum)." Weed Science 42, no. 2 (1994): 153–57. http://dx.doi.org/10.1017/s004317450008019x.

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Metolachlor has been evaluated both as a herbicide for use in cultivated oats (Avena sativaL.) and for its potential as an alternative herbicide for the control of herbicide-resistant rigid ryegrass. Eight herbicide-resistant and two susceptible biotypes of rigid ryegrass were tested for their susceptibility to metolachlor. Response to metolachlor was determined both in soil and an agar germination medium. The LD50for metolachlor in agar for a susceptible biotype (VLR1) was 0.13 μM. Five biotypes, SLR5 (6.9 fold), SLR31 (5.2 fold), SLR10 (2.5 fold), NLR12 (2.1 fold) and VLR69 (1.9 fold), were

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17

Brunton, David J., Peter Boutsalis, Gurjeet Gill, and Christopher Preston. "Resistance to Multiple PRE Herbicides in a Field-evolved Rigid Ryegrass (Lolium rigidum) Population." Weed Science 66, no. 5 (2018): 581–85. http://dx.doi.org/10.1017/wsc.2018.31.

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AbstractA population of rigid ryegrass (Lolium rigidumGaudin) from a field on the Eyre Peninsula, South Australia, was suspected of resistance to thiocarbamate herbicides. Dose–response studies were conducted on this population (EP162) and two susceptible populations (SLR4 and VLR1). The resistant population exhibited cross-resistance to triallate, prosulfocarb, EPTC, and thiobencarb with higher LD50to triallate (14.9-fold), prosulfocarb (9.4-fold), EPTC (9.7-fold), and thiobencarb (13.6-fold) compared with the susceptible populations SLR4 and VLR1. The resistant population also displayed resi

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18

Yu, Gang, Jin-Liang Liu, Li-Qin Xie, Xue-Liang Wang, Shi-Hong Zhang, and Hong-Yu Pan. "Characterization, cloning, and heterologous expression of a subtilisin-like serine protease gene VlPr1 from Verticillium lecanii." Journal of Microbiology 50, no. 6 (2012): 939–46. http://dx.doi.org/10.1007/s12275-012-2199-x.

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19

Brunton, David J., Peter Boutsalis, Gurjeet Gill, and Christopher Preston. "Resistance to very-long-chain fatty-acid (VLCFA)-inhibiting herbicides in multiple field-selected rigid ryegrass (Lolium rigidum) populations." Weed Science 67, no. 3 (2019): 267–72. http://dx.doi.org/10.1017/wsc.2018.93.

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AbstractFive populations of rigid ryegrass (Lolium rigidumGaudin) from fields across cropping regions in southern Australia were suspected of having resistance to thiocarbamates, chloroacetamides, and sulfonylisoxazoline herbicides. Resistant (R) populations 375-14, 198-15, 16.2, EP162, RAC1, and A18 and two susceptible (S) populations (SLR4 and VLR1) were included in a dose–response study. All suspected R populations expressed resistance to one or all herbicides (thiocarbamates, chloroacetamides, and pyroxasulfone). Population 198-15 exhibited the highest LD50to triallate (44.7-fold), prosulf

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20

Burnet, Michael W. M., Quentin Hart, Joseph A. M. Holtum, and Stephen B. Powles. "Resistance to Nine Herbicide Classes in a Population of Rigid Ryegrass (Lolium rigidum)." Weed Science 42, no. 3 (1994): 369–77. http://dx.doi.org/10.1017/s0043174500076645.

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Rigid ryegrass population VLR69 has become resistant to nine classes of herbicides after 21 yr of exposure to five herbicides in five different chemical classes. The population was exposed to diuron in 17 seasons and is resistant to diuron (4 fold) and chlorotoluron (8 fold) when compared with a reference susceptible population (VLR1). VLR69 had six seasons of exposure to chlorsulfuron and exhibits a high level of resistance to chlorsulfuron (> 20 fold) and triasulfuron (> 25 fold) and a lesser change in sensitivity to sulfometuron (7 fold); however, 4% of the population has a high level

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21

Burnet, Michael W. M., John T. Christopher, Joseph A. M. Holtum, and Stephen B. Powles. "Identification of Two Mechanisms of Sulfonylurea Resistance Within One Population of Rigid Ryegrass (Lolium rigidum) Using a Selective Germination Medium." Weed Science 42, no. 3 (1994): 468–73. http://dx.doi.org/10.1017/s0043174500076785.

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A biotype of rigid ryegrass (Lolium rigidumGaudin biotype VLR69) resistant to some ALS inhibitors was characterized to determine the mechanisms of resistance to the sulfonylurea herbicides. The biotype had a high level of resistance to chlorsulfuron (20×) and triasulfuron (25×), and an intermediate level of resistance to imazaquin (7×) and sulfometuron (7.5×) but exhibited a low level of resistance to imazapyr (2.5×). At 60 to 90 g ai ha-1sulfometuron, 4% of the population survived without apparent herbicidal effect The same response to sulfometuron was also observed when seeds of the resistan

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22

Damasceno, Samara, Pablo Augusto de Souza Fonseca, Izinara Cruz Rosse, et al. "Putative Causal Variant on Vlgr1 for the Epileptic Phenotype in the Model Wistar Audiogenic Rat." Frontiers in Neurology 12 (June 9, 2021). http://dx.doi.org/10.3389/fneur.2021.647859.

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Wistar Audiogenic Rat is an epilepsy model whose animals are predisposed to develop seizures induced by acoustic stimulation. This model was developed by selective reproduction and presents a consistent genetic profile due to the several generations of inbreeding. In this study, we performed an analysis of WAR RNA-Seq data, aiming identified at genetic variants that may be involved in the epileptic phenotype. Seventeen thousand eighty-five predicted variants were identified as unique to the WAR model, of which 15,915 variants are SNPs and 1,170 INDELs. We filter the predicted variants by pre-e

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23

Knapp, B., SJ Letteboer, E. van Wijk, et al. "Identification of novel interaction partners for Vlgr1b/GPR98 - a key component of the periciliary Usher syndrome protein network in photoreceptor cells." Cilia 1, S1 (2012). http://dx.doi.org/10.1186/2046-2530-1-s1-p29.

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