Relevant bibliographies by topics / Warburg hypothesis

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Warburg hypothesis'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Warburg hypothesis"

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Liboff, Abraham R. "The Warburg hypothesis and weak ELF biointeractions." Electromagnetic Biology and Medicine 39, no. 2 (2020): 45–48. http://dx.doi.org/10.1080/15368378.2020.1737810.

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Kaczanowski, Szymon, Joanna Klim, and Urszula Zielenkiewicz. "An Apoptotic and Endosymbiotic Explanation of the Warburg and the Inverse Warburg Hypotheses." International Journal of Molecular Sciences 19, no. 10 (2018): 3100. http://dx.doi.org/10.3390/ijms19103100.

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Otto Warburg, a Nobel prize winner, observed that cancer cells typically “switch” from aerobic to anaerobic respiration. He hypothesized that mitochondrial damage induces neoplastic transformation. In contrast, pathological aging is observed mainly in neuron cells in neurodegenerative diseases. Oxidative respiration is particularly active in neurons. There is inverse comorbidity between cancer and neurodegenerative diseases. This led to the creation of the “inverse Warburg hypothesis”, according to which excessive mitochondrial activity induces pathological aging. The findings of our studies s
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Gonzalez, Claudio D., Silvia Alvarez, Alejandro Ropolo, Carla Rosenzvit, Maria F. Gonzalez Bagnes, and Maria I. Vaccaro. "Autophagy, Warburg, and Warburg Reverse Effects in Human Cancer." BioMed Research International 2014 (2014): 1–10. http://dx.doi.org/10.1155/2014/926729.

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Autophagy is a highly regulated-cell pathway for degrading long-lived proteins as well as for clearing cytoplasmic organelles. Autophagy is a key contributor to cellular homeostasis and metabolism. Warburg hypothesized that cancer growth is frequently associated with a deviation of a set of energy generation mechanisms to a nonoxidative breakdown of glucose. This cellular phenomenon seems to rely on a respiratory impairment, linked to mitochondrial dysfunction. This mitochondrial dysfunction results in a switch to anaerobic glycolysis. It has been recently suggested that epithelial cancer cell
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Ganapathy, Vadivel, Muthusamy Thangaraju, and Puttur D. Prasad. "Nutrient transporters in cancer: Relevance to Warburg hypothesis and beyond." Pharmacology & Therapeutics 121, no. 1 (2009): 29–40. http://dx.doi.org/10.1016/j.pharmthera.2008.09.005.

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SHIOTA, G. "The hypothesis of Warburg revisited: A metabolic switch in carcinogenesis." Hepatology Research 36, no. 3 (2006): 156–57. http://dx.doi.org/10.1016/j.hepres.2006.08.012.

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Haowen, Jiang, Rachel L. Greathouse, Bo He, et al. "Abstract 2180: Deciphering the Warburg effect: Redox is the key to tumor differentiation." Cancer Research 82, no. 12_Supplement (2022): 2180. http://dx.doi.org/10.1158/1538-7445.am2022-2180.

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Abstract It was proposed by Warburg that the cause and consequence of the Warburg effect were the injury of respiration and cell dedifferentiation, respectively. However, the underlying mechanism was unclear due to limited understanding of metabolic control of epigenetics. Inhibition of mitochondrial electron transport chain (ETC) causes reduction of NAD+/NADH ratio. Recently it was discovered that this redox stress not only drives lactate production from pyruvate to enhance the Warburg effect, but also promotes the conversion of α-ketoglutarate (αKG) to 2-hydroxyglutarate (2HG), turning the s
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Schuster, Stefan, Daniel Boley, Philip Möller, Heiko Stark, and Christoph Kaleta. "Mathematical models for explaining the Warburg effect: a review focussed on ATP and biomass production." Biochemical Society Transactions 43, no. 6 (2015): 1187–94. http://dx.doi.org/10.1042/bst20150153.

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For producing ATP, tumour cells rely on glycolysis leading to lactate to about the same extent as on respiration. Thus, the ATP synthesis flux from glycolysis is considerably higher than in the corresponding healthy cells. This is known as the Warburg effect (named after German biochemist Otto H. Warburg) and also applies to striated muscle cells, activated lymphocytes, microglia, endothelial cells and several other cell types. For similar phenomena in several yeasts and many bacteria, the terms Crabtree effect and overflow metabolism respectively, are used. The Warburg effect is paradoxical a
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Alberghina, Lilia. "The Warburg Effect Explained: Integration of Enhanced Glycolysis with Heterogeneous Mitochondria to Promote Cancer Cell Proliferation." International Journal of Molecular Sciences 24, no. 21 (2023): 15787. http://dx.doi.org/10.3390/ijms242115787.

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The Warburg effect is the long-standing riddle of cancer biology. How does aerobic glycolysis, inefficient in producing ATP, confer a growth advantage to cancer cells? A new evaluation of a large set of literature findings covering the Warburg effect and its yeast counterpart, the Crabtree effect, led to an innovative working hypothesis presented here. It holds that enhanced glycolysis partially inactivates oxidative phosphorylation to induce functional rewiring of a set of TCA cycle enzymes to generate new non-canonical metabolic pathways that sustain faster growth rates. The hypothesis has b
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Hofmann, Peter. "Cancer and Exercise: Warburg Hypothesis, Tumour Metabolism and High-Intensity Anaerobic Exercise." Sports 6, no. 1 (2018): 10. http://dx.doi.org/10.3390/sports6010010.

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Rhodes, Christopher J. "The cancer hypothesis of pulmonary arterial hypertension: are polyamines the new Warburg?" European Respiratory Journal 56, no. 5 (2020): 2002350. http://dx.doi.org/10.1183/13993003.02350-2020.

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Dissertations / Theses on the topic "Warburg hypothesis"

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Oppermann, Henry, Yonghong Ding, Jeevan Sharma, et al. "Metabolic response of glioblastoma cells associated with glucose withdrawal and pyruvate substitution as revealed by GC-MS." Universitätsbibliothek Leipzig, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-213891.

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Background: Tumor cells are highly dependent on glucose even in the presence of oxygen. This concept called the Warburg effect is a hallmark of cancer and strategies are considered to therapeutically exploit the phenomenon such as ketogenic diets. The success of such strategies is dependent on a profound understanding of tumor cell metabolism. With new techniques it is now possible to thoroughly analyze the metabolic responses to the withdrawal of substrates and their substitution by others. In the present study we used gas chromatography coupled to mass spectrometry (GC-MS) to analyze how gli
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Book chapters on the topic "Warburg hypothesis"

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Narayanan, Samyukta, Anirudh Santhoshkumar, Srijit Ray, and Sitaram Harihar. "Reprogramming of Cancer Cell Metabolism: Warburg and Reverse Warburg Hypothesis." In Cancer Cell Metabolism: A Potential Target for Cancer Therapy. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-1991-8_2.

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Ristow, Michael, and José M. Cuezva. "Oxidative Phosphorylation and Cancer: The Ongoing Warburg Hypothesis." In Cellular Respiration and Carcinogenesis. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-435-3_1.

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Conference papers on the topic "Warburg hypothesis"

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Cha, Youngsu, Matteo Aureli, and Maurizio Porfiri. "On a Physics-Based Model of the Electrical Impedance of Ionic Polymer Metal Composites." In ASME 2012 Conference on Smart Materials, Adaptive Structures and Intelligent Systems. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/smasis2012-7982.

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In this paper, we analyze the chemoelectrical behavior of ionic polymer metal composites (IPMCs) in the small voltage range with a novel hypothesis on the charge dynamics in proximity of the electrodes. Specifically, this paper introduces a so-called composite layer which extends between the polymer membrane and the metal electrode. This homogeneous layer describes the charge distribution at the electrode via two species of charge carriers, that is, electrons and mobile counterions. Charge dynamics is described by adapting the multi-physics formulation based on the Poisson-Nernst-Planck (PNP)
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