Academic literature on the topic 'Wei sheng fang mian'

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Journal articles on the topic "Wei sheng fang mian"

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Cao, Liang, Nuzhat Shehla, Bin Li, et al. "Schinortriterpenoids from Tujia ethnomedicine Xuetong-The stems of Kadsura heteroclita." Phytochemistry 169 (January 31, 2020): 1–7. https://doi.org/10.1016/j.phytochem.2019.112178.

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Cao, Liang, Shehla, Nuzhat, Li, Bin, Jian, Yuqing, Peng, Caiyun, Sheng, Wenbing, Liu, Leping, Cai, Xiong, Man, Rongyong, Liao, Duan-Fang, Choudhary, M. Iqbal, Rahman, Atta-ur, Wang, Wei (2020): Schinortriterpenoids from Tujia ethnomedicine Xuetong-The stems of Kadsura heteroclita. Phytochemistry (112178) 169: 1-7, DOI: 10.1016/j.phytochem.2019.112178, URL: http://dx.doi.org/10.1016/j.phytochem.2019.112178
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Han, Haicheng, Rui Fang, Dan Wang, Yong Yang, Xiaoqing Fu, and Kangle Rui. "Elucidation of mechanisms of action of Wei-Sheng-Fang-Yi-Bao-Dan in the treatment of COVID-19 and depression using network pharmacology and molecular docking." Tropical Journal of Pharmaceutical Research 21, no. 9 (2022): 1939–49. http://dx.doi.org/10.4314/tjpr.v21i9.18.

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Purpose: To investigate the mechanisms of action of Wei-Sheng-Fang-Yi-Bao-Dan (WSFYBD) in the treatment of COVID-19 and depression using network pharmacology and molecular docking.
 Methods: First, the bioactive components and target genes of WSFYBD were retrieved from TCMSP database. The relevant gene targets of depression and COVID-19 were obtained from databases. The core WSFYBD genes for treatment were separately obtained by determining gene intersection. Cytoscape 3.8.0 software was used to draw the visual interactive networks. STRING database was employed to construct protein-protei
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Li, Qing-Jie, Xue-Liang Fang, Ying-Qin Li, et al. "Abstract 1999: DCAF7 acts as a scaffold to recruit USP10 for G3BP1 deubiquitylation and facilitates chemoresistance and metastasis in nasopharyngeal carcinoma." Cancer Research 84, no. 6_Supplement (2024): 1999. http://dx.doi.org/10.1158/1538-7445.am2024-1999.

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Abstract Though docetaxel plus cisplatin and 5-fluorouracil (TPF) induction chemotherapy becomes the standard care for locoregionally advanced nasopharyngeal carcinoma (NPC), some patients could not benefit from this therapy. The underlying mechanisms remain unclear. We found that DCAF7 was highly expressed in TPF resistant NPC patients, and promoted the cisplatin resistance and metastasis of NPC cells. Mechanistically, DCAF7 facilitates the interaction between USP10 and G3BP1, resulting in the removal of K48-linked ubiquitylation of G3BP1 at Lys76 mediated by USP10, thus preventing the degrad
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Teng, Qiu-Xu, Zi-Ning Lei, Wei Zhang, et al. "Abstract 1095: Establishment and characterization of a topotecan resistant lung cancer NCI H460/TPT10 cell line and a tumor xenograft model." Cancer Research 82, no. 12_Supplement (2022): 1095. http://dx.doi.org/10.1158/1538-7445.am2022-1095.

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Abstract While topotecan (TPT) is a chemotherapeutic drug in treating lung cancer, the development of TPT resistance in tumors reserves as a major obstacle to chemotherapeutic success. Therefore, a better understanding of the mechanisms of TPT resistance is critical. In this study, the first topotecan-resistant human non-small cell lung cancer (NSCLC) cell line, termed NCI-H460/TPT10, was established from the parental NCI-H460 cell line. NCI-H460/TPT10 cells exhibited a 394.7-fold resistance to TPT, and cross-resistance to SN-38, mitoxantrone, and doxorubicin, compared to parental NCI-H460 cel
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Lu, Shun, Ziming Li, Yan Wang, et al. "Abstract CT009: SHR-A1811, a HER2-directed antibody-drug conjugate (ADC), in advanced HER2-mutant non-small cell lung cancer (NSCLC): Updated phase 2 results from HORIZON-Lung." Cancer Research 85, no. 8_Supplement_2 (2025): CT009. https://doi.org/10.1158/1538-7445.am2025-ct009.

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Abstract Background: SHR-A1811 is a novel ADC consisting of a humanized HER2-directed monoclonal antibody, cleavable tetrapeptide-based linker, and DNA topoisomerase I inhibitor. We conducted a multicenter, open-label, phase 1/2 study to evaluate SHR-A1811 in HER2-altered NSCLC. In the registrational phase 2 portion in patients (pts) with HER2-mutant NSCLC, the primary endpoint of IRC-assessed ORR was met (DCO, Jun. 14, 2024). Here, we presented an updated analysis after an additional 6-mo follow-up, focusing on survival outcomes. Methods: Pts with advanced NSCLC and centrally confirmed HER2 m
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Fang, Yin-Ying, Chi-Fang Chen, and Sheng-Ju Wu. "Feature identification using acoustic signature of Ocean Researcher III (ORIII) of Taiwan." ANZIAM Journal 59 (July 25, 2019): C318—C357. http://dx.doi.org/10.21914/anziamj.v59i0.12655.

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Underwater acoustic signature identification has been employed as a technique for detecting underwater vehicles, such as in anti-submarine warfare or harbour security systems. The underwater sound channel, however, has interference due to spatial variations in topography or sea state conditions and temporal variations in water column properties, which cause multipath and scattering in acoustic propagation. Thus, acoustic data quality control can be very challenging. One of challenges for an identification system is how to recognise the same target signature from measurements under different te
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Chen, Yaoyu, Jinhong Chen, Yali Guo, et al. "Abstract B173: The anti-tumor activity of CDK2 inhibition alone or in combination with other anti-cancer agents in human cancers." Molecular Cancer Therapeutics 22, no. 12_Supplement (2023): B173. http://dx.doi.org/10.1158/1535-7163.targ-23-b173.

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Abstract Cyclin-dependent kinase 2 (CDK2) plays a crucial role in the regulation of the cell cycle and is involved in a variety of biological processes. It is responsible for phosphorylating proteins involved in G1 and S phase cell cycle progression, DNA damage response, intracellular transport, protein degradation, signal transduction, and DNA and RNA metabolism. CDK2 activity is particularly significant in cells exhibiting CCNE1 amplification or overexpression. Additionally, CDK2 activity has been linked to potential resistance against chemotherapy or targeted therapy. Several studies have i
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Lee, Chuan-Chun, Wan-Rong Wu, Liang-Chih Liu, et al. "Abstract 6051: Opposing function of USP5 in MRE11 endonuclease activity to evade anti-tumor immunity." Cancer Research 85, no. 8_Supplement_1 (2025): 6051. https://doi.org/10.1158/1538-7445.am2025-6051.

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Abstract Proliferating cancer cells frequently encounter replication stress, which is known to generate abnormal cytosolic DNA and trigger anti-tumor immune responses. Thus, the ability to evade immune surveillance is critical for the development and progression of malignant tumors for which the mechanisms are not fully understood. The MRE11 nuclease, essential for maintaining DNA integrity during cell proliferation, plays important roles in processing stalled replication forks during replication stress. This process produces single-stranded DNA (ssDNA) intermediates which activate the cGAS-ST
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Li, Ziming, Xiaomin Dang, Dingzhi Huang, et al. "Abstract CT246: Open-label, single-arm, multicenter, phase 2 trial of garsorasib in KRAS G12C-mutated non-small-cell lung cancer." Cancer Research 84, no. 7_Supplement (2024): CT246. http://dx.doi.org/10.1158/1538-7445.am2024-ct246.

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Abstract Background: Garsorasib (D-1553), a potent KRAS G12C inhibitor, has previously demonstrated promising anti-tumor activity in KRAS G12C-mutated non-small-cell lung cancer (NSCLC) in a phase 1 study. Here we present the efficacy and safety of garsorasib in a pivotal phase 2 study. Methods: In this open-label, multicenter, single-arm phase 2 study, patients received garsorasib 600 mg twice daily in 21-day cycles. Eligible criteria included patients with locally advanced or metastatic NSCLC harboring KRAS G12C mutation, who had disease progression after prior anti-PD-(L)1 therapy and plati
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Schomer, Nathan, Jinhong Chen, Wei Pang, et al. "Abstract A022: CDK2 inhibition demonstrates synthetic lethality in SCLC through apoptotic induction." Molecular Cancer Therapeutics 22, no. 12_Supplement (2023): A022. http://dx.doi.org/10.1158/1535-7163.targ-23-a022.

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Abstract Small cell lung cancer (SCLC), a malignant thoracic neoplasm genetically defined by dysregulation of the Retinoblastoma (RB1) gene and tumor suppressor protein 53 (TP53), remains one of the world’s deadliest tumors with a 5-year overall survival rate ranging from 5% to 10%. Despite numerous clinical trials and recent FDA approval of immune checkpoint inhibitors, the prognosis for SCLC has not significantly improved in decades. There remains a strong need for new therapeutic approaches. In this study, we found that the combined effect of dysregulation or deletion of RB1 and TP53, as is
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Dissertations / Theses on the topic "Wei sheng fang mian"

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Peng, Dun. "Xuan xue de chan sheng : si wei fang fa zhuan xing guo cheng de yan jiu = The establishment of "Xuanxue" : a study of the changing modes of thinking /." click here to view the abstract and table of contents, 1998. http://net3.hkbu.edu.hk/~libres/cgi-bin/thesisab.pl?pdf=b15646385a.pdf.

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Lee, Che-hung. "Guangzhou fang yan yong yu "du" gan rao xue sheng shu mian yu xue xi qing kuang chu tan A study on the influence of the Cantonese word "Dou" in the learning of written Chinese of Hong Kong students /." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B42554275.

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Books on the topic "Wei sheng fang mian"

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Chunjie, Chen, and Li Xiaodong, eds. Zhongguo ru shi wen ti bao gao: Ying xiang zheng fu, qi ye he bai xing sheng huo de fang fang mian mian = Zhongguo rushi wenti baogao. Zhongguo she hui ke xue chu ban she, 2002.

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National Geographic Society (U.S.), ed. Zhong ji ke xue bai ke: Yi kui yuan zi de mian mao, tan suo sheng wu de shi jie, ren shi yu zhou de ao miao : quan fang wei jie da "shen me shi ke xue". Da shi guo ji wen hua you xian gong si, 2017.

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Hu'nan dian shi tai "fu bei de zhan zheng sui yue" lan mu zu. Fu bei de zhan zheng sui yue: 6 wei Xiang ji kai guo da jiang hou bei yan zhong fu bei de chuan qi ren sheng. Wan juan chu ban gong si, 2009.

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translator, Huang Xiaohan, ed. Nao shen jing wai ke yi shi jiao ni chi shen qi da suan you: Fang yun xuan, huo hua da nao, ti sheng mian yi li! = Bokenai nō o tsukuru ninnikuyu. Shi mao chu ban you xian gong si, 2016.

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Yi lin za zhi she, ed. Fang xia jia mian ying de ren sheng. Ji lin she ying chu ban she, 2011.

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China). Wei sheng fang yi zhan Yangpu Qu (Shanghai. Wei sheng fang yi zhi, 1950-1990. [Shanghai Shi Yangpu Qu wei sheng fang yi zhan], 1993.

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1930-, Liu Fenghan, Wang Zhenghua, Cheng Yuhuang, and Guo shi guan (China : Republic : 1949- ), eds. Wei Yongning xian sheng fang tan lu. Guo shi guan, 1994.

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Bao, Qiguo. Huainan Shi wei sheng fang yi zhi. [Publisher not identified], 2003.

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Wen, Zhansheng. Gaoping Shi wei sheng fang yi zhi. Shanxi ren min chu ban she, 2009.

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Ganzhou Shi ji bing yu fang kong zhi zhong xin. Ganzhou Shi wei sheng fang yi zhi. Ganzhou Shi ji bing yu fang kong zhi zhong xin, 2003.

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Book chapters on the topic "Wei sheng fang mian"

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"Glossary Bailian Jiao White Lotus teachings baimiao paying homage at the temples ban band caijie street procession cha, ban small cymbals chi hui hold a feast for all the households chuige songs for winds chuigu shou drumers and wind players dangzi gong in frame daqu large pieces dizi transverse flute with kazoo membrane fang dengke releasing the lanterns fangshe or shishi pardon and distribution of food fang hedeng releasing the river lanterns gongche traditional notation gu large barrel drum guanshi, zan guan helper guanzi double-reed pipe guyueban, chuida ban wind-and-percussion band huahui assembly of performing troupes hui association huishou association head nanyue the southern music nao, bo large cymbals pai prelude paizi percussion pieces, cf. the melodic qupai qu pieces qupai labelled melodies shang miao going to the temple shan hui charitable associations she society she hui altar assembly shen body sheng free-reed mouth organ sheng-guan yue type of wind-and-percussion music shenghui outstanding association shifu masters tao suites wei tail xiangshou incense head xiaoqu small pieces xueshi learning the [ritual] business xuyuan make vows to the gods." In Tradition & Change Performance. Routledge, 2012. http://dx.doi.org/10.4324/9780203985656-10.

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