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1

Lean, Michael E. J. Clinical handbook of weight management. London: Martin Dunitz, Ltd., distributed in the USA, Canada and Brazil by Blackwell Science, 1988.

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2

Pearson, Dympna. Weight management: A practitioner's guide. Chichester, West Sussex: Wiley-Blackwell, 2012.

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3

Mullen, Mary Catherine. ADA pocket guide to pediatric weight management. Chicago: American Dietetic Association, 2010.

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4

Ellen, Shield Jo, and American Dietetic Association, eds. ADA pocket guide to pediatric weight management. Chicago: American Dietetic Association, 2010.

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5

Mullen, Mary Catherine. ADA pocket guide to pediatric weight management. [Chicago]: American Dietetic Association, 2010.

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6

Towell, Todd L. Practical weight management in dogs and cats. Chichester, West Sussex, UK: Wiley-Blackwell, 2011.

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7

Annette, Maggi, ed. Weight management for type II diabetes: An action plan. Minneapolis, MN: Chronimed, 1997.

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8

Laliberte, Michele. The cognitive behavioral workbook for weight management: A step-by-step program. Oakland, CA: New Harbinger Publications, 2009.

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9

D, Brownell Kelly, and LEARN Education Center, eds. The LEARN program for weight management 2000: Lifestyle, exercise, attitudes, relationships, nutrition. Dallas, Tex: American Health Pub. Co., 2000.

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10

Laliberte, Michele. The cognitive behavioral workbook for weight management: A step-by-step program. Oakland, CA: New Harbinger Publications, 2009.

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11

Laliberte, Michele. The cognitive behavioral workbook for weight management: A step-by-step program. Oakland, CA: New Harbinger, 2009.

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12

E, McCabe Randi, and Taylor Valerie, eds. The cognitive behavioral workbook for weight management: A step-by-step program. Oakland, CA: New Harbinger, 2009.

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13

Joseph, Mason. Let my heart attack save your life: A simple, sound workable weight management plan. Minneapolis, MN: Chronimed Pub., 1998.

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14

Franz, Marion J. ADA pocket guide to lipid disorders, hypertension, diabetes, and weight management. Chicago: American Dietetic Association, 2011.

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15

Aiyana, Juliette. Chinese medicine & healthy weight management: An evidence-based integrated approach. Boulder, Colo: Blue Poppy Press, 2007.

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16

Maximize your body potential: 16 weeks to a lifetime of effective weight management. Palo Alto, Calif: Bull Pub. Co., 1986.

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17

Aerobic walking: The weight-loss exercise : a complete program to reduce weight, stress, and hypertension. New York: Wiley, 1995.

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18

Van Itallie, Theodore B., 1919- and Simopoulos Artemis P. 1933-, eds. Obesity: New directions in assessment and management. Philadelphia: Charles Press, 1995.

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19

Royal College of Physicians of London. Nutrition Committee., ed. Anti-obesity drugs: Guidance on appropriate prescribing and management. London: Royal College of Physicians of London, 2003.

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20

F, Roizen Michael, ed. You on a diet: The owner's manual for waist management. Waterville, Me: Thorndike Press, 2006.

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21

Towell, Todd L. Practical Weight Management in Dogs and Cats. Wiley & Sons, Incorporated, John, 2011.

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22

Towell, Todd L. Practical Weight Management in Dogs and Cats. Wiley & Sons, Incorporated, John, 2011.

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23

Towell, Todd L. Practical Weight Management in Dogs and Cats. Wiley & Sons, Incorporated, John, 2011.

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24

Grace, Clare, and Dympna Pearson. Weight Management: A Practitioner's Guide. Wiley & Sons, Incorporated, John, 2012.

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25

Grace, Clare, and Dympna Pearson. Weight Management: A Practitioner's Guide. Wiley & Sons, Incorporated, John, 2012.

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26

Grace, Clare, and Dympna Pearson. Weight Management: A Practitioner's Guide. Wiley & Sons, Limited, John, 2013.

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27

Grace, Clare, and Dympna Pearson. Weight Management: A Practitioner's Guide. Wiley & Sons, Incorporated, John, 2012.

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28

McCabe, Randi E., Valerie Taylor, and Michele Laliberte. Cognitive Behavioral Workbook for Weight Management: A Step-By-Step Program. New Harbinger Publications, 2009.

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29

McCabe, Randi E., Valerie Taylor, and Michele Laliberte. Cognitive Behavioral Workbook for Weight Management: A Step-By-Step Program. New Harbinger Publications, 2009.

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30

Maggi, Annette, and Jackie Labat. Weight Management for Type II Diabetes: An Action Plan. Wiley & Sons, Incorporated, John, 2008.

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31

Maggi, Annette, and Jackie Labat. Weight Management for Type II Diabetes: An Action Plan. Wiley & Sons, Incorporated, John, 1997.

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32

McTiernan, Anne. Cancer Prevention and Management Through Exercise and Weight Control. Taylor & Francis Group, 2016.

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33

Cancer prevention and management through exercise and weight control. Boca Raton, FL: CRC/Taylor & Francis, 2005.

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34

Anne, McTiernan, ed. Cancer prevention and management through exercise and weight control. Boca Raton, FL: Taylor & Francis, 2005.

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35

McTiernan, Anne. Cancer Prevention and Management Through Exercise and Weight Control. Taylor & Francis Group, 2019.

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36

Anderson, Nina, Zakir Ramazanov, and Maria Del Mar Bernal Suarez. Stress and Weight Management: Effective Herbal Therapy Using Rhodiola Rosea and Rhododendron Caucasicum. National Bioscience, 2003.

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37

Wiffen, Philip, Marc Mitchell, Melanie Snelling, and Nicola Stoner. Therapy-related issues: palliative care. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199603640.003.0027.

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Anorexia and cachexia 578Constipation 578Fatigue 579Hypercalcaemia of malignancy 580Mouth care 581Noisy breathing 581Insomnia 582Spinal cord compression 582Malignant bowel obstruction 583Syringe drivers and compatibility of medicines 584End-of-life pathways 585Anaemia 586The dictionary definition of anorexia is a lack of appetite (for food). Cachexia is involuntary weight loss which can progress to an emaciated state. The majority of palliative care patients experience cachexia at some stage, and this difficult condition requires determination of the cause, if possible, and development of careful management. A number of Cochrane reviews have been published which summarize a fairly large volume of literature. The following interventions may be considered. ...
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38

McCarron, Robert M., Amir Ramezani, Ian Koebner, Samir J. Sheth, and Jessica Palka. Integrated Chronic Pain and Psychiatric Management. Edited by Robert E. Feinstein, Joseph V. Connelly, and Marilyn S. Feinstein. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190276201.003.0023.

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Both physical pain and psychiatric disorders are widely prevalent, and collectively they account for the most frequently presenting complaints in the primary care setting. These conditions are a complex challenge for both the patient and provider, with frequent high use of medical services and increased morbidity. The Integrated Behavioral Pain Medicine (IBPM) treatment model incorporates a multidisciplinary, biopsychosocial, team-based approach for patients who have chronic and largely treatment-refractory pain. IBPM uses an integrated care team of providers and coordinators, who collectively work with the chronic pain patient to individualize a pain management plan, which may include pharmacologic management, cognitive-behavioral therapy, trauma-focused therapy, biofeedback, mindfulness, acupuncture, nutrition, behavioral weight and sleep management, and physical therapy. Ideally, primary care providers will refer patients to an IBPM model of care, but if the treatment model is not available in a specific area, a piecemeal approach with partial use of services is recommended.
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39

MacCallum, Niall S. Management of oncological complications in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0376.

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Important treatment complications relevant to critical care are discussed. Cancer-related pain is complex and requires multidisciplinary care, particularly in the peri-operative setting. Chemotherapeutic complications include pancytopenia, cardiac, pulmonary, renal, gastrointestinal, hepatic, and neurotoxicity. Radiotherapy complications include cardiac, pulmonary, and gastrointestinal toxicity. In general, management includes assessing the risk-benefit to cytotoxic therapy withdrawal and supportive care. There is a paucity of proven treatment options for most complications, althoughcertain therapies are used to prevent and/or treat complications (e.g. tumour lysis syndrome). Thromboembolic disease is a common cause of mortality and morbidity; low molecular weight heparin therapy may be superior to oral anticoagulation.
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40

Nissenson, Allen R., John Moran, and Robert Provenzano. Overview of dialysis patient management and future directions. Edited by Jonathan Himmelfarb. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0267_update_001.

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Nearly 2 million patients worldwide have end-stage renal disease (ESRD) and require dialysis or kidney transplantation. The advent of clinical dialysis in the 1950s has had a huge impact on the way ESRD and acute kidney injury are managed, but several decades later, the morbidity and mortality in patients with ESRD remain unacceptably high and patients often have a poor quality of life. Many believe that we have focused attention on a few key treatment-related outcomes, and have done well with these (i.e. anaemia, adequacy of dialysis, metabolic bone disease), but achieving great results in only these domains has clearly not been sufficient to drive improvements in survival or patient-reported outcomes. Recent experience with integrated care management, focusing on comorbidity management, offers promise. In addition, a number of investigators have been challenging the current thrice-weekly, diffusion-based treatment paradigm and have been developing approaches to emulate the function of natural kidneys. Thus an ideal care delivery model would focus on the holistic needs of the patient with kidney disease, while the ideal form of renal replacement therapy would mimic native kidneys, operating continuously, removing solutes with a molecular-weight spectrum similar to that of native kidneys, removing water and solutes on the basis of individual patient needs, and would be biocompatible, wearable, and ideally implantable. It would also be low cost, reliable, and safe. A few years ago, these technical requirements would have seemed impossible to achieve, but with advances in the sciences of nanotechnology and microfluidics, renal replacement of the future may come closer to this ideal.
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41

Bessesen, Daniel, and Robert F. Kushner. Evaluation & Management of Obesity. Hanley & Belfus, 2002.

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42

Jordan, Henry A., and Jean Storlie. Nutrition and Exercise in Obesity Management. Springer, 2013.

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43

Nutrition and Exercise in Obesity Management. Springer, 2012.

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44

Ferro, Charles J., and Khai Ping Ng. Recommendations for management of high renal risk chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0099.

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Poorer renal function is associated with increasing morbidity and mortality. In the wider population this is mainly as a consequence of cardiovascular disease. Renal patients are more likely to progress to end-stage renal disease, but also have high cardiovascular risk. Aiming to reduce both progression of renal impairment and cardiovascular disease are not contradictory. Focusing on the management of high-risk patients with proteinuria and reduced glomerular filtration rates, it is recommended that blood pressure should be kept below 140/90, or 130/80 if proteinuria is > 1 g/24 h (protein:creatinine ratio (PCR) >100 mg/mmol or 0.9 g/g). These targets may be modified according to age and other factors. Angiotensin-converting enzyme inhibitors (ACEIs) or angiotensin receptor antagonists should form part of the therapy for patients with proteinuria > 0.5 g/24 h (PCR > 50 mg/mmol or 0.45 g/g). Use of ACEIs or angiotensin receptor blockers in patients with lower levels of proteinuria may be indicated in some patient groups even in the absence of hypertension, notably in diabetic nephropathy. Evidence that other agents that reduce proteinuria bring additional benefits is weak at present. The best studies of ‘dual-blockade’ with various combinations of ACEIs, ARBs, and renin inhibitors have shown additional hazard with little evidence of additional benefit. Hyperlipidaemia—regardless of lipid levels, statin therapy is indicated in secondary cardiovascular prevention, and in primary prevention where cardiovascular risk is high, noting that current risk estimation tools do not adequately account for the increased risk of patients with CKD. There is not substantial evidence that lipid lowering therapy impacts on average rates of loss of GFR in progressive CKD. Non-drug lifestyle interventions to reduce cardiovascular risk, including stopping smoking, are important for all. Acidosis—in more advanced CKD it is justified to treat acidosis with oral sodium bicarbonate. Diet—sodium restriction to < 100 mmol/day (6 g/day) and avoidance of excessive dietary protein are justified in early to moderate CKD. Recommendations to limit levels of protein to 0.8 g/kg body weight are suggested by some, but additional protective effects of this are likely to be slight in patients who are otherwise well managed. Low-protein diets may carry some risk. Lower-protein diets may however be used to prevent symptoms in advanced CKD not treated by dialysis.
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45

Jörres, Achim, Dietrich Hasper, and Michael Oppert. Fluid overload in acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0229.

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A central objective in the management of acute kidney injury is the restoration and maintenance of adequate systemic and renal perfusion, often requiring the parallel administration of fluids and vasoactive drugs. However, hypovolaemia and fluid overload may both predispose the patient to complications and poor outcomes. Therefore, body weight and daily fluid intake/output should be recorded, patients should continuously be assessed for clinical signs of under- or over-hydration, and adequate monitoring of haemodynamic parameters should be performed. Together these parameters constitute the basis for individualized fluid therapy that needs to be initiated promptly and should be re-evaluated at least on a daily basis.
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46

Taylor, Christopher, and Sally Connolly. Gastrointestinal disease and nutrition. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198702948.003.0007.

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This chapter discusses the common gastrointestinal and nutritional consequences of cystic fibrosis (CF) including hepato-biliary disease. The pathophysiology of obstructive gut disease (meconium ileus and distal ileal obstruction) is discussed with reference to CFTR dysfunction. The diagnosis and management of gastro-oesophageal reflux, an increasingly common problem in both children and adults with CF, is also considered in some depth. A new section on eosinophilic gut disease has been added. The importance of nutrition in maintaining lung function is emphasized with a section on pancreatic enzyme physiology and guidance on optimizing pancreatic enzyme replacement therapy. An investigation plan to exclude concomitant gut disease for a child with poor weight gain is given, together with a section on invasive nutritional support. The diagnosis of hepatic disease and biliary complications is considered together with suggested treatments and a long-term monitoring plan
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47

Sivan, Shobana, and Sankar D. Navaneethan. Acute Kidney Injury after Bariatric Surgery. Edited by Tomasz Rogula, Philip Schauer, and Tammy Fouse. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190608347.003.0020.

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Bariatric surgery is an effective and durable treatment option for weight loss and remission of diabetes in obesity. Incidence of acute kidney injury (AKI) after bariatric surgery has ranged between 2.8% and 8.5%, depending on the definition used in the studies. Published reports have used serum creatinine alone in determining the incidence of AKI and thereby have underestimated AKI prevalence. AKI has prognostic significance among bariatric surgery patients and is associated with increased healthcare utilization and increased mortality in patients with AKI when compared to patients who do not sustain AKI after bariatric surgery. AKI management in bariatric surgical patients is often similar to management in other postoperative patients, involving optimal volume management, avoidance of nephrotoxic agents, including contrast, and supportive care. Early involvement of nephrologists is helpful in instituting appropriate care for these patients.
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48

Oz, Mehmet, Michael F., M.D. Roizen, Ted Spiker, and Craig Wynett. You: on a Diet: The Owner's Manual for Waist Management. Thorndike Press, 2007.

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49

Hay, Phillipa J., and Angélica de M. Claudino. Evidence-Based Treatment for the Eating Disorders. Edited by W. Stewart Agras. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195373622.013.0025.

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This chapter comprises a focused review of the best available evidence for psychological and pharmacological treatments of choice for anorexia nervosa (AN), bulimia nervosa (BN), binge eating disorder (BED), and other specified and unspecified feeding and eating disorders (OSFED and UFED), discusses the role of primary care and online therapies, and presents treatment algorithms. In AN, although there is consensus on the need for specialist care that includes nutritional rehabilitation in addition to psychological therapy, no single approach has yet been found to offer a distinct advantage. In contrast, manualized cognitive behavior therapy (CBT) for BN has attained “first-line” treatment status with a stronger evidence base than other psychotherapies. Similarly, CBT has a good evidence base in treatment of BED and for BN, and BED has been successfully adapted into less intensive and non-specialist forms. Behavioral and pharmacological weight loss management in treatment of co-morbid obesity/overweight and BED may be helpful in the short term, but long-term maintenance of effects is unclear. Primary care practitioners are in a key role, both with regard to providing care and with coordination and initiation of specialist care. There is an emerging evidence base for online therapies in BN and BED where access to care is delayed or problematic.
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50

Managing Newborn Problems: A Guide For Doctors, Nurses And Midwives (Integrated Management of Pregnancy and Childbirth). World Health Organization, 2004.

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