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Journal articles on the topic 'Weight-recovered anorexia nervosa'

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Published: 4 June 2021

Last updated: 15 February 2022

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1

O'Dwyer, A.-M., J. V. Lucey, and G. F. M. Russell. "Serotonin activity in anorexia nervosa after long-term weight restoration: response to D-fenfluramine challenge." Psychological Medicine 26, no. 2 (1996): 353–59. http://dx.doi.org/10.1017/s0033291700034747.

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SynopsisAbnormalities in central serotonin function have been implicated in the pathogenesis of anorexia nervosa. It is difficult, however, to separate neuroendocrine abnormalities induced by weight loss and malnutrition from those related primarily to the disorder itself. To minimize these influences, this study assessed long-term weight restored anorexics. A correlation between persistent eating-related psychopathology, co-morbid illness and serotonin dysfunction was sought. Nine female weight-restored out-patients who had previously fulfilled DSM-III-R criteria for anorexia nervosa and nine

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2

Eckert, E. D., K. A. Halmi, P. Marchi, W. Grove, and R. Crosby. "Ten-year follow-up of anorexia nervosa: clinical course and outcome." Psychological Medicine 25, no. 1 (1995): 143–56. http://dx.doi.org/10.1017/s0033291700028166.

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SynopsisThe clinical course and outcome of anorexia nervosa are presented in a 10-year followup study of 76 severely ill females with anorexia nervosa who met specific diagnostic criteria and had participated in a well-documented hospital treatment study. Information was obtained on 100% of the subjects. A comprehensive assessment was made in 93% of the living subjects in specific categories of weight, eating and weight control behaviours, menstrual function, anorexic attitudes, and psychological, sexual, social and vocational adjustment. Five subjects had died, which gives a crude mortality r

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3

Singh, Aatma, Kiran Bains, and Harpreet Kaur. "Progression of anorexia nervosa: An insight into neurological and biological mechanisms influencing the personality patterns of anorexics." Journal of Applied and Natural Science 13, no. 2 (2021): 571–84. http://dx.doi.org/10.31018/jans.v13i2.2495.

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Anorexia nervosa has emerged as a prominent eating disorder affecting young women. This disorder's fundamental characteristic is an abnormally low weight achieved by severe calorie restriction and refusal to maintain body weight at or above the minimally normal weight for age and height. It is a complex disorder with its origins still not explicitly defined. In anorexic individuals, an imbalance in the molecular signalling and hypothalamic neuropeptides is believed to be significantly responsible for alterations in the biological mechanisms associated with body weight, appetite and energy home

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4

Ehrlich, Stefan, Harriet Salbach-Andrae, Deike Weiss, et al. "S100B in underweight and weight-recovered patients with anorexia nervosa." Psychoneuroendocrinology 33, no. 6 (2008): 782–88. http://dx.doi.org/10.1016/j.psyneuen.2008.03.004.

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5

Schneider, N., H. Salbach-Andrae, J. V. Merle, et al. "Psychopathology in underweight and weight-recovered females with anorexia nervosa." Eating and Weight Disorders - Studies on Anorexia, Bulimia and Obesity 14, no. 4 (2009): e205-e211. http://dx.doi.org/10.1007/bf03325118.

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6

Ehrlich, Stefan, Leonora Franke, Nora Schneider, et al. "Aromatic amino acids in weight-recovered females with anorexia nervosa." International Journal of Eating Disorders 42, no. 2 (2009): 166–72. http://dx.doi.org/10.1002/eat.20575.

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7

Hay, Phillipa J., Anne Hall, John W. Delahunt, Guy Harper, Alex W. Mitchell, and Clare Salmond. "Investigation of Osteopaenia in Anorexia Nervosa." Australian & New Zealand Journal of Psychiatry 23, no. 2 (1989): 261–68. http://dx.doi.org/10.3109/00048678909062144.

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Sixty-nine female patients, mean age 27.5 years (range 20-40), with a past or current history of anorexia nervosa (DSM Ill-R) had spinal trabecular bone density assessed by single energy quantitative CT scan. Current exercise and dietary calcium levels were assessed by detailed questionnaires and categorized. A semi-structured interview was used to record weight, menstruation, exercise and dietary calcium intake histories from early adolescence. Serum sex hormones and total calcium assays were measured. Bone density was significantly lower in the patients compared to 31 controls. Bone density

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8

Ward, Anne, Nigel Brown, Stafford Lightman, Iain C. Campbell, and Janet Treasure. "Neuroendocrine, appetitive and behavioural responses to d-fenfluramine in women recovered from anorexia nervosa." British Journal of Psychiatry 172, no. 4 (1998): 351–58. http://dx.doi.org/10.1192/bjp.172.4.351.

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BackgroundIncreased central serotonin (5-HT) function has been hypothesised to be a vulnerability trait in anorexia nervosa.MethodsEighteen women with a history of DSM–III–R anorexia nervosa and 18 female controls were examined. The subjects had recovered weight and menstrual function. A placebo-controlled d-fenfluramine test was used. Subjects ingested d-fenfluramine or placebo and after three hours were offered a ‘free’ meal. The amounts eaten were recorded and plasma Cortisol and prolactin levels were measured. Questionnaires related to eating attitudes and behaviour, to personality, and to

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9

Windauer, Ulricke, Pierre J. V. Beumont, W. Lennerts, Peter Talbot, and Stephen W. Touyz. "How Well are ‘Cured’ Anorexia Nervosa Patients?" British Journal of Psychiatry 163, no. 2 (1993): 195–200. http://dx.doi.org/10.1192/bjp.163.2.195.

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Eating behaviour, nutritional intake and psychopathology were examined in 16 weight-recovered anorexia nervosa patients. The Eating Disorders Examination (EDE) and a food diary were the main outcome measures, but body fat content and psychosocial adjustment were also assessed. Although body fat and overall psychosocial adjustment had returned to normal in most patients, 12 still had a restricted eating pattern with nutritional intake below 90% of their energy requirements. These results suggest that weight, menstruation, and psychosocial criteria are not sufficient to determine full recovery f

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10

Goldsmith, P. J., and B. Decadt. "Extreme Achalasia Presenting as Anorexia Nervosa." Case Reports in Surgery 2012 (2012): 1–2. http://dx.doi.org/10.1155/2012/985454.

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Background. Achalasia may lead to cachexia if not diagnosed in an early stage. Surgery in cachectic patients is hazardous and complications may result in a protracted recovery or even death. Different treatment options have been described. In this paper, we report a stepwise surgical laparoscopic approach which appears to be safe and effective.Methods. Over a one-year period, a patient with a body mass index (BMI) below 17 being treated for anorexia nervosa was referred with dysphagia. Because of the extreme cachexia, a laparoscopic feeding jejunostomy (LFJ) was fashioned to enable long-term h

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11

Castro-Fornieles, Josefina, Ana Isabel Garcia, Luisa Lazaro, et al. "Prefrontal brain metabolites in short-term weight-recovered adolescent anorexia nervosa patients." Progress in Neuro-Psychopharmacology and Biological Psychiatry 34, no. 6 (2010): 1049–53. http://dx.doi.org/10.1016/j.pnpbp.2010.05.020.

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12

Tam, Friederike I., Julius Steding, Jonas L. Steinhäuser, et al. "Hair endocannabinoid concentrations in individuals with acute and weight-recovered anorexia nervosa." Progress in Neuro-Psychopharmacology and Biological Psychiatry 107 (April 2021): 110243. http://dx.doi.org/10.1016/j.pnpbp.2021.110243.

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13

Tam, Friederike I., Maria Seidel, Ilka Boehm, et al. "Peptide YY3–36 concentration in acute- and long-term recovered anorexia nervosa." European Journal of Nutrition 59, no. 8 (2020): 3791–99. http://dx.doi.org/10.1007/s00394-020-02210-7.

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Abstract Purpose The gut–brain axis could be a possible key factor in the pathophysiology of anorexia nervosa. The neuropeptide peptide YY3–36, secreted by endocrine L cells of the gastrointestinal tract, is a known regulator of appetite and food intake. The objective of this study was to investigate peptide YY3–36 plasma concentrations at different stages of anorexia nervosa in a combined cross-sectional and longitudinal design to differentiate between effects of acute undernutrition and more enduring characteristics. Methods We measured fasting plasma peptide YY3–36 concentrations in young p

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Cetkovic, Aleksandar, Marina Djurovic, and Natasa Milic. "Leptin and inhibin B as predictors of reproductive recovery in patients with anorexia nervosa during weight gain." Srpski arhiv za celokupno lekarstvo 134, no. 11-12 (2006): 492–97. http://dx.doi.org/10.2298/sarh0612492c.

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Introduction: Anorexia nervosa represents an eating disorder that is associated with substantial psychological, social and physiological abnormalities, involving 0.5-2% of female population. Objective: The secretion patterns of inhibin B, as marker of gonadal activity, and leptin, as an indicator of energy balance and body composition, were analyzed in our cross-sectional study in order to asses the restoration of reproductive function in patients with anorexia nervosa (AN) during gaining of normal weight. Method: The study included 20 patients with low weight AN (BMI 14.3?0.3 kg/mI), 22 parti

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15

Miller, Karen K., Ellen E. Lee, Elizabeth A. Lawson, et al. "Determinants of Skeletal Loss and Recovery in Anorexia Nervosa." Journal of Clinical Endocrinology & Metabolism 91, no. 8 (2006): 2931–37. http://dx.doi.org/10.1210/jc.2005-2818.

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Abstract Context: Anorexia nervosa (AN) is complicated by severe bone loss. The effects of persistent undernutrition and consequent neuroendocrine dysfunction on bone mass and the factors influencing skeletal recovery have not been well characterized. Objective: The objective of the study was to determine the rate of bone loss at the spine and hip in women with AN and whether resumption of menstrual function and/or improvement in weight are determinants of skeletal recovery in AN. Design: The study had a longitudinal design. Setting: The study was conducted at a clinical research center. Study

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Bosanac, Peter, Simone Kurlender, Lillian Stojanovska, et al. "Neuropsychological study of underweight and “weight-recovered” anorexia nervosa compared with bulimia nervosa and normal controls." International Journal of Eating Disorders 40, no. 7 (2007): 613–21. http://dx.doi.org/10.1002/eat.20412.

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17

Ritschel, F., J. A. King, D. Geisler, et al. "Temporal delay discounting in acutely ill and weight-recovered patients with anorexia nervosa." Psychological Medicine 45, no. 6 (2015): 1229–39. http://dx.doi.org/10.1017/s0033291714002311.

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Background.Patients with anorexia nervosa (AN) are characterized by a very low body weight but readily give up immediate rewards (food) for long-term goals (slim figure), which might indicate an unusual level of self-control. This everyday clinical observation may be quantifiable in the framework of the anticipation-discounting dilemma.Method.Using a cross-sectional design, this study compared the capacity to delay reward in 34 patients suffering from acute AN (acAN), 33 weight-recovered AN patients (recAN) and 54 healthy controls. We also used a longitudinal study to reassess 21 acAN patients

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18

Seidel, Maria, Joseph A. King, Franziska Ritschel, et al. "Serum visfatin concentration in acutely ill and weight-recovered patients with anorexia nervosa." Psychoneuroendocrinology 53 (March 2015): 127–35. http://dx.doi.org/10.1016/j.psyneuen.2014.12.010.

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19

Ehrlich, S., L. Franke, R. Schott, et al. "Platelet Monoamine Oxidase Activity in Underweight and Weight-Recovered Females with Anorexia Nervosa." Pharmacopsychiatry 41, no. 06 (2008): 226–31. http://dx.doi.org/10.1055/s-2008-1078749.

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20

Germain, Natacha, Anaïs Fauconnier, Jean-Philippe Klein, et al. "Pulsatile gonadotropin-releasing hormone therapy in persistent amenorrheic weight-recovered anorexia nervosa patients." Fertility and Sterility 107, no. 2 (2017): 502–9. http://dx.doi.org/10.1016/j.fertnstert.2016.10.032.

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21

Zwipp, Johannes, Johanna Hass, Ilka Schober, et al. "Serum brain-derived neurotrophic factor and cognitive functioning in underweight, weight-recovered and partially weight-recovered females with anorexia nervosa." Progress in Neuro-Psychopharmacology and Biological Psychiatry 54 (October 2014): 163–69. http://dx.doi.org/10.1016/j.pnpbp.2014.05.006.

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22

Bryan, Kadi-Ann, Terrill Bravender, and H. Ryan Wagner. "49: Weight Gain Trajectory in Adolescents with Anorexia Nervosa: A Comparison of Recovered and Non-Recovered Outpatients." Journal of Adolescent Health 42, no. 2 (2008): 36–37. http://dx.doi.org/10.1016/j.jadohealth.2007.11.097.

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23

Seitz, Jochen, Katharina Bühren, Georg G. von Polier, Nicole Heussen, Beate Herpertz-Dahlmann, and Kerstin Konrad. "Morphological Changes in the Brain of Acutely Ill and Weight-Recovered Patients with Anorexia Nervosa." Zeitschrift für Kinder- und Jugendpsychiatrie und Psychotherapie 42, no. 1 (2014): 7–18. http://dx.doi.org/10.1024/1422-4917/a000265.

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Objective: Acute anorexia nervosa (AN) leads to reduced gray (GM) and white matter (WM) volume in the brain, which however improves again upon restoration of weight. Yet little is known about the extent and clinical correlates of these brain changes, nor do we know much about the time-course and completeness of their recovery. Methods: We conducted a meta-analysis and a qualitative review of all magnetic resonance imaging studies involving volume analyses of the brain in both acute and recovered AN. Results: We identified structural neuroimaging studies with a total of 214 acute AN patients an

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24

Giel, Katrin E., Annette Conzelmann, Tobias J. Renner, et al. "Attention allocation to illness‐compatible information discriminates women with active versus weight‐recovered anorexia nervosa." International Journal of Eating Disorders 53, no. 8 (2019): 1270–79. http://dx.doi.org/10.1002/eat.23209.

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25

Ramoz, N. "Is there epigenetics in anorexia nervosa?" European Psychiatry 28, S2 (2013): 21. http://dx.doi.org/10.1016/j.eurpsy.2013.09.050.

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Anorexia Nervosa (AN) is a young-onset psychiatric illness, for which the etiology remains unknown and presents a high heritability. Thus, the genetic component is estimated to be 70%. To identify the vulnerability genes to AN, different approaches of molecular genetic are performed, including linkage analysis, the candidate gene association study and, the Genome-Wide Association Study (GWAS). Some polymorphisms of candidate genes, such as the BDNF gene that encodes for the brain-derived neurotrophic factor, were found associated with AN in several studies. In addition to the DNA polymorphisms

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26

Tyszkiewicz-Nwafor, Marta, Agnieszka Slopien, Monika Dmitrzak-Węglarz, and Filip Rybakowski. "Adiponectin and resistin in acutely ill and weight-recovered adolescent anorexia nervosa: Association with psychiatric symptoms." World Journal of Biological Psychiatry 20, no. 9 (2018): 723–31. http://dx.doi.org/10.1080/15622975.2018.1492735.

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27

Djurovic, M., S. Pekic, M. Petakov, et al. "Gonadotropin response to clomiphene and plasma leptin levels in weight recovered but amenorrhoeic patients with anorexia nervosa." Journal of Endocrinological Investigation 27, no. 6 (2004): 523–27. http://dx.doi.org/10.1007/bf03347473.

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28

Ehrlich, Stefan, Roland Burghardt, Nora Schneider, et al. "The role of leptin and cortisol in hyperactivity in patients with acute and weight-recovered anorexia nervosa." Progress in Neuro-Psychopharmacology and Biological Psychiatry 33, no. 4 (2009): 658–62. http://dx.doi.org/10.1016/j.pnpbp.2009.03.007.

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29

Steinhäuser, Jonas L., Joseph A. King, Friederike I. Tam, et al. "Is Serum BDNF Altered in Acute, Short- and Long-Term Recovered Restrictive Type Anorexia Nervosa?" Nutrients 13, no. 2 (2021): 432. http://dx.doi.org/10.3390/nu13020432.

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Brain-derived neurotrophic factor (BDNF), a neurotrophin involved in the regulation of food intake and body weight, has been implicated in the development and maintenance of Anorexia nervosa (AN). The majority of previous studies reported lower BDNF levels in acutely underweight AN patients (acAN) and increasing levels after weight rehabilitation. Here, we investigated serum BDNF concentrations in the largest known AN sample to date, both before and after weight restoration therapy. Serum BDNF was measured in 259 female volunteers: 77 in-patient acAN participants of the restrictive type (47 re

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De-Mateo-Silleras, Beatriz, Sara R. Alonso-Torre, Paz Redondo-del-Río, Khursheed Jeejeebhoy, and Alberto Miján-de-la-Torre. "Effect of nutritional support on mitochondrial complex I activity in malnourished patients with anorexia nervosa." Applied Physiology, Nutrition, and Metabolism 38, no. 11 (2013): 1093–98. http://dx.doi.org/10.1139/apnm-2013-0082.

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Previous studies have shown a reduction in lymphocyte mitochondrial complex I activity (CIA) in malnourished patients, which is restored after refeeding. Our aim was to evaluate the usefulness of CIA as an indicator of nutritional status in anorexia nervosa patients. Twelve malnourished anorexia nervosa females (mean age, 24.5 years) were admitted to the Eating Disorders Unit. Basal and weekly anthropometrics, bioelectric impedance (BIA), body composition, and CIA were performed until discharge. Patients were matched to 25 healthy females and refeeding was adjusted according to the Unit’s prot

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De Álvaro, María Teresa García, María Teresa Muñoz-Calvo, Vicente Barrios, et al. "Regional fat distribution in adolescents with anorexia nervosa: effect of duration of malnutrition and weight recovery." European Journal of Endocrinology 157, no. 4 (2007): 473–79. http://dx.doi.org/10.1530/eje-07-0459.

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Objective: This study addresses the influence of the duration of malnutrition and the effect of weight recovery on regional fat mass distribution in moderately malnourished adolescents with anorexia nervosa (AN). Study design: We measured total and regional fat mass and leptin levels in 42 restrictive AN female adolescents and 23 controls. AN patients, followed over 2 years, were divided into three groups: prolonged moderate malnutrition (PM; secondary amenorrhea for over 1 year, n = 14); SM, short period of moderate malnutrition (secondary amenorrhea for less than 1 year, n = 13); and R, reco

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Schott, R., L. Franke, R. Burghardt, et al. "Relationships between Platelet MAO-B Activity and Personality Styles in Acute and Weight-Recovered Young Patients with Anorexia Nervosa." Pharmacopsychiatry 46, no. 02 (2012): 47–53. http://dx.doi.org/10.1055/s-0032-1321869.

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33

Pellegrino, F., A. M. Monteleone, M. Nigro, et al. "Investigation of Salivary Cortisol Response to Awakening in Underweight and Weight-Restored Patients with Anorexia Nervosa." European Psychiatry 41, S1 (2017): S283. http://dx.doi.org/10.1016/j.eurpsy.2017.02.133.

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IntroductionAnorexia nervosa (AN) is characterized by dysregulated eating that leads to chronic malnutrition, which may be responsible for several physical complications, including endocrine alterations, such as hyperactivity of the hypothalamus-pituitary-adrenal (HPA) axis.ObjectivesSeveral studies have shown a dysregulation of the cortisol awakening response (CAR) in symptomatic AN patients. However, it has not been established if the deranged CAR of underweight AN patients is a primary phenomenon or an alteration secondary to malnutrition.AimsThe aim of this study was to explore the salivar

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34

Maier, Simon, Kathrin Nickel, Evgeniy Perlov, et al. "Insular Cell Integrity Markers Linked to Weight Concern in Anorexia Nervosa—An MR-Spectroscopy Study." Journal of Clinical Medicine 9, no. 5 (2020): 1292. http://dx.doi.org/10.3390/jcm9051292.

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Objective: An insular involvement in the pathogenesis of anorexia nervosa (AN) has been suggested in many structural and functional neuroimaging studies. This magnetic resonance spectroscopy (MRS) study is the first to investigate metabolic signals in the anterior insular cortex in patients with AN and recovered individuals (REC). Method: The MR spectra of 32 adult women with AN, 21 REC subjects and 33 healthy controls (HC) were quantified for absolute N-acetylaspartate (NAA), glutamate + glutamine (Glx), total choline, myo-inositol, creatine concentrations (mM/L). After adjusting the metaboli

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Platte, Petra, Karl M. Pirke, Peter Trimborn, Karin Pietsch, Jürgen C. Krieg, and Manfred M. Fichter. "Resting metabolic rate and total energy expenditure in acute and weight recovered patients with anorexia nervosa and in healthy young women." International Journal of Eating Disorders 16, no. 1 (1994): 45–52. http://dx.doi.org/10.1002/1098-108x(199407)16:1<45::aid-eat2260160104>3.0.co;2-z.

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36

Ehrlich, Stefan, Leonora Franke, Susann Scherag, et al. "The 5-HTTLPR polymorphism, platelet serotonin transporter activity and platelet serotonin content in underweight and weight-recovered females with anorexia nervosa." European Archives of Psychiatry and Clinical Neuroscience 260, no. 6 (2009): 483–90. http://dx.doi.org/10.1007/s00406-009-0092-3.

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Fico, G., A. M. Monteleone, M. Nigro, G. Patriciello, U. Volpe, and P. Monteleone. "Ghrelin response to hedonic eating in underweight and short-term weight restored patients with anorexia nervosa." European Psychiatry 41, S1 (2017): S550. http://dx.doi.org/10.1016/j.eurpsy.2017.01.779.

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IntroductionRecently, anorexia nervosa (AN) has been conceptualized as a reward-related disorder, and brain imaging studies have shown functional and structural abnormalities in areas of the brain involved in reward processes in both acute and recovered AN patients. However, the role of endogenous biochemical mediators, such as Ghrelin, in the modulation of reward processes has been poorly investigated in this eating disorder.ObjectivesHedonic eating, that is the consumption of food exclusively for pleasure and not to maintain energy homeostasis, is a useful paradigm to investigate the physiol

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Jimerson, David C., and Barbara E. Wolfe. "Neuropeptides in Eating Disorders." CNS Spectrums 9, no. 7 (2004): 516–22. http://dx.doi.org/10.1017/s1092852900009603.

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AbstractThe past decade has witnessed a dramatic acceleration in research on the role of the neuropeptides in the regulation of eating behavior and body weight homeostasis. This expanding research focus has been driven in part by increasing public health concerns related to obesity and the eating disorders anorexia nervosa (AN) and bulimia nervosa (BN). Preclinical advances have been facilitated by the development of new molecular and behavioral research methodologies. With a focus on clinical investigations in AN and BN, this article reviews research on selected hypothalamic and gut-related p

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Badr, Sammy, Isabelle Legroux-Gérot, Jean Vignau, et al. "Comparison of regional bone marrow adiposity characteristics at the hip of underweight and weight-recovered women with anorexia nervosa using magnetic resonance spectroscopy." Bone 127 (October 2019): 135–45. http://dx.doi.org/10.1016/j.bone.2019.05.033.

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40

Lawson, Elizabeth A., Laura M. Holsen, Rebecca DeSanti, et al. "Increased hypothalamic–pituitary–adrenal drive is associated with decreased appetite and hypoactivation of food-motivation neurocircuitry in anorexia nervosa." European Journal of Endocrinology 169, no. 5 (2013): 639–47. http://dx.doi.org/10.1530/eje-13-0433.

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ObjectiveCorticotrophin-releasing hormone (CRH)-mediated hypercortisolemia has been demonstrated in anorexia nervosa (AN), a psychiatric disorder characterized by food restriction despite low body weight. While CRH is anorexigenic, downstream cortisol stimulates hunger. Using a food-related functional magnetic resonance imaging (fMRI) paradigm, we have demonstrated hypoactivation of brain regions involved in food motivation in women with AN, even after weight recovery. The relationship between hypothalamic–pituitary–adrenal (HPA) axis dysregulation and appetite and the association with food-mo

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Chaer, Rayane, Nour Nakouzi, Leila Itani, et al. "Fertility and Reproduction after Recovery from Anorexia Nervosa: A Systematic Review and Meta-Analysis of Long-Term Follow-Up Studies." Diseases 8, no. 4 (2020): 46. http://dx.doi.org/10.3390/diseases8040046.

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Reproductive health is compromised during anorexia nervosa (AN). However, it is still unclear whether this medical complication is reversible after recovery from AN. The purpose of this paper was to conduct a systematic review of the major reproductive health outcomes in females after recovery from AN. The review was conducted in adherence to preferred reporting items for systematic review and meta-analyses (PRISMA) guidelines. Data were collated using meta-analysis and a narrative approach. Of the 1186 articles retrieved, five studies met the inclusion criteria and were reviewed. These studie

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Doose, Arne, Joseph A. King, Fabio Bernardoni, et al. "Strengthened Default Mode Network Activation During Delay Discounting in Adolescents with Anorexia Nervosa After Partial Weight Restoration: A Longitudinal fMRI Study." Journal of Clinical Medicine 9, no. 4 (2020): 900. http://dx.doi.org/10.3390/jcm9040900.

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The capacity of patients with anorexia nervosa (AN) to resist food-based rewards is often assumed to reflect excessive self-control. Previous cross-sectional functional magnetic resonance imaging (fMRI) studies utilizing the delay discounting (DD) paradigm, an index of impulsivity and self-control, suggested altered neural efficiency of decision-making in acutely underweight patients (acAN) and a relative normalization in long-term, weight-recovered individuals with a history of AN (recAN). The current longitudinal study tested for changes in functional magnetic resonance imaging (fMRI) activa

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Föcker, M., N. Timmesfeld, S. Scherag, et al. "Comparison of metabolic profiles of acutely ill and short-term weight recovered patients with anorexia nervosa reveals alterations of 33 out of 163 metabolites." Journal of Psychiatric Research 46, no. 12 (2012): 1600–1609. http://dx.doi.org/10.1016/j.jpsychires.2012.08.015.

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44

Duriez, Philibert, Ida A. K. Nilsson, Ophelia Le Thuc, et al. "Exploring the Mechanisms of Recovery in Anorexia Nervosa through a Translational Approach: From Original Ecological Measurements in Human to Brain Tissue Analyses in Mice." Nutrients 13, no. 8 (2021): 2786. http://dx.doi.org/10.3390/nu13082786.

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Anorexia nervosa (AN) is a severe eating disorder where caloric restriction, excessive physical activity and metabolic alterations lead to life-threatening situations. Despite weight restoration after treatment, a significant part of patients experience relapses. In this translational study, we combined clinical and preclinical approaches. We describe preliminary data about the effect of weight gain on the symptomatology of patients suffering from acute AN (n = 225) and partially recovered (n = 41). We measured more precisely physical activity with continuous cardiac monitoring in a sub-group

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Bredella, Miriam A., Pouneh K. Fazeli, Lauren M. Freedman, et al. "Young Women with Cold-Activated Brown Adipose Tissue Have Higher Bone Mineral Density and Lower Pref-1 than Women without Brown Adipose Tissue: A Study in Women with Anorexia Nervosa, Women Recovered from Anorexia Nervosa, and Normal-Weight Women." Journal of Clinical Endocrinology & Metabolism 97, no. 4 (2012): E584—E590. http://dx.doi.org/10.1210/jc.2011-2246.

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Holsen, Laura M., Elizabeth A. Lawson, Kara Christensen, Anne Klibanski, and Jill M. Goldstein. "Abnormal relationships between the neural response to high- and low-calorie foods and endogenous acylated ghrelin in women with active and weight-recovered anorexia nervosa." Psychiatry Research: Neuroimaging 223, no. 2 (2014): 94–103. http://dx.doi.org/10.1016/j.pscychresns.2014.04.015.

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Ricca, V., G. Castellini, L. Lelli, F. Rotella, A. M. Monteleone, and M. Maggi. "Eating Disorders and Sexuality: A Complex Relationship." European Psychiatry 41, S1 (2017): S36—S37. http://dx.doi.org/10.1016/j.eurpsy.2017.01.170.

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IntroductionThe relationships between Eating Disorders (EDs) and sexuality are complex, and of interest for researchers and clinicians.ObjectiveTo identify psychopathological and clinical factors associated with restoration of regular menses and sexual function in EDs patients.AimsTo evaluate the role of sexuality as a moderator of the recovery process after an individual Cognitive Behavioural Therapy (CBT).Methods39 Anorexia Nervosa (AN) and 40 Bulimia Nervosa (BN) female patients were evaluated by means of a face-to-face interview, self-reported questionnaires, including Eating Disorder Exam

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Abraham, Suzanne F., Tani Brown, Catherine Boyd, Georgina Luscombe, and Janice Russell. "Quality of Life: Eating Disorders." Australian & New Zealand Journal of Psychiatry 40, no. 2 (2006): 150–55. http://dx.doi.org/10.1080/j.1440-1614.2006.01762.x.

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Objective: There is a lack of measurements with predictive validity that are specific for quality of life (QOL) in patients with eating disorders. Method: A total of 306 eating disorder patients treated as inpatients completed the Quality of Life for Eating Disorders (QOL ED): 109 at both admission and discharge from hospital, 65 at both admission and after 12months. Patients also completed well-validated measures of eating disorders, psychological dysfunction and general physical and mental QOL. QOL ED consists of 20 self-report questions that provide scores for the domains of behaviour, eati

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Lautenbacher, Stefan, Nicoline Kraehe, and Jürgen-Christian Krieg. "Perception of Body Size and Body Satisfaction in Recovered Anorexic Women: Comparison with Restrained and Unrestrained Eaters." Perceptual and Motor Skills 84, no. 3_suppl (1997): 1331–42. http://dx.doi.org/10.2466/pms.1997.84.3c.1331.

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The perception of body size, measured by three different methods, and body satisfaction were assessed in 23 formerly anorexic inpatients with an “intermediate” ( n = 9) or a “good” outcome ( n = 14) and compared with the data obtained from 21 restrained and 20 unrestrained eaters. Using the Kinaesthetic Size Estimation Apparatus, overestimation and uncertainty in the perception of body size became apparent in both groups of former patients. The other two methods, Video Distortion Technique and Image Marking Procedure, did not produce comparable results. There was only a trend towards higher sc

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Brodrick, Brooks B., Adrienne L. Adler-Neal, Jayme M. Palka, Virendra Mishra, Sina Aslan, and Carrie J. McAdams. "Structural brain differences in recovering and weight-recovered adult outpatient women with anorexia nervosa." Journal of Eating Disorders 9, no. 1 (2021). http://dx.doi.org/10.1186/s40337-021-00466-w.

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Abstract Background Anorexia nervosa is a complex psychiatric illness that includes severe low body weight with cognitive distortions and altered eating behaviors. Brain structures, including cortical thicknesses in many regions, are reduced in underweight patients who are acutely ill with anorexia nervosa. However, few studies have examined adult outpatients in the process of recovering from anorexia nervosa. Evaluating neurobiological problems at different physiological stages of anorexia nervosa may facilitate our understanding of the recovery process. Methods Magnetic resonance imaging (MR

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