Relevant bibliographies by topics / Wheat powdery mildew disease – Genetic aspects

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Academic literature on the topic 'Wheat powdery mildew disease – Genetic aspects'

Author: Grafiati
Published: 4 June 2021
Last updated: 10 February 2022

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Journal articles on the topic "Wheat powdery mildew disease – Genetic aspects"

1

Collins, N. C., R. E. Niks, and P. Schulze-Lefert. "Resistance to cereal rusts at the plant cell wall—what can we learn from other host-pathogen systems?" Australian Journal of Agricultural Research 58, no. 6 (2007): 476. http://dx.doi.org/10.1071/ar06065.

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The ability of plant cells to resist invasion by pathogenic fungi at the cell periphery (pre-invasion resistance) differs from other types of resistance that are generally triggered after parasite entry and during differentiation of specialised intracellular feeding structures. Genetic sources of pre-invasion resistance such as mlo for barley powdery mildew and Lr34 for resistance to wheat leaf rust have proven to be broad-spectrum in effect and durable in the field. Continued breeding for this type of resistance (often quantitative in effect) is therefore considered an important strategy to protect cereal crops long-term against potentially devastating fungal diseases such as rusts. Considerable progress has been made in characterising genes and processes underlying pre-invasion resistance using mutant analysis, molecular genetics, gene cloning, and the model plant Arabidopsis, as well as comparative functional analysis of genes in Arabidopsis and cereals. This review summarises the current knowledge in this field, and discusses several aspects of pre-invasion resistance potentially pertinent to use in breeding; namely, biological cost of the resistance and effectiveness of individual resistance genes against multiple pathogen types. We show that mutations in Mlo, Ror1, and Ror2 genes known to affect powdery mildew pre-invasion resistance have no detectable effect on partial resistance to barley leaf rust as measured by latency period.
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2

Lillemo, M., H. Skinnes, R. P. Singh, and M. van Ginkel. "Genetic Analysis of Partial Resistance to Powdery Mildew in Bread Wheat Line Saar." Plant Disease 90, no. 2 (February 2006): 225–28. http://dx.doi.org/10.1094/pd-90-0225.

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Powdery mildew, caused by Blumeria graminis (syn. Erysiphe graminis) f. sp. tritici, is an important disease of bread wheat (Triticum aestivum) in many countries. The CIMMYT bread wheat line Saar has exhibited a high level of partial resistance to powdery mildew in field trials conducted in Europe, Asia, and South America, and represents a valuable source of resistance in wheat breeding. A set of 114 random F5 inbred lines from the cross Saar × Avocet-YrA (susceptible) were evaluated in replicated field trials at two locations in southeastern Norway to determine the number of genes involved in partial resistance to powdery mildew. Narrow-sense heritability estimates were high (0.83 to 0.92). Based on both quantitative and qualitative genetic analyses, the minimum number of genes with additive effects segregating for powdery mildew resistance in the population was four. Transgressive segregation indicated that Avocet-YrA might have contributed one minor gene for resistance. It is concluded that partial resistance to powdery mildew in Saar is controlled by at least three genes. Such resistance conferred by multiple genes having additive effects is expected to be durable.
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3

Bliffeld, M., J. Mundy, I. Potrykus, and J. Fütterer. "Genetic engineering of wheat for increased resistance to powdery mildew disease." Theoretical and Applied Genetics 98, no. 6-7 (May 1999): 1079–86. http://dx.doi.org/10.1007/s001220051170.

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4

Zhou, Qingping, Lingli Li, and Long Wang. "Complex System Analysis of Blumeria graminis f.sp. tritici Isolates Collected in Central Hebei Province, China." Discrete Dynamics in Nature and Society 2021 (April 10, 2021): 1–8. http://dx.doi.org/10.1155/2021/6693759.

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Wheat powdery mildew (WPM), caused by Blumeria graminis f.sp. tritici, is a significant disease of wheat throughout the world and has resulted in substantial yield and economic losses in wheat production. It is particularly important to understand the population distribution and genetic resistance of B. graminis f.sp. tritici. In 2019, the cumulative incidence of wheat powdery mildew in China was nearly 8.7 million hm2, which seriously affected the safe production of wheat in China. However, the proportion of disease-resistant wheat varieties in actual production was relatively low, and effective disease-resistant genes were lacking. As one of the main wheat-producing provinces in China, it is of great significance for normal wheat production to understand powdery mildew resistance in Hebei province. In this study, using wheat seedling culture in vitro, the physiological races of wheat powdery mildew in central Hebei province were identified, and the population toxicity frequency was analyzed. The results were as follows: (1) 36 strains were purified and 20 physiological races were identified. Among them, the dominant race is 015, and the distribution frequency is 16.7%. Race 077 is the second dominant race. (2) The frequency of virulence genes VEra, V8, V1, V3c, and V3f in population toxicity frequency analysis was more than 70%, while the frequency of virulence genes V2, MID, V20, V21, V4b, and V4 was less than 16.7%, and 46% of virulence genes of powdery mildew were higher than 40%. It shows that the virulence gene frequency of powdery mildew in Hebei province is high, and the varieties containing Pm2 + MID, Pm20, Pm21, Pm1b, Pm1, and other disease resistance genes have a certain value inbreeding.
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5

Liang, S. S., K. Suenaga, Z. H. He, Z. L. Wang, H. Y. Liu, D. S. Wang, R. P. Singh, P. Sourdille, and X. C. Xia. "Quantitative Trait Loci Mapping for Adult-Plant Resistance to Powdery Mildew in Bread Wheat." Phytopathology® 96, no. 7 (July 2006): 784–89. http://dx.doi.org/10.1094/phyto-96-0784.

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Powdery mildew, caused by Blumeria graminis f. sp. tritici, is a major disease to wheat (Triticum aestivum) worldwide. Use of adult-plant resistance (APR) is an effective method to develop wheat cultivars with durable resistance to powdery mildew. In the present study, 432 molecular markers were used to map quantitative trait loci (QTL) for APR to powdery mildew in a doubled haploid (DH) population with 107 lines derived from the cross Fukuho-komugi × Oligoculm. Field trials were conducted in Beijing and Anyang, China during 2003-2004 and 2004-2005 cropping seasons, respectively. The DH lines were planted in a randomized complete block design with three replicates. Artificial inoculation was carried out in Beijing with highly virulent isolate E20 of B. graminis f. sp. tritici and the powdery mildew severity on penultimate leaf was evaluated four times, and the maximum disease severity (MDS) on penultimate leaf was investigated in Anyang under natural inoculation in May 2004 and 2005. The heritability of resistance to powdery mildew for MDS in 2 years and two locations ranged from 0.82 to 0.93, while the heritability for area under the disease progress curve was between 0.84 and 0.91. With the method of composite interval mapping, four QTL for APR to powdery mildew were detected on chromosomes 1AS, 2BL, 4BL, and 7DS, explaining 5.7 to 26.6% of the phenotypic variance. Three QTL on chromosomes 1AS, 2BL, and 7DS were derived from the female, Fukuho-komugi, while the one on chromosome 4BL was from the male, Oligoculm. The QTL on chromosome 1AS showed high genetic effect on powdery mildew resistance, accounting for 19.5 to 26.6% of phenotypic variance across two environments. The QTL on 7DS associated with the locus Lr34/Yr18, flanked by microsatellite Xgwm295.1 and Ltn (leaf tip necrosis). These results will benefit for improving powdery mildew resistance in wheat breeding programs.
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Wang, Z. L., L. H. Li, Z. H. He, X. Y. Duan, Y. L. Zhou, X. M. Chen, M. Lillemo, R. P. Singh, H. Wang, and X. C. Xia. "Seedling and Adult Plant Resistance to Powdery Mildew in Chinese Bread Wheat Cultivars and Lines." Plant Disease 89, no. 5 (May 2005): 457–63. http://dx.doi.org/10.1094/pd-89-0457.

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Powdery mildew, caused by Blumeria graminis f. sp. tritici, is a widespread wheat disease in China. Identification of race-specific genes and adult plant resistance (APR) is of major importance in breeding for an efficient genetic control strategy. The objectives of this study were to (i) identify genes that confer seedling resistance to powdery mildew in Chinese bread wheat cultivars and introductions used by breeding programs in China and (ii) evaluate their APR in the field. The results showed that (i) 98 of 192 tested wheat cultivars and lines appear to have one or more resistance genes to powdery mildew; (ii) Pm8 and Pm4b are the most common resistance genes in Chinese wheat cultivars, whereas Pm8 and Pm3d are present most frequently in wheat cultivars introduced from CIMMYT, the United States, and European countries; (iii) genotypes carrying Pm1, Pm3e, Pm5, and Pm7 were susceptible, whereas those carrying Pm12, Pm16, and Pm20 were highly resistant to almost all isolates of B. graminis f. sp. tritici tested; and (iv) 22 genotypes expressed APR. Our data showed that the area under the disease progress curve, maximum disease severity on the penultimate leaf, and the disease index are good indicators of the degree of APR in the field. It may be a good choice to combine major resistance genes and APR genes in wheat breeding to obtain effective resistance to powdery mildew.
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Weidner, Annette, Marion S. Röder, and Andreas Börner. "Mapping wheat powdery mildew resistance derived from Aegilops markgrafii." Plant Genetic Resources 10, no. 2 (May 14, 2012): 137–40. http://dx.doi.org/10.1017/s1479262112000123.

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The pattern of inheritance of powdery mildew resistance expressed by two bread wheat (cultivar ‘Alcedo’)/Aegilops markgrafii introgression lines was explored using F2 populations bred from crosses made with the powdery mildew-susceptible cultivar ‘Kanzler’. Disease reaction was tested at both seedling and adult plant stages. Two resistance loci, designated QPm.ipk-1A and QPm.ipk-7A, were identified as mapping to the distal ends of chromosome arms 1AS and 7AL, respectively. Whereas QPm.ipk-1A was expressed throughout the plant's life, QPm.ipk-7A was only effective at the seedling stage. The map location of both resistance loci indicated that resistances originated from A. markgrafii. The possible genetic relationship of these disease-resistant genes to known Pm genes is discussed in the context of synteny.
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8

Hartl, Lorenz, Volker Mohler, Friedrich J. Zeller, Sai LK Hsam, and Günther Schweizer. "Identification of AFLP markers closely linked to the powdery mildew resistance genes Pm1c and Pm4a in common wheat (Triticum aestivum L.)." Genome 42, no. 2 (April 1, 1999): 322–29. http://dx.doi.org/10.1139/g98-129.

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A total of 7654 DNA fragments were screened for linkage to wheat powdery mildew resistance gene Pm1c employing fluorescently based AFLP analysis and phenotypic pools from F3 families. F3 and derived F4 families were used for segregation analysis. Pool screening revealed several cosegregating and tightly linked (0.9 cM) AFLP markers for the Pm1c resistance gene. The previously reported RFLP locus Xwhs178 was integrated into the AFLP map in the vicinity of Pm1c. One AFLP marker, 18M2, was determined to be highly specific for the Pm1c gene in diverse genetic backgrounds. As Pm1c allele confers an effective resistance to powdery mildew, the marker 18M2 provides a valuable tool for enhancing marker assisted selection and pyramiding of powdery mildew resistance genes in wheat.Key words: Triticum aestivum, powdery mildew, disease resistance, AFLP, bulked segregant analysis
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9

Xu, Xiaodan, Wei Liu, Zhiyong Liu, Jieru Fan, and Yilin Zhou. "Mapping Powdery Mildew Resistance Gene pmYBL on Chromosome 7B of Chinese Wheat (Triticum aestivum L.) Landrace Youbailan." Plant Disease 104, no. 9 (September 2020): 2411–17. http://dx.doi.org/10.1094/pdis-01-20-0118-re.

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Chinese wheat landrace Youbailan has excellent resistance to powdery mildew caused by Blumeria graminis f. sp. tritici. In the present study, genetic analysis indicated that a recessive gene, tentatively designated pmYBL, was responsible for the powdery mildew resistance of Youbailan. pmYBL was located in the 695-to-715-Mb genomic region of chromosome 7BL, with 19 gene-linked single-nucleotide polymorphism (SNP) markers. It was flanked by SNP1-12 and SNP1-2 with genetic distances of 0.6 and 1.8 centimorgans, respectively. The disease reaction patterns of Youbailan and four cultivars (lines) carrying the powdery mildew (Pm) genes located on chromosome arm 7BL indicated that pmYBL may be allelic or closely linked to these genes. All of the SNP markers linked to pmYBL were diagnostic, indicating that these markers will be useful for pyramiding pmYBL using marker-assisted selection.
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10

Gupta, Vikas, R. Selvakumar, Satish Kumar, C. N. Mishra, V. Tiwari, and Indu Sharma. "Evaluation and identification of resistance to powdery mildew in Indian wheat varieties under artificially created epiphytotic." Journal of Applied and Natural Science 8, no. 2 (June 1, 2016): 565–69. http://dx.doi.org/10.31018/jans.v8i2.837.

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Wheat production is globally weighed down by several biotic factors of which rusts and powdery mildew are the most important. Powdery mildew, caused by Blumeria graminis f. sp. tritici, is becoming a disease of major importance in the North Western Plains Zone and Northern Hills Zone of the country. In the present context ofclimate variability, diseases like powdery mildew can assume greater importance in wheat breeding programs. Importance of basic studies on powdery mildew is the need of hour. A set of 370 Indian bread wheat, durum, dicoccum and triticale varieties were screened using mixture of natural occurring pathotypes from four locations(viz., Karnal, Ludhiana, Dhaulakuan and Yamunanagar) under polyhouse conditions. Data were recorded on the severity of infection based on 0-9 scale. Out of 370, only 23 varieties (Amrut, DDK 1025, DWR 1006, DWR 195, GW 1139, HD 4672, HD 4530, HD 2278, HD 1981, DDK 1001, HI 8627, Jay, TL 2942, DT 46, K 8020, DDK 1029, K 9107, K 816, Lok 1, MACS 6145, DDK 1009, NP 111 and NP 200) had shown immune reaction (0) whereas 150, 83 and 114 varieties have shown resistance (1-3), moderately susceptible (4-6) and highly susceptible (>6) response respectively against powdery mildew. Data indicated that there is an urgent need to broaden the genetic base of wheat by identifying and introgressing new sources of powdery mildew resistance. With limited sources of PM resistance available, above identified genotypes can be further used and characterized for resistance breeding programs in India.
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Dissertations / Theses on the topic "Wheat powdery mildew disease – Genetic aspects"

1

Chung, Young-Soo. "Inheritance of powdery mildew resistance genes in 10 winter wheat lines." Diss., Virginia Tech, 1994. http://hdl.handle.net/10919/38655.

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2

Hossain, Mohammad Abul. "Powdery mildew on barley : pathogen variability in South Australia : resistance genes in cv. Galleon /." Title page, contents and abstract only, 1986. http://web4.library.adelaide.edu.au/theses/09PH/09phh8287.pdf.

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Books on the topic "Wheat powdery mildew disease – Genetic aspects"

1

Forsström, Per-Olov. Broadening of mildew resistance in wheat. Alnarp: Swedish University of Agricultural Sciences, 2002.

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