Relevant bibliographies by topics / Wnt/Bcatenin pathway

Contents

  1. Journal articles
  2. Dissertations / Theses

Academic literature on the topic 'Wnt/Bcatenin pathway'

Author: Grafiati
Published: 1 April 2023
Last updated: 31 July 2025

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Wnt/Bcatenin pathway.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Wnt/Bcatenin pathway"

1

Chalamalasetty, Ravindra B., William C. Dunty,, Kristin K. Biris, et al. "The Wnt/bcatenin target, Mesogenin1 (Msgn1), directly regulates the Notch pathway during mammalian somitogensis." Developmental Biology 356, no. 1 (2011): 259. http://dx.doi.org/10.1016/j.ydbio.2011.05.493.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Fuchs, Quentin, Stephanie Legras, Benoit Lhermitte, et al. "DIPG-30. HIF2 OVEREXPRESSION DICTATES AGGRESSIVENESS ADDITIONALLY TO TP53 MUTATIONS IN H3.3K27M PEDIATRIC HIGH-GRADE GLIOMAS THROUGH RAS-MAPK AND WNT/BCATENIN PATHWAYS." Neuro-Oncology 25, Supplement_1 (2023): i19—i20. http://dx.doi.org/10.1093/neuonc/noad073.077.

Full text
Abstract:
Abstract Diffuse midline (DMG) and brainstem (DIPG) high-grade gliomas (HGG) bearing H3.3K27M driver mutation are aggressive and uncurable brain tumors. We evidenced by the past HIF-2a and its transcriptional gene, EPAS1, as a marker of resistance when targeting mTor/HIF-1a pathway. To understand HIF-2α potential role in chronically active hypoxic environment in DIPG/DMG, we investigate its status in a cohort of pediatric HGGs comprising notably H3.3K27M mutant tumors and in their paired in vitro derived models. Using concomitantly the low-input chromatin immunoprecipitation sequencing method
APA, Harvard, Vancouver, ISO, and other styles
3

Obrador-Hevia, A., F. Chin, S. Gonzalez, et al. "Wnt signaling somatic alterations in apc-associated fap adenomas." Journal of Clinical Oncology 27, no. 15_suppl (2009): e22046-e22046. http://dx.doi.org/10.1200/jco.2009.27.15_suppl.e22046.

Full text
Abstract:
e22046 Background: APC mutations are believed to constitutively activate the wnt pathway in colorectal tumors. Our goal was to comprehensively characterize Wnt signaling components in a set of APC-associated FAP tumors both at the DNA and RNA levels. Methods: Sixty adenomas from FAP cases harboring pathogenic APC mutations were included (10 adenomas and 1 normal mucosa per case). Somatic APC and KRAS alterations, β-catenin immunostaining and qRT-PCR of APC, MYC, AXIN2 and SFRP1 were analyzed. aCGH was also assessed in 26 FAP adenomas and 24 additional paired normal-adenoma-carcinoma samples. R
APA, Harvard, Vancouver, ISO, and other styles
4

Chung, Jihyun, Vrajesh Karkhanis, Said Sif та Robert A. Baiocchi. "Protein Arginine Methyltransferase 5 Supports MYC, Survivin and Cyclin D1 Activity in Aggressive Lymphomas By Regulating the WNT/β-Catenin Pathway". Blood 124, № 21 (2014): 58. http://dx.doi.org/10.1182/blood.v124.21.58.58.

Full text
Abstract:
Abstract Introduction: Aggressive histologic subtypes of lymphoma such as mantle cell (MCL) and activated B cell (ABC) are considered incurable and affected patients often have a short median survival despite multimodal therapy. It is well established that altered expression of oncogenes and epigenetic dysregulation of tumor suppressor and regulatory genes promote cellular transformation of normal B cells into malignant lymphoma. Hypermethylation of histone proteins (H3R8 and H4R3) by the protein arginine methyltransferase 5 (PRMT5) enzyme has been documented in multiple cancer types and has b
APA, Harvard, Vancouver, ISO, and other styles
5

Teofili, Luciana, Maurizio Martini, Eugenia Rosa Nuzzolo, et al. "Defective WNT Signaling and Genetic Profile Of Endothelial Cells In Patients With Low Risk Myelodysplastic Syndromes Suggest a Contribution Of Vascular Niches To Myelodysplasia." Blood 122, no. 21 (2013): 860. http://dx.doi.org/10.1182/blood.v122.21.860.860.

Full text
Abstract:
Abstract Endothelial cells are relevant to normal hematopoiesis. Vascular niches within the bone marrow allow hematopoietic stem cell proliferation and differentiation. In vivo, the contemporary administration of endothelial progenitor cells in BALB/c mice fosters the homing of transplanted hematopoietic cells (Salter et al. Blood 2009; 113:2104). In vitro, the differentiation of CD34+ progenitors is facilitated by the presence of an underlying confluent layer of endothelial colony forming cells (ECFCs Ingram et al. Blood. 2004;104: 2752), even though this effect is highly dependent from the p
APA, Harvard, Vancouver, ISO, and other styles
6

Sharifi, Sajjad, Shima Mehrabadi, Farzad Rahmani та ін. "PNU-74654 Enhanced the Antiproliferative Activity of Gemcitabine by Targeting Wnt/β-Catenin Pathway in Pancreatic Cancer". Clinical Cancer Drugs 11 (21 жовтня 2024). http://dx.doi.org/10.2174/012212697x293676240916114229.

Full text
Abstract:
Background: The Wnt/beta-catenin pathway is one of the pathways that is deregulated in pancreatic cancer and is reported to be associated with a poor prognosis. This indicates the need for the identification of novel agents to improve the efficacy of current therapy or have an improved efficacy. Therefore, in the present study, we explored the anticancer activity of PNU- 74654 alone or in combination with gemcitabine in 2 and 3-dimensional cell culture models of pancreatic cancer. Methods: The MTT assay was carried out to determine the viability of PC cancerous cells (PCCs), while the cytotoxi
APA, Harvard, Vancouver, ISO, and other styles

Dissertations / Theses on the topic "Wnt/Bcatenin pathway"

1

COSTA, ALICE. "Dissecting the role of G9a/GLP histone methyltransferases in Platinum-resistant ovarian cancer." Doctoral thesis, Università degli Studi di Trieste, 2021. http://hdl.handle.net/11368/2988156.

Full text
Abstract:
Epithelial Ovarian Cancer (EOC) is the most aggressive gynecological malignancy, mainly due to the advanced stage at diagnosis and the development of drug-resistant recurrences. Epigenetic modifications, such as histone methylation/acetylation, are emerging as key regulators of tumor growth and response to therapies. To verify if they are also involved in the onset of drug re-sistance in EOC, we performed a high-throughput Epigenetic Modifiers (EMs)-based screening using four different models of EOC isogenic Platinum-Resistant (PT-Res) cells. The screening identified G9a/GLP histone methyltran
APA, Harvard, Vancouver, ISO, and other styles
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography