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Books on the topic 'Worms – Intestinal and parasitic'

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Published: 4 June 2021
Last updated: 1 February 2022

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1

Arlene, Jones, ed. Parasitic worms of fish. London: Taylor & Francis, 1994.

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2

Williams, H. Harford. Parasitic worms of fish. Bristol, PA: Taylor & Francis, 1994.

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3

Williams, Harford. Parasitic worms of fish. London: Taylor& Francis, 1994.

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4

Souza, Ricardo M. Plant-Parasitic Nematodes of Coffee. Dordrecht: Springer Science+Business Media B.V., 2008.

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5

Alvarez-Pellitero, P. Mucosal intestinal immunity and response to parasite infections in ectothermic vertebrates. New York: Nova Science Publishers, 2011.

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6

1913-, Brooke Marion Murphy, and Centers for Disease Control (U.S.). Laboratory Training and Consultation Division., eds. Laboratory procedures for the diagnosis of intestinal parasites. 3rd ed. Atlanta, GA: U.S. DHHS, PHS, Centers for Disease Control, Laboratory Improvement Program Office, Laboratory Training and Consultation Division, 1985.

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7

L, Price Donald. Procedure manual for the diagnosis of intestinal parasites. Boca Raton: CRC Press, 1994.

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8

Miguel, Edward. Worms: Education and health externalities in Kenya. Cambridge, MA: National Bureau of Economic Research, 2001.

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9

Condie, Alison. An investigation of intestinal parasitic infection and haemoglobin levels of children in a Bir Zeit school. Bir Zeit, West Bank: Birzeit University, Community Health Unit, 1986.

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10

Parker, James N., and Philip M. Parker. The official patient's sourcebook on fasciolopsiasis. San Diego, Calif: Icon Health Publications, 2002.

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11

Parasites, worms, and the human body in religion and culture. New York: P. Lang, 2012.

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12

Gardenour, Brenda S. Parasites, worms, and the human body in religion and culture. New York: P. Lang, 2012.

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13

Vadillo, Clifford Abrico. Pepe-Bulate: Orihinal na isinulat ni Clifford Abrico Vadillo sa wikang Kinaray-a. Iloilo City, Philippines: Save the Children Federation, Inc., 2009.

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14

Ogbe, Martin Gbemi. Worms, disease, and the environment in the changing face of science: 8th inaugural lecture delivered at the Delta State University, Abraka on Tuesday September 28th, 2004. Abraka: Delta State University, 2004.

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15

Escobedo, Miguel A. Assessment of parasitic disease in children in five communities in the border region of far West Texas. Austin] Texas: Texas Dept. of Health, Office of Border Health, 2003.

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16

Smith, William Abbotts. On Human Entozoa: Comprising the Description of the Different Species of Worms Found in the Intestines and Other Parts of the Human Body, and the Pathology and Treatment of the Various Affections Produced by Their Presence. HardPress, 2020.

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17

Bibliography of the Helminth Parasites New Zealand - Cabi Ito. C.A.B. International, 1996.

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18

Myung, Yoon S. Intestinal Parasites & Worms in Adults & Children: Index of New Information. ABBE Publishers Association of Washington, D., 1998.

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19

Myung, Yoon S. Intestinal Parasites & Worms in Adults & Children: Index of New Information. ABBE Publishers Association of Washington, D., 1998.

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20

Nolan, T. J., T. B. Nutman, and G. A. Schad. Strongyloidosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0064.

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Strongyloidosis is an intestinal parasitism caused by the threadworm, Strongyloides stercoralis. The parasite, occurring in dogs, primates and man, is found throughout the moist tropics, as well as in temperate areas where poor sanitation or other factors facilitate the occurrence of faecally transmitted organisms. In some parts of the world, notably Africa and New Guinea, human infections caused by S. fülleborni have been reported. In Africa, the latter is primarily a parasite of primates, but in New Guinea, no animal host is known. S. stercoralis is unique among zoonotic nematodes, in that larvae passing in the faeces can give rise to a free-living generation of worms which, in turn, give rise to infective larvae. This life history alternative (i.e. heterogonic development) acts as an amplification mechanism, increasing the population of infective larvae in the external environment. The infective larvae are active skin penetrators; infection per os , while possible, is probably of limited importance. Because the parasitic female’s eggs hatch internally, a potential for autoinfection exists when precociously developing larvae attain infectivity while still in the host. This is another virtually unique feature of S. stercoralis infections in both its human and animal hosts. Autoinfection can occasionally escape control by the host, with massive re-penetration and larval migration. This can cause pulmonary or cerebro-spinal strongyloidosis as well as fulminant intestinal parasitism. Control of canine strongyloidosis has been achieved in kennels by strategic use of anthelmintics. Given the lack of epidemiological information community-based programs to control human strongyloidosis have not been attempted. The growing importance of human strongyloidosis depends upon the unique ability of S. stercoralis to replicate within its host and to behave as a potentially fatal opportunistic pathogen in immunocompromised hosts, particularly in those receiving corticosteroids.
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21

Swayanandan, S. Intestinal Worms. B. Jain Publishers, 2004.

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22

Parasitic Worms. Shire Publications, 1999.

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23

Nithikathkul, Choosak, Prasert Saichua, Louis Royal, and John H. Cross. Capillariosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0065.

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Capillaria species are members of the superfamily Trichinelloidae. These worms have a filamentous thin anterior end and a slightly thicker oesophagus which is surrounded by glandular cells or stichocytes. This oesophageal pattern is called stichosomal oesophagus. Capillaria species are parasites which are found in many vertebrate animals. More than two hundred species have been reported in several vertebrate species, including fish, amphibians, reptiles, birds, and mammals (Cross 1992; Chitwood et al. 1968), but only three species infect humans. These are Capillaria hepatica , C. aerophila and C. philippinensis (McCarthy and Moore 2000). Of these intestinal capillariosis, a fish-borne parasitic zoonosis caused by C. philippinensis , is the most important. Humans acquire the parasite, C. philippinensis, by eating uncooked or raw freshwater fish (Cross and Basaca-Sevilla 1991). The disease is endemic mainly in Philippines and Thailand where there are many reported fatalities.Although C . hepatica is found in rodents worldwide, only a few cases of hepatic capillariosis have been reported in humans from Europe, Asia, Africa, North and South America. The infection is acquired by the ingestion of embryonated eggs from the soil. Female worms deposit eggs in the liver tissue and granulomas develop around the egg. The eggs are released after the rodent is eaten and the liver digested. Eggs pass in the faeces and are deposited in the soil where they embryonate. Avoidance of contaminated soil would prevent human infection and destruction of rodents would control animal infections.Only 12 cases of human infection caused by Capillaria aerophila have been reported, the majority from Russia. The parasite is found within tissue of the respiratory passages of canines and felines worldwide.Anatrichosoma cutaneum (Nematoda, Trichosomoididae), also included in this chapter, is primarily a subcutaneous parasite of monkeys, but there are two reports of cutaneous infections in humans resulting in serpiginous lesions in the skin of the soles, palms, and nasal passages. In addition there is a further suspected case isolated from a breast nodule and a possible case of mucosal lesions in the mouth reported. Whole monkey colonies can be infected with this parasite and control is difficult.
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24

Sohn, Woon-Mok, and Jong-Yil Chai. Anisakiosis (Anisakidosis). Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0070.

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The term ‘anisakiosis (anisakidosis)’ or ‘anisakiasis’ collectively defines human infections caused by larval anisakids belonging to the nematode family Anisakidae or Raphidascarididae. Anisakis simplex, Anisakis physeteris, and Pseudoteranova decipiens are the three major species causing human anisakiosis. Various kinds of marine fish and cephalopods serve as the second intermediate hosts and the infection source. Ingestion of viable anisakid larvae in the fillet or viscera of these hosts is the primary cause of infection. The parasite does not develop further in humans as they are an accidental host. Clinical anisakiosis develops after the penetration of anisakid larvae into the mucosal wall of the alimentary tract, most frequently the stomach and the small intestine. The affected sites undergo erosion, ulceration, swelling, inflammation, and granuloma formation around the worm. The patients may suffer from acute abdominal pain, indigestion, nausea, vomiting, and in some instances, allergic hypersensitive reactions. Symptoms in gastric anisakiosis often resemble those seen in peptic ulcer or gastric cancer, and symptoms in intestinal anisakiosis resemble those of appendicitis or peritonitis. Treatments include removal of larval worms using a gastroendoscopic clipper or surgical resection of the mucosal tissue surrounding the worm. No confirmed effective anthelmintic drug has been introduced, though albendazole and ivermectin have been tried in vivo and in vitro. Prevention of human anisakiosis can be achieved by careful examination of fish fillet followed by removal of the worms in the restaurant or household kitchen. Immediate freezing of fish and cephalopods just after catching them on fishing boats was reported helpful for prevention of anisakiosis. It is noteworthy that anisakiosis is often associated with strong allergic and hypersensitivity reactions, with symptoms ranging from isolated angioedema to urticaria and life threatening anaphylactic shock.
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25

(1913-1918), Canadian Arctic Expedition, ed. Annelids, parasitic worms, Protozoans, etc. Part D: Gephyrea. Ottawa: J. de L. Taché, 1997.

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26

(1913-1918), Canadian Arctic Expedition, ed. Annelids, parasitic worms, Protozoans, etc. Part E: Acanthocephala. Ottawa: J. de L. Taché, 1997.

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27

(1913-1918), Canadian Arctic Expedition, ed. Annelids, parasitic worms, Protozoans, etc. Part M: Foraminifera. Ottawa: J. de L. Taché, 1997.

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28

1882-1959, Welch Paul S., and Canadian Arctic Expedition (1913-1918), eds. Annelids, parasitic worms, Protozoans, etc. Part A: Oligochaeta. Ottawa: J. de L. Taché, 1997.

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29

(1913-1918), Canadian Arctic Expedition, ed. Annelids, parasitic worms, Protozoans, etc. Part C: Hirudinea. Ottawa: T. Mulvey, 1997.

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30

Annelids, parasitic worms, Protozoan, etc. Part B: Polychaeta. Ottawa: T. Mulvey, 1997.

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31

Souza, Ricardo M. Plant-Parasitic Nematodes of Coffee. Springer, 2008.

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32

Prevention & Control of Intestinal Parasitic Infections (Technical Report Series,). World Health Organization, 1987.

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33

(1913-1918), Canadian Arctic Expedition, ed. Annelids, parasitic worms, Protozoans, etc. Part J: Polychaeta (supplementary). Ottawa: F.A. Acland, 1997.

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34

Annelids, parasitic worms, Protozoans, etc. Part G-H: Trematoda and Cestoda. Ottawa: T. Mulvey, 1997.

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35

Sanitation, Latrines and Intestinal Parasites in Past Populations. Taylor & Francis Group, 2015.

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36

WHO Expert Committee on Prevention and Control of Intestinal Parasitic Infections., ed. Prevention and control of intestinal parasitic infections: Report of a WHO expert committee. Geneva: World Health Organization, 1987.

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37

Pozio, Edoardo. Trichinellosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0068.

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Trichinellosis is caused by nematodes of the genus Trichinella. These zoonotic parasites show a cosmopolitan distribution in all the continents, but Antarctica. They circulate in nature by synanthropic-domestic and sylvatic cycles. Today, eight species and four genotypes are recognized, all of which infect mammals, including humans, one species also infects birds, and two other species infect also reptiles.Parasites of the genus Trichinella are unusual among the other nematodes in that the worm undergoes a complete developmental cycle, from larva to adult to larva, in the body of a single host, which has a profound influence on the epidemiology of trichinellosis. When the cycle is complete, the muscles of the infected animal contain a reservoir of larvae, capable of long-term survival. Humans and other hosts become infected by ingesting muscle tissuescontaining viable larvae.The symptoms associated with trichinellosis vary with the severity of infection, i.e. the number of viable larvae ingested, and the time after infection. The capacity of the worm population to undergo massive multiplication in the body is a major determinant. Progression of disease follows the biological development of the parasite. Symptoms are associated first with the gastrointestinal tract, as the worms invade and establish in the small intestine, become more general as the body responds immunologically, and finally focus on the muscles as the larvae penetrate the muscle cells and develop there. Although Trichinella worms cause pathological changes directly by mechanical damage, most of the clinical features of trichinellosis are immunopathological in origin and can be related to the capacity of the parasite to induce allergic responses.The main source of human infection is raw or under-cooked meat products from pig, wild boar, bear, walrus, and horses, but meat products from other animals have been implicated. In humans, the diagnosis of infection is made by immunological tests or by direct examination of muscle biopsies using microscopy or by recovery of larvae after artificial digestion. Treatment requires both the use of anthelmintic drugs to kill the parasite itself and symptomatic treatment to minimize inflammatory responses.Both pre-slaughter prevention and post-slaughter control can be used to prevent Trichinella infections in animals. The first involves pig management control as well as continuous surveillance programmes. Meat inspection is a successful post-slaughter strategy. However, a continuous consumer education is of great importance in countries where meat inspection is not mandatory.
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38

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Bacterial, fungal, and parasitic infections of the liver. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0059.

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Bacterial sepsis 428Spirochaetal infections 431Rickettsial infections 432Fungal infections 432Parasitic infections 434Granulomatous hepatitis 437Infectious agents can affect the liver either via direct invasion or by release of toxins. The liver's dual blood supply renders it uniquely susceptible to infection, receiving blood from the intestinal tract via the hepatic portal system, and from the systemic circulation via the hepatic artery. Because of this unique perfusion, the liver is frequently exposed to systemic or intestinal infections or the mediators of toxaemia. The biliary tree provides a further conduit for gut bacteria or parasites to access the liver parenchyma....
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39

Rhind, William. A Treatise On The Nature And Cure Of Intestinal Worms Of The Human Body. Kessinger Publishing, LLC, 2007.

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40

Rhind, William. A Treatise On The Nature And Cure Of Intestinal Worms Of The Human Body. Kessinger Publishing, LLC, 2007.

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41

L, Price Donald. Procedure Manual for the Diagnosis of Intestinal Parasites. Taylor & Francis Group, 2017.

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42

ADAS, ed. Grazing plans for the control of stomach and intestinal worms in sheep and in cattle. London: MAFF/ADAS, 1986.

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43

1914-, Ishikura H., and Kikuchi Kōkichi, eds. Intestinal anisakiasis in Japan: Infected fish, sero-immunological diagnosis, and prevention. Tokyo: Springer-Verlag, 1990.

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44

Belly-busting Worm Invasions!: Parasites That Love Your Insides! (24/7: Science Behind the Scenes). Franklin Watts, 2007.

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45

Tilden, Thomasine E. Lewis. Belly-busting Worm Invasions!: Parasites That Love Your Insides! (24/7: Science Behind the Scenes). Franklin Watts, 2007.

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46

Cleave, H. J. Van. Report of the Canadian Arctic Expedition, 1913-18, Vol. 9: Annelids, Parasitic Worms, Protozoans, Etc.; Part E, Acanthocephala (Classic Reprint). Forgotten Books, 2018.

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47

Savioli, Lorenzo, and D. W. T. Crompton. Handbook of Helminthiasis for Public Health. Taylor & Francis Group, 2006.

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48

Crompton, D. W. T., and Lorenzo Savioli. Handbook of Helminthiasis for Public Health. CRC, 2006.

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49

Cobbold, Thomas Spencer. Worms, a Series of Lectures on Practical Helminthology: Delivered at the Medical College of the Middlesex Hospital, with Cases Illustrating the Symptoms, Diagnosis and Treatment of Internal Parasitic Diseases. HardPress, 2020.

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50

Jex, Aaron R., Rachel M. Chalmers, Huw V. Smith, Giovanni Widmer, Vincent McDonald, and Robin B. Gasser. Cryptosporidiosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0053.

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Cryptosporidium species represent a genus of parasitic protozoa (Apicomplexa) that are transmitted via the faecal-oral route and commonly infect the epithelial tissues of the gastric or intestinal (or sometimes the respiratory) tract of many vertebrates, including humans. Infection occurs following the ingestion of viable and resistant oocysts, through direct host-to-host contact or in contaminated food, drinking or recreational water. Infection can be transmitted via anthroponotic (human-to-human, human-to-animal) or zoonotic (animal-to-human or animal-to-animal) pathways, depending upon the species of Cryptosporidium. Although infection can be asymptomatic, common symptoms of disease (cryptosporidiosis) include diarrhoea, colic (abdominal pain), nausea or vomiting, dehydration and/or fever. In humans, cryptosporidial infection in immunocompetent patients is usually short-lived (days to weeks) and eliminated following the stimulation of an effective immune response. However, infection in immunodeficient individuals (e.g., those with HIV/AIDS) can be chronic and fatal (in the absence of immunotherapy), as there are few effective anti-cryptosporidial drugs and no vaccines available. The present chapter provides an account of the history, taxonomy and biology, genomics and genetics of Cryptosporidium, the epidemiology, pathogenesis, treatment and control of cryptosporidiosis and the advances in tools for the identification and characterisation of Cryptosporidium species and the diagnosis of cryptosporidiosis.
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