Relevant bibliographies by topics / Worsening renal function

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Academic literature on the topic 'Worsening renal function'

Author: Grafiati
Published: 4 June 2025
Last updated: 20 July 2025

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Journal articles on the topic "Worsening renal function"

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Teerlink, John R., and Barry M. Massie. "Nesiritide and Worsening of Renal Function." Circulation 111, no. 12 (2005): 1459–61. http://dx.doi.org/10.1161/01.cir.0000160874.48045.54.

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Testani, Jeffrey, Martin St. John Sutton, and James Kirkpatrick. "Venous Congestion and Worsening Renal Function." Journal of the American College of Cardiology 54, no. 7 (2009): 661. http://dx.doi.org/10.1016/j.jacc.2009.03.074.

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van Kimmenade, Roland R. J., Tim J. ten Cate, and Hans-Peter Brunner-La Rocca. "Worsening Renal Function in Heart Failure." Journal of the American College of Cardiology 69, no. 1 (2017): 70–72. http://dx.doi.org/10.1016/j.jacc.2016.11.016.

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&NA;. "Worsening renal function with longer nesiritide infusions." Reactions Weekly &NA;, no. 1148 (2007): 5. http://dx.doi.org/10.2165/00128415-200711480-00017.

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&NA;. "Worsening renal function with longer nesiritide infusions." Inpharma Weekly &NA;, no. 1584 (2007): 25. http://dx.doi.org/10.2165/00128413-200715840-00074.

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Tsay, Julie, Daniel Pinkhas, Bryan C. Lee, et al. "Worsening Renal Function in Cardiac Mechanical Support." Heart, Lung and Circulation 29, no. 8 (2020): 1247–55. http://dx.doi.org/10.1016/j.hlc.2019.11.011.

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Warraich, Haider J., and Anju Nohria. "Is worsening renal function relevant without clinical context?" European Journal of Heart Failure 24, no. 2 (2022): 375–77. http://dx.doi.org/10.1002/ejhf.2416.

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Maqsood, Farhan, and Harold M. Szerlip. "Relationship of Venous Congestion to Worsening Renal Function." Journal of the American College of Cardiology 54, no. 7 (2009): 661–62. http://dx.doi.org/10.1016/j.jacc.2009.03.073.

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Núñez, Julio, Eduardo Núñez, Gema Miñana, Antoni Bayés-Genis, and Juan Sanchis. "Worsening Renal Function in Acute Decompensated Heart Failure." JACC: Heart Failure 4, no. 3 (2016): 232–33. http://dx.doi.org/10.1016/j.jchf.2015.10.013.

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Tan, M. H., S. K. Glendon Lau, W. H. Han, N. T. Ross, R. Visvanathan, and Y. Y. Ngau. "072 WORSENING RENAL FUNCTION IN HEART FAILURE PATIENTS." Kidney International Reports 2, no. 4 (2017): S35. http://dx.doi.org/10.1016/j.ekir.2017.06.112.

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Books on the topic "Worsening renal function"

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Wiles, Kate, Kate Bramham, and Catherine Nelson-Piercy. Kidney disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0044.

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This chapter describes the physiological adaptations to pregnancy in women with and without renal disease, reports pregnancy outcomes in women with both acute kidney injury and chronic kidney disease, and discusses a management strategy for antenatal and peripartum care. Acute kidney injury (AKI) is difficult to define in pregnancy because of the physiological increase in glomerular filtration. A normal creatinine can mask renal injury in pregnancy. This chapter considers important causes of AKI in pregnancy including pre-eclampsia, HELLP syndrome, thrombotic microangiopathy, acute fatty liver
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Ostermann, Marlies, and Ruth Y. Y. Wan. Diuretics in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0058.

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Fluid overload and chronic hypertension are the most common indications for diuretics. The diuretic response varies between different types and depends on underlying renal function. In patients with congestive heart failure, diuretics appear to reduce the risk of death and worsening heart failure compared with placebo, but their use in acute decompensated heart failure is questionable. Diuretics are also widely used in chronic kidney disease to prevent or control fluid overload, and treat hypertension. In acute kidney injury, there is no evidence that they improve renal function, speed up reco
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Cruz, Dinna N., Anna Giuliani, and Claudio Ronco. Acute kidney injury in heart failure. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0248.

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Acute kidney injury (AKI) occurring during heart failure (HF) has been labelled cardiorenal syndrome (CRS) type 1. CRS is defined as a group of ‘disorders of the heart and kidneys whereby acute or chronic dysfunction in one organ may induce acute or chronic dysfunction of the other’. This consensus definition was proposed by the Acute Dialysis Quality Initiative, with the aim to standardize those disorders where cardiac and renal diseases coexist. Five subtypes have been proposed, according to which organ is affected first (cardiac vs renal) and whether the dysfunction is acute or chronic. Ano
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Book chapters on the topic "Worsening renal function"

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Verbrugge, F. H., W. Mullens, and M. Malbrain. "Worsening Renal Function during Decompensated Heart Failure: The Cardio-abdomino-renal Syndrome." In Annual Update in Intensive Care and Emergency Medicine 2012. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-25716-2_52.

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Gracia, Ely, Javed Butler, and Sandeep K. Mallipattu. "Worsening Renal Function in a Patient with Acute Heart Failure and Volume Overload." In Cardiorenal Syndrome in Heart Failure. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-21033-5_10.

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Kumar, Shivram, and Kelly D. Flemming. "Neurologic Associations With Cardiac, Pulmonary, Renal, Hepatobiliary, and Hematologic Disease." In Mayo Clinic Neurology Board Review, edited by Kelly D. Flemming. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780197512166.003.0127.

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The brain has a higher demand for cardiac output than any other organ, and it strictly relies on oxygen and glucose metabolism. Consequently, the brain is exquisitely sensitive to homeostatic disturbances and extraneural organ dysfunction leading to cardiac, pulmonary, renal, hepatobiliary, and hematologic diseases. The primary neurologic manifestation of extraneural organic dysfunction is diffuse bihemispheric dysfunction or encephalopathy, which often lacks lateralizing or localizing signs. Common clinical findings are lethargy, difficulty with attention and orientation, sleep-wake disturbance, and psychomotor slowing. As organic dysfunction progresses, a moderate encephalopathy ensues, with worsening cognitive function, gross disorientation, hypoactive or hyperactive psychomotor state, frontal release signs, asterixis, and myoclonus. If organ failure (eg, hepatic or renal) progresses further, stupor and coma may result unless organ function improves. Patients with underlying organic brain disease from degenerative dementia can decompensate out of proportion to neurologically normal counterparts, resulting in encephalopathy even from minor organ dysfunction or infection.
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Costa, Rafaela Cristina Cardoso, Jaqueline Aparecida Sousa Pereira, Bruna Cardoso Lemes, et al. "Chronic kidney disease in felines and the use of SDMA for diagnosis: Literature review." In Uniting Knowledge Integrated Scientific Research For Global Development V.2. Seven Editora, 2024. http://dx.doi.org/10.56238/sevened2024.018-047.

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Due to the domestication process, tutors are increasingly concerned with ensuring better living conditions for their pets. This has reflected in the increased longevity of pets. Felines, due to anatomical, physiological and dietary aspects, have a certain predisposition to problems affecting the system renal.Com this, chronic kidney disease (CKD) is a progressive and irreversible disease, being an important cause of death in felines, especially with advanced age. Unfortunately, its initial signs are silent, becoming evident as the lesion progresses, thus allowing the worsening of the condition. Thus, when the diagnosis occurs, the evolution is already well established. Symmetrical dimethylarginine (SDMA) is a biomarker of renal function, correlated with glomerular filtration rate (TGF), and is used to evaluate the existence of Chronic Kidney Disease, because unlike the quantification of serum creatinine, it is not influenced by extrarenal mechanisms, for this reason it is considered more efficient. In 2015, SDMA became provisionally part of the International Society of Renal Interest (IRIS) classification of CKD as a complementary test of kidney function aiding in the classification of disease stages, thus allowing the identification of disease stages and substages. In this way, it is possible to better monitor and determine the most appropriate treatment for each patient.
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Gurgel, Mel Vitória Gama, Thalita Gonçalves Campos, and Juscélio Clemente de Abreu. "A reflection on anxiety and depression in patients undergoing hemodialysis treatment." In Health of Tomorrow: Innovations and Academic Research. Seven Editora, 2024. http://dx.doi.org/10.56238/sevened2023.007-071.

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This study aimed to analyze the relationship between End-Stage Renal Disease (ESRD) and the appearance of anxious and depressive symptoms among patients. Thus, ESRD is understood as a set of changes in kidney structure and function due to various causes, thus requiring renal replacement therapy (RRT), with hemodialysis being the main one used. However, due to the significant changes caused by this illness, the patient must undergo a series of adaptations to their lifestyle and daily routine. This can be explained by the limitations on physical activity, absence from work, increased dependence on others and changes in daily habits. As a result of being a tedious process, hemodialysis treatment can generate signs of anxiety, anguish and fear about the future, promoting depressive symptoms as side effects of the worsening of their condition, such as intense sadness and suffering. The combination of all these negative factors can stimulate the development of mental disorders among patients, either by reducing their quality of life or by the clinical effects of the disease itself.
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Cleland, John G. F., and Yura Mareev. "HFrEF pharmacological treatment: digitalis glycosides." In ESC CardioMed. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0428.

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About 25 years ago, a series of trials suggested that withdrawal of digoxin from patients with heart failure who were stable, had mild symptoms, a reduced left ventricular ejection fraction, and in sinus rhythm could lead to worsening symptoms. Withdrawal of digoxin was also associated with a decline in renal function and weight gain, suggesting important renal effects, as well as a decline in left ventricular ejection fraction and exercise capacity. However, stopping a medication in a patient who has responded to it may carry bias; the effect of initiating digoxin in a digoxin-naïve patient is unknown. A large outcome study showed no effect on mortality. Since then, however, the evidence both for and against digoxin has been superseded by important innovations in therapy, such as beta blockers and mineralocorticoid antagonists. For patients in atrial fibrillation, evidence of a clinical benefit other than ventricular rate control, itself a controversial issue, is lacking. Current European Society of Cardiology Guidelines on heart failure suggest a limited role for digoxin for the management of symptoms of heart failure when other treatments have failed. New studies investigating the role of digitalis glycosides in patients with heart failure receiving contemporary background therapy are ongoing.
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Moreira, Roberto de Souza, Maria Claudia Costa Irigoyen, José Manuel Condor Capcha, et al. "Protective effect of apolipoprotein AI mimetic peptide 4F on renal and cardiac injury and endothelial dysfunction induced by acute myocardial infarction in hypercholesterolemic rats receiving iodinated contrast." In Medicine: an exploration of the anatomy of the human body. Seven Editora, 2024. http://dx.doi.org/10.56238/sevened2024.005-019.

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The use of contrast after angiography in infarcted animals induces acute kidney injury, being associated with worsening prognosis and increased mortality. Hypercholesterolemia is an aggravating factor of endothelial injury in acute myocardial infarction (AMI) and the use of contrast in diagnosis and treatment can cause acute kidney injury. Treatment with the apolipoprotein AI mimetic peptide 4F can reverse endothelial injury by reducing LDL levels and preventing its oxidation. OBJECTIVES: To analyze the effect of Apo A-I (using mimetic peptide 4F) on cardiac and renal injury induced by acute myocardial infarction (AMI) with contrast therapy in hypercholesterolemic rats. METHODS: This was a prospective study with rats on a diet at 4% cholesterol for 8 days, divided into a SHAM group operated without coronary ligation (n=6) or infarcted animals with ligation of the left anterior descending coronary artery, with or without the use of contrast and treatment 6 hours after infarction induction: AMI (n=15), AMI+C (iopamidol 2.9 g/kg body weight, intrafemoral artery injection n=15), AMI+4F (4F, 10mg/kg body weight, peritoneal injection, n=8) and AMI+C+4F (n=8). All results are analyzed after 24 AMI and expressed as mean and standard error. RESULTS: It was observed that the AMI+4F and AMI+C+4F groups showed a better response to cardiac injury and renal injury compared to the AMI and AMI+C groups. There was an improvement in renal function through 12-hour creatinine clearance, increased expression of eNOS, increased VEGF, preservation of mitochondrial morphology, reduction of inflammation with a lower expression of CD68+ (macrophages), decreased positive tunnel cells associated with an increase in apolipoprotein AI (Apo AI) expression in renal tissue. The same happened in cardiac function with decreased plasma troponin, increased expression of eNOS, VEGF, isolectin B4, reduction in inflammation represented by lower TLR4 expression, positive tunnel cells, improved cholesterol profile, preservation of mitochondrial morphology and associated with increased expression of Apo AI in cardiac tissue. Hemodynamics were preserved with improvement in cardiac output, ejection fraction, baroreflex response, left ventricular end-diastolic pressure, and associated with a decrease in the infarct area measured by both echocardiogram and immunohistochemistry. We demonstrate that treatment with apolipoprotein AI can be a therapeutic option in cardiac and renal injury by reversing the inflammatory response through the efflux of HDL-dependent cholesterol.
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Mahmoodi, Marzieh, Fatemeh Mansouri, Maede Makhtoomi, Zainab Shateri, and Mehran Nouri. "More Consumption of Ultra-Processed and Sugar-Sweetened Beverages Could Increase Odds of Sarcopenia in Kidney Diseases." In Neuromuscular Diseases - How to Recognize and Treat Them [Working Title]. IntechOpen, 2025. https://doi.org/10.5772/intechopen.1006106.

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Chronic kidney disease (CKD) is a degenerative condition characterized by the gradual deterioration of renal function. Among the risk factors for the disease, older age, ethnicity, low birth weight, and family history of kidney disease are the most important. Other significant risk factors include smoking, obesity, hypertension, uncontrolled diabetes mellitus, exposure to heavy metals, chronic alcohol consumption, and the use of analgesic medications. Findings have also shown the relationship between CKD and sarcopenia. The prevalence of sarcopenia increases with the severity of CKD, independent of the definition of sarcopenia used. Muscle wasting in CKD is multifactorial. Several factors related to the development of chronic kidney failure lead to muscle loss, making CKD an independent risk factor for sarcopenia. Increasing age and female gender are known as non-modifiable risk factors for sarcopenia, while dietary intake is considered a modifiable risk factor. In the elderly, nutritional imbalances have been reported due to age-related physiological changes, such as loss of appetite, diminished senses of taste and thirst, and impaired nutrient absorption and utilization. Poor diet quality can potential increase the risk of worsening sarcopenia in CKD patients. Therefore, nutritional recommendations for CKD patients should emphasize nutrient-dense foods to support overall health and mitigate the risk of sarcopenia.
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Şentürk, Ömer, and Uğur Korkmaz. "Use of 5-ASA in Ulcerative Colitis in the Era of Biologics." In Unveiling Ulcerative Colitis - A Comprehensive Approach to Understanding and Management [Working Title]. IntechOpen, 2024. https://doi.org/10.5772/intechopen.1007709.

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In the era of biologic drugs, 5-aminosalicylic acid (5-ASA compounds) still constitutes the most important step in the treatment of patients with mild-to-moderate ulcerative colitis (UC). They can also be effective at high doses in moderately severe patients who are not at high risk. However, the use of many drugs daily can make it difficult for patients to comply with this group of drugs. Therefore, long-acting, single-dose drugs in the form of the Multi Matrix System (MMX) can play a very important role in treatment management. Although it is not comfortable for every patient, topical 5-ASAs can be used effectively and safely, especially in cases with proctitis and left colon involvement. Mesalazine preparations can also be used safely during pregnancy. In general, the oral dose that provides remission should be the dose selected for maintenance therapy. However, the dose can be reduced over time in topical treatment. Although more robust evidence is needed, 5-ASA preparations are also widely used in the prevention of colorectal cancer (CRC) in UC. 5-ASA drugs have little systemic toxicity. Although safe and well tolerated, patients should still be informed about rare but serious side effects, paradoxical worsening of symptoms at the beginning of treatment, and the need for long-term monitoring of renal function.
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Navsaria, Pradeep, Deidre McPherson, and Eduard Jonas. "Abdominal compartment syndrome." In Abdominal Trauma, Peritoneum, and Retroperitoneum, edited by Aditya J. Nanavati, Sanjay Nagral, Samiran Nundy, and Dirk J. Gouma. Oxford University PressOxford, 2022. http://dx.doi.org/10.1093/med/9780192862433.003.0005.

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Abstract The abdominal compartment syndrome (ACS) can be defined as a state of acute organ dysfunction, predominantly affecting the cardiovascular, respiratory, and renal systems from a sustained increase in intra-abdominal pressure (IAP) causing intra-abdominal hypertension (IAH). The early recognition, prompt diagnosis, and immediate intervention of ACS can improve organ dysfunction. Intra-abdominal pressure and the abdominal compartment syndrome are separate, distinct clinical entities. Intra-abdominal pressure is the steady-state pressure concealed within the abdominal cavity. Intra-abdominal hypertension is defined by a sustained or repeated pathological elevation in IAP ≥ 12 mmHg. Abdominal compartment syndrome is defined as a sustained increase of IAP over 20 mmHg that is associated with new or worsening organ function. The effects of IAH are not limited to the intra-abdominal organs but have an impact either directly or indirectly on other organ systems in the body. The definitive diagnosis of IAH/ACS requires a measurement of the IAP. The intra-vesicular technique is a minimally invasive, easy, precise, reliable, and reproducible way of measuring IAP. The development of IAH/ACS during a critical care unit stay is an independent predictor of a patient’s outcome. Medical interventions target three critical contributors to IAH; solid organ and hollow-viscera volume, space-occupying lesions, such as ascites, blood, abdominal packs and tumours, and conditions that limit abdominal wall compliance. Surgical decompression of the abdominal cavity is considered the definitive management of ACS. Temporary abdominal wall closure using negative pressure wound therapy has the highest fascial closure and lowest mortality rates.
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Conference papers on the topic "Worsening renal function"

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Nesović Ostojić, Jelena, Sanjin Kovačević, Nikola Mitović, Aleksandra Nenadović, Andrija Vuković, and Mirjana Jovanović. "When, how, and why is it necessary to assess kidney function in cardiovascular patients?" In 7th International Congress of Cardionephrology KARNEF 2025. Punta Niš, 2025. https://doi.org/10.46793/karnef25.182no.

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Renal function is an important aspect to evaluate in cardiovascular patients because kidney function and heart function are closely interconnected. Cardiorenal syndrome refers to this complex relationship, where either heart or kidney dysfunction can lead to worsening of the other, making their management more complex and challenging. Renal function in cardiovascular patietns should be assessed at the time of diagnosis of cardiovascular conditions (e.g., heart failure, coronary artery disease, or hypertension), as kidney function can impact the treatment plan and prognosis; during hospitalizat
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Santos, Hanna Caroline Antunes dos, Gabriela Vilaça Gutierrez, Ananda Ribeiro Fretes, Renata Froes Ramos de Lima, Alisson Pugliesi, and Ana Paula Toledo Del Rio. "HYPERTENSION, PROTEINURIA AND WORSENING RENAL FUNCTION IN A PATIENT WITH SLE AND SSC OVERLAP: LUPUS NEPHRITIS OR SCLERODERMMIC RENAL CRISIS? A CASE REPORT." In XL Congresso Brasileiro de Reumatologia. Sociedade Brasileiro de Reumatologia, 2023. http://dx.doi.org/10.47660/cbr.2023.2327.

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Gauza, Mateus de Miranda, Andréia Canello, Henrique Muller Genero, Rafael Marques Mendes, Maria Francisca Moro Longo, and Jordana Dolores Villar Lino. "Ramsay Hunt syndrome - a case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.298.

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Context: Ramsay Hunt syndrome is uncommon, with an incidence of 5 / 100,000 people (1), characterized by the triad of ipsilateral peripheral facial paralysis, otalgia and vesicles in the ear canal and / or auricular pavilion (1,2,3). It results from the reactivation of the Varicella Zoster virus in the geniculate ganglion and may extend to other cranial pairs, causing inflammation, edema and nerve dysfunction (1,3). As the second leading cause of peripheral facial palsy (1), its importance lies in a lower likelihood of recovery with a worse prognosis compared to Bell’s palsy (4). Case report:
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Santos, João Vitor Ribeiro dos, Mariana Spitz, and Ana Carolina Andorinho. "Stroke secondary to thrombotic microangiopathy." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.300.

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Introduction: Thrombotic thrombocytopenic purpura (TTP) is a hematological disease resulting from the ADAMTS 13 plasmatic protein deficit. It can be congenital or sporadic, and is usually autoimmune. Pathological platelet adhesion occurs, leading to microthrombi in capillary and arterial circulation, microangiopathic anemia and ischemia. The clinical picture includes thrombocytopenia, renal dysfunction, fluctuating neurological symptoms, microangiopathic hemolytic anemia, and fever. Methods: Case report of a 51-year-old male hypertensive patient, diagnosed with idiopathic thrombocytopenic purp
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Pelegrino, Paulo Sergio, and Alessandra Maria Felipe. "Co-Vivências, a residence model for the elderly that integrates specialized care and family living for active longevity. “Neurology Startups”." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.434.

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The institutionalization of the elderly is very strongly related to the diseases and complications associated with cognitive and functional impairment present in Alzheimer’s disease, Parkinson’s disease, other dementias, and stroke sequelae. The institutionalization of elderly people represents an aid to families. On the other hand, it has been a factor related to the worsening in cognition and functionality when compared to those who remain in their social environment and family life. The different modalities of long-term care facilities (LTCFs) for the elderly in Brazil contribute a lot to t
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Reports on the topic "Worsening renal function"

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Yuan, Shizhu, Yueming Liu, and Qiang He. Association between early worsening of renal function and poor outcomes in patients treated with renin angiotensin system inhibitors: A meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.7.0064.

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