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Relevant bibliographies by topics / Zacopride

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Journal articles
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Academic literature on the topic 'Zacopride'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 February 2022

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Journal articles on the topic "Zacopride"

1

Lin, Yuanyuan, Junhu Li, Baozhong Zhu, et al. "Zacopride Exerts an Antiarrhythmic Effect by Specifically Stimulating the Cardiac Inward Rectifier Potassium Current in Rabbits: Exploration of a New Antiarrhythmic Strategy." Current Pharmaceutical Design 26, no. 44 (2020): 5746–54. http://dx.doi.org/10.2174/1381612826666200701135508.

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Background: Zacopride, a potent antagonist of 5-HT3 receptors and an agonist of 5-HT4 receptors, is a gastrointestinal prokinetic agent. In a previous study, we discovered that zacopride selectively stimulated the inward rectifier potassium current (IK1) in the rat and that agonizing IK1 prevented or eliminated aconitine-induced arrhythmias in rats. Objective: Our aims were to confirm that the antiarrhythmic effects of zacopride are mediated by selectively enhancing IK1 in rabbits. Methods: The effects of zacopride on the function of the main ion channels were investigated using a whole-cell p

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Wade, P. R., G. M. Mawe, T. A. Branchek, and M. D. Gershon. "Use of stereoisomers of zacopride to analyze actions of 5-hydroxytryptamine on enteric neurons." American Journal of Physiology-Gastrointestinal and Liver Physiology 260, no. 1 (1991): G80—G90. http://dx.doi.org/10.1152/ajpgi.1991.260.1.g80.

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Two subtypes of excitatory 5-hydroxytryptamine (5-HT) receptor, 5-HT1P and 5-HT3, are found on type 2-AH neurons of the guinea pig myenteric plexus. The 5-HT1P receptor mediates a slow and the 5-HT3 receptor a fast depolarization of these cells, however, the role of these receptors in the physiology of the gut is unknown. Renzapride (BRL 24924), a substituted benzamide, has previously been found to antagonize responses of myenteric neurons mediated by both 5-HT1P and 5-HT3 receptors. The effects on myenteric type 2-AH neurons of a structurally similar benzamide, zacopride, which unlike renzapr

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&NA;. "Zacopride doesn??t meet expectations." Inpharma Weekly &NA;, no. 858 (1992): 12. http://dx.doi.org/10.2165/00128413-199208580-00018.

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SMITH, WILLIAM L., REEVIS S. ALPHIN, CARLOTTA B. JACKSON, and LAWRENCE F. SANCILIO. "The Antiemetic Profile of Zacopride." Journal of Pharmacy and Pharmacology 41, no. 2 (1989): 101–5. http://dx.doi.org/10.1111/j.2042-7158.1989.tb06402.x.

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Curtis, Michael J. "Activation of IK1 by Zacopride." Journal of Cardiovascular Pharmacology 64, no. 4 (2014): 343–44. http://dx.doi.org/10.1097/fjc.0000000000000140.

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Meyer, Leith C. R., Andrea Fuller, and Duncan Mitchell. "Zacopride and 8-OH-DPAT reverse opioid-induced respiratory depression and hypoxia but not catatonic immobilization in goats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 290, no. 2 (2006): R405—R413. http://dx.doi.org/10.1152/ajpregu.00440.2005.

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Neurophysiological studies have shown that serotonergic ligands that bind to 5-HT1A, 5-HT7, and 5-HT4 serotonin receptors in brain stem have beneficial effects on respiratory neurons during opioid-induced respiratory depression. The effect of these ligands on respiratory function and pulmonary performance has not been studied. We therefore examined the effects of 8-hydroxy-2-(di- n-propylamino)tetralin (8-OH-DPAT), an agonist of 5-HT1A and 5-HT7 receptors, and zacopride, an agonist of 5-HT4 receptors, to establish whether these ligands would reverse opioid-induced respiratory depression and hy

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SMITH, W. W., L. F. SANCILIO, J. B. OWERA-ATEPO, R. J. NAYLOR, and L. LAMBERT. "Zacopride, a potent 5-HT3 antagonist." Journal of Pharmacy and Pharmacology 40, no. 4 (1988): 301–2. http://dx.doi.org/10.1111/j.2042-7158.1988.tb05253.x.

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Ge, Jian, Janine M. Barnes, Patrick Towers, and Nicholas M. Barnes. "Distribution of S(−)-zacopride-insensitive []R(+)-zacopride binding sites in the rat brain and peripheral tissues." European Journal of Pharmacology 332, no. 3 (1997): 307–12. http://dx.doi.org/10.1016/s0014-2999(97)01091-1.

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Laporte, A. M., T. Koscielniak, M. Ponchant, D. Vergé, M. Hamon, and H. Gozlan. "Quantitative autoradiographic mapping of 5-HT3receptors in the rat CNS using [125I]iodo-zacopride and [3H]zacopride as radioligands." Synapse 10, no. 4 (1992): 271–81. http://dx.doi.org/10.1002/syn.890100402.

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Pisters, K. M. W., M. G. Kris, L. B. Tyson, R. A. Clark, and R. J. Gralla. "Dose-Ranging Trial of Zacopride: The Emetic Antiemetic?" JNCI Journal of the National Cancer Institute 84, no. 9 (1992): 717–18. http://dx.doi.org/10.1093/jnci/84.9.717.

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Dissertations / Theses on the topic "Zacopride"

1

Blum, Fabienne. "Pharmacologie animale du zacopride : nouvel antagoniste 5-HT3 au niveau central." Paris 5, 1991. http://www.theses.fr/1991PA05P022.

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Younes, Stephane Y. "Les récepteurs 5-HT4b adoptent différentes conformations ligand-spécifique ayant des propriétés de signalisation et de régulation distinctes." Thèse, 2012. http://hdl.handle.net/1866/8906.

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Les antidépresseurs actuels sont très similaires au niveau de leur mécanisme d’action et sont plus ou moins efficaces. Un des problèmes majeurs est leur long temps de latence à fournir une action thérapeutique dû aux adaptations des sites pré et post synaptiques. Dans un modèle animal, nous avons récemment découvert que l’agoniste RS67333 des récepteurs 5-HT4 était en mesure de produire en trois jours les mêmes effets antidépresseurs qui normalement prennent de deux à trois semaines à apparaître avec les antidépresseurs actuellement disponibles. De plus, nous avons constaté que les effets ant

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Trottier, Giacomo. "The biasing of the 5-HT4 receptor as an antidepressant target." Thèse, 2017. http://hdl.handle.net/1866/18903.

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La dépression majeure peut être très dommageable pour le 8% des Nord-Américains qui en souffriront au moins une fois durant leur vie. Les traitements actuels de la dépression ont été créés grâce à la compréhension de l'hypothèse de la monoamine; où les transporteurs de la sérotonine ou les enzymes sont bloqués ou inhibés afin de maintenir le neurotransmetteur (NT) dans la fente synaptique. Sur une période de plusieurs semaines, cette augmentation de NT encourage la plasticité synaptique, ce qui augmente la sensibilité de réponse de 5-HT dans la fente synaptique. Cette forme de traitement est e

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Book chapters on the topic "Zacopride"

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Pinkus, L. M., and J. C. Gordon. "Utilization of Zacopride and its R- and S-Enantiomers in Studies of 5-HT3 Receptor “Subtypes”." In Serotonin: Molecular Biology, Receptors and Functional Effects. Birkhäuser Basel, 1991. http://dx.doi.org/10.1007/978-3-0348-7259-1_44.

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Pecknold, J. C., L. Luthe, L. Iny, and M. H. Scott-Fleury. "Platelet [3H]-paroxetine and [3H]-imipramine binding in zacopride-treated patients with generalized anxiety disorder: preliminary results." In New Concepts in Anxiety. Macmillan Education UK, 1991. http://dx.doi.org/10.1007/978-1-349-11847-2_11.

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Machu, Tina. "Zacopride." In xPharm: The Comprehensive Pharmacology Reference. Elsevier, 2007. http://dx.doi.org/10.1016/b978-008055232-3.62872-7.

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