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Relevant bibliographies by topics / ZNF318

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Journal articles

Academic literature on the topic 'ZNF318'

Author: Grafiati

Published: 1 April 2023

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Journal articles on the topic "ZNF318"

1

Nizhnichenko, Vladimir A., Alexey V. Boyko, Talia T. Ginanova, and Igor Yu Dolmatov. "Muscle Regeneration in Holothurians without the Upregulation of Muscle Genes." International Journal of Molecular Sciences 23, no. 24 (2022): 16037. http://dx.doi.org/10.3390/ijms232416037.

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The holothurian Eupentacta fraudatrix is capable of fully restoring its muscles after transverse dissection. Although the regeneration of these structures is well studied at the cellular level, the molecular basis of the process remains poorly understood. To identify genes that may be involved in the regulation of muscle regeneration, the transcriptome of the longitudinal muscle band of E. fraudatrix has been sequenced at different time periods post-injury. An analysis of the map of biological processes and pathways has shown that most genes associated with myogenesis decrease their expression

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2

Mitchell, Emily, Michael Spencer Chapman, Nicholas Williams, et al. "Clonal Dynamics of Normal Haematopoiesis with Human Ageing." Blood 138, Supplement 1 (2021): 598. http://dx.doi.org/10.1182/blood-2021-150152.

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Abstract The haematopoietic system manifests several age-associated phenotypes including anaemia; loss of regenerative capacity, especially in the face of insults such as infection, chemotherapy or blood loss; and increased risk of clonal haematopoiesis and blood cancers. The cellular alterations that underpin these age-related phenotypes, which typically manifest in individuals aged over 70, remain elusive. We aimed to investigate whether changes in HSC population structure with age might underlie any aspects of haematopoietic system ageing. We sequenced 3579 genomes from single-cell-derived

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3

Sobocińska, Joanna, Joanna Nowakowska, Sara Molenda, et al. "Zinc Finger Proteins in Head and Neck Squamous Cell Carcinomas: ZNF540 May Serve as a Biomarker." Current Oncology 29, no. 12 (2022): 9896–915. http://dx.doi.org/10.3390/curroncol29120779.

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Head and neck squamous cell carcinoma (HNSCC) is one of the ten most common cancers. Most cancer cases originate from alcohol and tobacco consumption. However, studies have demonstrated that human papillomavirus (HPV) infection, particularly HPV-16, may also significantly influence disease progression. The KRAB-ZNF family of genes is involved in epigenetic suppression, and its involvement in carcinogenesis is the subject of extensive studies. The available literature data demonstrate that they may play different roles, both as tumor suppressors and oncogenes. In this study, six ZNF genes, ZFP2

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4

Dougherty, Michael P., Lynn P. Chorich, and Lawrence Clarke Layman. "Evaluation of Mayer-Rokitansky-Kuster-Hauser (MRKH) Patient Families by Whole Genome Sequencing." Journal of the Endocrine Society 5, Supplement_1 (2021): A501—A502. http://dx.doi.org/10.1210/jendso/bvab048.1025.

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Abstract Introduction: MRKH is a characterized by the congenital absence of the uterus and vagina in 46,XX individuals. A subset of these patients also has associated renal, skeletal, cardiac and/or auditory defects. Familial cases suggest a genetic component, but to date only pathogenic variants in WNT4 and HNF1B have been confirmed. We hypothesize that de novo heterozygous variants in candidate genes will be present in some patients with MRKH. Methods: DNAs from 30 quads (an MRKH proband and three relatives) were subjected to whole genome sequencing (WGS), and heterozygous variants in coding

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5

Ishikawa, S., M. Kai, Y. Takei, et al. "Isolation and mapping of a human zinc finger gene (ZNF188) homologous to ZNF187, a serum-response-element binding protein." Cytogenetic and Genome Research 77, no. 3-4 (1997): 185–89. http://dx.doi.org/10.1159/000134572.

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6

Yan, Feng-Juan, Yong-Jian Wang, Shi-Ran Yan, Jun Lu та Yuan-Lin Zheng. "ZNF300 stimulates fatty acid oxidation and alleviates hepatosteatosis through regulating PPARα". Biochemical Journal 476, № 2 (2019): 385–404. http://dx.doi.org/10.1042/bcj20180517.

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Abstract ZNF300 plays an important role in the regulation of HBV-related hepatocellular carcinoma. However, little is known about the role of ZNF300 in lipid metabolism and NAFLD. In the present study, we observed that ZNF300 expression was markedly decreased in free fatty acid (FFA)-induced fatty liver. Overexpressed ZNF300 alleviated hepatic lipid accumulation, whereas knockdown of ZNF300 enhanced the FFA-induced lipid accumulation. Investigations of the underlying mechanisms revealed that ZNF300 directly binds to and regulates the PPARα expression, thus promoting fatty acid oxidation. Furth

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7

Pieraccioli, Marco, Sara Nicolai, Consuelo Pitolli, et al. "ZNF281 inhibits neuronal differentiation and is a prognostic marker for neuroblastoma." Proceedings of the National Academy of Sciences 115, no. 28 (2018): 7356–61. http://dx.doi.org/10.1073/pnas.1801435115.

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Derangement of cellular differentiation because of mutation or inappropriate expression of specific genes is a common feature in tumors. Here, we show that the expression of ZNF281, a zinc finger factor involved in several cellular processes, decreases during terminal differentiation of murine cortical neurons and in retinoic acid-induced differentiation of neuroblastoma (NB) cells. The ectopic expression of ZNF281 inhibits the neuronal differentiation of murine cortical neurons and NB cells, whereas its silencing causes the opposite effect. Furthermore, TAp73 inhibits the expression of ZNF281

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8

Deng, Yu-Qin, Gang-Yong Kong, Song Li, Fen Li та Si-Lu Wen. "Upregulation of lnc-ZNF281 Inhibits the Progression of Glioma via the AKT/GSK-3β/β-Catenin Signaling Pathway". Journal of Immunology Research 2021 (11 травня 2021): 1–9. http://dx.doi.org/10.1155/2021/5573071.

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The purpose of this study is to elucidate the roles and potential underlying mechanisms of long noncoding RNA lnc-ZNF281 in glioma. We performed qRT-PCR to detect the expression levels of lnc-ZNF281 in glioma tissues. The effects of lnc-ZNF281 on the proliferative and migrative abilities of T98G and HS683 glioma cells were examined by cell proliferation assay, colony formation assay, wound-healing assay, and transwell assay. Also, the effects of lnc-ZNF281 on AKT/GSK-3β/β-catenin pathway were analyzed. The results showed that the expression of lnc-ZNF281 in glioma tissues was decreased compare

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9

Fahmé, Pia, Farah Ramadan, Diep Tien Le, et al. "The Intricate Interplay between the ZNF217 Oncogene and Epigenetic Processes Shapes Tumor Progression." Cancers 14, no. 24 (2022): 6043. http://dx.doi.org/10.3390/cancers14246043.

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The oncogenic transcription factor ZNF217 orchestrates several molecular signaling networks to reprogram integrated circuits governing hallmark capabilities within cancer cells. High levels of ZNF217 expression provide advantages to a specific subset of cancer cells to reprogram tumor progression, drug resistance and cancer cell plasticity. ZNF217 expression level, thus, provides a powerful biomarker of poor prognosis and a predictive biomarker for anticancer therapies. Cancer epigenetic mechanisms are well known to support the acquisition of hallmark characteristics during oncogenesis. Howeve

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10

Kubanov, A. A., A. A. Kubanova, A. E. Karamova, and A. A. Mineyeva. "Prevalence of genetic risk factors of psoriasis among the population of the Russian Federation." Vestnik dermatologii i venerologii 90, no. 6 (2014): 69–76. http://dx.doi.org/10.25208/0042-4609-2014-90-6-69-76.

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Goal. To assess the prevalence of polymorphisms of genes of the predisposition to psoriasis among the population of the Russian Federation. Materials and methods. The authors examined 546 psoriasis patients and 206 healthy people. The polymorphism of the following genes was assessed: genes encoding proteins of the signaling pathway of the nuclear transcription factor kappa-B - NF-κΒ (NFKBI, TRAF3IP2, TNFAIP3, REL, TYK2, TNIP1, IL-28RA) responsible for congenital immunity; genes participating in the IL-23 signaling pathway responsible for adaptive immunity (IL-23R, IL-12B); genes participating

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