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Relevant bibliographies by topics / Zoster virus (VZV)

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Academic literature on the topic 'Zoster virus (VZV)'

Author: Grafiati

Published: 4 June 2025

Last updated: 1 August 2025

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Journal articles on the topic "Zoster virus (VZV)"

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Arvin, A. M. "Varicella-zoster virus." Clinical Microbiology Reviews 9, no. 3 (1996): 361–81. http://dx.doi.org/10.1128/cmr.9.3.361.

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Varicella-zoster virus (VZV) is a ubiquitous human alphaherpesvirus that causes varicella (chicken pox) and herpes zoster (shingles). Varicella is a common childhood illness, characterized by fever, viremia, and scattered vesicular lesions of the skin. As is characteristic of the alphaherpesviruses, VZV establishes latency in cells of the dorsal root ganglia. Herpes zoster, caused by VZV reactivation, is a localized, painful, vesicular rash involving one or adjacent dermatomes. The incidence of herpes zoster increases with age or immunosuppression. The VZV virion consists of a nucleocapsid sur

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Ehrchen, Jan, and Matthias Hoy. "Varizella Zoster." Aktuelle Dermatologie 50, no. 07 (2024): 321–28. http://dx.doi.org/10.1055/a-2312-5252.

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ZusammenfassungDas Varizella-zoster-Virus (VZV) ist Erreger der Varizellen (Primärinfektion) und des Herpes zoster (Rezidiv). Die Erstinfektion erfolgt meist im Kindesalter über die respiratorische Route. Es kommt dabei zu einer systemischen Infektion mit dem VZV und an der Haut zu dem typischen VZV-Exanthem. Das VZV-Virus persistiert nach der Primärinfektion lebenslang in den sensorischen Spinalganglien. Im Laufe des Lebens kann es aus den Spinalganglien zu einer Reaktivierung des VZV kommen. Das VZV gelangt dabei entlang der sensorischen Nerven in das entsprechende Dermatom und verursacht in

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Bakradze, Ekaterina, Kathryn F. Kirchoff, Daniel Antoniello, et al. "Varicella Zoster Virus Vasculitis and Adult Cerebrovascular Disease." Neurohospitalist 9, no. 4 (2019): 203–8. http://dx.doi.org/10.1177/1941874419845732.

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The role of Varicella zoster virus (VZV) in neurological illness, particularly cerebrovascular disease, has been increasingly recognized. Primary infection by VZV causes varicella (chickenpox), after which the virus remains latent in neuronal ganglia. Later, during aging or immunosuppression, the virus can reactivate causing zoster (shingles). Virus reactivation can also spread to cerebral arteries causing vasculitis and stroke. Zoster is a recognized risk factor for stroke, but stroke can occur without preceding zoster rash. The diagnosis of VZV cerebral vasculitis is established by abnormal

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James, Stephanie, Alexander Choe, Igor Traktinskiy, Maria Nagel, and Donald Gilden. "Varicella zoster virus can reactivate and infect cerebral adventital fibroblasts, suppressing Jak/STAT and causing stroke. (168.35)." Journal of Immunology 188, no. 1_Supplement (2012): 168.35. http://dx.doi.org/10.4049/jimmunol.188.supp.168.35.

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Abstract Primary varicella zoster virus (VZV) infection causes varicella (chickenpox), after which virus becomes latent in ganglionic neurons. A declining cell-mediated immunity to VZV leads to virus reactivation, manifesting as zoster (shingles). Zoster is often complicated by chronic pain (post herpetic neuralgia) and other serious neurological disorders such as meningoencephalitis and myelitis. VZV vasculopathy results in transient ischemic attacks and stroke, and is the result of productive virus infection in cerebral arteries. More than 90% of the 6.75 billion world population harbors lat

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KANAI, Kyosuke, Souichi YAMADA, and Naoki INOUE. "Varicella-zoster virus (VZV)." Uirusu 60, no. 2 (2010): 197–208. http://dx.doi.org/10.2222/jsv.60.197.

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Reichelt, Mike, Leigh Zerboni, and Ann M. Arvin. "Mechanisms of Varicella-Zoster Virus Neuropathogenesis in Human Dorsal Root Ganglia." Journal of Virology 82, no. 8 (2008): 3971–83. http://dx.doi.org/10.1128/jvi.02592-07.

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ABSTRACT Varicella-zoster virus (VZV) is a human alphaherpesvirus that infects sensory ganglia and reactivates from latency to cause herpes zoster. VZV replication was examined in human dorsal root ganglion (DRG) xenografts in mice with severe combined immunodeficiency using multiscale correlative immunofluorescence and electron microscopy. These experiments showed the presence of VZV genomic DNA, viral proteins, and virion production in both neurons and satellite cells within DRG. Furthermore, the multiscale analysis of VZV-host cell interactions revealed virus-induced cell-cell fusion and po

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Navarrete Cuadrado, Katlin Susana, Maleivy Mastrodomenico Vargas, Hendry Guao Salinas, and Ornella Ruiz Pérez. "VARICELA ZOSTER." Biociencias 15, no. 1 (2020): 95–107. http://dx.doi.org/10.18041/2390-0512/biociencias.1.6364.

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La varicela es una enfermedad infecto-contagiosa, producida por el virus varicela-zoster (VVZ); es un virus alfaherpes que se relaciona más estrechamente con el virus del herpes simple, se presenta cuando el virus entra en contacto con la mucosa del tracto respiratorio superior o la conjuntiva de una persona susceptible, la transmisión de persona a persona se puede presentar por contacto directo con lesiones de VZV o por propagación en el aire. La reinfección con VZV es rara. El período de incubación es de 10 a 21 días; los síntomas que generalmente se presentan son fiebre, malestar general, a

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Çalışkan, Emel. "Evaluation of varicella zoster virus IgM, IgG and avidity results used in the diagnosis of varicella zoster virus." Anadolu Kliniği Tıp Bilimleri Dergisi 30, no. 2 (2025): 185–90. https://doi.org/10.21673/anadoluklin.1641218.

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Aim: Varicella zoster virus (VZV) is a disease that is rapidly transmitted through the respiratory tract and has a high seroprevalence worldwide. This study aimed to evaluate the results of VZV IgM, VZV IgG and VZV avidity serological tests studied in our laboratory. Methods: In this study, the VZV IgM, VZV IgG, and VZV avidity test results in serum samples sent to our laboratory for preliminary diagnosis or screening of chickenpox between November 2019 and December 2024 were retrospectively examined. Kruskal-Wallis and Chi-Square tests were used to evaluate the results according to mean age,

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Patil, Pankaj Shahajirao, Pankaj S. Patil, and Rajendra S. Bhambar. "Effective Natural Drug Remedies against Herpes Zoster: A Review." Journal of Drug Delivery and Therapeutics 10, no. 6-s (2020): 112–18. http://dx.doi.org/10.22270/jddt.v10i6-s.4599.

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Herpes zoster (HZ), also known as shingles, is a painful vesicular rash resulting from reactivation of the virus that also causes chickenpox – Varicella zoster virus (VZV). Typically, the rash runs its course in a matter of 4-5 weeks. The pain, however, may persist months, even years, after the skin heals. This phenomenon is known as postherpetic neuralgia (PHN). This review tried to provide more comprehensive and accurate data on the effects of different herbals on the VZV as a probable alternative treatment for VZV. Further clarification of the herbals interactions with VZV is required which

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Atılan, Kübra. "Varicella-Zoster Virus Infections: Epidemiology, Diagnosis and Vaccine Safety." Life and Medical Sciences 3, no. 1 (2024): 5–15. https://doi.org/10.54584/lms.2024.46.

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<strong>Özet</strong> <em>Orthoherpesviridae</em> ailesi üyesi bir alfaherpesvirus olan varicellovirus humanalpha3 (varicella-zoster virus; VZV) dünya genelinde yaygın ve onlarca yıldır kullanılan koruyucu aşısı bulunan bir insan patojenidir. Primer enfeksiyon, iyileşme sonrası VZV'nin gangliyonik nöronlarda latent olarak kaldığı suçiçeğine neden olur. Çocukluk döneminde genel olarak hafif seyirli enfeksiyonlarla ilişkili olan primer VZV enfeksiyonu, yenidoğanlar, bağışıklık sistemi baskılanmış kişiler veya yaşlılarda şiddetli ve pot

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Dissertations / Theses on the topic "Zoster virus (VZV)"

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Pothineni, Venkata Raveendra. "Proteome-wide production of monoclonal antibodies and study of intracellular localisation for Varicella-zoster virus (VZV)." Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-124114.

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Kleemann, Patrick [Verfasser]. "Mass spectrometric identification of Varicella-Zoster Virus (VZV) proteins recognized by human T cells / Patrick Kleemann." Mainz : Universitätsbibliothek Mainz, 2012. http://d-nb.info/1019669047/34.

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Gutzeit, Cindy. "Interference of Varicella-Zoster Virus (VZV) with the CD1 antigen presenting system on immature dendritic cells." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2009. http://dx.doi.org/10.18452/16059.

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Das human pathogene Varicella-Zoster Virus (VZV) gehört zur Familie der Herpesviren und ist weltweit verbreitet. Die Primärinfektion verursacht Varicellen, welche durch einen bläschenartigen Hautausschlag charakterisiert ist. Im Anschluss daran etabliert VZV eine lebenslange Latenz und verursacht nach Reaktivierung Herpes Zoster. Seit 2004 ist der Lebendimpfstoff aus attenuierten Virionen des VZV-Stammes V-Oka in Deutschland empfohlen. Im Gegensatz zur Infektion mit zirkulierenden virulenten VZV Stämmen tritt nach Verimpfung des Vakzin-Stammes V-Oka kein Exanthem auf. Die Haut ist der Hauptrep

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Henriquez, Rodney Fabian. "Varicella Zoster Virus infection of human matured monocyte derived dendritic cells & its impact on their effector functions." Thesis, The University of Sydney, 2015. http://hdl.handle.net/2123/13624.

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Varicella Zoster Virus (VZV) is a species specific alpha human herpes virus that causes both Varicella (chickenpox) and Herpes Zoster (shingles) in human hosts. It has been hypothesized that host dendritic cells (DC) play a pivotal role in the pathogenesis of VZV during the early stages of infection. DC have previously been shown to be permissive to VZV infection and capable of transmitting the virus to human T lymphocytes. VZV has also been shown to employ a variety of mechanisms to evade detection and clearance by the host immune system. The primary aim of this study was to identify the und

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Brändli, Lukas. "Inhibition einer Protein-Protein-Wechselwirkung : Strukturbasiertes Design, Synthese und Evaluation von wirkstoffähnlichen Inhibitoren der Dimerisierung der Varicella Zoster Virus Thymidin Kinase (VZV TK) /." kostenfrei, 2007. http://e-collection.ethbib.ethz.ch/view/eth:29808.

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Gutzeit, Cindy [Verfasser], Carmen [Gutachter] Scheibenbogen, Detlev H. [Gutachter] Krüger, and Andreas [Gutachter] Sauerbrei. "Interference of Varicella-Zoster Virus (VZV) with the CD1 antigen presenting system on immature dendritic cells / Cindy Gutzeit ; Gutachter: Carmen Scheibenbogen, Detlev H. Krüger, Andreas Sauerbrei." Berlin : Humboldt-Universität zu Berlin, 2009. http://d-nb.info/1208074458/34.

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Yang, Min. "Analysis of the regulation of a bi-directional varicella-zoster virus (VZV) promoter The ORF 28/29 promoter /." 2005. http://proquest.umi.com/pqdweb?did=888847091&sid=11&Fmt=2&clientId=39334&RQT=309&VName=PQD.

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Thesis (Ph.D.)--State University of New York at Buffalo, 2005.<br>Title from PDF title page (viewed on May 11, 2006) Available through UMI ProQuest Digital Dissertations. Thesis adviser: Ruyechan, William T. Includes bibliographical references.

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Gutzeit, Cindy [Verfasser]. "Interference of Varicella-Zoster virus (VZV) with the CD1 antigen presenting system on immature dendritic cells / von Cindy Gutzeit." 2009. http://d-nb.info/1002215315/34.

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Pothineni, Venkata Raveendra [Verfasser]. "Proteome-wide production of monoclonal antibodies and study of intracellular localisation for Varicella-zoster virus (VZV) / vorgelegt von Venkata Raveendra Pothineni." 2010. http://d-nb.info/1011065282/34.

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Diop, Ndeye Soukeyna. "Association entre l'utilisation de la prophylaxie antivirale et la virémie du cytomégalovirus et du virus Epstein-Barr chez les receveurs pédiatriques d'une greffe de cellules souches hématopoïétiques allogéniques." Thesis, 2020. http://hdl.handle.net/1866/25216.

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Les infections virales en particulier celles dues aux virus de la famille des Herpesviridae pendant la période d’aplasie et de lymphopénie à la suite d’une greffe de cellules souches hématopoïétiques (GCSH) peuvent occasionner des complications très graves, souvent associées à une morbidité et mortalité élevées. Les recommandations cliniques actuelles préconisent l’utilisation des antiviraux pour la prévention de certaines de ces infections. L’efficacité du famciclovir et de l’acyclovir contre les virus de l’herpès simplex (HSV), le virus varicella-zoster (VZV) et l’herpésvirus humain de type

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Books on the topic "Zoster virus (VZV)"

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Ilobi, Chuks P. Synthesis and immunological reactivity of varicella-zoster virus (VZV) antigens, and VZV-carrier cultures. University of Birmingham, 1988.

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Gilden, Don, Randall J. Cohrs, Ravi Mahalingam, and Maria A. Nagel. Varicella Zoster Virus Infection of the Nervous System. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0149.

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Varicella zoster virus (VZV) is a human herpesvirus that causes varicella (chickenpox), after which virus becomes latent in ganglionic neurons along the entire neuraxis. Reactivation of VZV due to a decline in the cell-mediated immune response to VZV in elderly or immunocompromised individuals causes zoster (shingles), frequently complicated by chronic pain (postherpetic neuralgia) and serious neurological disease (meningoencephalitis, myelitis and VZV vasculopathy due to retrograde spread of virus after zoster. Here, we describe clinical, laboratory and pathological features of neurological c

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Picone, Olivier, Christelle Vauloup-Fellous, and Laurent Mandelbrot. Varicella-Zoster Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0014.

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Chickenpox in a pregnant woman is uncommon, but it is a major concern for patients and their families, as well as for clinicians caring for pregnant women. Varicella infection during pregnancy is usually benign, but there can be serious consequences for both mother and child. Notably, fetal varicella syndrome (FVS) can happen when infection occurs before 21 weeks of gestation. It can present with serious neurological anomalies and unusual cicatricial skin lesions. Later in pregnancy, primary neonatal varicella may occur when the mother is infected in the peripartum period, and it can be life-t

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Miller, Aaron E., and Teresa M. DeAngelis. Varicella Zoster Myelopathy. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0010.

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Varicella zoster virus (VZV) myelopathy is a rare cause of infectious myelitis and can present in both immunocompetent as well as immunocompromised individuals. In this chapter, we review the two most common presentations of VZV myelopathy, the optimal diagnostic testing and current therapeutic approaches.

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Book chapters on the topic "Zoster virus (VZV)"

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Hengel, Hartmut. "Varicella-zoster-Virus (VZV)." In Lexikon der Infektionskrankheiten des Menschen. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-39026-8_1154.

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Neumann, G., H. H. Feucht, W. Becker, and M. Späth. "Varicella-Zoster-Virus (VZV)." In Gynäkologische Infektionen. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-05268-2_43.

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Tabery, Helena M. "Comparison of HSV and VZV Epithelial Keratitis." In Varicella-Zoster Virus Epithelial Keratitis in Herpes Zoster Ophthalmicus. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-14487-5_5.

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Tabery, Helena M. "Recurrent VZV Epithelial Keratitis in HZO; HZO sine Herpete." In Varicella-Zoster Virus Epithelial Keratitis in Herpes Zoster Ophthalmicus. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-14487-5_3.

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Takahashi, M., H. Kamiya, Y. Asano, et al. "Immunization of the elderly to boost immunity against varicella-zoster virus (VZV) as assessed by VZV skin test reaction." In Immunity to and Prevention of Herpes Zoster. Springer Vienna, 2001. http://dx.doi.org/10.1007/978-3-7091-6259-0_17.

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Gelb, Lawrence D., Susan G. Adams, and Dennis E. Dohner. "Differentiation between the Oka Varicella Vaccine Virus and American Wild-Type Varicella-Zoster Virus (VZV)." In Immunobiology and Prophylaxis of Human Herpesvirus Infections. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5853-4_7.

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Lönnqvist, B., P. Ljungman, P. Bolme, G. Gahrton, O. Ringdén, and B. Wahren. "Frequency of Reactivation of Varicella-Zoster Virus (VZV) Infection with and without Symptoms After Bone Marrow Transplantation." In 11th Annual meeting of the EBMT. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-662-40457-7_52.

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Azarkh, Yevgeniy, Don Gilden, and Randall J. Cohrs. "Molecular Characterization of Varicella Zoster Virus in Latently Infected Human Ganglia: Physical State and Abundance of VZV DNA, Quantitation of Viral Transcripts and Detection of VZV-Specific Proteins." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/82_2009_2.

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Weinberg, Jeffrey M. "Varicella-zoster virus." In Schlossberg's Clinical Infectious Disease, edited by Cheston B. Cunha. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190888367.003.0192.

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This chapter focuses on the varicella-zoster virus (VZV), a member of the herpesviruses family and the etiologic agent of varicella and varicella-zoster. Varicella is the exanthem caused by primary infection with VZV, and shingles is the clinical syndrome of segmental, unilateral exanthem and neuralgic pain due to reactivation of latent VZV infection. Both primary and reactivated VZV infection can lead to severe generalized virus dissemination in an immunodeficient person. The chapter discusses the availability of antiviral agents for management of VZV infection, which has raised the importanc

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Craighead, John E. "Varicella-Zoster Virus (VZV)." In Pathology and Pathogenesis of Human Viral Disease. Elsevier, 2000. http://dx.doi.org/10.1016/b978-012195160-3/50011-x.

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Conference papers on the topic "Zoster virus (VZV)"

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Dantas, Madson Caio dos Santos, and João Pedro Cardoso Prudêncio. "Acute cerebellar ataxia associated with varicella zoster virus encephalitis." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.423.

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Context: Varicella-zoster virus (VZV) primary infection causes a diffuse vesicular rash and affects mainly young people. VZV-associated encephalitis is a rare complication since the advent of vaccination, and can present as different neurological syndromes. This report aims to describe a case of acute cerebellar ataxia after VZV-associated encephalitis in a child, admitted to the Onofre Lopes University Hospital (HUOL) in Natal, Brazil. Case report: We present the case of a 9-year-old girl referred to HUOL with polymorphic skin lesions for 8 days. She evolved with headache, vomiting, drowsines

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Mendonça, Amanda Miranda de, Ana Luisa Vieira de Faria, Matheus Alves da Silva, et al. "Hemorrhagic demyelinating lesions and systemic thrombosis due to Varicella Zoster infection: a case report." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.426.

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Introduction: The investigation of hemorrhagic demyelinating cerebral lesions is challenging with diverse etiologies. We report a case of varicella zoster virus (VZV) infection with prominent hemorrhagic demyelinating component and systemic thrombosis. Objectives: Describe a case of hemorrhagic demyelinating lesions and systemic thrombosis secondary to varicella zoster central nervous system infection. Case report: A 45-year-old man presented with a one-week history of left leg strength loss associated with pyramidal liberation on the left side. Brain MRI showed two expansive hemorrhagic lesio

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Rabiee, Behnam, Chandani Patel, Mansab Jafri, et al. "Herpesviriae Infection of the Corneal Endothelium." In 27th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2023. https://doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.29_2023.

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Background - The corneal endothelium plays a vital role in maintaining corneal clarity by regulating the amount of fluid in the corneal stroma. - Corneal endotheliitis is defined as inflammation of the corneal endothelial layer that leads to corneal edema and haziness, and subsequent loss of vision. - Most common causes include cytomegalovirus (CMV), herpes simplex virus (HSV), and varicella zoster virus (VZV). - Because corneal endothelial cells cannot regenerate following injury, early diagnosis is essential in proper management and preventing loss of corneal endothelial cells. In this revie

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Gul, Sedat, Adeenah Ahmed, Corey McGraw, and Ruham Alshiekh Nasany. "MRI negative Varicella zoster virus (VZV) plexopathy in a young man with history of malignancy (P2-4.001)." In 2023 Annual Meeting Abstracts. Lippincott Williams & Wilkins, 2023. http://dx.doi.org/10.1212/wnl.0000000000202360.

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Rattu, Mohammad A., and Wayne Tamaska. "A Case of HHV-6 Viral Meningitis." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.87.

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Meningitis is the inflammation of the meninges and associated with abnormal cell count in the cerebrospinal fluid (CSF). The lack of bacterial growth in cultures, most commonly referred to as aseptic meningitis, is frequently caused by viruses [2]. Viruses have become more common as the prevalence of bacterial meningitis has decreased secondary to vaccination use, with viral meningitis being the most common form among countries. Viral meningitis will present with fever, headache, photophobia, neck stiffness and nausea and vomiting. Younger children however may not show any signs of meningeal i

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Tahboub, M., A. Al-Shyoukh, L. Numan, O. Abughanimeh, M. Younis, and A. Gohar. "A Lethal Combination of Disseminated Herpes Simplex Virus-1 (HSV-1) and Varicella Zoster Virus (VZV) Co-Infection in an Immunosuppressed Adult." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6587.

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Sharma, Kanika, PRAMOD JHA, Shreyashi Khanal, and Shitiz Sriwastava. "Herpes Simplex Virus (HSV), Varicella-Zoster Virus (VZV), and Cryptococcal Infections Associated With Sphingosine-1-Phosphate Receptor (S1PR) Modulators: A Review (P12-10.005)." In 2023 Annual Meeting Abstracts. Lippincott Williams & Wilkins, 2023. http://dx.doi.org/10.1212/wnl.0000000000203827.

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Almanzar, G., F. Baum, M. Steimer, et al. "POS0454 JANUS-KINASE-INHIBITORS (JAKi) SIGNIFICANTLY IMPAIR SIGNAL-TRANSDUCER-AND-ACTIVATOR-OF-TRANSCRIPTION (STAT) SIGNALING OF VARICELLA-ZOSTER-VIRUS-(VZV)-SPECIFIC T-CELL MEMORY RESPONSES." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.6038.

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Nogueira, Raniery Augusto dos Santos Beserra, Ana Beatriz Silva Barbosa, Francisco Das Chagas Diassis Jácome Valentim, Sâmya Pires Batista De Azevêdo, and Jamile Rodrigues Cosme De Holanda. "MECANISMOS DE MANUTENÇÃO DA LATÊNCIA DO VÍRUS VARICELA ZÓSTER: UMA ADAPTAÇÃO NECESSÁRIA." In I Congresso Nacional de Microbiologia Clínica On-Line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1182.

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Introdução: O Vírus Varicela Zóster pertence à subfamília α-herpesviridae e é um DNA-vírus de fita dupla, possuindo 68 Fases de Leitura Aberta (ORF) únicas. Sendo responsável por varicela, em um primeiro contato, o vírus mantém-se latente por um longo período em gânglios das raízes dorsais e de nervos cranianos, disseminando-se por essas estruturas após décadas, quando reativado, configurando Herpes Zóster, cujas complicações prejudicam a qualidade de vida dos pacientes. Posteriormente à infecção primária, o vírus classifica-se como neurotrópico e seus meios de sobrevida em neurônios, ainda nã

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