Книги: "Infections à Burkholderia cepacia"

1

Williams, Kerry Ann. Exopolysaccharide production in Burkholderia cepacia. Manchester: University of Manchester, 1997.

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2

McNeely, Damian. Biocontrol of multidrug resistant Burkholderia cepacia. [S.l: The Author], 2004.

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3

Desai, Maya. Antibiotic resistance of burkholderia cepacia in patients with cystic fibrosis. Birmingham: University of Birmingham, 1999.

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4

McKenney, David. The cell surface and extracellular virulence determinants of Burkholderia Cepacia. Manchester: University of Manchester, 1994.

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5

Baxter, Ian Anthony. Studies of the nature of Burkholderia cepacia in cystic fibrosis. Birmingham: Aston University. Department of Pharmaceutical and Biological Sciences, 1996.

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6

Lawrence, J. R. In-situ bioremediation of trichloroethylene using Burkholderia cepacia G4 PR1: Analysis of transport parameters for risk assessment. Gulf Breeze, FL: U.S. Environmental Protection Agency, National Health and Environmental Effects Research Laboratory, 1998.

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7

Lawrence, J. R. In-situ bioremediation of trichloroethylene using Burkholderia cepacia G4 PR1: Analysis of transport parameters for risk assessment. Gulf Breeze, FL: U.S. Environmental Protection Agency, National Health and Environmental Effects Research Laboratory, 1998.

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8

Lawrence, J. R. In-situ bioremediation of trichloroethylene using Burkholderia cepacia G4 PR1: Analysis of transport parameters for risk assessment. Gulf Breeze, FL: U.S. Environmental Protection Agency, National Health and Environmental Effects Research Laboratory, 1998.

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9

Lawrence, J. R. In-situ bioremediation of trichloroethylene using Burkholderia cepacia G4 PR1: Analysis of transport parameters for risk assessment. Gulf Breeze, FL: U.S. Environmental Protection Agency, National Health and Environmental Effects Research Laboratory, 1998.

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10

Lawrence, J. R. In-situ bioremediation of trichloroethylene using Burkholderia cepacia G4 PR1: Analysis of transport parameters for risk assessment. Gulf Breeze, FL: U.S. Environmental Protection Agency, National Health and Environmental Effects Research Laboratory, 1998.

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11

Lawrence, J. R. In-situ bioremediation of trichloroethylene using Burkholderia cepacia G4 PR1: Analysis of transport parameters for risk assessment. Gulf Breeze, FL: U.S. Environmental Protection Agency, National Health and Environmental Effects Research Laboratory, 1998.

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12

Snyder, Richard A. In situ bioremediation of trichloroethylene using Burkholderia cepacia G4 PR1: Analysis of microbial ecology parameters for risk assessment. Gulf Breeze, FL: U.S. Environmental Protection Agency, Research and Development, National Health and Environmental Effects Research Laboratory, 1998.

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13

Snyder, Richard A. In situ bioremediation of trichloroethylene using Burkholderia cepacia G4 PR1: Analysis of microbial ecology parameters for risk assessment. Gulf Breeze, FL: U.S. Environmental Protection Agency, Research and Development, National Health and Environmental Effects Research Laboratory, 1998.

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14

Snyder, Richard A. In situ bioremediation of trichloroethylene using Burkholderia cepacia G4 PR1: Analysis of microbial ecology parameters for risk assessment. Gulf Breeze, FL: U.S. Environmental Protection Agency, Research and Development, National Health and Environmental Effects Research Laboratory, 1998.

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15

Snyder, Richard A. In situ bioremediation of trichloroethylene using Burkholderia cepacia G4 PR1: Analysis of microbial ecology parameters for risk assessment. Gulf Breeze, FL: U.S. Environmental Protection Agency, Research and Development, National Health and Environmental Effects Research Laboratory, 1998.

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16

Govan, John, and Andrew Jones. Microbiology of CF lung disease. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198702948.003.0003.

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This chapter presents the microbiology of CF and describes the classical bacterial pathogens including Staphylococcus aureus, Haemophilus influenza, Pseudomonas aeruginosa and organisms of the Burkholderia cepacia complex. The dominant of these is P. aeruginosa. Infections with other opportunistic pathogens including non-tuberculous mycobacteria, Stenotrophomonas maltophila, and Achromobacter (Alcaligenes) xylosoxidans are also encountered. This chapter details measures to prevent the onset of chronic infection with these organisms include regular screening of respiratory tract samples for bacterial pathogens and the use of aggressive antibiotic therapy to eradicate initial infection before the pathogen can adapt to the environment of the CF lung. Patient-to-patient spread of transmissible strains of bacterial pathogens has led to the implementation of strict infection control measures at CF centres, including patient segregation. In addition to bacterial pathogens, the contribution of fungal infection in CF lung disease is increasingly recognized.

17

Weingart, Christine L. A phospholipase C produced by Burkholderia cepacia. 1997.

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18

Weingart, Christine L. A phospholipase C produced by Burkholderia cepacia. 1997.

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19

L, McCarty Perry, and National Health and Environmental Effects Research Laboratory (U.S.), eds. Bioaugmentation with Burkholderia cepacia PR1₃₀₁ for in situ bioremediation of trichloroethylene contaminated water. Gulf Breeze, FL: U.S. Environmental Protection Agency, National Health and Environmental Effects Research Laboratory, 1998.

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20

Bioaugmentation with Burkholderia cepacia PR1₃₀₁ for in situ bioremediation of trichloroethylene contaminated water. Gulf Breeze, FL: U.S. Environmental Protection Agency, National Health and Environmental Effects Research Laboratory, 1998.

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21

L, McCarty Perry, and National Health and Environmental Effects Research Laboratory (U.S.), eds. Bioaugmentation with Burkholderia cepacia PR1₃₀₁ for in situ bioremediation of trichloroethylene contaminated water. Gulf Breeze, FL: U.S. Environmental Protection Agency, National Health and Environmental Effects Research Laboratory, 1998.

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22

Ramachandran, Raja, and Vivekanand Jha. Renal involvement in other infections. Edited by Vivekanand Jha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0198_update_001.

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Renal involvement has been described in patients with many other infections and this chapter discusses several of these.Water-borne infections are a common cause of acute kidney injury (AKI) worldwide but especially in tropical regions. Cholera is notoriously dangerous but any other cause of fluid-depletion may achieve the same. Typhoid fever is more likely to cause AKI from its complications than directly, but a small proportion of patients have glomerulonephritis.Meliodosis is caused by the intracellular organism Burkholderia pseudomallei. It typically affects workers in paddy (rice) fields in the rainy season, and may cause a local, genitourinary infection or an acute melioidosis septicaemia with a high incidence of AKI and mortality. Those with other chronic conditions are at greatest risk.Obstetric infections as a result of unsafe deliberate abortion or post-partum are a very common (often the most common) cause of AKI in developing countries, and a major cause of avoidable death in young women.

Книги з теми "Infections à Burkholderia cepacia"

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