Готові списки джерел за темами / Premalignant

Зміст

  1. Статті в журналах
  2. Дисертації
  3. Книги
  4. Частини книг
  5. Тези доповідей конференцій
  6. Звіти організацій

Добірка наукової літератури з теми "Premalignant"

Автор: Grafiati
Опубліковано: 3 червня 2025
Оновлено: 31 липня 2025

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Статті в журналах з теми "Premalignant"

1

Namba, Ruria, Jeannie E. Maglione, Lawrence J. T. Young, et al. "Molecular Characterization of the Transition to Malignancy in a Genetically Engineered Mouse-Based Model of Ductal Carcinoma In situ." Molecular Cancer Research 2, no. 8 (2004): 453–63. http://dx.doi.org/10.1158/1541-7786.453.2.8.

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Abstract A transplantable model of human ductal carcinoma in situ that progresses to invasive carcinoma was developed from a genetically engineered mouse (GEM). Additional lines were established using early mammary premalignant lesions from transgenic MMTV-PyV-mT mice. These lines were verified to be premalignant and transplanted repeatedly to establish stable and predictable properties. Here, we report the first in-depth molecular analysis of neoplastic progression occurring in one premalignant transplantable GEM-derived line. Oligonucleotide microarrays showed that many genes are differentia
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2

Gündüz, Reyhan, Elif Ağaçayak, Gülcan Okutucu, Ulaş Alabalik, and Mehmet Sıddık Evsen. "Evaluation of definitive histopathological results of patients diagnosed with endometrial polyps: a tertiary care center experience." African Health Sciences 22, no. 1 (2022): 125–32. http://dx.doi.org/10.4314/ahs.v22i1.16.

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Objectives: To determine the premalignancy and malignancy prevalence in patients diagnosed with endometrial polyps and to investigate factors affecting premalignancy and malignancy.
 Methods: In our retrospective study, patients who were diagnosed with endometrial polyp with endometrial samples and who underwent polypectomy by hysteroscopy or hysterectomy within one year were included.
 Results: Premalignant / malignant histopathological results were detected in 7 (2.8%) patients. There were no statistically significant differences in histopathological results and endometrial samplin
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3

Blaisdell, Adam, Takiko Daikoku, Chin Siean Tay, Sudhansu Dey, and Adrian Erlebacher. "Neutrophil recruitment into premalignant lesions differentially influences tumor growth versus progression. (TUM4P.928)." Journal of Immunology 192, no. 1_Supplement (2014): 138.29. http://dx.doi.org/10.4049/jimmunol.192.supp.138.29.

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Abstract Neutrophils possess a diverse arsenal of enzymatic and chemical weapons that can inflict damage upon pathogens and host cells alike. Most tumors harbor neutrophils even from the earliest stages of progression (premalignancy), wherein these weapons could, in theory, either kill a tumor cell or enhance its malignant potential via mutagenesis. However, the interaction between neutrophils and premalignant tumor cells has not been well characterized. To this end, we utilized a mouse model of endometrial carcinoma with defined stages of premalignancy and malignancy that faithfully represent
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4

Stanton, Sasha E., Kristin G. Anderson, Tullia C. Bruno, et al. "SITC strategic vision: prevention, premalignant immunity, host and environmental factors." Journal for ImmunoTherapy of Cancer 13, no. 3 (2025): e010419. https://doi.org/10.1136/jitc-2024-010419.

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Cancer immunotherapy has improved the survival of a subset of patients by harnessing the power of the immune system to find and destroy malignant cells. The immune system also protects the host by destroying developing premalignant and malignant tumors. Advancing our knowledge of premalignant immunity and immune changes seen in lesions that develop into invasive cancer versus those that regress offers an exciting opportunity to leverage the immune system for immune prevention and immune interception of premalignancy. Understanding the immune environment of premalignant lesions and how chronic
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5

Ma, Peiwen, Pamela Beatty, John McKolanis, Robert Schoen, Randal Brand, and Olivera J. Finn. "Myeloid derived suppressor cells (MDSC) and anti-MUC1 immunosurveillance in pre-malignancy and cancer." Journal of Immunology 200, no. 1_Supplement (2018): 122.17. http://dx.doi.org/10.4049/jimmunol.200.supp.122.17.

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Abstract Recent work from our lab showed that in a colon cancer prevention trial of a tumor antigen MUC1 vaccine in patients with premalignant colon adenomas, those who did not make anti-MUC1 IgG, had higher pre-vaccination levels of circulating MDSC compared to vaccine responders. This was the first observation of increased MDSC in premalignancy. MDSC in premalignancy have not been compared to MDSC in cancer and our hypothesis is that there might be differences in the composition of various MDSC subpopulation and their immunosuppressive functions due to the shorter time of exposure to disease
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6

Spira, Avrum, Mary L. Disis, John T. Schiller, et al. "Leveraging premalignant biology for immune-based cancer prevention." Proceedings of the National Academy of Sciences 113, no. 39 (2016): 10750–58. http://dx.doi.org/10.1073/pnas.1608077113.

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Prevention is an essential component of cancer eradication. Next-generation sequencing of cancer genomes and epigenomes has defined large numbers of driver mutations and molecular subgroups, leading to therapeutic advances. By comparison, there is a relative paucity of such knowledge in premalignant neoplasia, which inherently limits the potential to develop precision prevention strategies. Studies on the interplay between germ-line and somatic events have elucidated genetic processes underlying premalignant progression and preventive targets. Emerging data hint at the immune system’s ability
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7

Nemakayala, Divyesh Reddy, Shilpa Tatineni, Ikponmwosa Enofe, Ling Wang, and Heather Laird-Fick. "Prevalence of gastric cancer and premalignant changes in a Michigan cohort, 2013-2017." Journal of Clinical Oncology 37, no. 15_suppl (2019): e15539-e15539. http://dx.doi.org/10.1200/jco.2019.37.15_suppl.e15539.

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e15539 Background: Gastric cancer (GC) is uncommon in the US. Incidence varies by geography, race/ethnicity, sex, age, and socioeconomic status (SES). H. pylori is a risk factor for premalignant lesions and GC. Prior studies estimate risk of progression from premalignant to GC at 0.1-0.5% per year. US prevalence of premalignant lesions is unclear. This study describes the prevalence of premalignant lesions and GC in a large community-based sample. Methods: Patients aged ≥18 years undergoing EGD with gastric biopsy in Sparrow Health System were identified. Pathology reports were abstracted for
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8

Zhou, Xian, Jianhao Zeng, Alexys T. Riddick, Victor H. Engelhard, and Hui Zong. "Premalignancy induced B cell-centered immune aggregates for immune-prevention against triple negative breast cancer." Journal of Immunology 210, no. 1_Supplement (2023): 88.10. http://dx.doi.org/10.4049/jimmunol.210.supp.88.10.

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Abstract Immune surveillance is believed to be a critical mechanism to eradicate premalignant mutant cells to prevent cancer. However, the cellular organization and mechanisms for immune surveillance remain largely unknown because premalignant mutant cells are “invisible” in patient samples and conventional animal models. To gain a glimpse into this process, our lab developed a genetically engineered mouse model recapitulating human basal-like breast cancer through unequivocal labeling of premalignant p53-Brca1 mutant cells with GFP. Using this model, we discovered prominent immune aggregates
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9

Tatineni, Shilpa, Divyesh Reddy Nemakayala, Ikponmwosa Enofe, Ling Wang, and Heather Laird-Fick. "Prevalence of esophageal malignant and premalignant lesions in a Michigan cohort, 2013-2017." Journal of Clinical Oncology 37, no. 15_suppl (2019): e15541-e15541. http://dx.doi.org/10.1200/jco.2019.37.15_suppl.e15541.

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e15541 Background: Esophageal cancer is diagnosed in roughly 4 per 100,000 US population. Older men are most frequently affected. Adenocarcinoma is most common and incidence is increasing. EGD with biopsy is important for diagnosis. Many patients present with metastases, limiting treatment options. EGD can identify Barrett’s esophagus, a precursor lesion for adenocarcinoma, but evaluation of biopsy specimens is difficult. This study describes findings from esophageal biopsies in a large community-based Michigan cohort. Methods: Patients aged ≥18 years undergoing EGD with esophageal biopsies in
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10

Huber, Michaell A. "PREMALIGNANT LESIONS." Journal of the American Dental Association 139, no. 4 (2008): 395–96. http://dx.doi.org/10.14219/jada.archive.2008.0172.

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Дисертації з теми "Premalignant"

1

Zhang, Li. "Proliferation, differentiation and apoptosis in human oral premalignant and malignant lesions." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0004/MQ37675.pdf.

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2

Santosh, Neetha. "Expression of Cornulin, DPEP1, SOX4 and BUB3 in Oral Premalignant Lesions." The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1467329505.

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3

Steinbeck, Rüdiger G. "Mitotic failure and genome stability in benign, premalignant and malignant human tissues /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19980611stei.

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4

Mui, Kin-cheong, and 梅堅祥. "Inactivation of DNA match repair proteins in premalignant lesions in Lynch syndrome." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44659635.

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5

McConnell, Dynes Tracey. "Vulval squamous cell carcinoma and premalignant skin conditions : a population-based study." Thesis, University of Aberdeen, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.301229.

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Incidence trends for invasive and non-invasive disease are explored. A nested case control study of those women who progress from non-invasive disease to carcinoma is performed. The incidence of vulval carcinoma in Grampian remains stable. Incidence rates of all non-invasive chronic vulval skin disorders, regardless of underlying aetiology are rising dramatically. Age specific incidence rates suggest that there may be significant ascertainment bias in rising incidence rates for each of these skin disorders. No distinction could be found between the relative risks for women with vulval litchen
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6

Yap, Jason Ker Wei. "Improving the outcome of patients with premalignant and malignant disorders of the vulva." Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/6533/.

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Research presented in this thesis was driven by the need to identify risk factors that predict local recurrence (LVR) in patients with vulval cancer (VSCC), and the need for more effective treatments for women with vulvar intraepithelial neoplasia (VIN). To identify the risk factors that predispose women to LVR, a multivariate analysis was performed on a well-characterized cohort of women treated for VSCC. This analysis revealed that the only independent predictor of LVR was the presence of Lichen Sclerosis (LS). These women were five times more likely to recur than those without LS. VIN is a
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7

Hamdoon, Z. G. "The clinical application of optical coherence tomography for head and neck premalignant/malignant lesions." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1402413/.

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The principle of Optical Coherence Tomography (OCT) is based on the property of light coherence. OCT generates cross-sectional images of two-dimensional objects to obtain in-vitro and in-vivo images of tissues. Non–commercially available OCT systems, which have a higher resolution and scanning rate, have been previously reported. However, some clinical research has already been conducted using the first commercially available OCT device (Niris system) to image the larynx; but applications on oral and skin tissue have not been tested yet. This thesis aims to explore, compare and validate three
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8

Weir, Lucinda Roberta. "An immunohistochemical and molecular study of putative premalignant liver cell populations induced by Aflatoxin B1." Thesis, University of Leicester, 1997. http://hdl.handle.net/2381/29536.

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Alterations in the phenotypic and genotypic expression in foci of altered hepatocytes (FSH) and tumours in livers of rats treated with aflatoxin B1 (AFB1) were analysed. Expression of the phase I and phase II drug metabolising enzymes, P450 2C11, glutathione S-transferase (GST) 7-7, -glutamyl transferase and novel aldehyde reductase, with high activity towards AFB1-dihydrodiol, were examined. A monoclonal antibody recognising GST Yc2, a phase II enzyme which displays high conjugating activity towards the AFB1-epoxide, was prepared and used to determine its expression in AFB1 treated livers. Ti
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9

Krupnik, Eduardo. "Evaluation of premalignant lesions of rat colon by [1]H nuclear magnetic resonance and infrared spectroscopy." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ35043.pdf.

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10

Odunsi, Adekunle Omatayo. "Immunogenetic analysis of HLA Class II in premalignant disease of the cervix and correlation with HPV status." Thesis, Open University, 1999. http://oro.open.ac.uk/54556/.

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The human papilloma virus (HPY) infection has a causal association with cervical intraepithelial neoplasia (CIN) and cervical cancer. However, pre-malignant or malignant transformation is not always observed with HPY infection. lILA molecules are important in the regulation of the immune response to foreign antigens. The role of genetic variation at the HLA class II loci (DR and DQ) in CIN was investigated in 176 British Caucasian patients and 420 controls (normal cervical cytology and negative for HPY 16, 18, 31 and 33). HLA DQB 1 *03 typing was performed by a novel polymerase chain reactionr
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Книги з теми "Premalignant"

1

Brennan, Peter A., Tom Aldridge, and Raghav C. Dwivedi, eds. Premalignant Conditions of the Oral Cavity. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-2931-9.

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2

L, Eastwood Gregory, ed. Premalignant conditions of the gastrointestinal tract. Elsevier, 1991.

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3

Albert, Singer, ed. Premalignant lesions of the lower genital tract. Elsevier, 1990.

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4

Julian, Katz, and Reynolds James C, eds. Clinical management of premalignant disorders of the gastrointestinal tract. W.B. Saunders, 2002.

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5

Harmsel, Willem Abraham ter. Studies on oncogenesis and prognostic factors in the premalignant and malignant uterine cervix. University of Leiden, 1998.

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6

Zeitels, Steven W. Premalignant epithelium and microinvasive cancer of the vocal fold: The evolution of phonomicrosurgical management. Layrngoscope Journal, 1995.

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7

Falk, Symposium (109th 1998 Titisee Germany). Colorectal cancer: Molecular mechanisms, premalignant state and its prevention : proceedings of the Falk Symposium 109 held in Titisee, Germany, October 14-15, 1998. Kluwer Academic Publishers, 1999.

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8

Falk Symposium (109th 1998 Titisee, Germany). Colorectal cancer: Molecular mechanisms, premalignant state and its prevention : proceedings of the Falk Symposium 109 held in Titisee, Germany, October 14-15, 1998. Kluwer Academic Publishers, 1999.

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9

Eitan, Yefenof, and Scheuermann Richard H, eds. Premalignancy and tumor dormancy. Springer, 1996.

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10

Eitan, Yefenof, and Scheuermann Richard H, eds. Premalignancy and tumor dormancy. R.G. Landes, 1996.

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Частини книг з теми "Premalignant"

1

Dallenbach-Hellweg, Gisela, and Hemming Poulsen. "Premalignant Lesions." In Atlas of Histopathology of the Cervix Uteri. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-662-21644-6_7.

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2

Li, Chia-Cheng, Zhe Li, Reshma S. Menon, and Sook-Bin Woo. "Premalignant Lesions." In Genomics, Personalized Medicine and Oral Disease. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-17942-1_12.

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3

Chan, Anthony W. H., Alberto Quaglia, Beate Haugk, and Alastair Burt. "Premalignant Lesions." In Atlas of Liver Pathology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9114-9_12.

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4

Sherban, Alex, and Matthew Keller. "Premalignant Neoplasms." In Roxburgh's Common Skin Diseases, 19th ed. CRC Press, 2022. http://dx.doi.org/10.1201/9781003105268-20.

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5

Dev, Bhawna, Sandhya Sundaram, Leena Dennis Joseph, Mehak Garg, and Mohana Priya. "Premalignant Lesions." In Holistic Approach to Breast Disease. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-0035-0_21.

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Braun-Falco, Otto, Gerd Plewig, Helmut H. Wolff, and Walter H. C. Burgdorf. "Premalignant Epithelial Tumors." In Dermatology. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-97931-6_55.

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7

Wong, Thomas T. F., Martin Mak, Hussain M. Alnajjar, and Wayne Lam. "Premalignant Penile Lesions." In Penile Cancer – A Practical Guide. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-32681-3_5.

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Khong, T. Yee, Annie N. Y. Cheung, and Wenxin Zheng. "Endometrial premalignant lesions." In Diagnostic Endometrial Pathology. CRC Press, 2019. http://dx.doi.org/10.1201/9781315228686-8.

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Schwartz, Robert A. "Other Premalignant Cutaneous Dysplasias." In Skin Cancer. Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3790-7_3.

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Roncucci, Luca. "Colorectal Cancer Premalignant Lesions." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27841-9_1269-2.

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Тези доповідей конференцій з теми "Premalignant"

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Firdowse, Humaira, Sakthiprian S, Karthikeyan S, et al. "Automatic Classification and Detection of Premalignant Cervical Lesions on the Colposcopic (VIA) Images Using Artificial Intelligence." In Frontiers in Optics. Optica Publishing Group, 2024. https://doi.org/10.1364/fio.2024.jw5a.26.

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Liebow, Charles, and M. J. Maloney. "Histochemical identification of malignant and premalignant lesions." In Optics, Electro-Optics, and Laser Applications in Science and Engineering, edited by Thomas J. Dougherty. SPIE, 1991. http://dx.doi.org/10.1117/12.44045.

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Nachmanson, Daniela, Mark F. Evans, Joseph Steward, et al. "Abstract 2176: Mutational profiling of premalignant breast microbiopsies." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-2176.

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Gao, DZ, GZ Yu, BZ Ma, Q. Zhang, L. Li, and YY Li. "Abstract P1-08-05: The Establishment of Rat Model of Premalignant Breast Disease and Histomorphological Study of Rats’ Premalignant Disease." In Abstracts: Thirty-Third Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 8‐12, 2010; San Antonio, TX. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/0008-5472.sabcs10-p1-08-05.

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Dhumal, Snehal, Arti Hole, Sheetal Korde, Aishwarya Naidu, Sakshi Bubna, and C. Murali Krishna. "Serum Raman spectroscopy: exploring delineation of oral premalignant disorders." In Optical Diagnostics and Sensing XX: Toward Point-of-Care Diagnostics, edited by Gerard L. Coté. SPIE, 2020. http://dx.doi.org/10.1117/12.2544815.

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Merenstein, C., S. Mazzilli, J. D. Campbell, et al. "Immune Alterations Associated with DiseaseProgression in Bronchial Premalignant Lesions." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4033.

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Rabinovitch, P. S., G. C. Burmer, R. C. Haggitt, D. S. Levine, C. E. Rubin, and B. J. Reid. "Flow Cytometric Changes That Precede Cancer: Premalignant Gastrointestinal Disease." In OE/LASE '89, edited by Gary C. Salzman. SPIE, 1989. http://dx.doi.org/10.1117/12.951906.

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Jayaprakash, Vijay, Gregory Loewen, Kirsten Moysich, et al. "Abstract B55: Factors predicting the progression of premalignant bronchial lesions." In Abstracts: Frontiers in Cancer Prevention Research 2008. American Association for Cancer Research, 2008. http://dx.doi.org/10.1158/1940-6207.prev-08-b55.

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Jayaprakash, Vijayvel, Gregory Loewen, Samjot Dhillon, et al. "Abstract A22: Spirometric surveillance for premalignant and malignant bronchial lesions." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Dec 6–9, 2009; Houston, TX. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1940-6207.prev-09-a22.

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Furukawa, Kinya, David H. Crean, Thomas S. Mang, Harubumi Kato, and Thomas J. Dougherty. "Fluorescence detection of premalignant, malignant, and micrometastatic disease using hexylpyropheophorbide." In Fifth International Photodynamic Association Biennial Meeting, edited by Denis A. Cortese. SPIE, 1994. http://dx.doi.org/10.1117/12.203403.

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Звіти організацій з теми "Premalignant"

1

Medina, Daniel, and Powel Brown. Prevention of Premalignant Progression of Human Breast Cancer. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada552885.

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2

Aldaz, C. M. Genomic Instability at Premalignant and Early Stages of Breast Cancer Development. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada382773.

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3

Krumm, Anton. Premalignant Genetic and Epigenetic Alterations in Tubal Epithelium from Women with BRCA1 Mutations. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada517362.

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4

Rosenberg, Carol L. Genes Differentially Expressed at the Transition from Premalignancy to Carcinoma. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada418732.

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5

Ospina Ospina, Viviana, Ana María Pérez-Gutiérrez, Juan Pablo Zapata-Ospina, Esteban Castrillón Martínez, Estefanía Muriel Lopera, and Miguel Mateo Cuervo. Abordaje del melanoma cutáneo y sus lesiones premalignas para el médico de atención primaria. Facultad de Medicina Universidad de Antioquia, 2024. http://dx.doi.org/10.59473/medudea.pc.2024.75.

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