Готові списки джерел за темами / Telomeres And Telomerase Biology

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Добірка наукової літератури з теми "Telomeres And Telomerase Biology"

Автор: Grafiati

Опубліковано: 3 червня 2025

Оновлено: 31 липня 2025

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Статті в журналах з теми "Telomeres And Telomerase Biology"

Brault, Marie Eve, and Chantal Autexier. "Telomeric recombination induced by dysfunctional telomeres." Molecular Biology of the Cell 22, no. 2 (2011): 179–88. http://dx.doi.org/10.1091/mbc.e10-02-0173.

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Telomere maintenance is essential for cellular immortality, and most cancer cells maintain their telomeres through the enzyme telomerase. Telomeres and telomerase represent promising anticancer targets. However, 15% of cancer cells maintain their telomeres through alternative recombination-based mechanisms, and previous analyses showed that recombination-based telomere maintenance can be activated after telomerase inhibition. We determined whether telomeric recombination can also be promoted by telomere dysfunction. We report for the first time that telomeric recombination can be induced in hu

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Lin, Chi-Ying, Hsih-Hsuan Chang, Kou-Juey Wu та ін. "Extrachromosomal Telomeric Circles Contribute to Rad52-, Rad50-, and Polymerase δ-Mediated Telomere-Telomere Recombination in Saccharomyces cerevisiae". Eukaryotic Cell 4, № 2 (2005): 327–36. http://dx.doi.org/10.1128/ec.4.2.327-336.2005.

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ABSTRACT Telomere maintenance is required for chromosome stability, and telomeres are typically replicated by the telomerase reverse transcriptase. In both tumor and yeast cells that lack telomerase, telomeres are maintained by an alternative recombination mechanism. By using an in vivo inducible Cre-loxP system to generate and trace the fate of marked telomeric DNA-containing rings, the efficiency of telomere-telomere recombination can be determined quantitatively. We show that the telomeric loci are the primary sites at which a marked telomeric ring-containing DNA is observed among wild-type

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Feigon, Juli. "Structural biology of telomerase mechanism and interactions at telomeres." Structural Dynamics 12, no. 2_Supplement (2025): A201. https://doi.org/10.1063/4.0000509.

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Telomerase is a unique RNA-containing reverse transcriptase that synthesizes the DNA at the 3’-ends of telomeres, the structures at the ends of linear chromosomes. It is a highly regulated determinant of tumorigenesis, cellular aging, and stem cell renewal. All telomerases contain a catalytic core comprising telomerase reverse transcriptase (TERT) and telomerase RNA (TER), along with other proteins involved in biogenesis, assembly, and activation. TER includes a template complementary to ∼1.5 telomere repeats used by TERT to repetitively synthesize the telomere repeat (dTTGGGG in Tetrahymena,

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Kishtagari, Ashwin, and Justin Watts. "Biological and clinical implications of telomere dysfunction in myeloid malignancies." Therapeutic Advances in Hematology 8, no. 11 (2017): 317–26. http://dx.doi.org/10.1177/2040620717731549.

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Telomeres at the ends of linear chromosomes protect the genome. Telomeres shorten with each round of cell division, placing a finite limit on cell growth. Telomere attrition is associated with cell senescence and apoptosis. Telomerase, a specialized ribonucleoprotein complex, maintains telomeres homeostasis through repeat addition of telomere sequences to the 3′ telomeric overhang. Telomere biology is closely related to cancer and normal aging. Upregulation of telomerase or activation of the alternative pathway of telomere lengthening is a hallmark of cancer cells, making telomerase an attract

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Dreesen, Oliver, and George A. M. Cross. "Telomerase-Independent Stabilization of Short Telomeres in Trypanosoma brucei." Molecular and Cellular Biology 26, no. 13 (2006): 4911–19. http://dx.doi.org/10.1128/mcb.00212-06.

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ABSTRACT In cancer cells and germ cells, shortening of chromosome ends is prevented by telomerase. Telomerase-deficient cells have a replicative life span, after which they enter senescence. Senescent cells can give rise to survivors that maintain chromosome ends through recombination-based amplification of telomeric or subtelomeric repeats. We found that in Trypanosoma brucei, critically short telomeres are stable in the absence of telomerase. Telomere stabilization ensured genomic integrity and could have implications for telomere maintenance in human telomerase-deficient cells. Cloning and

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Bechard, Laura H., Bilge D. Butuner, George J. Peterson, Will McRae, Zeki Topcu, and Michael J. McEachern. "Mutant Telomeric Repeats in Yeast Can Disrupt the Negative Regulation of Recombination-Mediated Telomere Maintenance and Create an Alternative Lengthening of Telomeres-Like Phenotype." Molecular and Cellular Biology 29, no. 3 (2008): 626–39. http://dx.doi.org/10.1128/mcb.00423-08.

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ABSTRACT Some human cancers maintain telomeres using alternative lengthening of telomeres (ALT), a process thought to be due to recombination. In Kluyveromyces lactis mutants lacking telomerase, recombinational telomere elongation (RTE) is induced at short telomeres but is suppressed once telomeres are moderately elongated by RTE. Recent work has shown that certain telomere capping defects can trigger a different type of RTE that results in much more extensive telomere elongation that is reminiscent of human ALT cells. In this study, we generated telomeres composed of either of two types of mu

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Prescott, John C., and Elizabeth H. Blackburn. "Telomerase RNA Template Mutations Reveal Sequence-Specific Requirements for the Activation and Repression of Telomerase Action at Telomeres." Molecular and Cellular Biology 20, no. 8 (2000): 2941–48. http://dx.doi.org/10.1128/mcb.20.8.2941-2948.2000.

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ABSTRACT Telomeric DNA is maintained within a length range characteristic of an organism or cell type. Significant deviations outside this range are associated with altered telomere function. The yeast telomere-binding protein Rap1p negatively regulates telomere length. Telomere elongation is responsive to both the number of Rap1p molecules bound to a telomere and the Rap1p-centered DNA-protein complex at the extreme telomeric end. Previously, we showed that a specific trinucleotide substitution in the Saccharomyces cerevisiae telomerase gene (TLC1) RNA template abolished the enzymatic activit

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Mondello, Chiara, and A. Ivana Scovassi. "Telomeres, telomerase, and apoptosis." Biochemistry and Cell Biology 82, no. 4 (2004): 498–507. http://dx.doi.org/10.1139/o04-048.

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Telomeres are specialized high-order chromatin structures that cap the ends of eukaryotic chromosomes. In vertebrates, telomeric DNA is composed of repetitions of the TTAGGG hexanucleotide, is bound to a set of specific proteins, and is elongated by the reverse transcriptase enzyme telomerase. Telomerase activity is promptly detected in cells with an indefinite replicative potential, such as cancer cells, while is almost undetectable in normal cells, which are characterized by a limited life span. Mounting evidence indicates that the maintenance of telomere integrity and telomerase protect cel

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Hahn, William C. "Role of Telomeres and Telomerase in the Pathogenesis of Human Cancer." Journal of Clinical Oncology 21, no. 10 (2003): 2034–43. http://dx.doi.org/10.1200/jco.2003.06.018.

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Specialized nucleoprotein structures, termed telomeres, cap the ends of human chromosomes. These terminal structures, composed of repetitive arrays of guanine-rich hexameric DNA together with specific telomere-binding proteins, play essential roles in protecting the chromosome from damage and degradation. In addition, several lines of evidence implicate telomere maintenance as an important regulator of cell life span. Activation of telomerase, a dedicated reverse transcriptase that synthesizes telomeric sequences, is strongly associated with cancer, and recent observations confirm that telomer

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Jády, Beáta E., Patricia Richard, Edouard Bertrand, and Tamás Kiss. "Cell Cycle-dependent Recruitment of Telomerase RNA and Cajal Bodies to Human Telomeres." Molecular Biology of the Cell 17, no. 2 (2006): 944–54. http://dx.doi.org/10.1091/mbc.e05-09-0904.

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Telomerase is a ribonucleoprotein enzyme that counteracts replicative telomere erosion by adding telomeric sequence repeats onto chromosome ends. Despite its well-established role in telomere synthesis, telomerase has not yet been detected at telomeres. The RNA component of human telomerase (hTR) resides in the nucleoplasmic Cajal bodies (CBs) of interphase cancer cells. Here, in situ hybridization demonstrates that in human HeLa and Hep2 S phase cells, besides accumulating in CBs, hTR specifically concentrates at a few telomeres that also accumulate the TRF1 and TRF2 telomere marker proteins.

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Дисертації з теми "Telomeres And Telomerase Biology"

Sherwood, Rebecca. "The Effect of the Copy Number of the Telomerase RNA Gene on the Elongation of Telomeres in Saccharomyces cerevisiae." Thesis, Boston College, 2008. http://hdl.handle.net/2345/532.

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Thesis advisor: Clare O'Connor<br>Telomeres are repeated sequences at the ends of chromosomes, which promote chromosome stability by preventing the loss of necessary nucleotides from the DNA with successive rounds of replication. Telomeres are elongated by the enzyme telomerase, which has both a protein component and an RNA component. In the yeast Saccharomyces cerevisiae, the TLC1 gene encodes the RNA component of the enzyme. Telomerase RNA interacts with several proteins to perform its function, including the Ku protein, which binds to the end of the DNA and helps to recruit telomerase to th

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Alotaibi, Mohammad Kdaimes H. "Genes required to maintain telomeres in the absence of telomerase in Saccharomyces cerevisiae." Thesis, University of Nottingham, 2012. http://eprints.nottingham.ac.uk/12589/.

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In the absence of telomerase, Saccharomyces cerevisiae telomeres erode leading to senescence. Rare cells can survive after this stage as they can elongate their telomeres utilizing homologous recombination. Two different types of survivors can be easily distinguished by Southern blot. Type I survivor cells, elongate the telomere by amplifying Y elements and require RAD51, RAD54, RAD55 and RAD57 for establishment. Type II survivors elongate their telomere by amplifying TG1-3 repeats, however, they require the following genes to be established: RAD50, MRE11 and XRS2, RAD59, SGS1 and KU80 in some

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Brault, Marie Eve. "Telomeres and telomerase: role in human cancer, the premature aging syndrome dyskeratosis congenita and frailty." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=117043.

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Telomeres and telomerase stand at a junction of cellular processes that govern aging, cancer and disease. Premature aging syndromes and age-related diseases are characterized by short telomeres which compromise cell function and viability, whereas cancer cells are able to reactivate telomerase or alternative lengthening of telomeres (ALT) mechanisms to maintain their telomeres and become immortal.Telomeres and telomerase represent very attractive targets for the development of anticancer therapies. However, there is concern that these therapies may lead to cell resistance, including the reacti

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Fakhoury, Johans. "Conserved and divergent mouse and human telomerase and telomere regulation: implications for the development and validation of telomerase and telomere-specific anticancer strategies." Thesis, McGill University, 2010. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=94905.

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Telomerase synthesizes telomeric sequences and is minimally composed of a reverse transcriptase (RT) (TERT) and RNA (TR). We reconstituted heterologous mouse and human TERT-TR and chimeric mTERT-hTERT-hTR complexes in vitro and in immortalized human alternative lengthening of telomere (ALT) cells. Our data suggest that species-specific determinants of activity, processivity, and telomere function map not only to TR, but also to the TERT component. hTERT-hTR, but not heterologous TERT-TR complexes, nor chimeric mTERT-hTERT-hTR complexes, significantly reduced the percentage of chromosomes witho

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McKevitt, Tom Patrick. "A study of telomere and telomerase biology in the dog and cat." Thesis, University of Glasgow, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.443374.

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D'Souza, Yasmin. "Processivity domains within human telomerase reverse transcriptase that regulate telomere length and immortalization." Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=116879.

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Short, repetitive G-rich DNA sequences present at telomeres are synthesized by telomerase, a ribonucleoprotein consisting of a catalytic subunit, the telomerase reverse transcriptase, TERT, and an integrally associated RNA, TR. Human TERT (hTERT) can repetitively reverse transcribe its short RNA template, acting processively to add multiple telomeric repeats onto the same DNA substrate. We investigated if threshold levels of telomerase activity and processivity are required to maintain telomere length and/or function and immortalize human cells with limited lifespan. Specifically, we assessed

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Mangosh, Tawna L. "SLX4 Interacting Protein (SLX4IP): A Vital Primer for Alternative Lengthening of Telomere (ALT)-like Processes Promoting Replicative Immortality in Castration-resistant Prostate Cancer with Androgen Receptor Loss." Case Western Reserve University School of Graduate Studies / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1623255136624147.

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Denham, Elizabeth. "The Effects of Relocating the Ku-binding Stem-loop of Telomerase RNA on Telomere Healing Events." Thesis, Boston College, 2008. http://hdl.handle.net/2345/528.

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Thesis advisor: Anne E. Stellwagen<br>Thesis advisor: Clare O'Connor<br>In most eukaryotes, the enzyme telomerase adds telomeric DNA repeats to the 3' ends of chromosomes in order to stabilize them and protect them from degradation. In the budding yeast Saccharomyces cerevisiae, telomerase is a ribonucleoprotein complex consisting of multiple protein subunits and an approximately 1.3 kb RNA component termed TLC1. Among the various proteins involved in telomerase, Ku is a heterodimer that binds both to double-stranded DNA and to a 48 nucleotide stem loop on the TLC1 RNA. Beyond its function of

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VENTURINI, LORENZA. "TELOMERE MAINTENANCE MECHANISMS IN TUMOR OF MESENCHYMAL ORIGIN: EVALUATION OF PROGNOSTIC SIGNIFICANCE AND CHARACTERIZATION OF RELEVANT MOLECULAR PATHWAYS." Doctoral thesis, Università degli Studi di Milano, 2012. http://hdl.handle.net/2434/171334.

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A limitless proliferative potential is one of the hallmarks of tumour cells and can be achieved through the activation of telomere maintenance mechanisms (TMM), which rely on telomerase reactivation (TA) or, alternatively, on recombination-based processes known as alternative lengthening of telomeres (ALT). Since a substantial fraction of tumours of mesenchymal origin utilizes ALT mechanisms, they represent an interesting model to study the molecular pathways involved in the activation of TMM. With the present work, we extended our knowledge about the prevalence and the prognostic significan

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Aisenberg, Jeremy Charles. "A Critical Review of Telomerase Biology and Model Systems for the Study of Telomerase." VCU Scholars Compass, 2006. http://hdl.handle.net/10156/2120.

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Книги з теми "Telomeres And Telomerase Biology"

Lue, Neal. Telomerases: Chemistry, biology, and clinical applications. Wiley, 2012.

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Double, John A., and Michael J. Thompson. Telomeres and Telomerase. Humana Press, 2002. http://dx.doi.org/10.1385/1592591892.

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Songyang, Zhou, ed. Telomeres and Telomerase. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6892-3.

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Songyang, Zhou, ed. Telomeres and Telomerase. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-092-8.

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Foundation, Ciba, ed. Telomeres and telomerase. John Wiley & Sons, 1997.

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Hiyama, Keiko, ed. Telomeres and Telomerase in Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-879-9.

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A, Double John, and Thompson Michael J, eds. Telomeres and telomerase: Methods and protocols. Humana Press, 2002.

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Chadwick, Derek J., and Gail Cardew, eds. Ciba Foundation Symposium 211 - Telomeres and Telomerase. John Wiley & Sons, Ltd., 1997. http://dx.doi.org/10.1002/9780470515433.

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Leonardo, Mancini, ed. Telomeres: Function, shortening, and lengthening. Nova Science Publishers, 2009.

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Rudolph, K. Lenhard, ed. Telomeres and Telomerase in Ageing, Disease, and Cancer. Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-73709-4.

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Частини книг з теми "Telomeres And Telomerase Biology"

Joseph, Nithila A., Chi-Fan Chen, Jiun-Hong Chen, and Liuh-Yow Chen. "Monitoring Telomere Maintenance During Regeneration of Annelids." In Methods in Molecular Biology. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2172-1_24.

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AbstractTelomere shortening is a hallmark of aging and eventually constrains the proliferative capacity of cells. The protocols discussed here are used for monitoring telomeres comprehensively in Aeolosoma viride, a model system for regeneration studies. We present methods for analyzing the activity of telomerase enzyme in regenerating tissue by telomeric repeat amplification protocol (TRAP) assay, for comparing telomere length between existing tissue and newly regenerated tissue by telomere restriction fragment (TRF) assay, as well as for visualizing telomeres by fluorescence in situ hybridiz

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Gomes, N. M. V., J. W. Shay, and W. E. Wright. "Telomeres and Telomerase." In The Comparative Biology of Aging. Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-3465-6_11.

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Nelson, Andrew D. L., Mark A. Beilstein, and Dorothy E. Shippen. "Plant Telomeres and Telomerase." In Molecular Biology. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-7570-5_4.

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Roka, Kleoniki, and Eftichia Stiakaki. "Telomere Length Biology in Blood Disorders and Childhood Cancer." In Telomeres. Jenny Stanford Publishing, 2024. http://dx.doi.org/10.1201/9781003568094-24.

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Bryan, Tracy M., and Scott B. Cohen. "Telomerase." In Handbook of Chemical Biology of Nucleic Acids. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-9776-1_47.

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Bryan, Tracy M., and Scott B. Cohen. "Telomerase." In Handbook of Chemical Biology of Nucleic Acids. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-16-1313-5_47-1.

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Marión, Rosa M., and Maria A. Blasco. "Telomeres And Telomerase in Adult Stem Cells and Pluripotent Embryonic Stem Cells." In Advances in Experimental Medicine and Biology. Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-7037-4_9.

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Siomos, Maria F., and Karel Riha. "Telomeres and Their Biology." In Plant Genome Diversity Volume 1. Springer Vienna, 2012. http://dx.doi.org/10.1007/978-3-7091-1130-7_5.

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Nelson, Andrew D. L., Mark A. Beilstein, and Dorothy E. Shippen. "Nucleus and Genome: Telomeres." In Molecular Biology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4939-0263-7_4-1.

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Baird, Duncan M., and Eric A. Hendrickson. "Telomeres and Chromosomal Translocations." In Advances in Experimental Medicine and Biology. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-0593-1_7.

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Тези доповідей конференцій з теми "Telomeres And Telomerase Biology"

Sannikova, A. V., M. R. Sharipova, E. V. Shakirov, and L. R. Valeeva. "THE ROLE OF TRFL PROTEINS IN THE REGULATION OF TELOMERE LENGTH MARCHANTIA POLYMORPHA." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-368.

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Telomeres are nucleoprotein structures, involved in protection of the physical ends of eukaryotic chromosomes. A decisive role in maintaining telomere stability is played by specific proteins telomere complex are TRF proteins. Here, we have shown the intraspecific variability of telomere length and the involvement of TRFL protein in telolere length maintanance in a liverwort M. polymorpha as a new model plant for telomere biology studies.

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Ferreira, Carlos Eduardo Gomes, Matheus Antonio Pereira Costa, Rafael Leite Carvalho, and Adriana Sarmento De Oliveira. "A BIOLOGIA DO ENVELHECIMENTO: TELÔMEROS, TELOMERASE E ATIVIDADE FÍSICA (UMA REVISÃO SISTEMÁTICA)." In I Congresso Nacional On-line de Biologia Celular e Estrutural. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1954.

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Introdução: Ao longo dos anos, mudanças na qualidade de vida vêm impactando diretamente a expectativa de vida humana. Estudos apontam certo envelhecimento mundial. Isso indica a importância de pesquisar a relação do impacto das atividades físicas no organismo e no envelhecimento, principalmente a nível celular, como em estruturas celulares consideradas possíveis marcadores: os telômeros. Compostos por uma curta e repetitiva sequência de DNA rica em guanina (5’-TTAGGG-3’)n, têm a função de proteger a integridade do DNA e a informação genética. Contudo, os telômeros são encurtados a cada ciclo c

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Johnson, Ruth A., Brooke R. Druliner, Jill Washechek Alleto, et al. "Abstract B1-46: How collection, processing and storage of PBL samples impact the measurement of telomerase activity." In Abstracts: AACR Special Conference: Computational and Systems Biology of Cancer; February 8-11, 2015; San Francisco, CA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.compsysbio-b1-46.

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Joyce, Brian T., Yang Li, Yinan Zheng, et al. "Abstract 2264: Telomere biology gene methylation and cancer risk." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-2264.

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"Natural variations in telomere lengths in different Bryophytes." In Bioinformatics of Genome Regulation and Structure/Systems Biology (BGRS/SB-2022) :. Institute of Cytology and Genetics, the Siberian Branch of the Russian Academy of Sciences, 2022. http://dx.doi.org/10.18699/sbb-2022-362.

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Shakirov, Yevgeniy. "Ribosome biogenesis pathway underlies establishment of telomere length set point in Arabidopsis." In ASPB PLANT BIOLOGY 2020. ASPB, 2020. http://dx.doi.org/10.46678/pb.20.1375852.

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Brosnan-Cashman, Jacqueline A., Ming Yuan, Anthony J. Rizzo, et al. "Abstract 3465: Context-dependent effects of ATRX loss on telomere biology in glioma cells." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3465.

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Fanous, J., D. Mcnierney, J. Chen, et al. "Multi-Organ Complexities in Telomere Biology Disorders: Liver Cirrhosis, Pulmonary Fibrosis, and Transplantation Challenges." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a5945.

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Shay, Jerry. "Abstract ED04-01: Aging and cancer: are telomeres and telomerase the connection?" In Abstracts: Frontiers in Cancer Prevention Research 2008. American Association for Cancer Research, 2008. http://dx.doi.org/10.1158/1940-6207.prev-08-ed04-01.

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Bechter, Oliver E., and Margit Dlaska. "Abstract 2993: Homologous recombination between telomeres is present in ALT and telomerase-positive immortal cells." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2993.

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