Relevant bibliographies by topics / Acute bilateral basal ganglia syndrome

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Academic literature on the topic 'Acute bilateral basal ganglia syndrome'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Acute bilateral basal ganglia syndrome"

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Park, Jong-Ho, Han-Joon Kim, and Seong-Min Kim. "Acute Chorea with Bilateral Basal Ganglia Lesions in Diabetic Uremia." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 34, no. 2 (2007): 248–50. http://dx.doi.org/10.1017/s0317167100006144.

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Uremia is a syndrome of clinical and metabolic abnormalities, which develops in parallel with the deterioration of renal function. Uremic encephalopathy is one of many manifestations of acute or chronic renal failure. It is usually applied to patients with cortical involvement, such as confusion, seizure, tremor, myoclonus, or asterixis. Some cases of acute extrapyramidal movement disorders associated with bilateral basal ganglia lesions, especially parkinsonism have been reported in uremic patients. Here, we report a diabetic uremic patient who developed acute chorea associated with bilateral
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Cao, Xin, Qiang Fang, and Hao Shi. "MRI Findings of Syndrome of Acute Bilateral Symmetrical Basal Ganglia Lesions in Diabetic Uremia: A Case Report and Literature Review." Case Reports in Radiology 2016 (2016): 1–6. http://dx.doi.org/10.1155/2016/2407219.

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The syndrome of acute bilateral basal ganglia lesions is an uncommon clinical occurrence exhibiting acute onset of movement abnormalities, which can be seen almost exclusively among patients with chronic renal failure, especially in the setting of concurrent diabetes mellitus. Symmetrical lesions located in basal ganglia demonstrated in MRI are typical manifestation of this syndrome. Our study includes routine MRI examination, MRS, 3D-ASL, and SWI findings, which have been rarely reported and will contribute to diagnosing more cases about this syndrome.
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De Witte, Lieve, Sebastiaan Engelborghs, Jo Verhoeven, Peter P. De Deyn, and Peter Mariën. "Disrupted Auto-Activation, Dysexecutive and Confabulating Syndrome Following Bilateral Thalamic and Right Putaminal Stroke." Behavioural Neurology 19, no. 3 (2008): 145–51. http://dx.doi.org/10.1155/2008/693671.

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Objective: Clinical, neuropsychological, structural and functional neuroimaging results are reported in a patient who developed a unique combination of symptoms after a bi-thalamic and right putaminal stroke. The symptoms consisted of dysexecutive disturbances associated with confabulating behavior and auto-activation deficits. Background: Basal ganglia and thalamic lesions may result in a variety of motor, sensory, neuropsychological and behavioral syndromes. However, the combination of a dysexecutive syndrome complicated at the behavioral level with an auto-activation and confabulatory syndr
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Nolasco, Remirr, Neshreen Kingking, Cristina Dorado, Jay Fonte, and Meliza Dadua-Ecal. "CHOREA HYPERGLYCEMIA BASAL GANGLIA SYNDROME." Journal of the ASEAN Federation of Endocrine Societies 37, no. 2 (2022): 40. https://doi.org/10.15605/jafes.037.afes.57.

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BACKGROUNDChorea hyperglycemia basal ganglia syndrome (C-H- BG) is a rare sequelae of acute hyperglycemia with a predilection for Asians and elderly women. Cases are often misdiagnosed with the more common intracerebral hemorrhage. The purpose of this report is to present this rare condition in a relatively young Asian male and discuss the approach to such cases. CASEA single case of C-H-BG was closely followed and recorded. A 53-year-old-male, Filipino, with type 2 diabetes mellitus and hypertension for more than 10 years, presented with sudden onset of hyperkinetic, involuntary, non-patterne
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Sarkar, Devendra Nath, Devavrata Sarkar, AKM Zahin, MA Ohab, Md Shahin Miah, and Md Mahmudul Haque. "Fahr's Syndrom e: A rare case- Presented as Acute Ischaemic Stroke." Medicine Today 29, no. 1 (2017): 45–46. http://dx.doi.org/10.3329/medtoday.v29i1.33862.

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Fahr's syndrome refers to a rare syndrome characterized by symmetrical and bilateral intracranial calcification. We present a 65-year-old female with Fahr disease, presenting with headache with acute ischaemic stroke with left sided hemiplegia. CT scan of brain reveals irregular variable size hyperdense areas are noted in both basal ganglia regions and in both cerebellar hemisphere.Medicine Today 2017 Vol.29(1): 45-46
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Wang, Han-Cheng, and Shih-Jung Cheng. "The syndrome of acute bilateral basal ganglia lesions in diabetic uremic patients." Journal of Neurology 250, no. 8 (2003): 948–55. http://dx.doi.org/10.1007/s00415-003-1122-0.

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Kirton, C. Adam, and Richard J. Riopelle. "Meige Syndrome Secondary to Basal Ganglia Injury: A Potential Cause of Acute Respiratory Distress." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 28, no. 2 (2001): 167–73. http://dx.doi.org/10.1017/s0317167100052896.

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ABSTRACT:Background:Meige syndrome is a movement disorder that includes blepharospasm and oromandibular dystonias. Its etiology may be idiopathic (primary) or it may arise secondary to focal brain injury. Acute respiratory distress as a feature of such dystonias occurs infrequently. A review of the literature on Meige syndrome and the relationship between dystonias and respiratory compromise is presented.Methods:A 60-year-old woman suffered a cerebral anoxic event secondary to manual strangulation. She developed progressive blepharospasm combined with oromandibular and cervical dystonias. Neur
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Popescu, Cyprian. "Hypoxic-Ischemic Injury of Basal Ganglia Associated with the COVID-19 Infection: Case Report." Case Reports in Neurology 13, no. 3 (2021): 668–71. http://dx.doi.org/10.1159/000518506.

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There is emerging evidence of a broad spectrum of neurological manifestations in COVID-19 patients. We report a case of a patient diagnosed with COVID-19 who presented bilateral lesions of the basal ganglia related to a severe acute respiratory distress syndrome. This observation allows expanding the neurological spectrum of COVID-19 particularly in patients with a more severe clinical course.
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Delgado, Silvia R., Leticia Tornes, Janice Maldonado, Jeffrey Hernandez, Yesica Campos, and Kottil Rammohan. "Neuroleptic Malignant Syndrome Associated with Refractory Acute Disseminated Encephalomyelitis." Case Reports in Neurology 8, no. 1 (2016): 92–96. http://dx.doi.org/10.1159/000446106.

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We present the case of a young man who was transferred to our hospital with worsening acute disseminated encephalomyelitis (ADEM) despite treatment with intravenous methylprednisolone, intravenous immunoglobulin and plasma exchange. He developed neuroleptic malignant syndrome (NMS) without the use of dopamine-modulating drugs. His progressive clinical improvement started after treatment with intravenous cyclophosphamide and methylprednisolone. In our patient, acute demyelination with severe bilateral inflammation of the basal ganglia could have caused a state of central dopamine depletion, cre
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Madadi, Shekiba, Kawsar Alami, Yudai Kaneda, and Pazhman Sediqi. "From seizures to cognitive dysfunction: A case report of Fahr syndrome in an Afghan patient." Medicine 103, no. 25 (2024): e38542. http://dx.doi.org/10.1097/md.0000000000038542.

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Rationale: Fahr syndrome is a rare, degenerative neurological condition characterized by bilateral idiopathic calcification of the periventricular region, especially the basal ganglia. This condition is often misdiagnosed as other neurological or psychiatric disorders due to its rarity and overlapping symptoms. Patient concerns: A 34-year-old man had been experiencing seizures and cognitive dysfunction for few years, which were further compounded by slurred speech and motor difficulties as acute conditions. Diagnosis: After investigations, severe hypocalcemia, and hypoparathyroidism were detec
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Books on the topic "Acute bilateral basal ganglia syndrome"

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Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 44-Year-Old Man with Bilateral Facial Droop. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0017.

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Facial neuropathy is most commonly seen as an idiopathic unilateral palsy known as Bell’s palsy. Generally, acute onset of typical lower motor neuron facial weakness that is not associated with other atypical or suspicious features, remains unilateral, and recovers completely requires no further workup. A recurrent or bilateral peripheral facial palsy makes an idiopathic cause less likely and prompts a more in-depth workup. The appropriate work-up of unilateral or bilateral facial palsy guided by the presence or absence of associated clinical findings is discussed. The major differentials for
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Book chapters on the topic "Acute bilateral basal ganglia syndrome"

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Alvarez, Catalina Sanchez, and Kenneth J. Warrington. "Headache and Hemiparesis in Middle Age." In Mayo Clinic Cases in Neuroimmunology, edited by Andrew McKeon, B. Mark Keegan, and W. Oliver Tobin. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780197583425.003.0061.

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A 53-year-old man with hypertension, hyperlipidemia, and a remote, cryptogenic, multifocal, posterior circulation ischemic stroke, came to the emergency department with 1 day of vertigo, ataxic gait, nausea, occipital headache, and painless binocular diplopia. Symptoms were present upon awakening on the day of presentation and progressed throughout the day. The erythrocyte sedimentation rate was 10 mm/h and C-reactive protein level was less than 3 mg/L. Cerebrospinal fluid examination indicated mild lymphocytic pleocytosis with 9 cells/µL, protein value of 45 mg/dL, and glucose level within no
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Frank, Yitzchak. "Cognitive and Behavioral Abnormalities Associated with Liver Disease and Wilson Disease." In Cognitive and Behavioral Abnormalities of Pediatric Diseases. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780195342680.003.0021.

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Liver diseases may be associated with neurological abnormalities, more commonly with central nervous system (CNS) dysfunction (Lewis and Howdle 2003; Lockwood 1995; Raskin and Rowland 1995; Steinberg and Frank 1993; Victor and Rothstein 1992). Consequently, cognitive and behavioral abnormalities are commonly present in patients with acute and chronic liver disease. Cognitive impairment may include impairments of memory, attention, and executive and motor functions, can be initially mild and then progress into overt hepatic encephalopathy (HE) (Collie 2005; O’Carroll 2007). Specific patterns of
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Conference papers on the topic "Acute bilateral basal ganglia syndrome"

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Leite, Izabel Feitosa da Mata, Adelina Mouta Moreira Neto, Guilherme de Aguiar Moraes, Lucas Cardoso Siqueira Albernaz, and Matheus de Campos Medeiros. "Neuro-Behçet’s Syndrome: case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.209.

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Context: Behçet’s Disease (BD) is a multisystem chronic disorder characterized by perivasculitis within several tissues, including the Central Nervous System – Neuro-Behçet’s Disease, which accounts for 3 – 9 % of the BD patients. Neuro- Behçet’s Syndrome may present as brainstem or pyramidal syndromes, myelopathies, meningoencephalitis, intracranial hypertension and movement or psychiatric disorders. The objective of the present work is to report a case of Neuro-Behçet’s Disease, a rare and disabling disorder. Case report: 24-year-old male presenting focal neurological deficits – hemiparesis
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Monteiro, Ana Karoline da Costa, Paulo Filho Soares Marcelino, Marcello Holanda de Andrade, Rairis Barbosa Nascimento, Marx Lincoln Lima de Barros Araújo, and Samuel Pinheiro da Silva. "Fahr’s Syndrome: A Case Report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.110.

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Context: Fahr’s syndrome is a clinical entity of primary or secondary causes characterized by neurological and/or psychiatric symptoms associated with abnormal calcifications in basal ganglia, cerebellum and cerebral cortex. Case report: G.M.A, female, 49 years-old, presented athetosis in the distal extremity of the right upper limb (RUL) in December 2020, without seeking for medical help. Known to be diabetic, hypertensive and with diastolic heart failure (HF), in February 2021 she presented decompensated HF associated with worsening of involuntary movements in the RUL. During investigation,
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Souza, Isabela Silva, Beatriz Cassarotti, Lucas de Oliveira Pinto Bertoldi, et al. "Fahr syndrome associated with post-thyroidectomy hypoparathyroidism." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.567.

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Context: Fahr’s syndrome is a rare disorder characterized by bilateral and symmetrical abnormal calcifications in basal ganglia and cerebral cortex. Those calcified deposits are due to changes in calcium and phosphorus metabolisms that can be caused by endocrine disorders, mitochondrial myopathies, dermatological and infectious diseases. Clinical manifestations may include a variety of extrapyramidal, cerebelar and neuropsychiatric syndromes. Case report: This study describes a 75-year-old female patient that underwent total thyroidectomy in 1985 due to a multinodular goiter and presented post
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Mikael, Luana de Rezende, Coralia Gabrielle Vieira Silveira, Camilla Duarte Ribeiro, et al. "Fahr’s syndrome as a differential diagnosis amid parkinsonian syndromes: a case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.465.

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Context: Parkinsonian syndromes are routinely identified by neurologists. However, the differential diagnosis among probable etiologies can be challenging and complex. In Fahr’s syndrome, calcifications of the basal ganglia secondary to disorders of calcium metabolism are observed. A possible clinical presentation associated with this entity is the presence of a parkinsonian syndrome. Case report: A 54-years-old female patient presented with a progressive tremor in the right upper and lower limbs associated with bradykinesia. Seizures were observed during the course of the disease. After exten
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Oliveira, Daniel Alves de, Vitor Maia Arca, Arthur Cesário de Holanda, et al. "Bilateral pallido-nigral lesions in a patient with subacute chorea after diabetic ketoacidosis: case-report." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.778.

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Case presentation: A 54-year-old female patient, with prior history of arterial hypertension and type 2 diabetes, presented a one-year history of bilateral weakness in lower limbs, cognitive impairment and irregular hyperkinectic movements in upper limbs and face. The clinical onset was subacute after hospitalization due to severe pneumonia and diabetic ketoacidosis, which required intensive care unit care and ventilatory assistance for more than 15 days. On physical examination, she presented cognitive impairment with dysexecutive predominance, spastic paraparesis and facial and upper limbs c
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Elkin, Jeanne, Jaclyn Tencer, and Sabrina Yum. "An Unusual Cause of Acute Onset Movement Disorder In An Adolescent: A Rare Pediatric Presentation Of Chorea-Hyperglycemia-Basal Ganglia Syndrome." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.849.

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