Relevant bibliographies by topics / ADH1B Polymorphism

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  1. Journal articles
  2. Dissertations / Theses
  3. Conference papers

Academic literature on the topic 'ADH1B Polymorphism'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "ADH1B Polymorphism"

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Yin, Guang, Keizo Ohnaka, Makiko Morita, Shinji Tabata, Osamu Tajima, and Suminori Kono. "Genetic Polymorphisms of Alcohol Dehydrogenase and Aldehyde Dehydrogenase: Alcohol Use and Type 2 Diabetes in Japanese Men." Epidemiology Research International 2011 (2011): 1–8. http://dx.doi.org/10.1155/2011/583682.

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This study investigated the association of ADH1B (rs1229984) and ALDH2 (rs671) polymorphisms with glucose tolerance status, as determined by a 75-g oral glucose tolerance test, and effect modification of these polymorphisms on the association between alcohol consumption and glucose intolerance in male officials of the Self-Defense Forces. The study subjects included 1520 men with normal glucose tolerance, 553 with prediabetic condition (impaired fasting glucose and impaired glucose tolerance), and 235 men with type 2 diabetes. There was an evident interaction between alcohol consumption and AD
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Chien, Tsuo-Hsuan, Chih-Lang Lin, Li-Wei Chen, Cheng-Hung Chien, and Ching-Chih Hu. "Patients with Non-Alcoholic Fatty Liver Disease and Alcohol Dehydrogenase 1B/Aldehyde Dehydrogenase 2 Mutant Gene Have Higher Values of Serum Alanine Transaminase." Journal of Personalized Medicine 13, no. 5 (2023): 758. http://dx.doi.org/10.3390/jpm13050758.

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Patients with non-alcoholic fatty liver disease (NAFLD) share similar pathophysiologies to those of patients with alcohol liver disease. Alcoholic metabolic enzyme-related genes (alcohol dehydrogenase 1B (ADH1B) and aldehyde dehydrogenase 2 (ALDH2)) may be associated with pathophysiology in NAFLD patients. In this study, the association between ADH1B/ALDH2 gene polymorphism and serum metabolic factors, body statures, and hepatic steatosis/fibrosis status was evaluated in patients with NAFLD. Using biochemistry data, abdominal ultrasonography, fibrosis evaluation (Kpa), and steatosis evaluation
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Pecikoza, Amar, Lejla Lasic, Gabrijela Radosavljević, et al. "Frequency of ADH1B RsaI (rs2066701) single nucleotide polymorphism in a population of Bosnia and Herzegovina." Genetics & Applications 2, no. 2 (2018): 28. http://dx.doi.org/10.31383/ga.vol2iss2pp28-34.

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Apart from its physiological role in the cellular oxidation of ethanol interesting feature of the ADH1B gene locus is its characteristic geographical distribution in which certain variants of ADH1B peak in different parts of the world. Therefore, ADH1B rs2066701 polymorphism is exploited as a genetic marker in tracing of the evolutionary processes and human migrations in the past. Taking into consideration the complexity of population genetic structure and several migrations in the history of the Balkan populations, including Bosnian and Herzegovinian, this study aimed to estimate the frequenc
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Gu, Sheng, Hui Li, Andrew Pakstis, et al. "Recent Selection on a Class I ADH Locus Distinguishes Southwest Asian Populations Including Ashkenazi Jews." Genes 9, no. 9 (2018): 452. http://dx.doi.org/10.3390/genes9090452.

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The derived human alcohol dehydrogenase (ADH)1B*48His allele of the ADH1B Arg48His polymorphism (rs1229984) has been identified as one component of an East Asian specific core haplotype that underwent recent positive selection. Our study has been extended to Southwest Asia and additional markers in East Asia. Fst values (Sewall Wright’s fixation index) and long-range haplotype analyses identify a strong signature of selection not only in East Asian but also in Southwest Asian populations. However, except for the ADH2B*48His allele, different core haplotypes occur in Southwest Asia compared to
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Kukowka, Arnold, Bogusław Brzuchalski, Mateusz Kurzawski, Damian Malinowski, and Monika Anna Białecka. "ADH1B, ADH1B/C and CYP2E1 Gene Polymorphism and the Risk of Fetal Alcohol Spectrum Disorder." Genes 14, no. 7 (2023): 1392. http://dx.doi.org/10.3390/genes14071392.

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Increasing alcohol consumption by women of childbearing age contributes to more frequent cases of fetal alcohol spectrum disorder. The cause of the syndrome is fetal alcohol exposure, particularly what is referred to as high prenatal alcohol exposure. Low metabolic activity of fetal enzymes shifts the burden of ethanol removal to maternal metabolism. One of the factors influencing the pathogenesis of FASD is the genetic background. It can determine the rate of elimination of ethanol, thus increasing or decreasing the time of fetal exposure to ethanol and also decreasing its concentration. Gene
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Chen, Yi-Chyan, Li-Fang Yang, Ching-Long Lai, and Shih-Jiun Yin. "Acetaldehyde Enhances Alcohol Sensitivity and Protects against Alcoholism: Evidence from Alcohol Metabolism in Subjects with Variant ALDH2*2 Gene Allele." Biomolecules 11, no. 8 (2021): 1183. http://dx.doi.org/10.3390/biom11081183.

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Alcoholism is a complex behavior trait influenced by multiple genes as well as by sociocultural factors. Alcohol metabolism is one of the biological determinants that can significantly influence drinking behaviors. Alcohol sensitivity is thought to be a behavioral trait marker for susceptibility to develop alcoholism. The subjective perceptions would be an indicator for the alcohol preference. To investigate alcohol sensitivity for the variants ADH1B*2 and ALDH2*2, sixty healthy young males with different combinatory ADH1B and ALDH2 genotypes, ADH1B*2/*2–ALDH2*1/*1 (n = 23), ADH1B*2/*2–ALDH2*1
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Bhutia, Yazum, Sanjiba Dutta, and Mingma Sherpa. "Asian flushing, ADH1B, aldehyde dehydrogenase 2 genotypic status among the unique ethnic population of the Himalayan state of Sikkim, India." Asian Journal of Medical Sciences 15, no. 12 (2024): 64–69. https://doi.org/10.71152/ajms.v15i12.4258.

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Background: Alcohol induced flushing a phenotypic instrument variable also known as “Asian flush” was first reported by Wolff (1972), commonly seen among East Asians. This phenotype is indicative of inactive aldehyde dehydrogenase 2 (ALDH2) and high activity of alcohol dehydrogenase (ADH1B). Aims and Objectives: This study aimed to examine the sensitivity and specificity of the simple flushing questionnaire for identifying inactive ALDH2 and to examine the flushing, ADH1B and ALDH2 status across the three unique ethnic groups in Sikkim, India. Materials and Methods: Two hundred and fifty conse
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Bhutia, Yazum, Sanjiba Dutta, and Mingma Lhamu Sherpa. "Asian flushing, ADH1B, aldehyde dehydrogenase 2 genotypic status among the unique ethnic population of the Himalayan state of Sikkim, India." Asian Journal of Medical Sciences 15, no. 12 (2024): 64–69. https://doi.org/10.3126/ajms.v15i12.70143.

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Background: Alcohol induced flushing a phenotypic instrument variable also known as “Asian flush” was first reported by Wolff (1972), commonly seen among East Asians. This phenotype is indicative of inactive aldehyde dehydrogenase 2 (ALDH2) and high activity of alcohol dehydrogenase (ADH1B). Aims and Objectives: This study aimed to examine the sensitivity and specificity of the simple flushing questionnaire for identifying inactive ALDH2 and to examine the flushing, ADH1B and ALDH2 status across the three unique ethnic groups in Sikkim, India. Materials and Methods: Two hundred and fifty conse
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Kawashima, Nozomu, Atsushi Narita, Xinan Wang, et al. "ALDH2 Polymorphism In Japanese Children With Acquired Aplastic Anemia." Blood 122, no. 21 (2013): 3717. http://dx.doi.org/10.1182/blood.v122.21.3717.3717.

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Abstract Introduction Aplastic anemia is a syndrome of bone marrow failure (BMF) characterized by peripheral pancytopenia and marrow hypoplasia. Injury to hematopoietic cells, such as immune-mediated cytotoxicity, can cause aplastic anemia; the successful treatment of aplastic anemia using immunosuppressive therapy supports this hypothesis. Another proposed mechanism is an intrinsic defect of hematopoietic stem cells, which is the presumed major cause of congenital BMF, but this mechanism has not been definitively established in patients with acquired aplastic anemia. Aldehyde dehydrogenase 2
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Jiang, Shupeng, Yongqing Tong, Rui Zhao, Ge Xiong, Bin Qiao, and Yan Li. "An improved PCR-CTPP assay for the detection of ADH1B Arg48His polymorphism." Journal of Clinical Laboratory Analysis 32, no. 2 (2017): e22268. http://dx.doi.org/10.1002/jcla.22268.

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Dissertations / Theses on the topic "ADH1B Polymorphism"

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Al-Bargash, Dana. "Effect of Alcohol Consumption and Alcohol Dehydrogenase 1C (ADH1C) Polymorphisms on Total Plasma Homocysteine Levels." Thesis, 2008. http://hdl.handle.net/1974/1500.

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BACKGROUND: Evidence supports that an elevated level of total plasma homocysteine (tHcy) signifies a breakdown in the methionine-homocysteine cycle. This may result in folate deficiency, DNA methylation and oxidative stress, all of which are potential mechanisms that may lead to cancer. Few studies examined the effects of alcohol consumption on tHcy levels. No studies considered polymorphisms in alcohol dehydrogenase 1C (ADH1C), a gene that encodes for an enzyme that metabolizes alcohol to acetaldehyde, a folate antagonist; ADH1C*1 encodes for an enzyme with a higher capacity to generate ace
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Conference papers on the topic "ADH1B Polymorphism"

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Huang, Shiang-Fu, Huei-Tzu Chien, Sou-De Cheng, Chun-Ta Liao, and Hung-Ming Wang. "Abstract 3255: The roles of ALDH2, ADH1B and ADH1C gene polymorphism in predicting upper digestive tract multiple primary malignancies in head and neck cancer patients." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-3255.

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Ito, Hidemi, Isao Oze, Satoyo Hosono, Miki Watanabe, Hideo Tanaka, and Keitaro Matsuo. "Abstract 4618: The risk prediction for esophageal cancer by drinking, smoking, and the polymorphisms of ALDH2 and ADH1B." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-4618.

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Duell, Eric J., Núria Sala, Xavier Muñoz, et al. "Abstract 3748: Polymorphisms in the alcohol dehydrogenase (ADH1) gene cluster, alcohol consumption, and interactions in relation to gastric cancer risk in the EPIC cohort." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-3748.

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Journal articles
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