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Relevant bibliographies by topics / Adipokine balance

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Journal articles
Dissertations / Theses
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Academic literature on the topic 'Adipokine balance'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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Journal articles on the topic "Adipokine balance"

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Rijnsburger, Merel, Niek Djuric, Inge A. Mulder, and Helga E. de Vries. "Adipokines as Immune Cell Modulators in Multiple Sclerosis." International Journal of Molecular Sciences 22, no. 19 (2021): 10845. http://dx.doi.org/10.3390/ijms221910845.

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Multiple sclerosis (MS), a chronic inflammatory and demyelinating disease of the central nervous system (CNS), is a major clinical and societal problem, which has a tremendous impact on the life of patients and their proxies. Current immunomodulatory and anti-inflammatory therapies prove to be relatively effective; however, they fail to concomitantly stop ongoing neurological deterioration and do not reverse acquired disability. The proportion to which genetic and environmental factors contribute to the etiology of MS is still incompletely understood; however, a recent association between MS e

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Hoppmann, J., N. Perwitz, B. Meier, et al. "The balance between gluco- and mineralo-corticoid action critically determines inflammatory adipocyte responses." Journal of Endocrinology 204, no. 2 (2009): 153–64. http://dx.doi.org/10.1677/joe-09-0292.

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Obesity is associated with chronic inflammation. Pro-inflammatory adipokines may promote metabolic disorders and cardiovascular morbidity. However, the key mechanisms leading to obesity-related inflammation are poorly understood. The corticosteroid metabolism in adipose tissue plays a crucial role in the pathogenesis of the metabolic syndrome. Both the glucocorticoid receptor (GR) and the mineralocorticoid receptor (MR) mediate corticosteroid action in adipose tissue. The significance of the interplay of these receptors in mediating an inflammatory adipokine response is virtually unexplored. I

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Nagl, Veronika, Bertrand Grenier, Philippe Pinton, et al. "Exposure to Zearalenone Leads to Metabolic Disruption and Changes in Circulating Adipokines Concentrations in Pigs." Toxins 13, no. 11 (2021): 790. http://dx.doi.org/10.3390/toxins13110790.

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Zearalenone (ZEN) is a mycotoxin classified as an endocrine disruptor. Many endocrine disruptors are also metabolic disruptors able to modulate energy balance and inflammatory processes in a process often involving a family of protein hormones known as adipokines. The aim of our study was to elucidate the role of ZEN as metabolic disruptor in pigs by investigating the changes in energy balance and adipokines levels in response to different treatment diets. To this end, weaned piglets (n = 10/group) were exposed to either basal feed or feed contaminated with 680 and 1620 µg/kg ZEN for 28 days.

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Ge, Qian, Laurence Ryken, Laurence Noel, Eléonore Maury, and Sonia M. Brichard. "Adipokines identified as new downstream targets for adiponectin: lessons from adiponectin-overexpressing or -deficient mice." American Journal of Physiology-Endocrinology and Metabolism 301, no. 2 (2011): E326—E335. http://dx.doi.org/10.1152/ajpendo.00153.2011.

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Adipokines play a central role in the pathogenesis of the metabolic syndrome. Among them, adiponectin (ApN), a master regulator of immune and fuel homeostasis, is decreased. Identifying downstream adipokines targeted by ApN may help in deciphering this syndrome. We have generated transgenic mice, allowing persistent and moderate overexpression of ApN (ApN-Overex) specifically in white adipose tissue (AT). We took advantage of this model to unravel the adipokine secretion profile triggered by ApN. AT was fractionated into adipocytes and stromal-vascular cells (SVC), which were cultured for 8 h.

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Helfer, Gisela, and Qing-Feng Wu. "Chemerin: a multifaceted adipokine involved in metabolic disorders." Journal of Endocrinology 238, no. 2 (2018): R79—R94. http://dx.doi.org/10.1530/joe-18-0174.

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Metabolic syndrome is a global public health problem and predisposes individuals to obesity, diabetes and cardiovascular disease. Although the underlying mechanisms remain to be elucidated, accumulating evidence has uncovered a critical role of adipokines. Chemerin, encoded by the gene Rarres2, is a newly discovered adipokine involved in inflammation, adipogenesis, angiogenesis and energy metabolism. In humans, local and circulating levels of chemerin are positively correlated with BMI and obesity-related biomarkers. In this review, we discuss both peripheral and central roles of chemerin in r

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Ahmed abed, Baydaa, Layla Othman Farhan, Isam Noori Salman, and Safaa Ehssan. "A review on the role of novel adipokine Isthmin-1 and Subfatin in human type 2 diabetes mellitus." University of Thi-Qar Journal of Science 10, no. 2 (2023): 181–86. http://dx.doi.org/10.32792/utq/utjsci/v10i2.1129.

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Adipokines are essential for maintaining cardiovascular and metabolic balance. The term "adipokines" refers to peptides in the pancreas, brain, vascular system, liver, immune system and muscle. adipokines regulate numbers of factors, such as energy expenditure, hunger, satiety, insulin sensitivity, and blood pressure, insulin secretion, adipogenesis and fat distribution, and that might make them play a role in future pharmacological treatment strategies for obesity and metabolic diseases. Isthmin-1 (Ism-1) is a recently discovered insulin-like adipokine that suppresses hepatic lipids. Accordin

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Goralski, Kerry B., and Christopher J. Sinal. "Type 2 diabetes and cardiovascular disease: getting to the fat of the matterThis paper is one of a selection of papers published in this Special Issue, entitled Young Investigators' Forum." Canadian Journal of Physiology and Pharmacology 85, no. 1 (2007): 113–32. http://dx.doi.org/10.1139/y06-092.

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The increasing national prevalence of obesity is a major public health concern and a substantial burden on the health care resources of Canada. In addition to the direct health impact of obesity, this condition is a well-established risk factor for the development of various prevalent comorbidities including type 2 diabetes, hypertension, and cardiovascular disease. Historically, adipose tissue has been regarded primarily as an organ for energy storage. However, the discovery of leptin in the mid 1990’s revolutionized our understanding of this tissue and has focused attention on the endocrine

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Di Zazzo, Erika, Rita Polito, Silvia Bartollino, et al. "Adiponectin as Link Factor between Adipose Tissue and Cancer." International Journal of Molecular Sciences 20, no. 4 (2019): 839. http://dx.doi.org/10.3390/ijms20040839.

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Adipose tissue is a key regulator of energy balance playing an active role in lipid storage as well as in synthesizing several hormones directly involved in the pathogenesis of obesity. Obesity represents a peculiar risk factor for a growing list of cancers and is frequently associated to poor clinical outcome. The mechanism linking obesity and cancer is not completely understood, but, amongst the major players, there are both chronic low-grade inflammation and deregulation of adipokines secretion. In obesity, the adipose tissue is pervaded by an abnormal number of immune cells that create an

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Donoso, M. A., M. T. Muñoz-Calvo, V. Barrios, G. Garrido, F. Hawkins, and J. Argente. "Increased circulating adiponectin levels and decreased leptin/soluble leptin receptor ratio throughout puberty in female ballet dancers: association with body composition and the delay in puberty." European Journal of Endocrinology 162, no. 5 (2010): 905–11. http://dx.doi.org/10.1530/eje-09-0874.

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IntroductionBallet dancers (BDs) have a negative energy balance related to physical training that results in alterations in body composition, sexual development, and adipokine secretion. Our aims were to study anthropometric parameters, body composition, and their relationship with adipokines throughout pubertal development.Subjects and methodsWe carried out a prospective follow-up study of 22 female Caucasian BDs (Tanner II stage) followed throughout puberty. Nutritional status was determined by measurement of height, weight, and body mass index (BMI). We calculated growth velocity, bone matu

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Frayn, Keith N. "Obesity and metabolic disease: is adipose tissue the culprit?" Proceedings of the Nutrition Society 64, no. 1 (2005): 7–13. http://dx.doi.org/10.1079/pns2004403.

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Obesity is a risk factor for the development of type 2 diabetes and CVD. Is adipose tissue the culprit in the relationship between obesity and metabolic disease? It iscertainly possible to argue that adipose tissue function is disturbed in obesity in sucha way that adverse consequences may follow. For instance, lipolysis is down regulated, the sensitivity of lipolysis to insulin is reduced and there are disturbances in the regulation of adipose tissue blood flow. However, when examined critically these changes canbe seen as adaptations to the increased adipose tissue mass, making the situation

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Dissertations / Theses on the topic "Adipokine balance"

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Shalimova, A., A. Belovol, L. Bobronnikova та ін. "The influence of α-lipoic acid to endothelial dysfunction and adipokines balance in patients with type 2 diabetes and essential hypertension in the presence of unfavorable genetic polymorphism". Thesis, Bioscientifica, 2016. http://essuir.sumdu.edu.ua/handle/123456789/74776.

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Smith, Jessica. "Adipose tissue secreted factors in specific populations and metabolic states : Implications for energy balance and metabolic health." Thesis, Université Laval, 2010. http://www.theses.ulaval.ca/2010/27012/27012.pdf.

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Helfer, Gisela, and Q.-F. Wu. "Chemerin: A multifaceted adipokine involved in metabolic disorders." 2018. http://hdl.handle.net/10454/16384.

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yes<br>Metabolic syndrome is a global public health problem and predisposes individuals to obesity, diabetes and cardiovascular disease. Although the underlying mechanisms remain to be elucidated, accumulating evidence has uncovered a critical role of adipokines. Chemerin, encoded by the gene Rarres2, is a newly discovered adipokine involved in inflammation, adipogenesis, angiogenesis and energy metabolism. In humans, local and circulating levels of chemerin are positively correlated with body mass index and obesity-related biomarkers. In this review, we discuss both peripheral and central rol

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Book chapters on the topic "Adipokine balance"

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Thompson, Cheryl L., and MacKenzie Reece. "Adipokines and Prostate Cancer." In Energy Balance and Cancer. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-64940-5_4.

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Connor, Elizabeth V., Ofer Reizes, and Caner Saygin. "Obesity, Adipokines, and Gynecologic Cancer." In Energy Balance and Cancer. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-63483-8_6.

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Kwon, Hyokjoon, and Jeffrey E. Pessin. "Adipokines, Inflammation, and Insulin Resistance in Obesity." In Textbook of Energy Balance, Neuropeptide Hormones, and Neuroendocrine Function. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-89506-2_9.

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Esra Bihter Gürler, Hülya Çevik ARAS, and Nagihan Bostanci. "Functional Changes in Metabolic Syndrome." In Metabolic Syndrome: A Comprehensive Update with New Insights. BENTHAM SCIENCE PUBLISHERS, 2025. https://doi.org/10.2174/9789815322132125010005.

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Metabolic Syndrome (MetS) is a condition characterized by the cooccurrence of several cardiovascular risk factors, including insulin resistance, obesity, dyslipidemia, and hypertension. The development of MetS is closely linked to visceral adiposity, which refers to fat accumulation around critical vital organs in the abdominal cavity. Visceral fat is metabolically active and produces adipokines, proteins that regulate energy balance and play a role in inflammation and atherosclerosis. Some adipokines, such as leptin and adiponectin, have beneficial effects on glucose homeostasis and are consi

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Barbhuiya, Pervej Alom, Ireenia Warjri, Priyam Jyoti Das, et al. "Diabetes, Obesity, and the Risk of Breast Cancer: An Attempt to Decipher the Interconnections." In Diabetes and Breast Cancer: An Analysis of Signaling Pathways. BENTHAM SCIENCE PUBLISHERS, 2024. http://dx.doi.org/10.2174/9789815256024124010009.

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Diabetes and obesity are linked to a higher risk of breast and other cancers. In addition to the direct effects of hyperglycaemia and insulin resistance on breast cancer cells, the complications of diabetes, obesity, and metabolic syndrome are characterised by a dysfunctional endothelium and increased inflammation in tissue phenomena that are intimately related and occur concurrently in breast cancer progression. However, the complexity of the underlying mechanisms, together with the interplay of diet and physical activity contributing to energy balance and the role of adipose tissue, hypergly

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