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Relevant bibliographies by topics / Alpha toxin

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Academic literature on the topic 'Alpha toxin'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

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Journal articles on the topic "Alpha toxin"

1

Liang, Xudong, Meiying Yan, and Yinduo Ji. "The H35A Mutated Alpha-Toxin Interferes with Cytotoxicity of Staphylococcal Alpha-Toxin." Infection and Immunity 77, no. 3 (December 22, 2008): 977–83. http://dx.doi.org/10.1128/iai.00920-08.

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ABSTRACT Staphylococcal alpha-toxin is an important virulence factor for Staphylococcus aureus to cause severe infections. In this study, we explored whether the toxoid of alpha-toxin may be utilized to block the toxicity of wild-type alpha-toxin. We created a series of H35A mutated alpha-toxin expression strains and revealed that the H35A mutation eliminates the activity of alpha-toxin using a human lung epithelial cell line (A549). More importantly, we found that either the pretreatment or simultaneous treatment of the epithelial cells with alpha-toxin-H35A completely disrupted the cytotoxic

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2

Bernheimer, Alan W. "STAPHYLOCOCCAL ALPHA TOXIN*." Annals of the New York Academy of Sciences 128, no. 1 (December 16, 2006): 112–23. http://dx.doi.org/10.1111/j.1749-6632.1965.tb11633.x.

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3

Palmer. "Staphyloccal alpha toxin." Journal of Applied Microbiology 84, S1 (July 1998): 125S—126S. http://dx.doi.org/10.1046/j.1365-2672.1998.0840s1125s.x.

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4

Jepson, Marie, Angela Howells, Helen L. Bullifent, Barbara Bolgiano, Dennis Crane, Julie Miller, Jane Holley, Pramukh Jayasekera, and Richard W. Titball. "Differences in the Carboxy-Terminal (Putative Phospholipid Binding) Domains of Clostridium perfringens andClostridium bifermentans Phospholipases C Influence the Hemolytic and Lethal Properties of These Enzymes." Infection and Immunity 67, no. 7 (July 1, 1999): 3297–301. http://dx.doi.org/10.1128/iai.67.7.3297-3301.1999.

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ABSTRACT The phospholipases C of C. perfringens (alpha-toxin) and C. bifermentans (Cbp) show >50% amino acid homology but differ in their hemolytic and toxic properties. We report here the purification and characterisation of alpha-toxin and Cbp. The phospholipase C activity of alpha-toxin and Cbp was similar when tested with phosphatidylcholine in egg yolk or in liposomes. However, the hemolytic activity of alpha-toxin was more than 100-fold that of Cbp. To investigate whether differences in the carboxy-terminal domains of these proteins were responsible for differences in the hemolytic an

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5

Zhu, H., and H. Bussey. "Mutational analysis of the functional domains of yeast K1 killer toxin." Molecular and Cellular Biology 11, no. 1 (January 1991): 175–81. http://dx.doi.org/10.1128/mcb.11.1.175.

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To determine the functional domains of K1 killer toxin, we analyzed the phenotypes of a set of mutations throughout regions encoding the alpha- and beta-toxin subunits that allow secretion of mutant toxins. A range of techniques have been used to examine the ability of mutant toxins to bind to beta-glucan cell wall receptor and to form lethal ion channels. Our results indicate that both the alpha and beta subunits are involved in beta-glucan receptor binding. Defects in ion channel formation and toxin immunity are confined to the hydrophobic alpha subunit of the toxin.

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Zhu, H., and H. Bussey. "Mutational analysis of the functional domains of yeast K1 killer toxin." Molecular and Cellular Biology 11, no. 1 (January 1991): 175–81. http://dx.doi.org/10.1128/mcb.11.1.175-181.1991.

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To determine the functional domains of K1 killer toxin, we analyzed the phenotypes of a set of mutations throughout regions encoding the alpha- and beta-toxin subunits that allow secretion of mutant toxins. A range of techniques have been used to examine the ability of mutant toxins to bind to beta-glucan cell wall receptor and to form lethal ion channels. Our results indicate that both the alpha and beta subunits are involved in beta-glucan receptor binding. Defects in ion channel formation and toxin immunity are confined to the hydrophobic alpha subunit of the toxin.

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Yarovinsky, Timur O., Martha M. Monick, Matthias Husmann, and Gary W. Hunninghake. "Interferons Increase Cell Resistance to Staphylococcal Alpha-Toxin." Infection and Immunity 76, no. 2 (December 10, 2007): 571–77. http://dx.doi.org/10.1128/iai.01088-07.

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ABSTRACT Many bacterial pathogens, including Staphylococcus aureus, use a variety of pore-forming toxins as important virulence factors. Staphylococcal alpha-toxin, a prototype β-barrel pore-forming toxin, triggers the release of proinflammatory mediators and induces primarily necrotic death in susceptible cells. However, whether host factors released in response to staphylococcal infections may increase cell resistance to alpha-toxin is not known. Here we show that prior exposure to interferons (IFNs) prevents alpha-toxin-induced membrane permeabilization, the depletion of ATP, and cell death

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TSIOURIS (Β.Σ. ΤΣΙΟΥΡΗΣ), V. S., I. GEORGOPOULOU (ΓΕΩΡΓΟΠΟΥΛΟΥ), and E. PETRIDOU (Ε. ΠΕΤΡΙΔΟΥ). "Update on the toxins of Clostridium perfringens and their actions." Journal of the Hellenic Veterinary Medical Society 61, no. 3 (November 17, 2017): 241. http://dx.doi.org/10.12681/jhvms.14892.

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Clostridia appeared as a distinct class, approximately 2.7 billion years ago, before the initial formation of oxygen. Clostridium perfringens is widely distributed throughout the environment due to its ability to form spores. Furthermore, it is a member of intestinal microbiota in animals and human. In 2002, the complete genome of C perfringens strain 13 was published. Genomic analysis has revealed that C. perfringens lacks the genetic machinery to produce 13 essential amino acids and it obtains these in vivo via the action of its toxins. Toxins of C perfringens can be divided into major, mino

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9

Nilsson, Ing-Marie, Orla Hartford, Timothy Foster, and Andrzej Tarkowski. "Alpha-Toxin and Gamma-Toxin Jointly PromoteStaphylococcus aureus Virulence in Murine Septic Arthritis." Infection and Immunity 67, no. 3 (March 1, 1999): 1045–49. http://dx.doi.org/10.1128/iai.67.3.1045-1049.1999.

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ABSTRACT Septic arthritis is a common and feared complication of staphylococcal infections. Staphylococcus aureus produces a number of potential virulence factors including certain adhesins and enterotoxins. In this study we have assessed the roles of cytolytic toxins in the development of septic arthritis by inoculating mice withS. aureus wild-type strain 8325-4 or isogenic mutants differing in the expression of alpha-, beta-, and gamma-toxin production patterns. Mice inoculated with either an alpha- or beta-toxin mutant showed degrees of inflammation, joint damage, and weight decrease simila

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10

Awad, Milena M., Darren M. Ellemor, Richard L. Boyd, John J. Emmins, and Julian I. Rood. "Synergistic Effects of Alpha-Toxin and Perfringolysin O in Clostridium perfringens-Mediated Gas Gangrene." Infection and Immunity 69, no. 12 (December 1, 2001): 7904–10. http://dx.doi.org/10.1128/iai.69.12.7904-7910.2001.

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ABSTRACT To examine the synergistic effects of alpha-toxin and perfringolysin O in clostridial myonecrosis, homologous recombination was used to construct an alpha-toxin deficient derivative of a perfringolysin O mutant of Clostridium perfringens. The subsequent strain was complemented with separate plasmids that carried the alpha-toxin structural gene (plc), the perfringolysin O gene (pfoA), or both toxin genes, and the resultant isogenic strains were examined in a mouse myonecrosis model. Synergistic effects were clearly observed in these experiments. Infection with the control strain, which

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Dissertations / Theses on the topic "Alpha toxin"

1

Guttenberg, Gregor [Verfasser], and Manfred [Akademischer Betreuer] Jung. "Clostridiale Glukosylierende Toxine: Untersuchungen zur Autoprozessierung von Clostridium sordellii Letalem Toxin und Clostridium novyi alpha-Toxin sowie funktionelle Charakterisierung von Clostridium perfringens TpeL-Toxin." Freiburg : Universität, 2012. http://d-nb.info/1123467994/34.

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2

Eaton, Julian Timothy. "Structural studies of Clostridium perfringens alpha toxin." Thesis, Birkbeck (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.417896.

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3

Justin, Neil. "Structural studies of clostridium perfringens alpha toxin." Thesis, Birkbeck (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392355.

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4

Reutemann, Mathias. "ICAM-1 abhängige Akkumulation neutrophiler Granulozyten und Leukotrien-vermittelte Kardiodepression in Staphylococcus aureus [alpha]-Toxin-perfundierten [Alpha-Toxin-perfundierten] Rattenherzen." [S.l.] : [s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=965811611.

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Carnegie, Andrew Mark. "Effects of C.perfringens alpha toxin on cell signalling." Thesis, Birkbeck (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.416052.

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6

Wegner, Judith. "Exotoxin-Schock, ausgelöst durch Staphylococcus-aureus-[alpha]-Toxin [Staphylococcus-aureus-alpha-Toxin], am Tiermodell Ratte Auswirkungen auf Gefässendothel, Leukozytenakkumulation und Thrombozytenaggregation /." [S.l.] : [s.n.], 2005. http://deposit.ddb.de/cgi-bin/dokserv?idn=97606538X.

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Sawicki, Maria. "Immunological studies on staphylococcal alpha toxin and its fragments." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0020/MQ52656.pdf.

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8

Penha, Marcelo De Luca. "Detecção dos genes das toxinas alfa, beta e épsilon de Clostridium perfringens isolados a partir de amostras clínicas de bovinos pela reação em cadeia da polimerase." Universidade de São Paulo, 2004. http://www.teses.usp.br/teses/disponiveis/10/10134/tde-06072005-101119/.

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O Clostridium perfringens é um microrganismo anaeróbio que está presente no solo e no trato intestinal dos mamíferos. Provoca intoxicação alimentar nos seres humanos, doenças enterotoxêmicas nos animais domésticos e gangrena gasosa em ambos os grupos. O C. perfringens é classificado em cinco tipos (A, B, C, D e E) mediante a produção de quatro toxinas principais (alfa, beta, épsilon e iota). Neste trabalho foi possível padronizar a técnica de PCR para detectar a presença dos genes cpa, cpb e etx a partir de culturas de C. perfringens. A sensibilidade analítica da técnica de PCR a partir de cul

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9

Leslie, Dario Lyall. "Genetic analysis of alpha toxin (phospholipase C) from Clostridium perfringens." Thesis, University of Newcastle Upon Tyne, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.346420.

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10

Sylvester, Ian David. "The characterisation and conjugation of the fungal toxin #alpha#-sarcin." Thesis, University of Warwick, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308083.

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Books on the topic "Alpha toxin"

1

Sylvester, Ian David. The characterisation and conjugation of the fungal toxin [alpha]-sarcin. [s.l.]: typescript, 1995.

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2

International Conference on Tumor Necrosis Factor and Related Cytokines (4th 1992 Veldhoven, Netherlands). Tumor necrosis factor: Molecular and cellular biology and clinical relevance. Edited by Fiers Walter and Buurman Wim A. Basel: Karger, 1993.

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3

Mechanical stimulation of skeletal muscle increases prostaglandin F[alpha] synthesis and cyclooxygenase activity by a pertussis toxin sensitive mechanism. Providence, RI: Dept. of Pathology and Laboratory Medicine, Brown University School of Medicine and the Miriam Hospital, 1992.

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4

H, Vandenburgh Herman, and United States. National Aeronautics and Space Administration., eds. Mechanical stimulation of skeletal muscle increases prostaglandin F[́alpha] synthesis and cyclooxygenase activity by a pertussis toxin sensitive mechanism. Providence, RI: Dept. of Pathology and Laboratory Medicine, Brown University School of Medicine and the Miriam Hospital, 1992.

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5

Chapple, Christopher R., and Altaf Mangera. Urgency incontinence and overactive bladder. Edited by Christopher R. Chapple. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0040.

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Overactive bladder (OAB) is very prevalent and may be very bothersome. In this chapter, we describe the various definitions associated with this condition, its assessment and management. The definitions for lower urinary tract symptoms have been standardized by the International Continence Society. OAB is a symptomatic diagnosis after all other pathology has been excluded. Important assessments include a frequency volume chart and in some cases urodynamic studies. Here we describe the important parameters which should be sought from a frequency volume chart. In addition, the relationship to th

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6

Carpenter, Douglas W. Childhood Trauma and the Non-Alpha Male - Gender Role Conflict, Toxic Shame, and Complex Trauma: Finding Hope, Clarity, Healing, and Change. Atlantic Publishing Group Inc., 2018.

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7

Fiers, Walter, and Wim A. Buurman. Tumor Necrosis Factor: Molecular and Cellular Biology and Clinical Relevance. S Karger Pub, 1993.

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Book chapters on the topic "Alpha toxin"

1

Bhakdi, Sucharit, Iwan Walev, Matthias Husmann, and Angela Valeva. "Staphylococcal alpha-toxin." In Microbial Protein Toxins, 91–110. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/b100513.

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2

Titball, Richard W., Helen Yeoman, and Sophie E. C. Hunter. "Gene Cloning and Organization of the Alpha-Toxin of Clostridium perfringens." In Brock/Springer Series in Contemporary Bioscience, 211–26. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-7087-5_14.

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3

Nagahama, Masahiro, Sadayuki Ochi, Keiko Kobayashi, and Jun Sakurai. "The Relationship between Histidine Redidues and Various Biological Activities of Clostridium perfringens Alpha Toxin." In Advances in Experimental Medicine and Biology, 251–55. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0361-9_19.

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4

Fernandez-Miyakawa, Mariano E., and Leandro M. Redondo. "Role of Clostridium perfringens Alpha, Beta, Epsilon and Iota toxins in Enterotoxemia of monogastrics and Ruminants." In Microbial Toxins, 1–26. Dordrecht: Springer Netherlands, 2016. http://dx.doi.org/10.1007/978-94-007-6725-6_16-1.

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5

Alvin, Joseph W., and D. Borden Lacy. "Role of Clostridium Perfringens Alpha, Beta, Epsilon and Lota Toxins in Enterotoxemia of Monogastrics and Ruminants." In Microbial Toxins, 1–18. Dordrecht: Springer Netherlands, 2016. http://dx.doi.org/10.1007/978-94-007-6725-6_26-1.

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Fernandez-Miyakawa, Mariano E., and Leandro M. Redondo. "Role of Clostridium perfringens Alpha, Beta, Epsilon, and Iota Toxins in Enterotoxemia of Monogastrics and Ruminants." In Toxinology, 93–118. Dordrecht: Springer Netherlands, 2018. http://dx.doi.org/10.1007/978-94-007-6449-1_16.

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7

Wilt, Susan G., Jia Min Zhou, Steve Wesselingh, Conrad V. Kufta, and Monique Dubois-Dalcq. "Tumor Necrosis Factor Alpha Derived from Human Microglia Enhances HIV-1 Replication and is Toxic for Rat Oligodendrocytes in Vitro." In Technical Advances in AIDS Research in the Human Nervous System, 151–62. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1949-2_12.

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8

Prévost, Gilles, Mira Y. Tawk, Gaëlle Zimmermann-Meisse, and Emmanuel Jover. "The staphylococcal alpha-toxin and leukotoxins." In The Comprehensive Sourcebook of Bacterial Protein Toxins, 739–72. Elsevier, 2015. http://dx.doi.org/10.1016/b978-0-12-800188-2.00025-2.

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9

Harshman, Sidney, Nancy Sugg, and Paul Cassidy. "[1] Preparation and purification of staphylococcal alpha toxin." In Microbial Toxins: Tools in Enzymology, 3–7. Elsevier, 1988. http://dx.doi.org/10.1016/s0076-6879(88)65004-x.

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Jolivet-Reynaud, Colette, Hervé Moreau, and Joseph E. Alouf. "[14] Purification of alpha toxin from clostridium perfringens: Phospholipase C." In Microbial Toxins: Tools in Enzymology, 91–94. Elsevier, 1988. http://dx.doi.org/10.1016/s0076-6879(88)65017-8.

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Conference papers on the topic "Alpha toxin"

1

Oshima, K., S. A. McMurtry, K. I. Michael, J. A. Hippensteel, G. Su, F. Zhang, A. R. Horswill, J. Liu, R. J. Linhardt, and E. P. Schmidt. "Circulating Heparan Sulfate Binds the Staphylococcus Aureus Toxin Alpha-Hemolysin (Hla), Attenuating Hla-Induced Pulmonary Endothelial Injury." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1977.

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2

Nawroth, Peter P., Jerry Brett, Susan Steinberg, Charles T. Esmon, and David M. Stern. "ENDOTHELIUM AND PROTEIN S: SYNTHESIS, RELEASE AND REGULATION OF ANTICOAGULANT ACTIVITY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642962.

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The protein C-protein S pathway is closely linked to the vessel wall. In terms of protein C, endothelium has been shown to provide the receptor thrombomodulin, which promotes thrombin-mediated formation of activated protein C. Optimal anticoagulant function of activated protein C requires protein S and a cellular surface. Recent studies have indicated that endothelium can facilitate assembly of the activated protein C-protein S complex and that bovine endothelium expresses specific binding site(s) for protein S which promote its anticoagulant function. Expression of protein S binding sites is

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Cournoyer, Michael E., David L. Wannigman, and Robert L. Dodge. "Pollution Prevention Benefits of Dissolvable Protective Clothing." In ASME 2011 14th International Conference on Environmental Remediation and Radioactive Waste Management. ASMEDC, 2011. http://dx.doi.org/10.1115/icem2011-59003.

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Programmatic operations at the Los Alamos National Laboratory Plutonium Facility (TA-55) involve working with various amounts of plutonium and other highly toxic, alpha-emitting materials. The spread of radiological contamination on surfaces, airborne contamination, and excursions of contaminants into the operator’s breathing zone are controlled through the radiological protection program. A key element of this program is the proper selection of protective clothing. Re-useable, launderable protective clothing has been the standard for several decades. Over the years, radioactive waste disposal

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Makhoul-Mansour, Michelle M., and Eric C. Freeman. "Photo-Triggered Soft Materials With Differentiated Diffusive Pathways." In ASME 2019 Conference on Smart Materials, Adaptive Structures and Intelligent Systems. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/smasis2019-5525.

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Abstract Controlled diffusive transport between regions within a compartmentalized structure is an essential feature of cellular-inspired materials. Using the droplet interface bilayer (DIB) technique, biomolecular soft materials can be constructed in an oil medium by connecting multiple lipid-coated microdroplets together through interfacial bilayers. While traditionally achieved through the incorporation of pore forming toxins (PFTs), signal propagation within DIB assemblies can be remotely controlled through the integration of photopolymerizable phospholipids (23:2 DiynePC) into the aqueous

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Cournoyer, Michael E., Archie E. Nixon, Keith W. Fife, Arnold M. Sandoval, Vincent E. Garcia, and Robert L. Dodge. "Transuranic (TRU) Waste Volume Reduction Operation at a Plutonium Facility." In ASME 2010 13th International Conference on Environmental Remediation and Radioactive Waste Management. ASMEDC, 2010. http://dx.doi.org/10.1115/icem2010-40132.

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Programmatic operations at the Los Alamos National Laboratory Plutonium Facility (TA-55) involve working with various amounts of plutonium and other highly toxic, alpha-emitting materials. The spread of radiological contamination on surfaces, airborne contamination, and excursions of contaminants into the operator’s breathing zone are prevented through use of a variety of gloveboxes (the glovebox, coupled with an adequate negative pressure gradient, provides primary confinement). Size-reduction operations on glovebox equipment are a common activity when a process has been discontinued and the

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Reports on the topic "Alpha toxin"

1

Shor, J. T., S. P. N. Singh, and L. V. Jr Gibson. Progress report and technology status development of an EG and G Berthold LB-150 alpha/beta particulate monitor for use on the East Tennessee Technology Park Toxic Substances Control Act Incinerator. Office of Scientific and Technical Information (OSTI), June 1998. http://dx.doi.org/10.2172/638188.

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