Relevant bibliographies by topics / Angioedema

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Angioedema'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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Journal articles on the topic "Angioedema"

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Šiupšinskienė, Nora, Žygimantas Tverskis, and Sigutė Norkienė. "ANGIOTENZINĄ KONVERTUOJANČIO FERMENTO INHIBITORIAUS SUKELTOS ANGIOEDEMOS GYDYMAS." Health Sciences 30, no. 7 (2021): 35–38. http://dx.doi.org/10.35988/sm-hs.2020.173.

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Angiotenziną konvertuojančio fermento (AKF) inhibitorių sukelta angioedema yra pavojinga gyvybei būklė, kuri pasireiškia maždaug 0,5 proc. pacientų, vartojančių AKF inhbitorius. Nors AKF inhibitorių sukeltos angioedemos patofiziologija susijusi su padidėjusiu bradikinino kiekiu kraujo plazmoje, standartinis gydymas paprastai apima kortikosteroidus ir antihistamininius medikamentus. Apžvalgos tikslas yra apibendrinti įrodymus, pagrindžiančius farmakoterapiją, gydant AKF inhibitorių sukeltą angioedemą.
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Fikri, C., Y. Zarouali, M. Aboudourib, O. Hocar, and S. Amal. "Histaminic Angioedema in Children: A Rare Entity Not to be ignored." Scholars Journal of Medical Case Reports 11, no. 11 (2023): 2061–63. http://dx.doi.org/10.36347/sjmcr.2023.v11i11.036.

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Angioedema is a common symptom to different etiologies, the diagnostic orientation is based on the associated signs. The most common are histamine, non IgE-mediated, benign and often associated with urticaria. Angioedema by anaphylaxis and bradykinin angioedemas are rare but potentially severe by upper airway involvement. We report the case of a 14 years old patient with a history of bilateral orbital swelling associated with deep urticaria, and a brother followed for angioedema. Presenting bilateral palpebral and jugal edema, associated with uticarian tasks in the trunk and back. The diagnosi
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Zuraw, Bruce L., and Sandra C. Christiansen. "Pathophysiology of Hereditary Angioedema." American Journal of Rhinology & Allergy 25, no. 6 (2011): 373–78. http://dx.doi.org/10.2500/ajra.2011.25.3661.

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Background Laryngeal angioedema may be associated with significant morbidity and even mortality. Because of the potential severity of attacks, both allergists and otolaryngologists must be knowledgeable about the recognition and treatment of laryngeal angioedema. This study describes the clinical characteristics and pathophysiology of bradykinin-mediated angioedema. Methods A literature review was conducted concerning the clinical characteristics and pathophysiology of types I and II hereditary angioedema (HAE), type III HAE, acquired C1 inhibitor (C1INH) deficiency, and angiotensin-converting
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Ertürk, Nihal, Şadan Soyyiğit, Gülhan Kurtoğlu Çelik, and Edip Varan. "Assessment of emergency physicians’ awareness and knowledge of hereditary angioedema." Intercontinental Journal of Emergency Medicine 2, no. 3 (2024): 50–55. http://dx.doi.org/10.51271/icjem-0038.

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Aims: Hereditary angioedema can occur with life-threatening attacks of severe laryngeal edema, and epinephrine is insufficient in the treatment of attacks. We sought an answer to the question, 'Do emergency physicians, who frequently encounter angioedema cases that are so important for the emergency department, have sufficient awareness about this issue?' Methods: In this study, the online questionnaire was conducted among physicians working in adult emergency departments between April and August 2022. The questionnaire form consisted of two parts. The first part contained three questions abou
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Cosme, Joana, Amélia Spínola, Manuel Branco Ferreira, and Manuel Pereira Barbosa. "Hospitalizations due to Angioedema without Urticaria in a Portuguese Center: Five Year Retrospective Study." Acta Médica Portuguesa 32, no. 11 (2019): 714. http://dx.doi.org/10.20344/amp.11893.

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Introduction: Hospitalizations due to angioedema are important especially in debilitating or life-threatening situations. The aim of this study was to evaluate the frequency and etiology of angioedema without urticaria in hospital admissions.Material and Methods: The admissions between 2009 and 2013 in Centro Hospitalar Lisboa Norte with a diagnosis grouped under the ICD9 codes of angioedema were retrospectively analysed. The episodes of angioedema with urticaria were excluded. The admissions were categorized into 2 groups: A — hospitalizations motivated by the angioedema; B — hospitalizations
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Haydon, Richard. "Urticaria-angioedema and nonurticarial angioedema." Current Opinion in Otolaryngology & Head and Neck Surgery 8, no. 3 (2000): 239–44. http://dx.doi.org/10.1097/00020840-200006000-00021.

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Young, Marielle C., and Aleena Banerji. "Angioedema without urticaria: Diagnosis and management." Allergy and Asthma Proceedings 46, no. 3 (2025): 185–91. https://doi.org/10.2500/aap.2025.46.250013.

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Angioedema is nonpitting swelling that involves the deeper subcutaneous and submucosal layers of tissue. Angioedema can be classified as histaminergic, bradykinin mediated, or idiopathic in etiology. Bradykinin-mediated angioedema presents without urticaria, whereas histaminergic angioedema is usually associated with urticaria (i.e., chronic spontaneous urticaria and angioedema) but manifests with isolated angioedema in ∼20% of patients and clinically overlaps with idiopathic angioedema. Bradykinin-mediated angioedema most commonly occurs in hereditary angioedema (HAE) with or without C1-ester
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Kulthanan, Kanokvalai, Sukhum Jiamton, Kanonrat Boochangkool, and Kowit Jongjarearnprasert. "Angioedema: Clinical and Etiological Aspects." Clinical and Developmental Immunology 2007 (2007): 1–6. http://dx.doi.org/10.1155/2007/26438.

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Angioedema is an abrupt swelling of the skin, mucous membrane, or both including respiratory and gastrointestinal tracts. This study aimed to report an experience of angioedema in a university hospital with respect to etiologies, clinical features, treatment, and outcome. One hundred and five patients were enrolled. About half had angioedema without urticaria. The common sites of involvement were periorbital area and lips. Forty five patients (49%) had systemic symptoms. The most common cause of angioedema was allergic angioedema. Nonsteroidal anti-inflammatory drug-induced angioedema and idio
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Holguín-Gómez, Luisa María, Luz Adriana Vásquez-Ochoa, and Ricardo Cardona. "Angioedema." Revista Alergia México 63, no. 4 (2016): 373. http://dx.doi.org/10.29262/ram.v63i4.220.

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El angioedema se define como el edema de piel o mucosas, incluidas las de los tractos respiratorio y gastrointestinal, de carácter autolimitado, que en la mayoría de los casos se resuelve en forma completa en menos de 72 horas. Ocurre por aumento de la permeabilidad de los capilares mucosos, submucosos y vénulas poscapilares, con la consiguiente extravasación del plasma. Existen diferentes tipos de angioedema: el histaminérgico (que puede ser mediado o no por inmunoglobulina E), el hereditario, por déficit de C1 inhibidor adquirido, por inhibidores de la enzima convertidora de la angiotensina,
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Southward, Jessica, Elizabeth Irvine, and Marina Rabinovich. "Probable Amlodipine-Induced Angioedema." Annals of Pharmacotherapy 43, no. 4 (2009): 772–76. http://dx.doi.org/10.1345/aph.1l527.

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Objective: To report a case of angioedema likely associated with amlodipine administration in a patient with a right thalamic hemorrhagic stroke. Case Summary: A 50-year-old female experienced angioedema during hospitalization for s right thalamic hemorrhagic stroke. She had no past history of angioedema and all of her medications were assessed for risk of angioedema. After careful evaluation, case reports linking calcium channel blockers (CCBs) and angioedema led to further examination of amlodipine as a cause. Amlodipine therapy had been initiated 24 hours prior to the development of angioed
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Dissertations / Theses on the topic "Angioedema"

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Gomide, Maria Abadia Consuelo Machado e. Silva. "Avaliação da qualidade de vida de pacientes com angioedema hereditário." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/5/5133/tde-27102011-093455/.

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INTRODUÇÃO: O angioedema hereditário (AEH) é uma doença rara, causada pela deficiência do inibidor de C1 esterase (C1-INH), que se manifesta por ataques recorrentes e imprevisíveis de edema em face, extremidades, genitais, tronco e trato gastrintestinal. O edema em vias superiores pode levar a asfixia e morte; enquanto que as crises de dor abdominal podem conduzir à laparatomias desnecessárias. Por suas características, o AEH afeta profundamente a qualidade de vida (QV) de seus portadores, tanto na esfera física, como psicológica e social. O presente estudo teve como objetivo avaliar a qualida
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Nordenfelt, Patrik. "Hereditary Angioedema in Sweden : a National Project." Doctoral thesis, Linköpings universitet, Institutionen för klinisk och experimentell medicin, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-142207.

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Background: Hereditary angioedema (HAE) due to C1-inhibitor deficiency, type I and II, is a rare disease with an estimated prevalence of 1/50,000. Angioedema in the larynx can be life threatening and angioedema in the abdomen and skin can give severe and disabling pain. Data on patients with HAE in Sweden were scarce before our study. Aim: To study the prevalence of HAE, and to investigate clinical manifestations, treatments, and Health-Related Quality of Life (HR-QoL) in adults and children in Sweden. Method: In studies, I and II, all patients received a written questionnaire followed by a ph
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Johansson, Caroline. "Genome-wide association study of drug-induced angioedema." Thesis, Uppsala universitet, Institutionen för biologisk grundutbildning, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-373135.

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Cumming, S. A. "The genetic basis of C1 inhibitor deficiency and hereditary angioedema." Thesis, University of Cambridge, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598213.

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Hereditary angioedema (HAE) is characterised by recurrent episodes of acute swelling in localised areas of the skin, mucous membranes or internal organs. Death from laryngeal oedema is a constant threat. HAE results from mutations within the C1 inhibitor gene leading to a plasma deficiency of C1 inhibitor. It can be divided into two phenotypic groups: Type I which is characterised by a low plasma level of normal C1 inhibitor and Type II which is characterised by normal or elevated plasma level of a dysfunctional mutant. The focus of my study was to determine the genotype-phenotype relationship
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HAN, EUN DUK. "DEFINING THE BIOLOGICAL FUNCTION OF C1 INHIBITOR IN HEREDITARY ANGIOEDEMA." University of Cincinnati / OhioLINK, 2002. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1022759212.

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Silvares, Maria Regina Cavariani [UNESP]. "Estudo da associação entre urticária crônica idiopática e autoimunidade tireóidea: aspectos clínicos, positividade do teste do soro autólogo da pele e expressão gênica do receptor de TSH." Universidade Estadual Paulista (UNESP), 2010. http://hdl.handle.net/11449/102647.

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Made available in DSpace on 2014-06-11T19:32:13Z (GMT). No. of bitstreams: 0 Previous issue date: 2010-08-20Bitstream added on 2014-06-13T21:03:39Z : No. of bitstreams: 1 silvares_mrc_dr_botfm.pdf: 1230751 bytes, checksum: a7b798cd6de13023c34b6f92ec176d41 (MD5)<br>Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)<br>A conexão pele-tireoide tornou-se uma fronteira de interesse da dermatoendocrinologia. Neste contexto, a relação entre urticária crônica idiopática (UCI) e autoimunidade tireóidea tem sido estudada nos seus diferentes aspectos. Além disso, de 30% a 40% dos casos
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Suffritti, C. "Marcatori di severità di malattia nei soggetti con angioedema da carenza C1 inibitore." Doctoral thesis, Università degli Studi di Milano, 2011. http://hdl.handle.net/2434/166930.

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Markers of disease severity in angioedema due to C1 inhibitor deficiency Background Hereditary angioedema (HAE) is characterized by recurrent attacks of edema affecting skin, gastrointestinal tract, and larynx. HAE results from an inherited deficiency (type I) or dysfunction (type II) of C1 inhibitor (C1-INH). C1-INH has a broad spectrum of activities inhibiting complement, contact system, coagulation and fibrinolysis. During acute attacks, unregulated active kallikrein cleaves high-molecular weight kininogen (HK) releasing bradykinin, the mediator of the increased vascular permeability
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Silvares, Maria Regina Cavariani. "Estudo da associação entre urticária crônica idiopática e autoimunidade tireóidea : aspectos clínicos, positividade do teste do soro autólogo da pele e expressão gênica do receptor de TSH /." Botucatu : [s.n.], 2010. http://hdl.handle.net/11449/102647.

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Resumo: A conexão pele-tireoide tornou-se uma fronteira de interesse da dermatoendocrinologia. Neste contexto, a relação entre urticária crônica idiopática (UCI) e autoimunidade tireóidea tem sido estudada nos seus diferentes aspectos. Além disso, de 30% a 40% dos casos de UCI apresentam etiologia autoimune. A tireoidite de Hashimoto (TH) representa a mais prevalente doença autoimune da tireoide (DAT), acarretando, frequentemente, hipotireoidismo. Os indícios que o quadro de UCI é mais grave e menos responsivo aos tratamentos convencionais quando associado à DAT, sugerem um papel da tireotrofi
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Aulak, Kulwant Singh. "Analysis of dysfunctional C1-inhibitor proteins isolated from individuals with type II hereditary angioedema." Thesis, University of Cambridge, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.333149.

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Correia, Alexandre Pires. "Avaliação de mutações no gene do inibidor de C1 esterase em pacientes com angioedema hereditário." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/5/5133/tde-24022010-173619/.

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A ativação dos sistemas complemento e de contato resulta na formação de peptídeos vasoativos tais como a bradicinina e anafilatoxinas. O inibidor de C1-esterase (C1-INH) é o principal regulador desses dois sistemas e a deficiência desta proteína resulta no Angioedema Hereditário (AEH). Trata-se de uma doença rara, de herança autossômica dominante, caracterizada pela deficiência de C1-INH, a qual ocorre devido a mutações no seu gene estrutural, levando a episódios graves de edema em tecido subcutâneo, gastrointestinal e respiratório, potencialmente fatais. Existem dois fenótipos variantes: AEH
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Books on the topic "Angioedema"

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Zuberbier, Torsten, Clive Grattan, and Marcus Maurer, eds. Urticaria and Angioedema. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-84574-2.

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Zuberbier, Torsten, Clive E. H. Grattan, and Marcus Maurer, eds. Urticaria and Angioedema. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-79048-8.

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Zuberbier, T. Urticaria and Angioedema. Springer-Verlag Berlin Heidelberg, 2010.

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W, Greaves M., and Kaplan Allen P, eds. Urticaria and angioedema. Marcel Dekker, 2004.

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W, Greaves M., and Kaplan Allen P, eds. Urticaria and angioedema. 2nd ed. Informa Healthcare USA, 2009.

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Bas, Murat. Angioedema congress book: 3 tables : XD-US. Thieme, 2009.

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Maurer, Marcus, Torsten Zuberbier, and Clive Grattan. Urticaria and Angioedema. Springer, 2016.

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Maurer, Marcus, Torsten Zuberbier, and Clive Grattan. Urticaria and Angioedema. Springer International Publishing AG, 2021.

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Kaplan, Allen, and Malcolm Greaves. Urticaria and Angioedema. CRC Press, 2009. http://dx.doi.org/10.1201/9780367802868.

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Greaves, Malcolm W., and Allen Kaplan. Urticaria and Angioedema. Taylor & Francis Group, 2019.

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Book chapters on the topic "Angioedema"

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Möller, P., and B. M. Henz. "Angioedema." In Urticaria. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-85267-1_4.

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Czarnetzki, Beate M. "Angioedema." In Urticaria. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70313-3_4.

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Mehlhorn, Heinz. "Angioedema." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_4512.

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Mehlhorn, Heinz. "Angioedema." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_4512-1.

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Borzova, Elena, and Clive E. H. Grattan. "Angioedema." In Urticaria and Angioedema. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-79048-8_14.

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Feteih, Abeer, Farida Almarzooqi, Michael Fein, Geneviève Genest, Hoang Pham, and Moshe Ben-Shoshan. "Angioedema." In The Manual of Allergy and Clinical Immunology. CRC Press, 2021. http://dx.doi.org/10.1201/9781003174202-13.

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Bouillet, L. "Angioedema." In Urticaria and Angioedema. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-84574-2_9.

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Ramos-Romey, Cristina J., and Timothy J. Craig. "Idiopathic Angioedema." In Challenging Cases in Allergic and Immunologic Diseases of the Skin. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-60761-296-4_4.

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Prematta, Michael J., and Timothy J. Craig. "Hereditary Angioedema." In Challenging Cases in Allergic and Immunologic Diseases of the Skin. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-60761-296-4_5.

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Mantovano, Pietro, Marco Cicardi, and Andrea Zanichelli. "Angioedema Ereditario." In Le malattie rare del sistema immunitario. Springer Milan, 2013. http://dx.doi.org/10.1007/978-88-470-5394-6_19.

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Conference papers on the topic "Angioedema"

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Parati, Monica, Beatrice De Maria, Luca Ranucci, et al. "Cardiovascular response to socially-evaluated cold pressor test in patients affected by hereditary angioedema." In 2024 13th Conference of the European Study Group on Cardiovascular Oscillations (ESGCO). IEEE, 2024. http://dx.doi.org/10.1109/esgco63003.2024.10766987.

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Peñasco, Paloma, Mª Sonsoles de la Hoz, Pilar Costa, Vanesa Hidalgo, Ana Isabel Sánchez, and Nuria Asensio. "Angioedema recidivante." In XXIX Congreso Nacional de Medicina General y de Familia y V Jornadas SEMG Andalucía. Grupo Pacífico, 2023. http://dx.doi.org/10.48158/semg23-246.

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"12TH C1-INHIBITOR DEFICIENCY & ANGIOEDEMA WORKSHOP." In 12TH C1-INHIBITOR DEFICIENCY & ANGIOEDEMA WORKSHOP. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88966-549-5.

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De Souza, Júlia Diniz, Citânia Cordeiro Da Nóbrega, Nefertite Augusta Guimarães De Oliveira, and Samara Rayssa Da Silva Mandu. "ANGIOEDEMA HEREDITÁRIO: PRÁTICAS IATROGÊNICAS." In I Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/968.

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INTRODUÇÃO: O angioedema hereditário (AEH) é uma doença autossômica dominante, caracterizada por episódios frequentes de edema subcutâneo e/ou submucoso. No Brasil, embora a prevalência do AEH seja baixa, quando não diagnosticada corretamente a mortalidade pode ser superior a 30%. A patologia atualmente ainda é de pouco conhecimento médico, por vezes, o diagnóstico é tardio, provocando práticas iatrogênicas que podem resultar no óbito. OBJETIVOS: Analisar as publicações científicas sobre as intervenções iatrogênicas causadas no AEH. MATERIAL E MÉTODOS: Foi elaborada uma revisão bibliográfica,
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Saed, Kaywan M., Kishan Patel, Paige DeLuca, Saipriya Gadiraju, James A. Espinosa, and Alan Lucerna. "Amlodipine-Induced Angioedema: A Case Report and Literature Review." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.68_2024.

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Angioedema is a medical condition characterized by rapid and localized swelling in the deeper layers of the skin and mucous membranes. Unlike hives (urticaria), which affect the superficial layers of the skin and appear as raised, itchy welts, angioedema involves swelling beneath the skin's surface and commonly affects areas such as the face, lips, tongue, throat, hands, feet, and genitals. This swelling can be dramatic and may cause discomfort, pain, or difficulty with breathing or swallowing, depending on the location and severity of the swelling. Here we present the case of 73-year-old pati
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Datar, P. B., E. U. R. Rahman, B. Bhattarai, et al. "Coronary Angioedema- An Unknown Complication." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3518.

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Rattu, Mohammad A., and Eric A. Maddock. "Quincke’s Phenomenon – The ACE Inhibitor Culprit." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.85_2024.

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Angioedema is defined as a non-pitting edema that involves the subcutaneous layer and additionally may include submucosal layers of tissue which pertain to the face, oral cavity, larynx, lips, extremities and gastrointestinal tract; this becomes a life-threatening situation particularly when there is involvement of the larynx. Angioedema may be classified as either histamine-mediated or bradykinin-mediated. Histamine-mediated, associated with mast-cell and basophil activation, is the most common. Bradykinin-mediated (secondary to hereditary, acquired C1-inhibitor deficiency, ACEI-associated an
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Greiner, B., J. Pelton, and D. Baker. "Hypothyroidism Associated Angioedema: A Case Report." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5173.

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Thomas, S. "Toxic Relationship with Labetalol Rare Angioedema." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1343.

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Sharma, P., J. P. Dugan, and S. K. Raju. "Medication Resistant Angioedema and Airway Compromise." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a1370.

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