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Journal articles on the topic 'Anhedonic depression'

Author: Grafiati

Published: 4 June 2021

Last updated: 13 February 2022

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1

Nizet, Laurie, Xavier Montana, Jean-Pol Lanquart, and Gwenolé Loas. "Research into an Association between Anhedonia and Decreased REM Latency in Moderately to Severely Depressed Patients." Sleep Disorders 2018 (July 2, 2018): 1–6. http://dx.doi.org/10.1155/2018/1636574.

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Anhedonia stands as a core symptom and potential trait marker of major depressive disorder (MDD). The importance of rapid eye movement sleep latency (REML) as a biological marker of depression has previously and repeatedly been studied. The aim of this paper is to analyse the relationship between anhedonia and REML in moderately to severely depressed patients. The shortened Beck Depression Inventory (BDI-13) was chosen to assess depressive symptoms and, among them, more particularly, anhedonic symptoms. Two-way ANCOVA was used for statistical analyses. A significant association between anhedon

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De Fruyt, Jürgen, Bernard Sabbe, and Koen Demyttenaere. "Anhedonia in Depressive Disorder: A Narrative Review." Psychopathology 53, no. 5-6 (2020): 274–81. http://dx.doi.org/10.1159/000508773.

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Since the introduction of DSM-III anhedonia has become a core depressive criterion and is defined as the loss of interest or pleasure. Although the origin of the word goes back to the end of the 19th century and numerous anhedonic symptoms are described in classic texts on depression, this centrality in the diagnosis of depression is only recent. Anhedonia is best described as a symptom complex with unclear boundaries cutting across the tripartite model of the mind (affect, volition, and cognition). Popular concepts of anhedonia pertain to the pleasure cycle and positive affectivity. These con

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Corral-Frías, N. S., Y. S. Nikolova, L. J. Michalski, D. A. A. Baranger, A. R. Hariri, and R. Bogdan. "Stress-related anhedonia is associated with ventral striatum reactivity to reward and transdiagnostic psychiatric symptomatology." Psychological Medicine 45, no. 12 (2015): 2605–17. http://dx.doi.org/10.1017/s0033291715000525.

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BackgroundEarly life stress (ELS) is consistently associated with increased risk for subsequent psychopathology. Individual differences in neural response to reward may confer vulnerability to stress-related psychopathology. Using data from the ongoing Duke Neurogenetics Study, the present study examined whether reward-related ventral striatum (VS) reactivity moderates the relationship between retrospectively reported ELS and anhedonic symptomatology. We further assessed whether individual differences in reward-related VS reactivity were associated with other depressive symptoms and problemati

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Mazo, G. E., and A. O. Kibitov. "Anhedonia as a basic syndrome and a target for treatment for depressive disorder." V.M. BEKHTEREV REVIEW OF PSYCHIATRY AND MEDICAL PSYCHOLOGY, no. 3 (October 6, 2019): 10–18. http://dx.doi.org/10.31363/2313-7053-2019-3-10-18.

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The article presents an analytical review aimed at determining the effect of anhedonia on the course and therapeutic prognosis of depression. Currently, there is no specific pharmacological approach to the treatment of anhedonia in depression.It is clear all antidepressants that affect the symptoms of depression in general act to a certain extent on anhedonia. Modern views on the biological mechanisms involved in the formation of anhedonia are described, on the basis of which a search for drugs with anti-anhedonic activity is possible. Vortioxetine can be considered as an antidepressant with a

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Chaturvedi, S. K. "Anhedonic Depression." British Journal of Psychiatry 149, no. 4 (1986): 521–22. http://dx.doi.org/10.1192/s0007125000139832.

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Von Duering, F. "Anhedonic brain while attending sexual and emotional pictures." European Psychiatry 33, S1 (2016): S161. http://dx.doi.org/10.1016/j.eurpsy.2016.01.316.

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Anhedonia is defined as the inability to gain pleasure from normally pleasurable experiences and reduced sexual desire. Rees et al. (2007) showed that limbic and paralimbic areas are responsible for sexual arousal and that anhedonia is associated with frontolimbic inhibition. In major depression, reduced ventral striatum and increased ventral prefrontal cortex areas was associated with anhedonia(Gorwood, 2009). Walter et al. (2009) indicated that there is a deviation in the neuronal activation pattern of the pregenual anterior cingulate cortex in anhedonic depression which is related to a glut

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Grippo, Angela J., Claudia M. Santos, Ralph F. Johnson, et al. "Increased susceptibility to ventricular arrhythmias in a rodent model of experimental depression." American Journal of Physiology-Heart and Circulatory Physiology 286, no. 2 (2004): H619—H626. http://dx.doi.org/10.1152/ajpheart.00450.2003.

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Depression is an important public health problem and is considered to be an independent risk factor for coronary artery disease. The pathophysiological mechanisms that link depression with adverse cardiovascular events (e.g., myocardial ischemia, myocardial infarction, and sudden death) are not well established. It is possible that an increased susceptibility to life-threatening cardiac arrhythmias in depressed patients influences the risk of morbidity and mortality in coronary artery disease. This idea was tested with the use of an experimental model of depression that was developed to induce

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Abramova, L. I., G. P. Panteleeva, I. Yu Nikiforova, and T. E. Novozhenova. "Differentiated approach and indications for optimization of agomelatine therapy for endogenous depression." Neurology, Neuropsychiatry, Psychosomatics 11, no. 2 (2019): 71–77. http://dx.doi.org/10.14412/2074-2711-2019-2-71-77.

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Objective: to develop and justify differentiated indications for the use of agomelatine (valdoxan) to treat the typological variants of endogenous depressions with varying severity on the basis of an analysis of its therapeutic efficacy.Patients and methods. An open prospective study was conducted using the clinical, psychopathological, and psychometric rating scales: the Hamilton Depression Rating Scale (HAMD-21); Udvalg for Kliniske Undersњgelser Scale (UKU); the Snaith-Hamilton Pleasure Scale (SHAPS) for assessing anhedonic disorders, and statistical methods. Examinations were made in 56 pa

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9

Steer, Roberta. "Self-Reported Inability to Cry as a Symptom of Anhedonic Depression in Outpatients with a Major Depressive Disorder." Psychological Reports 108, no. 3 (2011): 874–82. http://dx.doi.org/10.2466/02.09.13.15.pr0.108.3.874-882.

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To ascertain whether self-reported inability to cry would be associated with symptoms of anhedonic depression, the 21-item Beck Depression Inventory-II was administered to 1,050 outpatients diagnosed with a DSM-IV-TR major depressive disorder. 219 (21%) patients reported on the BDI-II Crying item that they were unable to cry, and 831 (79%) patients reported they were able to cry. Only BDI-II Loss of Interest was significantly associated with the inability to cry after the other BDI-II symptoms were controlled for using a multiple logistic-regression analysis. The inability to cry was discussed

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Stones, Michael J., Leah D. Clyburn, Margaret C. Gibson, and M. Gail Woodbury. "Predicting Diagnosed Depression and Anti-depressant Treatment in Institutionalized Older Adults by Symptom Profiles: A Closer Look at Anhedonia and Dysphoria." Canadian Journal on Aging / La Revue canadienne du vieillissement 25, no. 2 (2006): 153–59. http://dx.doi.org/10.1353/cja.2006.0042.

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ABSTRACTThe purpose of this study was to examine the relationships of diagnosis and treatment of depression with anhedonic and dysphoric symptom presentation, using the Minimum Data Set 2.0. Participants were from two sectors of long-term care: 70 nursing home residents and 92 residents in a Veterans' Care Service. The samples differed in their sex distribution and in cognition. A series of logistic regressions that controlled for demographics, type of facility, and cognition showed that dysphoric symptoms predicted diagnosed depression, whereas anhedonic symptoms predicted anti-depressant med

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Conan, C., J. M. Batail, I. Corouge, J. Palaric, G. Robert, and D. Drapier. "Apathy in depression: An arterial spin labeling study." European Psychiatry 41, S1 (2017): S344—S345. http://dx.doi.org/10.1016/j.eurpsy.2017.02.311.

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IntroductionApathy is usually defined as a lack of goal-directed behavior. Although it is observed in about 30% of depressed patients, neurovascular mechanisms underpinning apathy remain little-known.ObjectivesThe main objective of this study was to compare the cerebral perfusion of apathetic depressed patients with non-apathetic depressed patients by arterial spin labeling (ASL), a quantitative and non-invasive perfusion magnetic resonance imaging (MRI) technique. The secondary objectives were to study their clinical profile and their correlation with cerebral perfusion data.MethodsThis study

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Berenbaum, Howard, Keith Bredemeier, Renee J. Thompson, and Matthew Tyler Boden. "Worry, Anhedonic Depression, and Emotional Styles." Cognitive Therapy and Research 36, no. 1 (2010): 72–80. http://dx.doi.org/10.1007/s10608-010-9329-8.

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Young, Michael A., William A. Scheftner, Gerald L. Klerman, Nancy C. Andreasen, and Robert M. A. Hirschfeld. "The Endogenous Sub-type of Depression: A Study of its Internal Construct Validity." British Journal of Psychiatry 148, no. 3 (1986): 257–67. http://dx.doi.org/10.1192/bjp.148.3.257.

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The internal construct validity of the endogenous sub-type of major depression was investigated by statistically modelling the RDC endogenous and DSM-III melancholia diagnostic criteria. Data consisted of symptom ratings on 788 patients with major depression from NIMH Collaborative Depression Study. Results indicated that the symptoms in the criteria do not specify a dichotomous classification, melancholic-non-melancholic or endogenous-nonendogenous. Results did support the existence of two sub-typings, one related to anhedonia, and one related to vegetative symptoms. The vegetative sub-type r

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Robinson, Oliver J., and Henry W. Chase. "Learning and Choice in Mood Disorders: Searching for the Computational Parameters of Anhedonia." Computational Psychiatry 1 (December 2017): 208–33. http://dx.doi.org/10.1162/cpsy_a_00009.

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Computational approaches are increasingly being used to model behavioral and neural processes in mood and anxiety disorders. Here we explore the extent to which the parameters of popular learning and decision-making models are implicated in anhedonic symptoms of major depression. We first highlight the parameters of reinforcement learning that have been implicated in anhedonia, focusing, in particular, on the role that choice variability (i.e., “temperature”) may play in explaining heterogeneity across previous findings. We then turn to neuroimaging findings implicating attenuated ventral stri

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15

Wellen, Marcus. "Differentiation Between Demoralization, Grief, and Anhedonic Depression." Current Psychiatry Reports 12, no. 3 (2010): 229–33. http://dx.doi.org/10.1007/s11920-010-0106-x.

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Domaradzka, Ewa, and Małgorzata Fajkowska. "Structure of Affect in Types of Anxiety and Depression." Journal of Individual Differences 40, no. 2 (2019): 82–91. http://dx.doi.org/10.1027/1614-0001/a000279.

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Abstract. Although anxiety and depression are treated as separate conditions, the literature concerning their affective functioning is inconclusive. To address this issue, we analyzed types of anxiety (arousal and apprehension) and depression (valence and anhedonic). We hypothesized that the similarities and differences in the structure of affect would be related to the specific structural components and functions (reactive or regulative) of the types. We assessed the valence (negative and positive affect) and content (specific emotions) of affect by the Positive and Negative Affect Schedule –

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Song, Xin, and Iris Vilares. "Assessing the relationship between the human learned helplessness depression model and anhedonia." PLOS ONE 16, no. 3 (2021): e0249056. http://dx.doi.org/10.1371/journal.pone.0249056.

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The learned helplessness (LH) model is one of the most commonly used acute stress models to explain depression and it has shown good face and predictive validity. However, despite being able to induce depressed-like behaviors and corresponding psychophysiological changes, there is little evidence showing that the LH paradigm can produce anhedonia, a core symptom seen in all forms of depression in humans. So far a couple of studies showed that rodents bred for helplessness develop anhedonic-like behaviors in response to stress; yet, to the best of our knowledge, no similar human research has tr

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Mueller, Erik M., Christian Panitz, Diego A. Pizzagalli, Christiane Hermann, and Jan Wacker. "Midline theta dissociates agentic extraversion and anhedonic depression." Personality and Individual Differences 79 (June 2015): 172–77. http://dx.doi.org/10.1016/j.paid.2014.10.043.

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Young, Katherine S., Susan Y. Bookheimer, Robin Nusslock, et al. "Dysregulation of threat neurociruitry during fear extinction: the role of anhedonia." Neuropsychopharmacology 46, no. 9 (2021): 1650–57. http://dx.doi.org/10.1038/s41386-021-01003-8.

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AbstractDimensional models of anxiety and depression highlight common and distinct symptom clusters that are thought to reflect disruptions in underlying functional processes. The current study investigated how functioning of threat neurocircuitry relates to symptom dimensions of anxiety and depression. Participants were aged 18–19 years (n = 229, 158 female) and were selected to ensure a range of scores on symptom measures. Symptom dimensions of “General Distress” (common to anxiety disorders and depression), “Fears” (more specific to anxiety disorders), and “Anhedonia-apprehension” (more spe

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Millón, Carmelo, Antonio Flores-Burgess, Belén Gago, et al. "Role of the galanin N-terminal fragment (1-15) in anhedonia: Involvement of the dopaminergic mesolimbic system." Journal of Psychopharmacology 33, no. 6 (2019): 737–47. http://dx.doi.org/10.1177/0269881119844188.

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Background: Anhedonia is a core feature of depressive disorders. The galanin N-terminal fragment (1-15) plays a role in mood regulation since it induces depression and anxiogenic-like effects in rats. In this study, we analysed galanin N-terminal fragment (1-15) actions in anhedonic-like behaviours in rats using operant and non-operant tests and the areas involved with these effects. Methods: Galanin N-terminal fragment (1-15) effects were analysed in saccharin self-administration, sucrose preference, novelty-suppressed feeding and female urine sniffing tests. The areas involved in galanin N-t

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Sachs-Ericsson, Natalie J., Greg Hajcak, Julia L. Sheffler, et al. "Putamen Volume Differences Among Older Adults: Depression Status, Melancholia, and Age." Journal of Geriatric Psychiatry and Neurology 31, no. 1 (2017): 39–49. http://dx.doi.org/10.1177/0891988717747049.

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Background: Individuals with major depressive disorder (MDD) may exhibit smaller striatal volumes reflecting deficits in the reward circuit. Deficits may change with age and be more pronounced among the melancholic subtype. Limited research has investigated striatal volume differences in older adults and by depression subtypes. Method: We used baseline data from the Neurocognitive Outcomes of Depression in the Elderly study. We examined volumetric differences in the putamen and caudate nucleus among older adults (60 years and older), comparing healthy control participants (n = 134) to depresse

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Bean, C. A., and J. A. Ciesla. "1099 Sleep Restriction Leads To Greater Next-day Anxiety: The Moderating Role Of Anxiety And Depression." Sleep 43, Supplement_1 (2020): A418. http://dx.doi.org/10.1093/sleep/zsaa056.1094.

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Abstract Introduction Evidence from both experimental and daily-diary studies suggests that a single night of sleep restriction leads to higher levels of anxiety the following day. Depressive symptoms also increase the next day in healthy populations, although sleep restriction has demonstrated short-term antidepressant properties in depressed populations. Relatively little research has examined symptoms of anxiety and depression separately on days following naturally-occurring sleep restriction and whether any change from the previous day might be moderated by baseline levels of anxiety or de

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Vrieze, E., D. Pizzagalli, and S. Claes. "P.2.b.005 Anhedonic depression: an endophenotypic approach." European Neuropsychopharmacology 18 (August 2008): S315—S316. http://dx.doi.org/10.1016/s0924-977x(08)70430-9.

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Kentner, A. C., S. A. McLeod, E. F. Field, and Q. J. Pittman. "Sex-Dependent Effects of Neonatal Inflammation on Adult Inflammatory Markers and Behavior." Endocrinology 151, no. 6 (2010): 2689–99. http://dx.doi.org/10.1210/en.2009-1101.

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Inflammatory molecules, such as cyclooxygenase (COX), a prostaglandin synthetic enzyme, have been identified as a marker of depressive symptomology. Previously, we have observed elevated basal COX-2 expression in the hypothalamus of adult male rats treated neonatally with lipopolysaccharide (LPS), which might suggest a phenotype for disrupted hedonic behavior, a symptom of depression. However, COX-2 and its contribution to the expression of anhedonic behavior has not been investigated in these males or in female rats across the estrous cycle, which is the purpose of the current work. Here, we

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Bogdan, R., and D. A. Pizzagalli. "The heritability of hedonic capacity and perceived stress: a twin study evaluation of candidate depressive phenotypes." Psychological Medicine 39, no. 2 (2008): 211–18. http://dx.doi.org/10.1017/s0033291708003619.

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BackgroundAnhedonia and stress sensitivity have been identified as promising depressive phenotypes. Research suggests that stress-induced anhedonia is a possible mechanism underlying the association between stress and depression. The present proof-of-concept study assessed whether hedonic capacity and stress perception are heritable and whether their genetic and environmental contributions are shared.MethodTwenty monozygotic (MZ) and 15 dizygotic (DZ) twin pairs completed a probabilistic reward task that provides an objective behavioral measure of hedonic capacity (reward responsiveness) and c

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Lago, Natalia, Fernanda N. Kaufmann, María Luciana Negro-Demontel, et al. "CD300f immunoreceptor is associated with major depressive disorder and decreased microglial metabolic fitness." Proceedings of the National Academy of Sciences 117, no. 12 (2020): 6651–62. http://dx.doi.org/10.1073/pnas.1911816117.

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A role for microglia in neuropsychiatric diseases, including major depressive disorder (MDD), has been postulated. Regulation of microglial phenotype by immune receptors has become a central topic in many neurological conditions. We explored preclinical and clinical evidence for the role of the CD300f immune receptor in the fine regulation of microglial phenotype and its contribution to MDD. We found that a prevalent nonsynonymous single-nucleotide polymorphism (C/T, rs2034310) of the human CD300f receptor cytoplasmic tail inhibits the protein kinase C phosphorylation of a threonine and is ass

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Słupski, Jakub, Wiesław Jerzy Cubała, Natalia Górska, Anita Słupska, and Maria Gałuszko-Węgielnik. "Copper and anti-anhedonic effect of ketamine in treatment-resistant depression." Medical Hypotheses 144 (November 2020): 110268. http://dx.doi.org/10.1016/j.mehy.2020.110268.

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Liston, Conor. "115. Neuroimaging Biomarkers Predicting Distinct Subtypes of Anhedonic Behavior in Depression." Biological Psychiatry 83, no. 9 (2018): S47. http://dx.doi.org/10.1016/j.biopsych.2018.02.133.

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Kühne, Stephanie, Martha Schalla, Tiemo Friedrich, et al. "Nesfatin-130-59 Injected Intracerebroventricularly Increases Anxiety, Depression-Like Behavior, and Anhedonia in Normal Weight Rats." Nutrients 10, no. 12 (2018): 1889. http://dx.doi.org/10.3390/nu10121889.

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Nesfatin-1 is a well-established anorexigenic peptide. Recent studies indicated an association between nesfatin-1 and anxiety/depression-like behavior. However, it is unclear whether this effect is retained in obesity. The aim was to investigate the effect of nesfatin-130-59—the active core of nesfatin-1—on anxiety and depression-like behavior in normal weight (NW) and diet-induced (DIO) obese rats. Male rats were intracerebroventricularly (ICV) cannulated and received nesfatin-130-59 (0.1, 0.3, or 0.9 nmol/rat) or vehicle 30 min before testing. Nesfatin-130-59 at a dose of 0.3 nmol reduced su

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Pérez-Belmonte, Sergio, Laura Galiana, Patricia Sancho, Amparo Oliver, and José M. Tomás. "Subtypes of Depression: Latent Class Analysis in Spanish Old People with Depressive Symptoms." Life 10, no. 5 (2020): 70. http://dx.doi.org/10.3390/life10050070.

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Major depressive disorder (MDD) is one of the most disabling disorders and the one that most contributes to disability. When it occurs in older people, it is an additional burden to their potential physical and cognitive deficiencies, making MDD an important public health problem that supposes a large investment in health. There is a clear lack of consistency between the subtypes of depression found in the literature, ranging from two to seven classes, with three being the most commonly found non-melancholic, melancholic and psychotic, or putative psychotics. The aim of this research is to add

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Min, Hyewon, Seok-Man Kwon, and Seul-Ah Lee. "The Influence of Imagery Processing on the Experience of Pleasure and Anhedonic Depression." Korean Journal of Clinical Psychology 38, no. 2 (2019): 157–70. http://dx.doi.org/10.15842/kjcp.2019.38.2.003.

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Green, Amanda D., Alasdair M. Barr, and Liisa A. M. Galea. "Role of estradiol withdrawal in ‘anhedonic’ sucrose consumption: A model of postpartum depression." Physiology & Behavior 97, no. 2 (2009): 259–65. http://dx.doi.org/10.1016/j.physbeh.2009.02.020.

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Bredemeier, Keith, Howard Berenbaum, James R. Brockmole, Walter R. Boot, Daniel J. Simons, and Steven B. Most. "A load on my mind: Evidence that anhedonic depression is like multi-tasking." Acta Psychologica 139, no. 1 (2012): 137–45. http://dx.doi.org/10.1016/j.actpsy.2011.11.007.

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Franzen, Jessica, and Kerstin Brinkmann. "Anhedonic symptoms of depression are linked to reduced motivation to obtain a reward." Motivation and Emotion 40, no. 2 (2015): 300–308. http://dx.doi.org/10.1007/s11031-015-9529-3.

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Silvia, Paul J., Emily C. Nusbaum, Kari M. Eddington, Roger E. Beaty, and Thomas R. Kwapil. "Effort deficits and depression: The influence of anhedonic depressive symptoms on cardiac autonomic activity during a mental challenge." Motivation and Emotion 38, no. 6 (2014): 779–89. http://dx.doi.org/10.1007/s11031-014-9443-0.

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Shabel, Steven J., Chenyu Wang, Bradley Monk, Sage Aronson, and Roberto Malinow. "Stress transforms lateral habenula reward responses into punishment signals." Proceedings of the National Academy of Sciences 116, no. 25 (2019): 12488–93. http://dx.doi.org/10.1073/pnas.1903334116.

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Neuronal activity in the lateral habenula (LHb), a brain region implicated in depression [C. D. Proulx, O. Hikosaka, R. Malinow, Nat. Neurosci. 17, 1146–1152 (2014)], decreases during reward and increases during punishment or reward omission [M. Matsumoto, O. Hikosaka, Nature 447, 1111–1115 (2007)]. While stress is a major risk factor for depression and strongly impacts the LHb, its effect on LHb reward signals is unknown. Here we image LHb neuronal activity in behaving mice and find that acute stress transforms LHb reward responses into punishment-like neural signals; punishment-like response

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Min, Hyewon, Seok-Man Kwon, and Seul-Ah Lee. "Effects of Anticipatory and Consummatory Components of the Experience of Pleasure on Anhedonic Depression." Korean Journal of Clinical Psychology 38, no. 1 (2019): 1–12. http://dx.doi.org/10.15842/kjcp.2019.38.1.001.

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Lally, N., A. C. Nugent, D. A. Luckenbaugh, R. Ameli, J. P. Roiser, and C. A. Zarate. "Anti-anhedonic effect of ketamine and its neural correlates in treatment-resistant bipolar depression." Translational Psychiatry 4, no. 10 (2014): e469-e469. http://dx.doi.org/10.1038/tp.2014.105.

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Hundt, Natalie E., Rosemery O. Nelson-Gray, Nathan A. Kimbrel, John T. Mitchell, and Thomas R. Kwapil. "The interaction of reinforcement sensitivity and life events in the prediction of anhedonic depression and mixed anxiety-depression symptoms." Personality and Individual Differences 43, no. 5 (2007): 1001–12. http://dx.doi.org/10.1016/j.paid.2007.02.021.

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Kanekar, Shami, Robert Ettaro, Michael D. Hoffman, et al. "Sex-Based Impact of Creatine Supplementation on Depressive Symptoms, Brain Serotonin and SSRI Efficacy in an Animal Model of Treatment-Resistant Depression." International Journal of Molecular Sciences 22, no. 15 (2021): 8195. http://dx.doi.org/10.3390/ijms22158195.

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Background: Rates of major depressive disorder (MDD) increase with living at altitude. In our model, rats housed at moderate altitude (in hypobaric hypoxia) exhibit increased depression-like behavior, altered brain serotonin and a lack of antidepressant response to most selective serotonin reuptake inhibitors (SSRIs). A forebrain deficit in the bioenergetic marker creatine is noted in people living at altitude or with MDD. Methods: Rats housed at 4500 ft were given dietary creatine monohydrate (CRMH, 4% w/w, 5 weeks) vs. un-supplemented diet, and impact on depression-like behavior, brain bioen

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Sbrini, Giulia, Paola Brivio, Kari Bosch, Judith Regina Homberg, and Francesca Calabrese. "Enrichment Environment Positively Influences Depression- and Anxiety-Like Behavior in Serotonin Transporter Knockout Rats through the Modulation of Neuroplasticity, Spine, and GABAergic Markers." Genes 11, no. 11 (2020): 1248. http://dx.doi.org/10.3390/genes11111248.

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The serotonin transporter (5-HTT in humans, SERT in rodents) is the main regulator of serotonergic transmission in the brain. The short allelic variant of the 5-HTT gene is in humans associated with psychopathologies and may enhance the vulnerability to develop depression after exposure to stressful events. Interestingly, the short allele also increases the sensitivity to a positive environment, which may buffer the vulnerability to depression. Since this polymorphism does not exist in rodents, male SERT knockout (SERT−/−) rats were tested to explore the molecular mechanisms based on this incr

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Lecrubier, Y. "Pharmacological interventions in dysthymia." European Psychiatry 11, S3 (1996): 129s—133s. http://dx.doi.org/10.1016/0924-9338(96)85187-x.

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SummaryThe results of early therapeutic trials (1960s and 70s) in mild chronic depression (neurotic patients) are difficult to extrapolate to dysthymia. Most studies conducted in the early 1980s showed reference drugs (TCAs and MAOIs) to be poorly effective in chronic mild depression usually defined with RDC. The most recent trials using DSM-III-R criteria for dysthymic recruitment showed TCAs, new MAOIs (RIMA) and SSRI to be effective. AU these compounds have a rather similar pharmacological mechanism of action. It is therefore of both practical and theoretical interest that amisulpride, a be

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Fang, Xing, Shujun Jiang, Jiangong Wang, et al. "Chronic unpredictable stress induces depression-related behaviors by suppressing AgRP neuron activity." Molecular Psychiatry 26, no. 6 (2021): 2299–315. http://dx.doi.org/10.1038/s41380-020-01004-x.

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AbstractPrevious studies have shown that AgRP neurons in the arcuate nucleus (ARC) respond to energy deficits and play a key role in the control of feeding behavior and metabolism. Here, we demonstrate that chronic unpredictable stress, an animal model of depression, decreases spontaneous firing rates, increases firing irregularity and alters the firing properties of AgRP neurons in both male and female mice. These changes are associated with enhanced inhibitory synaptic transmission and reduced intrinsic neuronal excitability. Chemogenetic inhibition of AgRP neurons increases susceptibility t

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Bredemeier, Keith, Jeffery M. Spielberg, Rebecca Levin Silton, Howard Berenbaum, Wendy Heller, and Gregory A. Miller. "Screening for depressive disorders using the Mood and Anxiety Symptoms Questionnaire Anhedonic Depression Scale: A receiver-operating characteristic analysis." Psychological Assessment 22, no. 3 (2010): 702–10. http://dx.doi.org/10.1037/a0019915.

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Kalmbach, David A., Jeffrey A. Ciesla, Jeffrey W. Janata, and Sheryl A. Kingsberg. "Specificity of Anhedonic Depression and Anxious Arousal with Sexual Problems Among Sexually Healthy Young Adults." Journal of Sexual Medicine 9, no. 2 (2012): 505–13. http://dx.doi.org/10.1111/j.1743-6109.2011.02533.x.

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Ahles, Joshua J., Amy H. Mezulis, and Sheila E. Crowell. "Pre-ejection period reactivity to reward is associated with anhedonic symptoms of depression among adolescents." Developmental Psychobiology 59, no. 4 (2017): 535–42. http://dx.doi.org/10.1002/dev.21518.

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Sanders, Wesley, and Jamie Abaied. "Motivational systems and autonomic functioning: Overlapping and differential contributions to anhedonic depression and anxious arousal." Motivation and Emotion 39, no. 4 (2015): 602–12. http://dx.doi.org/10.1007/s11031-015-9470-5.

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Géa, Luiza P., Rafael Colombo, Eduarda D. da Rosa та ін. "Anhedonic-like behavior correlates with IFNγ serum levels in a two-hit model of depression". Behavioural Brain Research 373 (листопад 2019): 112076. http://dx.doi.org/10.1016/j.bbr.2019.112076.

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Henningsen, Kim, Jesper T. Andreasen, Elena V. Bouzinova, et al. "Cognitive deficits in the rat chronic mild stress model for depression: Relation to anhedonic-like responses." Behavioural Brain Research 198, no. 1 (2009): 136–41. http://dx.doi.org/10.1016/j.bbr.2008.10.039.

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Dunn, Barnaby D., Iolanta Stefanovitch, Kate Buchan, Andrew D. Lawrence, and Tim Dalgleish. "A reduction in positive self-judgment bias is uniquely related to the anhedonic symptoms of depression." Behaviour Research and Therapy 47, no. 5 (2009): 374–81. http://dx.doi.org/10.1016/j.brat.2009.01.016.

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