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1

G, Benditt David, and Benson D. Woodrow, eds. Cardiac preexcitation syndromes: Origins, evaluation, and treatment. Nijhoff, 1986.

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2

Schwartz, Peter J. The long QT syndrome. Futura Pub. Co., 1997.

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3

Charles, Antzelevitch, ed. The Brugada syndrome. Futura Pub. Co., 1999.

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4

Clinical approach to sudden cardiac death syndromes. Springer, 2010.

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5

Ramrakha, Punit, and Jonathan Hill, eds. Arrhythmias. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199643219.003.0010.

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The cardiac conduction system 478Bradycardia: general approach 480Sinus bradycardia 482Sinus pause 482Sick sinus syndrome 482Atrioventricular block 483Bundle branch block 484Tachycardia: general approach 486Tachycardia: emergency management 488Tachyarrhythmias: classification 490ECG diagnosis of tachyarrhythmias 492Supraventricular tachycardia 494...
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6

Long Qt Syndrome An Issue Of Cardiac Electrophysiology Clinics. W.B. Saunders Company, 2012.

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7

QT Prolongation and ventricular arrhythmias. New York Academy of Sciences, 1992.

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8

Brugada, Pedro, Ramon Brugada, Charles Antzelevitch, and Joseph Brugada. Brugada Syndrome: From Bench to Bedside. Wiley & Sons, Limited, John, 2007.

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9

Brugada, Pedro, Ramon Brugada, Charles Antzelevitch, and Joseph Brugada. Brugada Syndrome: From Bench to Bedside. Wiley & Sons, Incorporated, John, 2008.

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10

Brugada, Pedro, Ramon Brugada, Charles Antzelevitch, and Joseph Brugada. Brugada Syndrome: From Bench to Bedside. Wiley & Sons, Incorporated, John, 2008.

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11

(Editor), David G. Benditt, and D. Woodrow Benson (Editor), eds. Cardiac Preexcitation Syndromes: Origines, Evaluation, and Treatment. Springer, 1986.

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12

Benditt, David G., and D. Woodrow Benson. Cardiac Preexcitation Syndromes: Origins, Evaluation, and Treatment. Springer, 2012.

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13

Benditt, David G., and D. Woodrow Benson. Cardiac Preexcitation Syndromes: Origins, Evaluation, and Treatment. Springer London, Limited, 2012.

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14

Baranchuk, Adrian. Interatrial Block and Supraventricular Arrhythmias: Clinical Implications of Bayés' Syndrome. Cardiotext Publishing, 2017.

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15

Baranchuk, Adrian. Interatrial Block and Supraventricular Arrhythmias: Clinical Implications of Bayés' Syndrome. Cardiotext Publishing, 2017.

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16

Plesha, Carol A. Respiratory sinus arrhythmia as an indicator of success in coping with chronic pain. 1989.

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17

Brugada, Pedro, Josep Brugada, and Ramon Brugada. Clinical Approach to Sudden Cardiac Death Syndromes. Springer, 2014.

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18

Tülümen, Erol, and Martin Borggrefe. Monogenic and oligogenic cardiovascular diseases: genetics of arrhythmias—short QT syndrome. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0150.

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Short QT syndrome (SQTS) is a very rare, sporadic or autosomal dominant inherited channelopathy characterized by abnormally short QT intervals on the electrocardiogram and increased propensity to atrial and ventricular tachyarrhythmias and/or sudden cardiac death. Since its recognition as a distinct clinical entity in 2000, significant progress has been made in defining the clinical, molecular, and genetic basis of SQTS. To date, several causative gain-of-function mutations in potassium channel genes and loss-of-function mutations in calcium channel genes have been identified. The physiologica
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19

Wesley, Keith, and Robert J. Huszar. Huszar's Basic Dysrhythmias and Acute Coronary Syndromes: Interpretation and Management. Mosby, 2011.

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20

Sutter, Johan De, Miguel Mendes, and Oscar H. Franco. Cardioprotective drugs. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656653.003.0019.

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Cardioprotective drugs are important in the treatment of patients at risk for or with documented cardiovascular disease. Beta-blockers are indicated after acute coronary syndromes, stable coronary artery disease, heart failure, and arrhythmias. Angiotensin-converting enzyme inhibitors (ACEi) are important in congestive heart failure, stable angina, post-acute myocardial infarction, and secondary prevention after any event or revascularization. Angiotensin receptor blockers are mainly alternative drugs for the same indications in case of intolerance to ACEi. Calcium channel blockers are first l
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21

Sutter, Johan De, Miguel Mendes, and Oscar H. Franco. Cardioprotective drugs. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199656653.003.0019_update_001.

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Cardioprotective drugs are important in the treatment of patients at risk for or with documented cardiovascular disease. Beta-blockers are indicated after acute coronary syndromes, stable coronary artery disease, heart failure, and arrhythmias. Angiotensin-converting enzyme inhibitors (ACEi) are important in congestive heart failure, stable angina, post-acute myocardial infarction, and secondary prevention after any event or revascularization. Angiotensin receptor blockers are mainly alternative drugs for the same indications in case of intolerance to ACEi. Calcium channel blockers are first l
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22

Stacey, Victoria. Cardiology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199592777.003.0009.

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Acute coronary syndrome (ACS) - Investigations in ACS - Management of ACS - Management of STEMI/new LBBB - Atrial fibrillation - Arrhythmias - Transient loss of consciousness - Sudden cardiac death (SCD) - Heart failure - Hypertensive emergencies - Infective endocarditis - Pericardial disease - SAQs
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23

Bowker, Lesley K., James D. Price, Ku Shah, and Sarah C. Smith. Cardiovascular. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738381.003.0010.

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This chapter provides information on the ageing cardiovascular system, chest pain, stable angina, acute coronary syndromes, myocardial infarction, hypertension, treatment of hypertension, presentation of arrhythmias, management of arrhythmias, atrial fibrillation, rate/rhythm control in atrial fibrillation, stroke prevention in atrial fibrillation, bradycardia and conduction disorders, common arrhythmias and conduction abnormalities, heart failure assessment, acute heart failure, chronic heart failure, dilemmas in heart failure, heart failure with preserved left ventricular function, valvular
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24

Valgimigli, Marco, and Marco Angelillis. Treatment of non-ST elevation acute coronary syndromes. Edited by Stefan James. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0311.

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Treatment of patients presenting with a non-ST elevation acute coronary syndrome (NSTE-ACS) aims at immediate relief of ischaemia and the prevention of serious adverse events, including death, myocardial (re)infarction, and life-threatening arrhythmias. In NSTE-ACS, patient management is guided by risk stratification (troponin, electrocardiogram, risk scores, etc.). Treatment options include anti-ischaemic and antithrombotic drugs and coronary revascularization including percutaneous coronary interventions, or coronary artery bypass grafting. While long-term secondary prevention with aspirin m
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25

Wellens, Hein J. J., and Eduardo Back Sternick. Variants of Ventricular Preexcitation: Recognition and Treatment. Wiley & Sons, Limited, John, 2007.

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26

Wellens, Hein J. J., and Eduardo Sternick. Variants of Ventricular Preexcitation: Recognition and Treatment. Blackwell Publishing Limited, 2006.

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27

Wellens, Hein J. J., and Eduardo Back Sternick. Variants of Ventricular Preexcitation: Recognition and Treatment. Wiley & Sons, Incorporated, John, 2008.

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28

Wellens, Hein J. J., and Eduardo Back Sternick. Variants of Ventricular Preexcitation: Recognition and Treatment. Wiley & Sons, Incorporated, John, 2008.

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29

Huszars Basic Dysrhythmias And Acute Coronary Syndromes Interpretation And Management. Elsevier Health Sciences, 2011.

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30

Kočka, Viktor, Steen Dalby Kristensen, William Wijns, Petr Toušek, and Petr Widimský. Percutaneous coronary interventions in acute coronary syndromes. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0047.

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Three different guidelines of the European Society of Cardiology cover the field of percutaneous coronary interventions. Their main recommendations are the following:All patients with an ST-segment elevation myocardial infarction should undergo immediate coronary angiography and percutaneous coronary intervention as soon as possible after the first medical contact. Thrombolysis can be used as an alternative reperfusion therapy if the time delay to primary percutaneous coronary intervention is more than 2 hoursPatients with very high-risk non-ST-segment elevation acute coronary syndromes (recur
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31

Kočka, Viktor, Steen Dalby Kristensen, William Wijns, Petr Toušek, and Petr Widimský. Percutaneous coronary interventions in acute coronary syndromes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0047_update_001.

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Three different guidelines of the European Society of Cardiology cover the field of percutaneous coronary interventions. Their main recommendations are the following:All patients with an ST-segment elevation myocardial infarction should undergo immediate coronary angiography and percutaneous coronary intervention as soon as possible after the first medical contact. Thrombolysis can be used as an alternative reperfusion therapy if the time delay to primary percutaneous coronary intervention is more than 2 hoursPatients with very high-risk non-ST-segment elevation acute coronary syndromes (recur
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32

Kočka, Viktor, Steen Dalby Kristensen, William Wijns, Petr Toušek, and Petr Widimský. Percutaneous coronary interventions in acute coronary syndromes. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0047_update_002.

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Three different guidelines of the European Society of Cardiology cover the field of percutaneous coronary interventions. Their main recommendations are the following: All patients with an ST-segment elevation myocardial infarction should undergo immediate coronary angiography and percutaneous coronary intervention as soon as possible after the first medical contact. Thrombolysis can be used as an alternative reperfusion therapy if the time delay to primary percutaneous coronary intervention is more than 2 hours. Patients with very high-risk non-ST-segment elevation acute coronary syndromes (re
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33

Kočka, Viktor, Steen Dalby Kristensen, William Wijns, Petr Toušek, and Petr Widimský. Percutaneous coronary interventions in acute coronary syndromes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0047_update_003.

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Three different guidelines of the European Society of Cardiology cover the field of percutaneous coronary interventions. Their main recommendations are the following: All patients with an ST-segment elevation myocardial infarction should undergo immediate coronary angiography and percutaneous coronary intervention as soon as possible after the first medical contact. Thrombolysis can be used as an alternative reperfusion therapy if the time delay to primary percutaneous coronary intervention is more than 2 hours. Patients with very high-risk non-ST-segment elevation acute coronary syndromes (re
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34

Macleod, Dr Donald C., Dr Ian Scott, Professor Calum Archibald Macrae, et al. Cardiac diseases and resuscitation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.0004.

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Chapter 4 discusses cardiac diseases and resuscitation, including symptoms, signs, and diagnostic investigations in cardiac disease, adult cardiopulmonary resuscitation, cardiovascular risk assessment, heart failure, acute coronary syndromes, arrhythmias, hypertension and hypertensive emergencies, thromboembolic disease, valvular disease, infective endocarditis, cardiomyopathies, congenital heart disease, heart disease in pregnancy, diseases of arteries and veins, rheumatic fever, pericarditis, ST segment elevation, and myocardial infarction.
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35

Kisiel, Maria, and Alison Smith. Cardiac surgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642663.003.0026.

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Coronary heart disease is caused by the build-up of atherosclerotic plaques which, over time, narrow the lumen of the coronary arteries. Acute coronary syndrome describes a spectrum of conditions caused by coronary artery disease; these are unstable angina, ST-elevation myocardial infarction (STEMI), and non-ST-elevation myocardial infarction (NSTEMI). Coronary artery disease is the leading cause for cardiac surgical interventions, but other causes are hypertension, valve disease, arrhythmias, cardiomyopathies, infections, and congenital abnormalities. This chapter provides an overview of the
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36

Pocket Guide To Huszars Basic Dysrhythmias And Acute Coronary Syndromes Interpretation And Management. Mosby, 2011.

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37

AlJaroudi, Wael. Risk Assessment Before Noncardiac Surgery. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0014.

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Perioperative risk assessment is essential in screening patients before noncardiac surgery. Cardiovascular complications such as fatal and non-fatal myocardial infarction (MI), ventricular arrhythmia, pulmonary edema, and stroke are important in-hospital causes of morbidity and mortality intra and post-operatively. The optimal approach is to identify patients at increased risk so that appropriate testing and therapeutic interventions are undertaken a priori to minimize such risk. The initial preoperative evaluation includes identification of surgery-specific risk, patient exercise functional c
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38

Allan, Walter, and Maryann Anglim. Kara Mia: The Story of Sudden Loss & Slow Recovery in a Teenager with Long QT Syndrome. Seahorse Pr, 1997.

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39

Evans, Charlotte, Anne Creaton, Marcus Kennedy, and Terry Martin, eds. Cardiac. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198722168.003.0009.

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Retrieval of patients with cardiac emergencies makes up a large chunk of the workload of most retrieval services. Cases range from the routine to the most challenging unstable patients with complex physiology and high-level support requirements. The transfer of patients for time-critical interventions mean that the adrenaline levels of the retrieval clinician may approach those of the patient. Included are clinical and logistical considerations for patients with acute coronary syndromes, pulmonary oedema, cardiogenic shock, arrhythmias, and those requiring pacing. Aortic dissection and pulmona
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40

Sever, Mehmet Şükrü, and Raymond Vanholder. Acute kidney injury in polytrauma and rhabdomyolysis. Edited by Norbert Lameire. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0252_update_001.

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The term ‘polytrauma’ refers to blunt (or crush) trauma that involves multiple body regions or cavities, and compromises physiology to potentially cause dysfunction of uninjured organs. Polytrauma frequently affects muscles resulting in rhabdomyolysis. In daily life, it mostly occurs after motor vehicle accidents, influencing a limited number of patients; after mass disasters, however, thousands of polytrauma victims may present at once with only surgical features or with additional medical complications (crush syndrome). Among the medical complications, acute kidney injury (AKI) deserves spec
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41

Cheong, Adrian, Gabriel Steg, and Stefan K. James. ST-segment elevation myocardial infarction. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0043.

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Acute myocardial infarction with ST-segment elevation is a common and dramatic manifestation of coronary artery disease. It is caused by the rupture of an atherosclerotic plaque in a coronary artery, leading to its total thrombotic occlusion and resultant ischaemia and necrosis of downstream myocardium. The diagnosis of ST-segment elevation myocardial infarction is based on a syndrome of ischaemic chest pain symptoms, associated with typical ST-segment elevation on the electrocardiogram and an eventual rise in biomarkers of myocardial necrosis. The treatment of ST-segment elevation myocardial
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42

Cheong, Adrian P., Gabriel Steg, and Stefan K. James. ST-segment elevation MI. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0043_update_001.

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Acute myocardial infarction with ST-segment elevation is a common and dramatic manifestation of coronary artery disease. It is caused by the rupture of an atherosclerotic plaque in a coronary artery, leading to its total thrombotic occlusion and resultant ischaemia and necrosis of downstream myocardium. The diagnosis of ST-segment elevation myocardial infarction is based on a syndrome of ischaemic chest pain symptoms, associated with typical ST-segment elevation on the electrocardiogram and an eventual rise in biomarkers of myocardial necrosis. The treatment of ST-segment elevation myocardial
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