Relevant bibliographies by topics / Bones – Growth

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Bones – Growth'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 February 2023

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Journal articles on the topic "Bones – Growth"

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Levine, Milton I. "Fragile Bones, Soft Bones, and Linear Growth Failure." Pediatric Annals 16, no. 12 (December 1, 1987): 943–45. http://dx.doi.org/10.3928/0090-4481-19871201-04.

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Scheven, Ben A. A., and Nicola J. Hamilton. "Longitudinal bone growth in vitro: effects of insulin-like growth factor I and growth hormone." Acta Endocrinologica 124, no. 5 (May 1991): 602–7. http://dx.doi.org/10.1530/acta.0.1240602.

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Abstract. Longitudinal growth was studied using an in vitro model system of intact rat long bones. Metatarsal bones from 18- and 19-day-old rat fetuses, entirely (18 days) or mainly (19 days) composed of chondrocytes, showed a steady rate of growth and radiolabelled thymidine incorporation for at least 7 days in serum-free media. Addition of recombinant human insulin-like growth factor-I to the culture media resulted in a direct stimulation of the longitudinal growth. Recombinant human growth hormone was also able to stimulate bone growth, although this was generally accomplished after a time
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Hagan, Michael. "Sticks and Bones." Arithmetic Teacher 33, no. 1 (September 1985): 44–45. http://dx.doi.org/10.5951/at.33.1.0044.

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The proliferation of both teacher-made and professionally prepared games might easily tempt a teacher to conclude that using them to kindle a child's interest in mathematics is an end in itself. And yet the full riches of many games may lie undiscovered until children are challenged to go beyond their interests, to think about their actions, and to elaborate on them. From this perspective, the most valuable games are those that interest children and that have rich variations to encourage their growth.
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Soliz, Mónica, María Jose Tulli, and Virginia Abdala. "Forelimb musculoskeletal-tendinous growth in frogs." PeerJ 8 (February 25, 2020): e8618. http://dx.doi.org/10.7717/peerj.8618.

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The tendons unite and transmit the strength of the muscles to the bones, allowing movement dexterity, the distribution of the strength of the limbs to the digits, and an improved muscle performance for a wide range of locomotor activities. Tissue differentiation and maturation of the structures involved in locomotion are completed during the juvenile stage; however, few studies have investigated the ontogenetic variation of the musculoskeletal-tendinous system. We ask whether all those integrated tissues and limb structures growth synchronically between them and along with body length. We exam
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DeLuca, H. F. "TRIENNIAL GROWTH SYMPOSIUM— Vitamin D: Bones and beyond12." Journal of Animal Science 92, no. 3 (March 1, 2014): 917–29. http://dx.doi.org/10.2527/jas.2013-7237.

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McKee, Malcolm. "Growth deformities of the long bones in dogs." In Practice 32, no. 7 (July 2010): 282–91. http://dx.doi.org/10.1136/inp.c3914.

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Deltoro, J., and Ana M. López. "Allometric growth patterns of limb bones in rabbits." Animal Production 46, no. 3 (June 1988): 461–67. http://dx.doi.org/10.1017/s0003356100019073.

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ABSTRACTRelative growth of length and thickness of femur, tibia, humerus and radius-ulna was studied using data from 320 rabbits from both sexes of two lines (New Zealand White and California). A cross-sectional design was used with slaughter points fixed at weekly intervals from 1 to 20 weeks of age. Carcass length was chosen as independent variable and two models were fitted to the data. Model 1 assumed the existence of one allometric change at some moment of the post-natal development and model 2 of a continuous change of the allometric coefficient throughout the experimental period.Most of
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Efthimiou, J., and P. J. Barnes. "Effect of inhaled corticosteroids on bones and growth." European Respiratory Journal 11, no. 5 (May 1, 1998): 1167–77. http://dx.doi.org/10.1183/09031936.98.11051167.

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Garzón-Alvarado, D. A., J. M. García-Aznar, and M. Doblaré. "A reaction–diffusion model for long bones growth." Biomechanics and Modeling in Mechanobiology 8, no. 5 (December 24, 2008): 381–95. http://dx.doi.org/10.1007/s10237-008-0144-z.

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Lok, F., J. A. Owens, L. Mundy, J. S. Robinson, and P. C. Owens. "Insulin-like growth factor I promotes growth selectively in fetal sheep in late gestation." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 270, no. 5 (May 1, 1996): R1148—R1155. http://dx.doi.org/10.1152/ajpregu.1996.270.5.r1148.

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Insulin-like growth factor I (IGF-I) is required for normal fetal growth and skeletal maturation in late gestation, because null mutations of the IGF-I gene in mice reduce fetal weight and retard ossification of bones. To determine if, conversely, increased abundance of IGF-I promotes fetal growth and skeletal maturation, fetal sheep were infused intravascularly with recombinant human IGF-I (n = 7) (26 +/- 3 micrograms. h-1.kg-1) from 120 to 130 days gestation and compared with controls (n = 15). IGF-I infusion increased plasma IGF-I concentrations by 140% (P = 0.002) and weights of fetal live
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Dissertations / Theses on the topic "Bones – Growth"

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Yadav, Priti. "Modelling loading and growth of long bones Modelling loading and growth of long bones." Licentiate thesis, KTH, Biomekanik, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-177913.

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The long bones grow by the process of endochondral ossification, which occurs at the growth plate. This process is regulated by biological factors and mechanical factors. The biological factors which contribute to endochondral ossification process are genes, hormones, nutrients etc. The mechanical factor is the load acting on the bone. The major forces on the bone are due to joint contact load and muscle forces, which induce stresses in the bone. Carter and Wong proposed in a theory that cyclic or intermittent octahedral shear stress promotes the bone growth and cyclic or intermittent hydrosta
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Chim, Shek Man. "Identification and characterization of novel secreted factors involved in bone remodeling." University of Western Australia. School of Surgery, 2009. http://theses.library.uwa.edu.au/adt-WU2010.0110.

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[Truncated abstract] Bone remodeling is an important process to maintain mechanical integrity. It is accomplished by two important steps, bone resorption followed by new bone formation. Osteoclasts and osteoblasts are the principal cells in bone resorption and bone formation, respectively. A multitude of local and systemic factors regulates this process by controlling the cellular activities in bone remodeling compartments (BRC). An imbalance of osteoblastic bone formation and osteoclastic bone destruction will result in the development of skeletal diseases. Recent studies suggested that angio
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Ma, Li, and 马丽. "The influence of nicotine on angiogenesis and osteogenesis in bone regeneration." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41508440.

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Oest, Megan Elizabeth. "Dual Osteogenic and Angiogenic Growth Factor Delivery as a Treatment for Segmental Bone Defects." Diss., Georgia Institute of Technology, 2007. http://hdl.handle.net/1853/16264.

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A new model of a critically-sized segmental femoral bone defect in rats was developed to enable in vivo imaging and facilitate post-mortem mechanical testing of samples. The critically-sized nature of the model was assessed and confirmed. The efficacy of sustained co-delivery of osteogenic (BMP-2 and TGF- Ò3) and angiogenic (VEGF) growth factors in promoting functional bone repair was assessed. Effects of scaffold modification in terms of geometry and composition were evaluated. The results indicated that co-delivery of BMP-2 and TGF- Ò3 resulted in a dose-dependent improvement in functional b
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關健明 and Kin-ming Kwan. "Defining the function of type X collagen in skeletal development." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1997. http://hub.hku.hk/bib/B31237162.

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Foster, Bruce Kristian. "Epiphyseal plate repair using fat interposition to reverse physeal deformity : an experimental study." Title page, contents and summary only, 1989. http://web4.library.adelaide.edu.au/theses/09MD/09mdf754.pdf.

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Bibliography: leaves 169-197. Hypothesises that the physis has an internal mechanism of repair to restore physeal function. Aims to establish a defined degree of deformity by partial growth plate excision, then to examine different methods of reversal of such deformity to observe the process of growth plate repair. A secondary aim was to define the percentage of physis that could be resected yet still enable reversal of deformity.
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Chayanupatkul, Atinooch. "Bone formation in the temporomandibular joint in response to forward mandibular positioning." Thesis, Hong Kong : University of Hong Kong, 2002. http://sunzi.lib.hku.hk/hkuto/record.jsp?B25598776.

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Cha, Ming Chuan 1955. "The effect of zinc deficiency on the growth promoting actions of growth hormone and insulin-like growth factor-I /." Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=55484.

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The effect of zinc deficiency on the growth promoting effect of circulating IGF-I and the direct growth effect of GH on long bone growth were investigated. Food intake was decreased by lack of zinc in the diet. Tissue zinc content and plasma alkaline phosphatase activity were reduced by zinc deficiency. Systemic administration of human IGF-I increased the body weight, tail length and tibia epiphyseal cartilage width of control animals. This somatogenic action was impaired by zinc deficiency, as evidenced by continued weight loss, no increase in tail length and decreased tibial epiphyseal carti
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Dai, Zhijie, and 戴志洁. "The role of sodium/myo-inositol cotransporter 1 and myo-inositol in osteogenesis and bone formation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43783533.

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Dahlgren, Emma. "Effects of Different Load Magnitudes on Longitudinal Growth of Immature Bones." Thesis, KTH, Skolan för kemi, bioteknologi och hälsa (CBH), 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-230885.

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In vivo studies of mechanical loading on bone have suggested that load magnitude is one of the parameters that play a vital role in bone adaptation. This study examined how longitudinal growth of immature rat metatarsals is affected by different load magnitudes. The main hypotheses were that the longitudinal growth of immature bone would decrease with increased compressive load magnitude, and that the longitudinal growth would be more decelerated the higher the load mag- nitude. The three middle metatarsal bones in the back paws of 19-20 days old Sprague-Dawley rat fetuses were extracted. Meta
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Books on the topic "Bones – Growth"

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J, Massaro Edward, and Rogers John M, eds. The skeleton: Biochemical, genetic, and molecular interactions in development and homeostasis. Totowa, N.J: Humana Press, 2004.

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M, Shapiro Irving, Boyan Barbara, and Anderson H. Clarke, eds. The growth plate. Amsterdam: IOS Press, 2002.

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1946-, Revell William J., ed. Blood supply of bone: Scientific aspects. London: Springer, 1998.

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Enlow, Donald H. Facial growth. 3rd ed. Philadelphia: Saunders, 1990.

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Bones and cartilage: Developmental and evolutionary skeletal biology. Australia: Elsevier Academic Press, 2005.

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The complete book of bone health. Amherst, N.Y: Prometheus Books, 2011.

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Mone, Zaidi, ed. Skeletal development and remodeling in health, disease, and aging. Boston, Mass: Blackwell Pub. on behalf of the New York Academy of Sciences, 2006.

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Lin, Yunfeng. Osteogenesis. Rijeka, Croatia: InTech, 2012.

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NATO Advanced Study Institute on Advances in Bone Regulatory Factors: Morphology, Biochemistry, Physiology, and Pharmacology (1989 Erice, Italy). Bone regulatory factors: Morphology, biochemistry, physiology, and pharmacology. New York: Plenum Press, 1990.

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Mone, Zaidi, New York Academy of Sciences, and Mount Sinai School of Medicine, eds. Skeletal biology and medicine. Boston, Mass: Blackwell Pub., on behalf of the New York Academy of Sciences, 2007.

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Book chapters on the topic "Bones – Growth"

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Uhthoff, H. K., and J. J. Robichon. "The Growth of Tubular Bones." In The Embryology of the Human Locomotor System, 15–24. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75310-7_3.

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Snell, E. J. "Growth of bones from chick embryos." In Applied Statistics, 56–60. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-011-6946-2_12.

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Snell, E. J., and H. R. Simpson. "Growth of bones from chick embryos." In Applied Statistics, 50–53. Boca Raton: Chapman and Hall/CRC, 2021. http://dx.doi.org/10.1201/9781003059844-11.

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Kleber, Bernd-Michael, and Karl Hecht. "Influence of Hypokinesia and Weightlessness on Jaw Bones and Teeth." In Humoral Factors in the Regulation of Tissue Growth, 244–66. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4613-9272-9_11.

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Karbowski, A., J. Herwig, H. H. Matthiaß, and E. Buddecke. "Effects of Mechanical Factors on Structure and Function of the Growth Plate of Long Bones." In Generalized Bone Diseases, 337–44. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-73346-8_31.

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Wilson, Helen, Diana Calcraft, Cai Neville, Susan Lanham-New, and Louise R. Durrant. "Bone Health, Fragility and Fractures." In Perspectives in Nursing Management and Care for Older Adults, 115–34. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-63892-4_9.

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AbstractAchieving and maintaining skeletal health throughout the life trajectory is essential for the prevention of bone diseases such as rickets, osteomalacia and osteoporosis. Rickets and osteomalacia are usually a result of calcium and/or vitamin D deficiency, causing softening of bones and bone pain, and both conditions are treatable with calcium and vitamin D supplementation. Osteoporosis is a multifaceted disease mainly affecting older people, and its pathogenesis (and hence treatment) is more complex. Untreated osteoporosis results in fragility fractures causing morbidity and increased mortality.Nutrition is one of many factors that influence bone mass and risk of bone disease. Developing a nutritional sciences approach is a feasible option for improving bone health.The importance of adequate calcium and vitamin D in ensuring skeletal integrity throughout the life course has a sound evidence base. Poor vitamin D status in population groups of all ages is widespread across many countries (including affluent and non-affluent areas). Public health approaches are required to correct this given the fact that vitamin D is not just required for musculoskeletal health but also for other health outcomes.Dietary protein may be beneficial for bone due to its effect of increasing insulin-like growth-factor-1 (IGF-1). Recent meta-analyses show that dietary protein has a beneficial role to play in bone health at all ages.Other nutritional factors and nutrients (such as potassium, magnesium, vitamin K and acid-base balance) are also likely to have an important role in bone health, though the literature is less clear in terms of the association/relationship and more research is required.
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Kim, Su-Jung, and Ki Beom Kim. "Craniofacial Morphology Related to Obstructive Sleep Apnea: Growth of Craniofacial Bones and the Upper Airway." In Management of Obstructive Sleep Apnea, 105–33. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-54146-0_9.

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Brookes, Murray, and William J. Revell. "Growth cartilages." In Blood Supply of Bone, 152–76. London: Springer London, 1998. http://dx.doi.org/10.1007/978-1-4471-1543-4_11.

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Bartl, Reiner, and Christoph Bartl. "Growth and Ageing of Bone." In Bone Disorders, 39–41. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-29182-6_5.

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Anderson, H. Clarke, and Irving M. Shapiro. "The Epiphyseal Growth Plate." In Bone and Development, 39–64. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84882-822-3_3.

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Conference papers on the topic "Bones – Growth"

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Askew, Michael J., Gary B. Schneider, Kristina J. Grecco, Jason Hsu, Emily Mugler, and Donald A. Noe. "Effect of Pharmaceutical Bone Growth Stimulation With Novel Anabolic Peptides: Biomechanical and Bone Density Measurements in a Rat Model." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43044.

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Pharmaceutical bone growth stimulation holds promise for prevention and treatment bone disorders, and the enhancement of fracture healing. Bone growth hormones have begun to have limited clinical use, but can illicit adverse side effects. Recent studies have shown that short peptides (less than 15 amino acids) derived from the protein sequence of Vitamin D Binding Protein (DBP), can enhance bone formation (osteogenesis). These peptides may have potential as controllable bone growth stimulators without the adverse side effects and cost of bone growth hormones. Rats, injected every other day for
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Vincitorio, F. M., N. Budini, C. Mulone, M. Spector, C. Freyre, A. J. López Díaz, and A. Ramil. "Detection of fungi colony growth on bones by dynamic speckle." In 8th Ibero American Optics Meeting/11th Latin American Meeting on Optics, Lasers, and Applications, edited by Manuel Filipe P. C. Martins Costa. SPIE, 2013. http://dx.doi.org/10.1117/12.2025550.

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Kłodowski, Adam, Antti Valkeapa¨a¨, and Aki Mikkola. "Craig-Bampton Modal Reduction Applied to Human Tibia Tradeoff Between Accuracy and Speed." In ASME 2011 International Mechanical Engineering Congress and Exposition. ASMEDC, 2011. http://dx.doi.org/10.1115/imece2011-63618.

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Human bones adapt to external loading through bone growth and resorption processes (1). Strains within specific range induced by physical loading can lead to strengthening of affected bones. On the other hand, when external forces are too high, it can lead to bone fracture (2) or cause significant loads in the joints, which in turn can be damaging for the cartilage surfaces (3) and ligaments. Optimization of the gym equipment as well as the techniques of exercising is necessary to achieve bone growth stimulation without overloading the bones or the joints. This issue has been recently addresse
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Hart, Stephen A., and Marcelo J. Dapino. "Accelerated Bone Growth Remotely Induced by Magnetic Fields and Smart Materials." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-175966.

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Bone structure is exquisitely matched to its physical loading environment. From the cartilaginous skeletal framework formed in the embryo to the aging skeleton, bone architecture is directly related to function. Bone is a dynamic system, constantly remodeling itself by absorbing old tissue and forming new tissue. This capability allows bone architecture to become optimized to the loading environment. Julius Wolff [1] first postulated that bone structure adapts to changing stress environments in 1892. Exact understanding of the process of mechanotransduction, however, has remained elusive. In a
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Lerner, Amy L., David L. Gushue, and Emily A. Gedbaw. "Mechanical Stress Patterns in the Human Proximal Tibial Growth Plate During the Stance Phase of Normal Gait." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-32609.

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Since the mid 1800’s clinicians and researchers have considered the effect of mechanical stresses on bone growth and the development of growth disorders [1]. However, the specifics of this relationship remain poorly understood. Both clinical and experimental evidence support the concept that magnitude, frequency and duration of loads are critical in defining the response of cartilage to pressures [2, 3]. Yet, the range of physiologic pressures in the growth plate has not been identified, and most models of growing bones have considered single quasi-static loading conditions and/or elastic mate
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Chou, Kathy, Grace Kim, and Marjolein C. H. van der Meulen. "The Effects of Vitamin D Deficiency on Histomorphometry and Strength of Rat Vertebrae." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53519.

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Vitamin D3 is integral to both bone remodeling and calcium homeostasis [1]. With vitamin D deficiency, rickets develops during growth and osteomalacia results in adulthood [2]; in both cases, mineralization is altered and bones are more prone to fracture. Although the degenerative effects of vitamin D deficiency on trabecular architecture have been studied, investigations examining both compromised tissue material properties and mechanical properties in the vertebrae of growing animals are scarce. Therefore, the objective of this study was to investigate cancellous bone architecture and mechan
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Yanoso, Laura, Justin Jacobson, Tulin Dadali, David Reynolds, and Hani Awad. "Evaluation of Polylactic Acid/Beta-Tricalcium Phosphate Scaffolds as Segmental Bone Graft Substitutes." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192978.

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The use of processed structural allografts for treatment of massive segmental defects in long bones can be complicated by poor incorporation and remodeling of the devitalized graft, foreign-body reaction and micro-damage accumulation which often leads to catastrophic graft failure [1]. It is therefore useful to develop a bioengineered, biodegradable scaffold that is able to stimulate healing of the defect region. The use of bioengineered scaffolds has been limited due to their poor mechanical strength that does not permit withstanding large in vivo loads and due to their poor osteoinductive pr
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Nowlan, Niamh C., Paula Murphy, and Patrick J. Prendergast. "Mechanical Stimuli Resulting From Embryonic Muscle Contractions Promote Avian Periosteal Bone Collar Formation." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-172077.

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Mechanical forces due to muscle contractions play an essential role in embryonic skeletal development. In neuromuscular conditions such as congenital myotonic dystrophy, where movement of the fetus in utero is reduced or absent, the bones and joints of the newborn often show malformations [1]. In this paper, we examine the effect of muscle contractions on embryonic bone development. We propose the hypothesis that mechanical loading due to muscle contractions promotes periosteal ossification and we test this hypothesis using computational and experimental methods. A set of FE analyses were perf
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Charoenphan, Saiphon, and Apiwon Polchai. "Finite Element Modeling for Energy Release Rate in Human Cortical Bone." In ASME 7th Biennial Conference on Engineering Systems Design and Analysis. ASMEDC, 2004. http://dx.doi.org/10.1115/esda2004-58307.

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The energy release rates in human cortical bone are investigated using a hybrid method of experimental and finite element modeling techniques. An explicit finite element analysis was implemented with an energy release rate calculation for evaluating this important fracture property of bones. Comparison of the critical value of the energy release rate, Gc, shows good agreement between the finite element models and analytical solutions. The Gc was found to be approximately 820–1150 J/m2 depending upon the samples. Specimen thickness appears to have little effect on the plane strain condition and
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Uddin, Sardar M. Zia, and Yi-Xian Qin. "Anabolic Effects of Ultrasound as Countermeasures of Simulated Microgravity in In-Vitro and In-Vivo Functional Disuse Models." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53796.

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Microgravity (MG) during space flight has been known to cause adverse effect on bone quality. Data collected from studies done on spaceflights show loss of 1–1.6% bone mineral density (BMD) per space-flight-month[1]. Most BMD has been recorded in load-bearing bones [2]. Some studies has considered using drugs and different growth factors to maintain bone mass in microgravity conditions but it can be too expensive to maintain over longer periods of time besides the systematic effects of such treatments [3]. Considering the effects of microgravity are partially attributed to lack of mechanical f
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Reports on the topic "Bones – Growth"

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Tao, Yang, Victor Alchanatis, and Yud-Ren Chen. X-ray and stereo imaging method for sensitive detection of bone fragments and hazardous materials in de-boned poultry fillets. United States Department of Agriculture, January 2006. http://dx.doi.org/10.32747/2006.7695872.bard.

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As Americans become increasingly health conscious, they have increased their consumptionof boneless white and skinless poultry meat. To the poultry industry, accurate detection of bonefragments and other hazards in de-boned poultry meat is important to ensure food quality andsafety for consumers. X-ray imaging is widely used for internal material inspection. However,traditional x-ray technology has limited success with high false-detection errors mainly becauseof its inability to consistently recognize bone fragments in meat of uneven thickness. Today’srapid grow-out practices yield chicken bo
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Gilsanz, Vicente. Bone Growth, Mechanical Stimulus and IGF-I. Fort Belvoir, VA: Defense Technical Information Center, October 2005. http://dx.doi.org/10.21236/ada443762.

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Gilsanz, Vicente. Bone Growth, Mechanical Stimulus and IGF-I. Fort Belvoir, VA: Defense Technical Information Center, October 2004. http://dx.doi.org/10.21236/ada429530.

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Gilsanz, Vicente. Bone Growth Mechanical Stimulus and IGF-I. Fort Belvoir, VA: Defense Technical Information Center, October 2003. http://dx.doi.org/10.21236/ada430393.

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Gilsanz, Vicente. Bone Growth, Mechanical Stimulus and IGF-I. Fort Belvoir, VA: Defense Technical Information Center, October 2002. http://dx.doi.org/10.21236/ada416003.

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Gilsanz, Vicente. Bone Growth, Mechanical Stimulus and IGF-I. Fort Belvoir, VA: Defense Technical Information Center, October 2007. http://dx.doi.org/10.21236/ada481973.

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Nickerson, Nicole. Transforming Growth Factor Beta Signaling in Growth of Estrogen-Insensitive Metastatic Bone Lesions. Fort Belvoir, VA: Defense Technical Information Center, January 2012. http://dx.doi.org/10.21236/ada558405.

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Sumrall, Jr, and George L. Enlistment and Reenlistment Bonus Debts: Can the Growth Be Curbed? Fort Belvoir, VA: Defense Technical Information Center, April 1989. http://dx.doi.org/10.21236/ada209070.

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Knudsen, Beatrice S. Hepatocyte Growth Factor and Interleukin-6 in Prostate Cancer Bone Metastasis. Fort Belvoir, VA: Defense Technical Information Center, March 2005. http://dx.doi.org/10.21236/ada435856.

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Knudsen, Beatrice S. Hepatocyte Growth Factor and Interleukin-6 in Prostate Cancer Bone Metastasis. Fort Belvoir, VA: Defense Technical Information Center, March 2004. http://dx.doi.org/10.21236/ada428439.

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