Academic literature on the topic 'Brother Of the Regulator of Imprinted Sites'

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Journal articles on the topic "Brother Of the Regulator of Imprinted Sites"

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Chen, Kefei, Wenqing Huang, Bin Huang, et al. "BORIS, Brother of the Regulator of Imprinted Sites, Is Aberrantly Expressed in Hepatocellular Carcinoma." Genetic Testing and Molecular Biomarkers 17, no. 2 (2013): 160–65. http://dx.doi.org/10.1089/gtmb.2012.0242.

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Zhang, Yanmei, Yongfei Song, Chao Li, et al. "Brother of regulator of imprinted sites inhibits cisplatin‑induced DNA damage in non‑small cell lung cancer." Oncology Letters 20, no. 5 (2020): 1. http://dx.doi.org/10.3892/ol.2020.12114.

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Zambrano-Galván, Graciela, Miguel Reyes-Romero, Ronell Bologna-Molina, Oscar Eduardo Almeda-Ojeda, and Obed Lemus-Rojero. "CTCFL(BORIS) mRNA Expression in a Peripheral Giant Cell Granuloma of the Oral Cavity." Case Reports in Dentistry 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/792615.

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Peripheral giant cell granuloma (PGCG) is a relatively common benign reactive lesion of the oral cavity which can occur at any age.CTCFL/BORIS(CTCFlike/Brother of the Regulator of Imprinted Sites) andCTCF(CCCTC-binding factor) are paralogous genes with an important role in the regulation of gene expression, genomic imprinting, and nuclear chromatin insulators regulation.BORISexpression promotes cell immortalization and growth whileCTCFhas tumor suppressor activity; the expression pattern may reflect the reverse transcription silencing ofBORIS. The aim of this work was to describe a histopathol
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Lee, Hae Young, Jong In Kim, Sung Ho Cho, et al. "Expression of the Brother of the Regulator of Imprinted Sites Gene in the Sputum of Patients with Lung Cancer." Korean Journal of Thoracic and Cardiovascular Surgery 47, no. 4 (2014): 378–83. http://dx.doi.org/10.5090/kjtcs.2014.47.4.378.

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Akhtar, Mohammad Salman, Naseem Akhter, Arshi Talat, et al. "Association of mutation and expression of the brother of the regulator of imprinted sites (BORIS) gene with breast cancer progression." Oncotarget 14, no. 1 (2023): 528–41. http://dx.doi.org/10.18632/oncotarget.28442.

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Sati, Leyla, Caroline Zeiss, Krishna Yekkala, Ramazan Demir та James McGrath. "Expression of theCTCFLGene during Mouse Embryogenesis Causes Growth Retardation, Postnatal Lethality, and Dysregulation of the Transforming Growth Factor β Pathway". Molecular and Cellular Biology 35, № 19 (2015): 3436–45. http://dx.doi.org/10.1128/mcb.00381-15.

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CTCFL, a paralog ofCTCF, also known asBORIS(brother of regulator of imprinted sites), is a testis-expressed gene whose function is largely unknown. Its product is a cancer testis antigen (CTA), and it is often expressed in tumor cells and also seen in two benign human vascular malformations, juvenile angiofibromas and infantile hemangiomas. To understand the function ofCtcfl, we created tetracycline-inducibleCtcfltransgenic mice. We show thatCtcflexpression during embryogenesis results in growth retardation, eye malformations, multiorgan pathologies, vascular defects, and neonatal death. This
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Singh, Smriti, Sathiya Pandi Narayanan, Kajal Biswas, et al. "Intragenic DNA methylation and BORIS-mediated cancer-specific splicing contribute to the Warburg effect." Proceedings of the National Academy of Sciences 114, no. 43 (2017): 11440–45. http://dx.doi.org/10.1073/pnas.1708447114.

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Aberrant alternative splicing and epigenetic changes are both associated with various cancers, but epigenetic regulation of alternative splicing in cancer is largely unknown. Here we report that the intragenic DNA methylation-mediated binding of Brother of Regulator of Imprinted Sites (BORIS) at the alternative exon of Pyruvate Kinase (PKM) is associated with cancer-specific splicing that promotes the Warburg effect and breast cancer progression. Interestingly, the inhibition of DNA methylation, BORIS depletion, or CRISPR/Cas9-mediated deletion of the BORIS binding site leads to a splicing swi
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El-Sharkawy, Nahla M., Wafaa M. Radwan, Enas S. Essa, et al. "Increased expression of brother of the regulator of imprinted sites in peripheral blood neutrophils is associated with both benign and malignant breast lesions." Cytometry Part B: Clinical Cytometry 92, no. 5 (2016): 355–60. http://dx.doi.org/10.1002/cyto.b.21378.

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Suzuki, Teruhiko, Natsuki Kosaka-Suzuki, Svetlana Pack, et al. "Expression of a Testis-Specific Form of Gal3st1 (CST), a Gene Essential for Spermatogenesis, Is Regulated by the CTCF Paralogous Gene BORIS." Molecular and Cellular Biology 30, no. 10 (2010): 2473–84. http://dx.doi.org/10.1128/mcb.01093-09.

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ABSTRACT Previously, it was shown that the CTCF paralogous gene, BORIS (brother of the regulator of imprinted sites) is expressed in male germ cells, but its function in spermatogenesis has not been defined. To develop an understanding of the functional activities of BORIS, we generated BORIS knockout (KO) mice. Mice homozygous for the null allele had a defect in spermatogenesis that resulted in small testes associated with increased cell death. The defect was evident as early as postnatal day 21 and was manifested by delayed production of haploid cells. By gene expression profiling, we found
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Ghochikyan, Anahit, Mikayel Mkrtichyan, Dmitri Loukinov, et al. "Elicitation of T Cell Responses to Histologically Unrelated Tumors by Immunization with the Novel Cancer-Testis Antigen, Brother of the Regulator of Imprinted Sites." Journal of Immunology 178, no. 1 (2006): 566–73. http://dx.doi.org/10.4049/jimmunol.178.1.566.

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Book chapters on the topic "Brother Of the Regulator of Imprinted Sites"

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"Brother of the Regulator of Imprinted Sites." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_100366.

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"Brother of the Regulator of Imprinted Sites." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_741.

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Conference papers on the topic "Brother Of the Regulator of Imprinted Sites"

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Song, Yongfei, Chao Li, Mengdie Fang, et al. "Abstract 1418: Brother of regulator of imprinted sites (BORIS) inhibits cisplatin induced DNA damage in non-small-cell lung cancer." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1418.

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Song, Yongfei, Chao Li, Mengdie Fang, et al. "Abstract 1418: Brother of regulator of imprinted sites (BORIS) inhibits cisplatin induced DNA damage in non-small-cell lung cancer." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1418.

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