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Relevant bibliographies by topics / Cardiovascular damage

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Academic literature on the topic 'Cardiovascular damage'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Cardiovascular damage"

1

Collinson, Paul O., and David C. Gaze. "Biomarkers of Cardiovascular Damage." Medical Principles and Practice 16, no. 4 (2007): 247–61. http://dx.doi.org/10.1159/000102146.

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Mayhew, Maren S. "Aspirin for Preventing Cardiovascular Damage." Journal for Nurse Practitioners 6, no. 2 (2010): 147–48. http://dx.doi.org/10.1016/j.nurpra.2009.12.004.

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Funder, John W. "Mineralocorticoid Receptors and Cardiovascular Damage." Hypertension 47, no. 4 (2006): 634–35. http://dx.doi.org/10.1161/01.hyp.0000203732.03784.3b.

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Shakariants, G. A. Shakariants, V. Yu Kaplunova Kaplunova, I. S. Chekneva Chekneva, E. V. Privalova Privalova, and Yu N. Belenkov Belenkov. "Postradiation Damage of the Cardiovascular System." Kardiologiia 12_2014 (December 17, 2014): 97–104. http://dx.doi.org/10.18565/cardio.2014.12.97-104.

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Krüger, Thilo, Stephan Oelenberg, Nadine Kaesler, et al. "Warfarin Induces Cardiovascular Damage in Mice." Arteriosclerosis, Thrombosis, and Vascular Biology 33, no. 11 (2013): 2618–24. http://dx.doi.org/10.1161/atvbaha.113.302244.

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van Schooten, Frederik J., Ad M. Knaapen, and Alberto Izzotti. "DNA damage, mutagenesis and cardiovascular disease." Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis 621, no. 1-2 (2007): 1–4. http://dx.doi.org/10.1016/j.mrfmmm.2007.02.006.

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Lee, S. "Oxidative DNA Damage and Cardiovascular Disease." Trends in Cardiovascular Medicine 11, no. 3-4 (2001): 148–55. http://dx.doi.org/10.1016/s1050-1738(01)00094-9.

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Shukla, Praphulla C., Krishna K. Singh, Bobby Yanagawa, Hwee Teoh, and Subodh Verma. "DNA damage repair and cardiovascular diseases." Canadian Journal of Cardiology 26 (March 2010): 13A—16A. http://dx.doi.org/10.1016/s0828-282x(10)71055-2.

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Richardson, Kelly, Gregory Engel, Takuya Yamazaki, Sung Chun, and Victor F. Froelicher. "Electrocardiographic damage scores and cardiovascular mortality." American Heart Journal 149, no. 3 (2005): 458–63. http://dx.doi.org/10.1016/j.ahj.2004.06.025.

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Richardson, K., G. Engel, and T. Yamazaki. "Electrocardiographic Damage Scores and Cardiovascular Mortality." ACC Current Journal Review 14, no. 12 (2005): 22. http://dx.doi.org/10.1016/j.accreview.2005.11.035.

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Dissertations / Theses on the topic "Cardiovascular damage"

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Stanyer, Lee. "Beta-amyloid/plasma lipoprotein interactions : implications for vascular damage." Thesis, University College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.270774.

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Hedberg, P. (Pirjo). "Advances in routine measurement of cardiac damage and cardiovascular risk markers." Doctoral thesis, University of Oulu, 2005. http://urn.fi/urn:isbn:9514276388.

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Abstract The development of commercially available assays from the measurement of enzyme activity to mass concentrations of proteins, especially the assays of cardiac troponin I and T, has been the most important innovation in the field of cardiovascular laboratory diagnostics over the decade. The availability of a simple, rapid test using whole blood to facilitate processing and to reduce the turnaround time could improve the management of patients presenting with chest pain. The aim of this study was to evaluate the analytical and clinical performance of a new time-resolved fluorometry-base

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Hall, Luke Christian. "The Role Of Thiocyanate In Modulating Tissue Damage In Cardiovascular Disease." Thesis, The University of Sydney, 2017. http://hdl.handle.net/2123/17043.

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Myeloperoxidase (MPO) is an enzyme released by activated neutrophils, monocytes, and some tissue macrophages as part of the inflammatory response at sites of inflammation. Thiocyanate anions (SCN-) are a major and preferred substrate for MPO, with oxidation giving rise to increased concentrations of hypothiocyanous acid (HOSCN) at the expense of hypochlorous acid (HOCl) which is generated from chloride ions. There is considerable evidence that these oxidants contribute to damage at sites of inflammation and contribute to the pathophysiology of cardiovascular diseases. HOSCN is much less reacti

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DiPeri, Timothy P. "Neuromodulation Therapy Mitigates Heart Failure Induced Hippocampal Damage." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/honors/208.

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Cardiovascular disease (CVD) is the leading cause of death in the United States. Nearly half of the people diagnosed with heart failure (HF) die within 5 years of diagnosis. Brain abnormalities secondary to CVD have been observed in many discrete regions, including the hippocampus. Nearly 25% of patients with CVD also have major depressive disorder (MDD), and hippocampal dysfunction is a characteristic of both diseases. In this study, the hippocampus and an area of the hippocampal formation, the dentate gyrus (DG), were studied in a canine model of HF. Using this canine HF model previously, we

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Rarick, Kevin Richard. "Cardiovascular end-organ damage in response to increased blood pressure variability : impact of oxidative stress." Diss., University of Iowa, 2012. https://ir.uiowa.edu/etd/3370.

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Baroreflex sensitivity (BRS) is often reduced in elderly populations and patients with chronic cardiovascular diseases leading to a concomitant rise in blood pressure variability (BPV) that is associated with increased cardiovascular related morbidity and mortality. Thus, there is a need to better understand the mechanisms by which BPV causes cardiovascular end-organ damage. Animal studies using sinoaortic denervation (SAD) to increase BPV have demonstrated pathologic changes in the structure of the heart and blood vessels; however, there is a paucity of data investigating changes in functiona

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Vranyac-Tramoundanas, Alexandra, and n/a. "Domoic acid-induced cardiac damage : an in vitro and in vivo investigation." University of Otago. Department of Pharmacology & Toxicology, 2007. http://adt.otago.ac.nz./public/adt-NZDU20071012.143651.

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Cardiovascular pathology is seen in both animals and humans after domoic acid intoxication. Whether this damage is direct (i.e., cardiotoxic) or indirect (i.e., CNS/autonomic seizures) is not known. We have previously shown that acute in vitro domoic acid (0.05-0.25[mu]M; 10 min) treatment of isolated cardiac mitochondria compromises mitochondrial FADH and NAD⁺-linked respiratory control and mitochondrial energetics. Domoic acid was shown to traverse and bind the cellular membrane of H9c2 cardiac myoblasts. However it did not compromise cellular viability as assessed using cell quantification

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Kuwabara, Yasuhide. "Increased MicroRNA-1 and MicroRNA-133a Levels in Serum of Patients With Cardiovascular Disease Indicate Myocardial Damage." Kyoto University, 2012. http://hdl.handle.net/2433/157428.

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Genoni, Giulia. "Cardiovascular dysfunction and metabolic impairment in pediatric obesity. Markers of damage and effects of a behavioral intervention." Doctoral thesis, Università del Piemonte Orientale, 2018. http://hdl.handle.net/11579/104065.

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GIULIETTI, FEDERICO. "Predictors of moderate and severe obstructive sleep apnea and association of nocturnal oximetry parameters with cardiovascular organ damage." Doctoral thesis, Università Politecnica delle Marche, 2022. https://hdl.handle.net/11566/295289.

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Introduzione: L’apnea ostruttiva nel sonno (OSA) colpisce quasi un miliardo di persone nel mondo, con prevalenza crescente. La maggior parte del rischio di morbilità e mortalità associato all'OSA è legato ad aumentato rischio di sviluppo e/o progressione di malattie cardiovascolari (CV). Il marker più usato per definire sia gravità di malattia che rischio di danno d'organo è l'indice-apnea-ipopnea (AHI). La ricerca di predittori di OSA è in continuo sviluppo. Lo scopo di questo studio è stato valutare i predittori di malattia di grado moderato e grave in un ampio campione non selezionato di pa

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Uys, Aletta Sophia. "Comparing autonomic and cardiovascular responses in African and Caucasian men : the SABPA study / Aletta Sophia Uys." Thesis, North-West University, 2012. http://hdl.handle.net/10394/9851.

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Motivation Hypertension is a pertinent health problem for urban black African men (hereafter referred to as African). Sympathetic hyperactivity and a dominant α-adrenergic response pattern have both been implicated as contributing factors to their poor cardiovascular health. In addition to the deleterious effect of neurogenic hypertension on target organs, sympathetic hyperactivity may promote the accelerated progression of left ventricular hypertrophy and structural vascular disease. Aim The overarching aim of this study is to scrutinize autonomic control of the cardiovascular system in a coh

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Books on the topic "Cardiovascular damage"

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H, Birkenhäger W., and Hansson Lennart 1940-, eds. Assessment of hypertensive organ damage. Elsevier, 1997.

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Mancia, Giuseppe, and Enrico Agabiti Rosei. Assessment of Preclinical Organ Damage in Hypertension. Springer, 2016.

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Mancia, Giuseppe, and Enrico Agabiti Rosei. Assessment of Preclinical Organ Damage in Hypertension. Springer, 2015.

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Passive Smoking and Cardiovascular Pathology: Mechanisms and Physiopathological Bases of Damage. Nova Science Publishers, 2007.

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Hansson and Birkenhä. Assessment of Hypertensive Organ Damage. Elsevier Science Pub Co, 1997.

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Bhopal, Raj S. Epidemic of Cardiovascular Disease and Diabetes. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198833246.001.0001.

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Coronary heart disease (CHD) and stroke, collectively cardiovascular disease (CVD), are caused by narrowing and blockage of the arteries supplying the heart and brain, respectively. In type 2 diabetes (DM<sub>2</sub>) insulin is insufficient to maintain normal blood glucose. South Asians have high susceptibility to these diseases. Drawing upon the scientific literature and discussions with 22 internationally recognized scholars, this book focuses on causal explanations and their implications for prevention and research. Genetically based hypotheses are considered together with the developmenta

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Air Pollution and Cardiovascular Diseases. Exon Publications, 2024. https://doi.org/10.36255/air-pollution-cardiovascular-diseases.

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Air Pollution and Cardiovascular Diseases is a comprehensive guide that explains the connection between air pollution and heart health. The article is organized into clear sections, each providing essential information on the causes, mechanisms, health impacts, and preventive measures associated with cardiovascular diseases resulting from exposure to polluted air. It begins by defining air pollution and identifying the major pollutants, such as particulate matter (PM2.5), nitrogen dioxide (NO₂), and ozone (O₃), that are most harmful to cardiovascular health. The article explains how these poll

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Giacca, Mauro, and Borja Ibáñez. Advanced therapies to treat cardiovascular diseases: controversies and perspectives. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0028.

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There is a pressing need to develop novel therapies for myocardial infarction and heart failure, two conditions that affect over 20% of the world population. Despite important advances in achieving revascularization of the ischaemic myocardium and the usefulness of devices in assisting failing hearts, therapy for these conditions remains poor. The final extent of myocardial tissue loss after infarction is a major determinant of post-infarction mortality due to heart failure. In this chapter we review the current strategies aimed at counteracting injury due to acute myocardial ischaemia–reperfu

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Vinod, Nikhra. COVID-19 and Long Covid: Organs Damage and Dysfunctions, and Implications for Clinical Course. Heighten Science Publications Inc., 2021. http://dx.doi.org/10.29328/ebook1005.

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Like any other infectious disease, the prognosis of COVID-19 is influenced by infecting agent, the SARS-CoV-2 virus load and the extent of organs affliction and damage. COVID-19 having a propensity for multiorgan involvement carries an adverse prognosis during the clinical course as well as later during the post-recovery period persisting as Long Covid. The direct cytopathic effects of SARS-CoV-2 virus and the erratic and hyper-inflammatory response lead to tissue injury in various organs coupled with physiological dysfunctions and complications. In fact, the multi-system manifestations of COV

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Vannuzzo, Diego, and Simona Giampaoli. Primary prevention: principles and practice. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656653.003.0007.

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Cardiovascular primary prevention is a coordinated set of actions at community and individual level aimed at eradicating, eliminating, or compressing at later ages the impact of cardiovascular diseases and their related disability. Its aim is healthy ageing. Cardiovascular epidemiology has elucidated the role of cardiovascular risk factors, forming the basis of strategies to reduce cardiovascular risk and subsequent disease. There is evidence that cardiovascular primary prevention works if three strategies are implemented together: a population strategy (particularly through a widespread adopt

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Book chapters on the topic "Cardiovascular damage"

1

Mancusi, Costantino, and Eva Gerdts. "Hypertensive Cardiovascular Organ Damage." In Manual of Cardiovascular Disease in Women. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-65952-2_8.

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Land, Walter Gottlieb. "Cerebro-Cardiovascular Diseases." In Damage-Associated Molecular Patterns in Human Diseases. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-53868-2_11.

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Bronte, Enrico, Giuseppina Novo, Viviana Bazan, Christian Rolfo, Stefania Gori, and Antonio Russo. "Cardiovascular Damage in Clinical Trials." In Cardiovascular Complications in Cancer Therapy. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-93402-0_20.

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Galvano, Antonio, Giuseppina Novo, Mario Roselli, Antonio Giordano, and Antonio Russo. "Cardiovascular Damage Induced by Radiotherapy." In Cardiovascular Complications in Cancer Therapy. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-93402-0_4.

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Licata, Giuseppe. "Early Markers of Cardiovascular Damage." In Recent Advances in Geriatrics. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4899-1483-5_14.

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López-Amor, M. Fraile. "Studies of myocardial damage and viability." In Developments in Cardiovascular Medicine. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1984-9_6.

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Baxter, Gary F. "Cellular Mechanisms of Ischaemic Myocardial Damage." In Developments in Cardiovascular Medicine. Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-017-2380-0_1.

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Hibst, R., T. Kolbe, S. Weinbrenner, and V. Hombach. "Excimer laser angioplasty: efficiency and damage." In Developments in Cardiovascular Medicine. Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3802-4_17.

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Grotyohann, L., and J. R. Neely. "Glycolytic Products and Ischemic Myocardial Damage." In Developments in Cardiovascular Medicine. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2621-2_20.

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Paneni, Francesco, and Francesco Cosentino. "Environment, Epigenetic Changes, and Cardiovascular Damage." In Diabetes and Cardiovascular Disease. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-17762-5_4.

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Conference papers on the topic "Cardiovascular damage"

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Hopkins, Caroline G., Peter E. McHugh, and J. Patrick McGarry. "Computer Modeling of Cardiovascular Stent Coating Damage." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192880.

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In this paper computational simulations of stent coating debonding are presented. Finite element methods are implemented to model coating delamination during stent crimping, deployment and recoil. Gold, titanium and polymer coatings of differing thicknesses are explicitly modeled. The interfacial relationship between the stent surface and the coating during crimping and deployment is simulated using a cohesive zone model.

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Setjiadi, Dellaneira, Christian Delles, Gemma Currie, and Carlos A. Celis-Morales. "4 Relationship between blood pressure level and hypertensive mediated organ damage." In Scottish Cardiovascular Forum – 26th annual meeting. BMJ Publishing Group Ltd and British Cardiovascular Society, 2023. http://dx.doi.org/10.1136/heartjnl-scf-2023.4.

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Rohde, Ingo, Jennifer-M. Masch, Dirk Theisen-Kunde, Martin Marczynski-Bühlow, Georg Lutter та Ralf Brinkmann. "Cardiovascular damage after cw and Q-switched 2μm laser irradiation". У European Conferences on Biomedical Optics, редактори Lothar D. Lilge та Ronald Sroka. SPIE, 2013. http://dx.doi.org/10.1117/12.2033550.

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Padjen, I., M. Erceg, M. Cerovec, M. Mayer, R. Stevanović, and B. Anić. "FRI0282 Cardiovascular damage in deceased patients with systemic lupus erythematosus." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.5197.

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"Roles of γδT Cells in Cardiovascular Damage Induced by Lupus Erythematosus". У 2018 3rd International Conference on Life Sciences, Medicine, and Health. Francis Academic Press, 2018. http://dx.doi.org/10.25236/iclsmh.18.029.

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Grigioni, Mauro, Giuseppe D'Avenio, Umberto Morbiducci, Costantino Del Gaudio, and Carla Daniele. "Fluid dynamics studies of cardiovascular medical devices and blood damage prediction." In 2008 30th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2008. http://dx.doi.org/10.1109/iembs.2008.4649432.

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Douglas, Graeham R., Tho Wei Tan, Tim Bond, and A. Srikantha Phani. "Geometry Governs Mechanics of Cardiovascular Stents." In ASME 2009 International Mechanical Engineering Congress and Exposition. ASMEDC, 2009. http://dx.doi.org/10.1115/imece2009-12924.

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Cardiovascular stents are tubular lattice structures implanted into a stenosed artery to provide adequate lumen support and promote circulation. Commonly encountered complications are stent migration, NeoIntimal Hyperplasia (NIH), and damage to the arterial wall. Central to all these problems is the mechanical response of a stent to forces operating in situ including stent-artery interaction. The influence of geometry or repetitive pattern of the stent upon its mechanical response is the subject of this study. We focus on damage to the arterial wall caused by the stent which can lead to eventu

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Lahm, H., M. Dreßen, N. Beck, S. Doppler, R. Lange, and M. Krane. "MYBPHL Plasma Levels Are Increased after Induced Atrial Damage." In 49th Annual Meeting of the German Society for Thoracic and Cardiovascular Surgery. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1705469.

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Dias, Renan Italo Rodrigues, Ada Rhalinne Dias Arruda Silva Araújo, José de Moura Sampaio Neto, et al. "Diseases caused by electronic cigarettes and their irreversible damage." In V Seven International Multidisciplinary Congress. Seven Congress, 2024. http://dx.doi.org/10.56238/sevenvmulti2024-107.

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The increase in the consumption of electronic cigarettes has raised concerns due to potential harm to health. This study addresses the diseases caused by the use of electronic cigarettes and their irreversible damage. We seek to identify the harmful health effects associated with this practice, providing crucial insights for more effective preventative interventions and health policies. The growing use of e-cigarettes as an alternative to traditional smoking raises questions about the impacts on lung and cardiovascular health. Although considered less harmful, electronic cigarettes present sig

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Geng, Li, and Jian-Mei Li. "BS56 Redox-regulation of AngII-induced cerebral microvascular damage in aging." In British Cardiovascular Society Annual Conference ‘Digital Health Revolution’ 3–5 June 2019. BMJ Publishing Group Ltd and British Cardiovascular Society, 2019. http://dx.doi.org/10.1136/heartjnl-2019-bcs.217.

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Reports on the topic "Cardiovascular damage"

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Johnson, Ginger. Viral haemorrhagic fevers question bank: Qualitative questions for understanding transmission dynamics and experiences of care. Institute of Development Studies, 2025. https://doi.org/10.19088/sshap.2024.066.

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Viral haemorrhagic fevers (VHFs) are a group of severe illnesses caused by viruses that affect multiple organ systems and damage the cardiovascular system. VHFs include Ebola virus disease and Marburg virus disease. There is wide variation in how VHFs are diagnosed, how pathogenic they are, their geographic distribution, their known reservoirs (i.e., animals or insects that spread the disease) and the availability of vaccines or treatments. When using this Question Bank for a specific public health emergency in connection with an identified VHF, consider these factors in addition to an updated

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