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Relevant bibliographies by topics / Cardiovascular failure / Dissertations / Theses

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Dissertations / Theses on the topic 'Cardiovascular failure'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

Khand, Aleem U. "Arterial fibrillation and heart failure." Thesis, University of Glasgow, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288911.

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2

Davies, Lewis Ceri. "Cardiovascular control mechanisms in chronic heart failure." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.397946.

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3

Loughrey, Clodagh Maria. "Lipoprotein oxidation in chronic renal failure." Thesis, Queen's University Belfast, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318949.

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4

Piepoli, Massimo F. "Cardiovascular and ventilatory responses to exercise in chronic heart failure." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.243516.

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5

Lygate, Craig Alexander. "Comparative studies in experimental hypertension and cardiac failure." Thesis, University of Glasgow, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312137.

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6

DeGrande, Sean Thomas. "Phosphatase regulation in cardiovascular physiology and disease." University of Iowa, 2012. http://ir.uiowa.edu/etd/3443.

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Reversible protein phosphorylation is an essential component of metazoan signaling and cardiovascular physiology. Protein kinase activity is required for regulation of cardiac ion channel and membrane receptor function, metabolism, and transcription, and aberrant kinase function is widely observed across disparate cardiac pathologies. In fact, multiple generations of cardiac therapies (eg. beta-adrenergic receptor blockers) have targeted cardiac kinase regulatory cascades. In contrast, essentially nothing is known regarding the mechanisms that regulate cardiac phosphatase activity at baseline

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7

Khadour, Fadi H. "Cardiovascular nitric oxide generation, changes with exercise training and heart failure." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0007/NQ39553.pdf.

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8

Muller, Andrew Frank. "Studies of central and regional haemodynamic parameters in chronic heart failure." Thesis, University of Nottingham, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.305090.

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9

Murchie, Karen J. (Karen Jo) 1973. "Arterial function in hypertension and heart failure." Monash University, Faculty of Medicine, 2000. http://arrow.monash.edu.au/hdl/1959.1/8884.

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10

Donohoe, Paul Thomas. "Cardiac ion currents in a rat model of renal failure." Thesis, King's College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324668.

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11

Andersen, Kasper. "Physical Activity and Cardiovascular Disease." Doctoral thesis, Uppsala universitet, Institutionen för medicinska vetenskaper, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-217309.

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The aim was to investigate associations of fitness and types and levels of physical activity with subsequent risk of cardiovascular disease. Four large-scale longitudinal cohort studies were used. The exposures were different measures related to physical activity and the outcomes were obtained through linkage to the Swedish In-Patient Register. In a cohort of 466 elderly men without pre-existing cardiovascular disease, we found that skeletal muscle morphology was associated with risk of cardiovascular events. A high amount of type I (slow-twitch, oxidative) skeletal muscle fibres was associate

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12

Hsiao, Lien-Cheng. "Cardiac stem cell therapy for heart failure." Thesis, University of Oxford, 2012. https://ora.ox.ac.uk/objects/uuid:c4fcb449-2d05-4dc6-9a8d-f7450c0b200c.

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Cardiovascular disease is a leading cause of death worldwide and becomes increasingly prevalent in the elderly population. Independent of etiopathogenesis, heart failure (HF) is the final common stage of numerous heart diseases. Cardiac stem cell (CSC) therapy has emerged as a promising cell-based strategy for treatment of HF. However, cell replacement is not able to fully restore a structurally damaged myocardium in advanced and end-stage HF. The objective of this project was to test the following hypotheses: that a bioengineered heart extracellular matrix (ECM) with preserved intact geometri

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13

Fortin, Amanda Jay. "Cardiovascular effects of L-arginine in a rat model of heart failure." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq28566.pdf.

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14

DiPeri, Timothy P. "Neuromodulation Therapy Mitigates Heart Failure Induced Hippocampal Damage." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/honors/208.

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Cardiovascular disease (CVD) is the leading cause of death in the United States. Nearly half of the people diagnosed with heart failure (HF) die within 5 years of diagnosis. Brain abnormalities secondary to CVD have been observed in many discrete regions, including the hippocampus. Nearly 25% of patients with CVD also have major depressive disorder (MDD), and hippocampal dysfunction is a characteristic of both diseases. In this study, the hippocampus and an area of the hippocampal formation, the dentate gyrus (DG), were studied in a canine model of HF. Using this canine HF model previously, we

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15

Venkatasubramanian, Sowmya. "Cardiovascular effects of the sirtuin and urocortin systems in humans." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/22919.

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Background: Cardiovascular disease continues to remain a leading cause of morbidity and mortality in both developing and developed worlds. The sirtuin and urocortin systems are novel hormone systems in humans with an emerging role in cardiovascular physiology and pathophysiology. Through a series of studies, this thesis examines the cardiovascular effects of SRT2104 (a novel small molecule SIRT1 activator) in otherwise healthy cigarette smokers and in patients with type 2 diabetes mellitus, and of urocortins 2 and 3 in healthy volunteers and in patients with heart failure. Methods: Twenty-four

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16

Deshpande, Gaurang. "Testing metabolic and pharmacological agents for recovery following de novo acute heart failure in an isolated rat heart model." Doctoral thesis, University of Cape Town, 2014. http://hdl.handle.net/11427/18610.

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Acute heart failure is a potentially lethal clinical emergency without any effective therapy. Using an isolated rat heart model, we established and validated a model of de novo acute heart failure to study novel putative cardio protective therapies against acute heart failure, including glucose, insulin and the molecular agent sphingosine-1-phosphate(component of high density lipoprotein) for recovery. In isolated rat hearts, the protocol consisted of three phases: stabilization at normotensive perfusion pressure, hypotensive acute heart failure and recovery by restoring normotension. Low gluc

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17

Maurer, Michaela Burri Sonja. "Plasma homocysteine and cardiovascular risk in heart failure with and without cardiorenal syndrome /." [S.l.] : [s.n.], 2009. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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18

Shan, Qixian, and 單綺嫻. "The role of nitric oxide and adrenomedullin in cardiovascular failure in septic shock." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31242261.

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19

Young, Tim. "Cardiovascular autonomic responses in pre- and post-ganglionic models of chronic autonomic failure." Thesis, Imperial College London, 2008. http://hdl.handle.net/10044/1/4718.

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Multiple System Atrophy (MSA) and Pure Autonomic Failure (PAF) are contrasting models of Chronic Autonomic failure. PAF primarily involves the post-ganglionic autonomic nervous system, whilst in MSA the pre-ganglionic structures are impaired. My central hypothesis is that this underlying neuropathological difference between MSA and PAF will lead to differing cardiovascular responses. I will assess the cardiovascular effects of known pressor and vasomotor stimuli (mental arithmetic, cold pressor test, isometric exercise, water ingestion, inhaled CO₂ and inspiratory gasp) in MSA and PAF. Neuroho

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20

Dalzell, Jonathan. "The novel peptide apelin and its putative role in cardiovascular regulation in health and heart failure." Thesis, University of Aberdeen, 2012. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=192161.

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Heart failure is associated with significant morbidity, mortality and economic cost. Over the past 30 years our understanding of the pathophysiology of heart failure has advanced greatly. However, morbidity and mortality remain high and further improvements in therapy are necessary. Pre-clinical data suggest that the novel peptide apelin, acting through the APJ receptor, has anti-hypertensive, vasodilator, diuretic and inotropic actions and an antagonistic relationship with angiotensin-II. These findings are of obvious interest in heart failure, particularly as plasma and myocardial apelin con

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21

Stewart, Simon. "Optimising therapeutic efficacy in acute and chronic cardiac disease states /." Title page, contents and abstract only, 1999. http://web4.library.adelaide.edu.au/theses/09PH/09phs851.pdf.

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22

Silva, Luma Nascimento. "Comparação de três instrumentos para avaliação da fadiga em pacientes com insuficiência cardíaca." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/22/22132/tde-26012017-103129/.

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Objetivos: Comparar as distribuições das medidas dos instrumentos DUFS, DEFS e Pictograma de Fadiga de acordo com a gravidade da insuficiência cardíaca (IC) avaliada pela Classe Funcional da New York Heart Association (CF-NYHA) e a avaliar sua relação com a fração de ejeção do ventrículo esquerdo (FEVE). Método: Estudo metodológico, de corte transversal, cuja amostra foi composta por adultos com IC atendidos em um hospital universitário do Estado de São Paulo, de setembro de 2014 a março de 2015. O DUFS avalia a fadiga relacionada à cardiopatia (8 itens, intervalo total de 8 a 40;

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23

Long, Victor P. III. "Modulation of the Arrhythmia Substrate in Cardiovascular Disease." The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1459777728.

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24

Gibbons, Stephen. "Insights into the cardiovascular complications of a novel mouse model of diabetes mellitus : a mechanistic view." Thesis, University of Manchester, 2011. https://www.research.manchester.ac.uk/portal/en/theses/insights-into-the-cardiovascular-complications-of-a-novel-mouse-model-of-diabetes-mellitus-a-mechanistic-view(d1c68010-8059-4371-a50f-be3614703471).html.

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Heart failure (HF) is one of the commonest complications of Diabetes Mellitus (DM) with the prevalence of DM reported at around 30% in many pivotal heart failure studies. However the pathophysiological mechanisms that contribute to HF development in diabetes are poorly understood. To investigate this we used a novel human relevant mouse model of DM (GENA348) in which there is a point mutation in the glucokinase (Gck) gene, the glucose sensor which regulates insulin secretion. A mutation in the same gene is known to underlie Maturity Onset Diabetes of the Young Type 2 (MODY 2) in humans. The mu

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25

Wells, Matthew A. "Peri-transplant cardiovascular dynamics in an ovine model of heart transplantation following 24-hrs donor brain stem death." Thesis, Griffith University, 2021. http://hdl.handle.net/10072/405633.

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Background: Heart transplantation (HTx) for end-stage heart failure is one of the most challenging complex health problems faced in intensive care units across the globe. This is partly due to the shortage of viable donor hearts and primary graft failure post-transplant. The mechanisms behind these shortfalls are diverse and arise from donor brain stem death (BSD), a significant stressor on the heart and the main pool from which donor hearts are sourced. A compromised myocardium is then preserved in cold preservation solution on ice (CSS), as per the current clinical standard, initiating signi

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26

Mahmod, Masliza. "Multiparametric cardiovascular magnetic resonance for the assessment of cardiac function and metabolism in hypertrophy and heart failure." Thesis, University of Oxford, 2013. http://ora.ox.ac.uk/objects/uuid:ff24c167-e00d-4c6d-9809-82203979ba7a.

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Both hypertrophied and failing hearts are characterised by pathological left ventricular (LV) remodelling, impaired myocardial energy status and alteration in substrate metabolism. Cardiac magnetic resonance imaging (CMR) and magnetic resonance spectroscopy (MRS) are powerful tools in the characterisation of these disease conditions. More recent techniques have allowed assessment of myocardial steatosis using <sup>1</sup>H-MRS and tissue oxygenation using blood oxygen level dependent (BOLD) CMR. In hypertrophy and heart failure, studies on steatosis and the relationship with other parameters s

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27

Wan, Ray Kay. "A study of natural killer cells in renal failure and in patients at cardiovascular risk." Thesis, University of Glasgow, 2012. http://theses.gla.ac.uk/3132/.

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Cardiovascular disease (CVD) is the leading cause of death in the UK, accounting for more than a third of all deaths, with atherosclerotic coronary artery disease (CAD) being the commonest type of CVD. Recently, it has become recognised that in addition to traditional cardiovascular (CV) risk factors such as dyslipidaemia, hypertension, smoking, and diabetes, inflammation plays an important role in the development and progression of atherosclerosis. A concept has emerged that atherosclerosis to some extent can be viewed as a chronic inflammatory autoimmune disease in which the adaptive immune

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28

Spalding, M. (Michael). "The hemodynamic effects of aminophylline, adenosine, losartan and nitric oxide administered shortly after right heart infarct in a porcine model." Doctoral thesis, University of Oulu, 2001. http://urn.fi/urn:isbn:9514264010.

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Abstract Right heart failure may be caused by several etiologic factors such as pulmonary embolism, post coronary bypass, chronic obstructive pulmonary disease (COPD) and right heart infarction. Traditionally, treatment has consisted of fluid loading (volume expansion) and the use of inotropic agents. In the present series of studies, an experimental model of acute right heart failure was developed using right heart infarct. Treatment with drugs chosen to specifically improve right heart performance was then evaluated. The drugs used in this series were aminophlline, adenosine, nitric o

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29

Ahlers, Belinda A. "Regulated L-Arginine transport in heart failure." Monash University, Faculty of Medicine, 2003. http://arrow.monash.edu.au/hdl/1959.1/9521.

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30

Khou, Victor. "Mortality and Kidney Failure: An Epidemiological Enquiry Using Linked Health Data." Thesis, The University of Sydney, 2021. https://hdl.handle.net/2123/26201.

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Patients with kidney failure (KF) experience high mortality and morbidity. Treatment options include kidney replacement therapy (KRT) with dialysis or kidney transplantation, or palliation. In 2018, cardiovascular disease and withdrawal from KRT were the most common causes of death in KF in Australia and New Zealand, each accounting for almost 1/3 of deaths. Effective healthcare delivery requires accurate mortality and morbidity data from clinical-quality registers. Data linkage of these registers with administrative datasets can provide insights into disease burden and optimal resource alloca

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31

Socas, Leonardo Jensen. "Ferro intracelular: fator modificável de susceptibilidade cardiovascular?" Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/5/5166/tde-10112015-092645/.

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Mutações no gene Hfe causam a forma mais comum da hemocromatose hereditária, doença caracterizada por acúmulo progressivo de ferro nos tecidos parenquimatosos. Um estudo prévio conduzido em nosso laboratório (Am J Cardiol 88(4):388-91, 2001) encontrou associação entre mutação do gene Hfe e cardiomiopatia isquêmica, sugerindo que o acúmulo de ferro no tecido cardíaco pode ser um fator que potencializa as agressões ao sistema cardiovascular. O objetivo do presente trabalho foi testar a hipótese de que o ferro aumenta a susceptibilidade ao risco cardiovascular. A análise de dados de 318 pacientes

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32

Armfield, Jennifer, and Jennifer Armfield. "Project BOOST and Cardiovascular Disease Readmissions in a Rural Acute Care Facility." Diss., The University of Arizona, 2016. http://hdl.handle.net/10150/622926.

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Hospital readmissions are a source of reduced payment as mandated by the Centers for Medicare and Medicaid Services as part of the Affordable Care Act (ACA). The number of dollars used for hospital readmissions has sky rocketed above $17 million for heart failure alone. The changes in the ACA reimbursement guidelines has put stress on many hospitals as they are facing reduced income, increased use of resources, and increased length of stay. This project evaluated the implementation of Project BOOST, its components, and their predictability for hospital readmission. Sample groups were evaluated

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33

Aguirre, Tejedo Alfons. "Identificación y prevalencia de factores precipitantes de insuficiencia cardiaca aguda en los servicios de urgencias españoles y su relación con el pronóstico a corto y medio plazo." Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/481958.

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Introducción. Hay pocos estudios que analicen el papel que juegan los factores precipitantes (FP) en el manejo de la insuficiencia cardiaca aguda (ICA). Los datos respecto a la epidemiología de los FP y su relación con el pronóstico proceden de registros de pacientes hospitalizados, los cuales no incluyen a los pacientes dados de alta desde los servicios de urgencias hospitalarios(SUH) Hipótesis y objetivos. La epidemiología de los FP de ICA difiere de la publicada en la literatura. Los FP pueden tener relación con el pronóstico a medio y corto plazo. El objetivo fue evaluar la identificac

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34

Fonseca, Maria Cândida Faustino Gamito da. "Insuficiência cardíaca. Uma epidemia do século XXI. O desafio do diagnóstico." Doctoral thesis, Faculdade de Ciências Médicas. Universidade Nova de Lisboa, 2008. http://hdl.handle.net/10362/5040.

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Resumo: A insuficiência cardíaca, já denominada de epidemia do século XXI é, de entre as doenças cardiovasculares, a única cuja incidência e prevalência continuam a crescer, apesar dos imensos progressos feitos na área da terapêutica nas últimas duas décadas. Caracteriza-se por elevada mortalidade – superior à do conjunto das neoplasias malignas -, grande morbilidade, consumo de recursos e custos exuberantes. É um dos problemas mais graves de Saúde Pública dos Países industrializados, cujo manejo deverá constituir uma prioridade para os Serviços Nacionais de Saúde. Todavia, o reconhecimento un

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35

Горобець, Д. О. "Експрес-діагностика функціонального стану серця". Thesis, Сумський державний університет, 2017. http://essuir.sumdu.edu.ua/handle/123456789/61268.

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Актуальність теми: більшість людей, які живуть у сьогоднішньому світі, важко віднести до повністю здорових. Велику кількість людей у цивілізованих країнах відносять до третього стану по Галену: «Третій стан - нездоров'я, не хвороба». Фізіологічна напруга механізмів адаптації є таким «третім станом».

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36

Ho, Chao-Chung. "EFFECTS OF ANTIOXIDANT ON CARDIOVASCULAR PERFORMANCE, EXERCISE CAPACITY, AND FUNCTIONAL STATUS IN PATIENTS WITH CHRONIC HEART FAILURE." Case Western Reserve University School of Graduate Studies / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=case1157980994.

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37

Lymbury, Robyn S. "Cardiovascular Disease, Antioxidant Enzyme Systems, and Selenium." Thesis, Griffith University, 2008. http://hdl.handle.net/10072/367925.

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Globally, cardiovascular disease (CVD) is the leading cause of mortality, representing 30 % of all deaths in 2005. The prevalence of CVD is predicted to increase steadily over the coming years and this has been attributed to a number of factors including an increase in overall life expectancy, as well as a projected increase in the number of people with common risk factors. As in most developed countries, Australia is experiencing a rise in the proportion of aged population, with current predictions estimating that by 2051, the number of people aged 65 years and over will increase from the cur

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38

Woltz, Patricia. "Health-related Quality of Life and Mortality in Incident Heart Failure from the Cardiovascular Health Study (CHS)." Thesis, University of Maryland, Baltimore, 2014. http://pqdtopen.proquest.com/#viewpdf?dispub=3621931.

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<p> Heart failure (HF) is a debilitating clinical syndrome and the end stage of many heart diseases. Despite recent advances in treatment, it is a growing problem with a steadily increasing number of deaths annually. Costs of care have been rising and are projected to continue to do so over the next 20 years. Studies report that health related quality of life (HRQL) in HF adds prognostic information beyond that of traditional demographic and clinical risk factors, however findings vary. This research was conducted on data from The Cardiovascular Health Study (CHS), a large, population based co

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39

De, Mauri Andreana. "Lipoprotein-associated phospholipase A2 as cardiovascular risk factor and therapeutic target in nephropatic patients." Doctoral thesis, Università del Piemonte Orientale, 2020. http://hdl.handle.net/11579/115669.

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Lipoprotein-associated phospholipase A2 (Lp-PLA2) is a serine lipase associated with Low-Density Lipoproteins, that increases the oxidative stress in atherosclerotic lesions and makes the plaque instable. Lp-PLA2 is recognized as a risk factor for cardiovascular and peripheral artery disease in general population, in cardiac and dysmetabolic patients, but no data are available for subjects with renal failure. The aim of the present PhD thesis was to investigate the role of Lp-PLA2 among patients with advanced renal failure. Chapter 1. In 102 hemodialyzed patients, it has been demonstrated that

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40

Galiyeva, Dinara. "Cardiovascular risk factor prevalence, mortality and cardiovascular disease incidence in patients who initiated renal replacement therapy in childhood : systematic review and analyses of two renal registries." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/28837.

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Background. The incidence of starting renal replacement therapy (RRT) among young people (< 20 years of age) in 2013 in Scotland was 7.7 per million (age-related) population. Little knowledge exists about cardiovascular risk factors (CVRFs), long-term survival and cardiovascular disease (CVD) outcomes in patients who initiated RRT in childhood. The main source of routine data for these patients is available from the European Society of Paediatric Nephrology/European Renal Association- European Dialysis and Transplant Association (ESPN/ERA-EDTA) registry. In Scotland nationally comprehensive da

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41

Taniguchi, Fábio Papa [UNESP]. "Insuficiência renal aguda pela hemodiluição acentuada na extracopórea." Universidade Estadual Paulista (UNESP), 2006. http://hdl.handle.net/11449/99914.

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Made available in DSpace on 2014-06-11T19:30:24Z (GMT). No. of bitstreams: 0 Previous issue date: 2006-12-12Bitstream added on 2014-06-13T21:00:59Z : No. of bitstreams: 1 taniguchi_fp_dr_botfm.pdf: 574800 bytes, checksum: 10af142c1ac5d57d2fa2fe16744a2f5b (MD5)<br>A insuficiência renal aguda (IRA) no pós-operatório de cirurgia cardiovascular é causa de maior morbidade e mortalidade. A disfunção renal caracterizada pelo aumento da creatinina sérica deternina maior número de complicações operatórias e diminuição da sobrevida. Fatores de risco genéticos e no pré-operatório foram determinados, co

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42

Wang, Yee-moon Angela. "Utility of cardiac biomarkers in end-stage renal disease patients on maintenance peritoneal dialysis." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B41508968.

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43

Fenning, Andrew S. "Cardiac remodelling in rat models of chronic cardiovascular disease : angiotensin-converting enzyme inhibition in heart failure and diabetes /." [St. Lucia, Qld], 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe18264.pdf.

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44

Suliman, Mohamed Elsaeid M. "Sulfur amino acids and their metabolites in patients with renal failure : relation to nutritional status and cardiovascular disease /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4604-3/.

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45

Tan, Eng Shiong. "Advanced clinical approaches in complex cardiovascular disease invasive options at the crossroad of ischemia, heart failure, and arrhythmias /." [S.l. : [Groningen : s.n.] ; University Library Groningen] [Host], 2008. http://irs.ub.rug.nl/ppn/.

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46

Barnes, Gareth David. "Cardiovascular actions of apelin-receptor agonism during Renin-Angiotensin system activation, exercise and in patients with chronic stable heart failure." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/25415.

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The apelin-apelin receptor (APLNR) system is an important regulator of cardiovascular homeostasis both in health and disease. Principal actions of the apelin-APLNR system are positive inotropism, vasodilatation, diuresis and a potential anti-inflammatory role in vascular tissue. The significance of this system is highlighted in heart failure and pulmonary hypertension. Preclinical models of these diseases report downregulation of apelin- APLNR, whilst knockout strains develop more severe phenotypes, more rapidly. Moreover treatment with exogenous apelin retards or prevents disease progression.

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47

Rosell, Johan. "Long-term effects of adjuvant tamoxifen treatment on cardiovascular disease and cancer." Doctoral thesis, Linköpings universitet, Avdelningen för kliniska vetenskaper, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-112085.

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The aims of this thesis were to investigate the long-term effects of adjuvant tamoxifen treatment on breast cancer recurrence and mortality, cardiovascular disease, and the incidence of secondary cancer. Between 1982 and 1992, postmenopausal patients with early stage breast cancer were included in a randomized clinical study of 2 or 5 years of postoperative tamoxifen therapy. The trial was planned by the Swedish Breast Cancer Group, and it included 4610 patients. Follow-up on causes of death, hospitalizations and secondary cancers were obtained from national population-based registries. All-c

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48

Jewett, Benjamin E. "Inverse Changes in Ghrelin and A2A Receptor Gene Expression Levels in the Hippocampus of Heart Failure Canines Following Spinal Cord Stimulation." Digital Commons @ East Tennessee State University, 2015. https://dc.etsu.edu/honors/262.

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Myocardial infarction (MI), often referred to as a heart attack, is a serious health issue in the United States. There is a well-documented link between MI and major depressive disorder (MDD), with a high incidence of MDD occurring after an MI. Overlapping pathologies have been observed within the hippocampus of the brain in animal models of MI and depression. These observations suggest that pathobiological cross-talk between the heart and brain could have a role in the etiology of MDD that occurs after an MI. Spinal cord stimulation (SCS) has previously been shown to have both cardioprotectiv

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Clark, R. A. "Chronic Heart Failure Beyond City Limits: An Analysis of the Distribution, Management and Information Technology Solutions for People with Chronic Heart Failure in Rural and Remote Australia." Thesis, University of South Australia, 2007.

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50

Phoku, Nkosinathi Terrence. "Cardiovascular risk factors among 15-20 years old rural subjects residing in Dikgale Demographic Surveillance Site (DDSS), Limpopo Province." Thesis, University of Limpopo (Turfloop Campus), 2013. http://hdl.handle.net/10386/1048.

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Thesis (M.Sc. (Chemical Pathology) --University of Limpopo, 2013<br>Cardiovascular diseases (CVDs) are among one of the well documented conditions and pose a significant health burden in the world as they areconsidered to be of adult onset. However, recent studies have shown that in developed countries CVD risk factors are becoming prevalent in young people which isof great concern. Therefore, the aim of this study was to determine if CVD risk factors are present in young subjects aged 15-20 years of age residing in a rural area of a developing country. Methods: Subjects aged between 15-20 ye

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