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1

Miranda, Nayara Lima de, Fabiano Pinheiro Ribeiro Junior, Ana Carolina Leal Corrêa Lima, Vivian Lee Neves Borges, and Nathane Barbosa Xavier. "CARDIOVASCULAR MANIFESTATIONS OF CHRONIC RENAL FAILURE." Revista Ibero-Americana de Humanidades, Ciências e Educação 9, no. 12 (2024): 273–83. http://dx.doi.org/10.51891/rease.v9i12.12754.

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A insuficiência renal crônica (IRC) é uma condição clínica prevalente, associada a uma série de manifestações sistêmicas que vão além do comprometimento renal. Dentre essas, as manifestações cardiovasculares desempenham um papel significativo, contribuindo para a morbidade e mortalidade dos pacientes. A interligação entre a função renal comprometida e as complicações cardiovasculares é complexa e multifatorial, envolvendo alterações hemodinâmicas, disfunções metabólicas e ativação de vias inflamatórias. Compreender a extensão e os mecanismos por trás das manifestações cardiovasculares na IRC é
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Ortiz-León, Gabriela, Heiner José Barrantes-Vargas, Manuel Arguedas-Sandí, Jose Ángel Pacheco-Chaverri, and Marta Vílchez-Monge. "An Approximation of Heart Failure Using Cardiovascular Simulation Toolbox." Biomimetics 4, no. 3 (2019): 47. http://dx.doi.org/10.3390/biomimetics4030047.

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In this paper, we present the simulation of 5 different heart failures with the help of the Cardiovascular Simulation Toolbox (CVST) proposed by O. Barnea et al. at Tel-Aviv University. This is a modified version of the CVST, proposed by G.Ortiz; here, we show that the pathological failures can be covered by this tool. We varied the value of the tool blocks, included the results of the hemodynamic parameters and the P-V loop curves for each disease and compared them to the medical data to prove the effectiveness of the simulation. Based on these changes, we achieved an effective simulation of
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3

Díaz Navarro, Rienzi. "Heart failure: role of cardiovascular imaging." Medwave 16, Suppl4 (2016): e6552-e6552. http://dx.doi.org/10.5867/medwave.2016.6552.

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4

Jang, David H., Meghan B. Spyres, Lindsay Fox, and Alex F. Manini. "Toxin-Induced Cardiovascular Failure." Emergency Medicine Clinics of North America 32, no. 1 (2014): 79–102. http://dx.doi.org/10.1016/j.emc.2013.10.003.

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5

Vignon, Philippe. "Cardiovascular failure and weaning." Annals of Translational Medicine 6, no. 18 (2018): 354. http://dx.doi.org/10.21037/atm.2018.05.46.

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6

Nazarko, Linda. "Cardiovascular problems: heart failure." Nursing and Residential Care 10, no. 5 (2008): 233–36. http://dx.doi.org/10.12968/nrec.2008.10.5.29142.

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7

Macdonald, Peter S., and Michael F. O'Rourke. "Cardiovascular ageing and heart failure." Medical Journal of Australia 169, no. 9 (1998): 480–84. http://dx.doi.org/10.5694/j.1326-5377.1998.tb123374.x.

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8

Rostand, S. G., J. D. Brunzell, R. O. Cannon, and R. G. Victor. "Cardiovascular complications in renal failure." Journal of the American Society of Nephrology 2, no. 6 (1991): 1053–62. http://dx.doi.org/10.1681/asn.v261053.

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Cardiovascular diseases are a leading cause of death in end-stage renal disease (ESRD) largely as a result of the progressively increasing age of ESRD patients and the broad constellation of uremia-associated factors that can adversely affect cardiac function. Hypertension, one of the leading causes of renal failure, is a major culprit in this process, causing left ventricular hypertrophy, cardiac chamber dilation, increased left ventricular wall stress, redistribution of coronary blood flow, reduced coronary artery vasodilator reserve, ischemia, myocardial fibrosis, heart failure, and arrhyth
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9

Taylor, S. H. "Cardiovascular consequences of heart failure." European Heart Journal 9, suppl H (1988): 41–47. http://dx.doi.org/10.1093/eurheartj/9.suppl_h.41.

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10

Patel, Ankitkumar, and Steven Hollenberg. "Cardiovascular Failure and Cardiogenic Shock." Seminars in Respiratory and Critical Care Medicine 32, no. 05 (2011): 598–606. http://dx.doi.org/10.1055/s-0031-1287869.

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11

Mann, J. "Cardiovascular risk and renal failure." DMW - Deutsche Medizinische Wochenschrift 129, no. 46 (2004): 2479–81. http://dx.doi.org/10.1055/s-2004-835290.

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12

Tyralla, Karin, and Kerstin Amann. "Cardiovascular Changes in Renal Failure." Blood Purification 20, no. 5 (2002): 462–65. http://dx.doi.org/10.1159/000063551.

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13

Oxenham, Helen, and Norman Sharpe. "Cardiovascular aging and heart failure." European Journal of Heart Failure 5, no. 4 (2003): 427–34. http://dx.doi.org/10.1016/s1388-9842(03)00011-4.

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14

Triposkiadis, Filippos, Andrew Xanthopoulos, and Javed Butler. "Cardiovascular Aging and Heart Failure." Journal of the American College of Cardiology 74, no. 6 (2019): 804–13. http://dx.doi.org/10.1016/j.jacc.2019.06.053.

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15

Benham-Hermetz, Julia, Mark Lambert, and Robert CM Stephens. "Cardiovascular failure, inotropes and vasopressors." British Journal of Hospital Medicine 73, Sup5 (2012): C74—C77. http://dx.doi.org/10.12968/hmed.2012.73.sup5.c74.

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16

Richard, Christian, and Jean Louis Teboul. "Weaning failure from cardiovascular origin." Intensive Care Medicine 31, no. 12 (2005): 1605–7. http://dx.doi.org/10.1007/s00134-005-2698-x.

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17

Jardin, François, and Antoine Vieillard-Baron. "Weaning failure from cardiovascular origin." Intensive Care Medicine 32, no. 6 (2006): 937. http://dx.doi.org/10.1007/s00134-006-0145-2.

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18

Baldewijns, Karolien, Hans-Peter Brunner-La Rocca, Lieven de Maesschalck, Aleidis Devillé, and Josiane Boyne. "Unravelling heart failure nurses’ education: Content comparison of heart failure nurses’ education in three European Society of Cardiology states and the Heart Failure Association heart failure curriculum." European Journal of Cardiovascular Nursing 18, no. 8 (2019): 711–19. http://dx.doi.org/10.1177/1474515119863179.

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Aims: The European Society of Cardiology (ESC) guidelines state that heart failure nurse specialists (heart-failure nurses) with specific competences are essential for a successful heart-failure-management programme. Thus, the Heart Failure Association (HFA) of the ESC developed the heart failure nurse curriculum (HFA curriculum). Several ESC member states developed cardiovascular education programmes to enable nurses to deliver high specialist care, but little is known of whether these curricula are in line with the HFA curriculum. Therefore, this paper describes the extent to which cardiovas
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19

Madazimova, D. H., O. O. Zhabborov, N.T. Buvamuhammedova, and Ahudja Anand. "COVID-19 AND HEART FAILURE." Eurasian Journal of Academic Research 1, no. 8 (2021): 513–15. https://doi.org/10.5281/zenodo.5759783.

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Infectious disease caused by the SARS-CoV-2 virus (COVID-19) often occurs in patients with various cardiovascular risk factors and cardiovascular diseases (CVD), which can affect the course of the infectious process. On the other hand, with COVID-19, additional damage to the heart and blood vessels is possible, contributing to the occurrence of cardiovascular complications and aggravation of the course of CVD. This review examines the main facts concerning these interactions, as well as some practical approaches to the management of patients with the simultaneous presence of COVID-19 and cardi
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20

Husain, MA, A. AlQahtani, and Muneera AlTaweel. "The Conundrum of Obesity and Heart Failure." Series of Endocrinology, Diabetes and Metabolism 5, no. 2 (2023): 40–55. http://dx.doi.org/10.54178/jsedmv5i2002.

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Obesity is a significant global health concern; the prevalence continues to rise in developing and developed countries. According to World Health Organization (WHO) data, 39% of the world population over the age of 18 is overweight, including 13% being obese. Numerous studies have identified an association between obesity and cardiovascular diseases (CVD), including heart failure (HF). Obesity is additionally related to an increase in the occurrence of cardiovascular (CV) disorders, including hypertension (HTN), diabetes mellitus (DM), dyslipidemias, and sleep apnea syndrome. This review inves
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21

Nava, S., and F. Bellemare. "Cardiovascular failure and apnea in shock." Journal of Applied Physiology 66, no. 1 (1989): 184–89. http://dx.doi.org/10.1152/jappl.1989.66.1.184.

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A model of shock was developed in anesthetized dogs by limiting venous return with a balloon inflated in the right atrium. The change in ventilation (VE) in response to a sustained decrease in arterial pressure (Pa) to 50–60 Torr was studied by recording transdiaphragmatic pressure (Pdi) and diaphragm (Edi) and parasternal intercostal (Eic) electrical activity. Four dogs died of cardiac arrest after 20–60 min. In 11 dogs, VE, after an initial increase, decreased progressively until apnea occurred after 103 +/- 24 min, after 60% reductions in breathing frequency, Pdi, and Eic and a 30% fall in
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22

Zarco, Pedro. "Cardiovascular failure. Pathophysiological bases and management." Revista Española de Cardiología 56, no. 4 (2003): 426–27. http://dx.doi.org/10.1016/s0300-8932(03)76890-0.

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23

Karamitsos, Theodoros D., and Stefan Neubauer. "Cardiovascular Magnetic Resonance in Heart Failure." Current Cardiology Reports 13, no. 3 (2011): 210–19. http://dx.doi.org/10.1007/s11886-011-0177-2.

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24

Raman, Subha V. "Cardiovascular Magnetic Resonance in Heart Failure." Heart Failure Clinics 17, no. 1 (2021): xiii—xiv. http://dx.doi.org/10.1016/j.hfc.2020.10.001.

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25

I Bon, Elizaveta. "Pathologies of the Cardiovascular System." Clinical Cardiology and Cardiovascular Interventions 8, no. 3 (2025): 01–09. https://doi.org/10.31579/2641-0419/450.

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Purpose of the work: to study in detail the pathologies of the cardiovascular system in children and adults Heart failure is a syndrome of symptoms and signs caused by cardiac dysfunction, resulting in reduced life expectancy. To establish the diagnosis of heart failure, the guidelines of the European Society of Cardiology require the presence of symptoms and signs, objective evidence of cardiac dysfunction (preferably by echocardiography) and, in case of remaining doubt, a favourable response to treatment directed at heart failure. Numerous compensatory mechanisms are involved in supporting t
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26

de, Boer Rudolf A., and Johann Bauersachs. "The year in cardiovascular medicine 2022: the top 10 papers in heart failure and cardiomyopathies." Interventional Cardiology Forum 3 (November 2, 2023): 43–48. https://doi.org/10.3897/icf.3.e113433.

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27

Holland, Lee. "Cardiovascular medicines." British Homeopathic Journal 83, no. 04 (1994): 223–29. http://dx.doi.org/10.1016/s0007-0785(05)80797-5.

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AbstractA review of the main homoeopathic medicines whose centre of action includes the heart with a summary of their main characteristics. Special attention has been paid to Digitalis, Lilium tigrinum, Naja, Cactus, Spigelia, Kalmia latifolia and several others, including medicines commonly used in mother tincture form for heart failure and hypertension.
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28

Tøndel, Camilla, and Hans-Peter Marti. "Cardiovascular changes in young renal failure patients." Clinical Kidney Journal 15, no. 2 (2021): 183–85. http://dx.doi.org/10.1093/ckj/sfab223.

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ABSTRACT Progresses in medical care of severe kidney disease and congenital anomalies of kidney and urinary tract make it possible for a higher percentage of young renal failure patients to survive and enter adulthood. There is thus an increasing need to focus on the long-term effects of severely reduced kidney function early in life. Cardiovascular changes are known to contribute considerably in adulthood to the severe complications of renal failure. In young chronic kidney disease patients, there is limited knowledge of subclinical cardiovascular disease. In this issue of Clinical Kidney Jou
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29

Kiuchi, Shunsuke, and Takanori Ikeda. "Management of hypertension associated with cardiovascular failure." Journal of Cardiology 79, no. 6 (2022): 698–702. http://dx.doi.org/10.1016/j.jjcc.2021.11.012.

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30

Ketteler, Markus, Marie-Luise Gross, and Eberhard Ritz. "Calcification and cardiovascular problems in renal failure." Kidney International 67 (April 2005): S120—S127. http://dx.doi.org/10.1111/j.1523-1755.2005.09428.x.

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31

Fort, Joan. "Chronic renal failure: A cardiovascular risk factor." Kidney International 68 (December 2005): S25—S29. http://dx.doi.org/10.1111/j.1523-1755.2005.09906.x.

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32

Faconti, Luca, Philip J. Chowienczyk, and Ajay M. Shah. "Cardiovascular disease, heart failure and COVID-19." Journal of the Renin-Angiotensin-Aldosterone System 21, no. 2 (2020): 147032032092690. http://dx.doi.org/10.1177/1470320320926903.

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33

Malik, S., R. J. Winney, and D. J. Ewing. "Chronic Renal Failure and Cardiovascular Autonomic Function." Nephron 43, no. 3 (1986): 191–95. http://dx.doi.org/10.1159/000183828.

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34

Yoneyama, Kihei, Yuki Kitanaka, Osamu Tanaka, and Yoshihiro J. Akashi. "Cardiovascular magnetic resonance imaging in heart failure." Expert Review of Cardiovascular Therapy 16, no. 4 (2018): 237–48. http://dx.doi.org/10.1080/14779072.2018.1445525.

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35

Wardle, E. Nigel. "Premature cardiovascular disease and chronic renal failure." Lancet 356, no. 9232 (2000): 854–55. http://dx.doi.org/10.1016/s0140-6736(05)73432-2.

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36

Humphrey, Reed, and Matthew N. Bartels. "Exercise, cardiovascular disease, and chronic heart failure." Archives of Physical Medicine and Rehabilitation 82 (March 2001): S76—S81. http://dx.doi.org/10.1016/s0003-9993(01)70050-0.

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37

Humphrey, Reed, and Matthew N. Bartels. "Exercise, cardiovascular disease, and chronic heart failure." Archives of Physical Medicine and Rehabilitation 82, no. 3 (2001): S76—S81. http://dx.doi.org/10.1016/s0003-9993(01)80045-9.

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38

Dikow, Ralf, Martin Zeier, and Eberhard Ritz. "Pathophysiology of Cardiovascular Disease and Renal Failure." Cardiology Clinics 23, no. 3 (2005): 311–17. http://dx.doi.org/10.1016/j.ccl.2005.03.002.

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39

Baigent, Colin, Kate Burbury, and David Wheeler. "Premature cardiovascular disease in chronic renal failure." Lancet 356, no. 9224 (2000): 147–52. http://dx.doi.org/10.1016/s0140-6736(00)02456-9.

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40

Humphrey, Reed, and Matthew N. Bartels. "Exercise, cardiovascular disease, and chronic heart failure." Archives of Physical Medicine and Rehabilitation 82, no. 3B (2001): 0s76–0s81. http://dx.doi.org/10.1053/apmr.2001.22445.

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41

Mancia, Giuseppe, Gino Seravalle, Cristina Giannattasio, et al. "Reflex cardiovascular control in congestive heart failure." American Journal of Cardiology 69, no. 18 (1992): 17–23. http://dx.doi.org/10.1016/0002-9149(92)91251-x.

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42

Yorgun, Hikmet, Lale Tokgözoğlu, Uğur Canpolat, et al. "The cardiovascular effects of premature ovarian failure." International Journal of Cardiology 168, no. 1 (2013): 506–10. http://dx.doi.org/10.1016/j.ijcard.2012.09.197.

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43

Node, Koichi, and Makoto Akashi. "2. Glucose-metabolic Network and Cardiovascular Failure." Nihon Naika Gakkai Zasshi 111, no. 9 (2022): 1841–45. http://dx.doi.org/10.2169/naika.111.1841.

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44

Node, Koichi. "2. Glucose-metabolic Network and Cardiovascular Failure." Nihon Naika Gakkai Zasshi 111, Suppl (2022): 125b—126a. http://dx.doi.org/10.2169/naika.111.125b.

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45

Milazzo, Valeria, Cristina Di Stefano, Alberto Milan, et al. "Cardiovascular complications in patients with autonomic failure." Clinical Autonomic Research 25, no. 3 (2015): 133–40. http://dx.doi.org/10.1007/s10286-015-0275-0.

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46

Rehman Mir, Naeem-ur, Naeem Asghar, and Shaukat Javed. "HEART FAILURE;." Professional Medical Journal 24, no. 06 (2017): 912–18. http://dx.doi.org/10.29309/tpmj/2017.24.06.1198.

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Introduction: Atrial fibrillation (AF) and wider QRS duration have long beenidentified to worsen heart failure and LV dysfunction and increase cardiovascular morbidityand mortality. Therefore, it is necessary to identify those patients of heart failure who are atgreater risk for cardiovascular morbidity and mortality so that such subjects may be focusedfor preventive strategies. An association exists between QRS duration and AF with greaterincidences of cardiovascular events in patients of heart failure with LV systolic dysfunction.Study Design: Cross sectional survey. Setting: Department of C
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47

Lopukhov, Sergey V., and Eugene V. Filippov. "Assessment of cardiovascular risk in women with premature ovarian failure." Kazan medical journal 105, no. 3 (2024): 366–74. http://dx.doi.org/10.17816/kmj508732.

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BACKGROUND: Premature ovarian failure is a common disease that affects quality of life and cardiovascular risk in young women. AIM: To assess the relative risk of cardiovascular events in patients with primary and secondary premature ovarian failure. MATERIAL AND METHODS: The study included three groups of patients comparable in age: the first — women with primary premature ovarian failure at the age of 39.8±4.3 years; the second — women with secondary premature ovarian failure, age 40.1±4.1 years; the third was the control group at the age of 40.3±3.9 years, there were 133 people in each grou
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48

Ishigami, Junichi, Logan T. Cowan, Ryan T. Demmer, et al. "Incident Hospitalization with Major Cardiovascular Diseases and Subsequent Risk of ESKD: Implications for Cardiorenal Syndrome." Journal of the American Society of Nephrology 31, no. 2 (2020): 405–14. http://dx.doi.org/10.1681/asn.2019060574.

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BackgroundCardiorenal syndrome is a well known concept, bolstered by extensive investigations of CKD as a risk factor of cardiovascular disease. However, data on whether cardiovascular disease increases long-term risk of ESKD are sparse.MethodsWe assessed the association of incident hospitalization with major cardiovascular diseases (heart failure, atrial fibrillation, coronary heart disease, and stroke) with subsequent risk of ESKD among individuals enrolled in the Atherosclerosis Risk in Communities study; the analysis included 9047 individuals without prevalent cardiovascular disease at the
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49

Davis, Andrew M., Lisa M. Vinci, Tochi M. Okwuosa, Ayana R. Chase, and Elbert S. Huang. "Cardiovascular Health Disparities." Medical Care Research and Review 64, no. 5_suppl (2007): 29S—100S. http://dx.doi.org/10.1177/1077558707305416.

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Racial and ethnic disparities in cardiovascular health care are well documented. Promising approaches to disparity reduction are increasingly described in literature published since 1995, but reports are fragmented by risk, condition, population, and setting. The authors conducted a systematic review of clinically oriented studies in communities of color that addressed hypertension, hyperlipidemia, physical inactivity, tobacco, and two major cardiovascular conditions, coronary artery disease and heart failure. Virtually no literature specifically addressed disparity reduction. The greatest foc
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50

Steppan, Jochen, Julia Shields, and Ralph Lebron. "Pheochromocytoma Presenting as Acute Heart Failure Leading to Cardiogenic Shock and Multiorgan Failure." Case Reports in Medicine 2011 (2011): 1–4. http://dx.doi.org/10.1155/2011/596354.

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Pheochromocytoma is an endocrine tumor classically presenting with headache, paroxysmal hypertension, and palpitations. We discuss the case of a young male, presenting with acute heart failure and cardiogenic shock requiring stabilization with an intra-aortic balloon pump and a combination of ionotropes and vasopressors. Pheochromocytoma was diagnosed by CT scan, as well as urine and plasma metanephrines. After pretreatment with phenoxy-benzamine, the patient underwent adrenalectomy with subsequent cardiovascular stabilization and full recovery. Unfortunately, pheochromocytoma often remains un
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