Relevant bibliographies by topics / Cerebral glucose metabolism

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cerebral glucose metabolism'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Cerebral glucose metabolism"

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Millichap, J. Gordon. "Cerebral Glucose Metabolism and ADHD." Pediatric Neurology Briefs 4, no. 11 (1990): 83. http://dx.doi.org/10.15844/pedneurbriefs-4-11-4.

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Rosenkrantz, Ted S., Anthony F. Philipps, Isabella Knox, et al. "Regulation of Cerebral Glucose Metabolism in Normal and Polycythemic Newborn Lambs." Journal of Cerebral Blood Flow & Metabolism 12, no. 5 (1992): 856–65. http://dx.doi.org/10.1038/jcbfm.1992.117.

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In contrast to previous investigations, a recent study of polycythemic lambs suggested that cerebral glucose delivery (concentration × blood flow), not arterial glucose concentration, determined cerebral glucose uptake. In the present study, the independent effects of arterial glucose concentration and delivery on cerebral glucose uptake were examined in two groups of chronically catheterized newborn lambs (control and polycythemic). Arterial glucose concentration was varied by an infusion of insulin. CBF was reduced in one group of lambs (polycythemic) by increasing the hematocrit. At all art
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Flatt, Emmanuelle, Bernard Lanz, Yves Pilloud, et al. "Measuring Glycolytic Activity with Hyperpolarized [2H7, U-13C6] D-Glucose in the Naive Mouse Brain under Different Anesthetic Conditions." Metabolites 11, no. 7 (2021): 413. http://dx.doi.org/10.3390/metabo11070413.

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Glucose is the primary fuel for the brain; its metabolism is linked with cerebral function. Different magnetic resonance spectroscopy (MRS) techniques are available to assess glucose metabolism, providing complementary information. Our first aim was to investigate the difference between hyperpolarized 13C-glucose MRS and non-hyperpolarized 2H-glucose MRS to interrogate cerebral glycolysis. Isoflurane anesthesia is commonly employed in preclinical MRS, but it affects cerebral hemodynamics and functional connectivity. A combination of low doses of isoflurane and medetomidine is routinely used in
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Cook, Edwin H., John Metz, Bennett L. Leventhal, et al. "Fluoxetine effects on cerebral glucose metabolism." NeuroReport 5, no. 14 (1994): 1745–48. http://dx.doi.org/10.1097/00001756-199409080-00014.

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Van Bogaert, P., D. Wikler, P. Damhaut, H. B. Szliwowski, and S. Goldman. "Cerebral glucose metabolism and centrotemporal spikes." Epilepsy Research 29, no. 2 (1998): 123–27. http://dx.doi.org/10.1016/s0920-1211(97)00072-7.

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McCall, Anthony L. "Cerebral glucose metabolism in diabetes mellitus." European Journal of Pharmacology 490, no. 1-3 (2004): 147–58. http://dx.doi.org/10.1016/j.ejphar.2004.02.052.

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Theodore, W. H., G. DiChiro, R. Margolin, D. Fishbein, R. J. Porter, and R. A. Brooks. "Barbiturates reduce human cerebral glucose metabolism." Neurology 36, no. 1 (1986): 60. http://dx.doi.org/10.1212/wnl.36.1.60.

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Theodore, William H. "Antiepileptic Drugs and Cerebral Glucose Metabolism." Epilepsia 29, s2 (1988): S48—S55. http://dx.doi.org/10.1111/j.1528-1157.1988.tb05797.x.

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Kim, Y. K., J. S. Kim, S.-H. Jeong, K.-S. Park, S. E. Kim, and S.-H. Park. "Cerebral glucose metabolism in Fisher syndrome." Journal of Neurology, Neurosurgery & Psychiatry 80, no. 5 (2009): 512–17. http://dx.doi.org/10.1136/jnnp.2008.154765.

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Rosenkrantz, T. S., I. Knox, E. L. Zalneraitis, et al. "Cerebral metabolism and electrocortical activity in the chronically hyperglycemic fetal lamb." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 265, no. 6 (1993): R1262—R1269. http://dx.doi.org/10.1152/ajpregu.1993.265.6.r1262.

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Previous studies in the fetal lamb have demonstrated that hyperglycemia stimulates the fetal metabolic rate. The present study examined the effects of chronic fetal hyperglycemia on fetal cerebral metabolic rate and electrocortical activity. Nine chronically instrumented fetal lambs had measurements of cerebral blood flow and cerebral uptake/excretion of oxygen, glucose, lactate, and beta-hydroxybutyrate taken before and during a 48-h fetal glucose infusion. Electrocortical activity was also recorded. The fetal arterial glucose concentration was 19.8 +/- 2.0 mg/dl before glucose infusion and 4
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Dissertations / Theses on the topic "Cerebral glucose metabolism"

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Warnock, Geoffrey Iain. "Study of the central corticotrophin-releasing factor system using the 2-deoxyglucose method for measurement of local cerebral glucose utilisation." Thesis, University of Bath, 2007. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.492291.

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Stress is defined as a challenge to homeostatic equilibrium by physical or psychological events, generating a coping response consisting of central and peripheral changes, with the aim of exerting control over the threatening events. Corticotropin-releasing factor (CRF) is well known as a hypothalamic factor which controls the hypothalamic-pituitary-adrenocortical (HPA) axis during basal activity and stress. CRF also serves a neurotransmitter function in the brain, where it is implicated in a range of stress-related behaviours. The measurement of local cerebral glucose utilisation (LCGU) using
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Seaton, Tracy Ann. "Actions of thioctic acid on cerebral glucose utilisation and glutathione metabolism in relation to Parkinson's disease." Thesis, King's College London (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297268.

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Vasconcelos, Vívian Sarmento de. "Expressão do transportador de glicose GLUT3 e atividade da acetilcolinesterase em cérebro de ratos adultos submetidos à desnutrição no início da vida." Universidade Federal de Alagoas, 2009. http://repositorio.ufal.br/handle/riufal/632.

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The protein-energy malnutrition is one of the most serious nutritional problems that occur in developing countries. The energy and nutritional deficiency in early life can cause significant changes in different stages of development of central nervous system, since the cases of hyperplasia, hypertrophy and myelinization occur in more intense. The brain uses glucose as its main energy substrate. However, under certain physiological conditions, as during the weaning or during prolonged fasting, the cells may use other substrates in order to meet their metabolic needs. The transport of glucose to
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Katsumi, Yukinori. "Cholinergic projection from the basal forebrain and cerebral cortical glucose metabolism in rats. An animal positron emission tomography study." Kyoto University, 2000. http://hdl.handle.net/2433/180833.

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Nielsen, Henrietta M., Kewei Chen, Wendy Lee та ін. "Peripheral apoE isoform levels in cognitively normal APOE ε3/ε4 individuals are associated with regional gray matter volume and cerebral glucose metabolism". BIOMED CENTRAL LTD, 2017. http://hdl.handle.net/10150/622812.

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Background: Carriers of the APOE epsilon 4 allele are at increased risk of developing Alzheimer's disease (AD), and have been shown to have reduced cerebral metabolic rate of glucose (CMRgl) in the same brain areas frequently affected in AD. These individuals also exhibit reduced plasma levels of apolipoprotein E (apoE) attributed to a specific decrease in the apoE4 isoform as determined by quantification of individual apoE isoforms in APOE epsilon 4 heterozygotes. Whether low plasma apoE levels are associated with structural and functional brain measurements and cognitive performance remains
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Zetterling, Maria. "Clinical Studies in the Acute Phase of Subarachnoid Haemorrhage." Doctoral thesis, Uppsala universitet, Neurokirurgi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-129160.

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Patients admitted in similar clinical condition after spontaneous SAH can develop very different clinical courses. This could depend on the severity of the initial global ischemic brain injury at ictus. In the present study, we explored relations between clinical and radiological parameters at admission that indicate a more severe initial impact, and the following days hormone levels and brain metabolism. Early global cerebral oedema (GCE) on computed tomography occurred in 57 % of SAH patients and was associated with a more severe clinical condition. The brain’s glucose metabolism, measured w
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Keli, Barcelos Gleicy. "Valeur pronostique du « monitoring » du métabolisme énergétique cérébral chez les patients victimes d’une hémorragie sous-arachnoïdienne grave." Thesis, Lyon 1, 2012. http://www.theses.fr/2012LYO10300.

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Le ratio métabolique (MR) est un marqueur du métabolisme cérébral. Dans notre travail, nous avons démontré sa valeur pronostique chez 68 patients victimes d’une hémorragie sous-arachnoïdienne anévrysmale grave. En effet, une diminution du MR sous le seuil de 3,35 traduit un phénomène d’hyperglycolyse relative, dont le nombre d’événement est prédictive d’un pronostic défavorable avec une excellente sensibilité et spécificité. L’obtention de ces résultats est rendue possible, notamment après une phase de validation dans un modèle animal de procédures permettant de limiter les effets de facteurs
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Figueiras, Francisca Patuleia 1984. "Dual-tracer molecular neuroimaging : methodological improvements and biomedical applications." Doctoral thesis, Universitat Pompeu Fabra, 2012. http://hdl.handle.net/10803/84176.

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Positron emission tomography (PET) is a functional imaging method that allows studying physiological, biochemical or pharmacological processes in vivo. PET is being used in both research and clinical practice. In the brain, it has been used to investigate metabolism, receptor binding, and alterations in regional blood flow. This thesis involves both preclinical and clinical dual-tracer PET imaging studies of different neurological disorders. In this way, different radiotracers were used along the projects. The first project focused on the implementation and in vivo validation of the simultaneo
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Tholance, Yannick. "Suivi du métabolisme énergétique cérébral chez les patients victimes d'hémorragies sous-arachnoïdiennes graves : intérêt pour le pronostic individuel et le diagnostic des complications ischémiques." Thesis, Lyon 1, 2014. http://www.theses.fr/2014LYO10232/document.

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L'intérêt du suivi du métabolisme énergétique cérébral dans la prise en charge des patients victimes d'hémorragie sous-arachnoïdienne anévrismale (aSAH) grave reste actuellement controversé en raison de l'absence de valeurs seuils décisionnelles applicables en pratique. Ce travail avait pour objectif de réévaluer l'intérêt des paramètres biochimiques de trois techniques, la microdialyse intracérébrale (cMD), la mesure de la pression tissulaire cérébrale en oxygène (PbtO2) et le cathéter rétrograde jugulaire, pour prédire l’issue fonctionnelle de ces patients et diagnostiquer la survenue d'un i
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BOUJABIT, MIBARK. "Anatomie des mecanismes de l'evocation mnesique etude par analyse du metabolisme cerebral du 6-c14-glucose chez le rat." Paris 11, 1997. http://www.theses.fr/1997PA112409.

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Ce travail a pour objectif d'aborder l'etude des mecanismes de l'evocation mnesique mis en jeu lors de la presentation d'indices de rappel, apres un apprentissage partiel d'evitement actif en discrimination lumineuse, realise dans un labyrinthe en y. Pour cela nous avons etudie les modifications de l'activite metabolique du 6-c14-glucose dans 61 structures cerebrales, apres la presentation d'indices de rappel qui facilitent la performance de retention, comme l'exposition au stimulus discriminatif (sd) apres un intervalle de retention de 1 jour et au contexte experimental apres 21 jours. Les re
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Books on the topic "Cerebral glucose metabolism"

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L, Leenders K., and Paul Scherrer Institut, eds. PET pharmacokinetic course: Manual. Paul Scherrer Institut, 1997.

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Veech, Richard L., and M. Todd King. Alzheimer’s Disease. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0026.

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Deficits in cerebral glucose utilization in Alzheimer’s disease (AD) arise decades before cognitive impairment and accumulation of amyloid plaques and neurofibrillary tangles in brain. Addressing this metabolic deficit has greater potential in treating AD than targeting later disease processes – an approach that has failed consistently in the clinic. Cerebral glucose utilization requires numerous enzymes, many of which have been shown to decline in AD. Perhaps the most important is pyruvate dehydrogenase (PDH), which links glycolysis with the Krebs cycle and aerobic metabolism, and whose activ
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Xu, Kui, Joseph C. LaManna, and Michelle A. Puchowicz. Ketogenic Diet, Aging, and Neurodegeneration. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0024.

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The brain is normally completely dependent on glucose, but is capable of using ketones as an alternate energy source, as occurs with prolonged starvation or chronic feeding of a ketogenic diet. Research has shown that ketosis is neuroprotective against ischemic insults in rodents. This review focuses on investigating the mechanistic links to neuroprotection by ketosis in the aged. Recovery from stroke and other pathophysiological conditions in the aged is challenging. Cerebral metabolic rate for glucose, cerebral blood flow, and the defenses against oxidative stress are known to decline with a
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Narayana, Shalini, Babak Saboury, Andrew B. Newberg, Andrew C. Papanicolaou, and Abass Alavi. Positron Emission Tomography. Edited by Andrew C. Papanicolaou. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199764228.013.8.

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Positron emission tomography (PET) is an imaging method that utilizes compounds labeled with positron-emitting radioisotopes as molecular probes to evaluate different neurophysiological processes quantitatively and noninvasively. This chapter provides a background regarding positron emission, radiotracer chemistry, and detector and scanner instrumentation, as well as analytical methods for evaluating basic brain physiology, such as cerebral blood flow and oxygen and glucose metabolism. The methodological aspects of PET imaging, such as patient preparation and optimal scanning parameters, are d
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Gaitanis, John, Phillip L. Pearl, and Howard Goodkin. The EEG in Degenerative Disorders of the Central Nervous System. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0013.

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Nervous system alterations can occur at any stage of prenatal or postnatal development. Any of these derangements, whether environmental or genetic, will affect electrical transmission, causing electroencephalogram (EEG) alteration and possibly epilepsy. Genetic insults may be multisystemic (for example, neurocutaneous syndromes) or affect only the brain. Gene mutations account for inborn errors of metabolism, channelopathies, brain malformations, and impaired synaptogenesis. Inborn errors of metabolism cause seizures and EEG abnormalities through a variety of mechanisms, including disrupted e
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Litell, John M., and Nathan I. Shapiro. Pathophysiology of septic shock. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0297.

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The pathophysiology of sepsis is the result of a dysregulated host response to infection. Interactions between conserved pathogenic signals and host recognition systems initiate a systemic reaction to local infection. Pro- and anti-inflammatory intermediates and associated coagulatory abnormalities lead to altered macrovascular, microvascular, and mitochondrial function. Uncorrected, these processes yield similar patterns of failure in multiple organ systems. Mortality increases with successive organ failures. Although commonly thought to be a manifestation of impaired renal circulation, septi
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Klepper, Joerg, and Baerbel Leiendecker. Glut1 Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0005.

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Glut1 Deficiency (Glut1D, OMIM #606777) is caused by impaired glucose transport into the brain. The resulting cerebral “energy crisis” causes intractable seizures, developmental delay, and a complex movement disorder. The diagnosis is based on clinical features, low CSF glucose and/or mutations in the SLC2A1 gene. Paroxysmal exertion-induced dystonia (PED) and hereditary cryohydrocytosis have been described as allelic variants. Adults are increasingly being recognized through family pedigrees. The condition is effectively treatable by mimicking the metabolic state of fasting. High-fat carbohyd
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Book chapters on the topic "Cerebral glucose metabolism"

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Heiss, Wolf-Dieter. "Cerebral Glucose Metabolism." In PET and SPECT of Neurobiological Systems. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-42014-6_3.

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Heiss, Wolf-Dieter, and Olivier Zaro-Weber. "Cerebral Glucose Metabolism." In PET and SPECT of Neurobiological Systems. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-53176-8_4.

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Duarte, João M. N., and Rolf Gruetter. "Cerebral Glucose Transport and Homeostasis." In Neural Metabolism In Vivo. Springer US, 2011. http://dx.doi.org/10.1007/978-1-4614-1788-0_20.

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Hasselbalch, Steen G., and Olaf B. Paulson. "The Coupling of Cerebral Metabolic Rate of Glucose and Cerebral Blood Flow In Vivo." In Neural Metabolism In Vivo. Springer US, 2011. http://dx.doi.org/10.1007/978-1-4614-1788-0_14.

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Schuier, F. J. "Changes of Cerebral Glucose Metabolism in Movement Disorders." In Clinical efficacy of positron emission tomography. Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3345-3_8.

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Heiss, W. D., G. Pawlik, K. Herholz, R. Wagner, and K. Wienhard. "Determination of Regional Glucose Metabolism in the Brain by FDG and PET." In Cerebral Blood Flow and Metabolism Measurement. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70054-5_59.

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Reivich, M., M. Kushner, A. Alavi, and J. Greenberg. "Measurement of Local Cerebral Glucose Metabolism: Effect of Pathology and Functional Stimulation." In Cerebral Blood Flow and Metabolism Measurement. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70054-5_61.

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Cutler, Neal R., Ranjan Duara, Cheryl L. Grady, Arthur D. Kay, James V. Haxby, and Stanley I. Rapoport. "Cerebral Glucose Metabolism in Aging, Dementia, and Down Syndrome." In Child and Adolescent Psychiatry, Mental Retardation, and Geriatric Psychiatry. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4615-9367-6_47.

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Bale, Gemma, Ajay Rajaram, Matthew Kewin, et al. "Multimodal Measurements of Brain Tissue Metabolism and Perfusion in a Neonatal Model of Hypoxic-Ischaemic Injury." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-48238-1_32.

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AbstractThis is the first multimodal study of cerebral tissue metabolism and perfusion post-hypoxic-ischaemic (HI) brain injury using broadband near-infrared spectroscopy (bNIRS), diffuse correlation spectroscopy (DCS), positron emission tomography (PET) and magnetic resonance spectroscopy (MRS). In seven piglet preclinical models of neonatal HI, we measured cerebral tissue saturation (StO2), cerebral blood flow (CBF), cerebral oxygen metabolism (CMRO2), changes in the mitochondrial oxidation state of cytochrome c oxidase (oxCCO), cerebral glucose metabolism (CMRglc) and tissue biochemistry (L
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Thal, H. U., and W. J. Bock. "Clinical Interpretation of Dynamic Positron Emission Tomography with C-11-methyl-d-glucose Before and After STA-MCA Anastomosis." In Cerebral Blood Flow and Metabolism Measurement. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70054-5_63.

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Conference papers on the topic "Cerebral glucose metabolism"

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Machado, Luiza, Camila Santos, Bianca Leonardi, et al. "ACUTE PERIPHERAL INFLAMMATION IMPACT ON CEREBRAL GLUCOSE METABOLISM." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda072.

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Background: Neuroinflammation is a phenomenon already described in Alzheimer’s disease (AD). However, the effect of peripheral inflammation in AD is less understood. We recently demonstrated that severe sepsis causes acute brain metabolic disturbances. Nevertheless, whether mild acute peripheral inflammation affects brain metabolism remains unclear. Objective: We aimed at investigating the impact of mild acute peritonitis on glucose brain metabolism. Methods: Adult male wistar rats (n=6, per group) received a single intraperitoneal injection of 500 ml of carrageenan (CG, 500 µg of carrageenan
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Zhang, Jing, Kingwai Chu, Erin A. Hazlett, Monte S. Buchsbaum, and Jing Zhang. "A study of cerebral glucose metabolism and hemodynamic response in schizophrenia." In 2011 4th International Conference on Biomedical Engineering and Informatics (BMEI). IEEE, 2011. http://dx.doi.org/10.1109/bmei.2011.6098316.

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Deussing, M., J. Götzl, G. Werner, et al. "Opposite microglial phenotypes upon loss of PGRN or TREM2 result in reduced cerebral glucose metabolism." In NuklearMedizin 2019. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1683628.

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Sun, Haining, Bin Hu, Zhijun Yao, and Mike Jackson. "A PET study of discrimination of cerebral glucose metabolism in Alzheimer's disease and mild cognitive impairment." In 2013 6th International Conference on Biomedical Engineering and Informatics (BMEI). IEEE, 2013. http://dx.doi.org/10.1109/bmei.2013.6746897.

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Blazhenets, G., A. Dreßing, N. Schroeter, et al. "Altered regional cerebral glucose metabolism and its correlation with cognitive impairment in the subacute stage of COVID-19." In NuklearMedizin 2021 – digital. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1726699.

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Oliveira, Paloma Abrantes de, Diogo Abrantes de Oliveira, and Isabelle Magalhães Guedes Freitas. "Deep brain stimulation of the fornix as a therapeutic approach for Alzheimer’s disease: Systematic Review." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.168.

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INTRODUCTION: Alzheimer’s disease (AD) is a disorder characterized by cognitive impairment. The brain network in DA can be interrupted by deficiencies in glucose metabolismo. Deep brain stimulation (DBS) is used in Parkinson’s disease (PM), once it modulates motor circuits. Considering this potential, the benefits of this approach in DA must be evaluated1,2. OBJECTIVE: To investigate the potential benefit of stimulating the cerebral fornix (CF) through DBS for patients with AD. METHODS: Controlled and randomized clinical trials (ECCR), in English, performed on humans, in the last 5 years, inde
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Dai, Xiaoqian, Jie Tian, and Zhe Chen. "Improved estimation of parametric images of cerebral glucose metabolic rate from dynamic FDG-PET using volume-wise principle component analysis." In SPIE Medical Imaging, edited by Robert C. Molthen and John B. Weaver. SPIE, 2010. http://dx.doi.org/10.1117/12.843876.

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Mendes, Larissa Braga, Gabriel Pinheiro Martins de Almeida e. Souza, Marianna Pinheiro Moraes de Moraes, José Luiz Pedroso, and Orlando Graziani Povoas Barsottini. "Hypertrophic pachymeningitis associated with systemic lupus erythematosus: a case report." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.764.

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Introduction: Hypertrophic pachymeningitis is a chronic inflammatory process that results in focal or diffuse thickening of the dura mater. Nowadays, it’s an uncommon cause of neurological symptoms and has been mainly associated with infections, inflammatory diseases and carcinomatosis. This is a report of a single case based on retrospective analysis of the patient’s medical records. Case report: A 48-year-old woman was admitted in the emergency department with a two-month history of severe headaches that worsened with neck flexion and four episodes of focal to bilateral tonic-clonic seizures
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Reports on the topic "Cerebral glucose metabolism"

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Selph, Shelly S., Andrea C. Skelly, Ngoc Wasson, et al. Physical Activity and the Health of Wheelchair Users: A Systematic Review in Multiple Sclerosis, Cerebral Palsy, and Spinal Cord Injury. Agency for Healthcare Research and Quality (AHRQ), 2021. http://dx.doi.org/10.23970/ahrqepccer241.

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Objectives. Although the health benefits of physical activity are well described for the general population, less is known about the benefits and harms of physical activity in people dependent upon, partially dependent upon, or at risk for needing a wheelchair. This systematic review summarizes the evidence for physical activity in people with multiple sclerosis, cerebral palsy, and spinal cord injury regardless of current use or nonuse of a wheelchair. Data sources. We searched MEDLINE®, CINAHL®, PsycINFO®, Cochrane CENTRAL, Embase®, and Rehabilitation and Sports Medicine Source from 2008 thr
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