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Journal articles on the topic 'Chronic systolic heart failure'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Hőgye, Márta, and Tamás Forster. "Chronic heart failure with impaired left ventricular function (systolic heart failure)." Orvosi Hetilap 153, no. 51 (2012): 2021–29. http://dx.doi.org/10.1556/oh.2012.29502.

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Chronic heart failure is a common public health problem. The disease has a poor prognosis with high mortality rate and the incidence increases continuously. Prognosis of chronic systolic heart failure can be improved by several different medications as well as by special cardiac interventions based on the newly-published European and American guidelines. In case of severe systolic dysfunction, hospitalization and mortality can be reduced using angiotensin converting enzyme inhibitors, angiotensin receptor blocking drugs, beta-receptor blocking agents and aldosterone antagonists, as evidenced i

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2

Chatterjee, Kanu, and J. Eduardo Rame. "Systolic heart failure: Chronic and acute syndromes." Critical Care Medicine 36, Suppl (2008): S44—S51. http://dx.doi.org/10.1097/01.ccm.0000296267.84202.b0.

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3

Mörike, K., and J. Sindermann. "Drug Treatment for Chronic Systolic Heart Failure." Thoracic and Cardiovascular Surgeon 58, S 02 (2010): S170—S172. http://dx.doi.org/10.1055/s-0029-1240711.

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4

Geraci, Stephen A., Jessie M. Spencer, and Nancy D. Stubbs. "Office Management of Chronic Systolic Heart Failure." American Journal of Medicine 122, no. 4 (2009): 329–32. http://dx.doi.org/10.1016/j.amjmed.2008.12.011.

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5

Maack, Christoph, and Michael Böhm. "Pharmacological Treatment of Patients with Chronic Systolic Heart Failure." European Cardiology Review 9, no. 1 (2014): 43. http://dx.doi.org/10.15420/ecr.2014.9.1.43.

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Chronic heart failure is characterised by neuroendocrine activation as an attempt of the body to maintain pump function of the heart and blood pressure for the perfusion of peripheral tissues. While this neuroendocrine activation is beneficial in the short term, it induces maladaptive remodeling of the heart with continuous deterioration of left ventricular function. Accordingly, pharmacological treatment of patients with heart failure aims at protecting the heart from this neuroendocrine activation, which is represented in particular by the sympathetic nervous and the renin-angiotensin-aldost

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6

Xydonas, Sotirios, John Parissis, Louiza Lioni, et al. "Immunosenescence in patients with chronic systolic heart failure." Journal of Cardiovascular Medicine 17, no. 8 (2016): 624–30. http://dx.doi.org/10.2459/jcm.0000000000000372.

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7

Langford, Mary C. "Type 2 Diabetes and Chronic Systolic Heart Failure." Journal of Cardiovascular Nursing 19, Supplement (2004): S35—S44. http://dx.doi.org/10.1097/00005082-200411001-00005.

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8

&NA;, &NA;. "Type 2 Diabetes and Chronic Systolic Heart Failure." Journal of Cardiovascular Nursing 19, Supplement (2004): S45—S46. http://dx.doi.org/10.1097/00005082-200411001-00006.

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9

Bleske, Barry E. "Evolution and Pathophysiology of Chronic Systolic Heart Failure." Pharmacotherapy 20, no. 11 Part 2 (2000): 349S—358S. http://dx.doi.org/10.1592/phco.20.18.349s.34605.

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10

El-Armouche, Ali, Noriyuki Ouchi, Komei Tanaka, et al. "Follistatin-Like 1 in Chronic Systolic Heart Failure." Circulation: Heart Failure 4, no. 5 (2011): 621–27. http://dx.doi.org/10.1161/circheartfailure.110.960625.

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11

Fragasso, Gabriele, Giovanni Marinosci, Giliola Calori, et al. "Prognosis of mild/moderate chronic systolic heart failure." International Journal of Cardiology 145, no. 3 (2010): 584–86. http://dx.doi.org/10.1016/j.ijcard.2010.05.071.

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12

Magid, N. M., G. Opio, D. C. Wallerson, M. S. Young, and J. S. Borer. "Heart failure due to chronic experimental aortic regurgitation." American Journal of Physiology-Heart and Circulatory Physiology 267, no. 2 (1994): H556—H562. http://dx.doi.org/10.1152/ajpheart.1994.267.2.h556.

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Previously reported experimental models of aortic regurgitation generally have manifested normal systolic performance and have not developed heart failure [Magid et al. Am. J. Physiol. 263 (Heart Circ. Physiol. 32): H226–H233, 1992]. To determine whether more severe chronic experimental aortic regurgitation would generate systolic malperformance, heart failure, and emulate the human disease process, 11 New Zealand White rabbits underwent surgical induction of aortic regurgitation and 5 control animals underwent sham operation. Doppler echocardiography was performed serially for up to 3 yr, and

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13

Curtis, Stephanie L., Andrew Zambanini, Jamil Mayet, et al. "Reduced systolic wave generation and increased peripheral wave reflection in chronic heart failure." American Journal of Physiology-Heart and Circulatory Physiology 293, no. 1 (2007): H557—H562. http://dx.doi.org/10.1152/ajpheart.01095.2006.

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In human heart failure the role of wave generation by the ventricle and wave reflection by the vasculature is contentious. The aim of this study was to compare wave generation and reflection in normal subjects with patients with stable compensated heart failure. Twenty-nine normal subjects and 67 patients with heart failure (New York Heart Association class II or III) were studied by noninvasive techniques applied to the common carotid artery. Data were analyzed by wave intensity analysis to determine the nature and direction of waves during the cardiac cycle. The energy carried by an early sy

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14

Malov, Yu S., I. M. Borisov, E. P. Galova, and I. I. Yarovenko. "Diagnostic capabilities of electrocardiography systolic heart failure." Bulletin of the Russian Military Medical Academy 20, no. 3 (2018): 86–90. http://dx.doi.org/10.17816/brmma12254.

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The contractility of the myocardium is determined by the strength and speed of contraction of the ventricles. It can be estimated from the Q-T interval of the electrocardiogram, which is an electrical systole of the ventricles. Set the change in ventricular systole by the deviation of the actual interval of Q-T from the corrected one. Elongation of ventricular systole indicates a violation of myocardial contractility, which causes the development of systolic heart failure. When comparing this indicator with the results of echocardiography, reflecting the contractility of the left ventricle, th

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15

Chirinos, Julio A., and Nancy Sweitzer. "Ventricular–Arterial Coupling in Chronic Heart Failure." Cardiac Failure Review 03, no. 01 (2017): 12. http://dx.doi.org/10.15420/cfr.2017:4:2.

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Measures of interaction between the left ventricle (LV) and arterial system (ventricular–arterial coupling) are important but underrecognised cardiovascular phenotypes in heart failure. Ventriculo-arterial coupling is commonly assessed in the pressure–volume plane, using the ratio of effective arterial elastance (EA) to LV end-systolic elastance (EES) to provide information on ventricular–arterial system mechanical efficiency and performance when LV ejection fraction is abnormal. These analyses have significant limitations, such as neglecting systolic loading sequence, and are less informative

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16

Darahim, K. "Right ventricular systolic echocardiographic parameters in chronic systolic heart failure and prognosis." European Heart Journal 34, suppl 1 (2013): P659. http://dx.doi.org/10.1093/eurheartj/eht307.p659.

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17

Darahim, Khaled E. "Right ventricular systolic echocardiographic parameters in chronic systolic heart failure and prognosis." Egyptian Heart Journal 66, no. 4 (2014): 317–25. http://dx.doi.org/10.1016/j.ehj.2013.08.010.

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18

Chakraborty, Praloy, Sunil Verma, Pamila Dua, and Archana Saini. "Iron deficiency in chronic systolic heart failure(indic study)." Journal of the Practice of Cardiovascular Sciences 2, no. 2 (2016): 99. http://dx.doi.org/10.4103/2395-5414.191524.

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19

Abrahamsson, Putte, Karl Swedberg, Jeffrey S. Borer, et al. "Risk following hospitalization in stable chronic systolic heart failure." European Journal of Heart Failure 15, no. 8 (2013): 885–91. http://dx.doi.org/10.1093/eurjhf/hft032.

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20

MacDonald, Michael R., Mark C. Petrie, Nathaniel M. Hawkins, et al. "Diabetes, left ventricular systolic dysfunction, and chronic heart failure." European Heart Journal 29, no. 10 (2008): 1224–40. http://dx.doi.org/10.1093/eurheartj/ehn156.

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21

Zarudsky, A. A., I. U. Shkileva, E. V. Sheliakina, D. N. Perutsky, and E. A. Perutskaya. "HEMOGLOBIN CHANGES IN PATIENTS WITH SYSTOLIC CHRONIC HEART FAILURE." Journal of scientific articles Health and Education millennium 20, no. 1 (2018): 165–69. http://dx.doi.org/10.26787/nydha-2226-7425-2018-20-1-165-169.

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22

Hassapoyannes, Constantine A., Mary E. Bergh, M. Reza Movahed, Bruce M. Easterling, and Nowamagbe A. Omoigui. "Diastolic effects of chronic digitalization in systolic heart failure." American Heart Journal 136, no. 4 (1998): 688–95. http://dx.doi.org/10.1016/s0002-8703(98)70017-1.

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23

Volterrani, Maurizio, and Ferdinando Iellamo. "Eplerenone in chronic heart failure with depressed systolic function." International Journal of Cardiology 200 (December 2015): 12–14. http://dx.doi.org/10.1016/j.ijcard.2015.05.126.

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24

Aydogan, Mehmet, Sevket Balta, Sait Demırkol, Seyfettin Gumus, Murat Unlu, and Zekeriya Arslan. "Closest friends: Chronic pulmonary disease and systolic heart failure." International Journal of Cardiology 168, no. 3 (2013): 2965. http://dx.doi.org/10.1016/j.ijcard.2013.03.110.

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25

Hassapoyannes, C. A., B. M. Easterling, and M. R. Movahed. "Chronic digitalization and ventricular function in systolic heart failure." European Journal of Heart Failure 2 (June 2000): 58. http://dx.doi.org/10.1016/s1388-9842(00)80204-4.

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26

Kazmi Jahromi, Mitra, Fatemeh Nikparvar, Mansooreh Hoghooghi, et al. "Iron Status in Patients With Chronic Left Ventricular Systolic Failure." Disease and Diagnosis 11, no. 2 (2022): 86–90. http://dx.doi.org/10.34172/ddj.2022.16.

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Background: Heart failure is one of the most important public health problems with an increasing prevalence and identifying its contributing factors is imperative. This study was conducted to evaluate the iron status in patients with chronic systolic heart failure in Shahid Mohammadi Hospital of Bandar Abbas. Materials and Methods: A total of 80 patients with chronic left ventricular failure participated in this cross-sectional study. Data were collected using a researcher developed checklist containing demographic details and echocardiographic data (left ventricular ejection fraction, LVEF%).

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27

Dyudina, I. O., T. V. Ponomareva та L. P. Paraschenyuk. "Порівняльна характеристика даних лабораторного дослідження хворих з хронічною серцевою недостатністю ішемічного походження із систолічною дисфункцією лівого шлуночка та його збереженою фракцією викиду". Likarska sprava, № 3-4 (26 червня 2012): 47–52. http://dx.doi.org/10.31640/ls-2012-(3-4)-08.

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581 patients with chronic heart failure (CHF) of ischemic origin with systolic left ventricular dysfunction (SLVD) and preserved left ventricular ejection fraction (PLVEF) were examined. The aim of the study was to investigate the comparative aspect of laboratory examination of patients with chronic heart failure of ischemic origin with left ventricular systolic dysfunction and patients with heart failure and preserved left ventricular ejection fraction. The study found no differences in the groups on the basic parameters: the number of white blood cells, hemoglobin concentration, red blood ce

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28

Meyer, Philippe, Michel White, Marjan Mujib, et al. "Digoxin and Reduction of Heart Failure Hospitalization in Chronic Systolic and Diastolic Heart Failure." American Journal of Cardiology 102, no. 12 (2008): 1681–86. http://dx.doi.org/10.1016/j.amjcard.2008.05.068.

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29

Kumar, Arun, YL Shivamurthy, V. Mohan Kumar, et al. "2D Echocardiographic features in low T3 syndrome in chronic heart failure." Asian Journal of Medical Sciences 5, no. 3 (2014): 35–39. http://dx.doi.org/10.3126/ajms.v5i3.9522.

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Background: Thyroid abnormalities are common in chronic heart failure. Severity of heart failure rises by several fold in patients with thyroid dysfunction. Objectives: The purpose of this prospective study is to determine the correlation between low T3 syndrome and chronic heart failure with 2D echocardiography features & predicting the severity of chronic heart failure. Methods: In this descriptive, prospective cross sectional study, all patients who presented to the department of medicine with chronic heart failure during this study period of 12 months from January 2010-December 2011 in

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30

Altalhi, Hanan G. K., and Asgad A. Abdalgbar. "Accuracy of the Electrocardiogram in Identifying Chronic Heart Failure due to Left Ventricular Systolic Dysfunction." Al-Mukhtar Journal of Sciences 28, no. 1 (2013): 39–45. http://dx.doi.org/10.54172/mjsc.v28i1.143.

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There is a common assumption that a normal electrocardiography rules out chronic heart failure due to left ventricular systolic dysfunction (LVSD). In this research work, an assessment of the value of electrocardiography in identifying patients with chronic heart failure due to LVSD was investigated. Patients admitted to Al-Bayda central hospital with suspicion of heart failure for clinical assessment and electrocardiography, echocardiography; the accuracy of each screening tests were calculated for left ventricular systolic dysfunction and heart failure. A total of 120 patients' aged be

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31

Kuryata, O., A. Zabida, and O. Sirenko. "Galectin-3, advanced glycated end-products serum levels, endothelial function and cardiac hemodynamics in post infarction heart failure patients with reduced and preserved ejection fraction." Medicni perspektivi 23, no. 2 part 1 (2018): 69–74. https://doi.org/10.26641/2307-0404.2018.2(part1).129519.

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Epidemiological studies have reported that the rate of signs and symptoms of heart failure after myocardial infarction is approximately 25%. In addition, approximately 40% of myocardial infarctions are accompanied by left ventricular systolic dysfunction. Aim.. Aim of the study is to evaluate serum levels of galactin-3, AGEs and endothelial function, cardiac hemodynamics in post infarction chronic heart failure patients with different ejection fraction. Materials and methods. All patients are divided into two main groups according to ejection fraction:1<sup>st</sup> group-20 patients with

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32

A Upnitskiy, Alexander. "Pharmacotherapy of Chronic Heart Failure." Biomedical and Pharmacology Journal 11, no. 2 (2018): 993–1014. http://dx.doi.org/10.13005/bpj/1459.

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The aspects of chronic heart failure treatment in age-related risk group are described in the present article. It is established that the optimal treatment duration is 6-12 months. The authors focused on pharmacotherapeutic part only, excluding active application of invasive treatment and diagnostics methods. The analysis of long-term therapy in three parallel groups defined the role of both blood pressure control and pharmacodynamics specifics of the drugs in clinical positive dynamics of the disease development and reverse heart and vessels remodeling. Indication of spironolactone along with

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33

Katz, Stuart D., Carol Maskin, Guillaume Jondeau, Thomas Cocke, Robert Berkowitz, and Thierry LeJemtel. "Near-maximal fractional oxygen extraction by active skeletal muscle in patients with chronic heart failure." Journal of Applied Physiology 88, no. 6 (2000): 2138–42. http://dx.doi.org/10.1152/jappl.2000.88.6.2138.

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Systemic oxygen uptake and deep femoral vein oxygen content were determined at peak exercise in 53 patients with chronic heart failure with impaired systolic function (mean left ventricular ejection fraction 0.18; n = 41) or preserved systolic function (mean left ventricular ejection fraction 0.70; n = 12) and in 6 age-matched sedentary normal subjects. At peak exercise, deep femoral vein oxygen content in heart failure patients with impaired systolic function and preserved systolic function were similar, both significantly lower than that of normal subjects (2.5 ± 0.1, 2.9 ± 0.2, and 5.0 ± 0.

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34

Gurmani, Sumyia, Arij Arij, Fawad Fawad, et al. "Frequency of RV Diastolic Dysfunction in Chronic Heart Failure Patients." Pakistan Heart Journal 56, Supplement_2 (2023): S9. http://dx.doi.org/10.47144/phj.v56isupplement_2.2681.

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Objectives: In this cross-sectional study we aim to assess the frequency of RVDD in chronic heart failure patients with reduced EF who visited as outpatient in a tertiary care hospital. Methodology: We included consecutive patients with chronic heart failure who had reduced EF. We assessed these patients for RVDD if they had normal RV systolic function and pulmonary artery pressures. The RVDD was categorized as normal, impaired relaxation, pseudonormal filling and restricted filling according to ASE guideline. RVDD grades were compared with LV systolic dysfunction and diastolic dysfuncti

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35

Kalashnikova, Natalya M., Dmitriy N. Zaitsev, Anatoly V. Govorin, Natalya V. Mukha, and Marina V. Chistyakova. "Features of post-infarction chronic heart failure in patients with a new coronavirus infection COVID-19." Kazan medical journal 104, no. 1 (2023): 20–29. http://dx.doi.org/10.17816/kmj110849.

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Background. The coronavirus pandemic has caused a rapid increase in the number of cases and high deaths worldwide. A new coronavirus infection in the presence of an initial cardiac pathology can provoke decompensation of chronic heart failure. Aim. To study the clinical features of postinfarction chronic heart failure occurring against the background of a new coronavirus infection. Material and methods. The study included 80 patients with decompensated chronic heart failure. Three groups have been formed. In patients of the first group (n=40), who underwent inpatient treatment for

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36

Titova, O. N., N. A. Kuzubova, A. L. Aleksandrov, V. E. Perley, and A. Yu Gichkin. "Pulmonary and cardiac hemodynamics in COPD and cystic fibrosis by Doppler echocardiography." Russian Medical Inquiry 5, no. 7 (2021): 456–61. http://dx.doi.org/10.32364/2587-6821-2021-5-7-456-461.

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Aim: to assess the functionality of the right heart in patients with chronic obstructive pulmonary disease (COPD), mixed cystic fibrosis (CF), and cystic fibrosis lung disease by Doppler echocardiography. Patients and Methods: 30 adults with CF and 82 adults with COPD underwent ultrasonography to evaluate pulmonary and cardiac hemodynamics. All patients were divided into four groups based on the presence/absence of clinical signs of right ventricular failure and pulmonary hypertension. Results: in COPD, diastolic dysfunction of the right heart can be subclinical preceding systolic impairment.

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37

Fadeeva, M. V., A. V. Kudryavtseva, G. S. Krasnov, M. R. Skhirtladze, and V. T. Ivashkin. "Intestinal Microbiota in Patients with Chronic Heart Failure and Systolic Dysfunction." Russian Journal of Gastroenterology, Hepatology, Coloproctology 30, no. 2 (2020): 35–44. http://dx.doi.org/10.22416/1382-4376-2020-30-2-35-44.

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Aim. To study the composition of the intestinal microbiota in patients with chronic heart failure (CHF), as well as a relationship between the features of the CHF clinical course and changes in the microbial composition of the colon.Materials and methods. The study included 60 heart failure patients with systolic dysfunction diagnosed according to the results of echocardiographic examination. The control group consisted of 20 patients comparable to the main group by gender, age and underlying diseases in the absence of CHF. In all patients, the severity of CHF symptoms was assessed using a cli

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38

Shengbo, Y. "The prognostic value of atrial fibrillation in patients with chronic systolic heart failure or heart failure with preserved systolic function." Heart 97, Suppl 3 (2011): A223. http://dx.doi.org/10.1136/heartjnl-2011-300867.654.

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39

Akramova, E. G. "Echocardiographic features of congestive heart failure in patients with chronic obstructive pulmonary disease." Kazan medical journal 94, no. 2 (2013): 157–63. http://dx.doi.org/10.17816/kmj1580.

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Aim. To optimize echocardiographic diagnosis of congestive heart failure in patients with chronic obstructive pulmonary disease (COPD). Methods. Several modes of echocardiography using HD 11XE (Philips, USA) ultrasound scanner, 24-hour ECG monitoring using Microvit MT-101 (Schiller, Switzerland) 3-channel recorder and spirometry using АFD-02-«МFP» (Russia) machine were performed in 157 patients aged 39-76 years (male - 114, female - 43) who were divided into the following subgroups: (1) patients with COPD alone, (2) patients with COPD and concomitant arterial hypertension, (3) patients with CO

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40

Arfsten, Henrike, Georg Goliasch, Noemi Pavo, Hanno Ulmer, Martin Hülsmann, and Thomas Stefenelli. "Prescription Bias in the Treatment of Chronic Systolic Heart Failure." Annals of Internal Medicine 172, no. 1 (2019): 70. http://dx.doi.org/10.7326/m19-0476.

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41

Greenberg, Barry H. "A Treatment Approach for Patients With Chronic Systolic Heart Failure." Reviews in Cardiovascular Medicine 17, S1 (2016): 22–29. http://dx.doi.org/10.3909/ricm17s1s0003.

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42

Meyer, Philippe, Michel White, Marjan Mujib, et al. "Digoxin in Chronic Systolic and Diastolic Heart Failure: Similar Effects?" Journal of Cardiac Failure 14, no. 6 (2008): S79. http://dx.doi.org/10.1016/j.cardfail.2008.06.436.

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43

Ng, Austin Chin Chwan, Helen Siu Ping Wong, Andy Sze Chiang Yong, and Andrew Paul Sindone. "Impact of gender on outcomes in chronic systolic heart failure." International Journal of Cardiology 117, no. 2 (2007): 214–21. http://dx.doi.org/10.1016/j.ijcard.2006.04.079.

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44

Coats, Andrew J. Stewart. "Clinical utility of exercise training in chronic systolic heart failure." Nature Reviews Cardiology 8, no. 7 (2011): 380–92. http://dx.doi.org/10.1038/nrcardio.2011.47.

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45

Kim, Keun-Suh, Venkataramana Reddy, DK Petersen, S. Westrom, and Jun R. Chiong. "SYSTOLIC BLOOD PRESSURE AND TREATMENT OUTCOMES IN CHRONIC HEART FAILURE." Journal of the American College of Cardiology 55, no. 10 (2010): A62.E593. http://dx.doi.org/10.1016/s0735-1097(10)60594-3.

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46

Niewiński, Piotr, Dariusz Janczak, Artur Rucinski, et al. "Carotid body removal for treatment of chronic systolic heart failure." International Journal of Cardiology 168, no. 3 (2013): 2506–9. http://dx.doi.org/10.1016/j.ijcard.2013.03.011.

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47

Filippov, E. V., and K. A. Moseychuk. "Atorvastatin in patients with coronary artery disease and left ventricular systolic dysfunction." Medical Council, no. 16 (October 9, 2019): 28–33. http://dx.doi.org/10.21518/2079-701x-2019-16-28-33.

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Coronary artery disease (CAD) can manifest as a classic chest pain, or atypical angina. At the same time, the prevalence of CAD in a group of male patients with atypical angina over the age of 60 can reach 59--78%. It should be noted that the clinic manifestation of the chronic heart failure (CHF), which will be the main limiting factor, may take centre stage in diffuse coronary artery atherosclerosis. In patients with coronary artery disease and heart failure, who take atorvastatin, one should expect a decrease in the risk of adverse outcomes and hospitalizations due to heart failure. However

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48

Marinov, Nikolay, Iana Simova, and Ivo Petrov. "Acute myocarditis in a patient with parainfluenza infection, seven years after bone marrow transplantation." Bulgarian Cardiology 26, no. (2) (2020): 79–88. https://doi.org/10.3897/bgcardio.26.e52296.

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Myocarditis is an inflammatory disease of the myocardium, with heterogeneous etiology. Although rare, myocarditis can be a complication of the influenza infection. In the majority of cases, viral myocarditis is a self-limiting infection passing without permanent changes in the myocardium. We present a clinical case of a 30-year-old woman with acute heart failure in the course of respiratory infection accompanied by severe systolic dysfunction (left ventricular (LV) ejection fraction (EF) 16%). The patient was treated according to the current guideline recommendations for the treatment of acute

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49

RAUCHHAUS, M. "Diuretic use in patients with systolic chronic heart failure: a German competence network heart failure experience." European Journal of Heart Failure Supplements 7 (June 2008): 105. http://dx.doi.org/10.1016/s1567-4215(08)60290-8.

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50

Khondokar, Md Noornabi, Khurshed Ahmed, Mohammad Ashraf Hossain, et al. "Comparison between Spironolactone and Eplerenone on LV Systolic Function in Patients with Chronic Heart Failure." University Heart Journal 16, no. 2 (2020): 65–70. http://dx.doi.org/10.3329/uhj.v16i2.49649.

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Background:Chronic heart failure (CHF) is the most common and prognostically unfavorable outcome of many diseases of the cardiovascular system. Clinical trials have demonstrated mortality and morbidity benefits of mineralocorticoid receptor antagonists (MRAs) in patients with heart failure. These studies have used either eplerenone or spironolactone as the MRA. Eplerenone is a selective aldosterone antagonist expected to have a lower incidence of hormonal side effects than spironolactone. The present study is designed to compare these two drugs in chronic heart failure patients as no head to h

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