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Dissertations / Theses on the topic 'Chronically infected'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Bennett, Molly Marie Bennett. "Design, Fabrication, and Characterization of Electroceutical Bandages for Treatment of Chronically Infected Wounds." The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1462837586.

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2

Rahn, Wibke. "Molecular analysis of Helicobacter pylori-associated gastric inflammation in chronically infected and immune mice." [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=972341781.

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3

Roulston, Anne. "Regulation of NF-kB dependent cytokine gene expression in chronically HIV-1 infected myeloid cells." Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=28902.

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Myeloid cells are important reservoirs of HIV-1 infection. In response to pathogenic agents, macrophages secrete inflammatory cytokines which can stimulate viral replication and contribute to AIDS pathogenesis. Two promonocytic cell models were used to investigate the relationship between NF-$ kappa$B dependent cytokine gene expression, differentiation, and HIV infection: the U937 and PLB-985 cell lines and their chronically infected counterparts U9-IIIB and PLB-IIIB. Chronic HIV-1 infection of U9-IIIB cells did not generally lead to constitutive transcription of cytokine genes. However, when

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4

Banerjee, Camellia Tina. "Muscle ageing and anabolic response in the context of healthy and chronically HIV infected individuals." Thesis, Boston University, 2012. https://hdl.handle.net/2144/12275.

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Thesis (Ph.D.)--Boston University PLEASE NOTE: Boston University Libraries did not receive an Authorization To Manage form for this thesis or dissertation. It is therefore not openly accessible, though it may be available by request. If you are the author or principal advisor of this work and would like to request open access for it, please contact us at open-help@bu.edu. Thank you.<br>In the last thirty years of the HIV epidemic, HIV has transitioned from a deadly to a chronic, manageable disease wherein infected individuals are living longer. However, despite the efficacy of antiretroviral

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5

Desai, Annika. "Changes in Binding Properties of Helicobacter pylori Isolated over time from a Chronically Infected Patient." Thesis, Umeå universitet, Institutionen för odontologi, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-129986.

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Helicobacter pylori infects over 50 % of the world’s population, causing gastritis, and in some cases, peptic ulcer disease and gastric cancer. Adherence to the gastric surface occurs primarily through H. pylori outer membrane proteins (HOPs) and is essential for bacterial survival and establishment of infection. The Blood group Antigen-Binding Adhesin (BabA) is the best-characterized attachment protein, mediating adherence by binding to fucosylated carbohydrate structures on the surface of the gastric epithelium. H. pylori is highly adaptable to environmental changes that occur during stomach

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6

Calascibetta, F. "ANALYSIS OF VIRAL RESERVOIRS IN CHRONICALLY SIV-INFECTED SOOTY MANGABEYS TREATED WITH COMBINATION ANTIRETROVIRAL THERAPY (CART)." Doctoral thesis, Università degli Studi di Milano, 2015. http://hdl.handle.net/2434/252682.

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Background: Human Immunodeficiency Virus (HIV), the causative pathogen of Acquired Immunodeficiency Syndrome (AIDS), continues to be a major global public health issue. Although, potent antiretroviral therapy (ART) has dramatically reduced the morbidity and mortality in HIV-infected individuals, one of the most consistent obstacles to HIV eradication is the presence of stable viral reservoirs of latently infected cells. The aim of the present study is to understand the mechanisms of the complex virus-host interactions that lead to persistent infection, and to achieve the ideal combination of t

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7

Chuang, W. C. M. "Molecular and immunological study of genotype 2 Hepatitis C virus infection in chronically infected and recovered blood donors." Thesis, University of Cambridge, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.597684.

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Recovery from Hepatitis C virus (HCV) infection is considered infrequent (<20%) in Western populations but reaches 50% in West Africa where genotype 2 infection is predominant. The first objective of this project was to investigate the role of cellular response and host genetics involved in this phenomenon. Plasma samples from 104 Ghanaian blood donors reactive with anti-HCV assays were collected. 81% of subjects with chronic infection carried genotype 2 HVC. Cellular immune responses were studied in frozen peripheral blood mononuclear cell samples from 35 cases. HLA-A, -B and –DR types were d

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8

CAUCCI, Sara. "Replicative capacity and phenotypic sensitivity to antiretroviral compounds of HIV-1 strains from recently infected and chronically treated patients." Doctoral thesis, Università Politecnica delle Marche, 2020. http://hdl.handle.net/11566/273393.

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La terapia antiretrovirale purtroppo può indurre la selezione di ceppi virali resistenti ai farmaci, che possono essere trasmessi ad altre persone. In questo studio è stato messo a punto un saggio fenotipico per comprendere la dinamica evolutiva delle resistenze trasmesse, confrontando la capacità replicativa (RC), e quindi il potenziale epidemico, conferiti dai geni della proteasi (PR) e della trascrittasi inversa (RT) di ceppi virali provenienti da pazienti con infezione recente. Inoltre il saggio fenotipico è stato impiegato per studiare l'influenza del background genetico del gene dell’int

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9

Afridi, Suliman Qadir Verfasser], Ulrike [Akademischer Betreuer] [Protzer-Knolle, Markus [Gutachter] Gerhard, and Sabrina [Gutachter] Schreiner. "Genotypic and phenotypic analysis of norovirus evolution in chronically infected patients / Suliman Qadir Afridi ; Gutachter: Markus Gerhard, Sabrina Schreiner ; Betreuer: Ulrike Protzer." München : Universitätsbibliothek der TU München, 2019. http://d-nb.info/1206687312/34.

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10

TRIPODI, GIANLUCA. "Integration of hepatitis B virus DNA in chronically infected patients." Doctoral thesis, 2017. http://hdl.handle.net/11570/3104198.

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Abstract Background: Hepatitis B virus (HBV) infection is the main risk factor for hepatocellular carcinoma (HCC) worldwide. Several factors contribute to the development of cancer. Integration of HBV DNA into the human genome may activate genes that promote, or disrupt genes that normally suppress, cell proliferation, and has been found in >80% of HBV-related HCC. Some studies have however failed to confirm an association between HCC and integration at putativeoncogenic sites, but instead found similar integrations in tumourous and non-tumourous tissue. Thus, the rate of integration durin

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11

Kim, Yoona Amy. "Healthcare costs and resource utilization in treated versus untreated chronically infected hepatitis C patients." Thesis, 2014. http://hdl.handle.net/2152/26142.

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Successful treatment of chronic hepatitis C virus (HCV) leads to significant benefits in both hepatic and extrahepatic morbidity and mortality. However, treatment is costly and onerous. The purpose of this study was to evaluate the resource utilization and healthcare costs of chronic HCV patients who are treated versus those who are not treated. Patients eligible for this study were Texas Medicaid patients ≥18 and ≤63 years who had evidence of chronic HCV during the identification period (1/1/07-9/30/11) and continuous enrollment throughout the analysis period. High dimensional propensity s

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12

Baloyi, Tlangelani. "Drug resistance genotyping and phylogenetic analysis of HIV in chronically infected antiretroviral naive patients." Diss., 2018. http://hdl.handle.net/11602/1342.

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MSc (Microbiology)<br>Department of Microbiology<br>Background: Antiretroviral treatment (ART) has grown to be one of the most effective tool in the fight to control HIV/AIDS morbidity and mortality worldwide. However, due to the emergence of drug resistant HIV, ART efficacy can be jeopardized. Drug resistant HIV strain has a potential of becoming a major public threat, as its limit treatment options on people living with HIV. With several findings worldwide reporting drug resistant HIV to be currently being transmitted to ART-naïve persons, measures have been taken to genotype drug resi

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13

Alves, Tiago Nuno Ribeiro. "Immunological Characterization of Mucosal-Associated Invariant T (MAIT) Cells in chronically Hepatitis B infected patients." Master's thesis, 2019. http://hdl.handle.net/10451/39063.

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Tese de mestrado, Biologia Humana e Ambiente, Universidade de Lisboa, Faculdade de Ciências, 2019<br>A Organização Mundial de Saúde considera a doença da Hepatite B como a oitava causa de mortalidade a nível mundial e estima que aproximadamente 400 milhões de pessoas estarão infetadas nos dias de hoje. O vírus da hepatite B é caracterizado por ter um tamanho reduzido, possuir uma conformação genómica baseada em 4 “open reading frames” (ORFs), ser transmitido de forma singular e com um modo ação muito efetivo. Pacientes infetados com o vírus da hepatite B, dependendo da idade em que foram infet

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14

程新民. "The Immunological Characterization of the Epimastigote Antigens of Trypanosoma Cruzi with Chronically Infected Mouse Sera." Thesis, 1997. http://ndltd.ncl.edu.tw/handle/60146625265737702459.

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碩士<br>國立中山大學<br>生命科學研究所<br>85<br>Trypanosoma cruzi is a protozoan parasite that can cause Chagas' disease in the Central and South America. The disease was divided into two phases, acute and chronic. In the acute phase, a large number of bloodstream form (BSF) trypo-mastigotes appear in the infected host circulation. While the disease is usually asymptomatic and trypanosomes are hardly seen in the peripheral blood during the chronic phase. In this study, BALB/c mice were infected intraperitoneally with 1×106 BSF of T. cruzi. In the first week, the number of parasite was few (<1×104) in

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15

Mpanju, Onesmo. "Mechanisms of [gamma]-linolenic acid induction of apoptosis in T cells chronically infected with HIV-1." Thesis, 2001. http://hdl.handle.net/2429/13785.

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BACKGROUND: The polyunsaturated fatty acid (PUFA) gamma-linolenic acid (GLA) kills HIV-1- infected cells at concentrations that are relatively harmless to uninfected cells. The mechanism of this effect is unclear but evidence implicates oxidative stress. The aim of this study was to define mechanisms of GLA cytotoxicity in HIV-infected T cells. METHODS: The test model comprised uninfected A3.01 and HIV-1-infected 8E5 T cells. Cell proliferation was measured using WST-1 and Trypan blue dye-exclusion assays. Colorimetric analysis for lipid peroxidation, and supplementation with antioxidants and

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16

Rahn, Wibke [Verfasser]. "Molecular analysis of Helicobacter pylori-associated gastric inflammation in chronically infected and immune mice / vorgelegt von: Wibke Rahn." 2003. http://d-nb.info/972341781/34.

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