Relevant bibliographies by topics / CNS disorders; autoimmune diseases; neuro-immune diseases

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Academic literature on the topic 'CNS disorders; autoimmune diseases; neuro-immune diseases'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "CNS disorders; autoimmune diseases; neuro-immune diseases"

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Stimmer, Lev, Claire-Maëlle Fovet, and Ché Serguera. "Experimental Models of Autoimmune Demyelinating Diseases in Nonhuman Primates." Veterinary Pathology 55, no. 1 (2017): 27–41. http://dx.doi.org/10.1177/0300985817712794.

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Human idiopathic inflammatory demyelinating diseases (IIDD) are a heterogeneous group of autoimmune inflammatory and demyelinating disorders of the central nervous system (CNS). These include multiple sclerosis (MS), the most common chronic IIDD, but also rarer disorders such as acute disseminated encephalomyelitis (ADEM) and neuromyelitis optica (NMO). Great efforts have been made to understand the pathophysiology of MS, leading to the development of a few effective treatments. Nonetheless, IIDD still require a better understanding of the causes and underlying mechanisms to implement more eff
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Ren, Jingru, Jianchun Wang, Ran Liu, et al. "CSF oligoclonal bands in neurological diseases besides CNS inflammatory demyelinating disorders." BMJ Neurology Open 7, no. 1 (2025): e000912. https://doi.org/10.1136/bmjno-2024-000912.

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ObjectiveTo understand the distribution characteristics and clinical significance of cerebrospinal fluid (CSF) oligoclonal bands (OCBs) in different nervous system diseases besides typical central nervous system (CNS) inflammatory demyelinating disorders.MaterialA total of 2259 patients who underwent CSF examination for OCBs at Peking University First Hospital from January 2011 to December 2023 were tested. A cohort of 257 patients presenting with various types of OCBs but without CNS inflammatory demyelinating diseases was included in the analysis. Relevant medical history was collected from
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Restrepo-Martinez, Miguel, Vaughan Bell, and Jesus Ramirez-Bermudez. "Cognitive disorders in patients with neuroimmunological disease." Current Opinion in Psychiatry 38, no. 2 (2025): 126–33. https://doi.org/10.1097/yco.0000000000000977.

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Purpose of review Autoimmune diseases such as systemic lupus erythematosus (SLE), multiple sclerosis (MS), and autoimmune encephalitis can directly and indirectly affect brain function, leading to cognitive dysfunction or well characterized neurocognitive syndromes. However, these are often poorly characterized in the literature. Here, we review evidence on clinical manifestations, risk factors, their assessment and outcomes, and evidence for underlying mechanisms and associated biomarkers, if available. Recent findings Significant advances have been made in neurocognitive disorders associated
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Lee, Juhee, Han Sang Lee, and Kon Chu. "Successful treatment with rituximab for central nervous system vasculitis caused by Epstein-Barr virus-associated lymphoproliferative disorder with immunoglobulin M gammopathy." encephalitis 2, no. 1 (2022): 14–18. http://dx.doi.org/10.47936/encephalitis.2021.00143.

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Monoclonal gammopathy of undetermined significance (MGUS) is associated with several autoimmune conditions, including central nervous system (CNS) vasculitis. Epstein-Barr virus (EBV) is a pathogen capable of triggering a systemic immune response and is involved in the occurrence of a wide range of B-cell lymphoproliferative disorders. In systemic autoimmune diseases, EBV infection is suspected to play a central role in pathogenesis. Here, we present a case that was thought to be a systemic autoimmune disease and CNS vasculitis that developed after EBV infection, demonstrating that rituximab i
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Murdaca, Giuseppe, Francesca Paladin, Marco Casciaro, Carmelo Mario Vicario, Sebastiano Gangemi, and Gabriella Martino. "Neuro-Inflammaging and Psychopatological Distress." Biomedicines 10, no. 9 (2022): 2133. http://dx.doi.org/10.3390/biomedicines10092133.

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Inflammaging is a low degree of chronic and systemic tissue inflammation associated with aging, and is intimately linked to pro-inflammatory mediators. These substances are involved in the pathogenesis of chronic inflammatory diseases and related psychopathological symptoms. When inflammation and aging affect the brain, we use the term neuro-inflammaging. In this review, we focused on the neuro-inflammatory process typical of advanced ages and the related psychopathological symptoms, with particular attention to understanding the immune-pathogenetic mechanisms involved and the potential use of
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Huang, Bohan. "How immune system impact the nervous system." Highlights in Science, Engineering and Technology 74 (December 29, 2023): 399–404. http://dx.doi.org/10.54097/f2k6dq53.

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Immune system and nervous system are both important. Adaptive immune system and innate immune system are two categories of the immune system. Both types of system have a lot of organs that has different types of function. Both the CNS and the PNS share this characteristic. The nervous system disorders have some relationship with the immune cells being too activated. The T-cells, B-cells, macrophages, and glial cells will become to activate and accidently kill some cells in the nervous systems to cause the diseases. However, these types of disorders will have a varying cause and the scientists
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Gutman, Elisa Gouvea, Renan Amphilophio Fernandes, Jéssica Vasques Raposo-Vedovi, et al. "Molecular Mimicry between SARS-CoV-2 Proteins and Human Self-Antigens Related with Autoimmune Central Nervous System (CNS) Disorders." Microorganisms 11, no. 12 (2023): 2902. http://dx.doi.org/10.3390/microorganisms11122902.

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SARS-CoV-2 can trigger autoimmune central nervous system (CNS) diseases in genetically susceptible individuals, a mechanism poorly understood. Molecular mimicry (MM) has been identified in other viral diseases as potential triggers of autoimmune CNS events. This study investigated if MM is the process through which SARS-CoV-2 induces the breakdown of immune tolerance. The frequency of autoimmune CNS disorders was evaluated in a prospective cohort with patients admitted to the COVID-19 Intense Care Unity (ICU) in Rio de Janeiro. Then, an in silico analysis was performed to identify the conserve
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Rubin, Daniel, Ayush Batra, Ivana Vodopivec, and Henrikas Vaitkevicius. "Autoimmune Encephalitis in Critical Care: Optimizing Immunosuppression." Seminars in Respiratory and Critical Care Medicine 38, no. 06 (2017): 807–20. http://dx.doi.org/10.1055/s-0037-1608771.

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AbstractAutoimmune diseases affecting the nervous systems are a common cause of admission to the intensive care unit (ICU). Although there exist several well-described clinical syndromes, patients more commonly present with progressive neurologic dysfunction and laboratory and radiographic evidence of central nervous system (CNS) inflammation. In the critical care setting, the urgency to intervene to prevent permanent damage to the nervous system and secondary injury from the systemic manifestations of these syndromes often conflicts with diagnostic uncertainty. Furthermore, treatment is limit
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Poppell, Michael, Grace Hammel, and Yi Ren. "Immune Regulatory Functions of Macrophages and Microglia in Central Nervous System Diseases." International Journal of Molecular Sciences 24, no. 6 (2023): 5925. http://dx.doi.org/10.3390/ijms24065925.

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Macrophages can be characterized as a very multifunctional cell type with a spectrum of phenotypes and functions being observed spatially and temporally in various disease states. Ample studies have now demonstrated a possible causal link between macrophage activation and the development of autoimmune disorders. How these cells may be contributing to the adaptive immune response and potentially perpetuating the progression of neurodegenerative diseases and neural injuries is not fully understood. Within this review, we hope to illustrate the role that macrophages and microglia play as initiato
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Montarolo, Francesca, Serena Martire, Simona Perga, and Antonio Bertolotto. "NURR1 Impairment in Multiple Sclerosis." International Journal of Molecular Sciences 20, no. 19 (2019): 4858. http://dx.doi.org/10.3390/ijms20194858.

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The transcription factor NURR1 is a constitutively active orphan receptor belonging to the steroid hormone receptor class NR4A. Although a genetic association between NURR1 and autoimmune inflammatory diseases has never emerged from genome-wide association studies (GWAS), alterations in the expression of NURR1 have been observed in various autoimmune diseases. Specifically, its role in autoimmune inflammatory diseases is mainly related to its capability to counteract inflammation. In fact, NURR1 exerts anti-inflammatory functions inhibiting the transcription of the molecules involved in proinf
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Books on the topic "CNS disorders; autoimmune diseases; neuro-immune diseases"

1

Haghighi, Afshin Borhani, and Bernadette Kalman. Other Proven and Putative Autoimmune Disorders of the CNS. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0094.

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Behcet’s Disease (BD) is a multiorgan disorder characterized by oral and genital ulceration, uveitis, and dermatological symptoms. BD is most prevalent in the Mediterranean countries and East Asia, but also occurs in Europe and North America. The etiology remains unknown. Evidence suggests that BD is an autoimmune disorder with complex traits. Neuro-Behcet’s Syndome (NBS) develops in about 5% to 30% of patients with BD and presents with parenchymal or nonparenchymal pathology. The course of NBS is highly variable. Treatment strategies include modulations of the immune response and tissue degen
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Book chapters on the topic "CNS disorders; autoimmune diseases; neuro-immune diseases"

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Maltsev, Dmytro. "Efficacy of rituximab in autism spectrum disorders associated with genetic deficiency of the folate cycle with signs of antineuronal autoimmunity." In IMMUNODIAGNOSTICS AND IMMUNOTHERAPY OF NEUROPSYCHIATRIC DISORDERS IN CHILDREN. TECHNOLOGY CENTER PC, 2025. https://doi.org/10.15587/978-617-8360-21-4.ch12.

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Advances in genetics, molecular biology, and immunology over the past decades have significantly changed our understanding of the etiology and pathogenesis of autism spectrum disorders (ASD) in children. One of the key advances in this direction is the elucidation of the association of genetic deficiency of the folate cycle (GDFC) with ASD, evidence for which is based on the results of at least 5 meta-analyses of randomized controlled clinical trials and a number of additional controlled trials, the data of which have not yet been properly summarized. It has been established that GDFC leads to
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Maltsev, Dmytro. "Neuroradiological signs of encephalopathy in children with autism spectrum disorders associated with genetic deficiency of the folate cycle." In IMMUNODIAGNOSTICS AND IMMUNOTHERAPY OF NEUROPSYCHIATRIC DISORDERS IN CHILDREN. TECHNOLOGY CENTER PC, 2025. https://doi.org/10.15587/978-617-8360-21-4.ch7.

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One of the important advances in psychiatry and neurology in recent years is the elucidation of the association between genetic deficiency of the folate cycle (GDFC) and autism spectrum disorders (ASD) in children. The evidence for such an association is based on the results of at least five meta-analyses of randomized controlled clinical trials and a number of additional randomized controlled trials, the results of which are still not systematized. It has been shown that GDFC leads to the development of a number of typical biochemical disorders, which cause a state of a special form of immuno
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Zalewski, Nicholas L., and Sean J. Pittock. "Paraneoplastic and Idiopathic Autoimmune Neurological Disorders Affecting the Central Nervous System and Autoimmune Dysautonomia." In Neuroimmunology. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190050801.003.0011.

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This chapter is an examination of immune-mediated central nervous system (CNS) disorders, which have increasingly been recognized as a critical disease category in the field of neurology. The chapter looks at clinical presentation, diagnostic evaluation, and treatment. The chapter also looks to the future. The field of immune-mediated neurological diseases is rapidly growing. New autoantibodies are being discovered at a rapid rate, helping unveil the mystery behind the challenging neurological presentations in many patients.
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Scott, Elizabeth, Ian Hickie, and Kathryn Lovric. "Autoimmune Disease in Patients Hospitalised with Depressive Disorders." In Psychoimmunology CNS-Immune Interactions. CRC Press, 2019. http://dx.doi.org/10.1201/9780429279164-7.

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Liu, Yawei, Xinchen Nie, and Henrik Hasseldam. "Restoring Immune Balance in Multiple Sclerosis: The Impact of Skull Immunity." In Multiple Sclerosis - Pathways, Diagnosis and Therapeutic Targets [Working Title]. IntechOpen, 2025. https://doi.org/10.5772/intechopen.1009331.

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Multiple sclerosis (MS) is a chronic autoimmune disorder characterized by immune-mediated damage to the central nervous system (CNS). Recent research highlights an emerging and intriguing area of investigation, “skull immunity,” which refers to the unique immune cells within the skull bone marrow that directly interact with the CNS. Unlike peripheral bone marrow, skull immunity is specialized in regulating CNS immune homeostasis. The skull-derived immune response, alongside the glymphatic system and meningeal immunity, appears to significantly influence neuroinflammatory conditions such as MS.
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Mondanelli, Giada, and Claudia Volpi. "Serotonin Pathway in Neuroimmune Network." In Serotonin and the CNS - New Developments in Pharmacology and Therapeutics [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96733.

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Once considered merely as a neurotransmitter, serotonin (5-HT) now enjoys a renewed reputation as an interlocutor in the dense and continuous dialogue between neuroendocrine and immune systems. In the last decades, a role has been depicted for serotonin and its derivatives as modulators of several immunological events, due to the expression of specific receptors or enzymes controlling 5-HT metabolism in diverse immune cell types. A growing body of evidence suggests that the effects of molecules belonging to the 5-HT pathways on the neuroimmune communication may be relevant in the clinical outc
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